Chronic Obstructive Pulmonary Disease (COPD) is characterized by airflow obstruction that is generally progressive and may be partially reversible. It includes emphysema and chronic bronchitis. The primary cause is cigarette smoking which damages the lungs over many years through destruction of lung tissue and increased inflammation. Symptoms include dyspnea, cough, and limited physical activity. Treatment focuses on smoking cessation and medications to relieve symptoms along with respiratory therapy and oxygen as needed.
Emphysema is a type of COPD involving damage to the air sacs (alveoli) in the lungs. As a result, your body does not get the oxygen it needs. Emphysema makes it hard to catch your breath. You may also have a chronic cough and have trouble breathing during exercise. The most common cause is cigarette smoking
Emphysema is a type of COPD involving damage to the air sacs (alveoli) in the lungs. As a result, your body does not get the oxygen it needs. Emphysema makes it hard to catch your breath. You may also have a chronic cough and have trouble breathing during exercise. The most common cause is cigarette smoking
Chronic obstructive pulmonary disease (COPD)- Preeti sharmaEducate with smile
COPD is a type of obstructive lung disease and related conditions. it is very helpful presentation to you about information of COPD.
It includes all things that is definition, causes, symptoms, pathophysiology, diagnostic evaluation, types, treatment and role of nurses for COPD patient.
Acute and Chronic Bronchitis is amongst most common presenting illness for Family Physicians considering its prevalence in all ages. Revisiting it with perspective of a family physician helps improve understanding and management at Family PRactice
An undergraduate lecture on pathophysiology of Chronic Obstructive Pulmonary Disease for Medical Students by Dr Muhammad Omair Riaz (Consultant Immunologist)
Chronic obstructive pulmonary disease (COPD)- Preeti sharmaEducate with smile
COPD is a type of obstructive lung disease and related conditions. it is very helpful presentation to you about information of COPD.
It includes all things that is definition, causes, symptoms, pathophysiology, diagnostic evaluation, types, treatment and role of nurses for COPD patient.
Acute and Chronic Bronchitis is amongst most common presenting illness for Family Physicians considering its prevalence in all ages. Revisiting it with perspective of a family physician helps improve understanding and management at Family PRactice
An undergraduate lecture on pathophysiology of Chronic Obstructive Pulmonary Disease for Medical Students by Dr Muhammad Omair Riaz (Consultant Immunologist)
Bronchitis is an acute inflammation of the air passages within the lungs. It occurs when the trachea (windpipe) and the large and small bronchi (airways) within the lungs become inflamed because of infection or irritation from certain causes. Homeopathy is the best treatment with no side effects. For further information contact Ph. : +91-265-2250212,
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𝐓𝐡𝐞 𝐭𝐞𝐦𝐩𝐥𝐚𝐭𝐞 𝐜𝐨𝐯𝐞𝐫𝐬 𝐭𝐡𝐞 𝐟𝐨𝐥𝐥𝐨𝐰𝐢𝐧𝐠 𝐭𝐨𝐩𝐢𝐜𝐬
𝐃𝐞𝐟𝐢𝐧𝐢𝐭𝐢𝐨𝐧
Chronic obstructive pulmonary disease (𝐂𝐎𝐏𝐃) is a group of lung diseases that cause airflow blockage and breathing-related problems.
𝐩𝐫𝐞𝐯𝐚𝐥𝐞𝐧𝐜𝐞
COPD is more prevalent in developing countries, but it is also a growing problem in developed countries. In the United States, COPD is the third leading cause of death.
Forms of 𝐂𝐎𝐏𝐃
• Chronic bronchitis
• Emphysema
𝐏𝐚𝐭𝐡𝐨𝐩𝐡𝐲𝐬𝐢𝐨𝐥𝐨𝐠𝐲
In normal lungs, air flows freely in and out of the bronchi and alveoli. However, in people with COPD, the airflow is blocked. This can be caused by inflammation of the airways, mucus production, or damage to the air sacs.
𝐑𝐢𝐬𝐤 𝐅𝐚𝐜𝐭𝐨𝐫𝐬
There are several risk factors for COPD, including smoking, air pollution, and genetics. Smoking is the most common risk factor for COPD. Smoking is responsible for about 80% of COPD cases. Air pollution, particularly indoor air pollution from cooking and burning fuels, can also increase the risk of COPD. Genetics can also play a role in COPD. People with a family history of COPD are more likely to develop the disease.
𝐜𝐚𝐮𝐬𝐞𝐬
The main causes of COPD are smoking and air pollution. Smoking damages the lungs and makes it difficult to breathe
𝐒𝐭𝐚𝐠𝐞𝐬
• Stage 1
• Stage 2
• Stage 3
.
𝐒𝐲𝐦𝐩𝐭𝐨𝐦𝐬
The most common symptoms of COPD are:
• Shortness of breath
• Cough
• Wheezing
• Chest tightness
• Fatigue
𝐂𝐨𝐦𝐩𝐥𝐢𝐜𝐚𝐭𝐢𝐨𝐧𝐬
COPD can lead to several complications, including:
• Anemia
• Right-sided heart failure
• Muscle weakness
• Lung infections
• Bone thinning
• Collapsed lungs
𝐃𝐢𝐚𝐠𝐧𝐨𝐬𝐭𝐢𝐜 𝐦𝐞𝐭𝐡𝐨𝐝𝐬
COPD is diagnosed with a spirometry test, which measures how much air a person can exhale.
𝐓𝐫𝐞𝐚𝐭𝐦𝐞𝐧𝐭 𝐎𝐩𝐭𝐢𝐨𝐧𝐬
There is no cure for COPD, but there are treatments that can help manage the symptoms and slow the progression of the disease. Treatment options include:
• Bronchodilators
• Antibiotics
• Supplemental oxygen
• Vaccination
𝐏𝐫𝐞𝐯𝐞𝐧𝐭𝐢𝐯𝐞 𝐌𝐞𝐭𝐡𝐨𝐝𝐬
The best way to prevent COPD is to avoid smoking and air pollution. Several lifestyle changes can help reduce the risk of COPD, such as eating a healthy diet and exercising regularly.
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ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
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- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
2. COPD
Description
Characterized by presence of airflow
obstruction
Caused by emphysema or chronic
bronchitis
Generally progressive
May be accompanied by airway
hyperreactivity
May be partially reversible
4. Chronic Bronchitis
Description
Presence of chronic productive cough for
3 or more months in each of 2 successive
years in a patient whom other causes of
chronic cough have been excluded
5. COPD
Causes
Cigarette smoking
Primary cause of COPD***
Clinically significant airway obstruction
develops in 15% of smokers
80% to 90% of COPD deaths are related
to tobacco smoking
> 1 in 5 deaths is result of cigarette
smoking
6. COPD
Causes
Cigarette smoking
Nicotine stimulates sympathetic nervous
system resulting in:
↑ HR
Peripheral vasoconstriction
↑ BP and cardiac workload
7. COPD
Causes
Cigarette smoking
Compounds problems in a person with CAD
↓ Ciliary activity
Possible loss of ciliated cells
Abnormal dilation of the distal air space
Alveolar wall destruction
Carbon monoxide
↓ O2 carrying capacity
Impairs psychomotor performance and judgment
Cellular hyperplasia
Production of mucus
Reduction in airway diameter
Increased difficulty in clearing secretions
8. COPD
Causes
Secondhand smoke exposure associated
with:
↓ Pulmonary function
↑ Risk of lung cancer
↑ Mortality rates from ischemic heart
disease
9. COPD
Causes
Infection
Major contributing factor to the aggravation
and progression of COPD
Heredity
α-Antitrypsin (AAT) deficiency (produced by
liver and found in lungs); accounts for < 1% of
COPD cases
Emphysema results from lysis of lung tissues by proteolytic
enzymes from neutrophils and macrophages
12. Emphysema
Pathophysiology
Two types:
Centrilobular (central part of lobule)
Most common
Panlobular (destruction of whole lobule)
Usually associated with AAT deficiency
14. Emphysema
Pathophysiology
Small bronchioles become obstructed as a result
of
Mucus
Smooth muscle spasm
Inflammatory process
Collapse of bronchiolar walls
Recurrent infections production/stimulation
of neutrophils and macrophages release
proteolytic enzymes alveolar destruction
inflammation, exudate, and edema
15. Emphysema
Pathophysiology
Elastin and collagen are destroyed
Air goes into the lungs but is unable to
come out on its own and remains in the
lung
Causes bronchioles to collapse
16. Emphysema
Pathophysiology
Trapped air → hyperinflation and
overdistention
As more alveoli coalesce, blebs and bullae may
develop
Destruction of alveolar walls and capillaries →
reduced surface area for O2 diffusion
Compensation is done by increasing respiratory
rate to increase alveolar ventilation
Hypoxemia usually develops late in disease
21. Chronic Bronchitis
Pathophysiology
Pathologic lung changes are:
Hyperplasia of mucus-secreting glands
in trachea and bronchi
Increase in goblet cells
Disappearance of cilia
Chronic inflammatory changes and narrrowing
of small airways
Altered fxn of alveolar macrophages
infections
23. Chronic Bronchitis
Pathophysiology
Greater resistance to airflow increases
work of breathing
Hypoxemia and hypercapnia develop
more frequently in chronic bronchitis
than emphysema
24. Chronic Bronchitis
Pathophysiology
Bronchioles are clogged with mucus and
pose a physical barrier to ventilation
Hypoxemia and hypercapnia d/t lack of
ventilation and O2diffusion
Tendency to hypoventilate and retain CO2
Frequently patients require O2 both at
rest and during exercise
25. Chronic Bronchitis
Pathophysiology
Cough is often ineffective to remove
secretions because the person cannot
breathe deeply enough to cause air flow
distal to the secretions
Bronchospasm frequently develops
More common with history of smoking
or asthma
27. Chronic Bronchitis
Clinical Manifestations
Bronchospasm at end of paroxysms of coughing
Cough
Dyspnea on exertion
History of smoking
Normal weight or heavyset
Ruddy (bluish-red) appearance d/t
polycythemia (increased Hgb d/t chronic hypoxemia))
cyanosis
30. COPD
Diagnostic Studies
Chest x-rays early in the disease may not
show abnormalities
History and physical exam
Pulmonary function studies
reduced FEV1/FVC and ↑ residual
volume and total lung capacity
31. COPD
Diagnostic Studies
ABGs
↓ PaO2
↑ PaCO2(especially in chronic bronchitis)
↓ pH (especially in chronic bronchitis)
↑ Bicarbonate level found in late stages
COPD
35. COPD
Collaborative Care:
Oxygen Therapy
Chronic O2 therapy at home
Improved prognosis
Improved neuropsychologic function
Increased exercise tolerance
Decreased hematocrit
Reduced pulmonary hypertension
36. COPD
Collaborative Care: Respiratory
Therapy
Breathing retraining
Pursed-lip breathing
Prolongs exhalation and prevents bronchiolar
collapse and air trapping
Diaphragmatic breathing
Focuses on using diaphragm instead of accessory
muscles to achieve maximum inhalation and
slow respiratory rate
See text re how to teach
37. COPD
Collaborative Care: Respiratory
Therapy
Huff coughing (Table 28-21)
Chest physiotherapy – to bring secretions
into larger, more central airways
Postural drainage
Percussion
Vibration
41. COPD
Collaborative Care
Nutritional therapy
Full stomachs press on diaphragm causing
dyspnea and discomfort
Difficulty eating and breathing at the same time
leads to inadequate amounts being eaten
42. COPD
Collaborative Care
Nutritional therapy
To decrease dyspnea and conserve energy
Rest at least 30 minutes prior to eating
Use bronchodilator before meals
Select foods that can be prepared in advance
5-6 small meals to avoid bloating
Avoid foods that require a great deal of chewing
Avoid exercises and treatments 1 hour before and
after eating
43. COPD
Collaborative Care
Nutritional therapy
Avoid gas-forming foods
High-calorie, high-protein diet is
recommended
Supplements
Avoid high carbohydrate diet to prevent
increase in CO2 load
44. Nursing Management
Nursing Diagnoses
Ineffective airway clearance
Impaired gas exchange
Imbalanced nutrition: less than body
requirements
Disturbed sleep pattern
Risk for infection
45. Nursing Management
Nursing Implementation
Health Promotion
STOP SMOKING!!!
Avoid or control exposure to occupational
and environmental pollutants and irritants
Early detection of small-airway disease
Early diagnosis of respiratory tract
infections
48. Nursing Management
Nursing Implementation
Ambulatory and Home Care
Teach patient how to achieve optimal capability
in carrying out ADLs
Physical therapy
Nutrition
Education
Activity considerations
Exercise training of upper extremities to help
improve function and relieve dyspnea
49. Nursing Management
Nursing Implementation
n Ambulatory and Home Care
n Explore alternative methods of ADLs
Encourage patient to sit while
performing activities
Coordinated walking
51. Nursing Management
Nursing Implementation
Ambulatory and Home Care
Sexual activity
Plan during part of day when breathing is best
Slow, pursed-lip breathing
Refrain after eating or other strenuous
activity
Do not assume dominant position
Do not prolong foreplay
52. Nursing Management
Nursing Implementation
Ambulatory and Home Care
Sleep
Nasal saline sprays
Decongestants
Nasal steroid inhalers
Long-acting theophylline
Decreases bronchospasm and airway obstruction
53. Nursing Management
Nursing Implementation
Ambulatory and Home Care
Psychosocial considerations
Guilt
Depression
Anxiety
Social isolation
Denial
Dependence
Use relaxation techniques and support groups