As part of a class presentation, we attempted to make this to briefly explain what Torticollis meas, the Types of presentation of Torticollis, and Management strategies for a Physiotherapist for Congenital Torticollis especially.
I hope this helps. :)
The pictures and information had been taken from internet, complied to make a brief presentation for the purpose of class presentation.
I do not own any content.
Claw Hand,Definition,Causes,Types,Symptoms and ManagementDr.Md.Monsur Rahman
Dr.Md.Monsur Rahman, Bachelor of Physiotherapy (BPT), Master of Physiotherapy (MPT) in Musculoskeletal Disorders, ABC-Spine in Osteopathic Approach,
Maharishi Markandeshwar (Deemed to be University), Ambala -Haryana.
Waddling gait- definition|role of muscle|gait analysis|kinematic and spatiote...jasna ok
This powerpoint is about WADDLING GAIT,muscle that cause waddling gait , its causes, reasons for why this gait is called duck gait and pregnancy gait, gait analysis , and its physical therapy treatment
Erb’s Palsy, also known as brachial plexus palsy, occurs in the network of nerves that supply feeling and control to the shoulders and arms. Erb’s Palsy is an injury to the nerves in the neck and upper chest. The injury can result in a loss of movement and feeling in the arm, hand and fingers. This injury often occurs during childbirth if the baby's shoulders become stuck behind the mother's pubic bone and the appropriate delivery techniques are not used.
A description of the causes of sciatic nerve pain and subsequent treatment protocols using chiropractic care and acupuncture. Provided by Dr. Kirk Johnson of Johnson Chiropractic & Acupuncture P.A.
As part of a class presentation, we attempted to make this to briefly explain what Torticollis meas, the Types of presentation of Torticollis, and Management strategies for a Physiotherapist for Congenital Torticollis especially.
I hope this helps. :)
The pictures and information had been taken from internet, complied to make a brief presentation for the purpose of class presentation.
I do not own any content.
Claw Hand,Definition,Causes,Types,Symptoms and ManagementDr.Md.Monsur Rahman
Dr.Md.Monsur Rahman, Bachelor of Physiotherapy (BPT), Master of Physiotherapy (MPT) in Musculoskeletal Disorders, ABC-Spine in Osteopathic Approach,
Maharishi Markandeshwar (Deemed to be University), Ambala -Haryana.
Waddling gait- definition|role of muscle|gait analysis|kinematic and spatiote...jasna ok
This powerpoint is about WADDLING GAIT,muscle that cause waddling gait , its causes, reasons for why this gait is called duck gait and pregnancy gait, gait analysis , and its physical therapy treatment
Erb’s Palsy, also known as brachial plexus palsy, occurs in the network of nerves that supply feeling and control to the shoulders and arms. Erb’s Palsy is an injury to the nerves in the neck and upper chest. The injury can result in a loss of movement and feeling in the arm, hand and fingers. This injury often occurs during childbirth if the baby's shoulders become stuck behind the mother's pubic bone and the appropriate delivery techniques are not used.
A description of the causes of sciatic nerve pain and subsequent treatment protocols using chiropractic care and acupuncture. Provided by Dr. Kirk Johnson of Johnson Chiropractic & Acupuncture P.A.
This presentation was prepared by me, Dr. P. Chizororo, to help fellow professionals understand one of the most common malpresentations, Breech presentation. Visit my YouTube channel, Nexus Medical Media for all pre-clinical subjects
types of breech
how you can manage a woman with breech baby?
what is External cephalic version and who can do it ?
what is the risks of vaginal breech birth ?
Congenital disorders are commonly screened by pediatricians and certain disorders like club foot needs early intervention to get satisfactory results .I have tried to present common disorders in neonates for early diagnosis.
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micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
3. BRACHIAL PLEXUS
Proximal or Duchenne-Erb’s
paralysis -Injury to C5 &C6,
most common
Intermediate paralysis- Injury
to C7
Distal or Klumpke’s paralysis
- injury to C8 & T1,
extremely rare
Total brachial plexus
paralysis ( more often than
the Klumpke type)
5. Mechanism of injury
Bending or stretching of
the neck in a direction
away from the side of
injury
6. RISK FACTOR/CAUSES
NEONATAL
Large birthweight
( > 3500 g )
Low APGAR score
at 1 min, 5 min
& 10 min,
Breach fetal position
Congenital anomalies
MATERNAL
Age ( > 35 years )
Cephalo-Pelvic
Disproportion
Gestational Diabetes
Mellitus ( results in
Macrosomia )
BMI
Post date gestation
previous child with
OBPP
LABOR-RELATED
FACTORS
**Shoulder Dystocia
Increased duration of 2nd
stage of labour (>60min)
Induction of labour
-Oxytocin augment
Operative vaginal deliveries
-Vacuum extraction
-Direct compression of
fetal neck during
delivery by forceps
14. DIAGNOSING ERB’S PALSY
Erb’s palsy is diagnosed by a
thorough physical examination and
medical history. An affected baby
may hold its affected arm close to
the body with the elbow pronated.
In additional to a routine physical
examination, some doctors may
perform special imaging and
diagnostic studies such as a nerve
conduction study or magnetic
resonance imaging (MRI).
15. CLINICAL ASSESSMENT
U.E is flail & dangling
Look for other extremities
U.R: arm held in IR,add, active abd not possible,
elbow extended forearm pronated, thumb
flexed.
Complete paralysis- vasomotor impairment, pale
& marble like color
Horner’s sign
Associated # [clavicle, humerus]
16. DIFFERENTIAL DIAGNOSIS
Fracture Pseudoparalysis
Congenital Varicella of the Upper Limb
Cerebral Palsy (Monoplegia)
Intrauterine Upper-Limb Nerve Compression
by the Umbilical Cord or Amniotic Bands
Intrauterine Maladaption Palsy
18. Protective phase
Initial rest period of 7-10 days – to allow for
reduction of hemorrhage & edema around the
traumatized nerves
No ROM or other interventions are initiated
The involved UL is positioned across the
abdomen or aeroplane position.
Avoid lying on the involved limb
Positioning, splinting, kinesiotapping, gentle
massage therapy
19. CONSERVATIVE MANAGEMENT
PHYSIOTHERAPY – cornerstone of conservative mngt.
Maintain – PROM, Supple of muscle.
Improve Muscle strength
Stretch muscle groups to prevent contracture.
Facilitates normal movement patterns while inhibiting
substitutions.
Sensory Awareness
Positioning (abd, ER, F/A flexion, wrist ex.)
Splinting
Kinesiotapping
Electrical Stimulation
20. splinting
-Resting night splints –
prevent wrist & finger F
contracture
-Wrist cock-up – maintain
neutral wrist alignment
(Klumpke’s Paralysis)
-Statue of liberty splint –
prevent Add & IR
contracture
21. SPLINTING
Air splints – restraining uninvolved UE to
encourage involved UE
Aeroplane splint – Erb’s palsy
28. SURGICAL MANAGEMENT
If there is no change over the first 3 to 6 months,
doctors may suggest exploratory surgery on the nerves
to improve the potential outcome. Nerve surgery will
not restore normal function, and is usually not helpful
for older infants. Because nerves recover very slowly, it
may take several months, or even years, for nerves
repaired at the neck to reach the muscles of the lower
arm and hand. Many children with brachial plexus
injuries will continue to have some weakness in the
shoulder, arm, or hand. There may be surgical
procedures that can be performed at a later date that
might improve function
29. Towel test
Absence of biceps recovery by 3 months of age is an
indication of surgery
The infants that did not pass the towel test At 6
months also did not pass it at 9 months are the
potential candidates for surgery
Lefevre and Diament called it as hand to face test
In supine, the child face is covered with towel
Shoulder flexion, elbow flexion and extension and
finger flexion and extension are needed for the test.
He/she passes the test if he/she then removes the
towel from the face.
31. Indication for surgical correction
Surgical exploration should be done within 6
months of life
Exploration and nerve grafting or neurotization
if there is a complete plexus palsy at 3 months
or if there is a C5-C6 palsy with absence of
biceps at 3 months
Failure of recovery of elbow flexion and
shoulder abduction from the 3rd to the 6th
month of life.
32. Surgical Intervention
Neurosurgery 5-10%
OBPI
Nerve grafting
Neuroma dissection and
removal
Neurolysis
(decompression and
removal of scar tissue)
Direct end to end
anastomosis of nerve
ends
42. PROGNOSIS for Erb’s Palsy
Generally good for
spontaneous recovery,
although may be
incomplete
Depends on degree of
involvement
Majority of spontaneous
recovery by 9 months
43. BPI Neuronal Recovery
Axon regeneration 1 mm per day
4-6 months for upper arm
7-9 months for lower arm
Recovery is varied according to damage
2 years upper arm
4 years lower arm
Denervated muscle fibers survive for approximately 18
to 24 months.
44. PREVENTION
Birthing facility has a duty to be sure that their
obstetric teams have continuing education and
skill training, so that they have current
knowledge and skills to deal with these
challenges when they occur.
Mother/patients proper education.
Good advance planning by the obstetrician.
Good judgment .
Proper history taking
46. Alarmer method
Ask for help. This involves requesting the help of an
obstetrician, anesthesia and pediatrics for subsequent
resuscitation of the infant.
Leg hyperflexion (McRoberts' maneuver)
Anterior shoulder disimpaction (pressure)
Rubin maneuver/woodscrew
Manual delivery of posterior arm
Episiotomy
Roll over on all fours (GASKIN)
47. TRACTION
Many doctors use traction (pulling on baby's head) or fundal
pressure (where the nurse climbs on the bed and jumps down
onto your stomach) before anything else and these are not only
the least effective techniques, but dangerous to mother and baby.
50. McRobert’s Manuever
The McRoberts maneuver (where mom's legs are brought up as far back
toward her stomach as possible, which realigns the pubic bone and can slip
baby's shoulder out) ) should be tried first and if failing
51. Suprapubic Pressure
Suprapubic pressure (where the doctor or nurse makes a fist and pushes hard
on the baby's shoulder just above the pubic bone) can be applied.
53. Gaskin Manuever
The Gaskin Maneuver consists of having mom roll onto all fours (or assisting
if necessary). During the process, many babies become dislodged and pop
right out. If this doesn't happen, then the doctor actually has better access to
help wiggle the baby around until the shoulder releases and the rest of baby is
born (Woods or Rubin maneuver).
57. Manual Delivery of Post arm
Manual delivery of posterior arm: Insert hand into the vagina and flex the
posterior arm of the fetus, bringing it across the chest. The posterior arm is
then delivered over the perineum which allows the provider to rotate the
fetus to allow delivery of the anterior shoulder once the rotation has
disimpacted it from the pubic symphysis.