The document describes various benign and malignant pathologies of the gallbladder and biliary tract that can be identified on imaging. It discusses conditions such as gangrenous cholecystitis, porcelain gallbladder, gallbladder perforation, adenomyomatosis, gallbladder polyps, and gallbladder carcinoma. It also reviews benign tumors of the biliary tract such as adenomas. Malignant neoplasms covered include intrahepatic and hilar cholangiocarcinomas, as well as distal ductal cholangiocarcinomas. Imaging findings for differentiating these pathologies are presented.
Choledochal cysts should be considered in the differential diagnosis in all patients with a history of biliary colic, recurrent cholangitis or pancreatitis with associated dilatation of bile duct, particularly if they are <40 years of age. Delay in the diagnosis increases the incidence of associated biliary pathology and suboptimal surgical therapy
Choledochal cysts should be considered in the differential diagnosis in all patients with a history of biliary colic, recurrent cholangitis or pancreatitis with associated dilatation of bile duct, particularly if they are <40 years of age. Delay in the diagnosis increases the incidence of associated biliary pathology and suboptimal surgical therapy
PowerPoint presentation on Choledochal Cysts, also known as biliary cyst, uploaded by Dr. Vaskar Humagain, first presented in 31st December, 2013. This presentation contains all the information about Choledochal Cysts, the original and revised Todani classification of choledochal cysts, pathogenesis, other associated congenital anomalies, clinical features in infant and adult, management of choledochal cysts. Comments are highly welcome :)
ACUTE CHOLECYSTITIS- RUQ ABDOMINAL PAIN
#surgicaleducator #ruqabdominalpain #acutecholecystitis #usmle #babysurgeon #surgicaltutor
• Dear Viewers,
• Greetings from “Surgical Educator”
• Today I have uploaded a video on Acute Cholecystitis
• It is one of the common surgical problems you see in surgical wards.
• I have discussed the various causes for RUQ pain, etiology, pathology, clinical features, investigations, complications and treatment of Acute Cholecystitis.
• I have also included a mind map, a diagnostic and a treatment algorithm for Acute Appendicitis.
• I hope the video will be very useful and you will enjoy it.
• You can watch all my surgical teaching videos in the following links:
• surgicaleducator.blogspot.com youtube.com/c/surgicaleducator
• Thank you for watching the video.
Pancreatitis -a detailed study ( medical information )martinshaji
Pancreatitis is the Inflammation of the pancreatic parenchyma. Acute condition of diffuse pancreatic inflammation & auto digestion, presents with abdominal pain, and is usually associated with raised pancreatic enzyme levels in the blood &urine. this is a detailed study pancreatitis describing factors such as definition , epidemiology , etiology , pathophysiology , treatment , prevention , imaging techniques , diagnosis , lab investigations , images , drugs , control etc
please comment
thank u
PowerPoint presentation on Choledochal Cysts, also known as biliary cyst, uploaded by Dr. Vaskar Humagain, first presented in 31st December, 2013. This presentation contains all the information about Choledochal Cysts, the original and revised Todani classification of choledochal cysts, pathogenesis, other associated congenital anomalies, clinical features in infant and adult, management of choledochal cysts. Comments are highly welcome :)
ACUTE CHOLECYSTITIS- RUQ ABDOMINAL PAIN
#surgicaleducator #ruqabdominalpain #acutecholecystitis #usmle #babysurgeon #surgicaltutor
• Dear Viewers,
• Greetings from “Surgical Educator”
• Today I have uploaded a video on Acute Cholecystitis
• It is one of the common surgical problems you see in surgical wards.
• I have discussed the various causes for RUQ pain, etiology, pathology, clinical features, investigations, complications and treatment of Acute Cholecystitis.
• I have also included a mind map, a diagnostic and a treatment algorithm for Acute Appendicitis.
• I hope the video will be very useful and you will enjoy it.
• You can watch all my surgical teaching videos in the following links:
• surgicaleducator.blogspot.com youtube.com/c/surgicaleducator
• Thank you for watching the video.
Pancreatitis -a detailed study ( medical information )martinshaji
Pancreatitis is the Inflammation of the pancreatic parenchyma. Acute condition of diffuse pancreatic inflammation & auto digestion, presents with abdominal pain, and is usually associated with raised pancreatic enzyme levels in the blood &urine. this is a detailed study pancreatitis describing factors such as definition , epidemiology , etiology , pathophysiology , treatment , prevention , imaging techniques , diagnosis , lab investigations , images , drugs , control etc
please comment
thank u
Concentrate on HNI
Countries which charge lesser import duties, tariff & have relaxed import norms.
Countries with high growth rate.
Countries which dedicated biking clubs & communities & which have a biking culture.
Body MR imaging in the emergenecy setting. Focus on application where MRI provides diagnostic information for patient management in the emergency department, without radiation exposure.
Detailed Seminar on Carcinoma Pancreas with -
Anatomy, Epidemiology, Enteropathogenesis, Pathology, Staging , Diagnostic workup and different modalities of Treatment
Abstract
Hepatic angiosarcoma is a rare tumour that is often difficult to diagnose. Historically, most cases of hepatic angiosarcoma were seen in the setting of industrial epidemics caused by exposure of workers to toxins such as vinyl chloride. Cases associated with recognised exposure to carcinogens have fortunately been extremely rare for the last three or more decades. However, the tumour has by no means disappeared in the Australian community. In this case series, we describe three cases of hepatic angiosarcoma that were seen at our institution since 2002. The first case presented with cholestatic liver function tests and was found to have angiosarcoma on liver biopsy. In the second case, the patient was admitted for decompensated liver disease on a background of presumed hepatitis B cirrhosis. The diagnosis of hepatic angiosarcoma was made only at autopsy after the patient died from multi-organ failure. The third case presented with ascites and the diagnosis of disseminated angiosarcoma was again made at autopsy following a negative ante-mortem liver biopsy.
Squamous cell carcinoma in the native kidney of a renal transplant recipient ...Apollo Hospitals
We are reporting a case of squamous cell carcinoma of the native kidney in a renal
transplant recipient. A 54-year-old gentleman, a renal transplant recipient for three years,
presented with flank pain. On evaluation he was found to have a mass in the upper pole of
the left native kidney. Renal angiogram was done which showed a functioning transplanted
kidney with a large mass arising from the upper pole of the left native kidney. He
underwent nephrectomy. The histopathology reported a squamous cell carcinoma. He was
given adjuvant radiotherapy to the tumor bed using image guided radiotherapy thereby
delivering a differential dose to the high risk areas and preserving the surrounding normal
structures. He developed a urethral nodule which was found to be a squamous cell carcinoma.
The lesion was excised with clear margins. We present this case because it is rare
and to discuss adjutant management.
Squamous Cell Carcinoma in the Native Kidney of a Renal Transplant Recipient ...Apollo Hospitals
We are reporting a case of squamous cell carcinoma of the native kidney in a renal transplant recipient. A 54-year-old gentleman, a renal transplant recipient for three years, presented with flank pain. On evaluation he was found to have a mass in the upper pole of the left native kidney. Renal angiogram was done which showed a functioning trans-
planted kidney with a large mass arising from the upper pole of the left native kidney. He underwent nephrectomy. The histopathology reported a squamous cell carcinoma. He was given adjuvant radiotherapy to the tumor bed using image guided radiotherapy thereby delivering a differential dose to the high risk areas and preserving the surrounding normal structures. He developed a urethral nodule which was found to be a squamous cell carcinoma. The lesion was excised with clear margins. We present this case because it is rare and to discuss adjuvant management.
Annular pancreas is an uncommon condition in adults.
The ring formation generally originates from the failure of
normal clockwise rotation of ventral pancreas. First
described by Tiedmann in 1818, its incidence is
1:20,000 population. It has bimodal presentation i.e is seen
either in Infants or in 4th & 5th decade of life.
CARCINOMA COLON - Dr. ZAHID IQBAL MIR
Dr. Zahid Iqbal Mir, MBBS MS (General Surgery), DNB (General Surgery) has done his MBBS and masters in General Surgery from the prestigious Govt Medical College Jammu and DNB in General Surgery from NBEMS New Delhi. He is a passionate surgeon, earlier practising at Government Medical College, Jammu as Registrar in Department of General Surgery. Nowadays working as Senior Resident in Department of General Surgery, Government Medical College & Hospital, Sector 32, Chandigarh and a rising name in field of surgery.
He is an enthusiastic, enigmatic and dedicated teacher as well. He is not just a resolute learner, but also an awe inspiring guiding light for his juniors, which makes him the most loveable and respected senior.
Currently he is running “LOVE FOR SCALPEL” for PGMEE aspirants on most of the social platforms, which is gaining immense popularity among residents, medical graduates and undergraduates.
Similar to Patología benigna y maligna de vesícula y vias biliares (20)
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
2. Colecistitis gangrenosa
• Hemor r agi a i nt r amur al
• Necr osi s
• Mi cr oabscesos
• Debr i s i nt r al umi nal es
• Exudados f i br i nosos
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
3. Colecistitis gangrenosa
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
4. Colecistitis gangrenosa
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
5. Colecistitis gangrenosa
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
6. Colecistitis alitiásica
• Adul t os gr aves
• Vi scosi dad bi l i ar
• Necr osi s mur al
• Mor t al i dad
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
7. Colecistitis alitiásica
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
8. Colecistitis alitiásica
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
9. Colecistitis alitiásica
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
10. Colecistitis enfisematosa
• <1% de col eci st i t i s
• Di abét i cos 35%
• Cl o s t r i d i um s p , Kl e bs i e l l a
s p , E. Co l i .
• Ri esgo de per f or aci ón 5x
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
12. Colecistitis enfisematosa
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
14. Perforación vesicular
• 5 a 10% de col eci st i t i s
• Col eci st i t i s gangr enosa
• Fondo vesi cul ar
• Paci ent es t óxi cos
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
18. Vesícula en porcelana
• <1%
• Capa muscul ar , mucosa y/ o
submucosa
• 5x más f r ecuent e en hombr es
• 11- 33% Ca vesi cul ar .
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
25. Adenomiomatosis
• Pr ol i f er aci ón epi t el i al con
hi per t r of i a muscul ar y
di ver t í cul os mucosos-
submucosos.
• Di f usa, segment ar i a y
Local i zada ( f ondo) .
• Nódul os de Roki t ansky
Aschof fGore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
26. Adenomiomatosis
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
27. Adenomiomatosis
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
28. Adenomiomatosis
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
30. Pólipos
• Col est er ol : 5%,
adenomat osos: 0. 4%
• 0. 1 a 2. 5 cm de di ámet r o
• >1 cm: sospechar mal i gni dad
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
34. Pólipos
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
35. Carcinoma vesicular
• 5ª GI más f r ecuent e
• 1- 3% de col eci st ect omí as
• Li t i asi s, col eci st i t i s cr óni ca
y vesí cul a en por cel ana.
• Muj er es
• 60- 70 años.
• DX di f í ci l y t ar dí o
• Sobr evi da a 5 años: <5%
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
36. Carcinoma vesicular
• 3 f or mas de pr esent aci ón
– Engr osami ent o de par ed f ocal
– Tumor i nt r al umi nal pol i poi de >2 cm
( 25%) .
– Tumor aci ón subhepát i ca ( 40- 65%)
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
37. Carcinoma vesicular
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
38. Carcinoma vesicular
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
39. Carcinoma vesicular
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
40. Carcinoma vesicular
Gore RM, Yaghmai V, Newmark G et al. Imaging benign and malignant disease of
the gallbladder. Radiol Clin N Am 40. 1307– 1323. Evanston, IL. 2002.
42. Tumores benignos de vía biliar
• Rar os
• Adenoma de ví a bi l i ar
• Pequeños ( i nci dent al es)
• Def ect os de l l enado
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
43. Adenoma de vía biliar
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
44. Cistadenoma/Cistadenocarcinoma biliar
• I nt r ahepát i cos
• Muj er es de edad medi a
• Tumor aci ones gr andes
sol i t ar i as
• Tumor aci ones quí si t cas –
t umor aci ones nodul ar es
sept adas
• Cal ci f i caci ones f i nas o
i r r egul ar es abundant es
• Pr emal i gnosBarón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
45. Cistadenoma biliar hiliar
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
46. Cistadenoma biliar hiliar
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
47. Colangiocarcinoma
• Más común
• I nt r ahepát i cos ( 20- 30%)
• Semej an met ást asi s
• Hi l i ar es ( Kl at ski n) ( más
comunes)
• Conf l uenci a del hepát i co der echo o
i zqui er do
• USG. ” Vent ana hepàt i ca”
• Duct al es di st al es
• Menos común
• Mej or pr onóst i co
• USG
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
48. Colangiocarcinoma intrahepático
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
49. Colangiocarcinoma hiliar
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
50. Colangiocarcinoma hiliar
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
dsease. Radiol Clin N Am 40. 1325– 1743. Pittsburgh, PA. 2002.
51. Colangiocarcinoma ductal distal
Barón RL, Tublin V, Peterson M. Imaging the spectrum of biliary tract
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