This document discusses various orthopedic injuries to the lower limb, including fractures of the femoral neck, intertrochanteric fractures, femoral shaft fractures, distal femur fractures, patella fractures, tibial fractures, ankle fractures, and knee dislocations. For each injury, the document discusses epidemiology, mechanism of injury, clinical presentation, treatment options including operative and non-operative management, and potential complications. Surgical treatment involves procedures like open reduction internal fixation, intramedullary nailing, and arthroplasty, while non-operative care includes casting and bracing with restricted weight bearing.
MRI anatomy of ankle radiology ppt pk is nice presentation that covers cross sectional anatomy as well as relevant anatomy from standard radiology book like CT MRI whole body by Hagga . cross section of mri is taken from mrimaster.com. This will help for radiology resident as well radiographers.
Avascular necrosis of the femoral head
introduction
causes
anatomy of femur
blood supply of femur
Clinical Features
Investigations
Differential Diagnosis
treatments
Surgical Treatment
Prognosis
aseptic necrosis
ischemic necrosis.
Legg-Calvé-Perthes syndrome
Causes Of Avascular Necrosis
MRI anatomy of ankle radiology ppt pk is nice presentation that covers cross sectional anatomy as well as relevant anatomy from standard radiology book like CT MRI whole body by Hagga . cross section of mri is taken from mrimaster.com. This will help for radiology resident as well radiographers.
Avascular necrosis of the femoral head
introduction
causes
anatomy of femur
blood supply of femur
Clinical Features
Investigations
Differential Diagnosis
treatments
Surgical Treatment
Prognosis
aseptic necrosis
ischemic necrosis.
Legg-Calvé-Perthes syndrome
Causes Of Avascular Necrosis
The effect of intact fibula on functional outcome of reamed intramedullary in...Love2jaipal
detailed journal club presentation on The effect of intact fibula on functional outcome of reamed intramedullary interlocking nail in open and closed isolated tibial shaft fractures
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
2. INJURIES TO THE LOWER LIMB ( 2 )
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
2
3. FEMORAL NECK FRACTURES
Epidemiology
increasingly common due to aging
population
women > men
whites > blacks
most expensive fracture to treat
Mechanism
high energy in young patients
low energy falls in older patients
Healing potential
femoral neck is intracapsular, bathed in
synovial fluid
lacks periosteal layer
callus formation limited, which affects healing
3
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
4. Blood supply to femoral head
major contributor is medial femoral circumflex (lateral epiphyseal
artery)
some contribution to anterior and inferior head from lateral femoral
circumflex
some contribution from inferior gluteal artery
small and insignificant supply from artery of ligamentum teres
displacement of femoral neck fracture will disrupt the blood supply and
cause an intracapsular hematoma
4
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
5. FEMORAL NECK FRACTURES
Symptoms
impacted and stress fractures
slight pain in the groin or pain referred
along the medial side of the thigh and
knee
displaced fractures
pain in the entire hip region
Physical exam
impacted fractures
no obvious clinical deformity
minor discomfort with active or passive
hip range of motion, muscle spasms at
extremes of motion
pain with percussion over greater
trochanter
displaced fractures
leg in external rotation and abduction,
with shortening
5
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
6. FEMORAL NECK FRACTURES
Treatment
Nonoperative
observation alone
may be considered in some patients who
are non-ambulators, have minimal pain,
and who are at high risk for surgical
intervention
Operative
ORIF
Cannulated screw fixation (<50 yo)
Sliding hip screw
Hemiarthroplasty (for elders )
total hip arthoplasty (for active elders ) 6
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
8. FEMORAL NECK FRACTURES
Prognosis
mortality
~25-30% at one year (higher than vertebral compression
fractures)
predictors of mortality
pre-injury mobility is the most significant determinant for
post-operative survival
in patients with chronic renal failure, rates of mortality at
2 years postoperatively, are close to 45%
8
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
9. INTERTROCHANTERIC FRACTURES
Extracapsular fractures of the proximal femur between the
greater and lesser trochanters
Female : male ratio between 2:1 and 8:1
typically older age than patients with femoral neck fractures
Risk factors: osteoporosis, prior hip fracture, risk of
falls
More common than femoral neck fracture in patients
with preexisting hip arthritis
In contrast to intracapsular fractures, extracapsular trochanteric
fractures unite quite easily and seldom cause avascular
necrosis
Physical Exam : painful, shortened, externally rotated lower
extremity 9
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
11. INTERTROCHANTERIC FRACTURES
Treatment
Nonoperative
nonweightbearing with early out of bed to chair
Operative
Intertrochanteric fractures are almost always treated by early
internal fixatio because :
to obtain the best possible position And
to get the patient up and walking as soon as possible and
thereby reduce the complications associated with prolonged
recumbency
sliding hip compression screw
intramedullary hip screw
arthroplasty
11
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
12. INTERTROCHANTERIC FRACTURES
Complications
Implant failure and cutout
most common complication
usually occurs within first 3 months
Nonunion <2%
treatment
revision ORIF with bone grafting
proximal femoral replacement
Malunion
12
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
13. FEMORAL SHAFT FRACTURES
high energy injury that is associated with life-threating conditions
Fracture patterns
transverse
spiral
oblique
segmental
comminuted
Blood loss in closed femoral shaft fractures is 1000-1500ml
Must record and document distal neurovascular status
13
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
14. FEMORAL SHAFT FRACTURES
Treatment
Nonoperative
long leg cast or hip spica cast
nondisplaced femoral shaft fractures in patients with
multiple medical comorbidities
pediatric patients
Operative
antegrade intramedullary nail ( gold standard for
treatment of diaphyseal femur fractures )
retrograde intramedullary nail
external fixation with conversion to intramedullary nail
within 2-3 weeks
open reduction internal fixation with plate
14
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
16. FEMORAL SHAFT FRACTURES
Complications
Heterotopic ossification 25%
Pudendal nerve injury
Femoral artery or nerve injury
Malunion and rotational
malalignment
Delayed union
Nonunion
Infection
Fat embolism
16
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
Heterotopic ossification
17. DISTAL FEMUR FRACTURE
Distal femur fractures are fractures extending from
the distal metaphyseal-diaphyseal junction of the femur
to the articular surface of the femoral condyles.
They occur both in younger patients (as the result of high
energy trauma) or in older patients (from low energy
trauma as a pathological fracture secondary to
osteoporosis or malignancy).
The classification is commonly used to classify distal
femur fractures into
extra-articular (type A),
partial articular (type B),
and complete articular (type C). 17
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
19. DISTAL FEMUR FRACTURE
The majority of distal femur fractures are managed
surgically.
retrograde nailing or open reduction internal
fixation (ORIF).
Non-operative management requires a long period of
immobilisation and non-weight bearing, however is
sometimes indicated for fractures with minimal
displacement in a non-ambulatory or very co-
morbid patient.
19
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
20. PATELLA FRACTURE
A patella (kneecap) fracture is a traumatic injury caused by
direct trauma or rapid contracture of the quadriceps with a
flexed knee
most fractures occur in 20-50 year olds
male to female 2:1
Mechanism of injury
direct impact injury occurs from fall or dashboard injury
indirect eccentric contraction occurs from rapid knee
flexion against contracted quads muscle
Osteology
patella is largest sesamoid bone in body
superior 3/4 of posterior surface covered by articular cartilage
articular cartilage thickest in body (up to 1cm)
posterior articular surface comprised of medial and lateral
facets
lateral facet is larger
facets separated by vertical ridge 20
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
22. PATELLA FRACTURE
Treatment
Nonoperative
knee immobilized in extension (brace or cylinder cast) and full weight
bearing
Operative :depending on displacement and knee extension
function
ORIF
partial patellectomy
total patellectomy
22
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
23. DISLOCATION OF THE PATELLA
Anatomy
Static stability
medial patellofemoral ligament
(MPFL)
is primary restraint in first 20
degrees of knee flexion
patellar-femoral bony structures
account for stability in deeper
knee flexion
trochlear groove morphology,
patella height, patellar
tracking
Dynamic stability
provided by vastus medialis
23
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
24. DISLOCATION OF THE PATELLA
The patella can be dislocated by a sharp twisting movement of
the knee in very slight flexion
It is common in adolescents, particularly girls with loose
ligament
On examination soon after a dislocation, the knee will be
swollen because of the haemarthrosis and there will be
tenderness on the medial side of the patella because the medial
structures are torn (medial patella femoral ligament)
Treatment
All blood should be aspirated to help reduce pain.
Immobilization of the knee and early rehabilitation
If the patella has dislocated more than three times a
stabilizing operation will probably be required
24
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
26. DISLOCATION OF THE PATELLA
Recurrent dislocation of patella :
In over 70% of cases an underlying abnormality is found.
These include joint hypermobility, patella alta, patella maltracking and
axial malalignments
Anatomical factors
patella alta :causes patella to not articulate with sulcus, losing its
constraint effects
trochlear dysplasia
excessive lateral patellar tilt (measured in extension)
lateral femoral condyle hypoplasia
Treatment
Nonoperative
NSAIDS, activity modification, and physical therapy
Operative
Medial retinaculum and ligament repair
26
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
27. KNEE DISLOCATION
Complete separation of the tibia from the femur requires enough
trauma to tear at least two of the four major ligaments.
Both vascular and neurological functions must be assessed carefully
and recorded so that any deterioration will be noticed.
Damage to the popliteal vessels occurs in 50% of cases and an
angiogram is mandatory if there is doubt about the peripheral
vascularity.
Exploration of the popliteal artery and repair should be performed as
an emergency as there is a high chance of an amputation if this is
delayed more than 6 hours from the time of the injury
Types :
Anterior (30-50%) (most common)
Posterior (30-40%)
Lateral
Medial
Rotational
Treatment :
Emergent reduction followed by vascular assessment/consult
27
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
29. TIBIAL PLATEAU FRACTURES
Fractures of the tibial plateau are caused by a varus or valgus force
combined with axial loading (a pure valgus force is more likely to rupture
the ligaments).
Associated conditions
meniscal tears
ACL injuries
compartment syndrome
vascular injury
Radiology :
X-ray
CT scan
important to identify articular depression and comminution
Treatment :
Undisplaced fractures can be treated conservatively (splint).
Displaced fractures need open reduction and internal fixation +/- bone
graft .
29
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
31. TIBIAL SHAFT FRACTURES
Most common long bone fracture
account for 4% of all fracture seen in the Medicare population
Mechanism of injury
Indirect force: (low energy)
Twisting: spiral fractures of both bones
Angulation: oblique fractures with butterfly segment.
Direct force:
Transverse (low energy) or comminuted (high energy) fractures usually with skin and
soft tissue damage.
Treatment
Closed reduction / cast immobilization
indications
closed low energy fractures with acceptable alignment
< 5 degrees varus-valgus angulation
< 10 degrees anterior/posterior angulation
> 50% cortical apposition
< 1 cm shortening
No rotational malalignment
Open reduction internal fixation ( plate , IM nail)
External fixation ( mostly for open fractures )
31
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
33. TIBIAL SHAFT FRACTURES
Complications :
Knee pain
Malunion
Nonunion
Malrotation
Compartment syndrome
can occur in both
closed and open tibia
shaft fractures
Soft tissue damage.
Skin loss
33
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
34. DISTAL TIBIAL FRACTURES
These fractures occur at the distal end of the
tibia.
Tibial plafond fractures (Also known
as pilon fractures) are caused by high energy
axial load (motor vehicle accidents, falls from
height) and often characterized by articular
impaction and comminution with soft tissue
injury
Check Dorsalis Pedis and Posterior Tibial pulses
Ct scan is very important in planning for surgery
Treatment :
Undisplaced stable fractures can be treated
conservatively .
Displaced fractures need
open reduction and internal fixation (ORIF)
+/- bone graft .
External fixation and delayed ORIF
34
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
37. ANKLE FRACTURES
Typically a low-energy mechanism of injury, rotational as opposed to
axial load
Must always evaluate for deltoid or syndesmosis injury
injury patterns
isolated medial malleolus fracture
isolated lateral malleolus fracture
bimalleolar fractures
posterior malleolus fractures
syndesmotic injury
Radiographs recommended views
AP
lateral
Mortise (the leg internally rotated 15 degree ) for syndesmosis
37
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
39. ANKLE FRACTURES
Treatment
Nonoperative
short leg walking cast or cast boot for
nondisplaced stable fracture and tip
avulsions
Operative
ORIF for displaced unstable fractures
Complications
Wound problems surgery
Deep infections
up to 20% in diabetic patients
Malunion
Post-operative stiffness
Post-traumatic arthritis
39
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
40. ANKLE LIGAMENT INJURIES
ankle sprains involve an injury to the
Anterior Talo-Fibular Ligament (ATFL)
and calcaneofibular ligament (CFL)
and are the most common reason for
missed athletic participation
Types:
Stretching of the ligament.
Partial tear: healing restores full
function.
Complete tear: joint instability.
Usually involves lateral ankle
ligaments (ant. Talofibular lig.,
talocalcaneal lig., and post. Talofibular
lig.).
Medial calcaneal lig. (deltoid lig.) can
result from abduction or eversion
injury.
40
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
41. ANKLE LIGAMENT INJURIES
Clinical features:
Bruising, swelling, tenderness (usually distal
and anterior to lat. Malleolus in anterior
talofebular lig. Injury).
Treatment
Partial tears: RICE , elastic bandage and
gentle active exercise.
Complete tears: cast immobilization from
below knee to toes for 6 wks then
physiotherapy. if this regime fails; operative
repair is done.
Complications:
Recurrent sprains.
Recurrent giving way or instability.
41
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
42. FRACTURES OF TALUS
Rare and usually due to fall from
height , car accidents,…
May involve the body, neck, head
or dislocation of the talus.
Clinical features
Pain, swelling, deformity.
Radiology :
X-ray: difficult to diagnose.
May need to repeat several
days later to see the fracture.
CT in difficult cases.
42
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
43. FRACTURES OF TALUS
Treatment
Undisplaced : Below knee plaster
with knee plantigrade for 6-8 wks.
Displaced fractures or fracture
dislocations: urgent reduction by
closed manipulation; if fails,
ORIF
Complications
Non-union.
Avascular necrosis of body after
fracture of neck.
Secondary osteoarthritis. 43
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
44. CALCANEAL FRACTURES
Usually seen after fall from height .
Lover's fracture, also known as Casanova fracture is a type of calcaneal
fracture
Associated injuries: spine, pelvis, hip or base of skull.
Types: ( CT scan is important )
Extra-articular fractures: need closed treatment. Have good prognosis.
Intra-articular fractures: involve superior articular surface.
May be comminuted.
Special features
The foot swollen, bruised and the heel look broad. Movement is painful.
Signs of compartment syndrome: intense pain and diminished sensation.
Necessary to X-ray the knees, spine, and pelvis.
44
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
45. CALCANEAL FRACTURES
Treatment
Admit to hospital, elevate the leg
and apply ice-packs until swelling
subside.
Undisplaced fractures: closed
treatment.
Displaced fractures: ORIF with
screws or calcaneal plate and
screws.
Complications
Broadening of the heel
Stiffness
Osteoarthritis
Compartment syndrome 45
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
46. METATARSAL FRACTURES
Mechanism of injury:
Direct trauma, Twisting, Repetitive stress.
Treatment:
Walking plaster for 3 weeks.
Displaced fractures; Kirschner wire fixation.
46
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali
47. 5TH METATARSAL BASE FRACTURE
5th metatarsal base fractures are among the most common
fractures of the foot.
Some fractures may be predisposed to poor healing due to the
limited blood supply to the specific areas of the 5th metatarsal base.
Treatment can include protected weight bearing, immobilization or
surgery depending on location of fracture, degree of displacement,
and athletic level of patient.
47
Orthopedic
Surgery
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Dr.
Rami
Abo
Ali
48. 5TH METATARSAL BASE FRACTURE
Jones fracture :is a zone 2 fracture (a transverse fracture at the base of
the fifth metatarsal, 1.5 to 3 cm distal to the proximal tuberosity )
represents a vascular watershed area, making these fractures prone
to nonunion
zone 2 (Jones fracture) in elite or competitive athletes treated
surgically to minimizes possibility of nonunion or prolonged restriction
from activity
48
Orthopedic
Surgery
-
Dr.
Rami
Abo
Ali