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Myocardial Infarction
Dr. Nimra Wazeer PT
DPT (UMT)
Contents:
 Definition
 Types of infarcts
 Epidemiology
 Etiology
 Etiopathogenesis
 Pathophysiology
 Clinical manifestation
 Diagnosis
 Management:
 Non- Pharmacological
 Pharmacological
Myocardial Infarction:
 MI is defined as a diseased
condition which is caused by
reduced blood flow in a coronary
artery due to atherosclerosis &
occlusion of an artery by an
embolus or thrombus.
 MI or heart attack is the irreversible
damage of myocardial tissue caused
by prolonged Ischemia & Hypoxia.
Types of infarcts:
 According to anatomic region of left ventricle involved:
 Anterior
 Posterior
 Lateral
 Septal
 Circumferential
 Combinations: Anterolateral, Posterolateral, Anteroseptal
 According to degree of thickness of ventricular wall involved:
 Transmural (Full thickness)
 Laminar (Sub endocardial)
 According to age of infarcts:
 Newly formed (acute, recent, fresh)
 Advanced infarcts (old, healed)
Epidemiology:
 In Industrial countries MI count for 10-25% of all deaths.
 Incidence is higher in elderly people, about 5% occurs in people under
age 40.
 Males have higher risk.
 Women during reproductive period have low risk.
 Over last 30 years, the rate of diseases increased from 2-6% in rural
population and 4-12 % in urban population.
Etiology:
 Tobacco smoking
 Hypertension
 Drug abuse
 Obesity
 Stress
 Alcohol
 Age
 Gender
 Diabetes
 Hyperlipoproteinemia
 Family history
 Hyperhomocysteinemia
 Chronic kidney diseases
Etiopathogenesis:
 Mechanism of myocardial ischemia.
 Role of platelets
 Acute plaque rupture
 Non atherosclerotic causes
 Transmural versus subendocardial infarcts
Pathophysiology
Clinical manifestation:
 Chest pain/Chest discomfort
 Dyspnea
 Fatigue
 Other symptoms include:
 Increased sweating
 Weakness
 Nausea
 Vomiting
 Light headedness
 Palpitation
 Anxiety, sleeplessness, hypertension or hypotension, arrhythmia
 Chest pain is less in women, their common symptoms are weakness, fatigue &
dyspnea
Complications:
 Arrhythmia
 Cardiogenic shock (10%)
 Congestive heart failure
 Thromboembolism
 Rupture (5%)
 Cardiac aneurism (5%)
 Pericarditis
Diagnosis:
 Clinical features:
 Pain
 Indigestion
 Apprehension
 Shock
 Lower grade fever
 Serum cardiac markers:
 Creatinine phosphokinase (CK)
 Lactic dehydrogenase (LDH)
 Cardiac specific troponins (cTn)
 ECG changes:
 ST segment elevation
 T wave inversion
 Appearance of wide deep Q waves
 Magnetic resonance imaging
 Angiography
 Positron emission tomography (PET Scan)
 Chest Xray
Management:
 Non- Pharmacological:
 Counselling and education of patients
 Life style measures
 Smoking cessation
 Avoid alcohol intake
 Diet and nutrition
 Salt restriction
Pharmacological:
 Thrombolytic agents
 Anticoagulants
 Antiplatelet agents
 Antihypertensive agents
 Lipid lowering drugs
 Vasodilators
 Others
 Analgesics
 Antiulcer drugs
 Antidepressants
Thank
You 

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Myocardial Infarction.pptx

  • 1. Myocardial Infarction Dr. Nimra Wazeer PT DPT (UMT)
  • 2. Contents:  Definition  Types of infarcts  Epidemiology  Etiology  Etiopathogenesis  Pathophysiology  Clinical manifestation  Diagnosis  Management:  Non- Pharmacological  Pharmacological
  • 3. Myocardial Infarction:  MI is defined as a diseased condition which is caused by reduced blood flow in a coronary artery due to atherosclerosis & occlusion of an artery by an embolus or thrombus.  MI or heart attack is the irreversible damage of myocardial tissue caused by prolonged Ischemia & Hypoxia.
  • 4.
  • 5. Types of infarcts:  According to anatomic region of left ventricle involved:  Anterior  Posterior  Lateral  Septal  Circumferential  Combinations: Anterolateral, Posterolateral, Anteroseptal  According to degree of thickness of ventricular wall involved:  Transmural (Full thickness)  Laminar (Sub endocardial)  According to age of infarcts:  Newly formed (acute, recent, fresh)  Advanced infarcts (old, healed)
  • 6. Epidemiology:  In Industrial countries MI count for 10-25% of all deaths.  Incidence is higher in elderly people, about 5% occurs in people under age 40.  Males have higher risk.  Women during reproductive period have low risk.  Over last 30 years, the rate of diseases increased from 2-6% in rural population and 4-12 % in urban population.
  • 7. Etiology:  Tobacco smoking  Hypertension  Drug abuse  Obesity  Stress  Alcohol  Age  Gender  Diabetes  Hyperlipoproteinemia  Family history  Hyperhomocysteinemia  Chronic kidney diseases
  • 8. Etiopathogenesis:  Mechanism of myocardial ischemia.  Role of platelets  Acute plaque rupture  Non atherosclerotic causes  Transmural versus subendocardial infarcts
  • 10. Clinical manifestation:  Chest pain/Chest discomfort  Dyspnea  Fatigue  Other symptoms include:  Increased sweating  Weakness  Nausea  Vomiting  Light headedness  Palpitation  Anxiety, sleeplessness, hypertension or hypotension, arrhythmia  Chest pain is less in women, their common symptoms are weakness, fatigue & dyspnea
  • 11. Complications:  Arrhythmia  Cardiogenic shock (10%)  Congestive heart failure  Thromboembolism  Rupture (5%)  Cardiac aneurism (5%)  Pericarditis
  • 12. Diagnosis:  Clinical features:  Pain  Indigestion  Apprehension  Shock  Lower grade fever  Serum cardiac markers:  Creatinine phosphokinase (CK)  Lactic dehydrogenase (LDH)  Cardiac specific troponins (cTn)
  • 13.  ECG changes:  ST segment elevation  T wave inversion  Appearance of wide deep Q waves  Magnetic resonance imaging  Angiography  Positron emission tomography (PET Scan)  Chest Xray
  • 14. Management:  Non- Pharmacological:  Counselling and education of patients  Life style measures  Smoking cessation  Avoid alcohol intake  Diet and nutrition  Salt restriction
  • 15. Pharmacological:  Thrombolytic agents  Anticoagulants  Antiplatelet agents  Antihypertensive agents  Lipid lowering drugs  Vasodilators  Others  Analgesics  Antiulcer drugs  Antidepressants
  • 16.
  • 17.
  • 18.