anatomy
Abducens nerve palsy is the most common ocular motor paralysis. The abducens (sixth) cranial nerve controls the lateral rectus muscle, which abducts the eye. Abducens nerve palsy causes an esotropia due to the unopposed action of the antagonistic medial rectus muscle. The affected eye turns in toward the nose and is unable to abduct properly. The deviation is constant and is usually greater at distance fixation than at near. The esotropia is also worse when the patient is looking toward the affected side.
anatomy
Abducens nerve palsy is the most common ocular motor paralysis. The abducens (sixth) cranial nerve controls the lateral rectus muscle, which abducts the eye. Abducens nerve palsy causes an esotropia due to the unopposed action of the antagonistic medial rectus muscle. The affected eye turns in toward the nose and is unable to abduct properly. The deviation is constant and is usually greater at distance fixation than at near. The esotropia is also worse when the patient is looking toward the affected side.
1.BRIEF ANATOMY OF EYE
2.OPTIC NEUROPATHY
3. SIGNS OF OPTIC NEUROPATHY
4. CLASSIFICATION OF OPTIC NEUROPATHY
5. IN DETAIL ABOUT DIFFERENT OPTIC NEUROPATHY
6. MANAGEMENT OF OPTIC NEUROPATHY
Pupillary light reflex (PLR) : that controls the diameter of the pupil, in
response to the intensity of light that falls on the retinal ganglion cells of
the retina in the back of the eye.
- Light reflex
- Corneal reflex.
-Accommodation reflex:
Argyll Robertson pupils
Horner's syndrome:
Holmes–Adie syndrome
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anatomy of optic nerve and its blood supply and clinical corelation
Presentation Layout: optic nerve anatomy
Embryology of optic nerve
Introduction
Parts of optic nerve
Blood supply
Clinical significance
For Further Reading
Wolff’s Anatomy of the eye and orbit by Bron, Tripathi and Tripathi
Anatomy and Physiology of eye by A.K. Khurana 2nd edition
Comprehensive Ophthalmology by A.K. Khurana 5th edition
AAO- Fundamentals & Principles of Ophthalmology : sec 2
Walsh and Hoyt’s Clinical Ophthalmology
Internet
1.BRIEF ANATOMY OF EYE
2.OPTIC NEUROPATHY
3. SIGNS OF OPTIC NEUROPATHY
4. CLASSIFICATION OF OPTIC NEUROPATHY
5. IN DETAIL ABOUT DIFFERENT OPTIC NEUROPATHY
6. MANAGEMENT OF OPTIC NEUROPATHY
Pupillary light reflex (PLR) : that controls the diameter of the pupil, in
response to the intensity of light that falls on the retinal ganglion cells of
the retina in the back of the eye.
- Light reflex
- Corneal reflex.
-Accommodation reflex:
Argyll Robertson pupils
Horner's syndrome:
Holmes–Adie syndrome
Direct Download Link ❤❤https://healthkura.com/eye-ppt/28/❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com/eye-ppt/❤❤❤
anatomy of optic nerve and its blood supply and clinical corelation
Presentation Layout: optic nerve anatomy
Embryology of optic nerve
Introduction
Parts of optic nerve
Blood supply
Clinical significance
For Further Reading
Wolff’s Anatomy of the eye and orbit by Bron, Tripathi and Tripathi
Anatomy and Physiology of eye by A.K. Khurana 2nd edition
Comprehensive Ophthalmology by A.K. Khurana 5th edition
AAO- Fundamentals & Principles of Ophthalmology : sec 2
Walsh and Hoyt’s Clinical Ophthalmology
Internet
Cranial nerve assessment..Simple and Easy to perform for medics and Physiothe...pawan1physiotherapy
Cranial Nerve Assessment is a crucial step in neurological assessment. By following the simple theoretical aspects it can be made on your fingertips....here is an try to make the stuff easier for you....
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
The Gram stain is a fundamental technique in microbiology used to classify bacteria based on their cell wall structure. It provides a quick and simple method to distinguish between Gram-positive and Gram-negative bacteria, which have different susceptibilities to antibiotics
Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
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CDSCO and Phamacovigilance {Regulatory body in India}NEHA GUPTA
The Central Drugs Standard Control Organization (CDSCO) is India's national regulatory body for pharmaceuticals and medical devices. Operating under the Directorate General of Health Services, Ministry of Health & Family Welfare, Government of India, the CDSCO is responsible for approving new drugs, conducting clinical trials, setting standards for drugs, controlling the quality of imported drugs, and coordinating the activities of State Drug Control Organizations by providing expert advice.
Pharmacovigilance, on the other hand, is the science and activities related to the detection, assessment, understanding, and prevention of adverse effects or any other drug-related problems. The primary aim of pharmacovigilance is to ensure the safety and efficacy of medicines, thereby protecting public health.
In India, pharmacovigilance activities are monitored by the Pharmacovigilance Programme of India (PvPI), which works closely with CDSCO to collect, analyze, and act upon data regarding adverse drug reactions (ADRs). Together, they play a critical role in ensuring that the benefits of drugs outweigh their risks, maintaining high standards of patient safety, and promoting the rational use of medicines.
Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Best Ayurvedic medicine for Gas and IndigestionSwastikAyurveda
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Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
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NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
NVBDCP.pptx Nation vector borne disease control program
Optic Nerve
1. SET BY: NOFEL SALEEM ASSWIEL
MEDICAL STUDENT ,ADEN UNIVERSITY
THIRD YEAR 2013/2014
E-mail: Nofel166@gmail.com
@nasswiel
facebook/nasswiel
Symptoms & Signs in Clinical Medicine
2.
3.
4. Impressions of light >> optic nerve >>
optic chiasma >> optic tracts >> lateral
geniculate bodies >> optic radiation >>
occipital cortex
optic tracts >> pretectal area (pupillary
Reflex)
Medial parts of optic tracts
(communication) between optical
system & ocluomotor nuclei.
5.
6.
7.
8.
9.
10.
11.
12.
13.
14.
15.
16.
17.
18. Visual Acuity
Near vision
- Reading from a book
Far vision
- Snellen chart.
- Ishihara charts
26. Foster kennedy syndrome
Seen in patients with frontal lobe /
olfactory lobe tumors, meningiomas of
olfactory groove / sphenoidal wing.
characterized by
optic atrophy on the side of the tumor
(direct pressure on the nerve)
papilledema on the opposite side
(raised ICT).
29. Optic Neuritis
Definition
- Inflammation or demyelination
- At any point of the optic nerve.
Near the disc >> optic neuritis
behind the eyeball(lamina cribrosa) >>
retrobulbar neuritis.
Causes
- Multiple Sclerosis
- Meningitis
- Avitaminosis .
30. Optic Neuritis
Central scotoma
Oedematous disc>> less swelling
Red coloration of the disc
Inflammatory exudates > cloudy disc
36. GRADE I HTR
Narrowing of the vessel
Age- related
37. GRADE II HTR
Marked variation of
the caliber of the
vessel.
Veins is kinked, the
peripheral caliber is
engorged
Age- related
38. GRADE III HTR
Addition of Flame
shaped or round
retinal hemorrhages
& cotton wool spots,
hard exudates.
39. GRADE IV HTR
All changes of
grade 3
Addition of
papilloedema
Increase
hemorrhages and
exudates
40. Diabetic Retinopathy
Definition
Progressive dysfunction of the retinal blood
vessels caused by chronic hyperglycemia.
DR can be a complication of diabetes type
1 or diabetes type 2.
Initially, DR is asymptomatic, if not treated
though it can cause low vision and
blindness.
41. Diabetic Retinopathy
symptoms
Diabetic retinopathy is asymptomatic in early
stages of the disease
As the disease progresses symptoms may include
Blurred vision
Floaters
Fluctuating vision
Distorted vision
Dark areas in the vision
Poor night vision
Impaired color vision
Partial or total loss of vision
43. Diabetic Retinopathy
Next :
larger blot & dot
hemorrhages
appear
Then:
waxy-looking
exudates with
harder edges.
Microaneurysms
Hard exudates
44. Diabetic Retinopathy
Larger hemorrhages
appear with
irregular veins.
New-formed
vascular plexuses
.veins loops and
coils(may
protruded into the
vitreous)
Hard exudatesNeovascularization
Blot hemorrhage