GRACIOUS COLLGE OF NURSING
ABHANPUR , RAIPUR
GLAUCOMA
GLAUCOMA
PRESENTED BY
OM VERMA
ASSISTANT PROFESSOR
Pressures of between 11 and
21 mmHg are considered
normal, can exceed 70 mm Hg
normal, can exceed 70 mm Hg
in some glaucoma's.
DEFINITION
DEFINITION
“A disease of the eye in which the pressure of
fluid inside the eyeball is abnormally high,
caused by obstructed outflow of the fluid.
The increased pressure can damage the optic
The increased pressure can damage the optic
nerve and lead to partial or complete loss of
vision.”
According to luckmans
DEFINITION
Glaucoma is a group of ocular conditions
characterized by optic nerve damage. The
optic nerve damage is related to the intra
ocular pressure (IOP) caused by congestion
ocular pressure (IOP) caused by congestion
of aqueous humor in the eye.
According to Brunner & Suddarth’s
TYPES
CONGENITAL
It is characterized by elevation of intra ocular
pressure (IOP) associated with developmental
abnormalities of the angle of anterior chamber
depending upon the age of onset.
This developmental glaucoma's are termed as :
1. True or primary congenital glaucoma (IOP is raised
during intrauterine life)
2. Infantile glaucoma's (Diseases manifests prior to the
child)
3. Juvenile glaucoma (Children develop IOP pressure
rise between 3-16 yrs of life)
ACQUIRED
Glaucoma is defined as an acquired
loss of retinal ganglion cells and
axons within the optic nerve or optic
neuropathy that results in a
neuropathy that results in a
characteristic optic nerve head
appearance and a corresponding
progressive loss of vision.
ACQUIRED –
1. PRIMARY
2. SECONDARY
2. SECONDARY
ACQUIRED –
1. PRIMARY DIVIDED 2 TYPES
• Open angle/ Wide angle/ Chronic
• Angle Closure/Narrow angle/Closed
angle
OPEN ANGLE GLAUCOMA
1. Primary chronic open angle glaucoma
(COAG)
2. Normal tension (pressure) glaucoma or
low tension glaucoma
3. Childhood glaucoma
4. Secondary open angle glaucoma
5. Pigmentary glaucoma
6. Exfoliative glaucoma
OPEN ANGLE GLAUCOMA
1.Primary chronic open angle glaucoma
(COAG)
• It is the most common type of the
glaucoma. Its frequency increases greatly
with age. The aqueous fluid does not drain
with age. The aqueous fluid does not drain
from the eye properly. The pressure within
the eye, therefore, builds up painlessly and
without symptoms.
•
OPEN ANGLE GLAUCOMA
2. Normal tension (pressure) glaucoma or
low tension glaucoma :-
• This type of glaucoma is thought to be due
to decreased blood flow to the optic nerve.
This condition is characterized by
This condition is characterized by
progressive optic-nerve damage and loss of
peripheral vision (visual field) despite
intraocular pressures in the normal range or
even below normal.
OPEN ANGLE GLAUCOMA
3. Childhood glaucoma :-
• Childhood glaucoma is an uncommon
pediatric condition often associated with
significant visual loss.Itmay most commonly
significant visual loss.Itmay most commonly
be caused by trauma, surgery or other
acquired or secondary causes or abnormal
increase intra ocular pressure.
OPEN ANGLE GLAUCOMA
4. Secondary open angle glaucoma :-
• It can result from an eye (ocular)
injury,inflammation in the iris (iritis),retinal
injury,inflammation in the iris (iritis),retinal
vein blockage etc.
OPEN ANGLE GLAUCOMA
5. Pigmentary glaucoma :-
• In thisgranules of pigment detach from
theiris,which is the colored part of the
eye.These granules then may block the
trabecular meshwork, is a key element in
trabecular meshwork, is a key element in
the drainage system of the eye.Finally the
blocked drainage system leads to elevated
intraocular pressure which results in
damage to the optic nerve.
OPEN ANGLE GLAUCOMA
• Exfoliative glaucoma :-
• This type of glaucoma is characterized by
deposits of flaky material on the front
surface of the lens (anterior capsule) & in
the angle of the eye.The accumulation of
the angle of the eye.The accumulation of
this material in the angle is believed to
block the drainage system of the eye and
raise the eye pressure.
ANGLE CLOSURE GLAUCOMA
1. Acute angle closure glaucoma
2. Chronic angle closure glaucoma
Angle-closure glaucoma
• Angle-closure glaucoma may be acute or
chronic.The common element in both is
that all of the drainage angle becomes
anatomically closed,so that the aqueous
anatomically closed,so that the aqueous
fluid within the eye cannot even reach all
or part of the trabecular meshwork.
1. Acute angle closure glaucoma
• When the drainage angle of the eye suddenly
becomes completely blocked,pressure builds
up rapidly, and this is called acute angle-
closure glaucoma. The symptoms include
closure glaucoma. The symptoms include
Acute angle-closure glaucoma presents as a
sudden onset of severe unilateral eye pain or
a headache associated with blurred vision,
rainbow-colored halos around bright lights,
nausea, and vomiting.
2. Chronic angle closure glaucoma :-
• Chronic closed-angle glaucoma, also called chronic
angle-closure glaucoma, is a condition that can be a
cause of permanent vision loss if not diagnosed and
managed correctly. When the drainage angle of the
eye gradually becomes completely blocked, pressure
eye gradually becomes completely blocked, pressure
builds up gradually, and this is called chronic angle-
closure glaucoma. The drainage tissues gradually start
to scar. This condition is generally silent, and severe
glaucoma damage can occur without the person's
knowledge.
•
ETIOLOGY
1
Antihistamines
SLEEP APNEA
• sleep apnea: it tends to take longer for your
tears to be replenished, you're more likely to
have ocular irritation suffer from sleep apnea
are more likely to develop glaucoma low
are more likely to develop glaucoma low
oxygen concentration in the blood may
contribute to degradation of the optic nerve,
potentially leading to glaucoma.
Antihistamines drug used
• Many oral antihistamines have been
implicated in acute angle closure attacks in
patients with narrowed aqueous
circulation (e.g., narrow-angle glaucoma)
circulation (e.g., narrow-angle glaucoma)
RISK FACTORS
CLINICAL MANIFESTATION
PATHOPHYSIOLOGY :-
1. Initiating Events :- Precipitating factors include
illness,emotional stress,congenital narrow angles,
long term use of corticosteroids & mydriatics
(medications causing papillary dilation).These
events lead to second stage.
events lead to second stage.
2. Structural alterations in the aqueous outflow
system :- Tissue & cellular changes caused by
factors that affect aqueous humor dynamics lead
to structural alterations & to the third stage.
3. Functional alterations :- Conditions such as
increased IOP or impaired blood flow create
functional changes that lead to fourth stage.
4. Optic nerve damage :- Atrophy of the optic
nerve is characterized by loss of nerve fibers &
blood supply & this fourth stage inevitably
blood supply & this fourth stage inevitably
progresses to the fifth stage.
5. Vision loss :- Progressive loss of vision is
characterized by visual field defects.
• ASSESSMENT & DIAGNOSTIC
EVALUATION
ASSESSMENT & DIAGNOSTIC
EVALUATION
1. History taking
2. Tonometry
3. Ophthalmoscopy
4. Gonioscopy
4. Gonioscopy
5. Perimetry
6. Pachymetry
7. Dilated pupil
examination
• Optical coherence tomography (OCT) The
noninvasive method produces an image by
measuring the amount of a dim red light that
reflects off of retina and optic nerve. Optical
reflects off of retina and optic nerve. Optical
coherence tomography can measure the
thickness of retina and optic nerve. to look for
changes in your optic nerve that may indicate
glaucoma.
• Ophthalmoscopy is an examination of the
back part of the eye (fundus), which includes
the retina , optic disc, choroid , and blood
vessels.
• A perimetry test can help find certain
patterns of vision loss. This may mean a
certain type of eye disease is present. It is very
useful in finding early changes in vision caused
by nerve damage from glaucoma
• Ocular pressure test (tonometry) to measure
eye pressure.
• Pachymetry to measure corneal thickness.
• Slit-lamp exam to examine the inside of your
eye with a special microscope called a slit
lamp.
lamp.
• Visual acuity test (eye charts) to check for
vision loss.
• Dilated eye exam to widen pupils and view
your optic nerve at the back of your eyes.
PHARMACOLOGICAL MANAGEMENT
1. Cholinergics (Miotics): Pilocarpine,Carbachol
(1%,2% or 4%/3-4 times/day)-It increases
aqueous fluid outflow by contracting the ciliary
muscles & causing constriction of the pupil &
opening of the trabecular meshwork.
opening of the trabecular meshwork.
2. Adrenergic agonists : Dipivefrin,Epinephrine
(0.5%, 1% or 2%/1-2 times/day)-Reduces
production of aqueous humor & increases outflow.
3. Beta blockers : Betaxolol,Timolol (0.25 or
0.5%/2 times/day)-Decreases aqueous humor
production.
4. Alpha adrenergic agonists : Apraclonidin,
Brimonidine (0.5%/2-3 times/day)-Decreases
Brimonidine (0.5%/2-3 times/day)-Decreases
aqueous humor production.
5. Carbonic anhydrase inhibitors :
Acetazolamide, methazolamide (250 mg / tds
Decreases aqueous humor production.
SURGICAL MANAGEMENT
:-
( for papillary block glaucoma)
3. Filtering procedures :- To
create an opening or fistula in
the trabecular meshwork to
drain aqueous humor.
4. Trabeculectomy :- It is the
standard filtering technique
used to remove part of the
trabecular meshwork.
• . 5. Drainage implants or shunts :- an open
tubes implanted in the anterior chamber to
shunt aqueous humor to an attached place in
the conjunctival space
• A fibrous capsule develops around the
episcleral plate & filters the aqueous
humor,thereby regulating the outflow &
humor,thereby regulating the outflow &
controlling IOP.
6. Canaloplasty :- a micro-
catheter or tube placed in the Canal of
Schlemm (the natural
site of drainage for healthy eyes)
to enlarge the drainage canal. ,
relieving pressure inside the eye.
7. Diode laser cycloablation :-
When trabeculectomy or
When trabeculectomy or
glaucoma drainage tube has failed
then they may consider
cycloablation.it involves permanent
destruction of the ciliary body. ( which
produces the aqueous fluid ) it is
usually the last line of treatment for
uncontrolled glaucoma.
NURSING MANAGEMENT
1) Pre-operative care
2) Post-operative care
Administered Opioids,antiemetics, antibiotics as
directed.
Reassure patient that,with reduction in IOP,pain
Reassure patient that,with reduction in IOP,pain
& other sign & symptoms should subside.
Provide reassurance & calm presence to reduce
anxiety & fear.
Provide knowledge regarding glaucoma,their
sign & symptoms & Management.
COMPLICATION
• GLAUCOMA SURGERIES INCLUDE
• CHOROIDAL DETACHMENT;
• OCULAR DECOMPRESSION RETINOPATHY
• HAEMORRHAGIC CHOROIDAL DETACHMENT
• HAEMORRHAGIC CHOROIDAL DETACHMENT
• HYPOTONY MACULOPATHY
• VITREOUS HAEMORRHAGE
• RETINAL DETACHMENT.
• Choroidal detachments occur when there is
an accumulation of fluid or blood in the
suprachoroidal space, a potential space situated
between the choroid and the sclera.
• Ocular decompression retinopathy (ODR) is an
rare postoperative complication that occurs as a
rare postoperative complication that occurs as a
result of rapid intraocular pressure lowering
interventions. The clinical appearance is
characterized by multiple
round intraretinal hemorrhages, some with white
centers,
• Hemorrhagic choroidal detachment is a
hemorrhage in the suprachoroidal space or
within the choroid caused by the rupture of
choroidal vessels.
• Hypotony maculopathy is characterized by a
• Hypotony maculopathy is characterized by a
low IOP associated with fundus abnormalities,
including chorioretinal folds, optic nerve head
edema
• Vitreous hemorrhage is seen as blood floating in
vitreous, occluding the view of retina variably.
• Retinal detachment describes an emergency
situation in which a thin layer of tissue (the
situation in which a thin layer of tissue (the
retina) at the back of the eye pulls away from its
normal position. Retinal detachment separates
the retinal cells from the layer of blood vessels
that provides oxygen and nourishment to the
eye.
slide for glaucoma.pdf

slide for glaucoma.pdf

  • 1.
    GRACIOUS COLLGE OFNURSING ABHANPUR , RAIPUR GLAUCOMA GLAUCOMA PRESENTED BY OM VERMA ASSISTANT PROFESSOR
  • 2.
    Pressures of between11 and 21 mmHg are considered normal, can exceed 70 mm Hg normal, can exceed 70 mm Hg in some glaucoma's.
  • 3.
  • 4.
    DEFINITION “A disease ofthe eye in which the pressure of fluid inside the eyeball is abnormally high, caused by obstructed outflow of the fluid. The increased pressure can damage the optic The increased pressure can damage the optic nerve and lead to partial or complete loss of vision.” According to luckmans
  • 5.
    DEFINITION Glaucoma is agroup of ocular conditions characterized by optic nerve damage. The optic nerve damage is related to the intra ocular pressure (IOP) caused by congestion ocular pressure (IOP) caused by congestion of aqueous humor in the eye. According to Brunner & Suddarth’s
  • 6.
  • 7.
    CONGENITAL It is characterizedby elevation of intra ocular pressure (IOP) associated with developmental abnormalities of the angle of anterior chamber depending upon the age of onset. This developmental glaucoma's are termed as : 1. True or primary congenital glaucoma (IOP is raised during intrauterine life) 2. Infantile glaucoma's (Diseases manifests prior to the child) 3. Juvenile glaucoma (Children develop IOP pressure rise between 3-16 yrs of life)
  • 8.
    ACQUIRED Glaucoma is definedas an acquired loss of retinal ganglion cells and axons within the optic nerve or optic neuropathy that results in a neuropathy that results in a characteristic optic nerve head appearance and a corresponding progressive loss of vision.
  • 9.
    ACQUIRED – 1. PRIMARY 2.SECONDARY 2. SECONDARY
  • 10.
    ACQUIRED – 1. PRIMARYDIVIDED 2 TYPES • Open angle/ Wide angle/ Chronic • Angle Closure/Narrow angle/Closed angle
  • 11.
    OPEN ANGLE GLAUCOMA 1.Primary chronic open angle glaucoma (COAG) 2. Normal tension (pressure) glaucoma or low tension glaucoma 3. Childhood glaucoma 4. Secondary open angle glaucoma 5. Pigmentary glaucoma 6. Exfoliative glaucoma
  • 12.
    OPEN ANGLE GLAUCOMA 1.Primarychronic open angle glaucoma (COAG) • It is the most common type of the glaucoma. Its frequency increases greatly with age. The aqueous fluid does not drain with age. The aqueous fluid does not drain from the eye properly. The pressure within the eye, therefore, builds up painlessly and without symptoms. •
  • 13.
    OPEN ANGLE GLAUCOMA 2.Normal tension (pressure) glaucoma or low tension glaucoma :- • This type of glaucoma is thought to be due to decreased blood flow to the optic nerve. This condition is characterized by This condition is characterized by progressive optic-nerve damage and loss of peripheral vision (visual field) despite intraocular pressures in the normal range or even below normal.
  • 14.
    OPEN ANGLE GLAUCOMA 3.Childhood glaucoma :- • Childhood glaucoma is an uncommon pediatric condition often associated with significant visual loss.Itmay most commonly significant visual loss.Itmay most commonly be caused by trauma, surgery or other acquired or secondary causes or abnormal increase intra ocular pressure.
  • 15.
    OPEN ANGLE GLAUCOMA 4.Secondary open angle glaucoma :- • It can result from an eye (ocular) injury,inflammation in the iris (iritis),retinal injury,inflammation in the iris (iritis),retinal vein blockage etc.
  • 16.
    OPEN ANGLE GLAUCOMA 5.Pigmentary glaucoma :- • In thisgranules of pigment detach from theiris,which is the colored part of the eye.These granules then may block the trabecular meshwork, is a key element in trabecular meshwork, is a key element in the drainage system of the eye.Finally the blocked drainage system leads to elevated intraocular pressure which results in damage to the optic nerve.
  • 17.
    OPEN ANGLE GLAUCOMA •Exfoliative glaucoma :- • This type of glaucoma is characterized by deposits of flaky material on the front surface of the lens (anterior capsule) & in the angle of the eye.The accumulation of the angle of the eye.The accumulation of this material in the angle is believed to block the drainage system of the eye and raise the eye pressure.
  • 18.
    ANGLE CLOSURE GLAUCOMA 1.Acute angle closure glaucoma 2. Chronic angle closure glaucoma
  • 19.
    Angle-closure glaucoma • Angle-closureglaucoma may be acute or chronic.The common element in both is that all of the drainage angle becomes anatomically closed,so that the aqueous anatomically closed,so that the aqueous fluid within the eye cannot even reach all or part of the trabecular meshwork.
  • 20.
    1. Acute angleclosure glaucoma • When the drainage angle of the eye suddenly becomes completely blocked,pressure builds up rapidly, and this is called acute angle- closure glaucoma. The symptoms include closure glaucoma. The symptoms include Acute angle-closure glaucoma presents as a sudden onset of severe unilateral eye pain or a headache associated with blurred vision, rainbow-colored halos around bright lights, nausea, and vomiting.
  • 21.
    2. Chronic angleclosure glaucoma :- • Chronic closed-angle glaucoma, also called chronic angle-closure glaucoma, is a condition that can be a cause of permanent vision loss if not diagnosed and managed correctly. When the drainage angle of the eye gradually becomes completely blocked, pressure eye gradually becomes completely blocked, pressure builds up gradually, and this is called chronic angle- closure glaucoma. The drainage tissues gradually start to scar. This condition is generally silent, and severe glaucoma damage can occur without the person's knowledge. •
  • 22.
  • 23.
    SLEEP APNEA • sleepapnea: it tends to take longer for your tears to be replenished, you're more likely to have ocular irritation suffer from sleep apnea are more likely to develop glaucoma low are more likely to develop glaucoma low oxygen concentration in the blood may contribute to degradation of the optic nerve, potentially leading to glaucoma.
  • 24.
    Antihistamines drug used •Many oral antihistamines have been implicated in acute angle closure attacks in patients with narrowed aqueous circulation (e.g., narrow-angle glaucoma) circulation (e.g., narrow-angle glaucoma)
  • 25.
  • 26.
  • 27.
    PATHOPHYSIOLOGY :- 1. InitiatingEvents :- Precipitating factors include illness,emotional stress,congenital narrow angles, long term use of corticosteroids & mydriatics (medications causing papillary dilation).These events lead to second stage. events lead to second stage. 2. Structural alterations in the aqueous outflow system :- Tissue & cellular changes caused by factors that affect aqueous humor dynamics lead to structural alterations & to the third stage.
  • 28.
    3. Functional alterations:- Conditions such as increased IOP or impaired blood flow create functional changes that lead to fourth stage. 4. Optic nerve damage :- Atrophy of the optic nerve is characterized by loss of nerve fibers & blood supply & this fourth stage inevitably blood supply & this fourth stage inevitably progresses to the fifth stage. 5. Vision loss :- Progressive loss of vision is characterized by visual field defects.
  • 29.
    • ASSESSMENT &DIAGNOSTIC EVALUATION
  • 30.
    ASSESSMENT & DIAGNOSTIC EVALUATION 1.History taking 2. Tonometry 3. Ophthalmoscopy 4. Gonioscopy 4. Gonioscopy 5. Perimetry 6. Pachymetry 7. Dilated pupil examination
  • 31.
    • Optical coherencetomography (OCT) The noninvasive method produces an image by measuring the amount of a dim red light that reflects off of retina and optic nerve. Optical reflects off of retina and optic nerve. Optical coherence tomography can measure the thickness of retina and optic nerve. to look for changes in your optic nerve that may indicate glaucoma.
  • 32.
    • Ophthalmoscopy isan examination of the back part of the eye (fundus), which includes the retina , optic disc, choroid , and blood vessels. • A perimetry test can help find certain patterns of vision loss. This may mean a certain type of eye disease is present. It is very useful in finding early changes in vision caused by nerve damage from glaucoma
  • 33.
    • Ocular pressuretest (tonometry) to measure eye pressure. • Pachymetry to measure corneal thickness. • Slit-lamp exam to examine the inside of your eye with a special microscope called a slit lamp. lamp. • Visual acuity test (eye charts) to check for vision loss. • Dilated eye exam to widen pupils and view your optic nerve at the back of your eyes.
  • 34.
    PHARMACOLOGICAL MANAGEMENT 1. Cholinergics(Miotics): Pilocarpine,Carbachol (1%,2% or 4%/3-4 times/day)-It increases aqueous fluid outflow by contracting the ciliary muscles & causing constriction of the pupil & opening of the trabecular meshwork. opening of the trabecular meshwork. 2. Adrenergic agonists : Dipivefrin,Epinephrine (0.5%, 1% or 2%/1-2 times/day)-Reduces production of aqueous humor & increases outflow.
  • 35.
    3. Beta blockers: Betaxolol,Timolol (0.25 or 0.5%/2 times/day)-Decreases aqueous humor production. 4. Alpha adrenergic agonists : Apraclonidin, Brimonidine (0.5%/2-3 times/day)-Decreases Brimonidine (0.5%/2-3 times/day)-Decreases aqueous humor production. 5. Carbonic anhydrase inhibitors : Acetazolamide, methazolamide (250 mg / tds Decreases aqueous humor production.
  • 36.
  • 37.
    :- ( for papillaryblock glaucoma)
  • 38.
    3. Filtering procedures:- To create an opening or fistula in the trabecular meshwork to drain aqueous humor. 4. Trabeculectomy :- It is the standard filtering technique used to remove part of the trabecular meshwork.
  • 39.
    • . 5.Drainage implants or shunts :- an open tubes implanted in the anterior chamber to shunt aqueous humor to an attached place in the conjunctival space • A fibrous capsule develops around the episcleral plate & filters the aqueous humor,thereby regulating the outflow & humor,thereby regulating the outflow & controlling IOP.
  • 40.
    6. Canaloplasty :-a micro- catheter or tube placed in the Canal of Schlemm (the natural site of drainage for healthy eyes) to enlarge the drainage canal. , relieving pressure inside the eye. 7. Diode laser cycloablation :- When trabeculectomy or When trabeculectomy or glaucoma drainage tube has failed then they may consider cycloablation.it involves permanent destruction of the ciliary body. ( which produces the aqueous fluid ) it is usually the last line of treatment for uncontrolled glaucoma.
  • 41.
    NURSING MANAGEMENT 1) Pre-operativecare 2) Post-operative care Administered Opioids,antiemetics, antibiotics as directed. Reassure patient that,with reduction in IOP,pain Reassure patient that,with reduction in IOP,pain & other sign & symptoms should subside. Provide reassurance & calm presence to reduce anxiety & fear. Provide knowledge regarding glaucoma,their sign & symptoms & Management.
  • 42.
    COMPLICATION • GLAUCOMA SURGERIESINCLUDE • CHOROIDAL DETACHMENT; • OCULAR DECOMPRESSION RETINOPATHY • HAEMORRHAGIC CHOROIDAL DETACHMENT • HAEMORRHAGIC CHOROIDAL DETACHMENT • HYPOTONY MACULOPATHY • VITREOUS HAEMORRHAGE • RETINAL DETACHMENT.
  • 43.
    • Choroidal detachmentsoccur when there is an accumulation of fluid or blood in the suprachoroidal space, a potential space situated between the choroid and the sclera. • Ocular decompression retinopathy (ODR) is an rare postoperative complication that occurs as a rare postoperative complication that occurs as a result of rapid intraocular pressure lowering interventions. The clinical appearance is characterized by multiple round intraretinal hemorrhages, some with white centers,
  • 44.
    • Hemorrhagic choroidaldetachment is a hemorrhage in the suprachoroidal space or within the choroid caused by the rupture of choroidal vessels. • Hypotony maculopathy is characterized by a • Hypotony maculopathy is characterized by a low IOP associated with fundus abnormalities, including chorioretinal folds, optic nerve head edema
  • 45.
    • Vitreous hemorrhageis seen as blood floating in vitreous, occluding the view of retina variably. • Retinal detachment describes an emergency situation in which a thin layer of tissue (the situation in which a thin layer of tissue (the retina) at the back of the eye pulls away from its normal position. Retinal detachment separates the retinal cells from the layer of blood vessels that provides oxygen and nourishment to the eye.