Dermatitis is an inflammation of the skin that causes redness, swelling and itchiness. There are many types of dermatitis including atopic dermatitis, contact dermatitis and seborrheic dermatitis. Contact dermatitis occurs when the skin comes into contact with an irritant or allergen and can be either irritant or allergic in nature. Symptoms vary depending on the type but may include a rash, blisters, dry cracked skin and itchiness. Treatment involves identifying and avoiding triggers, using moisturizers and topical or oral medications like corticosteroids and antihistamines.
Dermatitis, also known as eczema, is a group of diseases that results in inflammation of the skin. These diseases are characterized by itchiness, red skin and a rash. In cases of short duration, there may be small blisters
Dermatitis, also known as eczema, is a group of diseases that results in inflammation of the skin. These diseases are characterized by itchiness, red skin and a rash. In cases of short duration, there may be small blisters
Eczema (Atopic Dermatitis): Symptoms, Causes, Types, and TreatmentEczema Less
Atopic dermatitis, commonly referred to as eczema, is a chronic inflammatory skin condition that affects millions of people worldwide. It is characterized by red, itchy rashes and can vary in severity from mild discomfort to significant impairment of daily life.
What is eczema?
Eczema (Dermatitis)-
A particular type of inflammatory reaction of the skin in which there is erythema (reddening), edema (swelling), papules (bumps), and crusting of the skin followed, finally, by lichenification (thickening) and scaling of the skin.
Eczema characteristically causes itching and burning of the skin.
What Causes eczema?
Allergy- One of the commonest cause of Eczema. Triggers include Dust, detergents, rubber, nickel plated jewelry etc.
Environment- More likely in urban areas due to high pollution levels. Extremely dry or cold weather tends to make skin scratchy, resulting in eczema.
Obesity- Obese children are 3 times more likely to get eczema. Obesity results in inflammation of fat tissues that spills into other parts of the body. Excess fat also results in poor circulation and skin ailments.
Smoking- One of the leading causes, especially on the fingers that hold the cigarettes, as well as lips.
Stress- Physical or emotional stress has been known to cause enhanced sensitivity and inflammatory skin changes.
Diaper rash- In babies eczema occurs because of chemical effect of urine/faeces on sensitive skin.
Genetic influence- More likely in individuals with a family history of Eczema or other allergic conditions like Asthma, Hay fever, etc.
eczema, is inflammation of the skin. It is characterized by itchy, erythematous, vesicular, weeping, and crusting patches. The term eczema is also commonly used to describe atopic dermatitis
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
2. INTRODUCTION
Dermatitis is an inflammation of the skin and which are
commonly swollen, reddened and irritatingly itchy. Although not
an alarming condition, this type of skin diseases can makeyou very
uncomfortable, unease and self-conscious.
Dermatitis is an itchy inflammation of the skin. It is not contagious
or dangerous, but it can be uncomfortable. There are many types of
dermatitis, including allergic dermatitis, eczema, and seborrheic
dermatitis (which causesdandruff).
A rash is an abnormal condition and reaction of the skin.
3. Dermatitis, also known as is eczema,inflammation of the skin. It is
characterized by itchy, erythematous, vesicular, weeping and crusting
patches. The term eczema is also commonly used to describe atopic
dermatitis, also known as atopic eczema.
The cause of dermatitis in unclear. One possibility is a dysfunctional
interplay between the immune system and skin.
Dermatitis is the inflammation of the skin characterised by erythema and
pain is called dermatitis.
DEFINITION
4. DEFINITION : -
ACCORDING TO BRUNNER AND SUDDHART -
Dermatitis is inflammation of the upper layers of theskin, causing itching,
blisters, redness, swelling, and often oozing, scabbing, and scaling.
ACCORDING TO LEWIS–
Dermatitis isan inflammation of the skin and which arecommonly swollen,
reddened and irritatinglyitchy.
ACCORDING TO LIPPINCOTT -
A medical condition in which the skin becomes red, swollen, and sore,
sometimes with small blisters, resulting from direct irritationof theskin byan
external agentoran allergicreaction to it.
5. Itaffects males and females and accounts for 10 to 20 percent of all visits to dermatologists (doctors who specialize in
the care and treatment of skin diseases). Although atopic dermatitis may occur at any age, it most often begins in
infancy and childhood. Women tends to develop thediseaseatanearlierage (20 to 40 years of age ) compared to men ( 60
to 70 years of age ), and womenareaffected more frequently.
INCIDENCE
6. STAGES OF DERMATITIS -
Acute dermatitis-
Acute dermatitis is characterised by erythema,
vesiculation and oozing, often with oedema.
8. CHRONIC DERMATITIS -
Chronic dermatitis is characterised by thickened dry
patches, often lichenified from chronicrubbing
(increased Skin markings).
Lichenification is often predominantly follicular in
pigmented skin.
11. 1.CONTACT DERMATITIS
Contact dermatitis is skin inflammation caused by
direct contact with a particular substance. The rash is
very itchy, is confined to a specific area, and often has
clearly defined boundaries.
14. IRRITANT CONTACT DERMATITIS : -
Irritant contact dermatitis,which accounts for 80% of
all cases of contact dermatitis, occurs when a chemical
substance causes direct damage to the skin; symptoms
are more painful than itchy. Typical irritating
substances are acids, alkalis (such as drain cleaners),
solvents (such as acetone in nail polish remover),
strong soaps, and plants (such as poinsettias and
peppers).
15. ALLERGIC CONTACT DERMATITIS
Allergic contact dermatitis is a reaction bythe body's system to a
immune substance contacting the skin. Sometimes a person can be
sensitized by Only one exposure, and other times sensitization occurs
only after many exposures to a substance. After a person is sensitized,
the next exposure causes itching and dermatitis within 4 to 24
hours.
16. ATOPIC DERMATITIS
Atopic dermatitis is chronic, itchy inflammation of the upper layers of the skin
that oftendevelops in peoplewho have hay fever or asthma and in people who
have family members with theseconditions.
Infants may develop red, oozing, crusted rashes on the face, scalp, diaper area,
hands, arms, feet, or legs. Infants may developred, oozing, crusted rashes on
the face, scalp, diaper area, hands, arms, feet, orlegs.
Treatment
The scalp can be treated with a shampoo containing
pyrithione zinc, selenium sulfide , an Antifungal drug,
salicylicacid and sulfur, ortar.
18. NUMMULAR DERMATITIS
Nummular dermatitis is a persistent, usually itchy, rash and
inflammation characterized by coin- shaped spots, often with tiny
blisters, scabs, and scales.
Most people benefit from skin moisturizers. Other treatments
include antibiotics taken by mouth, corticosteroid creams and
injections, and phototherapy.
Most people benefit from skin moisturizers. Other treatments
Include antibiotics taken bymouth, corticosteroid creams and
injections, and phototherapy.
20. 2.SEBORRHOEIC DERMATITIS
Seborrhoeic dermatitis (also known as "seborrheiceczema") is an inflammatoryskin
disorder affecting the scalp, face, and trunk. seborrheic dermatitis presents withscaly,
flaky, itchy, red skin. The condition's symptoms appeargraduallyand usually the first
signs of seborrheic dermatitis arethe flakes of skin called dandruff.
TREATMENT :
Dermatologist recommend topical treatments suchas shampoos, cleansers or
creams/lotionsthat contain antifungal , antiinf lammatory, sebosuppresive or keratolytic
ingredients.
22. STASIS DERMATITIS
Stasis dermatitis is inflammation on the lower legs from pooling of blood and
fluid. have varicose (dilated, twisted) veins and swelling (edema). It usually
occurs on the ankles but may spread upward to the knees.
have varicose (dilated, twisted) veins and swelling (edema). It usually occurs on
the ankles but may spread upward to the knees.
Treatment :
Long-term treatment is aimed at keepingblood from pooling in the veins around
the ankles. When sitting, the person should elevatethe legs above the level of
the heart. Antibiotics are used onlywhen the skin is already infected
24. PERIORAL DERMATITIS
Perioral dermatitis is a red,bumpy rash around the mouth
and on the chin that resembles acne or rosacea Perioral
dermatitis is distinguished from acne by the lack of
blackheads and whiteheads Treatment is with tetracyclines
or other antibiotics taken by mouth.
26. 3. GENERALIZED EXFOLIATIVE DERMATITIS
Generalized exfoliative dermatitis (erythroderma) issevere inflammation that
causes the entire skin surface to become red, cracked, and covered with scales.
Treatment –
People with severe exfoliative dermatitis
often need to be hospitalized and given
antibiotics (for infection), intravenous fluids
(to replace the fluids lost through theskin),
and nutritional supplements. Corticosteroids
(such as prednisone) given by mouthor
intravenously.
28. POMPHOLYX
Pompholyx/ dyshidrosis, is a chronic dermatitis characterized by
itchy blisters on the palms and sides of the fingers and sometimes
on the soles of the feet.
The blisters are often scaly,red, and oozing.. Wetcompresses with
potassium permanganate or aluminum acetate (Burow's solution)
may help the blisters resolve. Strong topical corticosteroid
30. Dermatitis herpitiform :
Dermatitis herpitiform is a particular typeof dermatitis
thatappearsas a result of a gastrointestinal condition,
known as celiac disease.
32. 4. LOCALIZED SCRATCH DERMATITIS
Localized scratch dermatitis (lichen simplex chronicus, neurodermatitis) is
chronic, itchy inflammation of the top layer of theskin. Localized scratch
dermatitis can occur anywhere on the body, including the anus (pruritus ani )
and the vagina (pruritus vulvae ), but is most common on the head, arms, and
legs. In the early stages, theskin looks normal, but it itches. Laterdryness
scaling, and dark patches develop as a result of the scratching andrubbing.
Applying surgical tape saturated witha corticosteroid
(applied in the morning and replaced in the evening)
helps relieve itching and inflammation and protects the
skin fromscratching.
35. There is a vasoconstriction of superficial blood vessels and the
skin blanches readily
Cold and low humilidity are poorly tolerated because of drifting effects
Heat and high humidity are poorly tolerated because vasodilatation increases the
inflammatory reaction thus aggravating the dermatitis and causing increased the itching
and discomfort
Lesion become localized to the flexor surface of the neck , to the eyelids , behind the
ears , in the anticubital and poplital areas and at the wrist
The erythema is now dusty in colour and excoriations may
become secondary secondiarily infected
PATHOPHYISIOLOGY :
36. by the late twenties or early thirties the lesions usually disappear , but they
may recur at a late date as chronic hand or foot eczema
Person with atopic dermatitis is highly suseptable to viral infections,
especially herpes, and to bacterial infections, such as those caused by
staphyloccus or hemolytic streptococcus
There is also an increased incidence of fungal infection such as tinae.
By the late twenties or early thristies the lesions usually dissapper ,
37.
38. various exogenous and endogeneous agent(dyes perfumes)
initiate inflammatory response of the skin
skin eruption present that are specific to causative agent
erythema, vesicles, scales and pruritis occur
PATHOPHYISIOLOGY :
Cause stretching In response to irritation and edema serous discharge and crusti
Long term irritation also causes thickened lethargy and darker skin
40. CLINICAL MANIFESTATION :
Red rash. This is the usual reaction. The rash appears immediately in
irritant contact dermatitis; in allergic contact dermatitis, the rash
sometimes does not appear until 24–72 hours after exposure to the
allergen.
Blisters or wheals. Blisters, wheals (welts), and urticaria (hives) often
form in a pattern where skin was directly exposed to the allergen or
irritant. Itchy, burning skin. Irritant contact dermatitis tends to be
more painful than itchy, while allergic contact dermatitis oftenitches.
41. ALLERGIC CONTECT DEREMATITIS
Erythema swelling and pruritic vesicles in area of allergen
CONTECT DERMATITIS
Acute Phase-
pt have erythema itching burning exposed to agent.
Sabacute phase-
crusting, drying, fissuring, burning, exposed to agent
Chronic phase-
after repeted reaction pt. Scratch the skin
CLINICAL MANIFESTATION :
42. Symptoms of dermatitis
The symptoms of dermatitis range from mild to severeand will look different
depending on what part of the body is affected. Not all peoplewith dermatitis
experience allsymptoms.
In general, the symptoms of dermatitis mayinclude:
rashes
blisters
dry, cracked skin itchy skin
painful skin, with stinging orburning redness
swelling
43.
44. MANAGEMENT :
MEDICAL MANAGEMENT
•Bathing Reduce how often you bath or shower, using lukewarm water.
Showers are better. Replace standard soap with a substitute such as a mild
detergent soap-free cleanser : your chemist or dermatologist can advise you.
• Clothing Wear soft smooth cool clothes; wool is best
avoided.
•Irritants Protect your skin from dust, water, solvents, detergents, injury. Avoid
exposure to environmental or food allergens. Common foods that cause allergic
reactions are dairy, soy, citrus, peanuts, wheat (sometimes all gluten
containing grains), fish, eggs, corn, and tomatoes.
•Emollients Apply an emollient liberally and often, particularly after bathing, and
when itchy. Ask your doctor or dermatologist to recommend some to try; avoid
perfumed products when possible.
45. •Topical steroids Apply a topical steroid cream or ointment to the itchy
patches fora 5 to 15 day course.
•Pimecrolimus cream Pimecrolimus is a new anti- inflammatory cream shown
to be very effective for atopic dermatitis, with fewer side effects than topical
steroids.
•Antibiotics Your doctor will recommend antibiotics such as flucloxacillin or
erythromycin if infection is complicating or causing the dermatitis. The
infection is most often with Staphylococcus aureus or Streptococcus pyogenes .
•Antihistamines Antihistamine tablets may help reduce the irritation, and are
particularly useful at night.
• Other treatments Systemic steroids , azathioprine , phototherapy , and other
complicated treatments may also be used for severecases.
46. MEDICAL CARE
Corticosteroids-A corticosteroid medication similar to
hydrocortisone may be prescribed to combat inflammation
in a localized area. This medication may be applied to
your skin as a cream or ointment. If the reaction covers a
relatively large portion of the skin or is severe, a
corticosteroid in pill or injection form may be prescribed.
Antihistamines-Prescription antihistamines may be given
if nonprescription strengths are inadequate .
47. Cleansing Properly
Use The RightProducts
Protecting The Skin
Environment
Watch YourDiet
DrinkWater
Prescriptiondrugs
Reducing provoking factors
NURSING MANAGEMENT :
49. PREVENTION
: Dermatitis relies on an irritant or an
allergen to initiate the reaction, it is important for the
patient to identify the responsible agent and avoid it. In an
industrial setting the employer has a duty of care to the
individual worker to provide the correct level of safety
equipment to mitigate the exposure to harmful irritants.
This can take the form of protective clothing, gloves or
barrier cream depending on the working environment.
52. CONCLUSIONS–
Atopic dermatitis can be treated by following a few basic rules
regarding skin hydration, use of a moisturizer, and topical steroid applications
to reduce inflammation. The distinction between the various types of contact
dermatitis is based on a number of factors. these findings have been
acknowledged not to distinguish [9], and even positive patch testing does not
rule out the existence of an irritant form of dermatitis as well as an
immunological one. It is important to remember, therefore, that the
distinction between the types of contact dermatitis is often blurred, with, for
example, certain immunological mechanisms also being involved in a case of
irritantcontactdermatitis.