This document discusses various developmental disturbances that can affect the oral cavity. It covers disturbances in size, number, eruption, shape, structure of enamel and dentin. Specific conditions discussed in detail include microdontia, macrodontia, anodontia, hypodontia, supernumerary teeth, impacted teeth, enamel hypoplasia, fluorosis, dentinogenesis imperfecta, dentin dysplasia, regional odontodysplasia and more. Diagrams and radiographs are provided to illustrate features of these different disturbances.
In this presentation, it describes about the periapical diseases, for dental students.
very useful for endodontic purpose.
remember it does not include the pulpal diseases.
In this presentation, it describes about the periapical diseases, for dental students.
very useful for endodontic purpose.
remember it does not include the pulpal diseases.
One of the most painful but easy-to-treat dental emergencies is a dry socket.
• Dry socket symptoms are experienced after a tooth extraction.
• This condition requires follow-up care by the doctor who performed the surgery, an oral surgeon or a dentist who is familiar with how to treat it.
For more information, contact :-
Dr Sachdeva's Dental Aesthetic And Implant Institute,
I 101, Ashok Vihar Phase 1, Delhi- 110052
Contact us at
• Phone : +919818894041,01142464041
• Our Websites:
• www.sachdevadentalcare.com
• www.dentalclinicindelhi.com
• www.dentalimplantindia.co.in
• www.dentalcoursesdelhi.com
• www.facialaestheticsdelhi.com
#drysocket #management #thirdmolarextraction #extractioncomplications
Brief notes on the inflammation of Alveolar bone that surrounds a tooth that has recently been extracted. It occurs as a complication of tooth extraction.
A detailed description about endo perio interrelationship, including introduction, development and etiology, historical aspects, definition, classification, diagnosis, differential diagnosis, management, special consideration in management,controversies prognosis, conclusion.
Tooth decay, also known as dental caries is an epidemic, microbiological contagious disease of the teeth that ends in localized dissolution and damage of the calcified structure of the teeth. ... The time factor is significant for the commencement and development of caries in teeth.
One of the most painful but easy-to-treat dental emergencies is a dry socket.
• Dry socket symptoms are experienced after a tooth extraction.
• This condition requires follow-up care by the doctor who performed the surgery, an oral surgeon or a dentist who is familiar with how to treat it.
For more information, contact :-
Dr Sachdeva's Dental Aesthetic And Implant Institute,
I 101, Ashok Vihar Phase 1, Delhi- 110052
Contact us at
• Phone : +919818894041,01142464041
• Our Websites:
• www.sachdevadentalcare.com
• www.dentalclinicindelhi.com
• www.dentalimplantindia.co.in
• www.dentalcoursesdelhi.com
• www.facialaestheticsdelhi.com
#drysocket #management #thirdmolarextraction #extractioncomplications
Brief notes on the inflammation of Alveolar bone that surrounds a tooth that has recently been extracted. It occurs as a complication of tooth extraction.
A detailed description about endo perio interrelationship, including introduction, development and etiology, historical aspects, definition, classification, diagnosis, differential diagnosis, management, special consideration in management,controversies prognosis, conclusion.
Tooth decay, also known as dental caries is an epidemic, microbiological contagious disease of the teeth that ends in localized dissolution and damage of the calcified structure of the teeth. ... The time factor is significant for the commencement and development of caries in teeth.
Management of cleft lip and palate 2. /certified fixed orthodontic courses ...Indian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy provides dental crown & Bridge,rotary endodontics,fixed orthodontics,
Dental implants courses.for details pls visit www.indiandentalacademy.com ,or call
00919248678078
Craniofacial syndromes /certified fixed orthodontic courses by Indian dental ...Indian dental academy
Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
At the end of this lecture, students should be able to:
Develop an understanding terms Cleft lip & Palate
Develop an understanding of incidence of the condition
Describe the etiology and pathogenesis
Describe classification and dental implications
Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Aesthetics is a major topic in modern dentistry. majority of patients presenting to the dental clinic today are concerned about their smile. A holistic dental care must encompass restoring function, anatomy and a confident smile.
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
micro teaching on communication m.sc nursing.pdfAnurag Sharma
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Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
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TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
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Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
2. ORAL PATHOLOGY
It is the speciality of dentistry and pathology that deals
with the nature, identification and management of
Dr. Ali Tahir
diseases affecting the oral & maxillofacial regions
It is a science that investigates the causes, processes &
effects of these diseases
The practice of oral pathology includes research,
diagnosis of disease using clinical, radiographic,
microscopic biochemical or other necessary
examinations & investigations
3. DISTURBANCES IN SIZE
Microdontia
When one or more teeth are smaller than
normal
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Generalized
True generalized: When all teeth are uniformly smaller
than normal
Cause: Pituitary Dwarfism, Down’s syndrome
Relative generalized: When mandible or maxilla are
somewhat larger (Macrognathia) but teeth are of normal
size
Localized
When one tooth is involved
Maxillary lateral incisors are the most common
5. DISTURBANCES IN SIZE
Macrodontia
When one or more teeth are larger than normal
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Generalized
True Generalized: When all teeth are uniformly larger
Relative Generalized: When maxilla/mandible is smaller
in size (Micrognathia) but teeth are of normal size
Regional
It is localized e.g; Hemifacial hypertrophy (unilateral),
segmental odontomaxillary dysplasia
Rhizomegaly :
When only roots are larger than normal
7. DISTURBANCES IN NUMBER
Anodontia
Congenital absence of all teeth
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Associated with Hereditary Ectodermal Dysplasia
Hypodontia
Congenital absence of one or more teeth
Third molars, maxillary lateral incisors are most
commonly absent teeth consecutively
9. DISTURBANCES IN NUMBER
Supernumerary Teeth
Teeth in excess of normal number
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More common in maxilla (90%)
Examples
Maxilla
Mesiodens
Paramolars
Lateral incisors
Mandible
Premolars
paramolars
Multiple supernumerary teeth are seen in Cliedocranial dysplasia
and gardner sydrome
11. DISTURBANCES IN ERUPTION
Premature Eruption
Natal Teeth:
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Erupted decidous teeth present at time of birth
Neonatal:
Deciduous teeth that erupt in first 30 days of life
Premature eruption of entire permanent dentition should
suspect the possibility of hyperthyroidism
13. DISTURBANCES IN ERUPTION
Delayed Eruption
Eruption later than the normal age of eruption
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Usually idiopathic
Or associated with systemic conditions such as
rickets, cliedocranial dysplasia, cretinism
Gingival fibromatosis
14. DISTURBANCES IN ERUPTION
Impacted teeth
Teeth with eruption that is impeded by a
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physical barrier
Causes:
Dental crowding
Supernumerary teeth
Odontogenic cysts
Odontogenic tumors (odontomas)
Most common are mandibular and maxillary
third molars followed by maxillary cuspids
15. IMPACTED TEETH
Classified according to their orientation as
Mesioangular
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Distoangular
Horizontal
Vertical
According to their stage of eruption
Completely impacted (within bone)
Partially impacted (partly in soft tissue)
16. IMPACTED TEETH
Partially impacted teeth that communicate with the oral
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cavity are more prone to pericoronitis
Complications
Root resorption of adjacent normal tooth
Infection & pain
Dentigerous cyst
External resorption of impacted tooth
18. DISTURBANCES IN ERUPTION
Eruption Sequestrum
A small spicule of calcified tissue that is extruded
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through the alveolar mucosa that overlies an
erupting molar
19. DISTURBANCES IN SHAPE
Dilaceration
A sharp bend or
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angulation involving the
root of the tooth
Causes:
Trauma during tooth
formation
Continued root
formation during a
curved or tortuous path
of eruption
20. DISTURBANCES IN SHAPE
Taurodontism
A molar with an
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elongated crown and
apically placed
furcation of roots
resulting in an enlarged
rectangular pulp
chamber.
Occurs because of late
invagination of
Hertwig’s Epithelial
Root Sheath
21. DISTURBANCES IN SHAPE
Dens Invaginatus
Developmental
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anomaly characterized
by a deep enamel lined
pit that extends for
varying depths into the
underlying dentin
displacing the pulp
chamber
22. DENS IN DENT AND SUPERNUMERARY
CUSP
Supernumerary
cusps
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Teeth containing
additional cusp
Example: Cusp of
caribili, Talon cusp
23. DISTURBANCES IN SHAPE
Dens Evaginatus
Characterized by
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cusp like
supernumerary
enamel protrusion
on occlusal or
lingual surface of
crown
24. Gemination
Single rooted tooth with
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unusual wide, partly
divided crown or two
separate crowns.
Cause: Because of
partial division of a
single tooth gem
Teeth count is normal
25. DISTURBANCES IN SHAPE
Fusion
Abnormally shaped
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tooth with wide crown
or a normal crown with
additional root(s)
Cause: Results from
the union of two tooth
germs
26. DISTURBANCES IN SHAPE
Concrescence
Union of the roots of
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two or more normal
teeth by confluence of
their cementum.
Cause: Trauma, Inter-
septal bone loss
27. DISTURBANCES IN SHAPE
Hypercementosis
One or more teeth with excessive deposition of
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cementum of roots
Causes: Increase/Decreased occlusal forces,
Paget’s disease, hyperpituitarism, chronic
inflammation
28. Cervical Enamel
Projections
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Apical extension of
coronal enamel beyond
the smooth cervical
margin
29. Hemispheric structures
that may consist
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entirely of enamel or
may contain underlying
dentin & pulp
Mostly present in roots
of maxillary or
mandibular molars
30. DISTURBANCES IN STRUCTURE OF ENAMEL
Acquired
Environmental factors
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Bacterial (syphilis), viral infections
Inflammation
Nutritional deficiencies
Chemical injuries
Trauma
Genetic
Amelogenesis Imperfecta
31. ACQUIRED DISTURBANCES
Focal enamel hypoplasia
Localized enamel hypoplasia involving one or two
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teeth
Example: Turner tooth, results from localized
inflammation or trauma during tooth development
Enamel has pitting areas or deformed with
yellowish or brownish discoloration
32. ACQUIRED DISTURBANCES
Generalized Enamel Hypoplasia
Systemic or Environmental factors inhibit
functioning ameloblasts.
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Enamel has horizontal lines of small pits or
grooves
Example:
Hutchinson’s incisors and mulberry molars due to
congenital syphilis
Neonatal line
Flourosis
Can also be seen in hypocalcemia (Vit. D
deficiency), measles, chicken pox, scarlet fever, Vit
A & C deficiency
34. ACQUIRED DISTURBANCES
Flourosis (Flouride mottling)
Minimal Flourosis:
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Smooth enamel surface with white flecks
Mild Flourosis:
Smooth enamel surface with white opaque areas
Moderate to severe:
Pitting and brownish discoloration
Severe:
Enamel is softer and weaker than normal, resulting
in excessive wear
Teeth are largely resistant to caries
36. HEREDITARY DISTURBANCES
Amelogenesis Imperfecta
A heterogeneous group of genetic disorders
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exhibiting faulty enamel formation (affects both
primary & permanent dentition)
Normal enamel formation:
1. Enamel matrix formation
2. Mineralization of enamel
3. Enamel maturation (secondary mineralization)
Accordingly three types of AI is identified
37. TYPES
Clinical Features:
Hypoplastic (focal or generalized)
Decreased enamel formation by disturbance in function of
ameloblasts
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Enamel is thinner than normal
Radiodensity is greater than that of dentin
Hypocalcified
Defect in mineralization of enamel
Enamel is off normal thickness but softer than normal
Can be easily removed with a blunt instrument
Radiodensity is lesser than that of dentin
Haypomaturation
Focal or generalized areas of immature enamel
crystallites
Enamel of normal thickness, but less harder and is
radiolucent e.g. snow capped teeth
40. DISTURBANCES IN STRUCTURE OF DENTIN
Acquired
Turner tooth
Regional odontodysplasia
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Genetic
Dentinogenesis Imperfecta
Type I
Type II
Type III
Dentin Dysplasia
Type I
Type II
Familial hypophosphatemia (Vit. D resistant rickets)
41. DENTINOGENESIS IMPERFECTA
A hereditary (autosomal dominant) defect
consisting of opalescent teeth composed of
irregularly formed & undermineralized dentin
that obliterates the pulp chambers & canals
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Types:
Type 1
Associated with osteogenesis imperfecta.
Characterized by bluish tint to sclera
Type 2
Hereditary opalescent dentin
Most common type
Type 3
Also called brandywine type
Clinically the same as type 1 & II
Multiple pulpal exposures of decidous dentition
42. DENTINOGENESIS IMPERFECTA
Clinical Features
Both dentitions are affected
Oplaescent teeth with bluish grey to brownish
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or yellowish discoloration
Abnormally soft dentin, enamel easily chipped
off
Despite the exposure of dentin, teeth are not
prone to caries
Histopathology
The mantle dentin is normal whereas remaining
dentin is severely dysplastic
Amorphous matrix with globular & inter-globular
areas of mineralization
Irregularly widely spaced disoriented dentinal
tubules
44. RADIOGRAPHICALLY
Type I & II
Bulb shaped crowns
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Constricted cemento-enamel junction
Thin roots
Varying degrees of obliteration of pulp chamber &
canals
Type III
Same features or may show extremely large pulp
chambers surrounded by thin shell of dentin
45. DENTIN DYSPLASIA
A hereditary defect in dentin formation where root
dentin is abnormal & gnarled, roots are
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shortened & tapered
Also called Rootless teeth
Autosomal dominant
Two types
Type I (radicular)
Type II (coronal)
46. DENTIN DYSPLASIA
Types:
1) Radicular Dentin Dysplaisa:
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Brownish or bluish translucency in cervical region
More common than type II
All teeth in both dentitions are affected
Teeth often show increased mobility & exfoliate prematurely
Histopathology:
Enamel and mantle dentin is normal
Remaining coronal and root dentin consists of nodular
masses composed of tubular dentin and osteo-dentin
Slit-like pulp remnants may be seen between nodular masses
Gives an appearance of ‘lava flowing around boulders’
48. RADIOGRAPHICAL FEATURES
Short blunt roots, may
be absent entirely
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Mandibular molars
have W-shaped roots
Dentition may show
obliteration of pulp
chambers & canals
Crescent shaped
remnants of pulp may
be seen
50. DENTIN DYSPLASIA TYPE II (CORONAL)
Both dentitions affected
Deciduous teeth with bluish-grey, brownish or
yellowish discoloration
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Same translucent, opalescent appearance as in DI
Permanent teeth have normal clinical appearance
Histopathology:
Deciduous teeth
Normal zone of mantle dentin that changes abruptly into
dense amorphous dentin with irregular arrangement of
tubules.
Permanent teeth
Globular and inter-globular areas of dentin in pulpal third
of dentin with atubular root dentin
Has pulp stones in pulp chamber
Narrow pulp canals
51. RADIOGRAPHIC FEATURES
Deciduous teeth show
obliteration of pulp
chambers & canals as
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seen in DD type I & DI
Roots are normal
Pulp chambers of
permanent teeth are
enlarged
Thistle-tube or flame
shaped pulp chamber
Pulpal calcifications in
coronal pulp chamber
52. REGIONAL ODONTODYSPLASIA (GHOST
TEETH)
Defective formation of enamel and dentin with
abnormal pulp and follicle calcifications with
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surrounding soft tissue hyperplaisa along-
with accumulations of spherical
calcifications and odontogenic rests
53. CLINICAL FEATURES
More common in maxilla
Affects several adjacent teeth in the same quadrant
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Mostly in permanent dentition
Deformed teeth with soft, leathery surface
Discolored, yellowish brown
Histopathology
Dysplastic, globular and interglobular dentin
Widened predentin layer
Enlarged pulp chamber with pulp stones
54. RADIOGRAPHIC FEATURES
Ghost teeth
Decreased
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radiodensity
Enamel & dentin are
very thin
Large pulp chambers
Pulp stones
56. DISTURBANCES IN STRUCTURE OF
CEMENTUM
Hypophosphatasia
Disorder of bone mineralization caused by
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deficiency in alkaline phosphatase in serum and
tissues
Autosomal recessive/dominant
Delayed formation and eruption of dentition
Premature loss of primary teeth
Spontaneous loss of permanent teeth
Radiographically
Enlarged pulp chambers & pulp canals
Histopathology
Absence or marked reduction of cementum