This document summarizes key findings on abdominal CT imaging. It describes the use of intravenous contrast agents to opacify vessels and enhance organ contrast. It outlines normal anatomy and measurements of abdominal organs and vessels. Common abdominal pathologies are discussed, including hernias, masses, fluid collections, vascular diseases and peritoneal abnormalities. Artifacts, window settings, and radiographic opacities are also reviewed.
Purpose of this presentation is to educate non radiologist about basic CT anatomy of abdominal viscera and basic interpretation of very common diseases
Acute abdoment contains all traumatic and non traumatic routine workup done at radiology center along with all the causes regarding abdominal pain refrence takent from manorama berry book of radiology
Purpose of this presentation is to educate non radiologist about basic CT anatomy of abdominal viscera and basic interpretation of very common diseases
Acute abdoment contains all traumatic and non traumatic routine workup done at radiology center along with all the causes regarding abdominal pain refrence takent from manorama berry book of radiology
65 year old female presents with a 2 week history of lower abdominal pain and dysuria.
A CT Abdomen and Pelvis with oral and IV contrast was performed. What is the major pathology present in this study which would explain this patient's symptoms? What is the most likely cause?
The significant abnormality in this scan involves the bladder. There is bladder wall thickening, most marked on the lateral aspect where it measures up to 20mm. Additionally, there is significant perivesical stranding and gas within the bladder lumen and wall. The kidneys are normal in appearance. There is no evidence of diverticular disease involving the adjacent sigmoid colon.
These radiological features are consistent with anaerobic cystitis.
Incidentally, did you note the surgical staple line along the stomach wall?
68 year old male presents with a 3 day history of severe
right-sided abdominal pain radiating down into his right scrotum. He has had associated vomiting on 3 occasions and his wife reports that the bedsheets and pillow case were drenched with sweat last night.
On examination, his vitals are: 38.6 degrees, 130bpm, 100/60mmHg, 24 breaths/min, 97% sats on room air. His peripheries are warm and vasodilated. Chest is clear. Abdomen demonstrates localised peritonism in the right lower quadrant. Testes are non-tender.
His urine dipstick is negative for blood, leukocytes or nitrites. Labs demonstrate a white cell count 18 and CRP 280. Renal function is normal.
Blood cultures are collected which quickly grow Clostridium.
A CT Abdomen and Pelvis is performed with IV contrast. Review the scan and identify the primary pathology which explains the patient's presentation.
ANSWER:
There is a small right indirect inguinal hernia containing an
enlarged 9.5mm inflamed appendix with associated fat stranding and minimal fluid. This finding of acute appendicitis contained within an inguinal hernia is consistent with Amyand's hernia. There are no features of small or large bowel obstruction. There is no free fluid or gas within the abdomen. There is no definite intraabdominal lymphadenopathy.
Incidentally, there are numerous other findings in this scan, including cholelithiasis without features of cholecystitis; multiple simple liver cysts; bilateral renal cortical cysts; a large hiatal hernia; a 12mm short-axis elongated lesion in the right para-aortic region posterior to the crus of the diaphragm, which may represent a lymph node. Additionally, there is subcutaneous emphysema involving the lower abdominal wall.
Amyand's hernia is a rare form of inguinal hernia in which the vermiform appendix becomes incarcerated within the hernia. Its incidence is less than 1%. The condition is named after Claudius Amyand, an English surgeon, who is attributed with performing the first successful appendicectomy on a young boy who had appendicitis contained within an inguinal hernia.
Right iliac fossa mass is a common clinical presentation and has a range of differentials that need to be excluded.
In this presentation will discuss RIF masses in briefly.
contact me / dr.3shaq@gmail.com
Local Advanced Lung Cancer: Artificial Intelligence, Synergetics, Complex Sys...Oleg Kshivets
Overall life span (LS) was 1671.7±1721.6 days and cumulative 5YS reached 62.4%, 10 years – 50.4%, 20 years – 44.6%. 94 LCP lived more than 5 years without cancer (LS=2958.6±1723.6 days), 22 – more than 10 years (LS=5571±1841.8 days). 67 LCP died because of LC (LS=471.9±344 days). AT significantly improved 5YS (68% vs. 53.7%) (P=0.028 by log-rank test). Cox modeling displayed that 5YS of LCP significantly depended on: N0-N12, T3-4, blood cell circuit, cell ratio factors (ratio between cancer cells-CC and blood cells subpopulations), LC cell dynamics, recalcification time, heparin tolerance, prothrombin index, protein, AT, procedure type (P=0.000-0.031). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and N0-12 (rank=1), thrombocytes/CC (rank=2), segmented neutrophils/CC (3), eosinophils/CC (4), erythrocytes/CC (5), healthy cells/CC (6), lymphocytes/CC (7), stick neutrophils/CC (8), leucocytes/CC (9), monocytes/CC (10). Correct prediction of 5YS was 100% by neural networks computing (error=0.000; area under ROC curve=1.0).
Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
- Video recording of this lecture in English language: https://youtu.be/kqbnxVAZs-0
- Video recording of this lecture in Arabic language: https://youtu.be/SINlygW1Mpc
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
Follow us on: Pinterest
Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
CDSCO and Phamacovigilance {Regulatory body in India}NEHA GUPTA
The Central Drugs Standard Control Organization (CDSCO) is India's national regulatory body for pharmaceuticals and medical devices. Operating under the Directorate General of Health Services, Ministry of Health & Family Welfare, Government of India, the CDSCO is responsible for approving new drugs, conducting clinical trials, setting standards for drugs, controlling the quality of imported drugs, and coordinating the activities of State Drug Control Organizations by providing expert advice.
Pharmacovigilance, on the other hand, is the science and activities related to the detection, assessment, understanding, and prevention of adverse effects or any other drug-related problems. The primary aim of pharmacovigilance is to ensure the safety and efficacy of medicines, thereby protecting public health.
In India, pharmacovigilance activities are monitored by the Pharmacovigilance Programme of India (PvPI), which works closely with CDSCO to collect, analyze, and act upon data regarding adverse drug reactions (ADRs). Together, they play a critical role in ensuring that the benefits of drugs outweigh their risks, maintaining high standards of patient safety, and promoting the rational use of medicines.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
2. INTRAVENOUS CONTRAST AGENTS
• Opacifying blood vessels
• Increasing the CT density of vascular abdominal organs
• Improving image contrast
Delayed images
• Contrast excretion by kidneys
• Late enhancement
• Prolonged retention of IV contrast agents
Low osmolar, non-ionic, iodine-based agents
• Lower rate of adverse reactions
3. CT OF THE ABDOMEN
• Soft tissue windows
• Liver windows
• Lung windows
• Bone windows
Spleen size
• N up to 14cm
Adrenals:
• N thickness up to 1cm
• No convex margins
Kidneys
• N up to 9-13 cm
Lymph nodes:
• Retroperitoneum
• Mesentery
• Omentum
• Porta hepatis
• Pelvis
Blood vessels
• Aorta
• IVC
• Celiac axis
• SMA and IMA
• Renal arteries and veins
• Splenic vein
• SMV
• Portal vein
4. CT ARTIFACTS
Patient motion
Volume averaging
• Display the average densities within the slice thickness instead of separate
individual densities
Beam hardening
• Increase in mean energy of the x-ray beam when it passes through an object
Noise: Quantum Mottle
• NORMALLY
– Data are generated by x-ray photons striking CT detectors
– More x-ray photons, the better the data
– Fewer x-ray photons, more limited the data
• Caused by:
– Reducing slice thickness to improve resolution and decrease volume averaging effect
– Therefore reducing number of photons used to create the image
5. ANATOMY
• RIGHT SUBPHRENIC SPACE communicates around the
liver with the ANTERIOR SUBHEPATIC and
POSTERIOR SUBHEPATIC (MORISON’S POUCH)
• LEFT SUBPHRENIC SPACE communicates with LEFT
SUBHEPATIC SPACE
• Right and left subphrenic spaces are separated by
FALCIFORM LIGAMENT and do not communicate
• LESSER SAC is the isolated peritoneal compartment
between the stomach and pancreas
6. ANATOMY
• Lesser sac communicates with greater sac through
FORAMEN OF WINSLOW
• Right subphrenic and subhepatic spaces communicates
freely with pelvic peritoneal cavity by RIGHT
PARACOLIC GUTTER
• The PHRENICOCOLIC LIGAMENT prevents
communication of left subphrenic/subhepatic spaces and
left paracolic gutter
• Pelvis communicates with both sides of the abdomen
7. ANATOMY
• Mesentery extends like fan from LIGAMENT OF
TREITZ in LUQ
• Disease from above the ligament is directed to RLQ
• Disease from below the ligament is directed to pelvis
• GREATER OMENTUM double layer of peritoneum
from greater curvature of the stomach
• Ground for PERITONEAL METASTASES
9. PERITONEAL FLUID
• Serous ascites (-10 to +15 H)
• Hemoperitoneum (45H)
• Exudative ascites by pancreatitis accumulates
in lesser sac
• Peritonitis: peritoneum enhances after contrast
10. PERITONEAL FLUID
Pseudomyxoma peritonei
• Complication of
mucocele of appendix
or mucinous
cystadenocarcinoma
• Loculations cause
mass effect on liver
and adjacent bowel
Mucinous
cystadenocarcinoma:
• Filling of peritoneal
cavity by gelatinous
mucin
11. PERITONEAL CARCINOMATOSIS
• Diffuse metastatic seeding of the peritoneal cavity
Most common cancers:
• Ovarian
• Stomach
• Pancreas
• Colon
Preferential sites:
• Pouch of Douglas
• Right paracolic gutter
• Greater omentum
17. CYSTIC ABDOMINAL MASSES
Cystic lymphangioma
• Lymphocele that is congenital
• Obstruction of lymphatic channels
• Thin walled and multiloculated
• Attenuation: water to fat density
MESENTERIC CYST:
• Cystic lymphangioma of the mesentery
OMENTAL CYSTS
• Cystic lymphangiomas of the greater omentum
18. CYSTIC ABDOMINAL MASSES
Enteric duplication cysts
• Lined with GI MUCOSA
• Attached to normal bowel
Cystic teratoma
• Retroperitoneum, mesentery, omentum
• Complex cystic and solid mass
• Water and fat attenuation and calcification
19. VESSELS
Abdominal aorta
• Left side of spine
• Bifurcates at level of iliac crest
• Does not exceed 3cm
• INFERIOR VENA CAVA
• Right of aorta
Common Iliac Vessels
• Bifurcates at PELVIC BRIM
21. VESSELS
CELIAC AXIS
• Level of aortic hiatus in diaphragm
SUPERIOR MESENTERIC ARETERY
• 1 cm below celiac axis
RENAL ARTERIES
• From lateral aspect of aorta within 1 cm of SMA
INFERIOR MESENTERIC ARETERY
• Anterior branch of aorta just above the bifurcation
22. ABDOMINAL AORTIC ANEURYSM
• Circumscribed dilatation of an artery
• All three layers of arterial wall (INTIMA, MEDIA, ADVENTITIA)
• Fusiform, saccular or spherical dilatation
• Outer to outer diameter >3cm
• Risk of aneurysm increases from 5-7cm and above
• N: aorta tapers distally
• AbN: aorta fails to taper distally
• Iliac arteries are aneurysmal if diameter >1.5cm
23. ABDOMINAL AORTIC ANEURYSM
• 90% begin below renal arteries (infrarenal
abdominal aortic aneurysm)
• Inflammatory and fibrotic changes in
perianeurysmal tissue may enhance ( this is
NOT chronic leak of aneurysm)
–May obstruct ureters
24. RUPTURE ABDOMINAL AORTIC ANEURYSM
• Lethal
• Abdominal pain, hypotension, pulsatile abdominal mass
• Unenhanced CT enough to confirm dx
CT findings
• Active arterial bleeding
• Streaks and puddles of IV contrast within retroperitoneal hematoma
Iliac artery aneurysms
• >3.5cm
• HYPERATTENUATING CRESCENT SIGN
• Crescent shaped high attenuation within wall of intraluminal thrombus of abdominal aortic aneurysm
• Sign of IMPENDING RUPTURE
• Acute blood DISSECTING into outer weak wall
25. INFECTED AORTIC ANEURYSM
• Rare
• Difficult to suspect
• Highly prone to rupture
• Also called MYCOTIC RUPTURE
• Bacterial instead of fungal
26. AORTIC DISSECTION
• Tear in the lumina
• Results in dilated segment with two lumina
• Most common: thoracic aorta
• Intimal flap separating true and false lumen
• Thrombosis in false lumen
• False lumen is usually larger and contains thrombus
Beak sign
• Junction of flap with outer wall of false lumen is an
acute angle
30. Morgagni Hernia
• Through foramen of Morgagni
• Adjacent to xiphoid process
• Right side > left
• Rarer than Bochdalek
• Small
• Anterior
• Low risk of prolapse
32. BOCHDALEK HERNIA
• Congenital
• Posterior attachment of the diaphragm
• Failure of PLEUROPERITONEAL MEMBRANE
closure
• Retroperitoneal structures (kidney, spleen) may
herniate
• Left side > right
• Pulmonary hypoplasia
• BBBBB (Bochdalek, Big, Back, Baby, Bad)
33. HIATUS HERNIA
• Herniation of stomach
• Through esophageal hiatus of diaphragm
• Types:
• Sliding hiatus hernia
• Rolling (para-esophageal) hiatus hernia
34. SLIDING HIATUS HERNIA
• 95%
• Gastro-esophageal junction (GEJ) displaced
more than 1cm above the hiatus
• Normal upper limit of esophageal hiatus 15mm
• Widened esophageal hiatus 3-4cm
• Gastric fundus above the diaphragm: as a
retrocardiac mass (with air-fluid level)
37. Groin Herniation
• Direct inguinal hernia
• Indirect inguinal hernia: more common than
direct
• Femoral hernia
• Obturator hernia
38. Direct Hernia
• Abdominal viscera
• Weakness of posterior inguinal canal medial to
inferior epigastric vessels, through
Hasselbach’s triangle
• Hasselbach’s triangle:
– Base: inguinal ligament
– Lateral: inferior epigastric vessel
– Medial: lateral edge of rectus sheath
39. Direct Hernia
• Hernial sac directly protrudes through inguinal
wall
• (Indirect: arise through deep ring and enter
inguinal canal)
• Seldom extend to the scrotum
• Due to weak transversalis fascia of Hasselbach
triangle
• Risks: Chronic increase in abdominal pressure
40. Direct Hernia
• Less susceptible to strangulation (unlike
indirect hernia) due to wide neck
• Lateral crescent sign (CT): crescent of fat
41. INDIRECT INGUINAL HERNIA
• More common than direct
• Males > females: persistence of processus
vaginalis during testicular descent
• Enters inguinal canal through deep ring
• Lateral to the inferior epigastric vessels
• Passes inferomedially to emerge via superficial
ring into the scrotum
43. Femoral Hernia
• Protrusion of peritoneal sac through femoral
ring into femoral canal
• Posterior and inferior to inguinal ligament
• May contain preperitoneal fat, omentum, small
bowel etc.
• Females
44. Femoral Hernia
• Inferior to inferior epigastric vessels
• Medial to common femoral vein
• Narrow funnel-shaped neck; compress femoral
vein engorged distal collateral ligaments
• Valsalva maneuver
• Femoral vein should also dilate
45. PANTALOON HERNIA
• Dual hernia
• Romberg’s hernia
• Saddle bag hernia
• Ipsilateral concurrent direct and indirect
inguinal hernias
• Hernial sac on both sides of inferior epigastric
vessel
50. Lumbar Hernia
• Defect in lumbar muscles or posterior fascia
• Below 12th rib and above iliac crest
• Two types:
Grynfeltt-Lesshaft hernia (superior)
• Through superior lumbar triangle
• More common
Inferior lumbar hernia (Petit Hernia)
• Inferior lumbar triangle
51. Lumbar Hernia
Superior lumbar triangle
• (Triangle of Grynfeltt Lesshaft Hernia)
• Medial: quadratus lumborum
• Superior: 12th rib
• Lateral: internal oblique muscle
• Floor: transversalis fascia and transversalis
muscle
• Roof: external oblique and latissimus dorsi
53. Foramen of Winslow
• Aka epiploic foramen
• Passage between GREATER and LESSER
SAC
• Greater sac: general peritoneal space
• Lesser sac: omental bursa
54. Spigelian Hernia
• Along semilunar line
• Through transversus abdominis aponeurosis
(Spigelian fascia)
57. Richter Hernia
• 10% of strangulated hernia
• Progress more rapidly to gangrene
• Obstruction less frequent
• Antimesenteric wall of bowel has herniated
• Most common entrapped part: TERMINAL
ILEUM
60. ABDOMINAL OPACITIES
• Foreign bodies
• Retained barium or fecal material in colonic
diverticulosis
• Appendicolith
• Dystrophic calcificiations
• Calculi
61. Peritoneal Calcification
Psammoma bodies
• Cystadenocarcinoma of the ovary
• Fine sand like calcification
Pseudomyxoma peritonei
• Ring or arc like calcificaitons in pelvis
Tuberculous peritonitis
Meconium peritonitis
Peritoneal oil granuloma
Result of continuous ambulatory peritoneal dialysis
62. Psammoma Bodies
• Round calcific collections
• Dystrophic calcification
• Necrotic cells form focus for surrounding calcific
deposition
• Lammelated concentric calcified structure
• Papillary thyroid carcinoma
• Papillary serous endometrial adenocarcinoma
• Meningioma
• Mesothelioma
• Serious cystadenoCA of the overy
• adenoCA of lung
64. Pseudomyxoma Peritonei
• Ascites due to rupture of mucinous tumour
(mucinous tumor of appendix, appendiceal
mucocele)
Two types:
• Peritoneal adenomucinosis (adenoma)
• Peritoneal mucinous carcinoma (mucinous
adenoCA)
65. Pseudomyxoma Peritonei
• Loculated collections of fluid with scalloping
of abdomino-pelvic organs
• Centrally displaced bowel loops and scattered
punctate or curvilinear calcifications
• Low attenuation loculated fluid in peritoneum,
omentum and mesentery
• Echogenic peritoneal masses or ascites with
echogenic particles
71. Acute Pancreatitis
• Alcohol abuse: most common cause of chronic
pancreatitis
• Gallstone passage/impaction: most common
cause of acute pancreatitis
73. Acute Pancreatitis
Necrosis absent:
• Acute peripancreatic fluid collection (APFC)
first 4 weeks
• Pseudocyst (encapsulated fluid) after 4 weeks
Necrosis present:
• Acute necrotic collections (ANC) first 4 weeks
• Walled off necrosis (WON) after 4 weeks
74. Acute Pancreatitis
Emphysematous pancreatitis
• Secondarily infected liquefactive necrosis
• Focal or diffuse parenchymal enlargement
• Edema
• Indistinct margins d/t inflammation
• Surrounding retroperitoneal stranding
• Liquefaction: lack of parenchymal enhancement
• Infected necrosis: presence of gas
• Abscess: cicumscribed fluid colleciton
• Hemorrhage: high attenuation fluid
75. Pancreatic Pseudocyst
• Most common cystic lesion of the pancreas
• Mass effect
– Biliary obstruction
– Gastric obstruction
• Secondary infection
• Disrupted pancreatic duct
• Takes 4-6 weeks
• Communication with pancreatic duct makes it
problematic (increase recurrence)
76. Pancreatic Pseudocyst
• Hypoechoic or anechoic
• Low attenuation surround by enhancing wall
• In contrast:
• Intraparenchymal fluid collections are called acute necrotic collections
(ANC) or walled off necrosis (WON)
• Small (4-6cm) likely to resolve spontaneously
Indication for drainage:
• Infection
• Large size (>4-6cm)
• Mass effect
• Growth
77. Diaphragmatic Apertures
• Passage between thoracic and abdominal cavities
Three main:
• Aortic hiatus
• Esophageal hiatus
• Vena cava foramen
• Lesser apertures:
78. Colonic Diverticulosis
• Multiple diverticula
• Left sided abdominal pain and constipation
Almost all are FALSE DIVERTICULA
• Mucosa herniating through a DEFECT IN THE
MUSCULARIS and covered by overlying serosa
• Increased intraluminal pressure
• Colon is shortened and hypertrophied (MYOCHOSIS
COLI)
• Most common: SIGMOID and descending colon
79. Foramen of Winslow
• Aka Epiploic foramen
• Passage between greater and lesser sac
• Greater sac: General peritoneal space
• Lesser sac: Omental bursa
80. Foramen of Winslow
Borders:
• Anterior: HEPATODUODENAL LIGAMENT
– Free border of lesser omentum
– Two layers
– Contains: CBD, hepatic artery, hepatic portal vein
• Posterior: peritoneum covering the IVC
• Superior: peritoneum covering caudate lobe of the liver
• Inferior: peritoneum covering commencement of
duodenum and hepatic artery
• Left lateral: GASTROSPLENIC and LIENORENAL
ligaments
81. Colorectal Carcinoma
• Most common CA of GIT
• Adenocarcinomas arising from pre existing adonomas
(malignant transformation; multi-hit hypothesis)
• Elderly
• Younger for Rectal CA
Some risk factors:
• Colonic adenoma (neoplastic polyps)
• Inflammatory Bowel Disease
• Dysplasia of colon within flat mucosa
• Pelvic irradiation
82. Colorectal Carcinoma
• Morphologically: sessile, exophytic, circumferential
(apple core), ulcerated, desmoplastic
• Right sided mass: larger mass, distant disease, iron
deficiency anemia
• Left sided mass: present earlier with altered bowel
habits
• From the cecum to the rectum
• Recto-sigmoid 55%
• Cecum and ascending colon 22%
This presentation demonstrates the new capabilities of PowerPoint and it is best viewed in Slide Show. These slides are designed to give you great ideas for the presentations you’ll create in PowerPoint 2011!
For more sample templates, click the File menu, and then click New From Template. Under Templates, click Presentations.