This document provides an overview of recurrent shoulder dislocation and operative intervention. It begins with a brief history of shoulder dislocation documentation and treatment. It then discusses the pathoanatomy, risk factors, classifications, and open vs arthroscopic surgical procedures for recurrent shoulder dislocation. Key open surgical techniques discussed include the Bankart procedure, capsular shift procedure, Putti-Platt procedure, Magnuson-Stack procedure, Bristow procedure, and Latarjet procedure. The document examines factors in determining the optimal treatment approach and whether open or arthroscopic stabilization is superior.
Posterolateral corner injuries of knee joint Samir Dwidmuthe
Missed posterolateral corner injuries of knee joint is a common cause for failure of ACL and PCL reconstruction only next to malpositioned tunnels.
Isolated PLC injuries are uncommon, making up <2% of all acute knee ligamentous injuries. Covey JBJS 2001
Incidence of PLC injuries associated with concomitant ACL and PCL disruptions are much more common (43% to 80%). Ranawat JAAOS 2008
A recent (MRI) analysis of surgical tibialplateau fractures demonstrated an incidence of PLC injuries in 68% of cases. Gardner JOT 2005
Take home message
PLC injuries to be ruled out in every case of ACL& PCL rupture.
Neurovascular integrity to be checked in every case.
Grade I & II can be managed conservatively.
Grade III Acute- Repair.
Grade III Chronic- Anatomic PLC recon.
Beware of varus knee alignment.
This video explains Lumbar Disc Replacement in Detail. When degenerative disc disease begins to affect the spine this is called degenerative disc disease. This video highlights the history, epidemiology, and treatment options both conservative and surgical. If you or someone you know needs to be seen in regards to Lumbar Disc Replacement feel free to look us up online www.beverlyspine.com or www.santamonicaspine.com OR call toll free 1-8SPINECAL-1
Posterolateral corner injuries of knee joint Samir Dwidmuthe
Missed posterolateral corner injuries of knee joint is a common cause for failure of ACL and PCL reconstruction only next to malpositioned tunnels.
Isolated PLC injuries are uncommon, making up <2% of all acute knee ligamentous injuries. Covey JBJS 2001
Incidence of PLC injuries associated with concomitant ACL and PCL disruptions are much more common (43% to 80%). Ranawat JAAOS 2008
A recent (MRI) analysis of surgical tibialplateau fractures demonstrated an incidence of PLC injuries in 68% of cases. Gardner JOT 2005
Take home message
PLC injuries to be ruled out in every case of ACL& PCL rupture.
Neurovascular integrity to be checked in every case.
Grade I & II can be managed conservatively.
Grade III Acute- Repair.
Grade III Chronic- Anatomic PLC recon.
Beware of varus knee alignment.
This video explains Lumbar Disc Replacement in Detail. When degenerative disc disease begins to affect the spine this is called degenerative disc disease. This video highlights the history, epidemiology, and treatment options both conservative and surgical. If you or someone you know needs to be seen in regards to Lumbar Disc Replacement feel free to look us up online www.beverlyspine.com or www.santamonicaspine.com OR call toll free 1-8SPINECAL-1
JOINT DISLOCATION of hip knee and shoulder PART-2.pptxrammmramm000
JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder
JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip knee and shoulder JOINT DISLOCATION of hip kn
Principles of splints and casts in orthopaedics by Dr. D. P. SwamiDR. D. P. SWAMI
Principles of splints/slabs and casts in orthopaedics. historical perspective, technique of slab/cast application, indications/ contraindications, care of slab/cast
TRAUMATOLOGY OF LOWER LIMB WITH MECHANISM OF INJURY, CLASSIFICATION, RADIOLOGY, NON OPERATIVE/ OPERATIVE TREATMENT, POTENTIAL PROBLEMS AND PREVENTIVE MEASURES
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Anti ulcer drugs and their Advance pharmacology ||
Anti-ulcer drugs are medications used to prevent and treat ulcers in the stomach and upper part of the small intestine (duodenal ulcers). These ulcers are often caused by an imbalance between stomach acid and the mucosal lining, which protects the stomach lining.
||Scope: Overview of various classes of anti-ulcer drugs, their mechanisms of action, indications, side effects, and clinical considerations.
2. Outline
• History and introduction
• Pathoanatomy
• Risk factors
• Classification
• Open vs arthroscopic procedures
• Landmark procedures
• Rehabilitation protocol
DPS
3. Introduction
• Documented in Egyptian tombs
as early as 3000 BC, with reduction
maneuver resembling Kocher technique
• Hippocrates detailed the oldest
known reduction method (Hippocratic Method)
• Most common joint dislocation
• Most mobile joint in the human body
DPS
4. Introduction
• Most commonly dislocated joint
50 % of all dislocations
2 % incidence in general population
• Acute dislocation - emergency and demands urgent relocation.
• Failure to reduce within the first 24 hours risk that it will be
impossible to achieve a stable closed reduction#
#Hovelius L, Augustini BG, Fredin H, Johansson O, Norlin R, Thorling J. Primary anterior dislocation of the shoulder in young patients. A ten-year prospective
study. J Bone Joint Surg Am 1996; 78: 1677–84.
DPS
5. Introduction
• In the younger-age group, the risk of
recurrence correlates strongly to
the violence of the initial injury
the age of the patient at the time of presentation
return to contact or collision sports
hyper laxity
• 16–30- year-old group being at particularly
high risk
DPS
6. Introduction
• Hovelius et al. found that the risk of re-
dislocation varied inversely with the age at the
time of primary dislocation
• Simonet et al in 1984, described a similar
recurrence rate. Both age and athletic activity
were shown to be important to the risk of
recurrence.#
#Simonet WT, Cofield RH. Prognosis in anterior shoulder dislocation. Am J Sports Med 1984; 12: 19–24.
DPS
8. Functional anatomy
• Shoulder joint is a complex
anatomical and biomechanical
structure which functions in a manner
that several stabilizers play role in
a special harmony in different stages of motion
Stability of the shoulder is established
by the glenohumeral articulation, labrum,
glenohumeral ligaments, rotator cuff, and
• deltoid muscle
Contact surface of the humeral head with the glenoid is about 30%, which means
that the joint has a limited osseous constraint so that the primary stability is due to
other soft tissue components rather than the osseous contact
DPS
9. Stabilizers
• Glenohumeral Stability
• Static restraints
– glenohumeral ligaments (below)
– glenoid labrum (below)
– articular congruity and version
– negative intraarticular pressure
• if release head will sublux inferiorly
• Dynamic restraints
– rotator cuff muscles
• the primary biomechanical role of the rotator cuff is stabilizing the
glenohumeral joint by compressing the humeral head against the glenoid
– rotator interval
– biceps long head
– periscapular muscles
DPS
10. Gleno-humeral ligaments
• Superior gleno humeral ligament -
attaches to the glenoid rim near the
apex of the labrum conjoined with
the long head of the biceps . On the
humerus, it is attached to the
anterior aspect of the anatomical
neck .
• restraint to inferior, anterior and
posterior stress at 0 degrees of
abduction
• Tightening of the rotator interval
(which includes the superior
glenohumeral ligament) decreases
posterior and inferior translation;
external rotation also may be
decreased DPS
11. • Middle gleno humeral ligament
–
has wide attachment
extending from the superior
glenohumeral ligament along the
anterior margin of the glenoid
down as far as the junction of the
middle and inferior thirds of the
glenoid rim.
On the humerus, it also is
attached to the anterior aspect of
the anatomical neck.
limits external rotation
when the arm is in the lower and
middle ranges of abduction but
has little effect when the arm is in
90 degrees of abduction DPS
12. • Inferior glenohumeral ligament –
glenoid margin from the 2- to 3-o’clock positions
anteriorly to the 8- to 9-o’clock positions posteriorly
humeral attachment is below the level of the
horizontally oriented physis into the inferior aspect of the
anatomical and surgical neck .
anterosuperior edge of this ligament usually is quite
thickened. There is a less thick and distinct posterior part
and thin axillary recess which create hammock type model.
external rotation, the hammock slides anteriorly and
superiorly. The anterior band tightens, and the posterior
band fans out. With internal rotation, the opposite occurs
DPS
13. “Circle Stability Concept”
• “Circle Stability Concept”
• For a full dislocation to occur, both
sides of the capsule and ligaments must be damaged.
The capsule preventing the direction of location
would be considered the primary restraint and the
opposite side would be considered the secondary restraint.
DPS
14. Mechanism of Injury
– Anterior (Abduction, ER, Extension)95% of all dislocations
– Posterior (Adduction, IR, Flexion, Axial Load)4% of all dislocations
– Epilepsy (Seizures), Electric Shock
– If seizure, look for bilateral
– Inferior (Luxatio Erecta)0.5% of all dislocations
– Hyperabduction or Axial force on overhead arm
• Superior (Rare)
• Intrathoracic (Rare)
DPS
15. Mechanism of Injury
Violent external rotation in abduction levers the
head of the humerus out of the glenoid socket,
avulsing anterior bony and soft tissue structures
in the process (Bankart lesion) *
• posterior part of the humeral head exits
the joint, it often collides with the anterior rim
of the glenoid, creating a bony indentation
at the back of the humeral head ( Hill Sachs lesion).#
*Bankart ASB. The pathology and treatment of recurrent dislocations of the shoulder joint. Br J Surg 1938: 26: 23–9.
# Bost FC, Inman VC. The pathological changes in recurrent dislocation of the shoulder: a report of Bankart’s operative procedure. J Bone
Joint Surg Am
1942; 23: 596–613.
DPS
16. Historical previews
• During the 1930s, many workers pursued what
was believed to be the essential lesion in
recurrent shoulder dislocation
• In a landmark paper in 1938, the British surgeon
Bankart described the lesion that still bears his
name.
• Bankart’s ‘essential lesion’ is an avulsion of
labrum from the anterior inferior glenoid with an
associated tear in the Labrum.
DPS
17. • capsular laxity in the absence of a Bankart
lesion is also well recognised #
• biomechanical studies have demonstrated that
the creation of a Bankart lesion in itself is
insufficient to permit shoulder dislocation.
• More recent cadaveric, arthroscopic and MRI
studies have shown that many patients have
sustained injury to several structures in the
shoulder.
#Hintermann B, Gachter A. Arthroscopic findings after shoulder dislocation. J Sports Med 1995; 23: 545–51.on
DPS
18. Classification
• According to direction of instability –
unidirectional
bidirectional
multidirectional
• Degree of instability –
sublaxation
dislocation
• Duration of instability –
acute
sub acute
chronic > 6 weeks
DPS
19. • Type of trauma –
macro trauma
micro trauma
secondary trauma
• Age of initial dislocation –
< 20 year - 90% recurrence
20 – 40 year
> 40 year - 10% recurrence
DPS
20. • Matsen’s simplified classification system -
1 - TUBS (Traumatic Unidirectional Bankart Surgery )
2 - AMBRII (Atraumatic, Multidirectional, Bilateral,
Rehabilitation, Inferior capsular shift, and Internal closure)
Micro traumatic or developmental lesions fall between the
extremes of macro traumatic and atraumatic lesions and
can overlap these extreme lesions
DPS
21. Clinico radiological evaluation
• A detailed history and a careful physical examination
of the patient are the primary steps of the clinical
assessment.
Mechanism of the first incident
time period from the first dislocation to recurrent
instability
activities leading to recurrence or apprehension
number of dislocations
history of reducibility without emergency visit
DPS
22. Clinico radiological evaluation
• Apprehension and relocation tests as provocative
examination are the fundamentals of clinical evaluation
• Anterior apprehension test is performed with the shoulder in 90 degrees of abduction and the elbow in 90 degrees of flexion, with forced external
rota- tion applied to the extremity as anterior stress is applied to the humerus. Relocation test is performed while the patient is supine and the shoulder in 90
degrees of abduction and external rotation.
DPS
23. Clinico radiological evaluation
• Anteroposterior, axillary lateral and scapular Y-
view images are the primary routine radiographic
evaluation along with West point axillary view
(glenoid rim fracture) or Stryker notch view (Hill-
Sachs lesion)
• 3D CT is gold-standard technique to detect
osseous pathologies as well as quantifying the
degree of bone loss
• Magnetic resonance imaging (MRI) is a very
useful tool in detecting soft tissue pathologies
DPS
24. Who is at risk
• young age
• participation in high demand contact sports activities
• previous history of ipsilateral traumatic dislocation
• presence of
Hill-Sachs
osseous Bankart lesion
ipsilateral rotator cuff
deltoid muscle insufficiency
underlying ligamentous laxity
DPS
25. Treatment
• common surgical interventions address the labral tears as
well as the capsular laxity which are generally the basic
underlying pathologies.
• Surgical repair of any accompanying rotator cuff tear should
also be included in the treatment process
• Although many different surgical techniques have been
described to treat traumatic recurrent anterior instability of
the shoulder, the best method still remains controversial.
• A successful clinical outcome basically requires an accurate
surgical technique applied via adequate exposure.
DPS
27. • The main objective of the treatment should be
considered as the most anatomical repair of the well
defined pathological condition leading to recurrent
instability.
• Achieving the best result for any particular patient
depends on the procedure which allows :
observation of the joint surfaces
provides the anatomical repair
maintains range of motion
with low rates of complications and recurrence
• Open and arthroscopic procedures are treatment options
DPS
32. History of anterior shoulder
stabilization surgery
• Split and shift
• Multi-pleated capsular plication
• Posteroinferior capsular plication
• Rotator interval closure
• Arthroscopic Latarjet
• Targeted management of Hill-Sachs lesions
• Humeral head or femoral head allograft
• Disimpaction
• Partial resurfacing arthroplasty
• Hemiarthroplasty
• Arthroscopic remplissage
DPS
33. Which one is superior
• Although open stabilization was reported as more effective
than arthroscopic stabilization in the aspect of post-operative
recurrence rates in 1990s, clinical outcomes have become
similar in time.
• Technological improvements in arthroscopic
instrumentation as well as the development of the innovative
surgical techniques as a result of the cumulative experience
with improved understanding of the factors leading failure in
such patients have played the key role#
# Burkhart SS, De Beer JF. Traumatic glenohumeral bone defects and their relationship to failure of arthroscopic
Bankart repairs: significance of the inverted-pear glenoid and the humeral engaging Hill-Sachs lesion. Arthroscopy
2000; 16: 677-694 [PMID: 11027751]
Porcellini G, Campi F, Pegreffi F, Castagna A, Paladini P. Predisposing factors for recurrent shoulder dislocation
after arthroscopic treatment. J Bone Joint Surg Am 2009; 91: 2537-2542 [PMID: 19884424 DOI:
10.2106/JBJS.H.01126]
DPS
34. • According to the results of prospective randomized clinical trial
comparing open and arthroscopic techniques, the difference in
quality of life between the patients in the two groups was neither
significant nor clinically important at two years follow-up; however
significantly lower risk of recurrence was obtained in patients for
whom open repair was preferred@
• Rhee et al compared the results of arthroscopic and open
stabilization in young contact athletes and reported recurrent
instability as 25% in the arthroscopic group and 13% in the open
stabilization group *
• Some authors mentioned that athletic activity plays a greater role in
postoperative recurrence than the surgical method used for
stabilization#
@Mohtadi NG, Chan DS, Hollinshead RM, Boorman RS, Hiemstra LA, Lo IK, Hannaford HN, Fredine J, SasyniukTM, Paolucci EO. A randomized
clinical trial comparing open and arthroscopic stabilization for recurrent traumatic anterior shoulder instability: two-year follow-up with
disease-specific quality-of-life outcomes. J Bone Joint Surg Am 2014; 96: 353-360 [PMID: 24599195 DOI: 10.2106/JBJS.L.01656]
* Rhee YG, Ha JH, Cho NS. Anterior shoulder stabilization in collision athletes: arthroscopic versus open Bankart repair. Am J Sports Med
2006; 34: 979-985 [PMID: 16436537]
# Cole BJ, L’Insalata J, Irrgang J, Warner JJ. Comparison of arthroscopic and open anterior shoulder stabilization. A two to six-year follow-
up study. J Bone Joint Surg Am 2000; 82-A:
1108-1114 [PMID: 10954100]
DPS
35. Open Surgical Techniques
• Two basic types of surgical approaches :
Anatomic repairs the goal is to restore the labrum to its
normal position and to reestablish the appropriate tension in
the shoulder capsule and ligaments
• Depending on the pathoanatomy
the classic Bankart procedure that was popularized by
Rowe
the capsular shift procedure which was popularized by
Neer
DPS
36. • Non-anatomic repairs :
The goal is to stabilize the shoulder by compensating for the capsulolabral and
osseous injury with an osseous or soft-tissue checkrein that blocks excessive translation
and restores stability.
The Putti-Platt procedure, which is an imbrication and shortening of the subscapularis
demonstrated excellent outcomes with non-anatomic stabilizations, but the reported
complications, such as loss of motion, recurrent instability, and premature arthritis#
The Magnuson-Stack procedure, which is an advancement of the subscapularis that
was popularized by De-Palma
The Bristow procedure
The Latarjet procedure which are transfers of the coracoid to the glenoid
• #Fredriksson AS, Tegner Y. Results of the Putti-Platt operation for recurrent anterior dislocation of the shoulder.
Int Orthop. 1991;15:185-8.
• Young DC, Rockwood CA Jr. Complications of a failed Bristow procedure and their management. J Bone Joint Surg
Am. 1991;73:969-81.
DPS
37. Patient selection
• an apprehension sign that is relieved by a relocation maneuver can be
virtually diagnostic of anterior shoulder instability and a Bankart lesion#.
• The anteroposterior laxity of the shoulder should be assessed with load
and shift testing, and the inferior laxity should be assessed with inferior
translation (sulcus testing).
• A large sulcus sign that recreates symptoms of instability is
Pathognomonic for multidirectional instability.
• a large sulcus sign in the adducted arm that does not decrease when the
arm is placed in external rotation indicates an insufficiency of the rotator
interval*
• #Speer KP, Hannafin JA, Altchek DW, Warren RF. An evaluation of the shoulder relocation test. Am J Sports Med.
1994;22:177-83.
• *Neer CS 2nd. Involuntary inferior and multidirectional instability of the shoulder:etiology, recognition, and treatment. Instr
Course Lect. 1985;34:232-8.
DPS
38. Indications
• Absolute indications :
substantial glenoid or humeral bone loss
capsular deficiency
irreparable rotator cuff deficiency
humeral avulsions of the glenohumeral ligaments and
capsular ruptures as these two injuries are extremely
difficult to address arthroscopically
• a previous failed arthroscopic or open repair because it
is easier to address the causes of the instability (which
may be multiple) with an open procedure
a prior failed thermal capsulorrhaphy
DPS
39. Contra indications
• Absolute contraindications: voluntary or
psychogenic instability and active infection.
• patients with concomitant severe arthritis.
• Paralysis
DPS
40. Open bankart repair
• Classically, the subscapularis tendon
is incised vertically at its lateral
insertion and sharply dissected
medially from the anterior capsule
• Make a vertical capsulotomy
approximately 0.5 cm lateral to the
glenoid
DPS
41. Open bankart repair…
Prepare this area with a curet to expose
bleeding bone and drill three holes; one at the
2-o' clock, one at the 4-o' clock, and one at the
6-o' clock positions for right shoulders (10-, 8-,
and 6-o' clock positions for left shoulders
Pass sutures through the holes and the lateral
capsular flap
Tie the flap down to the glenoid rim, and pass
these same sutures through the small medial
capsular flap, reinforcing the repair
DPS
42. Coracoid transfer procedures
• Latarjet 1958
• Later on popularized and modified
by Helfet, who named it for
his mentor Rowley Bristow.
• The aim of these procedures is
to stabilize the shoulder with the
static action of the transferred bone
block and the attached
coracobrachialis tendon
Only the tip of the coracoid process is transferred
in the Bristow procedure, whereas, in the Latarjet procedure,
the transfer includes a portion of the coracoacromial
osteotomy with the conjoined tendon left attached
transferred to the anterior glenoid, and fixed
DPS
43. Coracoid transfer procedures :Pit falls
• May fail to address :
essential lesion (i.e., theBankart lesion)
associated pathology (SLAP lesion)
• Recurrence rates have ranged from 0% (Allain et
al) to 6% (Hovelius et al.)
Loss of ROM: greater than that after an open
Bankart procedure
DPS
44. Glenoid Reconstruction with Iliac
Crest Bone Graft
• Bodey and Denham: first report 1983
• Glenoid grafting restores bone to recreate the
arc of the glenoid
DPS
45. Humeral Bone Deficiency
• Hill-Sachs lesions
• “engaging Hill-Sachs lesions.”-Burkhart and De
Beer described
• long axis of the humeral head defect aligns
parallel to the anterior glenoid rim, when the
shoulder is in a position of abduction and external rotation.
DPS
46. Humeral Bone Deficiency
• Surgical options :
Reconstruction of the humerus with an allograft
Restore the humeral articular arc
reconstruction of the glenoid with an anterior bone graft to lengthen
the glenoid articular arc and prevent the humeral defect from engaging
the glenoid rim
Rotation of the humeral head with an osteotomy to move the defect so
that it does not come into contact with the anterior aspect of the
glenoid
Burkhart S, Danaceau S. Articular arc length mismatch as a cause of failed Bankart repair. Arthroscopy. 2000;16:740-4.
Yagishita K, Thomas BJ. Use of allograft for large Hill-Sachs lesion associated with anterior glenohumeral dislocation. A case report.
Injury. 2002;33:791-4.
Weber BG, Simpson LA, Hardegger F. Rotational humeral osteotomy for recurrent anterior dislocation of the shoulder associated with a
large Hill-Sachs lesion. J Bone Joint Surg Am. 1984;66:1443-50.
DPS
47. Capsular Deficiency
• Capsular deficiency is more common in revision settings and after thermal capsulorrhaphy.
• Lazarus and Harryman described a method of using hamstring tendons for repair of such
deficiencies*
• . The long head of the biceps can be combined with the autograft for additional support.
• Gallie and Le Mesurier described the use of the iliotibial band for capsular reconstruction to treat
glenohumeral instability associated with an irreparable capsule#
• Moeckel et al. described the use of Achilles tendon allograft in ten patients who had persistent
anterior instability
• *Lazarus MD, Harryman DT 2nd. Open repair for anterior instability. In: Warner JJP, Iannotti JP, Gerber C, editors. Complex
and revision problems in shoulder surgery. Philadelphia: Lippincott-Raven; 1997. p 47-64.
• #Gallie WE, Le Mesurier AB. Recurring dislocation of the shoulder. J Bone Joint Surg Br. 1948;30:9-18.
DPS
48. Revision and Complex Problems
surgeon should be prepared to face:
distorted anatomic tissue planes
severe scarring
capsular deficiencies
osseous deficiencies due to erosion or
fracture, and subscapularis deficiencies
DPS
49. Open repairs: Complications and
Pitfalls
• Recurrence of Instability
• Stiffness
• Subscapularis Deficiency
• Arthrosis
• Hardware Problems
• Neurovascular Injuries
DPS
51. Arthroscopic Stapling
• In 1982, Detrisac and Johnson performed the first
arthroscopic shoulder stabilization procedure, using a
capsular stapling technique.*
• Abandoned because of hardware problems and an inability to
address capsular laxity.
• Lane and colleagues retrospectively reported 33% recurrence rate,
with 18.5% requiring a subsequent open reconstructive procedure.
Fifteen percent developed loose staples on follow-up radiographs#
• *Detrisac DA, Johnson LL: Arthroscopic shoulder capsulorraphy using metal staples. Orthop Clin North Am24(1):71-88, 1993.
• # Lane JG, Sachs RA, Riehl B: Arthroscopic staple capsulorraphy: A long-term follow-up. Arthroscopy 9(2):190-194, 1993.
DPS
52. Transglenoid Suture Technique
Morgan and associates first described the transglenoid suture technique in
1987*
• Failures were attributed to #
• plastic deformation in the capsular tissue
• component of the instability still existed. Seventy-
• immobilization periods of less than one week
• Caspari, in 1988, described a technique that allowed the surgeon to
advance and adjust tension in the capsuloligamentous structures$
• Caspari technique have experienced similar recurrence rates
• *Morgan CD, Bodenstab AB: Arthroscopic Bankart suture repair: Technique and early results. Arthroscopy 3:111-122, 1987.
• #Grana WA, Buckley PD, Yates CK: Arthroscopic Bankart suture repair. Am J Sports Med 21(3):348-353, 1993.
• $Green MR, Christensen KP: Arthroscopic Bankart procedure:Two- to five-year follow-up with clinical correlationto severity of glenoid labral lesion. Am J Sports Med
• 23(3):276-281, 1995.
DPS
53. Suture Anchors
• Weber and associates
• modified by both Wolf and Snyder who used
absorbable and non-absorbable sutures,
respectively.
• This technique has the advantage of allowing the
capsuloligamentous structures to be shifted
superiorly and be properly tensioned.
• Complications:
• intra-articular migration of a suture anchor
• articular damage
DPS
55. Arthroscopic Latarjet Procedure
• First Stage: Achieving Exposure
• Second Stage: Coracoid Preparation
• Third Stage: Coracoid Drilling and Osteotomy
• Fourth Stage: Coracoid Transfer
• Fifth Stage: Fixation of Bone Graft
DPS
56. Arthroscopic Latarjet Procedure
• Advantageous in those cases in which the preoperative
assessment fails to reveal an HAGL lesion or a large
bony avulsion from the anterior rim
• Allows surgeon to modify his or her plan
intraoperatively
• With regard to graft placement and fixation: provides
superior visualization for positioning the coracoid
DPS
58. ARTHROSCOPIC BANKART REPAIR
• Place the arm in 45 degrees abduction and 20 degrees
• forward flexion using 10 to 12 lb of traction.
• Place the posterior portal 2 cm inferior to the
• posterolateral edge of the acromion
• Thoroughly evaluate the glenohumeral joint for bony loss
• After identifying the quadrant or quadrants of injury
• to the labrum, create the planned portals shoulder
• just posterior to the biceps tendon and anterior to the
• leading edge of the supraspinatus tendon
DPS
59. Posterior Instability
– Indications:
– Failed non operative
– Irreducible dislocation
– Open dislocation
– Unstable reduction
– Surgical Options:
– Arthroscopic
– Open Anterior Procedure
– Open Posterior Procedure
DPS
60. Posterior Instability
– Arthroscopic
Capsular repair/capsulorrhaphy, Labral repair
86-96% success, 0-7% recurrent instability
• Open Anterior Procedure
Deltopectoral approach
Capsular release and transfer to remove
redundancy + imbrication
• RCT repair
– McLaughlin Procedure
– Neer Modification (reverse Hill-Sachs repair)
Transfer of subscap tendon into lesion
DPS
Anterior dislocations account for about 95% of recurrent dislocations, and posterior dislocations account for approximately 5%. Despite increased understanding of shoulder instability, 50% of posterior shoulder dislocations can be missed unless an adequate examination and appropriate radiographs are done. Inferior and superior dislocations are rare. Superior instability generally arises secondary to severe rotator cuff insufficiency.
1 - Secondary trauma to the rotator cuff and biceps tendon may cause asynchronous rotator cuff function. These injuries most commonly occur in pitchers, batters, gymnasts, weightlifters, tennis players and others who play racquet sports, and swimmers, especially with the backstroke or butterfly stroke.
2 - These differences can be explained by the greater elasticity in adolescent ligaments that results in greater plastic deformation before failure of the system. This deformation must be considered in surgical treatment approaches.
3 – but in older pt more a/w rotator cuff injury ( > 40 yr is 3o%, > 60 yr 80% ), # of GT 42 % )