Thoracic injuries can cause serious complications by compromising the airway, ventilation, and circulation. Key threats include tension pneumothorax, massive hemothorax, and cardiac tamponade. Proper assessment is important to identify these life-threatening injuries through primary and secondary surveys using inspection, auscultation, palpation, and adjuncts like ultrasound. Early interventions such as needle decompression, chest tube insertion, and surgery can help manage injuries and prevent further deterioration.
there is the introduction part of the torso trauma,
check out my next ppts for further more about torso trauma.
contents are in following order...
introduction
mechanism of injury
junctional zones of torso
tension pneumothorax
cardiac temponade
massive hemothorax
etc.
check out all slides
This is a presentation which contains basics of polytrauma management,ATLS, triage, critical decision making skills, application of Glasgow coma scale and complications of different management strategies, if not applied properly.
there is the introduction part of the torso trauma,
check out my next ppts for further more about torso trauma.
contents are in following order...
introduction
mechanism of injury
junctional zones of torso
tension pneumothorax
cardiac temponade
massive hemothorax
etc.
check out all slides
This is a presentation which contains basics of polytrauma management,ATLS, triage, critical decision making skills, application of Glasgow coma scale and complications of different management strategies, if not applied properly.
polytrauma lecture prepare by three medical student in Kerbala university / college of medicine department of surgery to presented as seminar
for download as ppt
https://drive.google.com/open?id=1bc3HMEeJyhrOwag-AvTFMmPVKi12O1PU
INITIAL ASSESSMENT OF TRAUMA PATIENTS....(INSPIRED FROM CTLS AND ATLS GUIDELI...Prerna Biswal
THIS PRESENTATION WAS MADE AT IMA HOUSE IN BHUBANESWAR,ODISHA, BY DR.NIBEDITA PANI,HOD ,DEPT. OF ANAESTHESIOLOGY AND DR.PRERNA BISWAL,PG,ANAESTHESIOLOGY,SCBMCH,CUTTACK,
polytrauma lecture prepare by three medical student in Kerbala university / college of medicine department of surgery to presented as seminar
for download as ppt
https://drive.google.com/open?id=1bc3HMEeJyhrOwag-AvTFMmPVKi12O1PU
INITIAL ASSESSMENT OF TRAUMA PATIENTS....(INSPIRED FROM CTLS AND ATLS GUIDELI...Prerna Biswal
THIS PRESENTATION WAS MADE AT IMA HOUSE IN BHUBANESWAR,ODISHA, BY DR.NIBEDITA PANI,HOD ,DEPT. OF ANAESTHESIOLOGY AND DR.PRERNA BISWAL,PG,ANAESTHESIOLOGY,SCBMCH,CUTTACK,
The document consists of the modern day care in case of thoracic. It touches all relevant aspects of the thoracic trauma. Latest recommendations and user friendly interface is the main highlight of this document. One can easily un
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
11. Early intervention – as required
• High concentration O2
• BVM ventilation , SPo2 monitoring
• Secure airway
• Finger thoraco/needle thoraco
• 3-sided occlusive dressing
• Chest tube
12. Circulation and haemorrhage control
-> massive hemothorax/cardiac tamponade
Assest:-
-> external source
-> pulse – volume, rate, regularity, paradox.
-> CRT, Skin colour, temperature of skin,
-> jugular vein
-> BP and PR
13. Management
Stop the bleeder – direct pressure
IV ascess – Large bore (2) , safe O/GXM
Warm Crystalloid/blood product
Massive hemothorax- chest tube
Cardiac tamponade – pericardiocentesis
* Surg Team early involvement
14. Adjuncts
• ECG
• ABG
• Spo2 monitoring
• ETCo2 monitoring
• E-FAST – Lung point/ Sliding Sign/Pericardial
Fluid
• X-ray – Chest – What to look for?
(loss of lung marking, intrapleural fluid, widening
mediastinum, rib fracture, s/c emphysema, ETT
placement, shift of midline)
15. 2ndary Survey
• Reexamine head to toe – look for missed
injuries – esp on chest region.
• Intervention as required
• Adjuncts
-> Ct Scans?angiography
Bronchoscopy
16. ATOM - TC
• AIRWAY OBSTRUCTION
• TENSION PNEUMOTHORAX
• OPEN PNEUMOTHORAX
• MASSIVE HEMOTHORAX
• TRACHEOBRONCHIAL TREE INJURY
• CARDIAC TAMPONADE
17. AIRWAY OBSTRUCTION
• Mechanism:-
-> laryngeal injury
-> Post. Dislocation of clavicular head
-> Penetrating Trauma to Neck
-> Edema/Blood/Vomitus/floppy tongue
18. • L: ICS, supraclavicular & Accesory muscle retraction /
FB in oropharynx
• L: Stridor, Air movement over nose, mouth and lung
field
• F: crepitus over Ant. Neck, Deformity in
sternoclavicular joint region
• Remove the obstruction - finger sweep,
jaw thrust, Suction, OPA,LMA, ETT
- Reduction of Post. Dislocation of head of clavicle by
shoulder extension
21. • 1 way valve air leak
• Air is trapped within pleural cavity
• Compressing the lungs
• Causes displacement of mediastinum
• Leads to raised intrapleural/intrathoracic
pressure, kinking of great vessels
• Reduction of venous return , reduced CO,
-> Obstructive shock
24. Treatment
• Principle : decompression – converting tension
pneumoT to simple pneumoT
1) Needle thoracocentesis
- 2nd Ics, midclav line VS Safety Triangle( 5th ICS ,
ant to mid axillary line)
2) Finger Thoracostomy
- Safety Triangle
- Mandatory chest tube
3) Chest Tube with under water seal
25. Open Pneumothorax
• Definition: large open chest wall defect
creating a direct communication btwn pleural
space and atmosphere, resulting in collapse of
the lungs.
• If opening is 2/3 size of trachea
crossectionally, then air will flow thru the
chest wall defect. (Sucking chest wound)
27. Diagnosis
• Open chest wound
• Respi distress
• Nisy air movement over chest wall
• Decreased breathing sound of affected side
• Hyper resonance
• Crepitus, tenderness
28. Treatment
• 3 way secure occlusive dressing ( temporary)
-> Disable air entry toward pleural space during
inspiration.
• Chest tube and seal the opening – dressing –
seal 4 ways or repair surgically.
29. Massive Hemothorax
Defination:
1) Rapid accumulation of > 1.5 L of blood
(initially) or more than 1/3 of patients total
Blood volume in the thoracic intra pleural space
chest cavity.
2) Continuous blood loss of 200 ml/hr for 2-4 hrs
32. Diagnosis
• Bruises/laceration/contusion to chest wall
• Distended neck vein – early
-> displacement of mediastinum due to massive hemothorax
• Collapsed Neck vein
-> severe hypovolemia – shock
• Reduced chest wall expansion
• Respi distress
• Decreased Spo2
• Decreased breathing sound
• Dullness
• Tachycardia, Hypotension, Shock
• EFAST: collection in lungs, +ve spine sign, -ve curtain sign
33. Treatment
Principle: Hemostatic resus and Thoracic decompression
• Activate MTP
• GXM – emergency cross match / Safe O/Tranexamic acid
• Chest Tube
• Get Primary( Surg) Team involved Early
• Indication for urgent thoracotomy
-> > 1.5L of blood drain in chest tube
-> continuous bleeding of 200 ml/ hr for 2-4 hrs
-> penetrating injury to region of mediastinal box, possibility
of cardiac, major vessel and hilar structural injury.
35. Pathophysio
• Blunt trauma
-> Potential shearing injuries occurring at
junction where fixed structure meets relative
mobile segment. ( Point of attachment )
- Areas near the cricoid cartilage and carina are
fixed to the thyroid cartilage and
the pericardium.
36. 2) Blast injury - causes severe injury at air fluid
interface – alveoli
3) Penetrating Trauma
- Direct laceration, tear or transfer of kinetic
injury with cavitation
37. When to suspect?
• Hemoptysis
• Respi distress
• S/c Emphysema over neck
• Tension pneumothorax
• Persistent pneumothorax despite on chest tube
• Continuous chest tube bubbling and multiple
chest tube required
Diagnostic -> Bronchoscopy
38. Treatment
• Definitive airway needed.
- > get Surgical and ENT / Anest team early
- > Fiberoptic intubation to intubate unaffected
bronchus
- Surgery
39. Cardiac tamponade
Definition: accumulation of fluid within the
pericardial sac -> increased intrapericardial
pressure -> reduced venous return due to
compromise of Right Ventricular Diastolic and
inflow function -> Reduced CO. ( Obstructive
Shock)
- 150 mls can cause cardiac tamponade
41. Diagnosis
• Beck Triad : Muffled Heart sound,
hypotension, Distended JV
• PEA
• Adjunct: E-FAST: echo for accurate diagnosis
-> collection over pericardium
-> collapsed RV during diastolic
-> distended IVC
42. Treatment
Pericardiocentesis
- To relieve the tamponade
Approaches
1) Subxiphoid ( Blind)
The needle insertion site is between the
xiphisternum and left costal margin. Once
beneath the cartilage cage, lower the needle to
a 15-to-30-degree angle, with the abdominal
wall directed towards the left shoulder.
43. Parasternal
Insertion site is in the fifth left intercostal space
close to the sternal margin. Advance the needle
perpendicular to the skin (at the level of the cardiac
notch of the left lung). Avoid the internal mammary
artery.
Apical
Needle insertion site is 1-2 cm lateral to the apex
beat within the fifth, sixth or seventh intercostal
space. Advance the needle over the superior border
of the rib to avoid intercostal nerves and vessels.
45. Flail Chest & pulmonary contusion
Definition: 3 or more adjacent rib fracture in at
least 2 or more places.
This causes ribs being separated and paradoxical
movements during inspiration.
-> flail segment moves inwards and during exp,
moves outwards.
46. Pulmonary contusion is the bruising of the lung
causing accumulation of blood/fluids – leading
to inadequate oxygenation, ventilation and V/Q
mismatch.
-> commonly associated with rib fracture
47.
48.
49.
50.
51. Diagnosis
Paradoxical movement of flail segment
Inadequate respiratory effort – due to pain
Tenderness and crepitation on palpation
Adjunct: CXR – rib fracture, lung contusion,
hemo/pneumothorax
52. Management
• Analgesia : Iv morphine, PCA/regional block
• Oxygenation Supplement
• Ventilation – Non invasive / Intubation
• w/o hypoxia, co2 retention, reduced breathing
effort
53. Esophageal injury
Def: Injury to the Esophagus
-> Mechanism
1) Penerating – common
2) Blunt – rare
- Forceful expulsion of gastric content into
esophagus – produced linear tear and leaking
into mediastinum. – causing mediastinitis
54.
55. Diagnosis
• Pain and shock that is out of porportion to
other obvious injuries
• Presence of left pneumo/hemothorax without
clear evidence of rib fracture – drainage may
have gastric content.
• Pneumomediastinum
• S/c Emphysema
57. Traumatic Diaphragmatic Injury (TDR)
• More common to the left side
• Suspected when Bowel shadow seen in
thoracic cavity on CXR
Mechanism
-> Blunt – may produce large tears
-> Penetrating – Small perforation that maybe
Asymtomatic
61. Definitive treatment is surgery.
Complications:
Pulmonary compromise
Strangulation or entrapment of peritoneal
contents
62. • Why Finger thoracocentesis prefered than
needle thoracocentesis?
- The latter is a blind procedure
- Success rate influenced by chest wall thickness
- Risk of iatrogenic injuries – pneumothorax or
injury to lung tissue in cases of wrongly
diagnose of clinical tension pneumothorax.
63. Finger and tUbe thoracostomy
• Indication:-
-> rapid thoracic decompression in tension
pneumothorax
-> Actual/near traumatic cardiac arrest
-> shocked state with no apparent cause in
trauma patient
64. Safety Triange
• Ant border: Lateral
boerfer of
pectoralis major
muscle
• Lateral brder: Mid
axillary Line
• Inferiror border: 5th
ICS
65. Steps
1. Indication for finger thoraco
2. Locate the safety triangle
3. Clean the area with povidone / chlorhexidine
4. Local Analgesia
5. On Sterile gloves
6. Scaplel no 10/11, make a 4cm incision thru
the skin to the superior border of inferior
ribs.
66.
67. 7. Use blunt forceps and dissect thru the muscles
and tissue – non stabbing manner
8. Feel the gift – a gush of air/blood/fluids
9. Remove blunt forceps and insert finger in the
tract and confirm the intrapleural space by
palpation and sweep.
10. Convert to Chest tube , use clamp and guide the
tip of chest tube into the tract , advance to desired
depth and connect to under water seal.
11. Secure the tube with sutures and apply sterile
dressing
12. Post procedure cxr and reassest patient.