Cbc revision-02
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1. The document describes several radiographic findings and histopathological features of oral lesions including a radiolucency in the posterior mandible surrounded by radiopaque areas consistent with a complex odontome. 2. It also mentions an adenomatoid odontogenic tumor characterized by spindle epithelial cells forming whorled masses and duct-like structures. 3. Finally, it discusses a benign cementoblastoma seen as a radiopacity in the lower anterior jaw, clinically appearing as a unilateral swelling and showing active cementoblasts surrounding cement trabeculae radiographically.
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1. The document describes various clinical presentations, radiographic findings, and histopathological features of different dental diseases.
2. Examples include caries lesions at different stages, pulp polyps, periapical abscesses, radicular cysts, dentigenous cysts, and central giant cell granulomas.
3. Histopathology slides show features like transverse dentinal clefts, pulp tissue with dilated blood vessels, and cyst cavities lined by stratified squamous epithelium.
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Ringkasan:
Presentasi kasus membahas delapan topik utama ilmu penyakit gigi dan mulut, yaitu anodontia, impacted teeth, maloklusi, debris, kalkulus, plaque, dental decay, dan pulpitis. Topik-topik tersebut dijelaskan definisi, etiologi, patogenesis, klasifikasi, manifestasi klinis, penegakan diagnosis, dan terapinya. Presentasi ini disampaikan oleh Tenri Ashari Wanahari untuk memenuhi kompetensi di Klinik Ilmu PenyakMore Related Content
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This document discusses odontomas, which are benign developmental malformations of dental tissues that have the basic histological structure of teeth but in a disorganized pattern. Odontomas are classified as compound or complex. Compound odontomas consist of separate small tooth-like structures, while complex odontomas are irregular hard and soft dental tissues showing no tooth resemblance. Odontomas typically present in people aged 10-20 as painless swellings and can cause complications like eruption issues or cyst formation if not removed by conservative surgery.
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The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable. Pierre robin syndrome & Achondroplasia
Pierre Robin syndrome is a condition characterized by three components: cleft palate, mandibular micrognathia or retrognathia, and glossoptosis. It is caused by the mechanical theory where cleft palate prevents palatal shelf fusion and mandibular hypoplasia constrains growth, leading to tongue displacement. Achondroplasia is caused by mutations affecting endochondral ossification, leading to disproportionate short stature and limb shortening, large head size, and restricted joint motion. Both conditions are diagnosed based on clinical features and imaging findings, and require multidisciplinary care.
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This document discusses the differential diagnosis and management of radiolucent lesions. It begins by listing various potential diagnoses for periapical and pericoronal radiolucencies. It then discusses giant cell lesions, fibro-osseous lesions, and odontogenic tumors. The management section describes the therapeutic goals of surgical procedures and details various surgical techniques used to treat oral lesions, including enucleation, marsupialization, enucleation with curettage, and resection. It provides indications, advantages, and disadvantages of each technique.
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The document reports a case of ameloblastoma in a 60-year old female patient. She presented with a swelling on her right cheek that had been growing over the past 2 years. Radiological investigations including OPG and CT scans showed a bicortical expanding lesion in the right body and ramus of the mandible. An incisional biopsy was performed and the provisional diagnosis of ameloblastoma was made.
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Dentine is the mineralized connective tissue that forms the
bulk of the tooth. It surrounds and protects the dental pulp. In
the crown it is covered by enamel, in the root by cementum.
Unlike enamel, dentine is sensitive and is formed throughout
life, giving rise to secondary dentine. Though the odontoblasts
that form the tissue have processes that lie in tubules within
the dentine, the cell bodies lie at the periphery of the pulp,
constituting a dentine/pulp complex. Being a living tissue,
dentine can react to trauma by forming tertiary dentine.
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This document reviews periodontic-endodontic lesions that originate from infections of the periodontium or dental pulp. Pulpal infections can spread to the periodontium through lateral and accessory canals, causing retrograde peri-odontitis. Conversely, periodontal disease can spread to the pulp through these same pathways. The diagnosis of whether a lesion originated from the periodontium or pulp can be difficult, as the clinical and radiographic features may be similar. Determining pulp vitality is important for differential diagnosis and treatment planning.
Pulp calcification
This document discusses pulp calcification and pulp stones. It notes that pulp stones are a physiological manifestation that may increase in number or size due to local or systemic pathology. The etiological factors involved in their formation are not fully understood. As people age, the pulp space decreases in size and the blood vessels, nerves, and cells in the pulp also decrease. Pulp stones can form due to factors like age, circulatory disturbances, orthodontic tooth movement, and genetic predisposition. They are typically composed of calcium and phosphorus. Pulp stones may block access to canal orifices or engage instruments, but can usually be removed during root canal treatment with magnification, access, and proper instruments.
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This document discusses the differential diagnosis of various cysts of the jaws. It describes the clinical presentation and radiographic appearance of odontogenic keratocyst, eruption cyst, gingival cyst of adult, lateral periodontal cyst, radicular cyst, nasopalatine duct cyst, residual cyst, calcifying epithelial odontogenic cyst, dentigerous cyst, and globulomaxillary cyst. Key information provided includes location, symptoms, appearance on imaging, and conditions included in the differential diagnosis for each cyst type.
Complex odontome associated with maxillary impacted
This document presents a case report of a complex odontome obstructing the eruption of a maxillary central incisor in a 12-year old female patient. Radiographs and surgical removal revealed several irregular calcified masses composed of dental tissues. Histopathological examination confirmed the diagnosis of a complex odontome. The odontome was removed and the impacted tooth was left to monitor eruption. Early diagnosis and removal of odontomes can allow for normal eruption of impacted permanent teeth.
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The document discusses various types of odontogenic cysts that develop in the jaws. It defines odontogenic cysts and provides classifications based on etiology and location. Key cysts discussed in detail include the dentigerous cyst, which forms around the crown of an unerupted tooth, and the lateral periodontal cyst, which occurs on the root surface of a vital tooth. For each cyst, the document outlines clinical features, radiographic appearance, histology, pathogenesis and treatment.
Ameloblastoma.
Ameloblastoma is a benign, epithelial odontogenic tumor that arises from odontogenic tissue such as dental lamina or enamel organ. It is most commonly found in the mandible, especially in the molar-ramus region. Radiographically, ameloblastomas often appear multilocular with a soap bubble or honeycomb appearance. Histologically, the two main types are the follicular and plexiform variants. Treatment options range from conservative surgery like curettage to more radical resection with at least 2cm margins. Segmental resection of affected bone followed by reconstruction with plates, wires or grafts is often required to prevent recurrence and facial disfigurement.
Dentin
Dentin forms the bulk of the tooth and contains dentinal tubules that run from the outer dentin to the pulp cavity. Dentinal tubules are hollow tubes that carry odontoblastic processes and nerves. They decrease in diameter and become more spaced out from the pulp cavity to the outer dentin. There are different types of dentin including primary dentin which surrounds the pulp and secondary dentin which is laid down after root formation. Tertiary dentin is formed in response to injury or trauma and can be reactionary or reparative depending on the stimulus. The dentin surrounding each tubule, called peritubular dentin, is more mineralized than the intertubular dentin
Oral pathology, lecture 1
Not all of these slides are completely done by me, some of the slides are already published and I just combine the already published slides with my own slides in order to gain an ideal , comprehensive scientific content. I hope to benefit from these slides and to spread the slides and the information widely.
Postgrad. 2013
This document provides an overview of dental histology and embryology. It discusses ground sections and decalcified sections of enamel, dentin, cementum, pulp, and periodontal ligament. Key topics covered include enamel tufts, Hunter-Schreger bands, incremental lines of Retzius in enamel. In dentin, it describes tubules, intertubular dentin, and lines of von Ebner and Owen. Cementum sections show cellular and acellular cementum as well as Sharpey's fibers. Periodontal ligament fiber groups and epithelial rests of Malassez are also summarized.
Path prac (2)
1. The document describes several dental pathology cases with information on clinical features, radiographic findings, and histopathological features.
2. Cases include examples like dentigerous cyst, ameloblastoma in various subtypes, odontogenic keratocyst, complex and compound odontome, odontogenic myxoma and others.
3. For each case, relevant clinical, radiographic and microscopic diagnostic details are provided.
Odontogenic tumors dina patho
This document provides information on various odontogenic tumors through descriptions of clinical presentations, radiographic findings, and histopathological images. It describes tumors such as ameloblastoma, adenomatoid odontogenic tumor, calcifying epithelial odontogenic tumor, and odontogenic myxoma. The tumors are classified based on subtypes defined by their histological patterns and clinical behavior, ranging from benign to locally aggressive to malignant.
Cysts of oral regions
This document defines and classifies different types of cysts that can occur in the oral cavity. It discusses epithelial cysts, which make up over 50% of oral cysts and includes radicular, dentigerous, and odontogenic keratocysts. Nonepithelial cysts are also mentioned. Specific cysts like paradental, nasopalatine, and solitary bone cysts are defined. Treatment options for jaw cysts include enucleation, marsupialization, a combination of both, and enucleation with curettage.
Peri apical radiolucency
Periapical radiopaque lesions can have several causes:
1. Condensing osteitis is caused by chronic apical inflammation from a necrotic tooth and appears as an amorphous radiopacity with a widened periodontal ligament space. It is treated with root canal treatment.
2. Cemento-osseous dysplasia involves abnormal bone formation replacing bone and fibrous tissues around vital teeth, appearing as radiolucencies, mixed densities or radiopacities with radiolucent rims. It requires follow-up only.
3. Idiopathic osteosclerosis has an unknown origin, appearing as an area of increased bone density around vital teeth. It is
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Accurate localization of impacted supernumerary tooth associated with dentige...
Accurate localization of impacted supernumerary tooth associated with dentige...
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Differential diagnosis and management of radiolucent lesions
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Complex odontome associated with maxillary impacted
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1. The document describes various clinical presentations, radiographic findings, and histopathological features of different dental diseases.
2. Examples include caries lesions at different stages, pulp polyps, periapical abscesses, radicular cysts, dentigenous cysts, and central giant cell granulomas.
3. Histopathology slides show features like transverse dentinal clefts, pulp tissue with dilated blood vessels, and cyst cavities lined by stratified squamous epithelium.
Makalah/Presentasi Kasus: Kepaniteraan Klinik Gigi & Mulut Universitas Sebela...
Makalah/Presentasi Kasus: Kepaniteraan Klinik Gigi & Mulut Universitas Sebela...Tenri Ashari Wanahari
Ringkasan:
Presentasi kasus membahas delapan topik utama ilmu penyakit gigi dan mulut, yaitu anodontia, impacted teeth, maloklusi, debris, kalkulus, plaque, dental decay, dan pulpitis. Topik-topik tersebut dijelaskan definisi, etiologi, patogenesis, klasifikasi, manifestasi klinis, penegakan diagnosis, dan terapinya. Presentasi ini disampaikan oleh Tenri Ashari Wanahari untuk memenuhi kompetensi di Klinik Ilmu PenyakPulpitis
This document discusses different types of pulpitis and periapical inflammation. It defines pulpitis as inflammation of the dental pulp that can be acute or chronic. Acute pulpitis is further divided into reversible and irreversible types based on whether the inflammation is localized or involves the entire pulp. Chronic pulpitis can be closed or open (hyperplastic). Periapical inflammation ranges from granulomas and cysts to abscesses. Diagnosis involves x-rays and pulp testing to evaluate the pulp chamber and periapical region. Treatment depends on the specific condition but may include removal of irritants, root canals, drainage or extraction.
Pulpitis
Pulpitis is inflammation of the dental pulp. There are two types: reversible pulpitis, where the pulp is still vital and can heal if the irritant is removed, usually by filling a cavity. Irreversible pulpitis occurs when the pulp is irreversibly damaged, such as by deep decay introducing bacteria. Management of irreversible pulpitis involves root canal treatment or extraction, while reversible pulpitis is treated by removing the irritant and filling the tooth.
Diseases of pulp
The document discusses diseases of the dental pulp, including causes, symptoms, diagnosis, and treatment. It covers reversible and irreversible pulpitis, pulp degeneration including calcific, atrophic, and fibrous degeneration, as well as necrosis of the pulp. The main causes of pulp disease are physical (trauma, wear), chemical (acids), and bacterial factors. Symptoms depend on the specific disease but may include tooth sensitivity to hot/cold or pain from irritation. Treatment involves removing the cause, performing root canals, or tooth extraction if not restorable.
Clinical features and histopathology of dental caries
This document provides an overview of the classification of dental caries based on various factors such as anatomical site, progression, extent of involvement, number of tooth surfaces affected, chronology, and whether caries was fully removed during treatment. It discusses 12 different classification systems for dental caries and provides details on types of caries such as pit and fissure, smooth surface, root surface, incipient, occult, and others based on these classification criteria. The document also covers the histopathology of caries in enamel and dentin.
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Dental caries ppt
Dental caries is caused by bacterial damage to teeth from plaque and acid production when exposed to fermentable carbohydrates like sugar. The major bacteria involved are Streptococcus mutans and lactobacilli, which adhere to teeth and produce acid from sugars, lowering the pH and demineralizing enamel and dentin. If left untreated, caries can spread to the pulp, causing inflammation and pain. Consequences of untreated dental caries include local infections and possible systemic illnesses.
Dental caries
The document discusses dental caries, including its etiology and classifications. It provides details on:
1) Dental caries is a progressive, subsurface demineralization of teeth caused by bacterial acids that leads to tooth decay.
2) Old and new theories on the etiology of dental caries including the roles of carbohydrates, microorganisms, acids, and dental plaque in the acidogenic/chemoparasitic theory.
3) Classifications of dental caries including based on nature of attack, progression, surfaces involved, direction of attack, number of surfaces, GV Black classification, location, and tissues involved.
Pulipitis
The document discusses diseases of the dental pulp, including pulpitis and necrosis. It defines the pulp as the formative organ of the tooth that builds dentin. Pulpitis is the most common cause of dental pain and can be reversible or irreversible depending on the severity of inflammation. Untreated pulpitis can lead to necrosis or death of the pulp. Causes include mechanical, thermal, chemical, and bacterial factors. Management involves removal of irritants and root canal treatment if needed.
Pulp diseases
This document discusses disease of the pulp, including classification and types. It describes reversible and irreversible pulpitis, with reversible pulpitis causing mild to moderate pain in response to stimuli that subsides after removal of the stimuli. Irreversible pulpitis causes persistent pain even after stimulus removal and can progress to pulp necrosis with total or partial death of the pulp. Other conditions discussed include chronic hyperplastic pulpitis, previously treated teeth, and teeth with previously initiated therapy.
Classification Of Dental Caries
The document classifies dental caries in several ways:
1. Based on anatomical site such as occlusal, root, and smooth surface caries. Occlusal caries are most prevalent.
2. Based on progression as acute, chronic, or arrested caries. Acute caries progresses rapidly while chronic caries is slow.
3. Based on the initial involvement of the tooth surface as primary or recurrent caries. Primary caries is the initial attack while recurrent caries occurs around restorations.
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- 1. Swelling seen in the posterior mandible Radiopaque areas surrounded by a radiolucent rim Complex odontome
- 3. 1- well defined radiolucency with impacted canine. 2- composed of spindle epithelial cells that form whorled masses, and duct like structure. 3- AOT ( adenomatoid odontogenic tumor)
- 4. Multilocular radiopacity due to large number of denticles ( looks like a bag of teeth ) Multiple teeth structure c. Diagnosis. Compound odontome
- 6. 1- multilocular radiolucency with well-defined radiopaque margin. 2- loose C.T with strands or nests of odontogenic epithelium. 3- Ameloblastic fibroma
- 7. a. Composed of loose C.T containing little collagen and large amounts of an intercellular substance, so it looks like dental pulp. b. Odontogenic myxoma
- 8. Benign cementoblastoma Active cementoblasts surrounding cement trabeculae
- 9. Clinical : unilateral swelling of the jaw X-ray : radiopacity in lower anterior of the jaw showing giantly form cementoma
- 10. Lined by pseudostratified squamous epithelium ,the wall contains lymphoid tissue
- 11. Dilaceration
- 12. Enamel pearl
- 14. Shell teeth