This document summarizes a presentation on the structure of enamel and its clinical importance. It discusses the mineral content, direction of rods, and other structural features of enamel. It also covers enamel defects like developmental defects, fluorosis, and tetracycline staining. Finally, it discusses clinical implications such as fluoridation, acid etching, enamel microabrasion, and macroabrasion. The document provides an overview of important enamel structures and properties and their relevance to clinical dentistry.
Aging causes irreversible changes to the dental hard tissues over time. The three main tissues - enamel, dentin, and cementum - all undergo changes as part of the aging process. Enamel becomes less permeable and more discolored with age. Dentin develops more dead tracts and sclerotic dentin. Cementum may experience hypercementosis and the formation of cementicles. The alveolar bone also undergoes resorption, decreasing in height and width over time. These morphological and functional changes to the dental tissues are a natural part of the biological aging process.
It is a presentation in detail about the strongest structure of the oral cavity "ENAMEL". It is a simple topic but people find it difficult to learn about it. I hope my presentation is a simple method to learn about it. I would like to thank my professors for assign me this project and i learn't a lot from it and still learning my basics daily.
Impression compounds are thermoplastic materials used for dental impressions. They are composed mainly of rosin, copal resin, carnauba wax, stearic acid, and talc. There are two main types - lower fusing impression compound and higher fusing tray compound. Impression compound is a viscous material that is softened in hot water before making impressions, allowing it to flow and capture detail but also maintain shape. While able to displace soft tissue, its high viscosity limits fine detail capture. Impression compound requires careful heating and cooling to avoid distortion, and constructs must be poured promptly due to its marginal dimensional stability.
The dental pulp contains zones including the odontoblastic zone, cell-free zone, and cell-rich zone. Principal cells include odontoblasts that synthesize dentin, fibroblasts that form the pulp matrix, and immune cells. Blood vessels enter the pulp and branches form capillaries. Nerves form the Raschkow plexus near the odontoblasts. The pulp provides nutrients and sensation to the tooth.
This document discusses the properties and use of zinc phosphate cement. It is the oldest luting cement, classified in two types - fine grained for luting and medium grained for luting and filling. The powder contains zinc oxide, magnesium oxide, and silica. The liquid contains phosphoric acid, water, and aluminum phosphate. When mixed, a reaction forms a zinc aluminophosphate gel. It has high compressive strength but no chemical adhesion and can irritate the pulp. Zinc phosphate cement is used for luting restorations and bases and as a temporary restoration material.
Coronal and radicular pulp
Apical foramen
Accessory canal
Functions of dental pulp
Components of dental pulp
Functions of pulpal extracellular matrix
Organization of cells in the pulp
The principle cells of the pulp
The pathways of collagen synthesis
Matrix and ground substances
Vasculature and lymphatic supply
Innervation of Dentin- pulp complex
Disorders of the dental pulp
Advances in pulp vitality testing
Dental amalgam is an alloy used as a dental restorative material. It consists of mercury combined with other metals like silver, tin, and copper. Amalgam undergoes a setting reaction when mixed with liquid mercury to form a hard material. It is indicated for restoring cavities. While it has advantages like strength and cost-effectiveness, it lacks esthetics and can release low levels of mercury vapor. Modern amalgams have improved properties like reduced creep and shrinkage. Careful manipulation is required to achieve optimal physical properties and reduce risks.
This document provides an overview of cementum, including:
- Its physical characteristics, composition, classification, and formation process (cementogenesis).
- The cells involved in cementum formation and maintenance, including cementoblasts and cementocytes.
- Its locations and junctions with other tissues like enamel and dentin.
- The functions of cementum in anchoring teeth, adaptation, and repair.
- Some developmental anomalies and abnormalities that can affect cementum.
Aging causes irreversible changes to the dental hard tissues over time. The three main tissues - enamel, dentin, and cementum - all undergo changes as part of the aging process. Enamel becomes less permeable and more discolored with age. Dentin develops more dead tracts and sclerotic dentin. Cementum may experience hypercementosis and the formation of cementicles. The alveolar bone also undergoes resorption, decreasing in height and width over time. These morphological and functional changes to the dental tissues are a natural part of the biological aging process.
It is a presentation in detail about the strongest structure of the oral cavity "ENAMEL". It is a simple topic but people find it difficult to learn about it. I hope my presentation is a simple method to learn about it. I would like to thank my professors for assign me this project and i learn't a lot from it and still learning my basics daily.
Impression compounds are thermoplastic materials used for dental impressions. They are composed mainly of rosin, copal resin, carnauba wax, stearic acid, and talc. There are two main types - lower fusing impression compound and higher fusing tray compound. Impression compound is a viscous material that is softened in hot water before making impressions, allowing it to flow and capture detail but also maintain shape. While able to displace soft tissue, its high viscosity limits fine detail capture. Impression compound requires careful heating and cooling to avoid distortion, and constructs must be poured promptly due to its marginal dimensional stability.
The dental pulp contains zones including the odontoblastic zone, cell-free zone, and cell-rich zone. Principal cells include odontoblasts that synthesize dentin, fibroblasts that form the pulp matrix, and immune cells. Blood vessels enter the pulp and branches form capillaries. Nerves form the Raschkow plexus near the odontoblasts. The pulp provides nutrients and sensation to the tooth.
This document discusses the properties and use of zinc phosphate cement. It is the oldest luting cement, classified in two types - fine grained for luting and medium grained for luting and filling. The powder contains zinc oxide, magnesium oxide, and silica. The liquid contains phosphoric acid, water, and aluminum phosphate. When mixed, a reaction forms a zinc aluminophosphate gel. It has high compressive strength but no chemical adhesion and can irritate the pulp. Zinc phosphate cement is used for luting restorations and bases and as a temporary restoration material.
Coronal and radicular pulp
Apical foramen
Accessory canal
Functions of dental pulp
Components of dental pulp
Functions of pulpal extracellular matrix
Organization of cells in the pulp
The principle cells of the pulp
The pathways of collagen synthesis
Matrix and ground substances
Vasculature and lymphatic supply
Innervation of Dentin- pulp complex
Disorders of the dental pulp
Advances in pulp vitality testing
Dental amalgam is an alloy used as a dental restorative material. It consists of mercury combined with other metals like silver, tin, and copper. Amalgam undergoes a setting reaction when mixed with liquid mercury to form a hard material. It is indicated for restoring cavities. While it has advantages like strength and cost-effectiveness, it lacks esthetics and can release low levels of mercury vapor. Modern amalgams have improved properties like reduced creep and shrinkage. Careful manipulation is required to achieve optimal physical properties and reduce risks.
This document provides an overview of cementum, including:
- Its physical characteristics, composition, classification, and formation process (cementogenesis).
- The cells involved in cementum formation and maintenance, including cementoblasts and cementocytes.
- Its locations and junctions with other tissues like enamel and dentin.
- The functions of cementum in anchoring teeth, adaptation, and repair.
- Some developmental anomalies and abnormalities that can affect cementum.
This document discusses the structure and properties of enamel. It begins by defining enamel as the outermost layer of tooth covering made of highly mineralized tissue. The structure of enamel is described including enamel rods, interrod substance, and rod sheaths. Physical properties like hardness, thickness and chemical composition consisting mainly of hydroxyapatite are covered. Incremental growth lines including cross striations, striae of Retzius and neonatal line are also summarized. Hypo-mineralized enamel structures such as enamel spindles, tufts and lamellae are defined. Finally, the surface structure of enamel including outer structureless enamel and perikymata grooves are described.
The document summarizes the development of teeth from the dental lamina. It discusses how the primary epithelial band forms and divides into the dental lamina and vestibular lamina. Tooth buds then develop from the dental lamina, forming the enamel organ, dental papilla, and dental follicle. Teeth progress through developmental stages including the bud stage, cap stage, bell stage, and root formation. The dental lamina gives rise to both primary and permanent teeth before degenerating.
This document provides information on cementum, including its definition, physical characteristics, chemical composition, formation (cementogenesis), classification, functions, anomalies, and clinical considerations. Cementum is the mineralized tissue covering tooth roots. It is softer than dentin and lacks enamel's luster. Cementum formation involves acellular and cellular stages. Cementum attaches the periodontal ligament fibers to the tooth root and allows for tooth repair. Abnormalities include hypercementosis, ankylosis, and cementomas. Cementum is an important part of the periodontium that aids in tooth attachment and repair.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
1. Gypsum materials like dental plaster and stone are commonly used in dentistry to make casts and models due to their low cost, ease of use, and ability to accurately reproduce fine details from impressions.
2. There are 5 types of gypsum materials defined by ISO standards for different applications based on their strength and expansion properties. Type I is rarely used for impressions while Types II-V are used for models, study casts, and fabricating crowns and bridges.
3. Gypsum materials set via a hydration reaction where calcium sulfate hemihydrate reacts with water to form calcium sulfate dihydrate. Factors like water-powder ratio, mixing time, and additives can affect properties like setting
The document discusses the complex process of tooth development from initiation to eruption. It begins with the formation of the primary epithelial bands and dental lamina between 6-7 weeks in utero, which give rise to the tooth buds. The buds progress through stages of proliferation, histodifferentiation, and morphodifferentiation to form the crown and root structures. Hertwig's epithelial root sheath is responsible for root formation and shape before teeth erupt into the oral cavity.
This document discusses denture base materials, specifically acrylic resins. It begins by defining denture base and classifying denture base resins as non-metallic, metallic, temporary or permanent. Ideal requirements of dental resins are listed. Composition and differences between heat cure and self cure acrylic resins are provided. Processing techniques like compression molding and the curing cycle are described. Other resin types like light activated are also mentioned. Common processing errors in acrylic resins like porosity, crazing and warpage are listed.
The document discusses the structure and development of dentin. It describes dentin as the layer beneath enamel that provides shape and structure to teeth. Dentin forms in stages that mirror tooth development from the lamina bud stage through late bell stage. Key features of dentin include dentinal tubules that contain odontoblastic processes and layers like peritubular dentin, intertubular dentin, and predentin near the pulp. Dentin is laid down in primary, secondary, and tertiary forms throughout life.
Amelogenesis is the formation of enamel. During amelogenesis, the ameloblast (enamel-forming cells) undergo various stages i.e the life cycle of ameloblast.
For more content check out my blog: www.rkharitha.wordpress.com "a little about everything dental"
The presentation discusses about tooth enamel in detail including its formation, characteristics, structure and histological features along with its clinical considerations. It is well supported with diagrams for better understanding of the text.
Suggestions and feedback will be well appreciated.
Radiographic Assessment of the Prevalence of Pulp Stones in Malaysians
Kannan et al.
JOE — Volume 41, Number 3, March 2015
Pulp stones are discrete calcified bodies found in the dental pulp.
They have calcium phosphorous ratios similar to dentin and can be seen in healthy, diseased, or even unerupted teeth
Radiographically, pulp stones appear as radiopaque structures in the pulp space that frequently act as an impediment during endodontic treatment
This document provides information on the anatomy of permanent mandibular molars. It describes the identifying features, anatomical aspects, and differences between upper and lower molars for the mandibular first, second, and third molars. Key details include the number and shape of cusps, developmental grooves, roots, and contact areas for each tooth. Differences between upper and lower molars are also summarized such as the number of roots, presence of an oblique ridge, and shape of cusps on the mesial aspect.
A Complete presentation explaining the complete morphology of Maxillary first molar, for the benefit of people like me who tried and failed to find everything in one package
This document discusses tarnish and corrosion of metals used in dentistry. It begins by defining tarnish and corrosion, then discusses the various causes of each in the oral environment. These include the mouth's moisture, temperature fluctuations, acidic foods and liquids. The document classifies corrosion into different types - chemical, electrochemical, galvanic, stress, and crevice corrosion. It also discusses how different metals used in dentistry, like amalgam, stainless steel, titanium and noble metals are affected by corrosion. The document concludes by covering some methods to protect against corrosion, like passivation, electroplating and maintaining oral hygiene.
Introduction
Classification
Composition
Properties Of GIC
Clinical Application Of GIC & GIC In Endodontics
Contraindication Of GIC
Types Of GIC
Recent Advances
Conclusion
References.
Theories of Mineralization
There are three main theories of mineralization:
1) Robinson's phosphate theory which involves alkaline phosphatase increasing local phosphate levels for hydroxyapatite formation.
2) Nucleation theory where nucleating substances like proteoglycans and collagen fibrils initiate crystal formation.
3) Matrix vesicle theory which is most accepted - matrix vesicles accumulate calcium and phosphate ions to form initial mineral complexes within their membranes before releasing crystals into the extracellular matrix.
The document discusses the periodontal ligament (PDL), which is the soft connective tissue that surrounds tooth roots and attaches cementum to alveolar bone. It defines PDL and describes its extent, average width, development from the dental follicle, orientation of collagen fibers, cellular elements including fibroblasts, cementoblasts, osteoblasts, and epithelial rests of Mallassez. The document also covers the biochemical composition and ground substance of PDL, as well as its blood supply, nerve supply, age-related changes, and role in healing after periodontal surgery.
The document discusses the peridontium and its components, which include the gingiva, periodontal ligament, cementum, and alveolar bone. It focuses on cementum, describing it as a hard connective tissue that covers tooth roots and provides attachment for collagen fibers. Cementum begins forming at the cementoenamel junction and continues to the root apex. It contains cementoblasts and cementocytes that aid in its formation and structure. Cementum comes in cellular and acellular varieties and demonstrates incremental lines from its continuous deposition over time.
PHYSICAL PROPERTIES
CHEMICAL PROPERTIES
STRUCTURE OF ENAMEL
DEVELOPMENT OF ENAMEL
EPITHELIAL ENAMEL ORGAN
AMELOGENESIS
LIFE CYCLE OF AMELOBLASTS
AGE CHANGES IN ENAMEL
DEFECTS OF AMELOGENESIS
CLINICAL IMPLICATIONS
Enamel significance in operative dentistry /certified fixed orthodontic cour...Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
Enamel is the hardest substance in the human body. It is composed primarily of hydroxyapatite crystals arranged in rods (prisms) that run from the dentin-enamel junction to the outer enamel surface. The basic structural unit of enamel is the enamel rod, which is hexagonal or oval in cross-section. Between the rods is the interrod enamel. Enamel gains its strength through the interweaving of tightly packed hydroxyapatite crystals in parallel alignment within the rods.
This document discusses the structure and properties of enamel. It begins by defining enamel as the outermost layer of tooth covering made of highly mineralized tissue. The structure of enamel is described including enamel rods, interrod substance, and rod sheaths. Physical properties like hardness, thickness and chemical composition consisting mainly of hydroxyapatite are covered. Incremental growth lines including cross striations, striae of Retzius and neonatal line are also summarized. Hypo-mineralized enamel structures such as enamel spindles, tufts and lamellae are defined. Finally, the surface structure of enamel including outer structureless enamel and perikymata grooves are described.
The document summarizes the development of teeth from the dental lamina. It discusses how the primary epithelial band forms and divides into the dental lamina and vestibular lamina. Tooth buds then develop from the dental lamina, forming the enamel organ, dental papilla, and dental follicle. Teeth progress through developmental stages including the bud stage, cap stage, bell stage, and root formation. The dental lamina gives rise to both primary and permanent teeth before degenerating.
This document provides information on cementum, including its definition, physical characteristics, chemical composition, formation (cementogenesis), classification, functions, anomalies, and clinical considerations. Cementum is the mineralized tissue covering tooth roots. It is softer than dentin and lacks enamel's luster. Cementum formation involves acellular and cellular stages. Cementum attaches the periodontal ligament fibers to the tooth root and allows for tooth repair. Abnormalities include hypercementosis, ankylosis, and cementomas. Cementum is an important part of the periodontium that aids in tooth attachment and repair.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
1. Gypsum materials like dental plaster and stone are commonly used in dentistry to make casts and models due to their low cost, ease of use, and ability to accurately reproduce fine details from impressions.
2. There are 5 types of gypsum materials defined by ISO standards for different applications based on their strength and expansion properties. Type I is rarely used for impressions while Types II-V are used for models, study casts, and fabricating crowns and bridges.
3. Gypsum materials set via a hydration reaction where calcium sulfate hemihydrate reacts with water to form calcium sulfate dihydrate. Factors like water-powder ratio, mixing time, and additives can affect properties like setting
The document discusses the complex process of tooth development from initiation to eruption. It begins with the formation of the primary epithelial bands and dental lamina between 6-7 weeks in utero, which give rise to the tooth buds. The buds progress through stages of proliferation, histodifferentiation, and morphodifferentiation to form the crown and root structures. Hertwig's epithelial root sheath is responsible for root formation and shape before teeth erupt into the oral cavity.
This document discusses denture base materials, specifically acrylic resins. It begins by defining denture base and classifying denture base resins as non-metallic, metallic, temporary or permanent. Ideal requirements of dental resins are listed. Composition and differences between heat cure and self cure acrylic resins are provided. Processing techniques like compression molding and the curing cycle are described. Other resin types like light activated are also mentioned. Common processing errors in acrylic resins like porosity, crazing and warpage are listed.
The document discusses the structure and development of dentin. It describes dentin as the layer beneath enamel that provides shape and structure to teeth. Dentin forms in stages that mirror tooth development from the lamina bud stage through late bell stage. Key features of dentin include dentinal tubules that contain odontoblastic processes and layers like peritubular dentin, intertubular dentin, and predentin near the pulp. Dentin is laid down in primary, secondary, and tertiary forms throughout life.
Amelogenesis is the formation of enamel. During amelogenesis, the ameloblast (enamel-forming cells) undergo various stages i.e the life cycle of ameloblast.
For more content check out my blog: www.rkharitha.wordpress.com "a little about everything dental"
The presentation discusses about tooth enamel in detail including its formation, characteristics, structure and histological features along with its clinical considerations. It is well supported with diagrams for better understanding of the text.
Suggestions and feedback will be well appreciated.
Radiographic Assessment of the Prevalence of Pulp Stones in Malaysians
Kannan et al.
JOE — Volume 41, Number 3, March 2015
Pulp stones are discrete calcified bodies found in the dental pulp.
They have calcium phosphorous ratios similar to dentin and can be seen in healthy, diseased, or even unerupted teeth
Radiographically, pulp stones appear as radiopaque structures in the pulp space that frequently act as an impediment during endodontic treatment
This document provides information on the anatomy of permanent mandibular molars. It describes the identifying features, anatomical aspects, and differences between upper and lower molars for the mandibular first, second, and third molars. Key details include the number and shape of cusps, developmental grooves, roots, and contact areas for each tooth. Differences between upper and lower molars are also summarized such as the number of roots, presence of an oblique ridge, and shape of cusps on the mesial aspect.
A Complete presentation explaining the complete morphology of Maxillary first molar, for the benefit of people like me who tried and failed to find everything in one package
This document discusses tarnish and corrosion of metals used in dentistry. It begins by defining tarnish and corrosion, then discusses the various causes of each in the oral environment. These include the mouth's moisture, temperature fluctuations, acidic foods and liquids. The document classifies corrosion into different types - chemical, electrochemical, galvanic, stress, and crevice corrosion. It also discusses how different metals used in dentistry, like amalgam, stainless steel, titanium and noble metals are affected by corrosion. The document concludes by covering some methods to protect against corrosion, like passivation, electroplating and maintaining oral hygiene.
Introduction
Classification
Composition
Properties Of GIC
Clinical Application Of GIC & GIC In Endodontics
Contraindication Of GIC
Types Of GIC
Recent Advances
Conclusion
References.
Theories of Mineralization
There are three main theories of mineralization:
1) Robinson's phosphate theory which involves alkaline phosphatase increasing local phosphate levels for hydroxyapatite formation.
2) Nucleation theory where nucleating substances like proteoglycans and collagen fibrils initiate crystal formation.
3) Matrix vesicle theory which is most accepted - matrix vesicles accumulate calcium and phosphate ions to form initial mineral complexes within their membranes before releasing crystals into the extracellular matrix.
The document discusses the periodontal ligament (PDL), which is the soft connective tissue that surrounds tooth roots and attaches cementum to alveolar bone. It defines PDL and describes its extent, average width, development from the dental follicle, orientation of collagen fibers, cellular elements including fibroblasts, cementoblasts, osteoblasts, and epithelial rests of Mallassez. The document also covers the biochemical composition and ground substance of PDL, as well as its blood supply, nerve supply, age-related changes, and role in healing after periodontal surgery.
The document discusses the peridontium and its components, which include the gingiva, periodontal ligament, cementum, and alveolar bone. It focuses on cementum, describing it as a hard connective tissue that covers tooth roots and provides attachment for collagen fibers. Cementum begins forming at the cementoenamel junction and continues to the root apex. It contains cementoblasts and cementocytes that aid in its formation and structure. Cementum comes in cellular and acellular varieties and demonstrates incremental lines from its continuous deposition over time.
PHYSICAL PROPERTIES
CHEMICAL PROPERTIES
STRUCTURE OF ENAMEL
DEVELOPMENT OF ENAMEL
EPITHELIAL ENAMEL ORGAN
AMELOGENESIS
LIFE CYCLE OF AMELOBLASTS
AGE CHANGES IN ENAMEL
DEFECTS OF AMELOGENESIS
CLINICAL IMPLICATIONS
Enamel significance in operative dentistry /certified fixed orthodontic cour...Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
Enamel is the hardest substance in the human body. It is composed primarily of hydroxyapatite crystals arranged in rods (prisms) that run from the dentin-enamel junction to the outer enamel surface. The basic structural unit of enamel is the enamel rod, which is hexagonal or oval in cross-section. Between the rods is the interrod enamel. Enamel gains its strength through the interweaving of tightly packed hydroxyapatite crystals in parallel alignment within the rods.
This Slide, gives a Brief introduction to the Anatomy of the tooth specifically the outer shell, the enamel, including the structures, development and abnormalities.
Created by Dr. Mohsen S. Mohamed
For Ozident.com
Enamel is the hardest tissue in the body that covers the tooth crown. It is acellular and highly mineralized, composed mostly of inorganic calcium phosphate in the form of hydroxyapatite crystals. Enamel has a density that decreases from the surface to the dentin junction. It contains enamel rods that weave a wavy path through the enamel and are surrounded by interrod cementing substance. The microscopic structure of enamel includes rods, rod sheaths, and interrod material that give it hardness and strength.
This document provides information about a seminar on enamel, dentin, and pulp presented by Dr. Ashish Kalhan. It discusses the key structures and properties of enamel, dentin, and pulp.
Enamel is the outermost covering of the tooth. It is the hardest tissue in the body and provides protection. It is made up of enamel rods arranged in a prism-like pattern. Dentin lies underneath the enamel and makes up the bulk of the tooth. It contains dentinal tubules that house odontoblast processes. The innermost living tissue is the pulp, which contains blood vessels, nerves, and odontoblasts.
The document discusses the physical and chemical properties of enamel
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
The infratemporal fossa is a complex irregular space deep to the mandible containing many neurovascular structures. It communicates superiorly with the middle cranial fossa and orbits. The fossa contains the lateral and medial pterygoid muscles, nerves like the mandibular nerve, vessels like the maxillary artery, and the otic ganglion. Due to its complex anatomy, tumors here present surgical challenges and infections can spread widely. Care is needed during surgery due to the vascular pterygoid plexus and proximity to critical structures.
Dentin forms in primary, secondary, and tertiary layers. Secondary dentin develops after root formation is complete and deposits more slowly with fewer, wavier tubules. Tertiary dentin forms in reaction to stimuli and has fewer, irregular tubules. Dentin is innervated by nerves that pass through tubules. Clinically, dentin permeability and adhesion of materials depends on its surface and tubule patency. Endodontic treatment success decreases with age as secondary and tertiary dentin decrease the pulp chamber and canal. Dentin tubule occlusion can reduce sensitivity, and mitochondrial DNA within tubules aids forensic identification.
La pulpa dental puede experimentar diferentes estados inflamatorios en respuesta a estímulos irritantes. Estos van desde una pulpa vital asintomática hasta una pulpa necrótica. Existen varias clasificaciones de la patología pulpar que incluyen etapas como pulpitis reversible e irreversible, siendo esta última sintomática o asintomática. No hay una correlación perfecta entre los hallazgos clínicos y histopatológicos, por lo que se requieren varias pruebas para diagnosticar correctamente el estado de la pulpa.
Dental caries is caused by an interaction between fermentable carbohydrates, bacteria in dental plaque, and susceptible tooth surfaces over time. Streptococcus mutans and Lactobacillus species in plaque produce acid as they metabolize carbohydrates, lowering the pH and demineralizing enamel. Factors like restoration of teeth, diet, saliva, age, and fluoride exposure influence caries risk. Caries was historically attributed to worms or humors but is now understood as a chemicoparasitic process initiated by acid from plaque bacteria.
This document provides information on dental caries (tooth decay). It defines dental caries as a multifactorial disease characterized by demineralization of tooth structure. Two bacteria, Streptococcus mutans and Lactobacillus, are responsible for initiating caries. Untreated caries can lead to pain, tooth loss, infection, and in severe cases death. The document discusses risk factors for caries like diet, fluoride exposure, socioeconomic status and behaviors. It also covers classifications of caries, epidemiology, pathogenesis, and treatments.
Enamel is the hardest tissue in the human body that covers the anatomical crown of a tooth. It is made up of hydroxyapatite crystals arranged in enamel rods or prisms. Enamel provides protection to the underlying dentin and allows for chewing and grinding of food. It is formed by ameloblasts, which deposit an organic matrix that mineralizes into enamel. Enamel can demineralize from acid produced by bacteria, leading to dental caries if left untreated.
At the end of this lecture, the student should be able to:
Develop understanding of the classification
Describe how a cyst develops.
Describe the origin and identifying characteristics of the radicular cyst.
Describe the origin and identifying characteristics of the Dentigerous cyst.
Describe the origin and identifying characteristics of the Odontogenic Keratocyst cyst.
Describe the radiographic characteristics of the dentigerous cyst and the odontogenic keratocyst.
Discuss the radiographic appearance of the lateral periodontal cyst. 8. List the factors involved in the nevoid basal cell carcinoma syndrome.
State the histologic finding that is a key diagnostic feature of Radicular, Dentigerous & Keratocysts.
Describe the origin and identifying characteristics of non odontogenic cysts.
Describe different treatment options available, and their clinical importance.
Difference between primary and permanent teethprincesoni3954
The presentation features the basic difference between primary and permanent dentition. The differences are tabulated under the headings of crown, roor and pulp.
Histopathology & microbiology of dental cariesAshish Karode
The document summarizes the histopathology of dental caries. It describes how dental caries is a microbial disease that causes demineralization of tooth enamel and dentin. It discusses the role of bacteria like Streptococcus mutans in producing acid that dissolves tooth structure. The summary describes the microscopic appearance of carious lesions in enamel and dentin, including the formation of zones of demineralization and bacterial invasion of dentin tubules over time. Advanced caries can ultimately lead to tooth cavitation and pulp involvement if left untreated.
Este documento habla sobre la pulpa dentaria. Explica que la pulpa dentaria es el tejido blando dentro del diente que se origina embrionariamente en la papila dental. La pulpa puede inflamarse (pulpitis) debido a infecciones bacterianas, traumas o estímulos químicos. Existen dos tipos principales de pulpitis: reversible e irreversible. La pulpitis reversible causa dolor leve que desaparece luego de remover el estímulo, mientras que la pulpitis irreversible puede evolucionar a abscesos o infecciones más
Dental caries is a common chronic dental disease caused by demineralization of tooth structure by acid produced by bacteria in dental plaque. Key factors for dental caries are the host, cariogenic bacteria such as Streptococcus mutans, and fermentable carbohydrates in the diet. Prevention strategies aim to reduce the cariogenic potential of the oral environment through measures like fluoride use, dietary modification, and plaque control. Treatment depends on the severity and activity of the carious lesion.
Clinical features and histopathology of dental cariesSAGAR HIWALE
This document provides an overview of the classification of dental caries based on various factors such as anatomical site, progression, extent of involvement, number of tooth surfaces affected, chronology, and whether caries was fully removed during treatment. It discusses 12 different classification systems for dental caries and provides details on types of caries such as pit and fissure, smooth surface, root surface, incipient, occult, and others based on these classification criteria. The document also covers the histopathology of caries in enamel and dentin.
This document provides an overview of mandibular growth and development from prenatal to postnatal periods. It discusses the anatomy of the mandible and theories around its evolution. The prenatal growth section describes the key stages from fertilization to embryo and fetal development. Mandibular growth mechanisms are explored through a brief history of theories proposed by researchers from the 18th century onwards. Growth progression, sites, and age-related changes in the mandible are examined.
The wasting diseases of teeth, namely attrition, abrasion and dental erosion have taken their toll in the population around the world due to the changing lifestyles, increase in the stress levels and many others factors that were persistent earlier but have suddenly increased drastically. This presentation brings to light the new factors that have attributed to this condition as well as discusses the previous ones.
This document discusses various causes of non-carious tooth destruction and disfigurement including attrition, abrasion, abfraction, erosion, enamel hypoplasia, hypocalcification, dentin hypoplasia, hypocalcification, discoloration, malformation, amelogenesis imperfecta, dentinogenesis imperfecta, and trauma. For each cause, signs and symptoms and treatment modalities are described. Endodontic therapy and restorative treatments like composite resin, glass ionomer cement, and metallic restorations are commonly used to address the defects and restore form and function.
Dental caries is caused by bacteria in the mouth that metabolize carbohydrates, producing acids that demineralize tooth enamel and dentin. It progresses through stages from early subsurface lesions to cavity formation and bacterial invasion. Risk factors include diet, oral hygiene, tooth composition, and saliva. Treatment involves preventing demineralization through fluoride, controlling plaque and bacteria, and restoring teeth through fillings or other methods.
Dental caries is caused by demineralization of tooth structure due to acid produced by oral bacteria. It is characterized by loss of both inorganic and organic components of the tooth. Dental caries has been defined and classified in various ways based on factors such as the anatomical site, severity, tissue involvement, number of surfaces affected, and chronology. The key etiological factors include the presence of cariogenic bacteria in dental plaque, a susceptible tooth substrate, and a cariogenic diet. Secondary factors like time, the dynamic process of demineralization and remineralization, and saliva also influence the development of dental caries.
The document summarizes traumatic dental injuries and their management. It discusses the classification, clinical features, treatment, and stabilization periods for various types of dentoalveolar injuries including enamel fractures, crown fractures, root fractures, luxations, and avulsions. Splinting is described as the best method for immobilizing mobile teeth or displaced teeth, with different splinting techniques and materials discussed. Prompt treatment of dental trauma is emphasized to save injured teeth.
Dental caries is caused by bacteria in the mouth that feed on sugars and produce acids. The acids demineralize the enamel and dentin of the teeth. Two main bacteria, Streptococcus mutans and Lactobacillus, are responsible for initiating caries. If left untreated, dental caries can lead to pain, tooth loss, and infection. Factors that influence the development of caries include diet, microorganisms, host factors, genetics, and immunology. Clinical and radiographic exams are used to detect caries. Treatment involves removing decay and restoring teeth. Preventive methods focus on nutrition, oral hygiene, fluoride, dental sealants, and altering bacterial growth.
This document provides an overview of enamel hypoplasia, including its definition, classification, etiology, clinical features, radiographic features, and management. Enamel hypoplasia is defined as an incomplete or defective formation of the enamel matrix of teeth. It can be hereditary or environmental in origin. Common causes include nutritional deficiencies, infections like syphilis, and dental fluorosis from excess fluoride intake. Clinical features range from mild pitting to severe absence of enamel. Treatment depends on severity and location, and may include desensitizing agents, composite restoration, crowns, or extractions for severely malformed teeth.
True generalized microdontia involves all teeth being smaller than normal and is seen in cases of pituitary dwarfism. Macrodontia refers to teeth being larger than normal. Geminated teeth arise from an attempt at division of a single tooth germ. Taurodontism is the enlargement of the tooth body and pulp chamber with displacement of the pulpal floor. Amelogenesis imperfecta represents hereditary defects of enamel formation. Dentinogenesis imperfecta affects dentin formation resulting in teeth that are gray to yellowish-brown.
Class on regresive altrations of teeth (RAOT)DrRam Thiramdas
The document discusses various regressive alterations of teeth (RAOT) that result from wear and tear over time rather than developmental abnormalities or infection. It describes several types of RAOT including attrition from tooth contact during chewing, abrasion from external abrasives, erosion from acid exposure, and abfraction from biomechanical forces. It provides details on the etiology, clinical features, and appearance of each type of RAOT. Treatment involves identifying and addressing the underlying causes as well as restorative measures to protect the tooth structure.
Interceptive orthodontics refers to early orthodontic treatment during mixed dentition to guide proper development of the dentition. Common procedures include serial extraction to relieve crowding, correction of developing crossbites, and control of abnormal habits like thumb sucking. Early intervention is beneficial as it is simpler and more stable than later treatment, prevents worsening of issues, and offers psychological benefits to children. The optimal age for screening and interceptive treatment is around 9-11 years old.
Tooth resorption is the progressive loss of dentine and cementum by the action of osteoclasts. This is a physiological process in the exfoliation of the primary dentition, caused by osteoclast differentiation due to pressure exerted by the erupting permanent tooth
This document provides an overview of enamel, including its physical and chemical properties, structure, development, and clinical aspects. Some key points:
1. Enamel is the hardest tissue in the body and covers the anatomical crown of teeth. It is composed primarily of hydroxyapatite crystals arranged in rods or prisms.
2. Enamel develops through a process called amelogenesis, where enamel matrix proteins are secreted by specialized cells called ameloblasts. The matrix then undergoes mineralization.
3. Enamel has a complex structure including rods, perikymata, and other features that contribute to its hardness and protection of the tooth. Its structure and composition can be altered by
Restoration of endodontically treated teethIAU Dent
This document summarizes the effects of endodontic treatment on teeth and considerations for restoring endodontically treated teeth. Key points include:
- Endodontic treatment can result in loss of tooth structure, altered physical properties making teeth more brittle, and discoloration.
- Remaining tooth structure, function, and aesthetics must be evaluated to determine the appropriate restoration. Teeth with minimal structure may be restored with composites while those with heavier function typically need crowns.
- Temporary cements must be completely removed before bonding permanent restorations to avoid inhibiting the bond. Teeth exposed to sodium hypochlorite also require treatment to reverse its oxidizing effects.
- Common restorative
Non-carious cervical lesions are caused by erosion from dietary or gastric acids, abrasion from toothbrushing or other habits, and abfraction from biomechanical forces. They present as broad shallow lesions on the facial or lingual surfaces for erosion, notched lesions on the facial surface for abrasion, and wedge-shaped lesions often subgingivally for abfraction. Treatment involves dentin desensitization, restorations with composites or glass ionomers, endodontics if pulpal involvement, periodontal therapy for gingival recession, and prevention through dietary counseling, fluoride application, and correcting habits.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
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Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
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Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
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3. MINERAL CONTENT
Enamel is the hardest tissue in the human
body. Its mineral portion is approximately 96%
of its weight,the rest is organic components
and water.
The mineral elements include hydroxyapatite
crystals, approximately 0.03μm to 0.2 μm,
surrounded by a thin film of firmly bound
water.
CLINICAL SIGNIFICANCE:-
Poorly mineralized enamel –more white
More mineralized –more translucent.
4. DIRECTION OF RODS
• The rods are oriented at right angles to the dentin
surface.
• In the cervical & central parts of the crown of a
permanent teeth, they are approximately horizontal.
• Near the incisal edge or tip of cusps they change
gradually to an increasingly oblique direction until they
are almost vertical in the region of the edge or tip of the
cusps.
• CLINICAL SIGNIFICANCE:-
• Follow the direction of enamel RODS during cavity
preparation so that enamel margins are supported.
4
6. • If the discs are cut in an oblique plane, the bundles of
rods seem to interwine more irregularly.
• Its optical appearance of enamel is called gnarled
enamel.
• CLINICAL SIGNIFICANCE:-
• This enamel is not subject to cleavage as regular
enamel.
• This enamel does not yield readily to pressure of hand
cutting instruments.
9. ENAMEL TUFTS
These projections arise in Dentine and extend into
enamel in the direction of long axis of crown,
hence may play a role in spread of caries.
10. ENAMEL LAMELLAE
Contains mostly organic material which is WEAK
AREA, therefore predisposes tooth to entry of
bacteria ,hence dental caries..
11. Perikymata :-
Transverse wave like grooves appear to be theTransverse wave like grooves appear to be the
external manifestations ofexternal manifestations of striae of retziusstriae of retzius..
Continuous around the tooth and parallel to eachContinuous around the tooth and parallel to each
other and to the CEJ.other and to the CEJ.
Seen in freshly erupted teeth or in tooth which isSeen in freshly erupted teeth or in tooth which is
not subjected to abrasive forces.not subjected to abrasive forces.
Average ofAverage of 30 perikymata/mm30 perikymata/mm in cervical regionin cervical region
andand 10/mm10/mm in occlusal region.in occlusal region.
These may contribute to adherance of
plaque material which results in caries.
13. NASMYTH’S MEMBRANE
Covers newly erupted tooth.
Membrane replaced by pellicle.
Microbes invade pellicle to form
plaque.
ENAMEL PEARLS
Occasionally found on root surface
towards cervical margin.
Importance: Predisposed to plaque
accumulation following gingival
recession.
14. They – act as
bacterial/ food traps
thickness of enamel
predispose tooth to
caries.
Fissure
16. Dental caries
Definition:
dental caries is defined as a multifactorial ,
transmissible ,infectious oral disease caused
primarily by the complex interaction of cariogenic
oral flora with fermentable dietary carbohydrates
on the tooth surface over time.
Sturdevant 6th
edition
18. Definition
White opaque chalky spots observed when the
tooth surface is desiccated are termed as
incipient caries Sturdevant 4th
edition
Radiographically seen as faint radiolucency
Chalky white spot
19. Definition:
Caries which becomes static or stationary and doesn't
show any tendency for further progression
Clinically intact ,discolored ( black or brown spots )
ARRESTED CARIES
21. For an ideal enamel wall , following are
the Noy’s structural requirements-
1) The enamel wall must rest on sound dentine
and all carious dentine must be removed
22. 2)Enamel which forms cavosurface angle must have
their inner ends resting on sound dentin
23. 3) The rods which form cavosurface angle must be
supported on sound dentine and their outer ends
must be covered by restorative material (possibly by
giving a bevel)
24. 4) Cavosurface angle must be beveled so that the
margins will not be exposed to injury in condensing
restorative material against it.
26. o Defective matrix formation.
o Enamel has not formed to full normal thickness
Hypoplastic type
Hypocalcified type
o Enamel is so soft that it can be removed by a
prophylaxis instrument.
o Defective mineralization of formed matrix
Hypomaturation type
o Immature Enamel crystals
o Defective enamel can be pierced by an explorer
point under firm pressure
27. 1) Small teeth with short root
2) Open contact
General features of Amelogenesis
Imperfecta
28. 3) Discoloration ranging from
yellow to dark brown.
4)Thin enamel
5)Enamel could look wrinkled
6)Delay in eruption
7)Occlusal surfaces and incisal edges severely
abraded
8)Sensitivity
29. 1. Enamel may be totally absent
2. Appear as thin layer, chiefly over the tips of the
cusps and the interproximal surfaces.
3. Same radiodensity as dentin , it become
difficult to differentiate between two
Radiographic features
31. II)Enamel
Hypoplasia
Incomplete or defective formation of the organic
matrix
Causes:
1.Nutritional defect
2.Exanthametous diseases
3.Congenital syphilis
4.Ingestion of fluoride
32. 1) Hutchinsons incisors
(screw driver shaped central incisors)
2) Mulberry molars
(small globular masses of enamel on occlusal surface)
Hypoplasia due to syphilis
34. Tetracycline
Generalized type of intrinsic stain
When the tetracycline is administered during
the time of enamel formation it forms a
complex chelating compound with the organic
and inorganic components of the enamel. The
created compound is very stable.
Discoloration depends upon:
Dosage
Length of time over which administration occurred
Form of tetracycline
35. According to Moffitt:
Critical period for tetracycline induced
discoloration in deciduous dentition
• 4 months in utero to 3 months postpartum
(maxillary and mandibular incisors)
• 5 months in utero to 9 months postpartum
(maxillary and mandibular canines)
In permanent dentition
• 3-5 months postpartum to 7 yrs of age
36. Discoloration varies from yellow –orange to dark blue
Drugs:
Chlortetracycline –grayish stains
Minocycline –grayish discoloration
Oxytetracycline –yellow stains
38. Fluorosis
Generalized intrinsic stain
Chronic ingestion of flouride ions interfers with
ameloblast function during formative stage of
tooth development and disturb their activity
39. Clinical features
1) Mild changes
• White flecking or spotting of
enamel
2)Moderate to severe changes
•Brown staining of surface
•Pitting
•Tendency of enamel to fracture
43. EXTRINSIC DISCOLORATIONS
Avoidance of the foods and beverages that cause stains
Using proper tooth brushing and flossing techniques
Professional tooth cleaning: Some extrinsic stains may
be removed with ultrasonic cleaning , enamel
microabrasion, enamel macroabrasion
INTRINSIC DISCOLORATIONS
Bleaching
Enamel microabrasion
Enamel macroabrasion
Veneering
44. Definition:
Surface tooth structure loss resulting from
direct frictional forces between contacting teeth
. (Marzouk 1st
edi)
Types of Attrition
1.Occluding surface attrition
2.Proximal surface attrition
Causes
1. Tooth to tooth contact
2.Parafunctional mandibular movements
45. Clinical features
1. Sensitivity
2. Flattening of incisal and occlusal surface
3. Flattening of inclined planes
4. Flattening of proximal contact areas
5. Facet formation
6. Reverse cusp
7. Loss of vertical dimension of teeth
8. Decay at occluding areas
9. Angular chelitis
10.Cheek bite
11.Temporo mandibular problems
Flattening of incisal
46. Treatment
1. Para functional activities should be controlled
with protecting occlusal splints.
2. Endodontic therapy for pulpally involved teeth
3. Occlusal equilibration, by selective grinding of
tooth surfaces
4. Restorative modalities(only metallic restoration)
47.
48. Abrasion
Definition:
Surface loss of tooth structure resulting from
direct friction forces between teeth and
external objects, or from frictional forces
between contacting teeth components in the
presence of an abrasive medium.
Causes
1. Improper use of tooth brush
2. Improper use of tooth pick and dental floss
3. Habitual opening of bobby pins with teeth.
4. Use of abrasive dentifrices
Marzouk 1st
edition
49. Clinical features
1. Linear in outline(following path of brush bristles)
2. Angular peripheries
3. Notching of central incisors
4. Wedge shaped ditch on proximal
exposed root surface
50. Treatment
1. Diagnosing the cause
2. Removing the causative factor(habits)
3. Desensitizing exposed dentin(if tooth is
sensitive)
4. Restorative treatment
51.
52. Definition
Loss of tooth structure resulting from chemico-
mechanical acts in the absence of specific
microorganisms Marzouk 1st
edition
Causes
1. Ingested acid(lemon and citrus fruits)
2. Chronic vomiting
3. Frequent regurgitation
Rate of erosion is 1micron per day
Erosion
53. Clinical features
1. Shallow, broad, smooth ,highly polished,
scooped out depression on the enamel surface
adjacent to cementoenameljunction
2. Confined to gingival third of labial surface
54. Treatment
1. Complete analysis of diet, chronic vomiting,
environmental factors should be performed
2. Restorative treatment
(tooth colored material can be used with
minimal or no tooth preparation)
55.
56. Abfraction
Definition:
Strong eccentric occlusal force resulting in
microfractures at the cervical area of tooth causing
wedge shaped defects
Sturdevant 6th
edition
Causes
Heavy force in eccentric occlusion
Clinical feature
Wedge shape defect
Defect has smooth surface
Treatment
Restoration
57.
58. Age changes & Clinical considerations
•Attrition is seen in aged people.
•Wear facets are common.
•Decrease in vertical dimension and flattening of
proximal contours.
•Color changes with age.
•Permeability decreases.
•Caries incidence is less in aged people.
•Surface composition: more amount of fluoride and
localized increase in nitrogen.
59.
60. Fluoridation
It decreases the solubility of enamel
It acts in the following way:
I. Forms fluoroapatite which is less soluble than
hydroxyapatite
II. Inhibits demineralization
III.Enhances remineralization
IV.Inhibits bacterial metabolism
61. Acid etching
ACID ETCHING TECHNIQUE- Buonocore in 1955
Micromechanical bonding b/w enamel and resin
based restorative material.
Mode of action-
Increases the porosity of exposed surfaces by
dissolution of crystals - creates a micro porous
layer from 5 to 50 µm deep
62. Three etching patterns predominate:-
(Preferential removal of rods)
TYPE II
TYPE III
(Junction b/w type 1 n type 2)
(Preferential dissolution of prism
core)
TYPE I
63. Enamel etching transforms the smooth enamel
surface into an irregular surface
Etched enamel has high surface energy
(72 dynescm) allow resin to wet the tooth surface
better when resin penetrates into micro porosities
and polymerized to forms resin tags
64. Resin tags interlocked with
the surface irregularities
created by etching which
form mechanical bond to
enamel.
Bond strength:16-20Mpa
65. Originally recommended 60 secs using 37% phosphoric
acid.
Currently,etching time for most etching gel is 15 sec
Aprismatic enamel requires double the etching time
required by prismatic
Etching time
66. Involves the surface dissolution of enamel by acid
along with the abrasiveness of the pumice to remove
superficial stains or defects
Commercially developed system for enamel
microabrasion.
[PREMA (Premier enamel micro abrasion)
Enamel microabrasion
In 1984 Mc Closkey reported this technique
In1986 Croll and cavanaugh modified this technique
67. PREMA contains a reduced concentration of
hydrochloric acid (approx 11%)+ silicon carbide
particles in a water soluble gel paste.
Mode of action
1. Physical removal of stained outer enamel layer by
stripping action of acid and abrasive action of
pumice
2. The etching action removes interprismatic
substance and changes light refraction
characteristics
3. There is oxidation of some pigments
69. Removal of localized superficial white spots and other
surface stains or defects is called macroabrasion
Sturdevant 6th
edition
12 fluted composite finishing bur or fine grit finishing
diamond in a high speed handpiece is used
Macro abrasion
71. CONCLUSION
Enamel is an important structural entity of the tooth
hence its protection is utmost important.
Its function is to form a resistant covering of the
teeth, rendering them suitable for mastication.
72. Marzouk : Operative Dentistry, First Edition
Orban :Oral Histology and Embryology,Tenth
Edition
Oral pathology SHAFER’S
Sturdevant :Art and Science of Operative
Dentistry, Fifth and sixth Edition
Ten Cates: Oral Histology , Seventh Edition
Enamel microabrasion,theodore p croll