ATAXIA IN CHILDREN -CAUSES, MANAGEMENT, INVESTIGATIONS, TYPES, COMMONEST ATAXIA IN CHILDREN IN DETAIL, HOW WILL YOU FIND OUT THE CAUSE FOR ATAXIA IN CHILDREN FLOWCHART, DEFINITION, TREATMENT
ATAXIA IN CHILDREN -CAUSES, MANAGEMENT, INVESTIGATIONS, TYPES, COMMONEST ATAXIA IN CHILDREN IN DETAIL, HOW WILL YOU FIND OUT THE CAUSE FOR ATAXIA IN CHILDREN FLOWCHART, DEFINITION, TREATMENT
Surgical approach to thalamus explained in details their surgical anatomy and lesion, Preop post op results with different surgical approach for thalamic lesions
Preoperative assessment of epilepsy clinical & radiological, eeg, megDr Fakir Mohan Sahu
Preoperative Assessment of Epilepsy and their localization, clinical semiology and Radiological evaluation MRI imaging, Video EEG MEG SPECT PET and patient management conference explained in simplified way
General Basic knowledge of Brain tumour explained in brief of classification, pathogenesis, clinical features, CT, MRI, management, Radiotherapy. Best for MBBS and PG preparation student.
Facial and Hearing Preservation in Acoustic Neuroma SurgeryDr Fakir Mohan Sahu
Vestibular Schwannoma Most common CPA (Cerebellopontine angle) tumor changed from prolongation of life to nerve preservation explained in brief with all pre- operative work up.
Details of Cerebrospinal Fluid special reference to cell count and alteration of CSF Hydrodynamics explained in brief and Different Diagnostic parameters to Hydrocephalus
263778731218 Abortion Clinic /Pills In Harare ,sisternakatoto
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New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
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2 Case Reports of Gastric Ultrasound
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Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
2. Outlines
• History
• Anthropometry/Development assessment
• Examining of head / Dysmorphic Child
• The neurological examination
• HMF
• Cranial Nerves
• Motor system
• Sensory system
• Reflexes
• Signs of meningeal irritation
• Cerebellar signs
• Pearls on examining infants and Older Child
3. Introduction
• The CNS in a children is a dynamic, developing and maturing system
• A daunting task to assessment of CNS
• Special tricks and adoption of “play attitude” is mandatory
• History is often imprecise as child cannot explain or express
4. Normal Neurologic Growth and Development
• What additional questions are important for a complete pediatric neurology
history?
– Antenatal
– Perinatal
– Neonatal Complications
– Neurodevelopment
– Immunizations
– Behaviour
– Family History
– Social History
5. Key points in History
• Presenting symptom
• Onset: Sudden/ Subacute/ Insidious
• Evolution: Improving/Slow Progression/Rapid progression
• Any symptom of raised ICP/Seizure/LOC
• Development before onset of symptom
• Mental status- Alert/ irritable/ lack of interest/ drowsy/ stuporous/ comatose
• Feeding History/Family history/ Consanguinity
• Etiological history- Perinatal events/trauma/drugs toxin/infections if any
6. Principles and art of Examination
• Pre-requisite/ Essential tools
• Setting- well lighted room, colourful comfortable, warm hands
• Position
• 0-3 months – Examination table
• 3mont – 1 yr - Mothers lap
• 1-3 yr -Standing / Mothers lap
• After 3 yr - Examination table
• Adolescent girl - Female attendants
7. General Physical Examinations
• General appearance/Posture/ nature of disability/ Build and Nutrition
• Handedness(established around 3 years of age)
• Anthropometry
• Skull and Spine- Size /shape /sutures/ fontanels/ spinal deformities
• Skin and Appendages –Neuroectodermal dysplasia: Adenoma sebaceum/café –au-lait/ shagreen
Patch/ Swelling
• Vitals Sign- Anaemia/Cyanosis/ Icterus/ Clubbing/ LN pathy/ Organomegaly
18. Developmental screening
• Gross motor Development
• Fine motor/ Visual
• Social/ Adaptive and language Development
• Red Alerts
19.
20.
21.
22.
23.
24. Red Alerts
• Lack of Social smile by 2 months
• Absence of stable head control by 4 months
• Inability to recognize the mother by 6 months
• Inability to sit when pulled to sit by 6 months / independent sitting by 8 months
• Lack of creeping by 9 months
• Inability to stands without support by 1yr
• Inability to walk without support by 18 months
• Lack of pincer grasp by 1 yr
• Absence of syllabic babbling by the age of 1 yr
• Failure to make meaningful sentences by 3 years
25. Anthropometry
• Weight
• Length/ Height
• Head circumference
• Assessment of - Mid arm circumference/ Subcutaneous fat/ Arm span/
Obesity
26. Head Circumference
• Brain growth takes place 70% during fetal life, 15% during infancy and
remaining 10% during pre-school years.
• Head circumference are routinely recorded until 5 years of age.
• If scalp edema or cranial moulding-
measurement of scalp edema may be inaccurate until fourth or fifth day
27. Expected head circumference in children
• Head Circumference Growth Velocity
Till 3 months 2 cm/month
3 months – 1 year 2cm/3 month
1 – 3 year 1cm/ 6 month
3 – 5 year 1cm/ year
• During first year there is 12 cm increase in head
circumference , while 1 – 5 year age , only 6 cm gain
occur in head size.
Age Head circumference (cm)
At birth 33 – 35
2 months 38
3 months 40
4 months 41
6 months 42 - 43
1 year 45 - 46
2 years 47 - 48
5 years 50 - 51
30. Head Examinations
• Size/ Shape/ Symmetry/Sutures
• Fontanels-Depressed/ Flat/ Buldged
• Six at birth
• Two large ant/post
• Anterior fontanel (2.5×2.5cm) closes by 12-18 mnths
• Posterior fontanel small at birth closes by 6 weeks
• Two Anterolateral/ two posterolateral
40. Higher Mental Function
• Emotional status- Assess behaviour/ perception and emotional
liability/ hyperactivity/ attention/ distractibility/ impulsiveness
• Memory and Orientation
• Immediate/Recent/ Remote(above 5yrs)
• Time / Place / Person
• Name of school/ teacher/ father/ friends
• Ability to obey simple commands
• Tell a brief story and repeat the same
• Repeat no forward/ Backward
6yr repeat forward five digits and backward three digits,
10 yr six digits forward and four digits backward
41. Speech
• Aphonia/Dysphonia- Volume or intensity of speech affected
• A child is asked to blow out a candle or count 1 -100
• Wernicke’s aphasia/ Broca’s Aphasia/ Dyslexia(slowness in reading, mirror
image writing, reading from right to left)
• Disorders of Articulation- “Pa” “Ta” “Ka”
• Stammering/ lalling or baby speech
• Scanning and Staccatto speech/Slurred
• Spastic / Slurring dysarthria
• Nasal Speech
• Autism Spectrum disorders
42. Signs of Meningeal irritations
• Absent-infants <3 months/ malnourished child/ seriously sick pt.
Early Sign Late Sign
45. Cranial nerve exam:
Olfaction CN(I)
• Olfactory sensation as transmitted by the olfactory nerve is not
functional in the newborn
• Present by 5 to 7 months of age.
• Anosmia/ Parosmia
46. Cranial Nerve-
• Optic nerve(II)
Visual Acuity-
Infants Blinking response/ Turning of head towards light
Above 3 yr – E Chart
Field of vision
tested after 3yrs(confrontation)
Perimetry feasible after 8-9 yrs
Colour vision
above 3 yr
Fundus exam
Optic disc infant- pale
Papilledema/Papillitis- elevated disc,
blurred edges, obliteration of physiological cuff
“E” chart
47. III, IV, VI CN
• Movements are complete in all directions by
approximately 4 months of age,
• Acoustically elicited eye movements appear at 5
months of age .
• Ptosis/ Diplopia
• Head tilt
• Dolls eye movement Infant
• Depth perception using solely binocular cues
appears by 24 months of age
• Stable binocular alignment and optokinetic
nystagmus.
49. Facial (VII) CN
• Impaired motor function is indicated by facial asymmetry.
• The McCarthy reflex, ipsilateral blinking produced by tapping the supraorbital
region, is diminished or absent in lower motor neuron facial weakness.
• Palpebral reflex, bilateral blinking induced by tapping the root of the nose, it
can be exaggerated by upper motor neuron lesions.
• In hemiparesis or peripheral facial nerve weakness, the contraction of the
platysma muscle is less vigorous on the affected side, This sign also carries
Babinski's name.
• Failure to pull the affected side of the mouth backward and downward when
crying.
• Sensory-----Ant.2/3 of tongue.
50. Vestibulocochlear(VIII)
Cochlear part(hearing)
• At birth ---Moro reflex.
• Younger deviate to sound.
• Later Rinne s test+ Weber test.
Vestibular part(Vertigo+ Nystagmus)
• Can be assessed easily in infants or small children by holding the youngster
vertically so he or she is facing the examiner, then turning the child several
times in a full circle.
• Clockwise and counterclockwise rotations are performed. The direction and
amplitude of the quick and slow movements of the eye are noted.
52. IX, X, XI CN
• Sensory ……loss of post 2/3 of tongue.
• Motor……pharyngeal O/E….
• 1-gag reflex…absent in bulber palsy UMNL
exaggarated in pseudo bulber palsy LMNL.
• 2-Uvula ….normally central & mobile.
• In unilateral lesion….uvula deviate to healthy side.
• In bilateral lesion…uvula is central but immobile.
Spinal accessory N.
• Sternomastoid-ability to rotate head to healthy side.
• Trapezius-dropping of shoulder in affected side
55. Muscle tone
• Hypotonia is characterized by decreased resistance to passive
movement and hyperextension at the joints.
• Hypertonia can be either spastic in nature or characterized by muscle
rigidity.
• UMNL =Pyramidal lesion….. spasticity(clasp knife) resistance on the start of movement.
• Extrapyramidal lesion….. rigidity(resistance is all over movement (cog-weal or lead)
56. Muscle power
• 1-Young child…….painful stimulation on the opposite side of the
tested muscle.
• 2-Older child….ask to move against resistance.
• 3-Test every joint for its muscle group.
• 4-Grading of muscle power
58. Involuntary movements
Usually with extrapyramidal lesion.
• Chorea….sudden irregular purposeless
dancing movement affect big proximal joint.
• Athetosis…slow twisting movement affect
distal joint.
• Dystonia….slow twisting movement in trunk.
• Tremors….rapid alternating movement
around small joint.
59. Incoordination
• 1st year ……grasp reflex & object transfer.
• -2nd year……button & unbutton.
• ->3years……U.L.
• 1- Finger to nose test
• 2-Finger to finger test
• 3-Dysdiadochokinesis…inability to perform rapidly
alternating movement
• 4-Rebound test L.L.
Heal to shin test
Toe finger test
Foot Tapping test
Inco-ordination = ataxia.
60. Sensory system
• Sensory examination in young children is often imprecise, and only
gross deficits can be detected.
• In children > 5 -6 years sensory function is evaluated in the same
manner as in an adult
• Touch
• Pain & Temperature
• JPS
61. Pearls on examining infants and Older Child
• Accuracy depends upon the observational ability and intelligence
• Concerns of Parents/ Attendants
• Dietary/ Immunization/ Perinatal/ Developmental history
• Approach of examination Unstructured
(unpleasant examination postponed to the end)
• Signs of meningeal irritation may be minimal or absent during first year
of life (especially first 3 months) and in malnourished children
62. Pearls on examining infants and Older Child
• Primitive reflexes are present in birth disappear by 4-5 months
• Developmental screening is a must/ early markers of Cerebral palsy
• Deep tendons reflexes are normally brisk during infancy
• KJ crossed adductor response/ Cremasteric reflex
• Plantar normally extensors up to 2yrs
• Fundus normally pale in infants/ Papilledema appears after 3 yrs