Pharyngitis is inflammation of the pharynx or back of the throat. It is commonly known as a sore throat and is usually caused by viruses. The most common type is acute pharyngitis caused by adenovirus, coronavirus or streptococcus bacteria. Chronic pharyngitis is a persistent inflammation that can be caused by irritants like dust, smoke or excessive voice use. Symptoms include pain on swallowing, fever and redness of the throat. Diagnosis involves examination of the throat and testing of secretions. Acute cases usually resolve within a week with pain relief and fluids, while antibiotics may be used for bacterial infections. Chronic pharyngitis management focuses on removing irritants and relieving symptoms
Pharyngitis- Easy PPT for Nursing StudentsSwatilekha Das
Pharyngitis- Easy PPT for Nursing Students
organs of the respiratory system
Definition
Acute Pharyngitis definition
Causes
Clinical features
Diagnosis
Treatment
Nutritional therapy
Chronic Pharyngitis
types
Clinical manifestations
Medical management
Tonsillitis slideshare for medical students NehaNupur8
complete and detail information about tonsillits , that is the inflammation of the tonsils ,present in the oral cavity , disease of oral cavity contains introduction, definition, types, causes, risk factors,pathophysiology , treatment , medical management, nursing management, nurses role, patient teaching sign and symptoms , drug therapy, diet management,
Tonsillitis is inflammation of the tonsils, two oval-shaped pads of tissue at the back of the throat — one tonsil on each side. Signs and symptoms of tonsillitis include swollen tonsils, sore throat, difficulty swallowing and tender lymph nodes on the sides of the neck
Pharyngitis- Easy PPT for Nursing StudentsSwatilekha Das
Pharyngitis- Easy PPT for Nursing Students
organs of the respiratory system
Definition
Acute Pharyngitis definition
Causes
Clinical features
Diagnosis
Treatment
Nutritional therapy
Chronic Pharyngitis
types
Clinical manifestations
Medical management
Tonsillitis slideshare for medical students NehaNupur8
complete and detail information about tonsillits , that is the inflammation of the tonsils ,present in the oral cavity , disease of oral cavity contains introduction, definition, types, causes, risk factors,pathophysiology , treatment , medical management, nursing management, nurses role, patient teaching sign and symptoms , drug therapy, diet management,
Tonsillitis is inflammation of the tonsils, two oval-shaped pads of tissue at the back of the throat — one tonsil on each side. Signs and symptoms of tonsillitis include swollen tonsils, sore throat, difficulty swallowing and tender lymph nodes on the sides of the neck
Otitis media is a group of inflammatory diseases of the middle ear. The two main types are acute otitis media (AOM) and otitis media with effusion (OME). AOM is an infection of rapid onset that usually presents with ear pain.
Nosebleeds are very common in young children, affecting most at some time or another. From the outset, it is important to be aware that nosebleeds will often settle down on their own, sometimes requiring medical treatment, but that major underlying causes (blood clotting problems or abnormalities in the nose) are very rare.
Rhinitis, also known as coryza, is irritation and inflammation of the mucous membrane inside the nose. Common symptoms are a stuffy nose, runny nose, sneezing, and post-nasal drip. The inflammation is caused by viruses, bacteria, irritants or allergens
Otitis media is a group of inflammatory diseases of the middle ear. The two main types are acute otitis media (AOM) and otitis media with effusion (OME). AOM is an infection of rapid onset that usually presents with ear pain.
Nosebleeds are very common in young children, affecting most at some time or another. From the outset, it is important to be aware that nosebleeds will often settle down on their own, sometimes requiring medical treatment, but that major underlying causes (blood clotting problems or abnormalities in the nose) are very rare.
Rhinitis, also known as coryza, is irritation and inflammation of the mucous membrane inside the nose. Common symptoms are a stuffy nose, runny nose, sneezing, and post-nasal drip. The inflammation is caused by viruses, bacteria, irritants or allergens
Nasal discharge, also known as rhinorrhea, is a common symptom that can be caused by a variety of conditions related to the ear, nose, and throat (ENT). It is the result of excess mucus production in the nasal cavity, which can be caused by inflammation or infection of the nasal passages.
Common causes of nasal discharge include allergies, colds, sinus infections, and nasal polyps. Allergies can cause the nasal passages to become inflamed and produce excess mucus, leading to a runny nose. Colds and sinus infections can also cause inflammation and infection, leading to nasal discharge.
Nasal polyps are growths in the nasal cavity that can obstruct airflow and cause chronic inflammation and excess mucus production. Other less common causes of nasal discharge include foreign bodies in the nasal cavity, tumors, and hormonal changes during pregnancy.
Treatment for nasal discharge depends on the underlying cause. For allergies, antihistamines and nasal corticosteroids may be recommended. For colds and sinus infections, decongestants, saline nasal sprays, and antibiotics may be used. Nasal polyps may require surgical removal.
In addition to nasal discharge, other symptoms that may be present with ENT-related conditions include nasal congestion, headache, facial pain or pressure, cough, and sore throat. If nasal discharge is persistent, accompanied by other symptoms, or affects quality of life, it is important to seek medical evaluation by an ENT specialist.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
1. PHARYNGITIS
It is the inflammation of the pharynx which is the back of the throat. It is also
referred to as “Sore throat”. It is a type of upper respiratory tract infection. It is one
of the most common reason to doctorvisit.
CLASSIFICATION
It is of 2 types: Acute Pharyngitis and chronic Pharyngitis.
ACUTE PHARYNGITIS
It is a sudden, painful inflammation of the throat.
ETIOLOGY
1. Virus: Most common cause (90%) of sore throat. Adenovirus, coronavirus,
Homophiles influenza, Mononucleosis. They do not respond to antibiotic
therapy.
2. Bacterial: Less common cause of sore throat. Strep throat is a common
bacterial infection of pharynx caused by Group A beta hemolytic
streptococci(GAS). Other bacteria are mycoplasma Pneumoniae, Neisseria
meningitis etc.
RISK FACTORS
1. Frequent exposure to cold and flu.
2. Allergies
3. Frequent sinusitis
4. GERD
5. Exposure to secondhand smoke.
PATHOPHYSIOLOGY
2. CLINICAL MENIFESTATIONS
1. Fiery red pharyngeal membrane and tonsils.
2. Fever(101 F)
3. Malaise
4. Sore throat pain ( In case of GAS bacteria)
5. Swollen , erythomatous tonsils
6. Halitosis.
DIAGNOSTIC EVALUATION
1. History taking
2. Physical examination
3. CBC
4. Throat swab culture.
5. Rapid streptococcalantigen test.
MEDICAL MANAGEMENT
Uncomplicated viral Pharyngitis infections subside promptly within 3 to 10
days after onset. Viral Pharyngitis is treated with supportive measures because
antibiotics have no effects on the causative agents.
A. PHARMACOLOGICAL THERAPY: In case of bacterial
Pharyngitis Penicillin is the treatment of choice and given for 5 days. Patient
who are allergic to penicillin and erythromycin, they are treated with
Cephalosporin and macrolides. Severe sore throat can be relieved by
analgesics such as Aspirin, Acetaminophen. If required acetaminophen with
codeine can be taken three to four times daily. Some patients find slat-water
gargling to be soothing. In severe cases, Gargles with benzocaine may
relieve symptoms.
B. Nutritional therapy: A liquid or soft diet is provided during the acute
stage of the disease. Sometimes throat is so sore that liquid can not be taken
in adequate amount. In severe situations IV fluids may be needed. Otherwise
patient is encouraged to drink as much fluid as possible.
4. CHRONIC PHARYNGITIS
Chronic Pharyngitis is a persistent inflammation of the pharynx. It is
common in the adults who work in dusty surroundings, use their voice to
excess, suffer from chronic cough or habitually use alcohol and tobacco.
CLASSIFICATION
1. Hypertrophic: It is characterized by thickening and congestion of the
pharyngeal mucous membrane.
2. Atrophic: Late stage . The membrane is thin, whitish, glistening.
3. Chronic granular: Numerous swollen lymph nodes on the pharyngeal
wall.
CLINICAL MENIFESTATIONS
1. Sense of irritation and fullness in the throat.
2. Mucus buildup which is expelled out by coughing.
3. Nasal congestion
4. Dysphagia.
MANAGEMENT
Nasal congestion is relieved by short term use of nasal sprays or
medications containing ephedrine sulphate.
For a patient with a history of allergy , antihistamine decongestants
such as Pseudoephedrine is prescribed orally every 4 -6 hours.
Aspirin or acetaminophen is recommended .
For adults with chronic Pharyngitis, tonsillectomy is an effective
option.
NURSING MANAGEMNT
To prevent the infection from spreading, the nurse instructs the
patient to avoid contactwith others until the fever subsides.
The nurse recommends avoidance of alcohol, tobacco,
secondhand smoke, and exposure to cold environment and
occupational pollutants.
The nurse encourage the patient to have plenty of drink or fluids.
5. Gargling with warm saline solution may relieve throat discomfort
. Lozenges keep the throat moistened.