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Neuromuscular
Adaptations
By Jill Costley
:
• Understand the role of the brain and nervous system in
relation to motor control
• Understand the structure of a muscle fibre
• Appreciate the implications and effects of training on the
neuromuscular system
The Nervous System
 Central Nervous System (CNS ) – brain and spinal cord
 Peripheral Nervous System (PNS) – nerves and sensory organs
outside of the CNS – sensory input and motor input
 Autonomic Nervous System (ANS) (Sympathetic and
Parasympathetic Nervous System) – subconscious control
(heart, glands and hollow organs
 Somatic Nervous System (SoNS) –direct conscious control
Three main functions:
1) Collecting information from internal and
external environment (input)
2) Processing the information
3) Providing an output via glands or muscle
Wilmore et al., 2008; Bompa & Haff, 2009; France, 2010; Plowman & Smith, 2011
The Brain and Motor Control
1. The Cerebrum (right and left cortices) – the cerebral cortex is the conscious region
of the brain
2. The Diencephalon - thalamus: receives all sensory information
- hypothalamus: is the control centre for homeostasis
3. The Cerebellum: coordinating movement
4. The Brain Stem: connects brain to spinal cord and is the location of reticular
formation (many analgesics temporarily alter fibres within the brain stem)
5. The Basal Ganglia – repetitive muscle contraction e.g. running and postural control
Wilmore et al., 2008
The Neuron - Structure
• Size and shape depends on function and location
• Cell body/ soma = neuron control centre
• Dendrites run to cell body • The axon extends away
from cell body
France, 2010; Watkins, 2014
• Action potentials generated at axon hillock
The Axon
Two primary roles:
• Conduction of the electrical signal
• Neurotransmitter secretion after stimulated by an action potential
 Faster transmission of
action potential if the
axon has a larger in
diameter and if it is
myelinated (saltatory
conduction)
 Glial cells – support the mechanical
and metabolic processes of neurons
1) Oligodendrocytes - CNS
2) Schwann cells - PNS
Bompa & Haff, 2009; France, 2010; Watkins, 2014
Potentials of the Axon
• Resting potential (approximately -70 mV) – positively
charged outer (high Na+ ions concentration); negatively
charged inner (high K+ concentration)
Potential difference maintained through: Na+ less permeable; the
sodium-potassium pump of the neuron’s membrane (three Na+ out for
every two K+ in)
• Depolarization – membrane permeability increases causing Na+
to move in
Intracellular charge = more positive (closer to 0mV)
• Graded potential – localized depolarization - signal diminishes
down the axon – not very capable of signal conduction – does
not reach stimulus threshold ( approximately -55/-50 mV)
All-or-None Principle
Rhoades & Bell, 2012; Kenney et al., 2015
Action Potential
• Depolarization to repolarization (including hyperpolarization)
Absolute refractory period – incapable of signal
transmission
Relative refractory period – signal transmission only if
higher threshold level is met
Rhoades & Bell, 2012;
Kenney et al., 2015
The Motor Unit
• A motor unit: ‘’A motor neuron
and the fibres it innervates’’
McGinnis, 2005; Rhoades & Bell, 2012
1. Alpha neurons (α-motor neurons) –
extrafusal fibres
2. Gamma neurons (γ-motor neurons) –
intrafusal fibre
 Innervating smaller number of
fibres – more precision in smaller
muscles
 Innervating larger number of fibres
– force production in larger muscles
The Neuromuscular Junction
The junction located between a synaptic bulb of the motor neuron and the
muscle it innervates
Electrical signal  Chemical signal
 Mechanical Work
1. Arrival of pre-synaptic action potential
2. Calcium ions into pre-synaptic bulb
3. Acetylcholine (ACh) released into
synaptic cleft
4. Binds to nicotinic ACh receptors on
postjunctional folds of muscle
5. Post-synaptic membrane permeability
altered - Na+ moves into muscle cell; K+
ions moves out
6. ACh hydrolysed by acetylcholinesterase
(AChE) = acetate and choline
7. Acetate and Choline moves back into
pre-synaptic cell where reforms into
ACh (energy from mitochondria – ATP)
Wilmore et al., 2008; Rhoades & Bell, 2012; Kenney et al., 2015
Ensures only unidirectional motion
The Muscle Fibre - Structure
• Sarcolemma – polarised allowing for the muscle’s irritable characteristic
• Nuclei
• Sarcoplasm – cytoplasm of skeletal muscle
• Sarcoplasmic Reticulum (SR) – storage, release and uptake of calcium ions
• Transverse tubules (T-tubules) – enables the transfer of electrical signals
throughout muscle fibre
Baechle & Earle, 2008
Skeletal Muscle - Proprioceptors
Proprioceptors – sensory receptors within skeletal muscle that are found
within the joints, muscles and tendons
Augustine, 2008; Baechle & Earle, 2008
1. Muscle Spindles –
• Intrafusal fibres enclosed in a sheath of
connective tissue
• Muscle length sensitive
• Intrafusal fibres lengthen as extrafusal
fibres lengthen  transmits
information via gamma motoneurons
• Extrafusal fibre contraction (safety
mechanism)
• The Stretch Reflex: extreme/ rapid
stretch  all motor units of muscle
may be activated
• Force output enhancement as polar
ends of intrafusal can contract i.e. the
SSC
2. Golgi Tendon Organs –
• Sensory receptors encapsulated within a group of muscle
tendon fibres
• Sensitive to changes in tension
• Inhibitory response created – agonist inhibited; antagonist
activated
• Strength-power training theoretically may decrease
inhibitory response
Stone et al., 2007; Baechle & Earle, 2008
NeuromuscularAdaptationsto Strength Training
Strength: ‘’the ability of the neuromuscular system to produce force
against an external resistance’’ (Stone et al., 2007)
Neural Changes –
• Inter-muscular coordination & Cross-education
• Synergist activation & co-activation of antagonists
• Neural inhibition (spindles and GTOs)
• Motor unit recruitment
• Motor Unit rate coding
• Motor unit synchronisation
Morphological Changes
• Muscle fibre type
• Muscle Architecture
• Cross-sectional area (CSA)
• Fascicle Length
• Angle of Pennation
6-10 wks for neural; <10 wks for morphological changes)
Baechle & Earle, 2008; Stone et al, 2007; Cormie et al., 2011; French, 2015
Motor Unit Recruitment
Affected by the force exerted, contractile speed,
contraction type and metabolic state
Henneman’s Size Principle (muscle fibre recruitment
from the smallest to the largest)
refers to the amount of motor units stimulated to produce
muscular contraction; the more motor units recruited, the
higher the degree of force that is produced
Henneman et al., 1965; Bergh et al., 1977; Ford et al., 2000; Baechle & Earle, 2008; Bompa &
Haff, 2009
Motor Unit Rate Coding
 refers to the frequency/ rate at which a motor unit
innervates its muscle fibres.
• Increase force output by increasing firing rate of motor unit;
smaller muscles rely more on this variable
Baechle & Earle, 2008; Wilmore et al., 2008; Bompa & Haff, 2009
• A twitch – one single
stimulus from a motor unit
• Summation – more than two
successive stimuli
• Tetanus - continued
successive stimuli which
enables muscular tension to
build
Motor Unit Synchronisation
• Asynchronisation/ Synchronisation
• Motor Unit Asynchronisation – ‘’recycling’’ of
motor units during low-intensity muscular
efforts
• Motor Unit Synchronisation – many motor
units are stimulated simultaneously
• Increases in rate of force development (RFD)
Sale, 1987; Bompa & Haff, 2009
Inter-muscular coordination (hypothesised):
Optimal timing and magnitude of force production from
agonist, synergist and antagonistic muscles through:
• Greater activation of synergists
• Reduction of antagonist coactivation: still required for
joint stability, safety and movement coordination
• Reciprocal Inhibition
Neuromuscular Adaptations
Cross-Education
Strength improvements in untrained heteronymous
contralateral limb:
• Predominately neural
• Dynamic exercises more effective than isometric
• Electrical stimulation
Gabriel et al., 2006
Motor Unit Recruitment – Adaptations to:
1. Heavy resistance training -
• All fibres get larger (Size Principle); greater in type 2b
• More effective selective recruitment; Inhibition of
smaller muscle fibres by the CNS for recruitment of
larger fibres
• A possible reduction in the number of motor units
required to undertake the same movement (Ploutz et
al., 1994)
• Greater motor unit recruitment maintained at maximal
force output
2. Aerobic training –
• Increased Type 1 muscle fibre motor unit recruitment
Sale, 1987; Ploutz et al., 1994; Baechle & Earle, 2008
Motor Unit Synchronisation - Adaptations to:
1. Strength training –
• more effective at motor unit synchronisation: may have
little effect on maximal force development
• Weightlifters have an enhanced ability to
synchronise motor units
2. Aerobic training –
• Greater asynchronisation ability; delays fatigue
Motor Unit Rate Coding - Adaptations to:
1. Strength training –
• Increase in firing rate: increases force and power
production
• Ballistic movements can enhance firing rate: enhances
(RFD)
Sale, 1987; Ploutz et al., 1994; Baechle & Earle, 2008; Cormie et al., 2011
Cross-SectionalArea(CSA)ofMuscle
Hypertrophy:
An increase in the cross-sectional area of muscle which is
thought to enhance maximal strength output
Resistance training –
• Transient hypertrophy – short term; occurs after a single
bout of training
• Chronic hypertrophy – long term; an absolute increase in
contractile units due to greater size (hypertrophy) and/ or
number (hyperplasia) of muscle fibres
Wilmore et al., 2008; Bompa & Haff, 2009
Morphological Adaptations
Folland & Williams, 2007; Baechle & Earle, 2008; Wilmore et al., 2008; Cormie et al., 2011; French,
2015
Muscle Fibre Type - Adaptations to:
1. Strength training –
• Conversion
• Reduction of Type IIb that increases IIa isoform
2. Combined (aerobic and resistance) – an almost complete
conversion of IIx (IIb) to IIa
3. Detraining – IIa to IIx (IIb)
Muscle Architecture - Adaptations to:
1. Strength training –
• Hypertrophy (previously described)– contractile unit increase
• Increase in force output
• Explosive exercises - increase Type II (specifically IIb) fibres
that changes CSA ratio between I and II
2. Detraining – Type 11b show greater atrophic response
Angle of Pennation - Adaptations to:
1. Strength training –
• increases fibre pennation in some muscles (Aagaard et
al., 2001; vastus lateralis) – possibly relates to
hypertrophy
2. Sprint training –
• Decrease the angle of pennation
Fascicle Length - Adaptations to:
1. Strength training –
• Increased fascicle length – velocity and power output;
sprinters show greater vastus lateralis and gastrocnemius
fascicle length compared to long distance runners (a
genetic predisposition??**)
Aagaard et al., 2001; Baechle & Earle, 2008; Wilmore et al., 2008; Cormie et al., 2011; French, 2015
NMJ - Adaptations to:
1. Strength training –
• no real impact on performance
2. Aerobic training –
• no real impact on performance
Aagaard et al., 2000; Stone et al, 2007 ; Baechle & Earle, 2008; Wilmore et al., 2008; Bompa &
Haff, 2009 Cormie et al., 2011; French, 2015
Proprioceptors - Adaptations to:
1. Strength training:
• A reduction or alteration in the inhibitory response may
allow a higher percentage of maximal strength potential
to be realised
 Majority of sports benefit from
development of power over a wide
range of loads (inc. unloaded): power
output maximized at or near the load
being trained
 Mixed-method approach for
developing maximal power
output: developments within
the whole force-velocity curve
Cormie et al, 2011; Haff & Nimphius, 2012
 Strength before power in beginners
 As athlete becomes more
advanced – do not ignore
strength!!
Summary –
• Many regions of the brain are involved in motor control
• The axon is the electrical conduction unit of the nervous
system
• The transmission of electrical energy into chemical energy to
create mechanical movement
• Strength training - neuromuscular adaptations: increase of
MU activation, activation of synergists, coactivation of
antagonists, transition of type IIb fibres to IIa and of many
muscular components
• Aerobic training – neuromuscular adaptations: increased
asynchronisation and Type I fibre recruitment
How this has impacted my practice
as a Placement S&C coach
1. Considering the sport to potential adaptations
found e.g. sprinting decreasing fascicle length
2. Developing a greater understanding of the
application of force, power and velocity
3. Studying programmes others have written
1. Cerebral Palsy, oligodendrocytes and the
impact on movement – the why, what,
and how
2. Biochemistry of the neuromuscular system
– a greater and more in-depth known of
the role of neurotransmitters
I would like to advance my knowledge in….
The End -
Thanks for Listening

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Neuromuscular Adaptations to Sports Training

  • 2. : • Understand the role of the brain and nervous system in relation to motor control • Understand the structure of a muscle fibre • Appreciate the implications and effects of training on the neuromuscular system
  • 3. The Nervous System  Central Nervous System (CNS ) – brain and spinal cord  Peripheral Nervous System (PNS) – nerves and sensory organs outside of the CNS – sensory input and motor input  Autonomic Nervous System (ANS) (Sympathetic and Parasympathetic Nervous System) – subconscious control (heart, glands and hollow organs  Somatic Nervous System (SoNS) –direct conscious control Three main functions: 1) Collecting information from internal and external environment (input) 2) Processing the information 3) Providing an output via glands or muscle Wilmore et al., 2008; Bompa & Haff, 2009; France, 2010; Plowman & Smith, 2011
  • 4. The Brain and Motor Control 1. The Cerebrum (right and left cortices) – the cerebral cortex is the conscious region of the brain 2. The Diencephalon - thalamus: receives all sensory information - hypothalamus: is the control centre for homeostasis 3. The Cerebellum: coordinating movement 4. The Brain Stem: connects brain to spinal cord and is the location of reticular formation (many analgesics temporarily alter fibres within the brain stem) 5. The Basal Ganglia – repetitive muscle contraction e.g. running and postural control Wilmore et al., 2008
  • 5. The Neuron - Structure • Size and shape depends on function and location • Cell body/ soma = neuron control centre • Dendrites run to cell body • The axon extends away from cell body France, 2010; Watkins, 2014 • Action potentials generated at axon hillock
  • 6. The Axon Two primary roles: • Conduction of the electrical signal • Neurotransmitter secretion after stimulated by an action potential  Faster transmission of action potential if the axon has a larger in diameter and if it is myelinated (saltatory conduction)  Glial cells – support the mechanical and metabolic processes of neurons 1) Oligodendrocytes - CNS 2) Schwann cells - PNS Bompa & Haff, 2009; France, 2010; Watkins, 2014
  • 7. Potentials of the Axon • Resting potential (approximately -70 mV) – positively charged outer (high Na+ ions concentration); negatively charged inner (high K+ concentration) Potential difference maintained through: Na+ less permeable; the sodium-potassium pump of the neuron’s membrane (three Na+ out for every two K+ in) • Depolarization – membrane permeability increases causing Na+ to move in Intracellular charge = more positive (closer to 0mV) • Graded potential – localized depolarization - signal diminishes down the axon – not very capable of signal conduction – does not reach stimulus threshold ( approximately -55/-50 mV) All-or-None Principle Rhoades & Bell, 2012; Kenney et al., 2015
  • 8. Action Potential • Depolarization to repolarization (including hyperpolarization) Absolute refractory period – incapable of signal transmission Relative refractory period – signal transmission only if higher threshold level is met Rhoades & Bell, 2012; Kenney et al., 2015
  • 9. The Motor Unit • A motor unit: ‘’A motor neuron and the fibres it innervates’’ McGinnis, 2005; Rhoades & Bell, 2012 1. Alpha neurons (α-motor neurons) – extrafusal fibres 2. Gamma neurons (γ-motor neurons) – intrafusal fibre  Innervating smaller number of fibres – more precision in smaller muscles  Innervating larger number of fibres – force production in larger muscles
  • 10. The Neuromuscular Junction The junction located between a synaptic bulb of the motor neuron and the muscle it innervates Electrical signal  Chemical signal  Mechanical Work 1. Arrival of pre-synaptic action potential 2. Calcium ions into pre-synaptic bulb 3. Acetylcholine (ACh) released into synaptic cleft 4. Binds to nicotinic ACh receptors on postjunctional folds of muscle 5. Post-synaptic membrane permeability altered - Na+ moves into muscle cell; K+ ions moves out 6. ACh hydrolysed by acetylcholinesterase (AChE) = acetate and choline 7. Acetate and Choline moves back into pre-synaptic cell where reforms into ACh (energy from mitochondria – ATP) Wilmore et al., 2008; Rhoades & Bell, 2012; Kenney et al., 2015 Ensures only unidirectional motion
  • 11. The Muscle Fibre - Structure • Sarcolemma – polarised allowing for the muscle’s irritable characteristic • Nuclei • Sarcoplasm – cytoplasm of skeletal muscle • Sarcoplasmic Reticulum (SR) – storage, release and uptake of calcium ions • Transverse tubules (T-tubules) – enables the transfer of electrical signals throughout muscle fibre Baechle & Earle, 2008
  • 12.
  • 13. Skeletal Muscle - Proprioceptors Proprioceptors – sensory receptors within skeletal muscle that are found within the joints, muscles and tendons Augustine, 2008; Baechle & Earle, 2008 1. Muscle Spindles – • Intrafusal fibres enclosed in a sheath of connective tissue • Muscle length sensitive • Intrafusal fibres lengthen as extrafusal fibres lengthen  transmits information via gamma motoneurons • Extrafusal fibre contraction (safety mechanism) • The Stretch Reflex: extreme/ rapid stretch  all motor units of muscle may be activated • Force output enhancement as polar ends of intrafusal can contract i.e. the SSC
  • 14. 2. Golgi Tendon Organs – • Sensory receptors encapsulated within a group of muscle tendon fibres • Sensitive to changes in tension • Inhibitory response created – agonist inhibited; antagonist activated • Strength-power training theoretically may decrease inhibitory response Stone et al., 2007; Baechle & Earle, 2008
  • 15. NeuromuscularAdaptationsto Strength Training Strength: ‘’the ability of the neuromuscular system to produce force against an external resistance’’ (Stone et al., 2007) Neural Changes – • Inter-muscular coordination & Cross-education • Synergist activation & co-activation of antagonists • Neural inhibition (spindles and GTOs) • Motor unit recruitment • Motor Unit rate coding • Motor unit synchronisation Morphological Changes • Muscle fibre type • Muscle Architecture • Cross-sectional area (CSA) • Fascicle Length • Angle of Pennation 6-10 wks for neural; <10 wks for morphological changes) Baechle & Earle, 2008; Stone et al, 2007; Cormie et al., 2011; French, 2015
  • 16. Motor Unit Recruitment Affected by the force exerted, contractile speed, contraction type and metabolic state Henneman’s Size Principle (muscle fibre recruitment from the smallest to the largest) refers to the amount of motor units stimulated to produce muscular contraction; the more motor units recruited, the higher the degree of force that is produced Henneman et al., 1965; Bergh et al., 1977; Ford et al., 2000; Baechle & Earle, 2008; Bompa & Haff, 2009
  • 17. Motor Unit Rate Coding  refers to the frequency/ rate at which a motor unit innervates its muscle fibres. • Increase force output by increasing firing rate of motor unit; smaller muscles rely more on this variable Baechle & Earle, 2008; Wilmore et al., 2008; Bompa & Haff, 2009 • A twitch – one single stimulus from a motor unit • Summation – more than two successive stimuli • Tetanus - continued successive stimuli which enables muscular tension to build
  • 18. Motor Unit Synchronisation • Asynchronisation/ Synchronisation • Motor Unit Asynchronisation – ‘’recycling’’ of motor units during low-intensity muscular efforts • Motor Unit Synchronisation – many motor units are stimulated simultaneously • Increases in rate of force development (RFD) Sale, 1987; Bompa & Haff, 2009
  • 19. Inter-muscular coordination (hypothesised): Optimal timing and magnitude of force production from agonist, synergist and antagonistic muscles through: • Greater activation of synergists • Reduction of antagonist coactivation: still required for joint stability, safety and movement coordination • Reciprocal Inhibition Neuromuscular Adaptations Cross-Education Strength improvements in untrained heteronymous contralateral limb: • Predominately neural • Dynamic exercises more effective than isometric • Electrical stimulation Gabriel et al., 2006
  • 20. Motor Unit Recruitment – Adaptations to: 1. Heavy resistance training - • All fibres get larger (Size Principle); greater in type 2b • More effective selective recruitment; Inhibition of smaller muscle fibres by the CNS for recruitment of larger fibres • A possible reduction in the number of motor units required to undertake the same movement (Ploutz et al., 1994) • Greater motor unit recruitment maintained at maximal force output 2. Aerobic training – • Increased Type 1 muscle fibre motor unit recruitment Sale, 1987; Ploutz et al., 1994; Baechle & Earle, 2008
  • 21. Motor Unit Synchronisation - Adaptations to: 1. Strength training – • more effective at motor unit synchronisation: may have little effect on maximal force development • Weightlifters have an enhanced ability to synchronise motor units 2. Aerobic training – • Greater asynchronisation ability; delays fatigue Motor Unit Rate Coding - Adaptations to: 1. Strength training – • Increase in firing rate: increases force and power production • Ballistic movements can enhance firing rate: enhances (RFD) Sale, 1987; Ploutz et al., 1994; Baechle & Earle, 2008; Cormie et al., 2011
  • 22. Cross-SectionalArea(CSA)ofMuscle Hypertrophy: An increase in the cross-sectional area of muscle which is thought to enhance maximal strength output Resistance training – • Transient hypertrophy – short term; occurs after a single bout of training • Chronic hypertrophy – long term; an absolute increase in contractile units due to greater size (hypertrophy) and/ or number (hyperplasia) of muscle fibres Wilmore et al., 2008; Bompa & Haff, 2009 Morphological Adaptations
  • 23.
  • 24. Folland & Williams, 2007; Baechle & Earle, 2008; Wilmore et al., 2008; Cormie et al., 2011; French, 2015 Muscle Fibre Type - Adaptations to: 1. Strength training – • Conversion • Reduction of Type IIb that increases IIa isoform 2. Combined (aerobic and resistance) – an almost complete conversion of IIx (IIb) to IIa 3. Detraining – IIa to IIx (IIb) Muscle Architecture - Adaptations to: 1. Strength training – • Hypertrophy (previously described)– contractile unit increase • Increase in force output • Explosive exercises - increase Type II (specifically IIb) fibres that changes CSA ratio between I and II 2. Detraining – Type 11b show greater atrophic response
  • 25. Angle of Pennation - Adaptations to: 1. Strength training – • increases fibre pennation in some muscles (Aagaard et al., 2001; vastus lateralis) – possibly relates to hypertrophy 2. Sprint training – • Decrease the angle of pennation Fascicle Length - Adaptations to: 1. Strength training – • Increased fascicle length – velocity and power output; sprinters show greater vastus lateralis and gastrocnemius fascicle length compared to long distance runners (a genetic predisposition??**) Aagaard et al., 2001; Baechle & Earle, 2008; Wilmore et al., 2008; Cormie et al., 2011; French, 2015
  • 26. NMJ - Adaptations to: 1. Strength training – • no real impact on performance 2. Aerobic training – • no real impact on performance Aagaard et al., 2000; Stone et al, 2007 ; Baechle & Earle, 2008; Wilmore et al., 2008; Bompa & Haff, 2009 Cormie et al., 2011; French, 2015 Proprioceptors - Adaptations to: 1. Strength training: • A reduction or alteration in the inhibitory response may allow a higher percentage of maximal strength potential to be realised
  • 27.  Majority of sports benefit from development of power over a wide range of loads (inc. unloaded): power output maximized at or near the load being trained  Mixed-method approach for developing maximal power output: developments within the whole force-velocity curve Cormie et al, 2011; Haff & Nimphius, 2012  Strength before power in beginners  As athlete becomes more advanced – do not ignore strength!!
  • 28. Summary – • Many regions of the brain are involved in motor control • The axon is the electrical conduction unit of the nervous system • The transmission of electrical energy into chemical energy to create mechanical movement • Strength training - neuromuscular adaptations: increase of MU activation, activation of synergists, coactivation of antagonists, transition of type IIb fibres to IIa and of many muscular components • Aerobic training – neuromuscular adaptations: increased asynchronisation and Type I fibre recruitment
  • 29. How this has impacted my practice as a Placement S&C coach 1. Considering the sport to potential adaptations found e.g. sprinting decreasing fascicle length 2. Developing a greater understanding of the application of force, power and velocity 3. Studying programmes others have written
  • 30. 1. Cerebral Palsy, oligodendrocytes and the impact on movement – the why, what, and how 2. Biochemistry of the neuromuscular system – a greater and more in-depth known of the role of neurotransmitters I would like to advance my knowledge in….
  • 31. The End - Thanks for Listening

Editor's Notes

  1. Hello
  2. Brain Muscle Effects
  3. Collect Process Produce Neurons CNS & PNS ANS & Somatic
  4. Cerebrum – cerebral cortex Diencephalon Cerebellum Brain Stem – reticular formation Spinal cord Basal ganglia
  5. Size Consideration Dendrites Threshold Split Axon terminals
  6. Axon Diameter Myelin Glial
  7. Contraction Potential Rest Maintained Depolarization Fates 0mV Transmission Graded All-or-none
  8. Action Influx Refractory Uni Synapse
  9. Motor unit Gamma Alpha Fine Force
  10. Junction Electrical/Chemical ACh (axo) Ca2+ 2 ACh Na+/K+ Resting
  11. Myofibril – myofilament (actin and myosin) Sarcolemma Propagation Nuclei Sarcoplasm - ^myoglobin ^glycogen SR Transverse tubules
  12. Calcium release – overview
  13. Proprioreceptors Sensory receptors Spindle – length composed extrafusal stretch stretch reflex degree - *speed
  14. GTO – tension Antagonist activation Composed of Decrease inhibitory
  15. Strength def Neural/morph Neural: 6-10 wks Morph e.g. hypertrophy – greater than 10 wks
  16. Recruitment **larger muscles** More = force inc Size principle smaller more sensitive
  17. Rate coding **smaller** Inc force with frequency Smallest electrical – twitch Summate Tetanus - tension
  18. Asynchronisation Lower intensity Recycle Synchronous RFD
  19. Hypothesised Animal/case Keep coactivation Reciprocal inhibition Neural* and morphological Electrical stimulation
  20. Recruitment adaptations Hypertrophy Selective recruitment MU required decreases Max force output – plutes – 9 – quads Aerobic – type 1
  21. Rate coding adaptations Strength – increases firing inc in force and power Ballistic – inc in RFD Synchro adaptations Strength – more effective weightlifters enhanced coord Aerobic - asynchro – delays fatigue
  22. Hypertrophy def Inc in CSA Strength – transient/ chronic (plus hyperplasia)
  23. Muscular adaptations summary – strength Mito and capillarisation dec – only relative Buffering inc Supercompensation effect Glycogen Enzyme
  24. Muscle fibre type Power and strength ahtletes – higher 2 – Olympic weightlifting – higher IIb able to lift greater weights and better at vertical jump Endurance – more type I – lower maximal force outputs Combined training – almost full conversion Architecture – IIb – hypertrophic and atrophic
  25. Longer fascicle – inc velocity and power output – eccentric & high velocity sprinters: longer gastro and v.l compared to endurance runners – inc max shortening velocity conflicting - genetic predisposition; chicken or egg?) Pennation – strength – inc pennation sprint – dec pennation (velocity rather than force?)
  26. NMJ – no real influence on performane total NMJ area, nerve terminal branch length, end plate perimeter length and area, and increased distance between each of the acetylcholine receptors. Proprioceptors – reduced inhibitory response
  27. maximal power : use a mixed-method approach It has also been suggested that development of maximal power output has been shown to be more effective over a variety of loads (unloaded and loaded at different loads) – this will enable developments within the whole force-velocity curve – this can be achieved through using sequential periodization (focusing on one variable of training – Long linear (other areas detrain; 4 weeks too long; okay for beginners), short linear (okay for mediocre athletes; 1 week; not enough stimulus of focus for elite), long undulated (waves; not a great periodization as high intensities too quickly for beginners; focuses on only one variable for elite), short undulated (shorter periods prevent detraining etc; more suitable to mediocre athletes; probably the best) - or completing submaximal loads during warm-up at a fast rate. It has also been Depends on athlete and situation
  28. Brain works in unison with body Axon – main conduction unit Electical to chemical to mechanical Strength training – MU and synergist activation, coactivation of antagonists, transition of type IIb fibres to IIa and of many muscular components Aerobic – asynchronisation, type 1 recruitnent
  29. From this presentation I have gained a better insight into the fact that specific exercise types are required to enhance certain aspects: you train to get specific adaptive responses that then produce the improvements in performance. I have developed a better understanding of the role that strength has with power and velocity how to tailor force-velocity training to get the most out of what you are trying to achieve and believe that I will now approach the thought of programming in a different way – think about what an athlete needs developed.
  30. During this presentation I have looked very briefly into cerebral palsy and what causes it. I was talking to …earlier in the week about his CP but he didn’t actually know very much about his condition; although I am not a doctor I feel that it would be interesting to look into the condition finding out why it happens, what are the consequences and effects upon training and how it affects his progression in sport…are there the same motor unit responses to that of unaffected individuals? Another area that I would to advance my knowledge in is the biochemistry involved in the neuromuscular system. The process that occurs at the neuromuscular junctions with the acetylcholine binding to the nicotinic acetylcholine junctions along with it’s hydrolysis into acetate and choline interests me a lot.
  31. Reference provided upon request via email