The document discusses flexor tendon injuries, including anatomy, classification by zones, surgical techniques for repair, and postoperative rehabilitation. It covers the superficial and deep flexor tendon groups, pulley system anatomy and its importance, and surgical approaches and repair methods for injuries in different zones of the hand. Primary goals of repair include restoring tendon continuity and gliding while preventing adhesions through techniques like circumferential suturing.
anatomy of median nerve,course in arm and struthers ligament, branches in the forearm, carpal tunnel and course in hand, high and low median nerve injuries, principles of surgical management, pronator teres syndrome, anterior interosseous nerve syndrome, open and endoscopic carpal tunnel release
Hand rehabilitation following flexor tendon injuriesAbey P Rajan
hand rehabilitation following flexor tendon injuries include introduction, clinical anatomy, tendon nutrition, tendon healing, post op. management, special cases, summary
A fingertip injury is defined as any soft tissue, nail or bony injury distal to the dorsal and volar skin creases at the distal interphalangeal joint and insertions of long flexor and extensor tendons of a finger or thumb.
The fingertips are exposed to all aspects of daily living,
recreation and work and it is perhaps no surprise they
are the most commonly injured part of the hand
anatomy of median nerve,course in arm and struthers ligament, branches in the forearm, carpal tunnel and course in hand, high and low median nerve injuries, principles of surgical management, pronator teres syndrome, anterior interosseous nerve syndrome, open and endoscopic carpal tunnel release
Hand rehabilitation following flexor tendon injuriesAbey P Rajan
hand rehabilitation following flexor tendon injuries include introduction, clinical anatomy, tendon nutrition, tendon healing, post op. management, special cases, summary
A fingertip injury is defined as any soft tissue, nail or bony injury distal to the dorsal and volar skin creases at the distal interphalangeal joint and insertions of long flexor and extensor tendons of a finger or thumb.
The fingertips are exposed to all aspects of daily living,
recreation and work and it is perhaps no surprise they
are the most commonly injured part of the hand
Basic principle of Knee Joint arthroscopy and techniques for beginners. Basic Steps of Knee Joint Diagnostic arthroscopy and common complication following knee joint arthroscopy.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
1. Flexors Tendon Injuries
Moderator : Assoc Prof Dr Suresh Uprety
Presenter : Dr Ajay Shah (Resident)
Department of Orthopedics and Trauma Surgery
IOM,TUTH
4. Zones of Flexors Tendon
Verdan Classification
• Zone I: From middle of middle phalanx distally
• Zone II: From MCP joints to middle of middle
phalanx
• Zone III: Central palm
• Zone IV: Tendons in carpal tunnel
• Zone V: Tendons proximal to carpal tunnel
Thumb Zone
• TI: Distal to IP joint
• TII: From A1 pulley to IP joint
• TIII: Thenar eminence
10. Camper’s chiasm
• At A1 pulley FDS tendon splits and
rotates laterally then dorsally around
FDP tendon
• Two splits tendon join together
proximal to PIP joint at camper’s
chiasm
• Functions
Provides a pathway for FDP tendon
Increases the stability and balance of
PIP joint
Prevents hyperextension of PIP joint
11. No Man’s Land
• Historic derivation of “no man's land”
dates back to 14th century
• It was used to describe an area outside
London used for executions
• Sterling Bunnel used this term in hand
surgery who most probably derived it
with his experience in the World War 1
12. • He described it as no man's land because of perennial anatomical
characteristics
• This zone has a fibro osseous digital canal where both the tendons interweave
in a complex manner
• Multiple pulleys increase its complexity because minimal swelling of the
epitenon can impair free motion of the tendon
• Any infection, fibrosis, scarring, overcrowding can lead to dense adhesions and
hence compromising the results
13. Pulley system
• Digital flexors synovial sheath is reinforced by a system of fibrous pulleys
• 5 annular pulleys (A)
A1: 8-10 mm over MCPJ
A2: 18-20mm over proximal phalanx
A3: 2-4 mm over PIPJ
A4: 10-12mm over middle phalanx
A5: 2-4 mm over DIPJ
14. • 3 Cruciform pulleys (C)
C1, C2, C3 proximal to A3,A4, A5
Allow shortening of the pulley system in flexion
• A2 and A4 are considered most important.
• Their disruption leads to bowstringing, reduced mechanical
efficiency and decreased flexion
15. • Function of Pulley System
Keep the tendon close to bone , providing an inline pull of tendon along
the axis of digit and toward palm
Keep the tendon adjacent to the joints, maintaining a standard lever
distance from joint center of rotation
• Gaps between annular and cruciform ligaments are necessary to allow
flexion of fingers
16. Biomechanics of Pulley
• If the tendon is allowed to move away from joint center of rotation and force is pulled through
flexor system , lever distance increases, causing increase in work performance at joint leading to
excess flexion at DIP joint
• Degree of flexion is directly proportional to tendon excursion and inversely proportional to
moment arm
17. Tendon Nutrition
Tendon nutrition 2 basic sources:
• Synovial fluid produced within the tenosynovial sheath
• Blood supply provided through longitudinal vessels in the paratenon,
intraosseous vessels at the tendon insertion, and vincular circulation
18. Blood supply
• Suture placement in volar half of tendon
• Intratendinous placement of crisscross sutures
disturb the microcirculation
19. Tendon healing
Tendon healing is believed to occur through the activity of extrinsic and intrinsic
mechanisms
• Extrinsic: Through the activity of peripheral fibroblasts derived from tendon
sheath and seems dominant mechanism contributing to the formation of scar and
adhesions.
• Intrinsic: Through the activity of the fibroblasts derived from the tendon
20. Phases of Tendon healing
• Following laceration, tendon healing proceeds through 3 sequential but
overlapping phases
21. Examination
• General condition of patient
• Possibility of other injuries
• Neurologic examination
Assessment of light touch and static two point
discrimination
• Vascular examination
CRT of volar digital pulp and nail bed is assessed
When necessary, digital Allen test can be done
22. Posture of Hand
• Normal flexion cascade: MCP joints slightly flexed – about 30 degrees in the
index, ranging to 70 degrees in the little
• Assure all fingers point to scaphoid area when flexed at PIPs joint
• When both flexor tendons of a finger are injured, the finger lies in an unnatural
position of hyperextension, especially compared with uninjured finger
• Passive extension of the wrist does not produce the normal flexion of the finger
23. Test for flexor digitorum profundus
Test for flexor digitorum superficialis
Test for flexor pollicis longus
Examination of individual Tendon
24. Objective of Tendon Repair
• Purpose of tendon suture
To approximate the ends of a tendon or to fasten one end of a tendon to
adjoining tendons or to bone
To hold this position during healing
• Pinching and grasping of the uninjured surfaces should be avoided
25. Principles of Tendon Repair
• All flexor tendons should be repaired at whatever level they are transected.
• Whenever possible the repair should be done primarily. When delayed a tendon
graft may be required
• A2 and A4 annular pulley areas of the flexor sheath should be preserved;
otherwise, tendon bowstringing and flexion deformity of the finger can develop
and excursion of the tendon becomes impaired.
• Tendon laceration of 60 % or more is treated as complete transection
26. • Initally dorsal epitenon sutures are placed then core sutures and then finally
volar epitenon sutures
• Diameter of the suture is also directly porportional to the strength of the
repair 3.0 to 4.0 calliber non absorbable suture is recommended
27. • Repair is supplemented with a running circumferential epitenon suture
technique.
Advantages:
smooth repair and improves tendon gliding
minimize the adhesion formation
less gap formation
• Suture technique must withstand gap formation of 3 mm at the repair site during
the initial 3 weeks following repair.
28. Strickland stressed six characteristics of an ideal tendon repair
• Easy placement of sutures in the tendon,
• Secure suture knots,
• Smooth juncture of tendon ends,
• Minimal gapping at the repair site,
• Minimal interference with tendon vascularity, and
• Sufficient strength throughout healing to permit application of early
motion stress to the tendon.
29. Timing of Flexor Tendon Repair
• Primary tendon repair
within the first 12 hours of injury
can be extended to within 24 hours of injury in rare situations
• Delayed primary repair: within 24 hours to approximately 10 days.
• Secondary after 10 to 14 days
• Late secondary repair after 4 weeks
30. Ideal properties of suture material
• Non reactive
• Hold securely when knotted
• Small caliber
• High tensile strength
• Easy to handle
Common material: Ethibond , Nylon, Prolene
31. Suture Configurations
Tendon repair types can be divided into three groups
• Group 1: Simple suture
• Group 2 : Bunnell suture
• Group 3: Pulvertaft technique (fish-mouth weave)
32. An abundance of research has shown that four-strand, six-strand, and
eight-strand core sutures
• Create stronger repairs
• Reduce the possibility of gap formation
• Permit greater active forces applied to the repaired tendons
• Allowing earlier active motion
33. End to End Suture Techniques
Kessler technique
• Modification of the Mason-Allen suture.
• Effective for tendon repair in the fingers and palm
• Knots being left exposed on the tendon surface.
34. Modified Kessler technique
• Modifications:
Only one knot inside the repair site
Knot is left in the cut surface of the tendon
• Easier to use a monofilament suture like a
4.0 Prolene to reapproximate tendon edges
35. Kessler Tajima stitch
• Separate sutures are used so that the tendon ends can be passed within the
flexor sheath using the free end of the suture as traction suture
• Knots are tied within the tendon
• However, use of two knots in the
repaired area could lead to a high risk of
early failure, suture slippage, and tendon torsion
36. Modified Kessler Tajima Suture (Strickland)
• Modified by addition of
Peripheral epitenon suture
Core mattress suture
• Knots are tied within the tendon
• Sutures are locked with each exit from
the tendon.
37. Pulvertaft Technique ( Fish mouth technique)
• Commonly used to suture tendons of unequal size
• Tendon of small diameter can be sutured to one of large diameter
38. End to Side Suture Technique
• Frequently used in tendon transfers when one motor must activate
several tendons
39. Tendon to Bone Attachment
• Frequently requires a pull-out technique (usually distal phalanx)
• For tendon to bone repairs ,core suture techniques used most often are
Kessler
Bunnell crisscross suture
40. Suture Anchor Tendon Attachment
• As effective as pull-out wire or suture but without
the potential complications with fingernail that
can occur with pull-out technique
• Two suture anchors are placed in the distal
phalanx from distal-volar to proximal-dorsal
• To purchase in the thickest portion of the distal
phalanx to provide greatest pullout strength
41. Surgical Incision
• Incisions should not compromise viability of skin flaps
• Should not create contractures or cosmetically unsightly scars
• Less exposure needed distally than proximally
• Zigzag (Brunner),mid radial ,mid ulnar or midline oblique incisions
42. Zone I Repair
• Contains only the FDP tendon
• Direct repair (primary tenorraphy): laceration >1 cm from FDP insertion
• Proximal tendon retrieved by feeding tube and passed underneath A4
pulley
43. Tendon advancement (<1cm stump)
• Pull-out suture
Free ends of sutures are passed
through or around distal phalanx, tied
over dorsum of fingernail with button.
Removed after 6 weeks
• Internal suture methods
Suture anchors or other methods to
affix the tendon directly to the bone
44. When the diagnosis of interruption of FDP tendon is delayed, and the tendon has
retracted into the palm, its vinculum has been disrupted and a decision must be
made regarding repair depending upon types
• Type I: FDP tendon is avulsed from its
insertion and retracts into palm
• Type II: Avulsed from its insertion but
stump remains within digital sheath
• Type III: Avulsed bony fragment attached
to tendon stump, remains within flexor
sheath
45. Modified Leddy and Packer classification for FDP avulsion
Type IV avulsions were first described by Robins and Dobyns in 1974. Inclusion
of type V avulsions in the classification was proposed by Al-Qattan in 2001
46. • Type 1, if it is within 7 to 10 days of the injury, the tendon should be threaded
back into the finger and reattached with a pull-out wire into distal phalanx
• Type 2, despite the passage of a few months, these tendons can be reattached as
well because circulation is thought to be maintained
• Type 3, usually fracture repair with k-wire or miniature screw fixation
• Type 4, fracture fixation followed by advancement of tendon to distal phalanx
• Type 5, FDP tendon with avulsed bone fragment is reduced using a pull-out
technique, and a K-wire is placed for avulsed fragment
47. Zone II Repair
Primary repair at this level frequently fails because of adhesion in the area of
pulley
Technical concerns during the repair procedure includes
• Management of lacerations of FDP and FDS tendons
• Appropriate orientation of the FDP with FDS slips
• Attachment of the FDS slips in the thin flat area
• Management of the flexor sheath, including annular thickening (pulleys)
• Postoperative management,
• Timing and technique for tenolysis
48. • Care should be taken when FDS has been injured in area just proximal to PIP joint
• Care also should be taken to deliver FDP tendon through the split portion of FDS
when profundus tendon has retracted proximally
49. • Method of repair however is controversial but following are the different options
of treatment:
Repair of the FDP tendon only with debridement of the FDS stump
Repair of both tendons or
Repair of FDP with repair of one slip of FDS tendon
Most hand surgeons prefer to repair the FDP and one slip of FDS , reasonably a
good option as the repair of both slips of FDS may produce overcrowding within the
sheath and pulleys and compromise the result
Coats RW, 2nd, Echevarría-Oré JC, Mass DP. Acute flexor tendon repairs in zone II. Hand Clin. 2005;21:173–9
50. • Core suture with two or more strands, locking components, and buried knots is
usually preferred
• A running, circumferential 5-0 or 6-0 suture is used to complete a smooth repair
and to minimize adhesion formation to the sheath
• Tenolysis may be required in an estimated 18% to 25% of patients after flexor
tendon repair
• Three months is considered to be earliest time for flexor tenolysis, assuming no
improvement in motion in previous 1 to 2 months
• Function in the finger can be improved by 50% by tenolysis
52. Zone III Repair
• If both tendons are lacerated, both are repaired, end to end with circumferential
re-enforcing sutures
• Muscles bellies of lumbricals and tendon are frequently affected so additional
incision often needed to expose further area
• If conditions permit, primary repair of sharply severed nerve is crucial
• If wound condition preclude tendon and nerve repair, the ends of the tendon and
nerve are sutured to adjacent fascia to prevent undue retraction
• Lumbricals muscle bellies usually are not sutured, causes lumbrical plus finger
53. Zone IV Repair
• Associated with median ,ulnar nerve or ulnar/radial vessel injury
• If the laceration occurs beneath the transverse carpal ligament, partial or complete release
of the transverse carpal ligament may be required
• Preserve, if possible, a portion of the transverse carpal ligament to avoid bowstringing
postoperatively
• If it cannot be preserved, release it in a Z-lengthening configuration
• Best suture configuration is intratendinous one with a locking core
• Immobilize the wrist in about 45 degrees of flexion, 50 to 60 degrees of flexion at MCP
joints and the IP joints in full extension
54. Zone V Repair
• In zone V, multiple tendons, nerves, and vessels frequently are injured so its important to identify
the tendons accurately
• Properly match the tendon ends by careful attention to their location and level in the wound,
their relation to neighboring structures
• Blood clots within the tenosynovium usually serve as clues in locating severed tendons
• Repairs done in the distal forearm do not absolutely require an intratendinous repair, double
right-angled or mattress suture may be used
• Immobilize the wrist in about 45 degrees of flexion, with fingers in 50 to 60 degrees of flexion at
MCP joints and the IP joints in full extension
56. Immobilization
Indication for immobilization after flexor tendon surgery is usually not required,
exceptions
• Children or adults unable to cooperate with hand therapy
• Unstable bone repair
• Concerns about the effect of tension on microneurovascular repairs.
• Unwilling to adhere to strict early mobilisation protocols
57. Passive Motion Protocol
• Modified Kleinert: Active extension, passive flexion by rubber bands.
• Duran: Controlled Passive Motion Methods
60. Flexor Tendons Reconstruction
When FDS and FDP tendons have been divided in the critical area of the pulleys,
flexor tendon grafting may be indicated if
• Injuries resulting in segmental tendon loss
• Neglected >3 to 6 weeks with tendon degeneration and scar within the tendon
sheath i.e. chronic flexor tendon injuries
• If a gap cannot be closed because of myocontracture
• Large section of tendon has been damaged in Zone II injury
• Delayed presentation of FDP avulsion injuries associated with significant tendon
retraction
61. Requirements before tendon graft
• Skin is pliable;
• Any wounds are well healed
• Tissues through which tendon is expected to glide must be relatively free
of scar
• Edema has subsided
• Joints allow a full passive range of motion
• Sensation in the finger is normal or at least one digital nerve is intact
63. Single-Stage Tendon Graft
• Considered when the flexor tendon sheath is relatively intact and unscarred and
the pulley system is competent
• Attempt can be made to dilate collapsed pulleys with urethral dilators
• However, if unsuccessful, a two-stage procedure with pulley reconstruction
should be undertaken
Age is a strong prognostic factor. Results are best in patients 10 to 30 years old, and
worst results in very young and in patients older than 50 years
Bora FW Jr: Profundus tendon grafting with unimpaired sublimis function in children. Clin Orthop Relat Res
1970;71 (71):118-123.
64. Two-Stage Tendon Graft
Indications
• Crushing injuries associated with fracture or skin damage
• Damaged pulley system
• Excessive scarring of the tendon bed
• Failure of previous operations
• Stiff joints
65. First stage
Excision of tendon and scar from the flexor tendon bed
Preserving or reconstructing the flexor pulley system
• Dacron-impregnated silicone rod is inserted to maintain the tunnel in the area of
excised tendons until passive motion and sensitivity have been restored to the digit
Second stage
• Done when the finger is soft, supple, and well healed with mobile joints
• Earliest time for the second stage is about 8 weeks, but usually 3 months is
required, depending on the patient’s needs and the surgeon’s judgment
• Consists of
Rod removal
Tendon graft insertion
66.
67. Pulley Reconstruction
Goal
• To enable maximum excursion and
strength with controlled gliding of flexor
tendons
• To minimize friction
Two main techniques are
• Weaving the pulley graft through
remaining pulley rim
• Creating a new pulley loop around bone
68. Graft Choices
• Palmaris longus tendon
• Plantaris tendon
• Extensor Digitorum longus to 2nd 3rd or 4th toes
• Flexor digitorum longus to 2nd toe
• Flexor digitorum superficialis
Intrasynovial grafts (FDS and toe flexors) have better
morphologic and functional characteristics than that of
extrasynovial grafts. Extrasynovial grafts have been
thought to lead to more adhesions
Flexor tendon grafting to the hand: An assessment of the intrasynovial
donor tendon. A preliminary single-cohort study. J Hand Surg Am 2000;
25(4):721-730.
69. Complications
Short term:
• Infection
• Injury to neurovascular structures or pulley system
• Abnormal scarring
Long term:
• Adhesion
• Rupture
• Joint contracture
• Triggering
Rarer complications
• Lumbrical plus finger
• Quadrigia effect
70. Adhesions
• Most common complication despite early motion protocols
• Higher risk with Zone II injuries
• Treatment
Physical therapy
Tenolysis when patients progressive gain in digital motion has plateaued,
usually 3- 6 months after repair
Adhesion formation with restriction of tendon excursion and the need for
tenolysis has reported rates from 12% to 47%
LaSalle WB, Strickland JW: An evaluation of the two-stage flexor tendon reconstruction technique. J
Hand Surg Am 1983;8(3):263-267.
71. Tendon rupture
• Noted by the patient as “popping” in the hand
• 7-10 days postop when tensile strength is weakest
• MRI may help in diagnosis
• Treatment
If < 1cm of scar is present, resect the scar and perform primary repair
If > 1cm of scar is present, perform tendon graft
• if the sheath is intact and allows passage of a pediatric urethral catheter or vascular
dilator, perform primary tendon grafting
• if the sheath is collapsed, place Hunter rod and perform staged graft
72. References
• Campbells Operative Orthopaedics,13th edition
• Green Operative Hand Surgery, 7th edition
• Apleys system of Orthopedics,9th edition
• Articles