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   To measure the Visual Acuity
   To Examine Pupillary Reactions
   To test the functions of Extra Ocular
    muscles
   To Evaluate the Visual Fields
   To Inspect the optic nerve head
 Identifying life threatening or vision
  threatening neurological disorders
 Other systemic diseases
   Visual Acuity testing
   Visual Field testing
   Pupillary Reactions
   Ocular-motility testing
   Opthalmoscopy
 One’s visual acuity is an
  indication of the clarity or
  clearness of one’s vision
 Word “acuity” is derived from
  Latin acuitas, which means
  sharpness.
 Snellen’s Chart
   Visual field defects affect Central Vision

 Gross confrontation field testing.
 Amsler grid test
 Swinging flash light test
 Dysfunction of Sympathetic
 & Parasympathetic System.
 Optic nerve dysfunction
   To examine the occular
    fundus
Aneurysm compressing upon
CN3

                            Temporal lobe Herniation compressing
                            CN3
Argyll Robertson Pupil
   Causes: Cerebral Aneurysms, Vaso-
    occlusive disease, HTN, DM, Trauma
    & Brain tumors
   Cause: Close head trauma, Small vessel
    dieases, HTN, DM

 Vertical Diplopia
 Downward gaze difficulty
 Tilting of head
   Cause: Head Trauma,
    Small vessel disease,
    Viral infection, Increase in
    ICP

 Loss of abduction
 Horizontal diplopia
 Greatest separation of
  gaze towards the affected
  side
 CN V
 CN VII
 CN VIII
   Autoimmune condition
   Interefering Neuromuscular transmission
   Edrophonium Chloride/ Tencsilon test
   Ptosis
   Diplopia
   Fatigue
   3rd, 4th, 6th nerve diseases
   Gaze paresis
   Internuclear opthalmoplegia
 Lesion of medial longitudinal fasiculus
 Cause:
  Small vessel disease         Old age
  Demyelinating dieases          Adults
  Pontine Glioma               Children
   Spontaneous, Rhythmic back and forth
    movement of one or both eyes

 Horizontal, Vertical, Rotatory &
  Combination
 Indicates Central Nervous system
  dysfunction, Multiple sclerosis, Brain
  Tumor, Degenration of CNS
   Referred as
    Pseudopapilledema

   Blurring of disc margin
   Elevated disc substance
   Absence of optic cup
   Spontaneous venous pulsation
   Associated with Hyperopia
   Optic disc drusen
  Swelling of optic disc
  secondary to increased
  ICP
 Causes: Intracranial pressure, Brain tumor
   (50%), Pseudotumor cerebri, Severe HTN
 Bilateral & Good central vision
 Characterisitics: Hyperemia of disc,
   Tortous vessels, Blurring & Elevation of the
   margins, Hemorrhages
 Inflammatory edema
   of the disc
 Opthalmoscopically
   indistinguishable to Papilledema
 Unilateral, Decreases visual acuity,
  impaired color vision
 Pale, Swollen disc,
  Splinter Hemorrhages
 Loss of Acuity &
  Visual Field(Altitudinal)
 Predominantly involve Superior &
  Inferior field
 Transient monocular
  visual loss
 Cause: Arterial
  Insufficiency
 Pallor of the optic disc
 Causes: Previous optic neuritis, long
  standing papilledema, Compression by
  mass lesion, Small vessel disease, Giant
  cell Arthritis, Glaucoma
 Disc appears white
 Decrease Visual Acuity & Visual field loss
 Scotoma
 Hemianopia
 Homonymous Hemianopia
 Bitemporal Hemianopia
   Testing of both visual acuity & visual field is critical in the
    evaluation of abnormal optic disc
   A patient with a unilateral optic nerve lesion should have
    equal pupils in ambient light but a positive swinging flash light
    test
   Blurred disc margin isn’t diagnostic for pailledema, other sign
    n symptoms must be considered
   Chiasmal diseases most likely to cause bitemporal field
    defects
   If one of the following is abnormal-pupil, lid position or ocular
    motility- look closely for the involvement of the others
   CN V & CN VII should be checked if ocular motility is
    abnormal & Cranial nerve palsy is suspected
   Slowly progressive visual loss or CN palsy should prompt
    consideration of compressive lesions such as Tumor &
    Aneurysms
Neuro-opthalmology

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Neuro-opthalmology

  • 1.
  • 2. To measure the Visual Acuity  To Examine Pupillary Reactions  To test the functions of Extra Ocular muscles  To Evaluate the Visual Fields  To Inspect the optic nerve head
  • 3.  Identifying life threatening or vision threatening neurological disorders  Other systemic diseases
  • 4. Visual Acuity testing  Visual Field testing  Pupillary Reactions  Ocular-motility testing  Opthalmoscopy
  • 5.  One’s visual acuity is an indication of the clarity or clearness of one’s vision  Word “acuity” is derived from Latin acuitas, which means sharpness.  Snellen’s Chart
  • 6. Visual field defects affect Central Vision  Gross confrontation field testing.  Amsler grid test
  • 7.  Swinging flash light test  Dysfunction of Sympathetic & Parasympathetic System.  Optic nerve dysfunction
  • 8.
  • 9.
  • 10.
  • 11.
  • 12. To examine the occular fundus
  • 13.
  • 14. Aneurysm compressing upon CN3 Temporal lobe Herniation compressing CN3
  • 15.
  • 17.
  • 18. Causes: Cerebral Aneurysms, Vaso- occlusive disease, HTN, DM, Trauma & Brain tumors
  • 19. Cause: Close head trauma, Small vessel dieases, HTN, DM  Vertical Diplopia  Downward gaze difficulty  Tilting of head
  • 20. Cause: Head Trauma, Small vessel disease, Viral infection, Increase in ICP  Loss of abduction  Horizontal diplopia  Greatest separation of gaze towards the affected side
  • 21.  CN V  CN VII  CN VIII
  • 22. Autoimmune condition  Interefering Neuromuscular transmission  Edrophonium Chloride/ Tencsilon test  Ptosis  Diplopia  Fatigue  3rd, 4th, 6th nerve diseases  Gaze paresis  Internuclear opthalmoplegia
  • 23.  Lesion of medial longitudinal fasiculus  Cause: Small vessel disease Old age Demyelinating dieases Adults Pontine Glioma Children
  • 24.
  • 25. Spontaneous, Rhythmic back and forth movement of one or both eyes  Horizontal, Vertical, Rotatory & Combination  Indicates Central Nervous system dysfunction, Multiple sclerosis, Brain Tumor, Degenration of CNS
  • 26.
  • 27.
  • 28. Referred as Pseudopapilledema  Blurring of disc margin  Elevated disc substance  Absence of optic cup  Spontaneous venous pulsation  Associated with Hyperopia  Optic disc drusen
  • 29.  Swelling of optic disc secondary to increased ICP  Causes: Intracranial pressure, Brain tumor (50%), Pseudotumor cerebri, Severe HTN  Bilateral & Good central vision  Characterisitics: Hyperemia of disc, Tortous vessels, Blurring & Elevation of the margins, Hemorrhages
  • 30.  Inflammatory edema of the disc  Opthalmoscopically indistinguishable to Papilledema  Unilateral, Decreases visual acuity, impaired color vision
  • 31.  Pale, Swollen disc, Splinter Hemorrhages  Loss of Acuity & Visual Field(Altitudinal)  Predominantly involve Superior & Inferior field
  • 32.  Transient monocular visual loss  Cause: Arterial Insufficiency
  • 33.  Pallor of the optic disc  Causes: Previous optic neuritis, long standing papilledema, Compression by mass lesion, Small vessel disease, Giant cell Arthritis, Glaucoma  Disc appears white  Decrease Visual Acuity & Visual field loss
  • 34.  Scotoma  Hemianopia  Homonymous Hemianopia  Bitemporal Hemianopia
  • 35. Testing of both visual acuity & visual field is critical in the evaluation of abnormal optic disc  A patient with a unilateral optic nerve lesion should have equal pupils in ambient light but a positive swinging flash light test  Blurred disc margin isn’t diagnostic for pailledema, other sign n symptoms must be considered  Chiasmal diseases most likely to cause bitemporal field defects  If one of the following is abnormal-pupil, lid position or ocular motility- look closely for the involvement of the others  CN V & CN VII should be checked if ocular motility is abnormal & Cranial nerve palsy is suspected  Slowly progressive visual loss or CN palsy should prompt consideration of compressive lesions such as Tumor & Aneurysms