Pneumonia is a serious infection that inflames the air sacs in the lungs. It can cause symptoms such as coughing, chest pain, fever, and difficulty breathing. It's important to seek medical attention if you suspect you have pneumonia. Here are some notes for dear medical students, i hope it helps you..
Asthma is a condition in which your airways narrow and swell and produce extra mucus. This can make breathing difficult and trigger coughing, wheezing and shortness of breath. For some people, asthma is a minor nuisance.
Bronchial Asthma: Definition,Pathophysiology and ManagementMarko Makram
Definition and Pathophysiology of Asthma in addition to classification and recent updates in the management of asthma based on GINA-2019 Guidelines, by Dr. Marco Makram.
Asthma is a condition in which your airways narrow and swell and produce extra mucus. This can make breathing difficult and trigger coughing, wheezing and shortness of breath. For some people, asthma is a minor nuisance.
Bronchial Asthma: Definition,Pathophysiology and ManagementMarko Makram
Definition and Pathophysiology of Asthma in addition to classification and recent updates in the management of asthma based on GINA-2019 Guidelines, by Dr. Marco Makram.
Acute and Chronic Bronchitis is amongst most common presenting illness for Family Physicians considering its prevalence in all ages. Revisiting it with perspective of a family physician helps improve understanding and management at Family PRactice
Bronchitis is an inflammation of the bronchial tubes, the airways that carry air to your lungs. It causes a cough that often brings up mucus. It can also cause shortness of breath, wheezing, a low fever, and chest tightness. There are two main types of bronchitis: acute and chronic
Slides are prepared as per INC Syllabus Unit V Drugs used on Respiratory systems and it is most benefited for 2nd yr B sc Nursing students and faculty of the subject.
Community Acquired Pneumonia and other types of pneumonia
for medical students
Detailed information on pneumonia including the following
Definition
Classification
Aetiology
Pathogenesis
Pathological states
Investigations
Treatment & follow up
Complications
Medication
Hospital acquired pneumonia and it’s treatment and management and prevention
Other types of pneumonia
And pneumonia in immune compromised patients
Acute and Chronic Bronchitis is amongst most common presenting illness for Family Physicians considering its prevalence in all ages. Revisiting it with perspective of a family physician helps improve understanding and management at Family PRactice
Bronchitis is an inflammation of the bronchial tubes, the airways that carry air to your lungs. It causes a cough that often brings up mucus. It can also cause shortness of breath, wheezing, a low fever, and chest tightness. There are two main types of bronchitis: acute and chronic
Slides are prepared as per INC Syllabus Unit V Drugs used on Respiratory systems and it is most benefited for 2nd yr B sc Nursing students and faculty of the subject.
Community Acquired Pneumonia and other types of pneumonia
for medical students
Detailed information on pneumonia including the following
Definition
Classification
Aetiology
Pathogenesis
Pathological states
Investigations
Treatment & follow up
Complications
Medication
Hospital acquired pneumonia and it’s treatment and management and prevention
Other types of pneumonia
And pneumonia in immune compromised patients
Pneumonia is an inflammation of the lung parenchyma caused by various microorganisms, including bacteria, mycobacteria, fungi, and viruses.
Pneumonitis is a more general term that describes the inflammatory process in the lung tissue that may predispose and Pneumonia is an inflammation of the lung parenchyma that is caused by a microbial agent.
place the patient at risk for microbial invasion.
Pneumonia is classified into four: community-acquired pneumonia (CAP) and hospital-acquired pneumonia (HAP), pneumonia in the immunocompromised host, and aspiration pneumonia.
PNEUMONIA,
DEFINITION
Pneumonia is an infection of the pulmonary parenchyma.
To the pathologist, pneumonia is an infection of the alveoli ,distal airways, and interstitium of the lung that is manifested by increased weight of the lungs, replacement of normal lung’s sponginess by consolidation ,and alveoli filled with white blood cells ,red blood cells and fibrin .To the clinician, pneumonia is a constellation of symptoms and signs in combination with at least one opacity on CXR.
Epidemiology
Between 5 and 10 million cases of infectious pneumonia occur annually in the United States and result in more than 1 million hospitalizations.
Pneumonia is a leading cause of death worldwide, the sixth leading cause of death in the United States, and the most common lethal infectious disease.
Pneumonia is a leading cause of illness and death in Nepal, particularly among young children and the elderly. This PowerPoint presentation provides a comprehensive overview of pneumonia in Nepal, including the causes, symptoms, risk factors, and treatment options.
Through powerful images and personal stories, we showcase the impact of pneumonia on individuals, families, and communities in Nepal. We highlight the challenges of accessing healthcare in remote and impoverished areas, the lack of awareness and education about the disease, and the importance of early diagnosis and treatment.
The presentation provides detailed information about the various types of pneumonia and the risk factors associated with each. We also discuss the diagnostic procedures, including chest x-rays and blood tests, and the treatment options, such as antibiotics and oxygen therapy.
In addition, we explore the efforts being made to prevent and control pneumonia in Nepal. We highlight the importance of vaccination, particularly among children and high-risk groups, and the role of community-based interventions in improving access to healthcare and promoting healthy behaviors.
Through this PowerPoint presentation, we aim to raise awareness about pneumonia in Nepal and the importance of early diagnosis and treatment. We showcase the latest research and innovations in pneumonia prevention and treatment, and the importance of collaboration and partnership to address the disease.
We urge the audience to take action in the fight against pneumonia, whether it be through spreading awareness, supporting organizations working on the ground, or advocating for policy change. Let us come together to create a world where no one has to suffer from the devastating effects of pneumonia.
Otitis externa, commonly known as swimmer's ear, is a painful condition characterized by inflammation of the external ear canal. It often occurs due to infection or irritation and can be described as follows:
Otitis externa is an inflammatory condition of the ear canal, typically caused by water exposure, bacterial or fungal infections, or skin conditions. Common symptoms include ear pain, itching, redness, and swelling of the ear canal. Discharge, temporary hearing loss, and discomfort when moving the earlobe or jaw may also occur. Prompt medical attention is advisable to prevent complications and alleviate symptoms.
In this slideshare we will se about GERD , i hope it helps u .
Gastroesophageal reflux diseases (GERD) is not a disease but a heterogenous syndrome resulting from esophageal reflux. Most cases are attributed to the inappropriate relaxation of lower esophageal sphincter (LES) in response to unknown stimulus.
It is a short description or short notes on ards, know we can easily know about this superficially.
It is a condition where in the alveoli, the alveoli is filled with fluid and then the gas exchange can't be done properly..
This is the notes of CORYNEBACTERIUM which is helpful to paramedical and medical students. In this notes the bacteriology of CORYNEBACTERIUM is given. Best of your luck and read this.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
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micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
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TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
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PNEUMONIA.pdf
1. PNEUMONIA
Definition :-
•Pneumonia or pneumonitis is an acute inflammation of the lung
tissue (lung parenchyma).
Etiology:-
-Pneumonia is resulting from inhalation or transport via the
blood-stream of infectious agents or noxious
fumes or from radiation treatment.
- Pneumonia is classified according to whether infection was
acquired in the community or in the hospital.
2. -Thus pneumoniais classified as “community acquired
pneumonia(CAP) or hospital acquired pneumonia (HAP).
-HAP is also called nosocomial pneumonia.
●The causes of pneumonia are as follows.
i. Community-acquired pneumonia caused by:
• Streptococcus pneumoniae
• Hemophilus influenzae
• Mycoplasma pneumoniae
• Respiratory viruses
4. • Enterobacter
• Escherichia coli
• Proteus
• Klebsiella
• Staphylococcus aureus
• Streptococcus pneumoniae
• Oral anaerobes.
The risk factors which predispose to pneumonia
are:
5. • Smoking
• Air pollution
• Altered consciousness: Alcoholism, head injury,
seizures, anesthesia, drug overdose
• Tracheal intubation (endotracheal intubations,
tracheostomy)
• Upper respiratory tract infection
• Chronic diseases: Chronic lung diseases, diabetes
mellitus, heart disease, uremia, cancer
• Immunosuppression
– Drugs (carciosteroids, cancer chemotherapy,
immunosuppressive therapy after organ
transplant)
– HIV
• Malnutrition
6. • Inhalation of aspiration of noxious substances
• Debilitating illness
• Bedrest and prolonged immobility
• Altered oropharyngeal flora.
The risk factors for hospital acquired pneumonia
are:
• Residence in an ICU
• Mechanical ventilation (those who required 48
hours or more ventilation)
• Endotracheal intubation or tracheostomy
• Recent surgery
• Debilitation, i.e. malnutrition
• Invasive devices
• Neuromuscular disease
• Depressed level of alertness
7. • Aspiration
• Antacid use
• Age 60 or older
• Prolonged hospital stay
• Any serious underlying disease.
Pathophysiology
Normally, the airway distal to the larynx is sterile because
of protective defence mechanisms. These mechanisms
include:
8. • Filtration of air
• Warming and humidification of inspired air
• Epiglottis closure over the trachea
• Cough reflex
• Mucociliary escalator mechanism
• Secretion of immunoglobulin A
• Alveolar macrophages.
Pneumonia results in inflammation of lung tissue.
Depending on the particular pathogen and the hosts’
9. physical status, the inflammatory process may involve
different anatomical areas of the lung parenchyma and
the pleurae.
The normal function of respiratory systemand primary
pathophysiology and clinical manifes tation are as follows:
i. Normally ‘mucociliary system’ cleanses inhaled
air by trapping particles. In pneumonia,
hypertrophy of mucous membrane lining lung,
resulting in hypersecretions leads to increased
sputum production and cough:
10. • Anerobic - Foul-smelling, sputum
• Klebsiella - Current Jelly color
• Staphylococcus - Creamy yellow
• Pseudomonas–Green
• Viral/mucopurulent
And bronchospasms from increased secretions, leads to
localised or diffuse wheezing dyspnea.
ii. Generally ‘alveolocapillary membrane’ exchan ges
oxygen-carbon dioxide. In pneumonia, there is increased
permeability resulting in excess fluid in interstitial space,
shows consolidation (in chest X-ray films)
11. localised/bacterial; diffuse/ viral, and also there is
decreased surface area for gas exchange leads to
hypoxaemia.
iii. Normally pleura maintains close approximation of
lungs and chest wall; minimizes friction during lung
expansion and contraction. In pneumonia there is
inflammation of the pleura which shows, chest pain,
especially on inspiration, pleural effusion, dullness on
percussion, decreased breath sounds and decreased vocal
fremitus.
12. iv. Normally respiratory muscle expands and contracts
chest wall and thus pleura and lungs. In pneumonia there
is hypoventilation and respiratory acidosis (in presence of
underlying disease) leads to decreased chest expansion
and hypercapnoea and low arterial blood pH.
v. The lung defense system protects normally sterile lung
from invasion. In pneumonia there is bacteremia, shows
elevated blood cell counting; leukocytes (15,000 to
25000/mm), neutrophilia and tachypnoea and fever.
In pneumococcal pneumonia there is congestion,
red hepatisation and gray hepatisation.
13. Clinical Manifestation
CAP has been traditionally thought to present any two
syndromes; typical and atypical, although the
distinctions are not clear.
●Typical pneumonic syndrome is characterised by
sudden onset of fever, chills, cough productive of
purulent sputum, and pleuritic chest pain (in some
cases).
14. On physical examination signs of pulmonary
consolidations such as dullness, percussion, increased
fermitus, bronchial breath sounds, and crackles may
be found.
In elderly or debilitated patient, confusion
or stupor may be predominant.
Usually two types of pneumonia caused by S. pneumoniae
and H. influenzae.
The atypical syndrome is characterised by a more
●gradual onset dry cough
15. ●and extrapulmonary manifestation Such as
●headache
●myalgia,
●fatigue,
●sore throat
● nausea
● vomiting
●diarrhea.
●On physical examination, crackles are often heard.
This type of classically produced by mycoplasm,
pneumonia and legonell chlamydia pneumoniae.
16. Viral pneumoniae are characterised by
an atypical presentation with
● chills,
● fever,
●dry non-productive cough
● and extrapulmonary Symptoms.
●Most of the pneumoniae run uncomplicated .
17. If occurs, the complications are pneumonia including
● pleurisy,
●pleural effusion,
● atelectasis,
● delayed resolution,
● lung abscess,
● empyema,
● pericarditis,
●arthritis,
● meningitis
●endocarditis.
19. • Chest X-ray film to confirm consolidation and
distribution and pleural effusions.
• Sputum studies for culture and sensitivity if unable
to obtain specimen by usual means, may use,
– Transtracheal aspiration,
– Bronchoscopy with aspiration, biopsy or bronchial
brushings.
• Arterial blood gas studies or pulse oximetry.
• Hematology: WBC count, cole agglutinin and
compliment fixation for viral studies.
• Thoracentesis to obtain pleural fluid specimen if
pleural effusion is present.
20. The possible nursing diagnosis on the basis and
assessment will be:
• Airway clearance, ineffective r/t decreased energy,
fatigue, tracheobronchial inflammation.
• Impaired gas exchange r/t alveolar capillary
membrane changes altered oxygen delivery.
• Pain r/t pleural inflammation, coughing paroxysms.
• Rest for infection r/t compromised lung defense
system.
• Knowledge deficient r/t condition, treatment.
• Anorexia r/t infection process-sputum production.
• Altered nutrition/body requirement r/t increased
metabolic needs.
21. Prompt treatment with the appropriate antibiotic
almost always cures bacterial and mycoplasmal pneu-
monia.
In uncomplicated cases, the patient responds to
drug therapy within 48 to 72 hours.
Indications for improvement including decreased
temperature, improved breathing, and reduced chest
pain. Abnormal physical finding lasts for more than 7 days.
22. The nursing intervention includes.
i. Maintaining effective airway clearance
• Monitor for increased respiratory distress
• Assist patient to cough effectively
• If unable to clear down airway, suction airway
using sterile technique
• Assist with nebuliser therapy
23. • Administer bronchiodilator as ordered. Monitor for side
effects and response to therapy
• Change position frequently to assist in mobilising
secretions
• Ensure fluid intake adequate to thin secretions.
ii. Facilitate breathing
Help the patient breathe deeply and expand the chest to
increase ventilation.
• Place the patient in position to facilitate breathing,
usually upright or semiupright position.
24. • A pillow may be placed lengthwise at the patient’s back
to provide support and thrust thorax slightly towards
allowing free use of the diaphragm.
• The patient who must be upright to breathe may find it
restful to place head and arms on a pillow placed on an
overbed table.
• For the patient with severe hypoxemia, side rails should
be in place. The patient can use them to assisting about in
bed.
• Some patients who breathe best when sitting
up in a large armchair while leaning on a Smaller chair.
Placed in front of that. This chair is blocked to prevent it
from slipping.
25. • Assist with ADL pacing activities to prevent fatigue and
respiratory distress.
iii. Administration of medication and treatment
• Before starting prescribed antibiotic, collect sputum for
culture and blood for culture if ordered.
• Maintain antibiotic blood levels by giving antibiotic at
scheduled time.
• Give medication prescribed to relieve pain. Codeine may
be ordered because it is less likely to inhibit cough reflex
than more potent narcotics.
• Begin oxygen therapy.
26. iv. Administering oxygen therapy
v. Promoting comfort
• Place in position of comfort, Preferably head of bed
elevated 45 to 90 degrees.
• Assess character and location of chest pain.
• Administer analgesic for chest pain e.g. acetylsalicylic
acid, acetominophen and codeine.
• Splint chest with hands when patient coughs.
• Administer frequent mouth care. Protect lips and nares
with lubricants.
• Keep patient warm and dry and avoid chilling.
27. vi. Preventing spread of infection.
• Standard precautions are used.
vii. Facilitating learning.
The major teaching emphasis is on prevention.
• Assess patient’s understanding of pneumonia
with questions concerning such information on how
pneumonia is transmitted and risk factor.
• Teach proper handling of secretions. Cover nose and
mouth with tissue when coughing or sneezing. Discard
tissues in paper or plastic bag for disposal. Expectorate
into specimen container provided
28. • Stress importance of handwashing after
coughing, sneezing and expectorating.
• Reinforce importance and follow-up care.
• Reinforce the need for immunization, i.e.,
inlfuenza vaccine and pneumococcal vaccine.
(Pneumonia Polysaccaride vaccine is given
only every 3 to 5 years.
viii. Promoting adequate hydration and nutrition
• Encourage oral fluids. If patient is receiving IV fluids,
monitor rate. Observe for signs of fluid volume deficit or
excess.
• Ask patient what foods he or she would like to eat.
29. • Offer small, frequent feedings.
• Encourage high-carbohydrate and high protein foods
Made by
Priyanshu Verma
Bsc nursing 3rd year student
Reference:- b.v. basavathappa