This document discusses penetrating neck trauma. It begins by outlining the anatomy of the neck and dividing it into three zones. It then discusses the mechanisms of injury, signs indicating injury to structures like blood vessels, and considerations for resuscitation and investigation. Hard signs that require emergency surgery include uncontrolled bleeding or shock. Soft signs may allow for further investigation with imaging or endoscopy before deciding on exploration. Surgical management depends on the injured zone, and may involve sternotomy, collar incisions, or mandible resection.
Splenic trauma - Causes, Complications, ManagementVikas V
Splenic Trauma - A detailed Presentation about Splenic Trauma, anatomy of the spleen, Causes of Trauma, Mechanism of Injury, Diagnosis, Management, Surgical management, Steps of Splenectomy, and Complications
Splenic trauma - Causes, Complications, ManagementVikas V
Splenic Trauma - A detailed Presentation about Splenic Trauma, anatomy of the spleen, Causes of Trauma, Mechanism of Injury, Diagnosis, Management, Surgical management, Steps of Splenectomy, and Complications
ATLS is two days course for those who manage trauma patients. These protocols have been followed by hospitals all over the world to treat trauma patients quickly and efficiently.
ATLS is two days course for those who manage trauma patients. These protocols have been followed by hospitals all over the world to treat trauma patients quickly and efficiently.
CHEST INJURY- BLUNT/ Trauma Surgery
Dear viewers,
Greetings from “Surgical Educator”
Today I have uploaded a video on CHEST INJURY- BLUNT- an important topic in trauma. Even the blunt chest trauma can turn into penetrating one because of jagged edges of the broken ribs. I haven’t talked elaborately but have included the essential minimum an undergraduate medical student should know. I have talked about pathophysiology, clinical approach, symptoms, signs, investigations, different individual types of Chest injuries and management of all the varieties of Chest injuries. My aim is, after watching this video all of you should be able to arrive at a correct working diagnosis of the type of chest injury and should also be able to institute immediate lifesaving treatment to the patients if there is a need. You can watch the video in the following links:
surgicaleducator.blogspot.com
youtube.com/c/surgicaleducator
Thank you for watching the video.
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Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
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These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
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2 Case Reports of Gastric Ultrasound
3. Introduction
❖ Penetrating neck injuries vs
blunt trauma
❖ Injury through the platysma
muscle
❖ Mechanisms of injury
http://www.innerbody.com/image_musfov/musc18-new.html
https://academic.amc.edu/martino/grossanatomy/site/medical/lab%20manual/gastrointestinal/Dissections/Anterior%20Triangle/Anterior%20Triangle1.htm
4. Ballistic injuries
❖ Kinetic energy = mass x
velocity squared / 2
❖ Greater tissue disruption from
high mass or velocity
projectiles
❖ Yaw
❖ Deformation/Fragmentation
5. Historically
❖ Carotid ligation vs observation
❖ High mortality associated with injury
❖ World War II - exploratory surgery in all patients
❖ High negative exploratory rates - zone based approach
❖ All zone 2 injuries explored, zone 1 + 3 explored based
of symptoms due to complications of the surgeries
6. Anatomical approach
❖ Zone 1: Clavicle to the cricoid
cartilage
❖ Zone 2: Cricoid cartilage to
angle of mandible
❖ Zone 3: Above angle of the
mandible
❖ Site of external wound vs
where projectile passes
7. Structures at risk
❖ Vascular - carotid arteries, vertebral arteries, jugular veins
❖ Aerodigestive - larynx/pharynx/trachea, oesophagus
❖ Neurological - Spinal cord
❖ Zone 1 can extend into thorax and include thyroid gland
❖ Zone 2 includes vagus and recurrent laryngeal
❖ Zone 3 includes cranial nerves IX, X, XI and XII
8. Hard and Soft Signs
HARD SIGNS SOFT SIGNS
Uncontrollable haemorrhage/Shock Haemorrhage responding to fluid resuscitation
Expanding or pulsatile haematoma Non pulsatile or expanding haematoma
Absent radial pulse Subcutaneous emphysema
Thrills/Bruits Dysphonia/Dysphagia
Neurological deficit indicating ischaemia
Haemoptysis/Haematemesis
Respiratory distress/Stridor
Visible air bubbles from wound
9. Resuscitation considerations
❖ Catastrophic bleeding
❖ C Spine - consider the mechanism, neurological findings
❖ Immobilised C Spine may make further assessment and
observation of penetrating neck injuries difficult
❖ Ramasamy et al 2009 - Review of British soldiers from Iraq war
- 56 surviving to surgery, 1 patient unstable C Spine
❖ C Spine fracture from gunshot wound vs stab wound was
1.35% and 0.12% retrospectively: study quoted UpTo date
Rhee et al
10. Resuscitation considerations
❖ Airway
❖ RSI vs surgical airway
❖ Visible trachae - secure to
prevent retraction into thorax
❖ Fiberoptic guided intubation
❖ Specific risks - false lumen out
of trachea, exacerbation of
injury
http://clinicalgate.com/penetrating-neck-trauma/
12. Investigation
❖ Bloods and crossmatch
❖ CXR
❖ Ultrasound
❖ CT with angiography/thin slice helical
❖ Direct visualisation - laryngoscopy/oesophagoscopy
13. Management
❖ Early surgical consultation
❖ If hard signs for theatre for exploration
❖ Soft signs - investigate - theatre/observe
❖ Possible surgical approaches:
❖ Zone 1 - median sternotomy
❖ Zone 2 - transverse cervical collar incision
❖ Zone 3 - dislocation/resection of mandible
❖ Endovascular approach
14. References
❖ Tintinalli’s Emergency Medicine 8th Edition
❖ UpTo Date Penetrating Neck Injuries: Initial evaluation
and management Feb 2017; Kim Newton
❖ Ramasamy et al. Learning lessons from conflict: Pre-
hospital cervical spine immobilisation following ballistic
neck trauma. Injury. 2009