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Cardiovascular system
History & Examination
Cardinal symptoms
 Chest pain
 DOE, PND, orthopnea
 Palpitation, syncope
 Edema or weight gain
 Claudication
 Cyanosis
Relevant other examination
 Vitals- Temperature, PR, BP, RR
 Pallor/cyanosis
 Clubbing
 Edema
 Hepatomegaly and/or splenomegaly
 Crepitations at lung base
Cardiac examination
 Pulse
 Blood pressure
 Jugular venous pulse (JVP)
 Inspection
 Palpation
 Auscultation
Pulse
Palpable- temporal, CAROTID,
brachial, RADIAL, femoral, popliteal,
posterior tibial, dorsalis pedis
Pulse
 Rate- normal 60-100 per minute
 Rhythm- regular or
irregular (atrial
fibrillation, ectopics)
 Volume- rough guide of pulse pressure
 Character
 Radio-femoral delay
R & R- radial, Vol. & Charac.- carotid
Character of pulse
 Slow rising- AS
 Collapsing- AR
 Bisferiens- AR & AS
 Pulsus paradoxus- heart sounds heard,
but no radial pulse- seen with
cardiac tamponade or severe asthma
 Pulsus alternans- alternate strong &
weak beats- seen in severe LVF
Blood pressure
 Measured by sphygmomanometer
 Patient comfortable
 Manometer at the level of arm
 Cuff length- >40% arm circumference
 Proper cuff width- 2/3rd
of arm- 12.5 cm.
 Korotkoff sounds heard by stethoscope
 Appearance- systolic BP
 Disappearance- diastolic BP
 Pulse pressure- systolic - diastolic
Classification of BP
 Normal- 90-119/60-79
 Pre-HT- 120-139/80-89
 Stage 1- 140-159/90-99
 Stage 2- >160/>100
 Systolic- >140/<90
AHA,2003
Jugular venous pulse
Internal jugular vein
At an angle of 45 degree
Look tangentially
JVP vs. Carotid pulse
 Better seen
 2 upstrokes/beat
 Upper level
 Less forceful
 Easily obliterated
 Changes with
posture/respiration
 HJ reflux +ve
 Better felt
 1 upstroke/beat
 No upper level
 More forceful
 Not obliterated
 No change with
posture/respiration
 No change
Jugular venous pulse
 a wave- atrial contraction with TV open,
precedes S1
 c wave- bulging of closed TV in atrium
 x descent- atrial relaxation with TV closed,
precedes S2
 v wave- atrial filling with TV closed
 y descent- atrial emptying with TV open
x, v- systolic; y, a- diastolic
Abnormal JVP
 a absent- A-fib.
 a large- TS, PS, PHT
 a cannon- arrythmia- CHB, VT, ectopics
 y & x prominent- constric. pericarditis
 y prominent, x absent- TR
 y slow- TS
Inspection
 Proper exposure
 Deformity
 Apex beat
 Scars
 Respiration
Palpation- apex beat
 Lowest & outermost point of definite cardiac
pulsation
 Lies just medial to MCL in 5th
ICS
 Size- 1-2.5 cm.
 Caused by left ventricle
 Normally <50% of systole
 Hyperkinetic- <50% systole, forceful, dilated LV,
regurgitant lesion (MR/AR), ±S3
 Sustained- >50% systole, forceful, hypertrophic LV,
stenotic lesion (AS/HOCM) or HT, ±S4
Palpation- other
 Parasternal impulse- enlarged LA/RV
 Thrill- palpable murmur (>grade 3)
 Other palpable sounds
 S1- apex, MS
 A2- R 2nd
ICS, systemic HT
 P2- L 2nd
ICS, pulmonary HT
 S3/S4- apex/L parasternal,
dilatation/hypertrophy of ventricle
 Aortic pulsation- chest/epigastrium,
consider aneurysm
Auscultation- normal
 Palpate carotid simultaneously
 Areas for auscultation
 Mitral- apex
 Tricuspid- L parasternal
 Aortic- R 2nd
ICS
 Pulmonary- L 2nd
ICS
 Only S1 & S2 are heard
 S1- closure of MV/TV, single, systole begins
 S2- closure of AV/PV, split (normally A2 before P2,
best heard in pulmonary area during inspiration),
systole ends
Auscultation areas
Auscultation- abnormal
 Altered intensity (soft/loud)
 All soft- emphysema, pericardial effusion
 Soft S1/A2/P2- calcific valve
 Loud S1- non-calcific MS, tachycardia, ASD/Ebstein’s, high cardiac
output states like anemia, exercise, hyperthyroidism
 Loud A2/P2- systemic/pulmonary HT
 Splitting of S2
 Single- AS/PS
 Wide- PS, RBBB
 Fixed- ASD
 Reverse- LBBB (appears on expiration, disappears on inspiration)
 Syst./pulm. HT- wide, narrow or reverse split
Auscultation- other sounds
 Systolic-
 Early ejection click- AV/PV
 Mid/late ejection click- MVP
 Diastolic-
 Opening snap- MS
 S3- poor LV compliance- CHF, DCMP
 S4- increased LV resistance- HT, AS, HOCM
Auscultation- murmurs
 Due to turbulent blood flow
 Characteristics-
 Timing- systolic/diastolic, early/mid/late/pan
 Location- apex, LLSB, aortic/pulmonary area
 Radiation- AS to carotids, MR to axilla
 Duration/shape
 Grade- 1-6, grade 4 causes palpable thrill
 Pitch- high-regurgitant/low-stenotic
 Quality- blowing high-pitched- regurgitant,
harsh rough rumbling low-pitched-
stenotic
 Variation- with respiration, position, valsalva etc.
Auscultation- murmurs
 Systolic-
 Ejection- AS/PS, flow murmurs (non pathological)
 Pan- MR/TR, VSD
 Late- MVP, HOCM
 Diastolic-
 Early- AR/PR
 Mid- MS/TS
 Continuous- PDA
Supported by
ECG, CxR, ECHO

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Cardiovascular system exam

  • 2. Cardinal symptoms  Chest pain  DOE, PND, orthopnea  Palpitation, syncope  Edema or weight gain  Claudication  Cyanosis
  • 3. Relevant other examination  Vitals- Temperature, PR, BP, RR  Pallor/cyanosis  Clubbing  Edema  Hepatomegaly and/or splenomegaly  Crepitations at lung base
  • 4. Cardiac examination  Pulse  Blood pressure  Jugular venous pulse (JVP)  Inspection  Palpation  Auscultation
  • 5. Pulse Palpable- temporal, CAROTID, brachial, RADIAL, femoral, popliteal, posterior tibial, dorsalis pedis
  • 6. Pulse  Rate- normal 60-100 per minute  Rhythm- regular or irregular (atrial fibrillation, ectopics)  Volume- rough guide of pulse pressure  Character  Radio-femoral delay R & R- radial, Vol. & Charac.- carotid
  • 7. Character of pulse  Slow rising- AS  Collapsing- AR  Bisferiens- AR & AS  Pulsus paradoxus- heart sounds heard, but no radial pulse- seen with cardiac tamponade or severe asthma  Pulsus alternans- alternate strong & weak beats- seen in severe LVF
  • 8. Blood pressure  Measured by sphygmomanometer  Patient comfortable  Manometer at the level of arm  Cuff length- >40% arm circumference  Proper cuff width- 2/3rd of arm- 12.5 cm.  Korotkoff sounds heard by stethoscope  Appearance- systolic BP  Disappearance- diastolic BP  Pulse pressure- systolic - diastolic
  • 9. Classification of BP  Normal- 90-119/60-79  Pre-HT- 120-139/80-89  Stage 1- 140-159/90-99  Stage 2- >160/>100  Systolic- >140/<90 AHA,2003
  • 10. Jugular venous pulse Internal jugular vein At an angle of 45 degree Look tangentially
  • 11. JVP vs. Carotid pulse  Better seen  2 upstrokes/beat  Upper level  Less forceful  Easily obliterated  Changes with posture/respiration  HJ reflux +ve  Better felt  1 upstroke/beat  No upper level  More forceful  Not obliterated  No change with posture/respiration  No change
  • 12. Jugular venous pulse  a wave- atrial contraction with TV open, precedes S1  c wave- bulging of closed TV in atrium  x descent- atrial relaxation with TV closed, precedes S2  v wave- atrial filling with TV closed  y descent- atrial emptying with TV open x, v- systolic; y, a- diastolic
  • 13. Abnormal JVP  a absent- A-fib.  a large- TS, PS, PHT  a cannon- arrythmia- CHB, VT, ectopics  y & x prominent- constric. pericarditis  y prominent, x absent- TR  y slow- TS
  • 14. Inspection  Proper exposure  Deformity  Apex beat  Scars  Respiration
  • 15. Palpation- apex beat  Lowest & outermost point of definite cardiac pulsation  Lies just medial to MCL in 5th ICS  Size- 1-2.5 cm.  Caused by left ventricle  Normally <50% of systole  Hyperkinetic- <50% systole, forceful, dilated LV, regurgitant lesion (MR/AR), ±S3  Sustained- >50% systole, forceful, hypertrophic LV, stenotic lesion (AS/HOCM) or HT, ±S4
  • 16. Palpation- other  Parasternal impulse- enlarged LA/RV  Thrill- palpable murmur (>grade 3)  Other palpable sounds  S1- apex, MS  A2- R 2nd ICS, systemic HT  P2- L 2nd ICS, pulmonary HT  S3/S4- apex/L parasternal, dilatation/hypertrophy of ventricle  Aortic pulsation- chest/epigastrium, consider aneurysm
  • 17. Auscultation- normal  Palpate carotid simultaneously  Areas for auscultation  Mitral- apex  Tricuspid- L parasternal  Aortic- R 2nd ICS  Pulmonary- L 2nd ICS  Only S1 & S2 are heard  S1- closure of MV/TV, single, systole begins  S2- closure of AV/PV, split (normally A2 before P2, best heard in pulmonary area during inspiration), systole ends
  • 19. Auscultation- abnormal  Altered intensity (soft/loud)  All soft- emphysema, pericardial effusion  Soft S1/A2/P2- calcific valve  Loud S1- non-calcific MS, tachycardia, ASD/Ebstein’s, high cardiac output states like anemia, exercise, hyperthyroidism  Loud A2/P2- systemic/pulmonary HT  Splitting of S2  Single- AS/PS  Wide- PS, RBBB  Fixed- ASD  Reverse- LBBB (appears on expiration, disappears on inspiration)  Syst./pulm. HT- wide, narrow or reverse split
  • 20. Auscultation- other sounds  Systolic-  Early ejection click- AV/PV  Mid/late ejection click- MVP  Diastolic-  Opening snap- MS  S3- poor LV compliance- CHF, DCMP  S4- increased LV resistance- HT, AS, HOCM
  • 21. Auscultation- murmurs  Due to turbulent blood flow  Characteristics-  Timing- systolic/diastolic, early/mid/late/pan  Location- apex, LLSB, aortic/pulmonary area  Radiation- AS to carotids, MR to axilla  Duration/shape  Grade- 1-6, grade 4 causes palpable thrill  Pitch- high-regurgitant/low-stenotic  Quality- blowing high-pitched- regurgitant, harsh rough rumbling low-pitched- stenotic  Variation- with respiration, position, valsalva etc.
  • 22. Auscultation- murmurs  Systolic-  Ejection- AS/PS, flow murmurs (non pathological)  Pan- MR/TR, VSD  Late- MVP, HOCM  Diastolic-  Early- AR/PR  Mid- MS/TS  Continuous- PDA