This document discusses trauma from occlusion (TFO). It begins by defining TFO as pathologic alterations or adaptive changes that develop in the periodontium as a result of undue force from chewing muscles. It describes primary TFO resulting from sudden impacts and secondary TFO from gradual changes that occur with reduced bone support. Clinical features include tooth pain and mobility. Radiographic features include widened ligament space and buttressing bone. Treatment focuses on reducing tooth mobility, eliminating prematurities, and using splints. While TFO alone may increase mobility, inflammation is required for attachment loss.
The document discusses bone loss patterns in periodontal disease. It notes that the balance between bone formation and resorption maintains bone height and density under normal conditions. The most common cause of bone destruction in periodontal disease is the extension of gingival inflammation into the supporting bone and tissues. This can lead to horizontal bone loss when inflammation travels along the bone crest or vertical bone loss when it travels directly into the periodontal ligament space. Bone destruction patterns include osseous craters in the interdental bone, bulbous bone contours, reversed architecture with loss of interdental bone, and furcation involvement in multi-rooted teeth.
INTRODUCTION
DEFINITION
TYPES OF TRAUMA FROM OCCLUSION
GLICKMAN CONCEPT
WAERHAUG CONCEPT
STAGES OF TISSUE RESPONSE TO INJURY
CLINICAL AND RADIOGRAPHIC FEATURES OF TFO
CLINICAL DIAGNOSIS OF TFO
TFO AND IMPLANTS
TREATMENT OF TFO
CONCLUSION
REFRENCES
Bone loss occurs when resorption exceeds formation due to an imbalance between inflammatory and anti-inflammatory signals. Bacterial products stimulate immune cells like macrophages and T cells to release cytokines that promote osteoclast formation through the RANKL pathway. Mature osteoclasts attach to bone and secrete acids and enzymes to degrade the mineralized matrix and organic components. While inflammatory mediators increase resorption, anti-inflammatory cytokines from T cells and other cells inhibit osteoclasts and support new bone formation to maintain equilibrium. Periodontitis results from this inflammatory process overwhelming the protective mechanisms and leading to net bone destruction.
Bone loss and patterns of bone destructionvidushiKhanna1
- introduction
- bone resorption
- factors causing bone destruction in periodontal disease
-- destruction by extension of gingival inflammation
--- histopathology
--- pathways of spread of inflammation
--- radius of action
--- periods of destruction
---- mechanism of destruction
-- bone destruction caused by TFO
-- bone destruction caused by systemic disorders
- factors determining bone morphology in periodontal disease
-- normal variation of alveolar bone
-- exostosis
-- butressing bone formation
-- food impaction
-- agressive periodontitis
- patterns of bone destruction
-- horizontal bone loss
-- vertical or angular defects
-- osseous craters
-- bulbous bone contours
-- reversed architecture
-- ledges
- furcation involvement
-- classification
-conclusion
CLINICAL FEATURES OF GINGIVITIS AND ITS CORRELATION WITH MICROSCOPIC FINDINGSShilpa Shiv
This document summarizes the clinical features of gingivitis. It describes the stages of gingival inflammation from initial lesion to early lesion. In stage I (initial lesion), microscopic changes include dilation of capillaries and increased leukocyte migration and accumulation. In stage II (early lesion), clinical signs like erythema and bleeding on probing appear within 1 week. Microscopically, collagen destruction increases and polymorphonuclear leukocytes continue migrating into the gingival tissues. The document also discusses experimental gingivitis studies, prevalence of gingivitis, and microscopic characteristics at each stage.
This document discusses gingival recession, including its definitions, classifications, etiology, factors affecting treatment outcomes, and treatments. It provides an overview of several classification systems for gingival recession, including those proposed by Sullivan and Atkins, Miller, Mahajan, Cairo, and Ashish Kumar. Miller's classification is the most widely used but has limitations, so modifications have been suggested. The document also proposes a new comprehensive classification system that aims to address the limitations of previous systems.
This document discusses trauma from occlusion (TFO), which refers to pathologic alterations or adaptive changes in the periodontium resulting from excessive occlusal forces. It covers the historical understanding of TFO, definitions, classifications, clinical features, and the periodontal response and adaptation to excessive forces. It also examines Glickman's concept of co-destruction between TFO and plaque-associated periodontal disease. The document provides details on injury, repair, remodeling processes in the periodontium in response to TFO.
This document discusses trauma from occlusion (TFO). It begins by defining TFO as pathologic alterations or adaptive changes that develop in the periodontium as a result of undue force from chewing muscles. It describes primary TFO resulting from sudden impacts and secondary TFO from gradual changes that occur with reduced bone support. Clinical features include tooth pain and mobility. Radiographic features include widened ligament space and buttressing bone. Treatment focuses on reducing tooth mobility, eliminating prematurities, and using splints. While TFO alone may increase mobility, inflammation is required for attachment loss.
The document discusses bone loss patterns in periodontal disease. It notes that the balance between bone formation and resorption maintains bone height and density under normal conditions. The most common cause of bone destruction in periodontal disease is the extension of gingival inflammation into the supporting bone and tissues. This can lead to horizontal bone loss when inflammation travels along the bone crest or vertical bone loss when it travels directly into the periodontal ligament space. Bone destruction patterns include osseous craters in the interdental bone, bulbous bone contours, reversed architecture with loss of interdental bone, and furcation involvement in multi-rooted teeth.
INTRODUCTION
DEFINITION
TYPES OF TRAUMA FROM OCCLUSION
GLICKMAN CONCEPT
WAERHAUG CONCEPT
STAGES OF TISSUE RESPONSE TO INJURY
CLINICAL AND RADIOGRAPHIC FEATURES OF TFO
CLINICAL DIAGNOSIS OF TFO
TFO AND IMPLANTS
TREATMENT OF TFO
CONCLUSION
REFRENCES
Bone loss occurs when resorption exceeds formation due to an imbalance between inflammatory and anti-inflammatory signals. Bacterial products stimulate immune cells like macrophages and T cells to release cytokines that promote osteoclast formation through the RANKL pathway. Mature osteoclasts attach to bone and secrete acids and enzymes to degrade the mineralized matrix and organic components. While inflammatory mediators increase resorption, anti-inflammatory cytokines from T cells and other cells inhibit osteoclasts and support new bone formation to maintain equilibrium. Periodontitis results from this inflammatory process overwhelming the protective mechanisms and leading to net bone destruction.
Bone loss and patterns of bone destructionvidushiKhanna1
- introduction
- bone resorption
- factors causing bone destruction in periodontal disease
-- destruction by extension of gingival inflammation
--- histopathology
--- pathways of spread of inflammation
--- radius of action
--- periods of destruction
---- mechanism of destruction
-- bone destruction caused by TFO
-- bone destruction caused by systemic disorders
- factors determining bone morphology in periodontal disease
-- normal variation of alveolar bone
-- exostosis
-- butressing bone formation
-- food impaction
-- agressive periodontitis
- patterns of bone destruction
-- horizontal bone loss
-- vertical or angular defects
-- osseous craters
-- bulbous bone contours
-- reversed architecture
-- ledges
- furcation involvement
-- classification
-conclusion
CLINICAL FEATURES OF GINGIVITIS AND ITS CORRELATION WITH MICROSCOPIC FINDINGSShilpa Shiv
This document summarizes the clinical features of gingivitis. It describes the stages of gingival inflammation from initial lesion to early lesion. In stage I (initial lesion), microscopic changes include dilation of capillaries and increased leukocyte migration and accumulation. In stage II (early lesion), clinical signs like erythema and bleeding on probing appear within 1 week. Microscopically, collagen destruction increases and polymorphonuclear leukocytes continue migrating into the gingival tissues. The document also discusses experimental gingivitis studies, prevalence of gingivitis, and microscopic characteristics at each stage.
This document discusses gingival recession, including its definitions, classifications, etiology, factors affecting treatment outcomes, and treatments. It provides an overview of several classification systems for gingival recession, including those proposed by Sullivan and Atkins, Miller, Mahajan, Cairo, and Ashish Kumar. Miller's classification is the most widely used but has limitations, so modifications have been suggested. The document also proposes a new comprehensive classification system that aims to address the limitations of previous systems.
This document discusses trauma from occlusion (TFO), which refers to pathologic alterations or adaptive changes in the periodontium resulting from excessive occlusal forces. It covers the historical understanding of TFO, definitions, classifications, clinical features, and the periodontal response and adaptation to excessive forces. It also examines Glickman's concept of co-destruction between TFO and plaque-associated periodontal disease. The document provides details on injury, repair, remodeling processes in the periodontium in response to TFO.
Trauma from occlusion occurs when excessive occlusal forces exceed the adaptive capacity of the periodontium, causing injury. It can be acute from a sudden impact or chronic from gradual changes in occlusion over time. Clinical signs include increased tooth mobility, bone loss, and widening of the periodontal ligament space seen radiographically. Theories suggest trauma alters the pathway of inflammation and may increase periodontal destruction. Treatment involves eliminating traumatic occlusal contacts through procedures like occlusal adjustment and splinting to allow the tissues to heal.
Splinting is one of the oldest forms of aids to periodontal therapy. By redistribution of forces on the affected teeth the splint minimizes the effects caused by loss of support. Splinting teeth to each other allows weakened teeth to be supported by neighbouring teeth. This presentation reviews the rationale, techniques, advantages and ill effects of stabilization of teeth by splinting as an aid to periodontal therapy. With the acceptance and clinical predictability of adhesive procedures, the use of conservative bonding techniques to splint teeth offers a useful alternative to more invasive restorative procedures. Loss of tooth-supporting structures results in tooth mobility. Increased tooth mobility adversely affects function, aesthetics, and the patient’s comfort. Splints are used to overcome all these problems. When faced with the dilemma of how to manage periodontally compromised teeth, splinting of mobile teeth to stronger adjacent teeth is a viable option. This prolongs the life expectancy of loose teeth, gives stability for the periodontium to reattach, and improves comfort, function and aesthetics.
2017 classification of periodontal and periimplant diseasesDr. Bibina George
The document summarizes the key changes in the 2017 classification of periodontal and peri-implant diseases compared to the 1999 classification. The 2017 classification introduces staging and grading systems for periodontitis to indicate disease severity and risk of progression. It also includes classifications for peri-implant health, mucositis, and implantitis, as well as mucogingival deformities. The document reviews these changes and provides references for the revised classification system.
RADIOGRAPHIC AIDS IN THE DIAGNOSIS OF PERIODONTAL DISEASERupal Patle
The document discusses the radiographic evaluation of periodontal disease. It begins by stating that radiographs are useful for diagnosis, prognosis, and treatment evaluation but are an adjunct to clinical examination. Radiographs reveal changes to calcified tissues from past cellular activity but not current activity. Interdental septa and the lamina dura normally appear as thin radiopaque borders and variations in technique can distort radiographic findings. Early signs of periodontal disease on radiographs include fuzziness or breaks in the lamina dura continuity. Progressive bone destruction appears as wedge-shaped radiolucencies and reduced crest height. Furcation involvement and abscesses may also be visualized but radiographs have limitations. Clinical probing with radiopa
Coronoplasty is a procedure that reshapes teeth to eliminate occlusal interferences and establish a functional relationship favorable for the periodontium. It is indicated for trauma from occlusion on a single tooth or few teeth. The objectives are to change afferent impulses, reduce tooth mobility, and verticalize occlusal forces. The procedure involves analyzing the occlusion, determining the endpoint, selecting an occlusal scheme, and making adjustments like grooving, spheroiding, or pointing to reduce supracontacts. The goals are light contact between incisors and firm contact between posterior teeth without asymmetry or sharp sounds.
This document provides information on scaling and root planing procedures. It discusses the different types of periodontal instruments used, including probes, explorers, scalers, curettes, and ultrasonic instruments. It covers the principles of instrumentation, including proper positioning, illumination, use of sharp instruments, and techniques for instrument stabilization and activation. The goal of scaling and root planing is to remove biofilm, calculus, and rough surfaces from teeth to produce a smooth, clean surface and reduce inflammation.
1. Osseous surgery involves modifying the alveolar bone support of teeth and includes techniques like osteoplasty and ostectomy. It aims to eliminate pockets and correct unphysiological bone architecture.
2. Factors in selecting a technique include the amount and location of bone loss, root trunk length, and anatomical limitations. Techniques range from non-resective procedures like osteoplasty to resective procedures like ostectomy.
3. Outcomes of osseous surgery generally include pocket elimination and establishment of physiological bone contours and architecture, though some bone loss from remodeling is expected in the range of 0.06mm to 1.2mm.
The document discusses treatment planning in periodontics. It begins by defining treatment planning and outlining the short and long-term goals. These include eliminating infection and inflammation and reconstructing a healthy dentition. The treatment plan is the blueprint and involves decisions about emergency treatment, extractions, nonsurgical and surgical therapies, restorations, and maintenance. Phases of treatment are discussed including preliminary, nonsurgical, surgical, and maintenance phases. Factors in deciding whether to extract or preserve a tooth are also outlined.
This document discusses the process and techniques for coronoplasty, which is a selective reduction of occlusal areas to influence mechanical contact situations and neural sensory input patterns. It describes the 10 basic steps for performing coronoplasty, which include adjusting the intercuspal position and retruded contact position, removing prematurities, establishing stable simultaneous contacts, testing and adjusting incisal contacts and excursions, and finishing with polishing rough surfaces. The goal is to establish an optimal occlusion with stable contacts, smooth excursions, and decreased tooth mobility and stress on the teeth and temporomandibular joints.
The document summarizes the biology of tooth movement during orthodontic treatment. It discusses how application of force leads to bone remodeling through pressure and tension on the periodontal ligament. Optimal force causes bone resorption on the pressure side and deposition on the tension side through cellular processes. Tooth movement occurs in initial, lag, and post-lag phases as the hyalinized tissue is removed and bone remodeling allows for further movement.
mucogingival surgery or plastic surgery of muco-gingival tissue is a surgical procedure targeted to correct and eliminate anatomic, developmental and traumatic alterations of gingiva.
AGGRESSIVE PERIODONTITIS
PRESENTER
DR. REBICCA RANJIT
DEPT. OF PERIODONTOLOGY & ORAL IMPLANTOLOGY
Why is there localisation of disease to 1st molars and incisors in LAP?
Often subjects present with attachment loss that does not fit the specific diagnostic criteria (AP or chronic periodontitis).
Schenkein et al. 1995: cigarette smoking was shown to be a risk factor for patients with generalized forms of AgP.
Smokers with GAP had more affected teeth and greater mean levels of attachment loss than patients with GAP who did not smoke.
IgG2 serum levels as well as antibody levels against A.a. are significantly depressed in subjects with GAP who smoked.
The biological width refers to the dimensions of the junctional epithelium and connective tissue attachment above the alveolar crest, which averages 2.04mm. Placement of restoration margins within 1mm of the gingival sulcus is ideal to preserve this biological width, while subgingival placement can lead to inflammation, recession, or bone loss by violating the biological width. When a violation occurs, it can be corrected by surgery to remove bone away from the margin by the ideal biological width distance, or by orthodontic extrusion. Maintaining the biological width is essential for periodontal health.
This document discusses trauma from occlusion (TFO) and its effects on the periodontium. It defines TFO as injury to the periodontal tissues caused by excessive occlusal forces. It describes the different types of occlusal forces and their effects, including acute vs chronic TFO, and primary vs secondary TFO. It also discusses the stages of tissue response to increased occlusal forces: injury, repair, and adaptive remodeling. The document examines various theories on the interaction between TFO and plaque-associated periodontal disease.
Trauma from occlusion
In Periodontics
definition of trauma from occlusion by WHO (1978)
and many more definitions by different authors
Factors involved in the etiology of trauma from occlusion
which includes 1.) precipitating factors : such as Magnitude, Direction, Duration of force applied, Frequency of force applied
2.) Predisosing factors : intrinsic factors & extrinsic factors
# Terminologies which are used which have been used to describe occlusion trauma
Glickmans theory of co-destruction
occlusal forces during jaw movement
classification of trauma from occlusion which includes acute , chronic , primary secondary
Stages of tissue response to excessive occlusion forces
stage 1 INJURY
slightly excessive forces
greater then slightly excessive forces
severely high forces
stage 2: REPAIR
stage 3: ADAPTIVE REMODELLING OF THE PERIDONTIUM
Examination And Diagnosis of trauma from occlusion
signs of trauma from occlusion :
tooth mobility
tooth migration
wear pattern abfraction
V shaped or angled gingival recession
Buccal bone dehisence
fermitus test
SYMPTOMS OF TRAUMA FROM OCCLUSION
radiographic features of trauma from occlusion
effects of insufficent occlusal forces
reversibility of traumatic lesion
effects of excessive occlusion forces on dental pulp
Influence of trauma from occlusion on progression of marginal periodontitis
pathological migration
pathogenisis
management of trauma from occlusion
treatment of trauma from occlusion
conclusion
Apexogenesis & apexification in pediatric dentistryDr. Harsh Shah
SDDCH Parbhani
Presented by : Vipul GIratkar
Dept. of Pediatric dentitstry
Guided by . Dr. Rehan Khan
DIscussion regarding apexification and apexogenesis
The document discusses the relationship between pulpal and periodontal diseases. It states that diseases of the pulp or periodontium can lead to secondary diseases in the other via the apical foramen, lateral canals, or dentinal tubules. Both endodontic and periodontal examinations are important to diagnose the origin of lesions. Treatment should focus first on the primary disease, but both may need treatment if the secondary disease is established.
traumatic injuries in children: trauma to teeth and softJeena Paul
This document discusses traumatic injuries to children's teeth and soft tissues. It notes that trauma occurs frequently in children, with the highest incidence between ages 2-3. Common causes of trauma include falls, accidents, and sports. Examination of injured children should involve a thorough history, clinical examination of soft tissues and teeth, and radiographs to check for fractures or displaced teeth/bone. Proper documentation of findings is important for diagnosis and treatment planning.
This document outlines a treatment plan for periodontal disease. It includes 5 phases: emergency, etiotropic (non-surgical), surgical, restorative, and maintenance. The etiotropic phase involves nonsurgical therapies like scaling, root planing, and oral hygiene instruction. The surgical phase uses various periodontal surgeries to further treat pockets and furcations. The restorative phase focuses on final restorations. Lastly, the maintenance phase provides periodic recall visits to monitor the patient's condition. The overall goal is to resolve inflammation and reduce pocket depths through a coordinated approach involving multiple dental specialists.
Phase I periodontal therapy is the first in the chronologic sequence of procedures that constitute periodontal treatment. It is also referred to as cause related therapy or non-surgical periodontal therapy.
This document defines trauma from occlusion (TFO) and outlines its causes, classification, clinical features, radiographic findings, and treatment. TFO occurs when occlusal forces exceed the adaptive capacity of the periodontium, causing tissue injury. It can be acute from a sudden impact or chronic from gradual changes in occlusion. Factors that increase traumatic forces are magnitude, direction, and duration of forces. TFO is classified as primary, secondary, or combined based on causative factors. Clinical features include tooth mobility and pain. Radiographic findings show increased periodontal ligament space and bone loss. Treatment goals are to maintain periodontal health and function through occlusal adjustment, habit management, stabilization, orthodontics, reconstruction,
When occlusal forces exceed the adaptive capacity of the tissues, tissue injury results. The resultant injury is termed as trauma from occlusion.
TFO refers to tissue injury, not the occlusal force. An occlusion that produces such injury is termed as traumatic occlusion.
Trauma from occlusion occurs when excessive occlusal forces exceed the adaptive capacity of the periodontium, causing injury. It can be acute from a sudden impact or chronic from gradual changes in occlusion over time. Clinical signs include increased tooth mobility, bone loss, and widening of the periodontal ligament space seen radiographically. Theories suggest trauma alters the pathway of inflammation and may increase periodontal destruction. Treatment involves eliminating traumatic occlusal contacts through procedures like occlusal adjustment and splinting to allow the tissues to heal.
Splinting is one of the oldest forms of aids to periodontal therapy. By redistribution of forces on the affected teeth the splint minimizes the effects caused by loss of support. Splinting teeth to each other allows weakened teeth to be supported by neighbouring teeth. This presentation reviews the rationale, techniques, advantages and ill effects of stabilization of teeth by splinting as an aid to periodontal therapy. With the acceptance and clinical predictability of adhesive procedures, the use of conservative bonding techniques to splint teeth offers a useful alternative to more invasive restorative procedures. Loss of tooth-supporting structures results in tooth mobility. Increased tooth mobility adversely affects function, aesthetics, and the patient’s comfort. Splints are used to overcome all these problems. When faced with the dilemma of how to manage periodontally compromised teeth, splinting of mobile teeth to stronger adjacent teeth is a viable option. This prolongs the life expectancy of loose teeth, gives stability for the periodontium to reattach, and improves comfort, function and aesthetics.
2017 classification of periodontal and periimplant diseasesDr. Bibina George
The document summarizes the key changes in the 2017 classification of periodontal and peri-implant diseases compared to the 1999 classification. The 2017 classification introduces staging and grading systems for periodontitis to indicate disease severity and risk of progression. It also includes classifications for peri-implant health, mucositis, and implantitis, as well as mucogingival deformities. The document reviews these changes and provides references for the revised classification system.
RADIOGRAPHIC AIDS IN THE DIAGNOSIS OF PERIODONTAL DISEASERupal Patle
The document discusses the radiographic evaluation of periodontal disease. It begins by stating that radiographs are useful for diagnosis, prognosis, and treatment evaluation but are an adjunct to clinical examination. Radiographs reveal changes to calcified tissues from past cellular activity but not current activity. Interdental septa and the lamina dura normally appear as thin radiopaque borders and variations in technique can distort radiographic findings. Early signs of periodontal disease on radiographs include fuzziness or breaks in the lamina dura continuity. Progressive bone destruction appears as wedge-shaped radiolucencies and reduced crest height. Furcation involvement and abscesses may also be visualized but radiographs have limitations. Clinical probing with radiopa
Coronoplasty is a procedure that reshapes teeth to eliminate occlusal interferences and establish a functional relationship favorable for the periodontium. It is indicated for trauma from occlusion on a single tooth or few teeth. The objectives are to change afferent impulses, reduce tooth mobility, and verticalize occlusal forces. The procedure involves analyzing the occlusion, determining the endpoint, selecting an occlusal scheme, and making adjustments like grooving, spheroiding, or pointing to reduce supracontacts. The goals are light contact between incisors and firm contact between posterior teeth without asymmetry or sharp sounds.
This document provides information on scaling and root planing procedures. It discusses the different types of periodontal instruments used, including probes, explorers, scalers, curettes, and ultrasonic instruments. It covers the principles of instrumentation, including proper positioning, illumination, use of sharp instruments, and techniques for instrument stabilization and activation. The goal of scaling and root planing is to remove biofilm, calculus, and rough surfaces from teeth to produce a smooth, clean surface and reduce inflammation.
1. Osseous surgery involves modifying the alveolar bone support of teeth and includes techniques like osteoplasty and ostectomy. It aims to eliminate pockets and correct unphysiological bone architecture.
2. Factors in selecting a technique include the amount and location of bone loss, root trunk length, and anatomical limitations. Techniques range from non-resective procedures like osteoplasty to resective procedures like ostectomy.
3. Outcomes of osseous surgery generally include pocket elimination and establishment of physiological bone contours and architecture, though some bone loss from remodeling is expected in the range of 0.06mm to 1.2mm.
The document discusses treatment planning in periodontics. It begins by defining treatment planning and outlining the short and long-term goals. These include eliminating infection and inflammation and reconstructing a healthy dentition. The treatment plan is the blueprint and involves decisions about emergency treatment, extractions, nonsurgical and surgical therapies, restorations, and maintenance. Phases of treatment are discussed including preliminary, nonsurgical, surgical, and maintenance phases. Factors in deciding whether to extract or preserve a tooth are also outlined.
This document discusses the process and techniques for coronoplasty, which is a selective reduction of occlusal areas to influence mechanical contact situations and neural sensory input patterns. It describes the 10 basic steps for performing coronoplasty, which include adjusting the intercuspal position and retruded contact position, removing prematurities, establishing stable simultaneous contacts, testing and adjusting incisal contacts and excursions, and finishing with polishing rough surfaces. The goal is to establish an optimal occlusion with stable contacts, smooth excursions, and decreased tooth mobility and stress on the teeth and temporomandibular joints.
The document summarizes the biology of tooth movement during orthodontic treatment. It discusses how application of force leads to bone remodeling through pressure and tension on the periodontal ligament. Optimal force causes bone resorption on the pressure side and deposition on the tension side through cellular processes. Tooth movement occurs in initial, lag, and post-lag phases as the hyalinized tissue is removed and bone remodeling allows for further movement.
mucogingival surgery or plastic surgery of muco-gingival tissue is a surgical procedure targeted to correct and eliminate anatomic, developmental and traumatic alterations of gingiva.
AGGRESSIVE PERIODONTITIS
PRESENTER
DR. REBICCA RANJIT
DEPT. OF PERIODONTOLOGY & ORAL IMPLANTOLOGY
Why is there localisation of disease to 1st molars and incisors in LAP?
Often subjects present with attachment loss that does not fit the specific diagnostic criteria (AP or chronic periodontitis).
Schenkein et al. 1995: cigarette smoking was shown to be a risk factor for patients with generalized forms of AgP.
Smokers with GAP had more affected teeth and greater mean levels of attachment loss than patients with GAP who did not smoke.
IgG2 serum levels as well as antibody levels against A.a. are significantly depressed in subjects with GAP who smoked.
The biological width refers to the dimensions of the junctional epithelium and connective tissue attachment above the alveolar crest, which averages 2.04mm. Placement of restoration margins within 1mm of the gingival sulcus is ideal to preserve this biological width, while subgingival placement can lead to inflammation, recession, or bone loss by violating the biological width. When a violation occurs, it can be corrected by surgery to remove bone away from the margin by the ideal biological width distance, or by orthodontic extrusion. Maintaining the biological width is essential for periodontal health.
This document discusses trauma from occlusion (TFO) and its effects on the periodontium. It defines TFO as injury to the periodontal tissues caused by excessive occlusal forces. It describes the different types of occlusal forces and their effects, including acute vs chronic TFO, and primary vs secondary TFO. It also discusses the stages of tissue response to increased occlusal forces: injury, repair, and adaptive remodeling. The document examines various theories on the interaction between TFO and plaque-associated periodontal disease.
Trauma from occlusion
In Periodontics
definition of trauma from occlusion by WHO (1978)
and many more definitions by different authors
Factors involved in the etiology of trauma from occlusion
which includes 1.) precipitating factors : such as Magnitude, Direction, Duration of force applied, Frequency of force applied
2.) Predisosing factors : intrinsic factors & extrinsic factors
# Terminologies which are used which have been used to describe occlusion trauma
Glickmans theory of co-destruction
occlusal forces during jaw movement
classification of trauma from occlusion which includes acute , chronic , primary secondary
Stages of tissue response to excessive occlusion forces
stage 1 INJURY
slightly excessive forces
greater then slightly excessive forces
severely high forces
stage 2: REPAIR
stage 3: ADAPTIVE REMODELLING OF THE PERIDONTIUM
Examination And Diagnosis of trauma from occlusion
signs of trauma from occlusion :
tooth mobility
tooth migration
wear pattern abfraction
V shaped or angled gingival recession
Buccal bone dehisence
fermitus test
SYMPTOMS OF TRAUMA FROM OCCLUSION
radiographic features of trauma from occlusion
effects of insufficent occlusal forces
reversibility of traumatic lesion
effects of excessive occlusion forces on dental pulp
Influence of trauma from occlusion on progression of marginal periodontitis
pathological migration
pathogenisis
management of trauma from occlusion
treatment of trauma from occlusion
conclusion
Apexogenesis & apexification in pediatric dentistryDr. Harsh Shah
SDDCH Parbhani
Presented by : Vipul GIratkar
Dept. of Pediatric dentitstry
Guided by . Dr. Rehan Khan
DIscussion regarding apexification and apexogenesis
The document discusses the relationship between pulpal and periodontal diseases. It states that diseases of the pulp or periodontium can lead to secondary diseases in the other via the apical foramen, lateral canals, or dentinal tubules. Both endodontic and periodontal examinations are important to diagnose the origin of lesions. Treatment should focus first on the primary disease, but both may need treatment if the secondary disease is established.
traumatic injuries in children: trauma to teeth and softJeena Paul
This document discusses traumatic injuries to children's teeth and soft tissues. It notes that trauma occurs frequently in children, with the highest incidence between ages 2-3. Common causes of trauma include falls, accidents, and sports. Examination of injured children should involve a thorough history, clinical examination of soft tissues and teeth, and radiographs to check for fractures or displaced teeth/bone. Proper documentation of findings is important for diagnosis and treatment planning.
This document outlines a treatment plan for periodontal disease. It includes 5 phases: emergency, etiotropic (non-surgical), surgical, restorative, and maintenance. The etiotropic phase involves nonsurgical therapies like scaling, root planing, and oral hygiene instruction. The surgical phase uses various periodontal surgeries to further treat pockets and furcations. The restorative phase focuses on final restorations. Lastly, the maintenance phase provides periodic recall visits to monitor the patient's condition. The overall goal is to resolve inflammation and reduce pocket depths through a coordinated approach involving multiple dental specialists.
Phase I periodontal therapy is the first in the chronologic sequence of procedures that constitute periodontal treatment. It is also referred to as cause related therapy or non-surgical periodontal therapy.
This document defines trauma from occlusion (TFO) and outlines its causes, classification, clinical features, radiographic findings, and treatment. TFO occurs when occlusal forces exceed the adaptive capacity of the periodontium, causing tissue injury. It can be acute from a sudden impact or chronic from gradual changes in occlusion. Factors that increase traumatic forces are magnitude, direction, and duration of forces. TFO is classified as primary, secondary, or combined based on causative factors. Clinical features include tooth mobility and pain. Radiographic findings show increased periodontal ligament space and bone loss. Treatment goals are to maintain periodontal health and function through occlusal adjustment, habit management, stabilization, orthodontics, reconstruction,
When occlusal forces exceed the adaptive capacity of the tissues, tissue injury results. The resultant injury is termed as trauma from occlusion.
TFO refers to tissue injury, not the occlusal force. An occlusion that produces such injury is termed as traumatic occlusion.
The document provides information on trauma from occlusion and coronoplasty. It defines trauma from occlusion as damage to the periodontium caused by excessive occlusal forces. Coronoplasty involves selective reduction of occlusal surfaces to influence mechanical contact conditions and sensory input, with the aim of reducing excessive tooth mobility and providing functional stimulation for periodontal health. The document discusses the diagnosis, classification, and clinical features of trauma from occlusion, as well as the objectives, methods, and techniques used in performing coronoplasty.
This document provides information about trauma from occlusion including its classification, types (acute vs chronic), causes (primary, secondary, combined), and stages of tissue response (injury, repair, adaptive remodeling). It discusses the histological changes that occur at each stage and clinical signs of trauma from occlusion like tooth mobility and bone loss patterns seen radiographically. It also discusses two concepts (Glickman and Waerhaugs) on the influence of trauma from occlusion on the progression of marginal periodontitis and the development of infrabony pockets.
This document discusses trauma from occlusion (TFO), which refers to injury to the periodontium resulting from excessive occlusal forces. It defines key terms, classifies TFO, and describes the stages of tissue response and role of occlusion in periodontal disease pathogenesis. Several human and animal studies are summarized that investigated the relationship between occlusal forces and periodontal disease, generally finding that excessive forces alone do not cause attachment loss but may alter the pathway of destruction in the presence of plaque. The document provides an overview of TFO and its relationship to periodontal disease.
*RAMA DENTAL COLLEGE HOSPITAL AND RESEARCH CENTRE
DEPARTMENT OF PERIODONTOLOGY
TRAUMA FROM OCCLUSION
When occlusal forces exceed the adaptive capacity of the tissues, tissue injury results the resultant injury is termed as trauma from occlusion.
Trauma from occlusion occurs when occlusal forces exceed the tolerance of the periodontal tissues, causing injury. The document defines occlusion and discusses the forces involved. It describes physiologic, non-physiologic, and therapeutic occlusion and outlines the stages of tissue response to increased forces - injury, repair, and adaptive remodeling. Clinical signs of trauma from occlusion include increased tooth mobility, which can be initial or secondary mobility. Both physiologic adaptation and pathological response to excessive forces present as increased mobility clinically.
Trauma from occlusion in Periodontics.pptxSUBHRADIPKAYAL
Contents
1. Definitions
2. Introduction
3. Classification of Trauma from occlusion
4. Stages of tissue response
5. Clinical features
6. Radiological features
7. Trauma from occlusion and plaque associated periodontal disease
8. Treatment of TFO
9. References
Definitions
• When occlusal forces exceed the adaptive capacity of tissues, tissue injury results. The resultant injury is termed as trauma from occlusion. - Carranza 10th edition
• Trauma from occlusion is a term used to describe pathologic alterations or adaptive changes which develop in the periodontium as a result of undue force produced by the masticatory muscles. - Lindhe 6th edition
• Stillman (1917) as “a condition where injury results to the supporting structures of the teeth by the act of bringing the jaws into a closed position”.
• WHO (1978) defined trauma from occlusion as “damage in the periodontium caused by stress on the teeth produced directly or indirectly by teeth of the opposing jaw”.
• Injury resulting in tissue changes within the attachment apparatus as a result of occlusal force(s). - AAP Glossary of periodontal terms 2001; 4th Edition
Introduction
• The periodontal ligament has a cushioning effect on forces applied to teeth as means to accommodate forces exerted on the crown.
• When there is increase in occlusal forces, changes occur in the periodontium in order to accommodate such forces.
• Changes occur in magnitude, direction, duration and frequency of increased occlusal forces.
Increased magnitude of occlusal forces
• Widening of periodontal ligament space.
• An increase in number and width of periodontal ligament fibers.
• An increase in the density of alveolar bone.
Changes in direction of occlusal forces
• Reorientation of the stresses and strains within the periodontium.
• The principal fibers of the periodontal ligament are arranged so that they best accommodate occlusal forces along the long axis of the tooth.
• Lateral (horizontal) and torque (rotational) forces are more likely to injure the periodontium.
Duration and frequency of occlusal forces
• Constant pressure on the bone is more injurious than intermittent forces.
• The more frequent the application of an intermittent force, the more injurious the force is to the periodontium.
Classification
According to mode of onset
1. Acute
2. Chronic
According to the capacity of the periodontium to resist to occlusal forces
1. Primary
2. Secondary
Acute trauma from occlusion
• Acute trauma from occlusion results from an abrupt occlusal impact such as that produced by biting on a hard object. Restorations or prosthetic appliances that interfere with or alter the direction of occlusal forces on the teeth may also induce acute trauma.
• Clinical features
1. Tooth pain
2. Sensitivity to percussion
3. Tooth mobility
Chronic trauma from occlusion
• It is more common than acute trauma from occlusion and is of greater clinical significance.
This document defines and describes primary and secondary occlusal trauma, which are injuries to the periodontium resulting from excessive occlusal forces. Primary trauma occurs with normal bone and attachment levels, while secondary trauma occurs with bone or attachment loss. Histological studies in animals and humans show that occlusal forces can cause widening of the periodontal ligament space, bone remodeling, root resorption, and other changes depending on the force magnitude and direction. Clinical indicators include tooth mobility, occlusal prematurities, sensitivity, and radiographic changes like widening of the periodontal ligament space. Diagnosis involves examining history, teeth for attrition, decreased vertical dimension, and radiographs. Treatment may include extra
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The adaptive capacity of the periodontium allows it to accommodate forces from occlusion. When occlusal forces exceed this capacity, trauma from occlusion occurs, potentially leading to periodontal injury. The magnitude, direction, duration and frequency of forces influence the periodontium's response. Signs of trauma from occlusion include pain, mobility, increased periodontal pocketing, bone loss and root resorption. Trauma can alter the progression of periodontal disease from a suprabony to an infrabony pattern and increase the rate of attachment loss. Pathologic tooth migration may result when the balance of factors maintaining normal position is disturbed by periodontal disease.
INTRODUCTION
DEFINITION
TYPES OF TRAUMA FROM OCCLUSION
GLICKMAN CONCEPT
WAERHAUG CONCEPT
STAGES OF TISSUE RESPONSE TO INJURY
CLINICAL AND RADIOGRAPHIC FEATURES OF TFO
CLINICAL DIAGNOSIS OF TFO
TFO AND IMPLANTS
TREATMENT OF TFO
CONCLUSION
REFRENCES
Trauma from occlusion occurs when occlusal forces exceed the adaptive capacity of the periodontal tissues, resulting in tissue injury. There are two types: acute trauma from occlusion caused by abrupt changes in forces, and chronic trauma from occlusion caused by gradual changes over time from tooth wear or drift. Signs include excessive tooth pain, mobility, and periodontal pocketing or bone loss visible radiographically. Treatment involves occlusal adjustments, splinting, orthodontics, or extractions to reduce traumatic forces on the tissues.
This document defines and discusses trauma from occlusion (TFO). It describes the physiologic capacity of the periodontium to adapt to occlusal forces. Factors that can increase traumatic forces include magnitude, direction, and duration of forces. TFO is classified as acute, chronic, primary, secondary, or combined. Clinical features may include mobility, pain, fremitus, and radiographic findings like increased periodontal ligament space. Treatment aims to maintain the periodontium in comfort and function through approaches like occlusal adjustment, parafunctional habit management, stabilization, orthodontics, reconstruction, or extractions.
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Trauma from occlusion refers to tissue injury caused when occlusal forces exceed the adaptive capacity of the tissues. It can be classified as acute or chronic depending on the mode of onset, and primary or secondary depending on whether occlusion is the primary cause of periodontal destruction. Clinical features include increased tooth mobility while radiographic features show widened periodontal spaces and bone loss. The tissue response progresses through injury, repair, and adaptive remodeling stages. Plaque initiates gingivitis and pocket formation independently of trauma from occlusion. Management involves relieving injurious forces for permanent repair.
- Trauma from occlusion occurs when occlusal forces exceed the adaptive capacity of the periodontium, causing injury. It can be acute or chronic.
- The magnitude, direction, duration, and frequency of forces impact the periodontium's ability to adapt. Excessive pressure or tension can damage tissues.
- Primary trauma from occlusion is caused by changes in occlusal forces, while secondary trauma occurs when reduced bone support impairs the tissues' resistance to normal forces.
- The periodontium responds to trauma in three stages - injury, repair through new tissue formation, and adaptive remodeling to better withstand forces. Trauma can cause reversible damage if forces are reduced, or lead to irreversible injury if
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1) Trauma from occlusion refers to injury to the periodontium caused by excessive occlusal forces. It can be either acute due to sudden impact or chronic due to gradual excessive forces.
2) Primary trauma results from changes in occlusal forces, while secondary trauma occurs when the periodontium has a reduced capacity to withstand normal occlusal forces, such as in cases of periodontitis.
3) Signs of trauma from occlusion include mobility, pain, bone loss, and widening of the periodontal ligament space seen radiographically. Management involves identifying and eliminating the source of trauma, such as through occlusal adjustments.
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2. “ A condition where injury results to the
supporting structures of the teeth by the act
of bringing the jaws into a closed position.”
(stillman1917)
“damage in the periodontium caused by the
stress on the teeth produced directly or
indirectly by the teeth of opposing jaws”
(WHO in 1978)
3. “When occlusal forces exceed the adaptive
capacity of periodontal tissues, the tissue
injury results.This resultant injury is termed
as a trauma from occlusion.” ( a/c to Orban and Glickman)
4. There are many factors involved in the etiology
ofTFO. Broadly they can be divided into 2
categories:
1. Precipitating factor
2. Predisposing factor
5. 1. Precipitating factor
Destructive occlusion forces are the
precipitating or primary etiology of theTRO.
The occlusal forces when within the normal
range can be well adapted by tooth-
supporting soft tissues. But when these
forces exceed the adaptive capacity of the
tooth supporting tissues, pathological
changes can be seen in the soft tissue.
6. These forces are usually described in terms of
magnitude, direction, duration and
frequency.
Magnitude( the amount)- when it is
increased the periodontium reponds
a) With thickening of the periodontal ligament,
b) An increase in the number and width of the
periodontal ligament fibres and
c) An increase in the density of the alveolar
bone
7. Direction – the principal fibres of periodontal
ligament play an important role in
withstanding the occlusal forces and
transferring them to alveolar bone.
But , if the direction of occusal forces is
changed, these fibres are not able to
efficiently bear the forces hence injury
results.
8. Duration – constant pressure on the bone is
the more injurious than inttermittent forces.
Frequency - the more frequent the
application of an intermittent force, the more
injurious to the periodontium.
9. 2.Predisposing factor – these can be divided
into intrinsic and extrinsic factors.
Intinsic factors
a. the orientation of the long axis of the teeth in
relation to forces to which they are exposed.
b. The morphological characteristics of the
roots.
10. The size , shape, and number of the roots
determine how occlusal forces are
dessipated.
In general , short ,conical, slender or fusedroots
are more vulnerable toTFO.
c.The morphology of the alveolar process, i.e.
the quality and quantity of the alveolar bone
play an important role in absorbing the
occlusal forces.
11. Extrinsic factors
a. Local factor such as plaque which predispose
to alveolar bone loss.
b. Fabrication of long span bridges on few
teeth, thus overloading them.
c. Injudicious bone resection during surgical
periodontal therapy or oral surgical
procedure.
12. d. Parafunctional habits as a results of neurosis.
e. Other factors includes the food impaction,
overhanging filling, poorly coloured crown
and bridges, and ill fitting partial dentures.
13. 1. Depending on the onset and duration
Acute trauma from occlusion -produced by
biting on a hard object, in addition could also
be due to iatrogenic factors(faulty
restoration/prosthetic appliance.)
Chronic trauma from occlusion- due to tooth
wear, drifting movement, extrusion of teeth
combined with parafunctional habits such as
bruxium and clenching.
14. 2. Depending on the cause
Primary trauma from occlusion- it is a tissue
injury, which is elicited around a tooth with
normal height of periodontium.
example- insertion of high fillings, insertion of
prosthetic replacement, orthodontic
movement in functionally unacceptable
position.
15. Secondary trauma from occlusion - it is
related to situation in which occlusal forces
cause injury in a periodontium of reduced
height.
Example- periodontitis
16. When evaluating a patient suspected of having
occlusal trauma there are a number of clinical
and radiographic symptoms that may be
present.These indicators of trauma from
occlusion may include one or more of the
following
Clinical
1) Mobility (progressive)
2) Pain on chewing or percussion
3) Fremitus
4) Occlusal prematurities/discrepancies
17. 5)Wear facets in the presence of other clinical
indicators
6)Tooth migration
7) Chipped or fractured tooth (teeth)
8)Thermal sensitivity
Radiographic
1)Widened PDL space
2) Bone loss
(furcations; vertical; circumferential)
3) Root resorption
18. The response of tissue to increased occlusal
forces is explained under 3 stages:
1) Injury
2) Repair
3) Adaptive remodeling of the periodontuim
19. When tooth is exposed to excessive occlusal
forces, the periodontal tissue to withstand
and hence they distribute, while maintaining
the stability of tooth.
When tooth is subjected to horizontal forces the
tooth rotate or tilts in the direction of force.
This tilting results in the pressure and tension
zones, within the marginal and apical parts of
periodontium.
20.
21.
22.
23. Slightly excessive force
The slightly excessive force stimulate bone
resorption in the area of the pressure, causing
the widening of periodontal ligament space.
In the area of tension , elongation of
periodontal ligament fibres takes place.The
blood vessels on the pressure side are
compressed whereas on the tension side they
are enlarged. in the due course of time, slow
remodeling of the alveolar socket takes place.
24. Greater than slightly excessive forces
Marked change in tooth structures are seen.The
excessive compression of periodontal ligament
produces area of hyalinization. Areas of necrosis
can be seen in periodontal ligament due to
excessive trauma to the periodontal ligament
fibres and connective tissue cells including
fibroblast. Within 30 minutes of application of
such traumatic forces on the tooth, vascular
changes can be seen.
25. severely high forces
Severely high forces results in thrombosis,
hemorrhage, tearing of the periodontal
ligament, widening of periodontal ligament
space and alveolar bone resorption.
Under severely high force, on the pressure side
, there is a disturbance of blood flow in the
compressed PDL and cell death in the
compressed area of the PDL(hyalinization).
26. The 1st sign of hyalinization is the presence of
pyknotic nuclei in the cells, followed by areas of
acellularity, or cell free zones.
Due to injury caused by occlusal trauma , there is a
temporary reduction of mitotic activity of the
cells, including fibroblast and osteoblasts. In the
presence of excessive occlusal forces, all the
above changes take place, but if the forces are
removed or tooth moves away from the forces,
the periodontium is completely restored.
27. Depending on the types of forces there can be
many histologic changes
28.
29.
30. The diagnosis and assessment (flowchart) of
occlusal trauma is not merely made based on a
single examination, due to the necessarily
progressive nature of injury. Orthodontic
correction is usually restricted to cases where
tooth malpositions are the prime cause of
trauma.There are certain additional factors such
as morphology, prognosis of the teeth involved,
direction and magnitude of movement required
that will influence the decision of whether or not
orthodontic tooth movement is indicated.
31.
32.
33.
34. The treatment ofTFO involves removal of the
excessive occlusal forces and bringing the tooth/
teeth in a comfortable position. Many treatment
modalities have been adviced to treatTFO.
These include,
Occlusal adjustment
Management of parafunctional habits
Splinting of teeth
Orthodontic tooth movement
Occlusal reconstruction
Extraction of selected teeth