Nystagmus is an involuntary, rhythmic movement of the eyes. This document discusses evaluating a case of nystagmus, including defining nystagmus, classifying its types, obtaining a patient history, measuring visual acuity, amplitude, frequency, and null/neutral zones. Treatment options are also summarized such as correcting refractive error, using contact lenses, added lenses, prism correction, vision therapy, and surgery.
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Retinoscopy and Objective Refraction and Subjective Refraction in spherical ametropia and astigmatism
Retinoscopy (Principle & Techniques of Retinoscopy) and objective refraction, Subjective Refracition
Best presentation about retinoscopy and objective refraction techniques, and basis of subjective refraction. If you want to master the technique of retinoscopy, this presentation can be your guidance and partner in your journey to retinoscopy, objective refraction and subjective refraction.
Presentation Layout:
Retinoscope, types of retinoscope and uses of retinoscope
-Introduction to retinoscopy and objective refraction
-Retinoscopy
- In spherical ametropia
- In astigmatism
- Others: strabismus, amblyopia, pediatric pt.,
cycloplegic refraction
-Static and Dynamic Retinoscopy
-Problems seeing reflex during retinoscopy
-Errors in retinoscopy
Objective of retinoscopy and objective refraction
-To locate the far point of the eye conjugate to the retina
- Myopia or hyperopia
-Bring far point to the infinity by using appropriate lenses
- Determines amount of ametropia by retinoscopy and objective refraction
References:
-Clinical Procedures in Optometry by Eskridge, Amos and Bartlett ,
-Primary Care Optometry by Grosvenor T.,
-Borish’s Clinical Refraction by Benjamin W. J.,
-Theory And Practice Of Optics And Refraction by AK Khurana
-Retinoscopy-Student Manual by ICEE Refractive Error Training Package (2009)
-Clinical Optics and Refraction By Andrew Keirl, Caroline Christie
-Clinical Refraction Guide - A Kumar Bhootra
-Clinical Procedures in Primary Eye Care by David B. Elliott
-Internet
Follow me to get in touch with optometric and ophthalmic updates.
DIRECT DOWNLOAD LINK ❤❤https://healthkura.com/retinoscopy/❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com ❤❤❤
Retinoscopy and Objective Refraction and Subjective Refraction in spherical ametropia and astigmatism
Retinoscopy (Principle & Techniques of Retinoscopy) and objective refraction, Subjective Refracition
Best presentation about retinoscopy and objective refraction techniques, and basis of subjective refraction. If you want to master the technique of retinoscopy, this presentation can be your guidance and partner in your journey to retinoscopy, objective refraction and subjective refraction.
Presentation Layout:
Retinoscope, types of retinoscope and uses of retinoscope
-Introduction to retinoscopy and objective refraction
-Retinoscopy
- In spherical ametropia
- In astigmatism
- Others: strabismus, amblyopia, pediatric pt.,
cycloplegic refraction
-Static and Dynamic Retinoscopy
-Problems seeing reflex during retinoscopy
-Errors in retinoscopy
Objective of retinoscopy and objective refraction
-To locate the far point of the eye conjugate to the retina
- Myopia or hyperopia
-Bring far point to the infinity by using appropriate lenses
- Determines amount of ametropia by retinoscopy and objective refraction
References:
-Clinical Procedures in Optometry by Eskridge, Amos and Bartlett ,
-Primary Care Optometry by Grosvenor T.,
-Borish’s Clinical Refraction by Benjamin W. J.,
-Theory And Practice Of Optics And Refraction by AK Khurana
-Retinoscopy-Student Manual by ICEE Refractive Error Training Package (2009)
-Clinical Optics and Refraction By Andrew Keirl, Caroline Christie
-Clinical Refraction Guide - A Kumar Bhootra
-Clinical Procedures in Primary Eye Care by David B. Elliott
-Internet
Follow me to get in touch with optometric and ophthalmic updates.
This deals with the types of Nystagmus both in pediatrics and adults, physiological and pathological types. Also the different diagnostic techniques and the management plan are presented in this.
Nystagmus is a condition of involuntary (or voluntary, in some cases)eye movement, acquired in infancy or later in life, that in extremely rare cases may result in reduced or limited vision. Due to the involuntary movement of the eye, it has been called "dancing eyes"Contents
1 Causes
1.1 Early-onset nystagmus
1.2 Acquired nystagmus
1.3 Other causes
2 Diagnosis
2.1 Pathologic nystagmus
2.2 Physiological nystagmus
3 Treatment
4 Epidemiology
Speaker Name: Anjali
Topic: "Demystifying Nystagmus"
Hello Everyone, Namaste!! We would like to notify you all that Mero Eye Foundation is going to conduct an "EYE TALKS-Webinar", and we will be having our session live broadcast on YouTube (Session No. 118)
DATE: at, 07:300 PM NPT, 07:15 PM IST, 22nd May 2021.
YouTube links: https://youtu.be/b4G12rRvXFc
This presentation explain about retinoscope, the instrument, its history, its types, the procedure and different cases also the advantages and disadvantages of the instrument and the working lens
astigmatism
AstigmatismAstigmatism Walter Huang, ODWalter Huang, OD Yuanpei UniversityYuanpei University Department of OptometryDepartment of Optometry
2. DefinitionDefinition When parallel rays of light enter the eyeWhen parallel rays of light enter the eye ((with accommodation relaxedwith accommodation relaxed) and do) and do notnot come to a single point focus on or nearcome to a single point focus on or near the retinathe retina
3. OpticsOptics Power in thePower in the horizontalhorizontal plane projects aplane projects a verticalvertical focal line imagefocal line image Power in thePower in the verticalvertical plane projects aplane projects a horizontalhorizontal focal line imagefocal line image
4. OpticsOptics Refraction of light taking place at a toricRefraction of light taking place at a toric surface: the conoid of Sturmsurface: the conoid of Sturm
TONOMETRY • Tonometry is the procedure performed to determine the intraocular pressure (IOP).
3. CLASSIFICATION TONOMETRY DIRECT INDIRECT Indentation Applanation Manometer
4. APPLANATION Contact Non-contact Goldmann Perkins Air-puff Pulse air
5. INDENTATION TONOMETER • It is based on fundamental fact that plunger will indent a soft eye more than hard eye. • The indentation tonometer in current use is that of Schiotz . • It was devised in 1905 and continued to refine it through 1927.
6. PROCEDURE • Patient should be anaesthetising with 4% lignocaine or 0.5% proparacaine. • with the patient in supine position, looking up at a fixation target while examiners separates the lids and lower the tonometer plate to rest on the cornea so that plunger is free to move. •
. Introduction Biomicroscope derives its name from the fact that it enables the practitioner to observe the living tissue of eye under magnification. It not only provides magnified view of every part of eye but also allows quantitative measurements and photography of every part for documentation.
3. • The lamp facilitates an examination which looks at anterior segment, or frontal structures, of the human eye, which includes the –Eyelid –Cornea –Sclera –Conjunctiva –Iris –Aqueous –Natural crystalline lens and –Anterior vitreous.
4. Important historical landmarks De Wecker 1863 devised a portable ophthalmomicroscope . Albert and Greenough 1891,developed a binocular microscope which provided stereoscopic view. Gullstrand ,1911 introduced the illumination system which had for the first time a slit diapharm in it Therefore Gullstrand is credited with the invention of slit lamp.
1. Introduction Gross anatomy Layers Blood supply, drainage and nerve supply
2. INTRODUCTION • Sclera forms posterior 5/6th of external tunic , connective tissue coat of eyeball. • it continues with duramater and cornea • Its whole surface covered by tenon’s capsule • Anteriorly covered by- bulbar conjunctiva • Inner surface lies in contact with choroid • With a potential suprachoroidal space in between
3. Equa THICKNESS OF SCLERA
4. • Thickness varies with individual, with age • Thinner- children, elder, F> M • Thickest posteriorly • Gradually becomes thinner when traced anteriorly • Thin at insertion of extraocular muscle
The pupil is an opening located in the center of the iris that allows light to enter the retina. • Its function is to control the amount of light entering the eye and it does this via contraction (miosis) and dilation (mydriasis) under the influence of the autonomic nervous system
3. • The iris is a contractile structure, consisting mainly of smooth muscle, surrounding the pupil. Light enters the eye through the pupil, and the iris regulates the amount of light by controlling the size of the pupil.
4. The iris contains two groups of smooth muscles: a circular group called the sphincter pupillae. and a radial group called the dilator pupillae.
5. Parasympathetic pathway • First Order – Retina to Pretectal Nucleus in B/S (at level of Superior colliculus) Second Order – Pretectal nucleus to E/W nucleus (bilateral innervation!) Third Order – E/W nucleus to Ciliary Ganglion Fourth Order – Ciliary Ganglion to Sphincter pupillae (via short ciliary nerves) • • •
The tear film constitutes Three layers :- An outermost lipid (oily) layer An aqueous (watery) layer that makes up 90% of the tear film volume; and A mucin layer that coats the corneal surface.
3. To form smooth optical surface on cornea. To keep the surface of cornea & conjunctiva moist It serve as lubricant It transfer oxygen Provide antibacterial action Wash debris out It provides a pathway for WBC in case of injury
4. Functions of lipid layer Retards evaporation of tear film Prevents the overflow of tears
5. Function of Aqueous Layer Flushes, buffers and lubricates the corneal surface Delivers oxygen and other nutrients to the corneal surface Wash out debris Delivers antibacterial enzymes and antibodies such as lysozyme.
6. Functions of Mucin Layer Spreads tears over corneal surface. Protects the cornea against foreign substances . Makes corneal surface smooth by filling in surface irregularities
Introduction Transparent,avascular,watch-glass like structure. Forms 1/6th part of outer fibrous coat (Sclera) It is the major refracting surface of the eye
3. Dimensions + Avg horizontal dia =11.75 mm (ant surface) + Avg vertical dia = 11 mm (ant surface ) + Avg dia (post surface)= 11.5 mm + Thickness(centre) =0.52mm + Thickness(peripheral) = 0.67mm + Radius of curvature (ant surface) = 7.8mm + Radius of curvature (post surface)= 6.5mm + Refractive power (ant surface) = +48D + Refractive power(post surface)= - 5D + R.I = 1.376
4. Histology + Epithelium + Bowman’s membrane + Stroma + Dua’s layer + Descemet’s membrane + Endothelium
5. Epithelium + Made up of stratified squamous epithelium + Thickness - 50-90 um + 5-6 layers of cells + Regenerative, entire epithelial layer is replaced every 6-8 days + Made up of 3 types of cells - basal,wing, flattened cells + Cells are attached by to each other by means of desmosomes & maculi occludents
6. Bowman’s membrane + Acellular,Non regenerative + Made up of condensed collagen fibrils. + Thickness - 12um + Resistant to infection & injury.
LIMBUS… • The limbus forms the border between the transparent cornea and opaque sclera, contains the pathways of aqueous humour outflow, and is the site of surgical incisions for cataract and glaucoma
2. Anatomical Limbus: Circumcorneal transitional zone of the conjunctivocorneal & corneoscleral junction Conjunctivo-corneal junction: • Bulbar conjunctiva is firmly adherent to underlying structures • Substantia propria of the conjunctiva stops here but its epithelium continues with that of the cornea. Sclero-corneal junction: • Transparent corneal lamellae become continuous • With the oblique, circular and opaque fibres of sclera
3. CONTINUE…. • In the area near limbus, the conjunctiva, tenon’s capsule & the episcleral tissue are fused into a dense tissue which is strongly adherent to corneo scleral junction.It is preferred site for obtaining a firm hold of the eyeball during ocular surgery. • The limbus is a common site for the occurrence of corneal epithelial neoplasm. • The Limbus contains radially oriented fibrovascular ridge known as the palisades of Vogt that may harbour a stem cell population. The palisades of Vogt are more common in the superior and inferior quadrants around the eye
Diagnosis, Management, and Surgery by Adam J. Cohen, Michael Mercandetti & Brian G. Brazzo. The dry eye , a practical approach by Sudi Patel & Kenny J Blades. Jack J Kanski’s clinical ophthalmology Clinical Anatomy of the Eye by Richard S. Snell & Michael A. Lemp.
3. It is concerned with the tear formation & transport. Lacrimal passage includes : Lacrimal gland Conjunctival sac Lacrimal puncta Lacrimal canaliculi Lacrimal sac Nasolacrimal duct
4. The following components of the lacrimal apparatus are discussed : Embryology Osteology Secretory system Excretory system Physiology
5. Ectodermal origin Solid epithelial buds(first 2 months) Supero
Extraocular musles(EOM) They are six in number Four recti: Superior rectus Inferior rectus Medial rectus Lateral rectus Two oblique muscles: Superior oblique Inferior oblique
3. SUPERIOR RECTUS MUSCLE . Origin Superior part of common annular tendon of Zinn Course Passes anterolaterally beneath the levator At 23 degrees with the globe ‘s AP axis Pierces Tenon s capsule Insertion into sclera by flat tendinous 10 mm broad insertion 7.7 mm behind sclero-corneal junction. 42 mm long 9 mm wide
4. Nerve supply Sup division of 3rd N Blood Supply Lateral Muscular br. of Ophthalmic A APPLIED: SR loosely bound to LPS muscle. • During SR resection- eyelid may be pulled forward narr owing palpebral fissure • In hypotropia pseudoptosis may be present Origin of SR and MR are closely attached to the dural sheat h of the optic nerve pain during upward & inward movements of the globe in RETROBULBAR NEURITIS
Diseases of sclera
2. anatomy • Sclera posterior 5/6th opaque part of the external fibrous tunic of the eyeball.
3. • outer surface }covered by Tenon's capsule. • anterior part } covered by bulbar conjunctiva.
4. Its inner surface lies in contact with choroid with a potential suprachoroidal space in between
5. Thickness of sclera. • thinner }children and in females Sclera • thickest} posteriorly (1mm) • gradually becomes thin when traced anteriorly. • thinnest } insertion of extraocular muscles (0.3 mm). • Lamina cribrosa is a sieve-like sclera from which fibres of optic nerve pass.
6. Apertures of sclera • Anterior • Anterior ciliary vessels • Middle • four vortex veins (vena verticosae) • Posterior • Optic nerve • Long & short ciliary nerves
7. Layers of sclera sclera episclera Sclera proper Lamina fusca thin, dense vascularised layer of connective tissue fibroblasts, macrophages and lymphocytes avascular structure dense bundles of collagen fibres. innermost blends with suprachoroidal and supraciliary laminae of the uveal tract. brownish in colour presence of pigmented cells.
Main physiologic function of cornea is to act as a major refracting medium, so that a clear retinal image is formed. • Normal corneal transparency is result of • 1.anatomical factor such as uniform and regular arrangement of corneal epithelium, peculiar arrangement of corneal lamella and corneal vascularity 2.Physiological factor [ie] relative state of corneal dehydration.
3. • Therefore, any process which upsets the anatomy or physiology of cornea will cause LOSS OF TRANSPARENCY to some degree.
4. FACTORS AFFECTING CORNEAL TRANSPARENCY • CORNEAL EPITHELIUM &TEAR FLIM • ARRANGEMENT OF STROMAL LAMELLA • CORNEAL VASCULARIZATION • CORNEAL HYDRATION • CELLULAR FACTORS AFFECTING TRANSPARENCY
CONJUNCTIVA: ANATOMY , PHYSIOLOGY, SYMPTOMATOLOGY AND CLASSIFICATION Pranay Shinde DNB Resident Deen Dayal Upadhyay Hospital,New Delhi
2. ANATOMY It is the mucous membrane covering the under surface of the lids and anterior part of the eyeball upto the cornea.
3. Parts of conjunctiva • Palpebral; covering the lids—firmly adherent. • Forniceal; covering the fornices—loose—thrown into folds. • Bulbar; covering the eyeball—loosely attached except at limbus.
4. Palpebral conjunctiva • Subtarsal sulcus 2mm from posterior edge of the lid margin. • Richly vascular. • Extremely thin. • Strongly bound to the tarsal plate.
5. Palpebral conjunctiva is subdivided into three parts: 1)Marginal 2)Tarsal 3)Orbital
6. Conjunctival fornices • Transitional region between palpebral and bulbar conjunctivae. • Superior fornix 10 mm from limbus. • Inferior fornix 8 mm from limbus. • Lateral fornix 14mm from limbus. • Medially absent. • Ducts of lacrimal glands open into lateral part of superior fornix.
q Colour Vision Deficiency Presented by : Optometrist (intern) Asma Al-Jroudi Saudi Arabia, Riyadh, King Abdulaziz University Hospital 30 Dec 14
2. • What Is Color Vision Deficiency? • Causes Of Color Vision Deficiency • Types Of Color Vision Deficiency • Tretments Of Color Vision Deficiency • Ishihara’s Test • Conclousion
3. What is Colour Blindness? • Color blindness, or color vision deficiency, is the inability or decreased ability to see color, or perceive color differences, under normal lighting conditions. •This condition results from an absence of color- sensitive pigment in the cone cells of the retina, the nerve layer at the back of the eye.
4. What is Colour Blindness? • Cones are the coulored light receptors in back of the eye: Red light receptors, Blue light receptors and Green light receptors. • Colour blindness occurs when one or more of the cone types are defected.
5. Causes of Color Blindness • Genetic: Many more men are affected than women. • Acquired : Chronic illness, Accidents, Medications and Age.
ANATOMY & PHYSIOLOGY Lecturer: Tatyana V. Ryazantseva
2. Outer eye: Eyelids The eyelids fulfill two main functions: protection of the eyeball secretion, distribution and drainage of tears
3. Lid movement The levator extends from an attachment at the orbital apex to attachments at the tarsal plate and skin. ● The lids are securely attached at either end to the bony orbital margin by the medial and lateral palpebral ligaments. Trauma to the medial ligament causes the lid to flop forward and laterally, impairing function and cosmesis.
4. Innervation - Sensory innervation is from the trigeminal (fifth) cranial nerve, via the ophthalmic division (upper lid) and maxillary division (lower lid). - The orbicularis oculi is innervated by the facial (seventh) nerve. - The levator muscle in the upper lid is supplied by the oculomotor (third) nerve.
5. Blood supply and lymphatics The eyelids are supplied by an extensive network of blood vessels which form an anastomosis between branches derived from the external carotid artery via the face and from the internal carotid artery via the orbit.
6. Blood supply and lymphatics Lymphatic fluid drains into the preauricular and submandibular nodes. Preauricular lymphadenopathy is a useful sign of infective eyelid swelling (especially viral).
Anatomy and Physiology of Aqueous Humor Sumit Singh Maharjan
2. Anatomy
3. Angle of anterior chamber
4. Angle of the Anterior chamber
5. Gonioscopic grading of Angle
6. Aqueous Outflow system
7. Trabecular meshwork
8. Functions of Aqueous Humor • Maintenance of Intraocular pressure • Metabolic role cornea lens vitreous and retina • Optical function • Clearing function
9. Physicochemical properties • volume: 0.31ml (0.25ml in Ant. Chamber and 0.06 in post chamber) • Refractive index: 1.336 • Density: slightly greater than water, its viscocity is 1.025-1.040 • Osmotic pressure: slightly hyperosmotic to plasma by 3-5mosm/l • PH: 7.2 • Rate of formation: 2-2.5microliter/min
10. Biochemical composition • Water: 99.9% • Proteins: 5-16mg/100ml • Amino acids: aqueous/plasma concentration varies from 0.08-3.14 • Non colloidal constituents: conc. of ascorbate, pyruvate, lactate in higher am
Vitreous humour
1. Vitreous Humour
2. General features Vitreous humour is an inert ,transparent , colourless, jellylike, hydrophilic gel that serves the optical functions and also acts as important supporting structures for the eyeball. The vitreous cavity is bounded by anteriorly by the lens and ciliary body and posteriorly by the retina Its weighs nearly 4g Vitreous is an extacellular material composed of approximately 99 per cent water
3. Structure The vitreous body is the largest and simplest connective tissue present as a single piece in the human body Divided into three parts- 1. The hyaloid layer or membrane 2. The cortical vitreous and 3. The medullary vitreous
Vitamins all
1. Vitamins. Definition - Organic compound required in small amounts. Vitamin A Vitamin B1, B2, B3, B5, B6, B7, B9, B12 Vitamin D Vitamin E Vitamin K A few wordsabout each.
2. Sourcesin diet - Many plants(photoreceptors), also meat, especially liver. Fat soluble, so you can get too much, or too littleif absorption isaproblem. Vitamin A - Retinol Retinol (vitamin A) Someuses: Vision (11-cis-retinol bound to rhodopsin detectslight in our eyes). Regulating genetranscription (retinoic acid receptorson cell nuclei arepart of a system for regulating transcription of mRNAsfor anumber of genes).
Tear film
1. TEAR FILM
2. The outer most layer of the cornea. It is the exposed part of the eyeball. FUNCTION It provide smooth optical surface It serves to keep the surface of cornea and conjunctiva moist. It serves as a lubricant for the preocular surface and lids It transfer oxygen from the air to the cornea Prevent infection due to the presence of antibacterial substance like lysozymes,and other protein. It wash away debris and irritants Provides pathway to WBC in case of injury.
3. LAYERS OF TEAR FILM It consist of three layers: 1.Lipid layer 2.Aqueous layer 3.Mucoid layer 1.LIPID LAYER
1. EVALUATION OF A CASE OF
NYSTAGMUS
Presenter-Himanshu Sapra
Moderator-Mrs-Sagun Jha
(Consultant Optometrist)
2. • DEFINITION
• TYPE OF NYSTAGMUS
• HISTORY
• HOW TO TAKE THE VISUAL ACUITY
• HOW TO MEASURE THE FREQUENCY
• HOW TO MEASURE THE AMPLITUDE
• WHAT IS NULL POINT
• WHAT IS NEUTRAL ZONE
• DOCUMENTATION
• TREATMENT
OBJECTIVE
3. DEFINITION
• Nystagmus is rhythmic rapidity to and fro movement of two eyes is called nystagmus
type of nystagmus described based on certain characteristics like
– rate (rapid or slow),
– amplitude ( coarse or fine),
– direction (horizontal ,
– vertical or rotational).
C L I N I C A L M A N A G E M E N T O F B i n o c u l a r V i s i o n Heterophoric, Accommodative, and Eye Movement D i s o r d
e r s(c) 2015 Wolters Kluwer. All Rights Reserved.
5. C L I N I C A L M A N A G E M E N T O F B i n o c u l a r V i s i o n Heterophoric, Accommodative, and Eye Movement D i s o r d e
r s(c) 2015 Wolters Kluwer. All Rights Reserved.
6. PATIENT HISTORY
• The usual case history must be expanded to include a number of additional questions
when evaluating patients with nystagmus.
• These include questions relating to the onset (i.e., at birth, before 6 months of age, or
specifically when).
• Association of the onset with any infection, drugs or medications, metabolic disease,
or trauma. Answers to such questions can help establish the often elusive cause of
nystagmus.
7. CONT….
• Information must be sought about observations by the parents or patient concerning
variability of the frequency, amplitude, position of gaze, or time characteristics of the
nystagmus.
• Symptoms should be assessed, especially those related to visual acuity, visual
discomfort, periodic blurring of vision, or oscillopsia.
• Except for the severely visually impaired, children with congenital nystagmus
seldom complain of visual symptoms.
8. HOW TO TAKE THE VISUALACUITY IN NYSTAGMUS
PATIENT
• Acuity testing of patients who read optotypes can proceed in the standard
manner.
• It is often more reliable to determine line or single-letter acuity with
children rather than to assess full-chart Snellen acuity.
• When visual acuity is reduced, a Bailey–Lovie chart (with logarithmic
proportionally spaced letters) or low vision chart may be needed.
• Determine acuity thresholds at distance (6 m) and at near (40 cm) for
each eye independently, as well as for binocular viewing.
9. CONT….
• Allow the patient to assume the preferred head position for
distance and near testing.
• Preschool children may need to be evaluated with hand-held
figure cards and it may not be possible to determine an exact
clinical acuity for children younger than 2 years. However, an
indication of visual ability can be determined by comparing
visual and hand–eye behavior with each eye.
• Monocular acuity differences are frequently seen in patients
with nystagmus.
10. CONT….
• Clinical differentiation needs to be between amblyopia (functionally reduced acuity in
one eye) and latent nystagmus (in which the nystagmus increases in amplitude with
occlusion).
• If there is high astigmatism, anisometropia, or strabismus— and amblyopia is
suspected—a comparison of the acuity thresholds may help make a differential
diagnosis.
• Latent nystagmus is characterized by an occlusion-induced increase in jerk nystagmus
that has a fast phase in the direction of the uncovered eye.
11. CONT….
• When nystagmus increases with occlusion, assess acuity using a method that does not
disassociate the eyes, use a plus lens and blur one eye while measuring the acuity of
the other eye.
12. TERMINOLOGIES
• Saccade/ Pursuit
• Jerk / Pendular
• Amplitude
• Frequency
• Null zone
• Neutral zone
• Conjugate/Disjugate
• Dissociated
13. DIRECTION
• PENDULAR MOVEMENT:-
• consist of to-and-fro eye movements of approximately equal
velocity in each direction . A pendular waveform can be sinusoidal
(smooth transition to the opposite direction) ortriangular (an abrupt
direction shift).
• Pendular nystagmus may be the horizontal and vertical
• JERKY MOVEMENT:-
• Jerk movements have both quick and slow components (Fig. 18.1A).
There may be intervals, known as foveations, in which the eye
movement is relatively slower for a short duration as the target
crosses the fovea and then the velocity increases.
14. • Right or left beating nystagmus.
• Upbeat or downbeat nystagmus
• Rotatory
15. AMPLITUDE
• Amplitude is the extent of excursion of the nystagmus.
• HOW TO MEASURE THE AMPLITUDE.
• The amplitude can be estimated using a millimeter ruler or a reticule in the magnifier
used to evaluate the type of nystagmus.
• As the patient fixates a target at 6 m, measurement of the overall excursion is made by
holding the ruler in front of the eye with the best acuity (either eye, if the movements
are conjugate).
16. • One millimeter of movement at the plane of the cornea translates to about 22 Δ (12
degrees of visual angle) . Therefore, if 2 mm of movement is noted, the eyes are
moving approximately 24 degrees.
• • Fine/ small : less than 5 degree.
• Medium/ moderate : 5 -15 degree.
• Coarse/ large : greater than 15 degree.
17. FREQUENCY
• • Frequency is the number of complete to and fro movements in one
second.
• HOW TO MEASURE THE FREQUENCY
• One Hertz (Hz) (1 cycle per second) means that the waveform
completes one full rotation in 1 second. Frequencies greater than 2
Hz are considered fast, and frequencies less than 1 Hz are
considered slow.
• Frequencies slower than 2 Hz can be timed with a stopwatch as the
oscillations are counted.
This picture taken from Association of Optometrists Article
18. • More rapid frequencies can be estimated with observation under low magnification of
the slit lamp.
19. NULL POINT
• The gaze position of least eye movement is the “null point” and tends to be where
vision is best. Tilting or turning the head into this direction where the movements are
least can thus optimize vision.
• NEUTRAL ZONE:-
• The neutral zone is that eye position in which a reversal of direction of jerk
nystagmus occurs and in which none of the several bidirectional waveforms, or
pendular nystagmus is present.
• It is not synonymous with the null zone.
• The null and neutral zones usually overlap.
20. HOW TO CORRECT THE ABNORMAL HEAD POSTURE
• The prisms are inserted with the base opposite the preferred direction of gaze.
• For instance, with a head turn to the left, the null zone is in dextroversion, and a prism
base-in before the right eye and base-out before the left eye will correct the head turn.
• Likewise, a compensatory chin elevation caused by a null zone in deorsumversion
will be improved with prisms base-up before each eye.
• A combination of vertical and horizontal prisms can be used when the null zone is in
an oblique position of gaze.
How to cite this article Kavitha Kalaivani N. An approach to Nystagmus management, Sci J Med & Vis Res Foun 2015;
XXXIII:138–140.