Please find the power point on Management and pathophysiology of Normal Labour . I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
The increased cardiac output related to pregnancy can lead to heart failure, and the increased heart rate in the third trimester can lead to ischemic events. The potential obstetrical complications include preeclampsia or other hypertensive related disorders, premature birth, and small-for-gestational-age births.
The increased cardiac output related to pregnancy can lead to heart failure, and the increased heart rate in the third trimester can lead to ischemic events. The potential obstetrical complications include preeclampsia or other hypertensive related disorders, premature birth, and small-for-gestational-age births.
Placenta previa is a condition in which the placenta lies very low in the uterus and covers all or part of the cervix. The cervix is the opening to the uterus that sits at the top of the vagina. Placenta previa happens in about 1 in 200 pregnancies.
Placenta praevia risk factors include a previous delivery, age older than 35 and a history of previous surgeries, such as a caesarean section (C-section) or uterine fibroid removal.
The main symptom is bright red vaginal bleeding without pain during the second-half of pregnancy. The condition can also cause severe bleeding before or during delivery.
Limited physical activity is recommended. A C-section is often required in severe cases.
Please find the power point on Management of Preterm labor. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Pathophysiology of Normal Labor:
A series of events that take place in female genital organs to expel the product of conception that are fetus, placenta, membranes) out of womb through the vagina into the outer world. We further describe pathogenesis and features of different stages of labor
Placenta previa is a condition in which the placenta lies very low in the uterus and covers all or part of the cervix. The cervix is the opening to the uterus that sits at the top of the vagina. Placenta previa happens in about 1 in 200 pregnancies.
Placenta praevia risk factors include a previous delivery, age older than 35 and a history of previous surgeries, such as a caesarean section (C-section) or uterine fibroid removal.
The main symptom is bright red vaginal bleeding without pain during the second-half of pregnancy. The condition can also cause severe bleeding before or during delivery.
Limited physical activity is recommended. A C-section is often required in severe cases.
Please find the power point on Management of Preterm labor. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Pathophysiology of Normal Labor:
A series of events that take place in female genital organs to expel the product of conception that are fetus, placenta, membranes) out of womb through the vagina into the outer world. We further describe pathogenesis and features of different stages of labor
This presentation contains :-
1.Introduction of normal labour
2. Definiation of normal labour
3.Criteria of normal labour
4. Physiology of normal labour
5. Pathophysiology of labor
6.Estrogen
7. Prostaglandin
8. Oxytocin
9. True labor and false labor difference
10. Uterine contraction in labor
11. Stages of labour
12. Management of 1 st stage
13. management of 2 nd stage
14. mamagement of 3 rd stage of labor
15. Cervix dilation
16. Friedman's curve
17. Fetal skull
18. Diameter of fetal skull
19. Sutures in fetal head
20. Moulding
21. Mechanism of labour
Childbirth, labour, delivery, birth, partus, or parturition is the culmination of a pregnancy period with the expulsion of one or more newborn infants from a woman's uterus. The process of normal childbirth is categorized in three stages of labour: the shortening and dilation of the cervix, descent and birth of the infant, and birth of the placenta.
Inorganic (non metallic) irritant Poisons by Sunil Kumar Dahasunil kumar daha
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Please find the power point on Carbonmonoxide poisioning and Its management. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
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Please find the power point on Management of alcohol withdrawl seizure . I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
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Please find the power point on Acute management of seizure. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Lymphoma by Sunil Kumar Daha (Hodgkins and Non-Hodgkins)sunil kumar daha
Please find the power point onLymphoma . I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Approach to a patient with fever of unknown origin sunil kumar daha
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Fever in a hospitalized patient and its managementsunil kumar daha
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Please find the power point on Typhus and its managemen. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
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The prostate is an exocrine gland of the male mammalian reproductive system
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A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
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micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
2. DEFINITION
Labor : series of events that take place in genital
organs to expel the product of conception (fetus,
placenta, membranes) out of womb through the
vagina into the external world.
Delivery: the expulsion of viable fetus out of the
uterus (vaginal (spontaneous or aided) or
abdominal
3. NORMAL LABOR/
EUTOCIA
spontaneous in onset and at term
with vertex presentation
without undue prolongation
Natural termination with minimal aids
without having any complications affecting the
health of the mother and/or the baby
4.
5. ESTROGEN
Increases release of oxytocin from maternal
pituitary
Promotes synthesis of myometrial receptors for
oxytocin,PGincrease in gap junctions in
myometrial cells
Stimulates synthesis of myometrial
contraction protein actinomyosin through
cAMP.
Increases excitability of myometrial cell
6. PROSTAGLANDIN
Initiates and maintain labor
Major site of production:
Amnion,chorion, decidual cells and
myometrium
Enhances gap junction formation
Triggered by estrogen, glucocorticoids,
separation or rupture of membrane
7. OXYTOCIN
Peptide hormone
Hypothalamus-posterior pituitary
Fetal production: Maternal serum increase in
second stage of labor
Oxytocin receptors: Fundal location, increase
during pregnancy
Actions
1. Stimulate uterine contractions
2. Stimulate PG production from amnion/decidua
8. TRUE AND FALSE LABOR
True labor
o Painful contractions at regular
intervals at term
o Contraction frequency,
intensity, duration increases
gradually
o Associated with Show
o Progressive effacement and
dilatation of cervix
o Descent of presenting part
o Formation of “bags of water”
o Not relieved by enema/
sedatives (analgesics)
False labor
o Dull pain confined to
groin and abdomen
o Pain interval doesn’t
shorten
o Pain intensity remains
same
o No cervical dilatation
o No hardening of uterus
o Relieved by enema or
sedative
9. DURING PREGNANCY…
Marked hypertrophy and hyperplasia of uterine
muscles
Length of uterus + cervix = 35 cm at term
Uterus assumes pyriform/ ovoid shape
Cervical canal occluded by thick, tenacious mucus
plug
10. UTERINE CONTRACTION IN
LABOR
Irregular involuntary painless spasmodic uterine
contraction (Braxton-Hicks) throughout pregnancy which
changes during labor
Pacemaker situated in : tubal ostia contraction waves
initiate
Pain of contraction distributed along the cutaneous nerve
distribution of T10 –L1
11. PATTERN OF
CONTRACTIONo Good synchronization of contraction waves from both
halves of the uterus
o Fundal dominance with gradual diminishing
contraction wave through midzone down to lower
segment in 10-20 sec
o Wave of contraction follow regular pattern
o Upper segment of uterus contracts longer and
stronger than lower part
o Intra-amniotic pressure rises beyond 20mm Hg
during uterine contraction
o Good relaxation occurs in between contraction(intra-
amniotic pressure less than 8)
12. RETRACTION
Phenomenon of uterus in labor in which muscle
fibers are permanently shortened
Effects of retraction:
1. Formation of lower uterine segment + dilatation and
effacement of cervix
2. Decent of presenting part expulsion of fetus
3. Reduce surface area separation of placenta
4. Effective homeostasis after separation of placenta
13. STAGES OF LABOR
First phase
- latent
- Active
Second phase
Propulsive
Expulsive
Third phase
Fourth phase
14. FIRST STAGE
Concerned with formation of birth canal
Main events:
1. Dilatation of cervix
2. Effacement of cervix
3. Lower uterine segment formation
15. FACTORS RESPONSIBLE IN
DILATATION
Uterine contraction and retraction
Longitudinal fiber of upper segment
attach to circular fiber of lower
segment if uterus contracts
canal opens + shortens polarity of
uterus
Fetal axis pressure
o longitudinal lie of fetus circular
muscles contraction transmitted
from podalic pole to head
o Not in transverse lie
Bag of membrane
Vis-a-tergo
16. EFFACEMENT OF CERVIX
“processes by which muscular fibers of cervix pulled
upward and merge with fibers of lower uterine
segment”
Primigravidae: effacement before dilation of
cervix
Multiparae: effacement and dilatation occur at
same time
18. Friedman’s Curve
Friedman's Curve describes progress of two variables
over time:
• dilation of cervix
• descent of baby
Labor is “dysfunctional” when cervix stops dilating or fetal
descent stops or both
Possible diagnosis of "failure to progress"
C-section indicated
May be due to CPD (Cephalo Pelvic Disproportion) or
epidural anesthesia
20. SECOND STAGE OF
LABOR
“Begins when cervical dilatation is complete and
ends with fetal delivery.”
Median duration
50 minutes in primigravida
20 minutes in multiparous
Uterine contractions and accompanying expulsive
forces last:
60-90 seconds and
recur every 60 seconds
21. Propulsive phase:
Period of full dilation until head touches pelvic floor
Expulsive phase:
Since the time mother has irresistible desire to ‘bear
down’ and push until the baby is delivered
22. DURATION OF LABOR
Mean length of 1st and 2nd stage labor
12 hours in primigravida
6 hours in multipara
23. THIRD STAGE OF LABOR
Includes separation, descent and expulsion of
placenta with its membrane.
Signs of placental separation:
1. Hardening of uterus
2. Sudden gush of blood
3. Rise of Uterus (because the placenta, having separated, passes
down in the lower uterine segment and vagina)
4. Lengthening of umbilical cord
Signs of placental separation appear within 1-5 minutes within
delivery of newborn.
24. FOURTH STAGE OF LABOR
The placenta, membranes and umbilical cord
should be examined for completeness and for
anomalies
observation: 1 hour after birth of baby
Laceration of birth canal(vagina and perineum):
first degree laceration
Second degree laceration
third degree laceration
fourth degree laceration
25. Degree of Lacerations
First degree laceration:
Involved the perineal skin, vaginal mucus membrane but not underlying
fascia and muscle
2nd degree laceration:
Involve in addition, the fascia and muscle of perineal body but not anal
sphincter
3rd degree laceration:
Extent further to involve the anal sphincter
4th degree laceration:
Laceration extend through the rectum’s mucosa to exposed its lumen
26. MANAGEMENT OF FIRST STAGE
LABOR
1. Monitoring fetal well-being during labor
Fetal heart should be monitored every 30 mins in 1st stage
and every 15 mins in 2nd stage of labor
2. Uterine contractions
to evaluate the frequency, duration, and intensity of uterine
contractions.
3. Maternal vital signs
Maternal temperature, pulse, and blood pressure are
evaluated at least every 4 hours
with prolonged membrane rupture(>18 hours) antimicrobial
administration for prevention of group B streptococcal
infections is recommended
4. Subsequent vaginal examinations
27. CONTD..
5. Oral intake
Food should be withheld during active labor and
delivery
6. Maternal position
position that she finds most comfortable, which
will be lateral recumbency most of the time
7. Urinary bladder function
Bladder distention-avoided, because it can hinder
descent of the fetal presenting parts
28. MANAGEMENT OF SECOND STAGE
LABOR1. Preparation for delivery
Put the patient in dorsal lithotomy position or lying flat on bed
Clean the vulva, and perineum with antiseptic solution
Encourage organized pushing down which she is feeling to do so
2. spontaneous delivery
With each contraction, perineum bulges increasing
Ritgen maneuver- when head distends the vulva and perineum
enough to open the vaginal introitus to 25 cm or more
A towel-draped ,gloved hand –used to exert forward pressure on the chin of
fetus through the perineum
This maneuver allow delivery of head and also favors the neck
extension so that head is delivered with small diameter
29. CONT..
Clearing the nasopharynx:
Once the thorax –delivered and the newborn can inspire
Face quickly wiped and the nares and mouth cleared
Nuchal cord :
Found in 25% of deliveries and ordinarily no harm
If coil of umbilical cord felt-it should be slipped over the
head if loose enough
If too tight, the loop should be cut between two clamps
30. CONT...
Clamping the cord:
Umbilical cord is cut between two clamps placed 4 to 5 cm from the foetal
abdomen and later an umbilical cord clamp-applied 2 to 3 cm from the fetal
abdomen
Plastic clamp –safe
Timing of cord clamping:
If after delivery of the newborn –placed below the level of the vaginal
introitus for 3 min and Fetoplacental circulation – not immediately occluded
by cord clamping, then approx. 80 ml of blood shift from placenta to
neonate this reduces the frequency of iron deficiency anemia later in
infancy
31. MANAGEMENT OF THIRD
STAGE LABOR Delivery of the placenta:
-Traction on the umbilical cord must not be used to pull the placenta
out of uterus.
-uterine inversion is one of the complication associated with delivery
Manual removal of placenta:
- Adequate analgesia is mandatory and aseptic surgical technique
should be used
-occasionally, placenta will not separate especially common in case
of preterm delivery
-if there is brisk bleeding and the placenta can not be delivered-
indicated
32. CONT...
1.Oxytocin
Given before delivery of placenta will decrease blood loss(they
may entrap an undiagnosed, undelivered 2nd twins)
The spontaneously labouring uterus is typically sensitive to oxytocin
and dosing should be titered to achieved adequate contraction
After delivery of the foetus, dosing should be fixed
It should be given as a dilute solution by continuous iv. Infusion or
im
10 USP unit i.m. (oral not effective)
T1/2 3-4 minutes- iv. Infusion (large bolus should not be given)
33. CONT..
CVS effect:
IV bolus of 10 unit of oxytocin caused marked fall in BP with an
abrupt increase in CO.
These hemodynamic changes could be dangerous for women
hypovolemic from haemorrhage or those with cardiac disease.
Water intoxication:
Has antidiuretic action
With high dose of oxytocin- produce water intoxication if the
oxytocin administered with large volume of electrolyte free aqueous
dextrose solution
Oxytocin given with NS or ringer solution
34. 2. Ergonovine and methylergonovine:
Ergot alkaloids
Stimulation of myometrium contraction
Given IV (0.1mg),IM or orally(0.25mg)
They are dangerous for mother and foetus prior to delivery-
tendency of relaxation
IV administration sometimes initiation of transient hypotension-
severe in gestational hypertension
3. prostaglandins:
Analogs not used routinely for management of 3rd stage labour
35. MANAGEMENT OF FOURTH
STAGE LABOR
1. Examine the placenta for their completeness,
- anomalies, ( single umbilical arteryMultiple births)
- length, and
- number of vessels in the cord and record the placental weight
Suture the episiotomy or any laceration
Estimate blood loss, count swabs, and take cord blood for Hb, blood
group, Rh, bilirubin, and Coomb’s test for Rh negative mother
Check BP, P, T and firmness of the uterus before transferring the patient
Allow no food during the first hour, sips of water may be taken
36.
37. FETAL SKULL
oMade of thin pliable tabular (flat) bones
forming the vault
oCompressible to some extent
oAreas of skull:
Vertex
Brow
Face
38. o Vertex: quadrangular area bounded
anteriorly by bregma and coronal sutures
Posteriorly by lambda and lambdoid suture
Laterally by lines passing through parietal eminences
o Brow:
One side anterior fontanels and coronal sutures
Other side root of nose and supra-orbital ridges of either side
o Face:
One side root of nose and supra-orbital ridges
On other side junction of floor of mouth with neck
39. SUTURES
Frontal: between the two frontal bones
Sagittal: between the two parietal
bones
Two coronal: between the frontal and
parietal bones
Two lambdoid: between the posterior
margins of the parietal bones and upper
margin of the occipital bone
40. Diameter Measurement(cm) Attitude of
head
Presentatio
n
Suboccipito-bregmatic (nape
of neck to center of bregma)
9.5 cm Complete
flexion
Vertex
Suboccipito-frontal (nape of
neck to ant. end of ant.
fontanelle )
10.5 cm Incomplete
flexion
Vertex
Occipito-frontal(occipital
eminence to glabella)
11.5 cm Marked
deflexion
Vertex
Mento-verticle (mid point of
chin to highest point on sagittal
suture)
14 cm
(13cm in oxford
hand book)
Partial
extension
Brow
Submento-verticle (junction of
floor of mouth and neck to
highest point on sagittal suture)
11.5 cm Incomplete
extension
Face
Submento-bregmatic (junction
of floor of mouth and neck to
center of bregma)
9.5 cm Complete
extension
Face
ANTERO-POSTERIOR DIAMETER OF HEAD
THAT MAY ENGAGE
41. a. Biparietal diameter:
i. 9.5 cm
ii. Extends between two parietal eminences
b. Super-subparietal diameter:
i. 8.5 cm
ii. Extends from a point placed below one parietal eminence
to a point placed above other parietal eminence of
opposite side
c. Bitemporal diameter:
i. 8 cm
ii. Distance between antero-inferior ends of coronal suture
d. Bimastoid diameter:
i. 7.5 cm
ii. Distance between tips of mastoid processes
TRANSVERSE DIAMETER
CONCERNED IN MECHANISM OF
LABOR
42. Attitude of head Plane of engagement Circumference
Complete flexion Biparietal-suboccipito-bregmatic
Shape - almost round
27.5 cm
Deflexed Biparietal-occipito-frontal
Shape – oval
34 cm
Incomplete extension Biparietal-mento-vertical
Shape - bigger oval
37.5 cm
Complete extension Biparietal-submento-bregmatic
Shape - almost round
27.5 cm
CIRCUMFERENCE
Circumference of the plane of diameter of engagement
differs according to attitude of head
Circumference of head in different attitude:
43. MOULDING
“The alteration of the shape of the fore coming head
while passing through the resistant birth passage during
labor”
o There is little alteration in size of head as the volume
of content inside skull is incompressible
o An alternation of 4mm in skull diameter commonly
occur during normal delivery
o Disappears within few hours after birth
44. MECHANISM:
Compression of engaging diameter of head with corresponding
elongation of the diameter at right angle to it
GRADING OF MOULDING
Grade 1:
Bones touching but not overlapping
Grade 2:
Bones overlapping but easily separated
Grade 3:
Fixed overlapping of bones
45. IMPORTANCE OF MOULDING
Slight moulding is inevitable and beneficial
Enables head to pass more easily through the
birth canal
Extreme moulding may produce Severe
intracranial disturbance in the form of tearing
of tentorium cerebelli or subdural
haemorrhage
Shape of moulding gives information about
position of head occupied in pelvis
47. o the passage of the widest
diameter of the presenting part
to a level below the plane of the
pelvic inlet
o In the cephalic presentation with a
well-flexed head, the largest
transverse diameter of the fetal
head is the biparietal diameter (9.5
cm)
ENGAGEMENT
48. Engagement can be confirmed clinically
by palpation of the presenting part
abdominally and/or vaginally
The head is assumed to be engaged if
the leading edge has reached the ischial
spines and there is no significant
molding or scalp edema
49. Head in Synclitism: the sagittal suture corresponds
to the diameter of engagement with the head enters
the brim
Anterior asynclitism: Anterior parietal presentation
Posterior asynclitism: Posterior parietal presentation
Mild degree of asynclitism are common but severe
degrees indicate cephalopelvic disproportion
PRESENTATION
50. downward passage of the presenting
part through the pelvis
The greatest rate of descent occurs during the
deceleration phase of the first stage and during the
second stage of labor
Forces involved:-
1. Pressure of amniotic fluid
2. Pressure of fundus upon breech with contraction
3. Maternal abdominal muscles
4. Extension and straightening of fetal body
DESCENT
51. o Occurs passively as the head
descends
o due to resistance related to the shape of bony pelvis
& by the soft tissues of the pelvic floor
o Although flexion of the fetal head onto the chest is
present to some degree in most fetuses antepartum,
complete flexion usually only occurs during the
course of labor
o A deflexed head presents a larger diameter, which
may be too large to negotiate the pelvic bone
FLEXION
52. o Rotation of the presenting part
from its original position (usually transverse with
regard to the birth canal) to the anteroposterior
position as it
passes through the pelvis
o As with flexion, internal rotation is a passive
movement resulting from the shape of the pelvis
and the resistance of the pelvic floor musculature
INTERNAL ROTATION
53. o Occurs once the fetus has
descended to the level of the
introitus
o This descent brings the base of the occiput into
contact with the inferior margin of the symphysis
pubis
o At this point, the birth canal curves upwards
o The fetal head is delivered by extension and
rotates around the symphysis pubis
EXTENSION
54. o After the fetal head deflexes (extends),
it rotates to the correct anatomic
position in relation to the fetal torso;
left or right rotation depends on the
orientation of the fetus
o Passive movement resulting from a release of the
forces exerted on the fetal head by the maternal bony
pelvis and its musculature and mediated by the basal
tone of the fetal musculature
EXTERNAL ROTATION
55. Cunningham et.al., Williams OBSTETRICS,
24E, McGraw-Hill Education, 2014,
DC Dutta’s textbook of Obstetrics
References
ABNORMAL LABOR/ DYSTOCIA: DEVIATION FROM NORMAL LABOR
Bags of water: detached membrane with liquor that presents below the presenting part; almost certain sign of labor
False pain/ suurious labor is usual in primimother, 1-2 weeks prior to labor, may be due to stretching of cervix or lower uterine segment
Prelabor: lightening (good sign:presenting part sinks to true pelvis) + false pain + ripening of cervix (soft, effaced, dilatable)
Myometrial hypoxia during contraction
Stretching of peritoneum over fundus
Stretching of cervix during dilatation
Stretching of ligaments surrounding uterus
Compression of nerve ganglion
Normal polarity of uterus: contraction starts from the fallopian tube fundus contracts more than lower segment when fundus contracts to push fetus, lower segment and cervix dialate in response to force
Bag of membrane:—The membranes (amnion and chorion) are attached loosely to the decidua lining the uterine cavity except over the internal os. In vertex presentation, the girdle of contact of the head (that part of the circumference of the head which first comes in contact with the pelvic brim) being spherical, may well fit with the wall of the lower uterine segment. Thus, the amniotic cavity is divided into two compartments (Fig. 12.5). The part above the girdle of contact contains the fetus with bulk of the liquor called hindwaters and the one below it containing small amount of liquor called forewaters. With the onset of labor, the membranes attached to the lower uterine segment are detached and with the rise of intrauterine pressure during contractions there is herniation of the membranes through the cervical canal. There is ball-valve like action by the well flexed head. Uterine contractions generate hydrostatic pressure in the forewaters that in turn dilate the cervical canal like a wedge. When the bag of forewater is absent (PROM) the pressure of the presenting part pushes the cervix centrifugally
Vis- a-tergo: The final phase of dilatation and retraction of the cervix is achieved by downward thrust of the presenting part of the fetus and upward pull of the cervix over the lower segment. This phenomenon is lacking in transverse lie where a thin cervical rim fails to disappear.
The Ritgen maneuver is an obstetric procedure used by midwives and doctors in order to control the delivery of the fetal head. It involves applying an upward pressure from the coccygeal region to extend the head during actual delivery, thereby protecting the musculature of theperineum.
Delayed cord clamping until 1 min after birth increase the newborn HB conc. 2.2g/dl compared with clamping with in 60 sec.
Early cord clamping reduced the risk of phototherapy by 40%
Placental delivery is recommended to prevent postpartum hemorrhage
United States Pharmacopeia: Abbreviated USP
The cardinal movements of labor refer to changes in the position of the fetal head during its passage through the birth canal (figure 5). Because of asymmetry in both the shape of the fetal head and the maternal bony pelvis, such rotations are required if the fetus is to successfully negotiate the birth canal.