A simple description of a virus, how it works and affects our bodies, and how the body defends itself against the virus and what is a virus originally, and how it multiplies in our bodies.
Introduction to virology for Medical studentsNCRIMS, Meerut
Introduction to virology for MBBS students
A virus is an obligate intracellular parasite containing genetic material surrounded by protein
Virus particles can only be observed by an electron microscope
Most viruses range in sizes from 20 – 250 nanometers
Viruses are obligate intracellular parasites
Viruses are non-living entities
Viruses cannot make energy or proteins independent of a host cell (Depends on host cell for replication)
Viral genome are either RNA or DNA but not both.
Viruses have a naked capsid or envelope with attached proteins
Do not possess cellular organization
Viruses do not have the genetic capability to multiply by division.
They are NOT cultiviable on ordinary media.
Much smaller than bacteria
“Filterable agents” – can pass through filters that can hold back bacteria
Vary widely in size:
Largest – poxvirus (300nm)
Smallest – parvovirus (20nm)
General Characters and Classification of Viruses. Includes ICTV classification and Baltimore classification of viruses. A brief explanation of the Viral structure and Lifecycle.
What Causes A Virus Pandemic and How to Prevent Future Ones.pdfAnshuman Jamdade
“The single biggest threat to man’s continued dominance on the planet is the virus”. Joshua Lederberg, Ph.D., Nobel laureate, Film introduction: Outbreak (1995).
What is Virus?
The human race was at its knees. You know how the Covid-19 virus pandemic impacted millions of lives worldwide. Viruses are the smallest and simplest infectious agents that can replicate only inside the living cells of an organism. As viruses lack their own structure, they are unable to replicate on their own and must infect a host cell to reproduce. When a virus infects a host cell, it inserts its genetic material in the host cell’s genes in order to create copies of itself. As the virus multiplies, the infected host cell bursts to release new viruses into the surrounding environment. These new viruses can then go on to infect other cells and even infect other organisms, leading to the spread of the virus and infection. Viruses can infect all life forms, from humans, animals, and plants to micro-organisms including bacteria and fungi. Most viral infections if occur in healthy individuals are usually asymptomatic or with mild symptoms.
Why do viruses mutate so frequently?
Like all other living forms, viruses also go through mutations throughout their lifespan. However, their genetic structure especially of RNA viruses lacks proofreading skills, which makes them undergo random “copying errors” (i.e., genetic mutations) during replication. This also makes them prone to high mutation rates. That’s why most pandemic infections are usually viral in origin. The more it circulates, the more it can change. However, the more virulent virus may be less transmissible, because it reduces the chances of transmission by killing the host. Viruses usually mutate in immunocompromised individuals. If viruses don’t get host cells, their population in the environment may decrease or remain stable.
Viruses may swap genetic material with the host to make a new “mixed” virus with unique properties. This may lead to horizontal gene transfer from a host to a virus or from a virus to a host, which plays an important role in the mutation and evolution of all organisms. All living forms including humans, plants, and animals are evolved from/by micro-organisms; however, micro-organisms are evolved to keep control of macroorganisms.
Why viruses are more dangerous?
Viruses are more unstable like an ion because they lack their own structure to reproduce. They must need a host to grow and replicate. Viruses enter the host cell by camouflaging and tricking it. They first incorporate their genome with the host genome and then multiply by “commandeering” and “hijacking” the host cell to produce more viruses. The infected cell doesn’t know that the commandeering is by the virus, and thus unknowingly becomes a virus factory. New viruses then burst out of a host cell and enter into new cells to repeat the process. This makes a host helpless, functionless, and even defenseless. You felt sick because your body is
Introduction to virology for Medical studentsNCRIMS, Meerut
Introduction to virology for MBBS students
A virus is an obligate intracellular parasite containing genetic material surrounded by protein
Virus particles can only be observed by an electron microscope
Most viruses range in sizes from 20 – 250 nanometers
Viruses are obligate intracellular parasites
Viruses are non-living entities
Viruses cannot make energy or proteins independent of a host cell (Depends on host cell for replication)
Viral genome are either RNA or DNA but not both.
Viruses have a naked capsid or envelope with attached proteins
Do not possess cellular organization
Viruses do not have the genetic capability to multiply by division.
They are NOT cultiviable on ordinary media.
Much smaller than bacteria
“Filterable agents” – can pass through filters that can hold back bacteria
Vary widely in size:
Largest – poxvirus (300nm)
Smallest – parvovirus (20nm)
General Characters and Classification of Viruses. Includes ICTV classification and Baltimore classification of viruses. A brief explanation of the Viral structure and Lifecycle.
What Causes A Virus Pandemic and How to Prevent Future Ones.pdfAnshuman Jamdade
“The single biggest threat to man’s continued dominance on the planet is the virus”. Joshua Lederberg, Ph.D., Nobel laureate, Film introduction: Outbreak (1995).
What is Virus?
The human race was at its knees. You know how the Covid-19 virus pandemic impacted millions of lives worldwide. Viruses are the smallest and simplest infectious agents that can replicate only inside the living cells of an organism. As viruses lack their own structure, they are unable to replicate on their own and must infect a host cell to reproduce. When a virus infects a host cell, it inserts its genetic material in the host cell’s genes in order to create copies of itself. As the virus multiplies, the infected host cell bursts to release new viruses into the surrounding environment. These new viruses can then go on to infect other cells and even infect other organisms, leading to the spread of the virus and infection. Viruses can infect all life forms, from humans, animals, and plants to micro-organisms including bacteria and fungi. Most viral infections if occur in healthy individuals are usually asymptomatic or with mild symptoms.
Why do viruses mutate so frequently?
Like all other living forms, viruses also go through mutations throughout their lifespan. However, their genetic structure especially of RNA viruses lacks proofreading skills, which makes them undergo random “copying errors” (i.e., genetic mutations) during replication. This also makes them prone to high mutation rates. That’s why most pandemic infections are usually viral in origin. The more it circulates, the more it can change. However, the more virulent virus may be less transmissible, because it reduces the chances of transmission by killing the host. Viruses usually mutate in immunocompromised individuals. If viruses don’t get host cells, their population in the environment may decrease or remain stable.
Viruses may swap genetic material with the host to make a new “mixed” virus with unique properties. This may lead to horizontal gene transfer from a host to a virus or from a virus to a host, which plays an important role in the mutation and evolution of all organisms. All living forms including humans, plants, and animals are evolved from/by micro-organisms; however, micro-organisms are evolved to keep control of macroorganisms.
Why viruses are more dangerous?
Viruses are more unstable like an ion because they lack their own structure to reproduce. They must need a host to grow and replicate. Viruses enter the host cell by camouflaging and tricking it. They first incorporate their genome with the host genome and then multiply by “commandeering” and “hijacking” the host cell to produce more viruses. The infected cell doesn’t know that the commandeering is by the virus, and thus unknowingly becomes a virus factory. New viruses then burst out of a host cell and enter into new cells to repeat the process. This makes a host helpless, functionless, and even defenseless. You felt sick because your body is
This ppt gives a brief review of Giant Viruses and their role in medicine?!
Mamavirus is a large and complex virus in the Group I family Mimiviridae. The virus is exceptionally large, and larger than many bacteria.
Prepare for and Survive a Pandemic/Biological WarfareBob Mayer
Pandemics are natural occurring-- usually. However, mankind has also used biological warfare since the advent of war. Biological weapons are also a favorite of terrorists and what is on the horizon is truly frightening. Accidents also happen. How do you prepare for and deal with this event?
International Journal of Virology Studies & Research (IJVSR) ISSN:2330-0027 is a comprehensive, peer reviewed journal devoted to Virology Studies & Research. IJVSR, published by SciDoc is an open access journal that includes high quality papers, which covers all major areas of Virology Studies & Research. SciDoc with its Open Access publication model spreads all the day-to-day developments and research to readers around the world.
International Journal of Virology Studies & Research (IJVSR) ISSN:2330-0027 is a comprehensive, peer reviewed journal devoted to Virology Studies & Research. IJVSR, published by SciDoc is an open access journal that includes high quality papers, which covers all major areas of Virology Studies & Research. SciDoc with its Open Access publication model spreads all the day-to-day developments and research to readers around the world.
IJVSR aims to publish all the latest and outstanding research articles, reviews and letters in all areas of Virology. It contains a series of timely, in-depth written articles by scholars & researchers in the field, covering a wide range of the integration of multidimensional challenges of research of Virology
http://scidoc.org/IJVSR.php
International Journal of Virology Studies & Research (IJVSR) ISSN:2330-0027 is a comprehensive, peer reviewed journal devoted to Virology Studies & Research. IJVSR, published by SciDoc is an open access journal that includes high quality papers, which covers all major areas of Virology Studies & Research. SciDoc with its Open Access publication model spreads all the day-to-day developments and research to readers around the world.
IJVSR aims to publish all the latest and outstanding research articles, reviews and letters in all areas of Virology. It contains a series of timely, in-depth written articles by scholars & researchers in the field, covering a wide range of the integration of multidimensional challenges of research of Virology
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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2. Outline
1. About Virus.
2. The size of Viruses.
3. What is inside the virus?
4. Shapes of Virus.
5. How Viruses reproduces ?
6. The cycle life of virus.
7. VIRUS IN HUMAN.
8. Human Viral Disease.
9. The benefits of virus
3. About Virus
• viruses are everywhere from our surroundings to the food that we
eat, such as in the soil and living organism like humans.
• Viruses can infect all types of life forms including bacteria.
• Over 5,000 species of viruses have been discovered.
A virus is a small infectious agent that replicates only inside the living cells of an organism .
4. Viruses are very tiny germs.
.(in other words, they are sub-microscopic);
their sizes range from 20 to 300 nm.
The size of Viruses
5. What is inside the virus?
Most scientists accept that viruses are not living. Because they do
not have cell membrane, organelles (like mitochondria), any nucleus
and cytoskeleton .
It is genetic material made from either DNA or RNA, that carry
genetic information and a protein casing, called the capsid, which
surrounds and protects the genetic material.
A complete virus particle, known as a vision.
"organisms at the edge of life"
7. How Viruses reproduces ?
1. Viruses reproduce by injecting their genetic material into a host cell.
2. Forcing the cell to make copies of the virus, instated of what the cell
would normally be doing.
o This is because they do not have ribosomes or other components
necessary to express
9. VIRUS IN HUMAN
• A lot of viruses exist in human body but most Of them do not cause
diseases.
• Human immune system can generate antibodies against viruses, the
antibody will recognize the virus and destroy it.
Immune system
Virus
10. Human Viral Disease
Human diseases caused by viruses include,
• HIV
• Influenza
• Ebola
• Hepatitis
• Common Gold
• ..etc.
11. The benefits of virus
• From billion wears ago war has
been killing trillions every single
day well we don't even notice the
war.
• Is fought by the single deadliest
entity on our planet the Virus.
• Up to 40% of all bacteria in the
oceans are killed by them every
single dayOCEAN BACTERIA
40 60
killed alive
12. bacteria were our faces tiny monsters that
hunted us mercilessly
But we found a solution in nature.
suddenly we had a powerful superweapon
antibiotic was so effective that we stopped
thinking of bacteria as monsters .
in the past, a single cut or a sip from the wrong water could kill you.
13. • we used antibiotics more and more for less and less serious causes
we lost respect for the bacteria .
• but bacteria are living things that evolve and they started to become
immune against antibiotics our solution .
• this continued until we had created what are called superbugs
bacteria immune to almost everything we have.
this immunity is spreading across the world as we talk know.
We change bacteria
15. In 2050 superbugs could kill more humans a year than cancer
the days when a cut or a cough could kill you or your loved ones are
coming back
BUT!
how could injecting millions of viruses into an infection be a good idea?
phages our tiny killer virus robots could save us, we can inject them
into our bodies to help cure infections.
16. Use bacteriophages.
• bacteriophage are very specialized killers of bacteria
• so specialized in fact that humans are completely immune to them,
we are too different we encounter billions of bacteriophages every
day.
• with bacteria develop ways of defending themselves more complex
than that bacteriophages evolved too but there has been an arms
race between Virus and bacteria for billions of years and so far Virus
doing great this makes phages smart weapons that are constantly
getting better at killing.
This tested with “Pseudomonas Aeruginosa” that infect chest
17. summary
• Can't classify a virus as a prokaryote or a eukaryote either because it's
not a cell.
• Virus exist everywhere.
• Virus very small.
• made from either DNA or RNA.
• The cycle life of virus tow type lytic cycle and lysogenic cycle.
• We can use virus as medicine, against superbugs.