This document provides guidance on pediatric advanced life support (PALS). It discusses respiratory and circulatory failure, which often lead to cardiac arrest in children. Asphyxial cardiac arrest caused by lack of oxygen is more common than primary cardiac issues. Shock is also a common precursor and progresses from compensated to decompensated states. Foreign body airway obstruction, drowning, and hypothermia/hyperthermia are covered. The document provides detailed guidance on airway management, ventilation, vascular access, defibrillation, and the management of arrhythmias like tachycardia and bradycardia in a pediatric setting.
Presentation on NRP (Neonatal Resuscitation Program)Moninder Kaur
NRP is neonatal resuscitation program. Approximately 10% of newborns require some assistance to begin breathing at birth. Less than 1% require extensive resuscitative measures. Although the majority of newly born infants do not require intervention to make the transition from intrauterine to extra-uterine life, because of the large total number of births, a sizable number will require some degree of resuscitation.
Presentation on NRP (Neonatal Resuscitation Program)Moninder Kaur
NRP is neonatal resuscitation program. Approximately 10% of newborns require some assistance to begin breathing at birth. Less than 1% require extensive resuscitative measures. Although the majority of newly born infants do not require intervention to make the transition from intrauterine to extra-uterine life, because of the large total number of births, a sizable number will require some degree of resuscitation.
pediatric assessment in emergency rooms , how to pass the PALS exam , part 1 search for the other 3 parts, for any comment send to sayedahmed 1900@ g mail .com
Advance life support refer to a constellation of interventions needed to support the vital physiological process during a critical illness, while we await response with definitive therapy. These life support measures are instituted to prevent cardiac arrest.
To recognise physiological derangements that arise out of multiple etiologies and stabilize them first.
EVALUATE – IDENTIFY – INTERVENE
The steps of evaluation are
1.Initial impression
2. Primary assessment
3. Secondary assessment
4. Diagnostic test
Gives insight to overall physiological status and functioning of the brain.
TICLS
Tone: Look for general posture of the child has adopted
Interactive: Is the child responsive and interacting appropriately, unresponsive or lethargic.
Consolable: Irritable, consolable or inconsolable
Look\Gaze: How is the child looking at mother, any vacant gaze
Speech: Is the child able to speak or vocalise as is appropriate for age or is there a paucity\weak\hoarseness of voice.
IDENTIFY = Abnormality in any of these parameters point towards a brain dysfunction
Impaired consciousness is a significant alteration in the awareness of self and environment with varying degree of wakefulness.
Unconsciousness persisting for at lest 1 hr – Coma.
Younger children more likely to have coma or altered sensorium secondary to non-traumatic etiology, where as traumatic brain injury is more common in older children.
Always rule out reversible causes of coma, like hypoglycemia, hyperglycaemia and electrolyte imbalance.
Any severe systemic illness can cause altered consciousness as a result of hypoxic ischemic insult, which if on-going can aggravate raised ICT.
Basic life support is a course run by American Heart Association that teaches about handling cardiac arrest in Out of Hospital and In Hospital Situations. This Presentation covers important aspects of the same.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
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These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
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ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
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2. PEDIATRIC
ADVANCED LIFE
SUPPORT
“Assessment and maintenance of pulmonary and circulatory
function in the period before, during and after an instance of
cardiopulmonary arrest in a seriously ill or injured child, by a
system of critical care procedures and facilities.”
AHA 2014
Timely intervention in seriously ill or injured children is the key to
preventing progression toward cardiac arrest and to saving lives.
3. INTRODUCTION
In contrast to adults, cardiac arrest in infants and children
does not usually result from a primary cardiac cause. More
often it is the terminal result of progressive respiratory
failure or shock, also called an asphyxial arrest. Asphyxia
begins with a variable period of systemic hypoxemia,
hypercapnea, and acidosis, progresses to bradycardia and
hypotension, and culminates with cardiac arrest.
4. BASIC
CONSIDERATIONS FOR
PALS
Paediatric advanced life support (PALS) usually takes place
in the setting of an organized response in an advanced
healthcare environment.
In these circumstances, multiple responders are rapidly
mobilized and are capable of simultaneous coordinated
action.
5. ASPHYXIAL CARDIAC
ARREST
Cardiac arrest caused by asphyxiation (lack of oxygen in
blood)
Carbon dioxide accumulates in the lungs while oxygen in the
lungs is depleted resulting in cardiac arrest.
Causes: drowning, choking, airway obstruction, sepsis,
shock
6. ASPHYXIAL CARDIAC
ARREST
“One large pediatric study demonstrated that CPR with chest
compression and mouth-to-mouth rescue breathing is more
effective than compression alone when the arrest was from a
noncardiac etiology.”
“Ventilations are more important during resuscitation from
asphysia-induced arrest, than during resuscitation from VF
or pulseless VT.”
7. SHOCK
Shock results from inadequate blood flow and oxygen delivery to meet
tissue metabolic demands.
The most common type of shock in children is hypovolemic,
including shock due to hemorrhage.
Distributive, cardiogenic, and obstructive shock occur less frequently.
Shock progresses over a continuum of severity, from a compensated to
a decompensated state.
Compensatory mechanisms include tachycardia and increased
systemic vascular resistance (vasoconstriction) in an effort to maintain
cardiac output and perfusion pressure respectively.
Decompensation occurs when compensatory mechanisms fail and
results in hypotensive shock.
8. Typical signs of compensated shock include
● Tachycardia
● Cool and pale distal extremities
● Prolonged (2 seconds) capillary refill (despite warm
ambient temperature)
● Weak peripheral pulses compared with central pulses
● Normal systolic blood pressure
As compensatory mechanisms fail, signs of inadequate
end-organ perfusion develop. In addition to the above, these
signs include
● Depressed mental status
● Decreased urine output
● Metabolic acidosis
● Tachypnea
● Weak central pulses
● Deterioration in color
9. ● Tachycardia is a common sign of shock, but it can also result
from other causes, such as pain, anxiety, and fever.
● Pulses are weak in hypovolemic and cardiogenic shock, but
may be bounding in anaphylactic, neurogenic, and septic shock.
● Blood pressure may be normal in a child with compensated
shock but may decline rapidly when the child decompensates.
Like the other signs, hypotension must be interpreted within the
context of the entire clinical picture.
10. HYPO/HYPERTHERMI
A
Potential complications of hypothermia include diminished cardiac output,
arrhythmia, pancreatitis, coagulopathy, thrombocytopenia,
hypophosphatemia, hypovolemia from cold diuresis, hypokalemia, and
hypomagnesemia.
● Monitor temperature continuously, if possible, and treat fever (38°C)
aggressively with antipyretics and cooling devices because fever adversely
influences recovery from ischemic brain injury
● Treat post ischemic seizures aggressively; search for a correctable
metabolic cause such as hypoglycemia or electrolyte imbalance.
● Avoid rewarming from 32 to 34°C faster than 0.5°C per 2 hours unless the
patient requires rapid rewarming for clinical reasons.
11. FOREIGN-BODY AIRWAY
OBSTRUCTION
90% of childhood deaths from foreign body aspiration occur
in children < 5 years of age; 65% are infants.
Balloons, small objects, foods (hot dogs, round candies,
nuts and grapes) are the most common causes of foreign-
body airway obstruction
12. FBAO
If FBAO is mild, do not interfere.
• Allow the victim to clear the airway by coughing.
If the FBAO is severe (victim unable to make a sound) you
must act the relieve the obstruction.
13. FBAO
For a child perform subdiaphragmatic abdominal thrusts
until the object is expelled or the victim becomes
unresponsive.
For an infant, deliver repeated cycles of 5 back blows
followed by 5 chest compressions until the object is
expelled or the victim becomes unresponsive.
14. FBAO –
UNRESPONSIVE
Start Chest Compression
After 30 chest compressions open airway
If you see a foreign body remove it
DO NOT perform a blind finger sweep
Give 2 breaths
Followed by 30 chest compressions
15. DROWNING
Outcomes after drowning is determined by the duration of
submersion, the water temperature, and how promptly and
effectively CPR is provided.
16. PEDIATRIC CARDIAC
ARREST
Pediatric cardiopulmonary arrest results when respiratory
failure or shock is not identified and treated in the early
stages.
Early recognition and intervention prevents deterioration to
cardiopulmonary arrest and probable death.
17. CARDIAC ARREST
Pediatric cardiac arrest is:
• Uncommon
• Rarely sudden cardiac arrest caused by primary cardiac
arrhythmias.
• Most often asphyxial, resulting from the progression of
respiratory failure or shock or both.
19. RESPIRATORY
FAILURE
Respiratory failure is characterized by inadequate ventilation,
insufficient oxygenation, or both. Anticipate respiratory failure
if any of the following signs is present:
● An increased respiratory rate, particularly with signs of
distress (eg, increased respiratory effort including nasal
flaring, retractions, seesaw breathing, or grunting)
● An inadequate respiratory rate, effort, or chest excursion
(eg, diminished breath sounds or gasping), especially if
mental status is depressed
● Cyanosis with abnormal breathing despite supplementary
Oxygen.
20. BREATHING
Breathing is assessed to determine the child’s ability to
oxygenate.
Assessment:
• Respiratory rate
• Respiratory effort
• Breath sounds
• Skin color
21. BREATHING CHILD
If child is breathing, put in recovery position, call emergency
response system, return quickly and re-assess child’s
condition
Turn child on-side (recovery position)
22. INADEQUATE
BREATHING WITH
PULSE
If pulse > 60 per minutes but there is inadequate breathing
give rescue breathing at a rate of about 12 to 20 breathes per
minute.
Reassess pulse about every 2 minutes
• Carotid or femoral for child
• Brachial for infant
23. UNRESPONSIVE AND
NOT BREATHING
If the child is unresponsive and not breathing (or only
gasping) begin CPR.
Start with high-quality chest compression. (30 chest
compressions)
After one cycle 2 minutes check for pulses
Call for help when able
24. AIRWAY
Airway must be clear and patent for successful ventilation.
• Position
• Suction
• Administration of oxygen
• Bag-mask ventilation
• Note: suctioning is helpful if secretions, blood or debris is
present. Use with caution if upper airway swelling is edema
(eg. croup, epiglottitis)
30. RAPID SEQUENCE
INTUBATION (RSI)
To facilitate emergency intubation and reduce the incidence
of complications, skilled, experienced providers may use
sedatives, neuromuscular blocking agents, and other
medications to rapidly sedate and neuromuscularly block the
pediatric patient.
Use RSI only if you are trained, and have experience using
these medications and are proficient in the evaluation and
management of the pediatric airway.
If you use RSI you must have a secondary plan to manage
the airway in the event that you cannot achieve intubation.
31. CRICOID PRESSURE
DURING INTUBATION
There is insufficient evidence to recommend routine cricoid
pressure application to prevent aspiration during
endotracheal intubation in children.
Do not continue cricoid pressure if it interferes with
ventilation or the speed or ease of intubation.
32. END-TIDAL CO2
(PETCO2)
Continuous capnography or capnometry monitoring, if
available, may be beneficial during CPR, to help guide
therapy, especially the effectiveness of chest compressions
If the PETCO2 is consistently 10 to 15 mm Hg, focus efforts
on improving chest compressions and make sure that the
victim does not receive excessive ventilation.
33. VENOUS ACCESS
Peripheral IV access is acceptable during resuscitation if it can
be placed rapidly, but placement may be difficult in a critically ill
child.
Although a central venous catheter can provide more secure
long-term access, its placement requires training and
experience, and the procedure can be time consuming.
Therefore central venous access is not recommended as the
initial route of vascular access during an emergency.
If both central and peripheral accesses are available, administer
medications into the central circulation since some medications
(eg, adenosine) are more effective when administered closer to
the heart, and others (eg, calcium, amiodarone, procainamide,
sympathomimetics) may be irritating when infused into a
peripheral vein.
34. VASCULAR ACCESS
Vascular access is essential for administering medications
and drawing blood samples. Obtaining peripheral venous
access can be challenging in infants and children during an
emergency; intraosseous (IO) access can be quickly
established with minimal complications by providers with
varied levels of training.
35. VASCULAR ACCESS –
NEW GUIDELINES
New guidelines: in children who are six years or younger
after 90 seconds or 3 attempts at peripheral intravenous
access – Intraosseous vascular access in the proximal tibia
or distal femur should be initiated.
38. INTRAOSSEOUS (IO) ACCESS
IO access is a rapid, safe, effective, and acceptable route for
vascular access in children and it is useful as the initial vascular
access in cases of cardiac arrest.
All intravenous medications can be administered
intraosseously, including epinephrine, adenosine, fluids,
blood products and catecholamines.
Onset of action and drug levels for most drugs are comparable
to venous administration.
40. ENDOTRACHEAL DRUG
ADMINISTRATION
Vascular access (IO or IV) is the preferred method for drug
delivery during CPR, but if it is not possible, lipid-soluble
drugs, such as lidocaine, epinephrine, atropine, and
naloxone
(mnemonic “LEAN”) can be administered via an endotracheal
tube.
However, the effects may not be uniform with tracheal as
compared with intravenous administration.
41. NEUROLOGIC
SYSTEM
A primary goal of resuscitation is to preserve brain function. Limit the
risk of secondary neuronal injury by adhering to the following
precautions:
● Do not routinely provide excessive ventilation or hyperventilation. It
has no benefit and may impair neurologic outcome by adversely
affecting cardiac output and cerebral perfusion. Intentional brief
hyperventilation may be used as temporizing rescue therapy in response
to signs of impending cerebral herniation (eg, sudden rise in measured
intracranial pressure, dilated pupil not responsive to light, bradycardia,
hypertension).
● Therapeutic hypothermia (32°C to 34°C) may be considered for children
who remain comatose after resuscitation from cardiac arrest.
43. CIRCULATION
ASSESSMENT
Heart rate (most accurate assessment)
Blood pressure
End organ profusion
• Urine output (1-2 mL / kg / hour)
• Muscle tone
• Level of consciousness
44. CIRCULATORY
ASSESSMENT
Heart rate is the most sensitive parameter for determining
perfusion and oxygenation in children.
• Heart rate needs to be at least 60 beats per minute to
provide adequate perfusion.
• Heart rate greater than 140 beats per minute at rest
needs to be evaluated.
45. BLOOD PRESSURE
25% of blood volume must be lost before a drop in blood
pressure occurs.
Minimal changes in blood pressure in children may indicate
shock.
46. BLOOD PRESSURE
GUIDELINES
Hypotension is defined as systolic blood pressure
Neonates: < 60 mm Hg
Infants: <70 mm Hg
Child (1 to 10): < 70 mm Hg
Child (>10): < 90 mm Hg
47. IV SOLUTIONS
Crystalloid solution for fluid loss (hypovolemia)
• Normal saline 20ml/kg bolus over 20 minutes
• Ringers Lactate
Blood loss:
Colloid: 5% albumin
Blood
Fresh-frozen plasma
48. GASTRIC
DECOMPRESSION
Gastric decompression with a nasogastric or oral gastric
tube is necessary to ensure maximum ventilation.
• Air trapped in stomach can put pressure on the diaphragm
impeding adequate ventilation.
• Undigested food can lead to aspiration.
49. ELECTROCARDIOGRAPHY
Monitor cardiac rhythm as soon as possible so both normal
and abnormal cardiac rhythms are identified and followed.
Continuous monitoring is helpful in tracking responses to
treatment and changes in clinical condition.
51. DEFIBRILLATORS
Defibrillators are either manual or automated (AED),
with monophasic or biphasic waveforms.
AEDs in institutions caring for children at risk for
arrhythmias and cardiac arrest (eg, hospitals, EDs) must
be capable of recognizing pediatric cardiac rhythms and
should ideally have a method of adjusting the energy
level for children.
52. DEFIBRILLATION
“Children with sudden witnessed collapse (eg, a child
collapsing during an athletic event) are likely to VF or
pulseless VT and need immediate CPR and rapid
defibrillation. “
VF and pulseless VT are referred to as “shockable rhythms”
because they respond to electric shocks.
VT – ventricular tachycardia
VF – ventricular fibrillation
53. DEFIBRILLATION
DOSING
The recommended first energy dose for defibrillation is 2
J/kg.
If second dose is required, it should be doubled to 4 J/kg.
AED with pediatric attenuator is preferred for children < 8
years of age.
54. DEFIBRILLATION
SEQUENCE
Turn AED on
Follow the AED prompts
End CPR cycle (for analysis and shock)
Resume chest compressions immediately after the shock.
Minimize interruptions in chest compressions.
State CLEAR when giving the shock and have visual / verbal
communication with any other rescue personal
55. DEFIBRILLATOR
GUIDELINES
AHA recommends that automatic external defibrillation be
use in children with sudden collapse or presumed cardiac
arrest who are older than 8 years of age or more than 25 kg
and are 50 inches tall.
Electrical energy is delivered by a fixed amount range 150 to
200. (2-4J/kg)
57. BRADYCARDIA WITH
POOR PERFUSION
•If pulse is less than 60 per minutes
and there are signs of poor perfusion
• Pallor
• Mottling
• cyanosis
despite support of oxygenation and ventilation – start
CHEST COMPRESSION
58. BRADYCARDIA
The most common dysrhythmia in the pediatric population.
Etiology is usually hypoxemia
Initial management: ventilation and oxygenation.
If this does not work IV or IO epinephrine 0.01 mg / kg
(1:10,000)
ET tube (not recommended) 0.1 mg / kg
Search for and treat possible contributing factors
60. PULSELESS ARREST
● As soon as the child is found to be unresponsive with no
breathing, call for help, send for a defibrillator, and start CPR
(with supplementary oxygen if available). Attach ECG monitor or
AED pads as soon as available. Throughout resuscitation,
emphasis should be placed on provision of high-quality CPR
● While CPR is being given, determine the child’s cardiac
rhythm from the ECG or, if you are using an AED, the device will
tell you whether the rhythm is “shockable” (eg VF or rapid VT)
or “not shockable” (eg, asystole or PEA). It may be necessary to
temporarily interrupt chest compressions to determine the
child’s rhythm. Asystole and bradycardia with a wide QRS are
most common in asphyxial arrest.
VF and PEA are less common but VF is more likely to be present
in older children with sudden witnessed arrest.
61. MANAGEMENT
Consider vagal maneuvers
Establish vascular access
Adenosine
• First dose 0.1 mg/kg IV (maximum of 6 mg)
• Second dose 0.2 mg/kg IV (maximum of 12 mg)
Synchronized cardioversion: 0.5 to 1 J / kg
63. PULSELESS ARREST –
ASYSTOLE
CAB: Start CPR
Give oxygen when available
Attach monitor / defibrillator
Check rhythm / check pulse
If asystole give epinephrine 0.01 mg / kg of 1:10,000
Resume CPR may repeat epinephrine every 3-5 minutes until
shockable rhythm is seen
64. PULSELESS ARREST –
VF AND VT
Start CAB
Give oxygen
Attach monitor / defibrillator
Check rhythm: VF / VT
Give one shock at 2 J/kg
If still VF / VT
Give 1 shock at 4 J/kg
Give Epinephrine 0.01 mg/kg of 1:10,000
Consider: amiodarone at 5 mg / kg
65. NEW GUIDELINE
EPINEPHRINE
Still remains primary drug for treating patients for
cardiopulmonary arrest, escalating doses are de-emphasized.
Neurologic outcomes are worse with high-dose epinephrine.
67. If CPR is in progress, stop chest compressions briefly,
administer the medications, and follow with a flush of at least
5 mL of normal saline and 5 consecutive positive-pressure
ventilations. Optimal endotracheal doses of medications are
unknown; in general expert consensus recommends
doubling or tripling the dose of lidocaine, atropine or
naloxone given via the ETT. For epinephrine, a dose ten
times the intravenous dose (0.1 mg/kg or 0.1 mL/kg of 1:1000
concentration) is recommended
68. EPINEPHRINE
Action: increase heart rate, peripheral vascular resistance
and cardiac output; during CPR increase myocardial and
cerebral blood flow.
Dosing: 0.01 mg / kg 1: 10,0000
69. AMIODARONE
Used in atrial and ventricular antiarrhythmic
Action: slows AV nodal and ventricular conduction, increase the
QT interval and may cause vasodilation.
Dosing: IV/IO: 5 mg / kg bolus
Used in pulseless arrest
70. ADENOSINE
Drug of choice of symptomatic SVT
Action: blocks AV node conduction for a few seconds to
interrupt AV node re-entry
Dosing
• First dose: 0.1 mg/kg max 6 mg
• Second dose: 0.2 mg/kg max 12 mg
• Used in tachycardia with pulses after synchronized
cardioversion
71. GLUCOSE
10% to 25% strength
Action: increases glucose in hypoglycemia
Dosing: 0.5 – 1 g/kg
75. DOPAMINE
Therapeutic classification: inonotropic
May be used to treat shock; effects are dose dependent
Increases force of contraction and cardiac output, increases
peripheral vascular resistance, BP and cardiac output
Dosing: IV/IO infusion: 2-20 mcg/kg/min
76. POST-
RESUSCITATION
CARE
Re-assessment of status is ongoing.
Laboratory and radiologic information is obtained.
Etiology of respiratory failure or shock is determined.
Transfer to facility where child can get maximum care.
77. REFERENCES
Duncan BW, Ibrahim AE, Hraska V, del Nido PJ, Laussen PC, Wessel DL, Mayer JE,
Jr., Bower LK, Jonas RA. Use of rapid-deployment extracorporeal membrane
oxygenation for the resuscitation of pediatric patients with heart disease after
cardiac arrest. J Thorac Cardiovasc Surg. 1998;116:305–311.
Hoskote A, Bohn D, Gruenwald C, Edgell D, Cai S, Adatia I, Van Arsdell G.
Extracorporeal life support after staged palliation of a functional single ventricle:
subsequent morbidity and survival. J Thorac Cardiovasc Surg. 2006;131:1114 –
1121.
Ibrahim AE, Duncan BW, Blume ED, Jonas RA. Long-term follow-up of pediatric
cardiac patients requiring mechanical circulatory support. Ann Thorac Surg.
2000;69:186 –192.
Prodhan P, Fiser RT, Dyamenahalli U, Gossett J, Imamura M, Jaquiss RD, Bhutta
AT. Outcomes after extracorporeal cardiopulmonary resuscitation (ECPR) following
refractory pediatric cardiac arrest in the intensive care unit. Resuscitation.
2009;80:1124 –1129.
Pediatric Advanced Life Support2010,2014 American Heart Association Guidelines
for Cardiopulmonary Resuscitation and Emergency Cardiovascular Care