The document summarizes India's National Leprosy Eradication Programme. It discusses that leprosy is caused by Mycobacterium leprae bacteria and mainly affects the skin and peripheral nerves. The key milestones of the programme included introducing multi-drug therapy in 1982 and achieving elimination at the national level in 2005. The current strategies include integrating leprosy services into general healthcare, promoting early detection and complete treatment, involving ASHA workers, and reducing stigma through information campaigns. The goal is to continue driving down prevalence rates toward total eradication of the disease in India.
National Leprosy Eradication Programme (NLEP)Kavya .
Chronic infectious disease caused by Mycobacterium leprae.
It usually affects the skin and peripheral nerves
Long incubation period generally 5-7 years.
Classified as paucibacillary or multibacillary
permanent disability
Timely diagnosis and treatment of cases
This ppt contains all the information about Revised NationalTuberculosis Control programme (RNTCP) It is useful for students of the medical field learning Preventive and social medicine, Swasthavritta (Ayurved) and everyone who is interested in in knowing about it.
National Leprosy Eradication Programme (NLEP)Kavya .
Chronic infectious disease caused by Mycobacterium leprae.
It usually affects the skin and peripheral nerves
Long incubation period generally 5-7 years.
Classified as paucibacillary or multibacillary
permanent disability
Timely diagnosis and treatment of cases
This ppt contains all the information about Revised NationalTuberculosis Control programme (RNTCP) It is useful for students of the medical field learning Preventive and social medicine, Swasthavritta (Ayurved) and everyone who is interested in in knowing about it.
This ppt contains all the information about National Leprosy Eradication programme (NLEP). It is useful for students of the medical field learning Preventive and social medicine, Swasthavritta (Ayurved) and everyone who is interested in in knowing about it
On 19 November 1985, GOI renamed EPI program, modifying the schedule as ‘Universal Immunization Program’ dedicated to the memory of Late Prime Minister Mrs Indira Gandhi.
UIP has two vital components: immunization of pregnant women against tetanus, and immunization of children
Pulse Polio is an immunisation campaign established by the government of India to eliminate poliomyelitis (polio) in India by vaccinating all children under the age of five years against the polio virus.
This PPT has all the necessary information about 'National Rural Health Mission'. It is useful for students of Medical field learning 'Preventive & Social Medicine' as well as anyone who is interested in knowing about it.
Copyright Disclaimer - Use of these PowerPoint Presentation for any commercial purpose is strictly prohibited. The presentations uploaded on this profile are protected under Copyright Act,1957.
This ppt contains all the information about National Leprosy Eradication programme (NLEP). It is useful for students of the medical field learning Preventive and social medicine, Swasthavritta (Ayurved) and everyone who is interested in in knowing about it
On 19 November 1985, GOI renamed EPI program, modifying the schedule as ‘Universal Immunization Program’ dedicated to the memory of Late Prime Minister Mrs Indira Gandhi.
UIP has two vital components: immunization of pregnant women against tetanus, and immunization of children
Pulse Polio is an immunisation campaign established by the government of India to eliminate poliomyelitis (polio) in India by vaccinating all children under the age of five years against the polio virus.
This PPT has all the necessary information about 'National Rural Health Mission'. It is useful for students of Medical field learning 'Preventive & Social Medicine' as well as anyone who is interested in knowing about it.
Copyright Disclaimer - Use of these PowerPoint Presentation for any commercial purpose is strictly prohibited. The presentations uploaded on this profile are protected under Copyright Act,1957.
After the successful NSP 2017-2025,Goi is lauching NSP 2017-2025 for elimination of TB on 24th march( World TB day ) 2017. Module is on MOHFW site but i have try to keep it brief,hope its ll be useful specially for academic and administrative purposes.
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Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
2. Leprosy
• Mycobacterium leprae
• Mainly involves peripheral nerves and skin
• Cardinal features
• Hypopigmented patch
• Loss of cutaneous sensation
• Thickened nerve
• Acid fast bacilli
• Based on no. of skin lesions and nerves involved
• Paucibacillary
• Multibacillary
3. Problem statement
• WORLD : According to WHO data
• 2,15,656 new cases detected during 2013
• 1,80,618 cases registered prevalence at beginning
of 2014
• Among new cases 71.68% multibacillary
• Prevalence rate dropped from 21.1 cases per
10,000 in 1985 to 0.32 case per 10,000 population
at beginning of 2014
• Leprosy has been eliminated from 119 of 122
countries
4.
5.
6. Milestones in NLEP
1955 - National Leprosy Control Programme.
1970s - Introduction of Multi Drug Therapy. National
programme remained with Dapsone treatment.
1982 – Introduction of MDT under National Programme.
1983 - National Leprosy Eradication Programme (NLEP)
1991 - World Health Assembly resolved to eliminate
leprosy at a global level by the year 2000.
1993-2000- The 1st phase of the World Bank
supported National Leprosy elimination Project
7. Milestones in NLEP (Continue)
1998-2004- Modified Leprosy Elimination Campaign
2001-2004- The 2nd phase of the World Bank supported
National Leprosy elimination Project
2005 - India achieved elimination of Leprosy as a Public
Health Program at National Level.
2012 – PIP under 12th plan
9. Programme Strategy
1. Integrated leprosy services through General
Health Care system.
2. Early detection & complete treatment
3. Carrying out house hold contact survey for early
detection of cases.
4. Involvement of Accredited Social Health
Activist (ASHA) in the detection & completion of
treatment of Leprosy cases on time.
10. 5. Strengthening of Disability Prevention & Medical
Rehabilitation (DPMR) services.
6. Information, Education & Communication (IEC)
activities in the community to improve self-
reporting to Primary Health Centre (PHC) and
reduction of stigma.
7. Intensive monitoring and supervision at block
Primary Health Centre/Community Health Centre.
11. Institutions
• Four premier Leprosy Institutes
A.Central Leprosy Teachning & Research Institute
Chengalpattu (Tamilnadu)
B. Regional Leprosy Training & Research Institute
Raipur (Chhattisgargh)
C. Regional Leprosy Traning & Research Institute
Aska (Orissa)
D. Regional Leprosy Training & Research Institute
Gouripur, Bankura (West Bengal)
12. 1a.DECENTRALIZATION OF NLEP
SERVICES
• State level societies are formed & funding to districts
is done by these.
• In smaller states/UTS - district societies
13. 1b.INTEGRATION OF NLEP WITH
GENERAL HEALTH CARE SYSTEM
• Integration means to provide “comprehensive”
essential services from one service point:
ADVANTAGES:
• Patients detected early
• Patients treated early
• Transmission of infection interrupted early
• Development of deformities prevented
• Stigma reduced further
NRHM & NLEP:
Link person-ASHA
Performance based incentive:
14. 2. Early detection & complete treatment
(i) To improve access to services.
(ii) To involve women including leprosy affected persons in
case detection.
(iii) To organize skin camps for detecting leprosy patients
while providing services for other skin conditions.
(iv)To undertake contact survey to identify the source in
the neighbourhood of each child or M.B. case.
(v) To increase awareness through the ANM, AWW,
ASHA and other Health Workers
15. 2.Multi Drug Therapy (MDT)
• Highly effective cure
• Combination of 2-3 drugs :
Clofazimine, Rifampicin,
Dapsone
• Cure patient in 6
month(PB)/12 month(MB)
• Kill the lepra bacilli and
stop transmission
• Can be delivered without
special staff and institution
• Available free of cost
16. 4. ASHA Involvement
• To bring out suspected cases from village for
diagnosis and treatment
• Receive incentives
• At confirmation and diagnosis - 250/-
• On completion of full treatment –
• Additional 400/-(PB)
• Additional 600/-(MB)
• Activities to be performed by ASHA
• Search for suspected case
• Follow up all cases
• Advice and motivate, self care practices
• Spreading awareness .
17. 5. Disability Prevention & Medical
Rehabilitation (DPMR)
Main activities carried out under DPMR are as follows :
• Integrating DPMR services with NRHM
• Dressing material, supporting medicine, ulcer kit to
leprosy affected person
• Microcellular rubber footwear
• NGOs, Medical Colleges strengthen for Reconstructive
surgery for correction of disability
• Amount of 5000/- for leprosy affected person from
BPL family undergoing Reconstructive Surgery
• Support to Govt. Institution in form of 5000/- per
reconstructive surgery conducted
• To develop a referral system
18. Disability Prevention & Medical Rehabilitation (DPMR)
Primary level care
• PHC
• CHC
• Sub Divisional Hospital
• Urban Leprosy
Centers/Dispensaries
Secondary level care
• All District hospitals
• District nucleus unit
Tertiary level care
• Central Govt. Institute
• ICMR Institute JALMA, Agra
• ILEP supported leprosy hospitals
• All PMR institute and Dept. of
Medical College
DPMR planned to be carried out in a 3 tier system :-
19.
20. .
0 1 2
EYES Normal
vision,lid
gap,blinking.
Corneal reflex
weak
Reduced vision,
lagophthalmos.
HANDS Normal
sensation &
m.power.
Loss of feeling
in the palm
Visible damage:
wounds, claw
hand, loss of
tissue etc.
FEET Normal
sensation &
m.power.
Loss of feeling
in the sole
Visible damage:
wound, foot
drop, loss of
tissue.
WHO DISABILITY GRADING
25. 6. IEC :Information Education &
Communication
Focus on –
• Behaviour change in community against stigma and
discrimination against leprosy affected person
• Making the public aware about the
• Availability of MDT
• Correction of deformity through surgery
• Leprosy affected person can live a normal life
with family
26. 7. MONITORING & EVALUATION
• PRIMARY INDICATOR:
-Annual New Case Detection Rate (ANCDR)
-Treatment Completion Rate (cohort analysis)
27. • INDICATORS FOR CASE DETECTION:
- Proportion of new cases with Gr II disability
- Proportion of child cases(15yrs) among new cases
- Proportion of MB cases among new cases
- Proportion of Female cases among new cases
• INDICATORS FOR QUALITY OF SERVICE:
- Proportion of new cases correctly diagnosed.
- Proportion of defaulters.
- Number of relapses during a year.
- Proportion of cases with new disabilities.
28. MODIFIED LEPROSY ELIMINATION
CAMPAIGN (MLEC)
• Organising camps for 1 or 2 wks duration
• Services available:
case detection, Tt & referral
• Mass media
• Quite effective in case finding & has been employed
during phase-II.
• Five MLEC rounds conducted
29. BLOCK LEPROSY AWARENESS
CAMPAIGNS (BLAC)
• Carried out for 15 days in identified priority areas
during Sep-Nov each yr.
• Made huge impact on:
oHidden case detection
oBetter case management
oImprovement in spreading the awareness
oBringing down PR in high endemic areas.
30. SPECIAL ACTION PROJECTS FOR THE
ELIMINATION OF LEPROSY(SAPEL)
• For people living in special difficult to access areas or
situation or neglected communities.
• Strategies:
early detection & prompt MDT with proper IEC.
31. LEPROSY ELIMINATION
CAMPAIGNS(LEC) FOR URBAN AREAS
• GOI provides assistance to urban areas with 1lakh
population.
• Activities done
• Identify human resources available with Govt., Civil
societies, NGOs and Private Medical Practitioners
for leprosy services
• Build capacity
• Examination of all household contacts of all new
cases at least once before the completion of
treatment of index case.
• Identify one referral centre
• Mobile Health Clinics
• System of record keeping and reporting
• regular MDT supply
• sensitization meetings for IEC and advocacy
Activities to be performed by ASHAs
(i) Search for suspected cases of leprosy i.e. before any sign of disability appears. Such early detection will help in prevention of disability and also cut down transmission potential.
(ii)Follow up all cases for completion of treatment in scheduled time. During follow up visit also look for symptoms of any reaction due to leprosy and refer them to the Health Workers/PHC for treatment. This will again reduce chances of disability occurring in cases under treatment.
(iii) Advise and motivate self-care practices by disabled cases for proper care of their hands and feet during the follow up period. This will improve quality of life of the affected persons and prevent deterioration of disabilities.
(iv) Spreading awareness.