The document discusses various types of thoracic trauma injuries that can occur. It covers immediately life-threatening injuries like tension pneumothorax, massive haemothorax, and pericardial tamponade that require urgent treatment to control bleeding and decompress the chest. It also discusses potentially life-threatening injuries such as aortic injuries, flail chest, pulmonary contusion, and diaphragmatic injuries that may require procedures like chest drain insertion or surgery if complications occur.
CHEST INJURY- BLUNT/ Trauma Surgery
Dear viewers,
Greetings from “Surgical Educator”
Today I have uploaded a video on CHEST INJURY- BLUNT- an important topic in trauma. Even the blunt chest trauma can turn into penetrating one because of jagged edges of the broken ribs. I haven’t talked elaborately but have included the essential minimum an undergraduate medical student should know. I have talked about pathophysiology, clinical approach, symptoms, signs, investigations, different individual types of Chest injuries and management of all the varieties of Chest injuries. My aim is, after watching this video all of you should be able to arrive at a correct working diagnosis of the type of chest injury and should also be able to institute immediate lifesaving treatment to the patients if there is a need. You can watch the video in the following links:
surgicaleducator.blogspot.com
youtube.com/c/surgicaleducator
Thank you for watching the video.
The document consists of the modern day care in case of thoracic. It touches all relevant aspects of the thoracic trauma. Latest recommendations and user friendly interface is the main highlight of this document. One can easily un
CHEST INJURY- BLUNT/ Trauma Surgery
Dear viewers,
Greetings from “Surgical Educator”
Today I have uploaded a video on CHEST INJURY- BLUNT- an important topic in trauma. Even the blunt chest trauma can turn into penetrating one because of jagged edges of the broken ribs. I haven’t talked elaborately but have included the essential minimum an undergraduate medical student should know. I have talked about pathophysiology, clinical approach, symptoms, signs, investigations, different individual types of Chest injuries and management of all the varieties of Chest injuries. My aim is, after watching this video all of you should be able to arrive at a correct working diagnosis of the type of chest injury and should also be able to institute immediate lifesaving treatment to the patients if there is a need. You can watch the video in the following links:
surgicaleducator.blogspot.com
youtube.com/c/surgicaleducator
Thank you for watching the video.
The document consists of the modern day care in case of thoracic. It touches all relevant aspects of the thoracic trauma. Latest recommendations and user friendly interface is the main highlight of this document. One can easily un
chest trauma is one of the leading cause of death in poly trauma patients. ER doctor should be aware of how to suspect and how to deal with life threatening conditions resulting from chest trauma
The history of developments in the field of surgery since the dawn of civilization, leading to modernization of the field to the current scientific era.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
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Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
3. INTRODUCTION
• a training program for medical providers in the management of acute trauma
cases
• developed by the American College of Surgeons
• goal is to teach a simplified and standardized approach to trauma patients
• premise of the ATLS program is to treat the greatest threat to life first
• no need of a definitive diagnosis and a detailed history
• with the most time-critical interventions performed early – best utilization of
“golden hour” which lies between life and death after a traumatic event
4. ATLSPROTOCOL
• Scene safety
• Primary survey
– identify and manage the most immediately life-threatening pathologies first
• Resuscitation
• Secondary survey
– detailed head to toe examination
5. PRIMARY SURVEY
• ‘c’ : Exsanguinating external haemorrhage
• A :Airway & cervical spine control
• B : Breathing and ventilation
• C : Circulation and haemorrhage control
• D : Disability (neurological evaluation)
• E : Exposure (assess for other injuries) + environmental control
6. C: EXSANGUINATING EXTERNAL
HAEMORRHAGE
• Controlled immediately by the application of packs and pressure directly onto the bleeding
wound and artery.
• Haemostatic dressings that contain agents that augment local coagulation are now available.
• In case of failure to control bleeding on a limb, application of a tourniquet proximal to the
wound.
• The time at which the tourniquet is applied must be recorded.
• Requires urgent surgical control of the bleeding in order to re-perfuse the limb.
7. A: AIRWAY CONTROL
• General measures
– Suctioning secretions or blood
– Clearing any foreign bodies, dentures
etc.
– Chin lift
– Jaw thrust
• No head tilt to be done in trauma
patients – Why?
9. A: CERVICAL
SPINE CONTROL
• Immobilize the patient
• Avoid hyperextension of
neck
• Apply cervical collar
• Can be stabilized manually
by assistant during airway
management
10. B : BREATHING AND VENTILATION
• Expose the chest & assess RR & respiration type.
• Give O2 inhalation
• Check chest wall, lungs & diaphragm by inspection, palpation, percussion & auscultation
• Pulse oximeter
• Look for conditions that impair ventilation
– Tension pneumothorax
– Massive haemothorax
– Flail chest
– Rib fractures
– Open pneumothorax
– Pulmonary contusion
12. INTRODUCTION
• Thoracic injury accounts for 25% of all injuries.
• In a further 25%, it may be a significant contributor to the subsequent death of
the patient.
• In most of these patients, the cause of death is haemorrhage.
• About 80% of patients with chest injury can be managed non-operatively.
• The key is early physiological resuscitation followed by diagnosis.
13. CLINICAL INDICATORS OF BLEEDING
• Physiological
– Increasing respiratory rate
– Increasing pulse rate
– Falling blood pressure
– Rising serum lactate
• Anatomical
– Visible bleeding
– Injury in close proximity to major vessels
– Penetrating injury with a retained weapon
14. INVESTIGATIONS
• Ultrasound – eFAST
– extended Focused Assessment with Sonar forTrauma is becoming the most common investigation.
– The technique uses sonographic assessment in the chest, looking for
• cardiac tamponade
• free blood/air in the hemithorax on each side
– Also assessment for blood in the abdominal cavity, in the paracolic gutters, subdiaphragmatic spaces
and pelvis.
• Underwater chest drain
– In the physiologically grossly unstable patient in respiratory distress, there is no time for radiological
investigations
– Insertion of an underwater chest drainage tube can be a diagnostic procedure as well as a
therapeutic one.
15. INVESTIGATIONS (CONTD.)
• Chest radiograph
– In those cases where the patient is haemodynamically unstable but with acceptable oxygenation, an
anteroposterior (AP) supine chest radiograph is usually the simplest initial investigation,
– Will provide good information regarding
• tracheal deviation
• injury to the lung parenchyma
• mediastinal pathology
• injury to skeletal elements causing pneumothorax, haemothorax or lung contusion.
– The presence of thoracic skeletal injury should alert the clinician to the possibility of adjacent
thoracic or abdominal visceral injury.
16. INVESTIGATIONS (CONTD.)
• Computed tomography scan
– Only indicated in stable patients.
– CT scan with contrast allows for three-dimensional reconstruction of the chest and
abdomen, as well as of the bony skeleton.
– It has become the principal and most reliable examination for major injury in thoracic
trauma.
– Has replaced angiography as the diagnostic modality of choice for the assessment of the
thoracic aorta and mediastinal vessels.
19. AIRWAY OBSTRUCTION
• Early preventable trauma deaths are often due to lack of or delay in airway control.
• Causes:
– Dentures, teeth, secretions and blood
– Bilateral mandibular fracture,
– Expanding neck haematomas producing deviation of the pharynx and mechanical compression of the
trachea
– Laryngeal trauma such as thyroid or cricoid fractures
– Tracheal injury
20. AIRWAY OBSTRUCTION (CONTD.)
• Management
– Early intubation is very important,
particularly in cases of neck haematoma
or possible airway oedema.
– Airway distortion can be insidious and
progressive and can make delayed
intubation more difficult if not impossible.
– Tracheostomy if required
21. TENSION PNEUMOTHORAX
• Develops when a ‘one-way valve’ air leak
occurs either from the lung or through
the chest wall.
• Collapsing the affected lung.
• Mediastinum displaced to the opposite
side.
• Decreased venous return.
• Compression of the opposite lung.
22. TENSION PNEUMOTHORAX (CONTD.)
• Causes:
– penetrating chest trauma
– blunt chest trauma with parenchymal lung injury
– iatrogenic lung punctures (e.g. due to subclavian central venepuncture) and
– mechanical positive pressure ventilation
• Clinical presentation:
– Dramatic
– The patient is increasingly restless with tachypnoea, dyspnoea and distended neck veins
– Clinical examination may reveal tracheal deviation, hyper-resonance and decreased or absent breath
sounds over the affected hemithorax
– Is a clinical diagnosis and treatment should never be delayed by waiting for radiological confirmation
23. TENSION PNEUMOTHORAX (CONTD.)
• Treatment:
– Needle thoracostomy
• immediate decompression
• by rapid insertion of a large-bore
cannula into the second intercostal
space in the mid-clavicular line of the
affected side.
– Followed by tube thoracostomy
(inter-costal drain)
• insertion of a chest tube through the
fifth intercostal space in the anterior
axillary line.
25. PERICARDIAL
TAMPONADE
• Accumulation of a relatively small amount of blood into
the non-distensible pericardial sac.
• Compression of the heart and obstruction of the venous
return, leading to decreased filling of the cardiac
chambers during diastole.
• M/C cause penetrating trauma
• All patients with penetrating injury anywhere near the
heart plus shock must be considered to have cardiac
injury until proven otherwise
26. PERICARDIAL
TAMPONADE (CONTD.)
• Clinical presentation
– Classically, Beck’s triad
• decline in arterial pressure
• central venous pressure elevation
• muffled heart sounds
– However, in cases in which major bleeding from other
sites has taken place, the neck veins may be flat.
• Needs to be differentiated from a tension
pneumothorax in the shocked patient with
distended neck veins.
27. PERICARDIAL
TAMPONADE (CONTD.)
• Investigations:
– eFAST
• showing fluid in the pericardial sac.
• This is the most expeditious and reliable diagnostic tool.
– Chest radiograph
• shows an enlarged heart shadow.
28. PERICARDIAL TAMPONADE (CONTD.)
• Treatment
– Operative repair –The correct immediate treatment
• left antero-lateral thoracotomy – preferred
• clamshell thoracotomy (if both hemithorax also to be explored)
• sternotomy
• subxiphoid window
– in the operating theatre if time allows
– otherwise in the emergency room – Emergency Department Thoracotomy
30. PERICARDIAL
TAMPONADE (CONTD.)
• Treatment (contd.)
– Needle pericardiocentesis
• always under ECG control
• only as a desperate temporary measure in a
transport situation
• high potential for iatrogenic injury to the heart
• “dry tap” does not rule out tamponade
32. OPEN PNEUMOTHORAX (CONTD.)
• Treatment:
– 3-way occlusive dressing
– initial management consists of
promptly closing the defect with a
sterile occlusive plastic dressing,
taped on three sides to act as a
flutter-type valve
– a chest tube is inserted as soon as
possible in a site away from the
injury site.
– definitive management includes
proper cleaning and suturing of
wound
33. MASSIVE
HAEMOTHORAX
• collection of blood in pleural cavity
• causes:
– in blunt injury
• m/c is continuing bleeding from torn
intercostal vessels or
• occasionally from the internal mammary
artery secondary to fractures of the ribs
– in penetrating injury a variety of viscera, both
• thoracic and
• abdominal (with blood leaking through a hole
in the diaphragm from the positive pressure
abdomen into the negative pressure thorax)
34. MASSIVE
HAEMOTHORAX (CONTD.)
• Clinical presentation:
• haemorrhagic shock
• flat neck veins
• unilateral absence of
breath sounds
• dullness to percussion
• dyspnoea and cyanosis –
late.
36. MASSIVE HAEMOTHORAX (CONTD.)
• Treatment:
– intercostal drain
– correcting the hypovolemic shock
– urgent thoracotomy
• >1500 mL of blood drained
• ongoing haemorrhage >200 mL/h
over 3–4 hours
– clamping a chest drain to tamponade
a massive haemothorax is not helpful
37. FLAIL CHEST
• three or more ribs fractured in
two or more places
• leads to paradoxical motion of a
chest wall segment
• on inspiration, the loose segment is
displaced inwards and therefore
less air moves into the lungs
• on expiration, the segment moves
outwards
38. FLAIL CHEST (CONTD.)
• diagnosis is made clinically in
patients who are not ventilated,
not by radiography
• observe for paradoxical motion
of a chest wall segment
• high risk of developing a
pneumothorax or haemothorax
• CT scan, with contrast to display the
vascular structures and a 3-D reconstruction
of the chest wall, is the gold standard for
diagnosis of this condition – but rarely
indicated for only this
39. FLAIL CHEST (CONTD.)
• Treatment:
– oxygen administration
– powerful analgesia (including opiates)
– physiotherapy
– if ICD in situ, topical intrapleural local analgesia introduced via the tube
– ventilation is reserved for cases developing respiratory failure despite
adequate analgesia and oxygen
• Traditionally, mechanical ventilation (IPPV) was used to ‘internally splint’ the chest, but had a
price in terms of ICU resources and ventilation-associated morbidity – not recommended now
40. FLAIL CHEST (CONTD.)
• Treatment (contd.):
– surgery to stabilize the flail segment using internal fixation of the ribs may
be useful in a selected group of patients with isolated or severe chest
injury and pulmonary contusion
– strapping of chest wall by adhesive bandages is not recommended now.
42. THORACIC AORTIC
DISRUPTION
• common cause of sudden death after an automobile
collision or fall from a great height
• fixed distal to the ligamentum arteriosum, shear forces
from a sudden impact disrupt the intima and media
• if the adventitia is intact, the patient may remain
haemodynamically stable
• salvage is frequently possible if aortic rupture is identified
and treated early
43. THORACIC AORTIC
DISRUPTION (CONTD.)
• Clinical features:
– should be suspected in patients with
• gross asymmetry in systolic blood
pressure (between the two upper limbs,
or between upper and lower limbs)
• widened pulse pressure (SBP – DBP)
• chest wall contusion
• sudden deceleration
44. THORACIC AORTIC
DISRUPTION
(CONTD.)
• Investigations:
– erect chest radiography
most commonly shows a
widened mediastinum
– confirmed by a CT scan of
the mediastinum
– transoesophageal
echocardiography, in
unstable patients who
cannot be moved to the
scanner
45. THORACIC AORTIC DISRUPTION
(CONTD.)
• Treatment:
– initially, control of the systolic arterial
blood pressure <120 mmHg
– an endovascular intra-aortic stent can
be placed
– operatively repaired by
• direct repair
• excision and grafting using a Dacron
graft.
46. TRACHEOBRONCHIAL
INJURIES
• severe subcutaneous emphysema with
respiratory compromise
• chest drain placed on the affected side
will reveal a large air leak
• the collapsed lung may fail to re-
expand
49. MYOCARDIAL CONTUSION
• should be suspected in any patient sustaining blunt trauma who
develops early ECG abnormalities
• 2-D echocardiography may show wall motion abnormalities
• no role of enzyme estimations in diagnosis
• are at risk of developing sudden dysrhythmias and should be closely
monitored
50. PULMONARY CONTUSION
• very common injury and the major cause of hypoxaemia after blunt
trauma
• causes:
– more frequently following blunt trauma, usually associated with a flail segment or
fractured ribs
– following gunshot wounds, there is an area of contusion from the shock wave of
the bullet
• clinical features:
– worsening hypoxaemia for the first 24–48 hours
– haemoptysis or blood in the endotracheal tube
51. PULMONARY CONTUSION (CONTD.)
• investigations:
– chest radiographic findings may be typically
delayed
– contrast CT scanning is confirmatory
• treatment:
– In mild contusion
• oxygen administration
• pulmonary toilet
• adequate analgesia
– In more severe cases
• mechanical ventilation is necessary
• normovolaemia is critical for adequate
tissue perfusion
52. DIAPHRAGMATIC INJURIES
• any penetrating injury below the 5th ICS should raise suspicion of diaphragmatic and
visceral injury
• blunt injury to the diaphragm is usually caused by a compressive force applied to the
pelvis and abdomen, rupture is usually large, with herniation of the abdominal
contents into the chest
• diagnosis can easily be missed in the acute phase, and may only be discovered at
operation, or through the presentation of complications
• most diaphragmatic injuries are silent and the presenting features are those of injury
to the surrounding organs
53. DIAPHRAGMATIC
INJURIES (CONTD.)
• thorax is at negative pressure and the
abdomen is at positive pressure
• breach of the diaphragm leads to
herniation of abdominal contents into
the chest
• may present much later, and
strangulation of any of the contents
can occur – with a high mortality rate
54. DIAPHRAGMATIC INJURIES (CONTD.)
• Presentation:
– most are silent, presenting features of injury to the surrounding organs
• Investigations:
– no single standard investigation
– chest radiography after placement of a NG tube may be helpful
– contrast studies, CT scan, ultrasound and diagnostic peritoneal lavage all
lack positive or negative predictive value
– most accurate evaluation is by
• video-assisted thoracoscopy (VATS) or
• laparoscopy – allows repair and additional evaluation of the abdominal organs
55. DIAPHRAGMATIC INJURIES (CONTD.)
• Treatment:
– operative repair is
recommended in all cases
– must be repaired via the
abdomen and not the chest, to
rule out penetrating hollow
viscus injury
57. OESOPHAGEAL INJURY
• most from penetrating trauma
• blunt injury is rare
• Presentation:
– a high index of suspicion is required
– odynophagia (pain on swallowing)
– subcutaneous or mediastinal emphysema
– air in the peri-oesophageal space
– pleural effusion
– unexplained fever
• Mediastinal and deep cervical emphysema are evidence of an
aerodigestive injury until proven otherwise.
58. OESOPHAGEAL INJURY (CONTD.)
• A combination of following confirm the
diagnosis in the great majority of cases
– oesophagogram in the decubitus position
and
– oesophagoscopy
• The treatment is operative repair of any
defect and drainage
• The mortality rate rises exponentially if
treatment is delayed
60. EMERGENCY THORACIC SURGERY
• timely surgical intervention can be the key step in saving an injured
patient’s life
• could be:
– Emergency Department Thoracotomy (EDT)
• urgently done bedside in the emergency room
– Planned emergency thoracotomy
• takes place in the more controlled environment of the operating theatre
• clinical decision as to whether a patient requires ED surgery can be
complex
• far better to perform a thoracotomy in the operating room, with good
light and assistance and the potential for bypass, than it is to attempt
heroic emergency surgery in the resuscitation area
61. EMERGENCY DEPARTMENT
THORACOTOMY (EDT)
• reserved for those patients suffering penetrating injury in whom signs of life
are still present
• EDT is considered futile if:
– CPR in the absence of endotracheal intubation >5 min
– CPR with endotracheal intubation >10 min
– when there have been no signs of life at the scene
62. EMERGENCY DEPARTMENT
THORACOTOMY (CONTD.)
• Aim:
– internal cardiac massage
– control of ongoing severe haemorrhage
– control of massive air leak
– clamping of the thoracic aorta
• to preserve the blood supply to the heart and brain, and cutting off the arterial supply distally,
in a moribund patient with a major distal penetrating injury
63. HOME WORK
• Please watch following two videos on before next slides, as it will be
easier to understand them:-
1. ChestTube ATLS
– https://youtu.be/qR3VcueqBgc
2. The fundamentals of Chest Tube Physiology
– https://youtu.be/1CYAYNJNias
64. TUBE THORACOSTOMY - INTRODUCTION
• aka
– inter-costal drain (ICD)
– chest tube
– tube thoracocentesis
• inserted to provide outlet of
air/fluid from pleural cavity
• always placed in Under
Water-Seal Drainage
66. INDICATIONS
IN TRAUMA
• Pneumothorax
– Simple
– Tension
– Open
• Haemo-pneumothorax
• Haemothorax
• Diaphragm injury
NON-TRAUMA
• Spontaneous pneumothorax
• Subcutaneous emphysema
• Pleural effusion “with respiratory
distress”
• “Recurrent” pleural effusion
• Empyema thoracis
• Chylothorax
WITH OTHER
• With any thoracic
surgery
• Diaphragmatic hernia
repair
67. ANATOMICAL PLACEMENT
• in the 5th intercostal space slightly
anterior to the mid axillary line
• Zone of safety
– recommended by BritishThoracic
Society
– is a region bordered by
• the lateral border of pectoralis major
• a horizontal line inferior to the axilla
• the anterior border of latissimus
dorsi
• a horizontal line superior to the
nipple
68. ANATOMICAL
PLACEMENT (CONTD.)
• inserted just at the upper border of the rib
• Directed
– Medially
– Posteriorly
– Apically – if air
– Downward – if fluid/blood
• Size – Measured in French gauge (Fr)
– Adult – 28-40 Fr
– Children – 16-28 Fr
– Infants – 12-20 Fr
70. PRE-PROCEDURE PREPARATION
• Obtain informed consent from patient/guardian
• Explain the procedure to the patient in detail
• Establish intra-venous access
• Patient to be placed on oxygen inhalation and continuous Pulse oximetry and ECG monitoring
• Confirm the side on which ICD to be placed by History, Clinical examination and X-ray/CT
• Position the patient
– 45 degree recumbent
– with ipsilateral hand above the head
• Identify anatomical landmarks
• Charge the drain bag/bottle with sterile water
71. PROCEDURE
1. Part painting and draping
– Paint the area of insertion with Povidone-iodine solution (or any other antiseptic) using sponge-
holding forceps
– Cover rest of the surrounding area with sterile drape sheets
2. Anaesthesia
– Local anaesthesia
– Lignocaine with adrenaline infiltrated at the site of insertion, first in skin then deeper, till pleura
72. PROCEDURE (CONTD.)
3. Steps
– a 1-2 cm transverse incision is given on the skin using BP knife
– blunt dissection is done in the subcutaneous and muscle plane using curved
artery forceps and finger
– intercostal muscles also dissected similarly just at the upper border of rib
– confirmed that pleura is reached by inserting finger (will feel as soft
smooth structure)
– if needed further local anaesthetic can be given directly in the pleura
– using the tip of artery forceps in a controlled manner under finger
guidance, pleura is punctured; a gush of air/blood/fluid will confirm this
73. PROCEDURE (CONTD.)
3. Steps (contd.)
– opening is kept occluded with finger
– the external end of ICD tube is
clamped with one artery forceps
– the internal end is grasped in other
artery forceps and inserted via the
incision
– tube is guided medially, posteriorly
and apically/downward as needed
74. PROCEDURE (CONTD.)
3. Steps (contd.)
– external end is connected with
UWSD bag/bottle and unclamped
– evacuation of fluid/bubbles via the
tube and movement of column of fluid
with breathing confirms correct
placement
– the tube is now fixed with non-
absorbable suture using needle holder
and toothed forceps
– dressing is done
– post-procedure x-ray is taken to
confirm position and lung re-
expansion
75. POST-PROCEDURE MONITORING
• continuous oxygen saturation monitoring
• periodic chest examination to look for bilateral movement and air-entry
• periodic examination of ICD to look for:
– output – amount and character (blood, serous, pus, etc.)
– column movement
– persistent air leak
• periodic dressing and examination of drain site for infection and displacement of tube
76. COMPLICATIONS
IMMEDIATE
• bleeding (due to injury of the intercoastal
artery)
• injury of the lung
• diaphragmatic injury and intra-peritoneal
placement
• liver/spleen injury
• subcutaneous emphysema
• injury to the intercostal nerves
DELAYED
• occluded tube – M/C
• leaks
• inadequate drainage
• persistent collection of air/fluid – may
need second tube
• pulmonary edema secondary to lung re-
expansion
• infection (specially pneumonia)