Orbital fracture, types, blow in fracture ,blow out fracture ,clinical features ,superior orbital fissure syndrome ,management ,complications ,reconstruction techniques ,Oculocardiac reflex
Dr. Ahmed M. Adawy
Professor Emeritus, Dept. Oral & Maxillofacial Surg.
Former Dean, Faculty of Dental Medicine
Al-Azhar University
The term “blow out” refers to partial herniation of the orbital contents through one of its walls. This usually occurs via blunt force trauma to the eye. Most often, the orbital floor is fractured in conjunction with the inferior orbital rim “impure” blowout fracture, but “pure” orbital floor fractures, with intact orbital rim can be seen. An extensive and careful history, physical examination, together with CT scans is vital for the diagnosis of orbital floor fractures. The timing of treatment, surgical approaches, and reconstruction of the orbital floor are presented.
Orbital Fractures - The Role of an OphthalmologistAnkit Punjabi
Orbital fractures are a common finding in maxillofacial trauma. although a multi-disciplinary approach is essential, the role of ophthalmologist cannot be overemphazised. here we discuss the same.
Frontal sinus fractures are currently managed by various medical specialists, including otolaryngologists/head and neck surgeons, maxillofacial surgeons, plastic surgeons, and neurosurgeons. As a result, consensus does not exist regarding the timing, indications, and treatment modality of these injuries.
abscess advanced trauma life support anterior open bite antibiotics braces csf leaks dental diseases doxycycline dr dr shabeel drshabeel’s face eye trauma gingival infection medical medicine periodontal gum surgery pharmacy pn
Dr. Ahmed M. Adawy
Professor Emeritus, Dept. Oral & Maxillofacial Surg.
Former Dean, Faculty of Dental Medicine
Al-Azhar University
The term “blow out” refers to partial herniation of the orbital contents through one of its walls. This usually occurs via blunt force trauma to the eye. Most often, the orbital floor is fractured in conjunction with the inferior orbital rim “impure” blowout fracture, but “pure” orbital floor fractures, with intact orbital rim can be seen. An extensive and careful history, physical examination, together with CT scans is vital for the diagnosis of orbital floor fractures. The timing of treatment, surgical approaches, and reconstruction of the orbital floor are presented.
Orbital Fractures - The Role of an OphthalmologistAnkit Punjabi
Orbital fractures are a common finding in maxillofacial trauma. although a multi-disciplinary approach is essential, the role of ophthalmologist cannot be overemphazised. here we discuss the same.
Frontal sinus fractures are currently managed by various medical specialists, including otolaryngologists/head and neck surgeons, maxillofacial surgeons, plastic surgeons, and neurosurgeons. As a result, consensus does not exist regarding the timing, indications, and treatment modality of these injuries.
abscess advanced trauma life support anterior open bite antibiotics braces csf leaks dental diseases doxycycline dr dr shabeel drshabeel’s face eye trauma gingival infection medical medicine periodontal gum surgery pharmacy pn
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Orbital anatomy and orbital fracture/oral surgery courses by indian dental ac...Indian dental academy
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Bony orbits are Quadrangular truncated pyramids with Anterior cranial fossa above and the maxillary sinuses below.
in this presentation we study the detailed anatomy of the arbit, the bones, relations of each wall, the contents, the apertures, orbital fissures and structures passing, fascia, septa and the surgical spaces of the orbit
Head and Neck Trauma by Dr. Kenneth DickieKenneth Dickie
Dr. Kenneth Dickie from Royal Centre of Plastic Surgery in Barrie, Ontario explained the care for Head & Neck Trauma.
f you have any questions, please contact Dr. Kenneth Dickie at http://royalcentreofplasticsurgery.com/
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
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- Prix Galien International Awards Ceremony
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Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
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TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
2. CONTENTS
❑ Anatomy of Orbit
❑ Classification of fracture
❑ Pathophysiology
❑ Blow In fracture
❑ Blow Out Fracture – Pathophysiology & Clinical features.
❑ Superior Orbital Fissure Syndrome - Pathophysiology &
Clinical features.
❑ CLINICAL EVALUATION – Forced Duction Test
❑ Management
❑ Reconstruction
❑ Complications
3. Anatomy of Orbit
❑ Orbit – conical cavity
❑ Base – Anteriorly
❑ Apex – Directed at Optic Foramen.
❑ Orbital Volume – 30cc ; Globe – 7cc
❑ Bones – 7 (Maxillary, Zygomatic, Frontal, Ethmoid,
Lacrimal, Palatine, and Sphenoid )
❑ Four Walls – roof, lateral, medial, floor
❑ Medial wall & Floor – Thin
❑ Lateral wall & Roof – Stronger.
❑ Floor weakened – due to – Infraorbital canal passing
through it.
4. ❑ Medial wall – Formed by – Lamina Papyracea of Ethmoid bone
& Lacrimal bone.
❑ 6 EXTRAOCULAR MUSCLES –
❑ Originate from Common Tendinous fibrous ring
❑ Annulus of Zinn – Fibrous ring – Common origin of 4Recti Muscle
❑ OPTIC NERVE – direct extension of Gray Matter
❑ Very sensitive to Compression and once damaged fails to regenerate.
❑ SUPERIOR ORBITAL FISSURE
❑ Transmits – 3rd , 4th & 6th Cranial Nerves
❑ Opthalmic Division of 5th Cranial Nerve
Anatomy of Orbit
6. Floor Of Orbit
❑ Extends – Rim to approx.
2/3rd of the depth of orbit.
❑ Posteromedial aspect of Orbital
floor transits into Medial Orbital Wall
– to form
post. medial bulge.
❑ Only three of four orbital wall extend
into the apex – Medial, Superior,
Lateral.
❑ Ant. 3rd – Diameter widens
behind Superior Inferior margins
‘Post Entry Concavity’ –
Saggital plane – ‘Lazy S’ shape.
Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page 463- 464
7. Surface Landmarks –
❑ Infraorbital Groove –
bony sulcus
❑ Inferior Orbital Fissure
–converts into canal –
continues to Infraorbital
Foramen.
❑ Origin Of Inferior
Oblique Muscle
❑ Posterior Lateral
Promontry – Posterior
medial bulge ending in a
raised promontory shaped
lateral plateau.
Floor Of Orbit
8.
9. Medial wall
❑ Convex Rectangular shape
❑ Runs parallel to saggital
plane
❑ Medial wall – paper thin
Lamina paprycea
– (0.2-0.4mm)
Reinforced along the Maxillary-
Ethmoidal suture --- forming an
Internal Orbital Buttress.
Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page –465
10. Surface Landmarks
❖ Lacrimal Fossa –Anterior 1/3rd of MedialWall
❖ Frontoethmoidal suture – Roof of ethmoid sinus at the level of
cribriform plate.
❖ Ant. & Post. Ethmoidal Foramen – along frontoethmoidal suture in
midorbit.
❖ Distances :
❖ Medial orbital rim – Ant. Ethmoid Foramen – 22- 25mm
❖ Post. Ethmoid Foramen to Ant Ethmoid Foramen – 12mm
11. Lateral wall
❑ Forms – Triangle with an ant. base.
❑ Forms 450
angle to its medial wall
counterpart.
❑ SURFACE LANDMARKS –
❑ Whitnall’s tubercle – small bony
projection – actual insertion is
‘Common lateral retinaculum’.
❑ Superior & Inferior Orbital Fissure
❑ Zygomaticosphenoid suture –
imp. landmark to verify proper
reduction of complex ZMC fracture.
Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page –465
12. Roof Of Orbit
❑ Domed contour
❑ Comprised of – Orbital plate of Frontal Bone
❑ Extremely thin
❑ In apex – Roof terminates into Lesser Wing of Sphenoid
– where Oval shaped Optic foramen forms Optic canal.
❑ SURFACE LANDMARKS:
❑ Trochlear fossa : Zone of attachment of tendon of
Superior Oblique muscle
❑ Lacrimal Gland Fossa
13. Apex of Orbit
❑ Post. 3rd – made by sphenoid
bone
❑ Superior Orbital Fissure –
separates the Lesser wing of
sphenoid & Greater Wing of
Sphenoid
❑ Optic Canal – within the lesser
wing of sphenoid
❑ Annulus of Zinn – All Extraocular
muscles except Inf. Oblique arises
from this tendinous ring.
14.
15. Limit of Dissection
❑ Inferiorly(Floor) – Upto 28-30mm (safe limit) –optic canal
is at around 40mm.
❑ Laterally – Superior Orbital Fissure
❑ Superiorly – Orbital roof dissection is stopped at periorbital
surrounding Recurrent Meningeal Artery – passing through
bony canal within the Sphenofrontal suture line
❑ Medially – Posterior extent – Posterior Ethmoidal vessels,
running in the Fronto-Ethmoidal Suture line Anterior to Optic
foramen.
16. Distance of Vital Orbital Structures
from Bony landmark
STRUCTURE REFERENCE
LANDMARK
MEAN DISTANCE (mm)
Midpoint of
inferior orbital
fissure
Infraorbital foramen 24
Anterior
Ethmoidal
Foramen
Anterior Lacrimal crest 24
Superior Orbital Fissure Zygomaticofrontal
suture
35
Superior orbital Fissure Supraorbital Notch 40
Optic Canal
(medial aspect)
Anterior Lacrimal Crest 42
Optic Canal (Superior
Aspect)
Supraorbital notch 45
17. Pathophysiology of Orbital
Fractures
Trauma
Thick Rims protect the Eyeball
Absorb shock by Fracturing themselves
Orbital walls (especially Medial Wall & Floor) fracture
in an isolated way
Gets displaces Inwards or Outwards
Called as ‘Blow-In’ or ‘Blow-Out’fractures
18. Classification
ORBITAL WALL FRACTURES
1. Blow Out Fracture
2. Pure Blow Out Fracture
3. Impure Blow Out Fracture
4. Blow In Fracture
ISOLATED ORBITAL WALL FRACTURE :
1. Roof
2. Floor
3. Medial wall
4. Lateral wall
19. ❑ ISOLATED FRACTURES OF THE ORBITAL
RIM:
1. SUPERIOR RIM
2. INFERIOR RIM
3. MEDIAL RIM
4. LATERAL RIM
❑ COMPLEX COMMINUTED FRACTURES :
Nasoethmoidal and fronto-naso-orbital
fractures
20. ❑ PURE Blow Out OR Blow In –
Orbital Walls fracture in Isolation
❑ IMPURE Blow Out or Blow In Fracture –
Walls + Rim
22. Blow Out Fracture
❑ Term coined by – Smith and Regan – 1957
❑ First described by MacKenzie in 1844
❑ PATHOPHYSIOLOGY
❑ Buckling Theory –
❑ If a force strikes at any part of orbital rim,
❑ it will cause walls to undergo a rippling effect &
the force striking the rim will transfer to weaker
portion especially floor & cause them to distort
& eventually fracture
23. Pathophysiology of blow out fracture
of the orbit
❑ Hydraulic Theory (Pfeiffer in
1943) – A generalized increase
in orbital content pressure
results in direct compression and
fracture of the thin orbital bone.
❑ Therefore direct injury to globe
forcing it into the orbit is
necessary.
❑ Medial Wall & Floor –Thin &
Fragile
❑ Fracture readily – Provide
natural compensation.
24. Clinical Features
❑ Circumorbital Edema
❑ Subconjunctival Bleeding – due to fracture →
subperiosteal bleeding → escapes in subconjuctival
plane.
❑ Enopthalmous → Increase in size of Orbit →
Eyeball sinks
❑ Periorbital fat herniates through fractured walls-‘Hanging-
drop’Appearance
❑ Unilateral Epistaxis – bleeding into antrum
❑ Numbness in area of distribution of Infraorbital Nerve
❑ Diplopia or Vertical gage – Inferior Rectus or Inferior
Obliqus gets entrapped in fracture → Inability of eyeball
to move in vertical direction.
27. Pathophysiology
Raised Intraorbital Pressure (due to
Hematoma/Displaced fractured segments)
Compression of contents of Sup. Orbital Fissure
Paresis of Nerve
Neurological deficit in their distribution
28. ❑ Due to paresis of these nerves → all these
extraocular muscles undergo paralysis →
eyeball fails to move → ‘External
Opthalmoplegia’
❑ As affected eye does not move → whereas
contralateral normal eyeball moves → focal
axis gets disturbed → two images -→
Diplopia
29. Clinical Manifestations
❑ External Opthalmoplegia – Eyeball fails to move
❑ Diplopia – Two images of one object
❑ Internal Opthalmoplegia – Fixed Dilated pupils
(parasympathetic – III cranial nerve –
Occulomotor )
❑ Ptosis of Upper Eyelid – upper eyelid drops
down like a curtain – parasympathetic supply.
❑ Orbital Apex Syndrome – If Optic Nerve
involvement is present.
30. BLOW IN FRACTURE
Fragmented bones of the orbital floor are
displaced into the orbit.
Proptosis – Exopthalmous.
More commonly seen in fractures of orbital roof.
32. Forced Duction Test
❑ Prior to the test, a cotton-
tipped applicator soaked
with topical anesthetic
drops and held against
the limbus for few
minutes.
❑ Fine-toothed forceps
are then used to grasp
the conjunctiva and
Tenon’s capsule just
posterior to the limbus.
❑ The patient is then
asked to look in the
direction of restriction of
movement of the eye .
33. Snellen Chart
• To assess visual
acuity of the eye
Tonometer
• Measures IOP
• Greatly increased IOP
causes pulsatile optic
disk
Hess Chart
34. Opthalmoscopy
Examination of the fundus
TYPES:
Direct --- General examination
Indirect--- Better view of the eye
Pressure – eye ball – blunt instrument—edges
of fundus -- examination
Retinal detachment requires
ophthalmologic attention.
35. Hyphaema
❑ Layering of blood in the anterior chamber.
❑ Direct blow to eye --fist or squash ball
❑ C/F: Pain
Blurred vision
Photophobia
Visual acuity decreased
Pupil dilated
❑ Vision worse supine, clears upright
❑ Can cause increased IOP
36. ❑ Head of bed elevated
❑ Atropine –dilates pupil– prevents iris movements
❑ Mannitol
❑ Anti fibrinolytic agent 50mg/kg every 4 hrs reduces
recurrent bleeding.
37. IMAGING
❑ Caldwell view
❑ Waters view
❑ Lateral skull
❑ CT Scan – Orbit
- To visualize the fractured segments
- Also helps evaluate the Intraorbital volume.
By using a software to compare the normal
orbital volume to the affected.
❑ MRI
38. Imaging
Ultrasound
High resolution CT is used the
management of enophthalmos
Sinuscopy
3D reconstruction volumetric
assessment
43. SAGITAL SECTION THROUGH ORBIT &
GLOBE
C- Palpebral
Conjuntiva
IO- Inferior Oblique
muscle
IR- Inferior Rectus
Muscle
OO- Orbicularis Oculi
OS – Orbital Septum
P -
Periosteum/Periorbita TP-
Tarsal Plate.
44. Orbicularis Oris Muscle
Innervated by Cranial Nerve VII
Upper Eyelid – Levator
Palpebral Superioris – Cranial
Nerve III
Orbicularis Oculi -
Orbital and
Palpebral Portions
Palpebral Portion is divided into
fibers -
Pretarsal Portion
Preseptal Portion
45. Subciliary
Incision
The incision is approximately 2 mm below eyelashes, can be
extended laterally as necessary (top dashed line). It is made
through skin only.
46. Subcutaneous dissection of skin, leaving
pretarsal portion of orbicularis muscle
attached to tarsus. Dissection 4-6mm
inferiorly in this plane is adequate.
Subcutaneous dissection
through lid margin
47. Use scissors to dissect through orbicularis oculi muscle
over lateral orbital rim to identify periosteum.
48. Incision through the bridge of orbicularis
oculi muscle.
Saggital plane through orbit showing
incision of the bridge of orbicularis oculi
muscle.
49. ❑ Incision through periosteum
along ant. maxilla, 3 to 4 mm
inferior to infraorbital rim.
❑ Note the pretarsal muscle still
remaining on the inferior tarsus
and orbital septum, which restricts
orbital fat from entering field.
❑ Subperiosteal dissection of anterior
maxilla and orbital floor. Periosteal
elevator entering the orbit is
placed almost vertically as
dissection proceeds behind rim.
❑ In ant. region, floor of the orbit is at a
lower level than crest of the rim,
necessitating dissection inferiorly.
50. Sagital plane through orbit showing subperiosteal dissection of
the anterior maxilla and orbital floor.
51. Transconjuctival approach
•Fig.1 - Incision of conjuctiva below the tarsalplate.
•Fig 2 - Incision through periosteum. To facilitate this maneuver, a traction
suture is placed through the cut end of conjunctiva to retract the tissueand
maintain the position of corneal shield.
• Small retractors are placed so that lower lid is retracted to the level of ant.
surface of infraorbital rim.
• Intervening tissue along infraorbital rim is the periosteum. Theincision
is made through the periosteum just post. to infraorbitalrim.
52. •Saggital plane through the orbit and globe demonstrating
level and plane of incision. The conjunctiva and lower lid
are incised with scissors.
53. SUBPERIOSTEAL DISSECTION OF THE ORBITAL FLOOR.
Note the traction suture placed through the cut end of the
conjunctiva, which assists in retracting the conjunctiva and
maintains the corneal shield in place.
54. Surgical Exposure
❑ After periorbital
dissection is performed,
adequate exposure and
illumination of the
fractured area is
imperative.
❑ Malleable retractors,
spoons and special
orbital retractors
designed for the globe.
55. ❑ Transition between anterior
mid orbit , the orbital floor
slopes upwards giving rise to
posterior medial bulge &
slightly convex bony platform.
❑ Elevator passes transversely
along inferior orbital fissure.
❑ Infraorbital neurovascular
bundle can be visualized first
shining through the thin bony
roof of its canal.
❑ Then it becomes directly visible
in infraorbital groove.
56. EXTENT OF DISSECTION
❑ Taking into account extent of
fracture, the periorbital dissection
stops at medial border of the
inferior orbital fissure leaving
the soft tissue invagination intact.
❑ Laterally, the dissection is
continued to posterior edge of
the floor to orbital process of
the palatine bone. The suture
between the maxilla and the
palatine bone is indistinguishable
in the adult skull.
❑ Medially the periorbital
dissection extends to the zone
over the internal orbital
buttress.
57. ❑ For an EXTENDED ACCESS
to posterior orbital floor, the
contents of the inferior orbital
fissure must be addressed
and transected to allow for
this additional access.
❑ The transsection is prepared
with a dissection along the
inferior portion of the lateral
orbital wall in order to create a
second tunnel alongside the
vertical soft tissue.
58. ❑ The intervening soft tissue
invagination is transected in a
stepwise fashion using
bipolar electrocautery and
sharp dissection across the
fissure above the level of
Müller’s muscle, stripping
the periorbita along the
lateral edge of the inferior
orbital fissure.
59. Limit of Dissection
❑ Inferiorly – Upto 28-30mm.
❑ Laterally – Superior Orbital Fissure
❑ Superiorly – Orbital roof dissection is stopped
at periorbital surrounding Recurrent Meningeal
Artery – passing through bony canal within the
Sphenofrontal suture line
❑ Medially – Posterior extent – Posterior
Ethmoidal vessels , running in the Fronto-
Ethmoidal Suture line Anterior to Optic
foramen.
60. ❑ The subperiosteal dissection is
continued using a periosteal or
freer elevator in a lateromedial
direction and lifted up and
retracted by and by with the
ribbon retractor until the
entrance of apex is reached.
❑ Closure (layer-by-layer)
62. Autogenous Material
• Graft transferred from one part
of patient’s body to another
• Ex. –1. Septal cartilage
2. Ear cartilage
3. Maxillary wall bone
4. Iliac crest
5. Rib
6. Split calvaria
63. Bone Graft
Advantages:
1. Provides strength and stability to the orbital
floor
2. Relative resistance to infection
3. Lack of host response against the graft
4. Lack of concern for late extrusion
5. Long term efficacy and reliability
6. Can be easily accessed and harvested from
same operative area ( cartilage – ear, nose;
bone- maxilla, mandible )
64. Disadvantages :
1. Need for graft donor site
2. Increased operative time
3. Complications related to donor site wound
4. Donor site morbidity ( rib – pneumothorax,
infection; calvarial – dural tears,
subarachnoid haemorrhage, etc)
5. Unpredictable graft resorption
6. Limited ability to contour same type of bone
7. Limited availability
65. Allogenic Materials
Graft transferred from another
person (homogenic/ allogenic) or
from any animal of different
species (xenograft)
Ex. 1. Lyophilized dura
2. Lyophilized cartilage
3. Banked bone
Advantages:
1. No need for graft donor site
2. Decreases operative time
3. Eliminates complications
related to donor site wound
morbidity
4. Inertness
5. Resistant to resorption
68. Porous Polyethylene
Sheet (PPE)
Disadvantages :
• Not Radiopaque (Not visible on Post OP Radiographs)
•Lack of Rigidity when very thin wafer of PPE is used. When a
more thick rigid wafer is used there is a risk of causing
dystopia.
69. Polyethylene and titanium mesh
❑ By combining titanium mesh with
porous polyethylene –
❑ Material becomes radioopaque
❑ More rigid than porous PPE.
❑ ADVANTAGE :
❑ Stability
❑ Contouring
❑ Adequate in large three wall
fractures
❑ Radiopacity
❑ No Donor Site Needed
❑ DISADVANTAGES : Less
drainage from orbit.
70. Resorbable materials
❑ Thermoplastic and Non
thermoplastic materials.
❑ Advantages :
❑ Availability Handling/
❑ Contourability (only for
thermoplastics)
❑ Smooth surface and smooth edges
❑ Disadvantages:
❑ No radiopacity
❑ Degradation of material with possible
contour loss Sterile infection /
inflammatory response
❑ Difficult to shape according to patients
anatomy (only for non- thermoplastics)
❑ Less drainage from the orbit.
71. COMPLICATIONS
EARLY COMPLICATIONS :
1. Hemorrhagic or orbital hematoma – treated by
-lateral cathotomy immediately, lateral canthal
Tendonlysis ,
Lateral canthotomy – indicated when –
- Decreased visual acuity
- Intarocular pressure more than 40mmHg
- Proptosis
- Opthalmoplegia
72. Retrobulbar hemorrhage
❑ Rare, rapidly progressive
life threatening emergency
that results in accumulation
of blood in the retrobulbar
space
❑ Increased IOP → stretching
of the optic nerve &
blockage of ocular
perfusion
❑ Proptosis , marked
subconjuctival ecchymosis
& edema.
73. Symptoms– Pain , decreasing visual acuity, diplopia.
Treatment includes
• I.v mannitol(200 ml of 20% solution)
• i.v acetazolamide 500 mg
• i.v papaverine 40 mg
• i.v hydrocortisone 100 mg
• Dexamethasone sodium phosphate-3-4mg/kg body
weight followed by 1-3 mg /kg 6th hourly.
• If no evidence of improvement stop after 48 hours.
3. Blindness
74. OCULOCARDIAC REFLEX/ TRIGEMINOCARDIAC/
TRIGEMINOVAGAL REFLEX
- The oculocardiac reflex pathway begins with the
- afferent fibres of the long & short ciliary nerves that
travel with
- the opthalmic division of the trigeminal nerve to
- the gasserion ganglion via
- the sensory nucleus of the trigeminal nerve.
- In the floor of the 4th ventricle short internuncial
fibres in the reticular formation connect them with the
efferent pathway from the motor nucleus of the
vagus nerve to the depressor nerve ending in the
mucle tissue of the heart.
75. CLINICAL FEATURES –
- Bradycardia
- Faintness
- Further stimulation can lead to cardiac
dysrhythmias
- Atrioventricular blocks
- Asystole
- Bradycardia has been attributed to Trigeminal
derived vagal reflex
76. LATE COMPLICATIONS
❑ Altered vision
❑ Diplopia
❑ Ectropion – Lower eyelid turns outwards.
❑ Epiphora – Overflow of tears onto the face
❑ Enopthalmous
77. References
❑ Rowe & William’s Maxillofacial Injuries
❑ Maxillofacial Surgery-Peter Ward Booth
Vol-1
❑ Peterson’s Principles of Oral and
Maxillofacial Surgery ; 2nd Edition
❑ Textbook of Oral & Maxillofacial Surgery –
R.M. Borle