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Disorder of the dental pulp
 Inflammation is the most important disease process
affecting the dental pulp
pulpitis
 Clincal features:
a clinical diagnosis of acute pulpitis is usually made
when the patient complains of a severe throbbing
pain at times lacinating in type precipitated by hot
or cold stimuli or on lying down and which often
keeps the patient awake , the pain generally lasts for
about 10-15 mins but may be more or less
continuous.
 In contrast a clinical diagnosis of chronic pulpitis
is associated with spontaneous attacks of dull
aching pain which can last for an hour or more.
Pathway of Pulpitis
Etiology
 Dental caries is the most common cause of pulpitis.
 By other MO’s reaching the pulp via other routes.
 Traumatic injury.
Microbial
 Bacteria generally reach the pulp as a result of dental
caries.
 Inflammation of the pulp starts before the leading
organisms reach the pulp.
 Pulpitis is seen histologically when the leading
organisms in the dentine are within abuot 1mm of
the pulp in permanent or twice this distance in
deciduous teeth.
Chemical and thermal injury
Chemical and thermal injury to the pulp may occur
during restorative procedures.
 Irritant substances may be directly applied to an
exposed pulp or may diffuse through dentine after
insertion of a restorative material.
Pulp polyp
 Chronic hyperplastic pulpitis.
 In deciduous or recently erupted permanent
teeth with wide open carious cavities and a good
apical blood supply, pulpitis may be associated with
a hyperplastic response characterized by the
production of exuberant granulation tissue. The
polyp may become epithelialized by the spontaneous
grafting of oral epithelial cells present in the saliva.
 Clinically ,an ulcerated pulp polyp present as a dark
red yellow flecked (because of the fibrous
exudates) fleshy mass protruding from the pulp
chamber , which bleed readily on probing, in
contrast , an epithelialized polyp is firmer ,
pinkish white in color and does not bleed readily
 they both usually devoid of sensation on gentle
probing.
The effect of cavity preparation and restorative
material
 The speed, heat , pressure and coolants may all
irritate the pulp tissue.
 The main threat to the pulp is from frictional heat
generated during the cutting process.
 Histological changes in pulp reaction to restorative
techniques and materials are aspiration or
displacement of odontoblast or their nuclei into the
dentinal tubules and a reduction in the number of
odontoblasts.
Healing of pulp
 Animal experiments gave shown that it is possible
for pulpitis to heal if the irritant agent are removed
from the dentine.
 In some where the pulp is exposed during cavity
preparation , and following pulpotomy ,it is possible
to maintain pulp vitality by pulp-capping . Ideally
capping agent should be no irritant Should
stimulate the formation of a calcific barrier ,
and have and antibacterial action as most pulp
exposures are contaminated by saliva.
Pulp calcification
 Pulp stones are calcified bodies with an organic
matrix and occur most frequently in the coronal
pulp.
 True/false pulp stone.
 True pulp stones contain tubules and may have an
outer layer of predentine and adjacent odontoblasts.
 False pulp stones are composed of concentric layers
of calcified material with no tubular structure.
 According to their location in pulp , stones may be
described as free , adherent of interstitial when
they have become surrounded by reactionary or
secondary dentine.
 Pulp stones increase in size and number with
age and after tooth operative procedure.
Disorder of-the-dental-pulp
Disorder of-the-dental-pulp

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Disorder of-the-dental-pulp

  • 1. Disorder of the dental pulp
  • 2.  Inflammation is the most important disease process affecting the dental pulp
  • 3. pulpitis  Clincal features: a clinical diagnosis of acute pulpitis is usually made when the patient complains of a severe throbbing pain at times lacinating in type precipitated by hot or cold stimuli or on lying down and which often keeps the patient awake , the pain generally lasts for about 10-15 mins but may be more or less continuous.
  • 4.  In contrast a clinical diagnosis of chronic pulpitis is associated with spontaneous attacks of dull aching pain which can last for an hour or more.
  • 6. Etiology  Dental caries is the most common cause of pulpitis.  By other MO’s reaching the pulp via other routes.  Traumatic injury.
  • 7. Microbial  Bacteria generally reach the pulp as a result of dental caries.  Inflammation of the pulp starts before the leading organisms reach the pulp.  Pulpitis is seen histologically when the leading organisms in the dentine are within abuot 1mm of the pulp in permanent or twice this distance in deciduous teeth.
  • 8. Chemical and thermal injury Chemical and thermal injury to the pulp may occur during restorative procedures.  Irritant substances may be directly applied to an exposed pulp or may diffuse through dentine after insertion of a restorative material.
  • 9. Pulp polyp  Chronic hyperplastic pulpitis.  In deciduous or recently erupted permanent teeth with wide open carious cavities and a good apical blood supply, pulpitis may be associated with a hyperplastic response characterized by the production of exuberant granulation tissue. The polyp may become epithelialized by the spontaneous grafting of oral epithelial cells present in the saliva.
  • 10.
  • 11.  Clinically ,an ulcerated pulp polyp present as a dark red yellow flecked (because of the fibrous exudates) fleshy mass protruding from the pulp chamber , which bleed readily on probing, in contrast , an epithelialized polyp is firmer , pinkish white in color and does not bleed readily  they both usually devoid of sensation on gentle probing.
  • 12. The effect of cavity preparation and restorative material  The speed, heat , pressure and coolants may all irritate the pulp tissue.  The main threat to the pulp is from frictional heat generated during the cutting process.
  • 13.  Histological changes in pulp reaction to restorative techniques and materials are aspiration or displacement of odontoblast or their nuclei into the dentinal tubules and a reduction in the number of odontoblasts.
  • 14. Healing of pulp  Animal experiments gave shown that it is possible for pulpitis to heal if the irritant agent are removed from the dentine.
  • 15.  In some where the pulp is exposed during cavity preparation , and following pulpotomy ,it is possible to maintain pulp vitality by pulp-capping . Ideally capping agent should be no irritant Should stimulate the formation of a calcific barrier , and have and antibacterial action as most pulp exposures are contaminated by saliva.
  • 16. Pulp calcification  Pulp stones are calcified bodies with an organic matrix and occur most frequently in the coronal pulp.  True/false pulp stone.  True pulp stones contain tubules and may have an outer layer of predentine and adjacent odontoblasts.
  • 17.  False pulp stones are composed of concentric layers of calcified material with no tubular structure.  According to their location in pulp , stones may be described as free , adherent of interstitial when they have become surrounded by reactionary or secondary dentine.  Pulp stones increase in size and number with age and after tooth operative procedure.