This document discusses mental retardation and learning disabilities. It defines mental retardation as significantly below average intellectual functioning and deficits in adaptive skills. It classifies the severity of mental retardation into four levels based on IQ scores: mild, moderate, severe, and profound. Learning disabilities are defined as disorders that cause a discrepancy between academic performance and intellectual ability in one or more areas such as language processing. The document discusses the prevalence, causes, characteristics, and management of both conditions.
mental retardation power point presentationjagan _jaggi
Intellectual disability (ID), once called mental retardation, is characterized by below-average intelligence or mental ability and a lack of skills necessary for day-to-day living. People with intellectual disabilities can and do learn new skills, but they learn them more slowly.
The DSM-IV definition utilizes four degrees of severity that reflect the level of intellectual impairment: IQ levels between 50–55 to approximately 70 characterize mild mental retardation, 35—40 to 50–55 characterize moderate mental retardation, 20–25 to 35–40 characterize severe mental retardation,
Challenged children are one who deviated from normal health status either physically, mentally or socially and requires special care, treatment and education.
Definition
Children with special health care needs are those who have or are at increased risk for a chronic physical, developmental, behavioral, or emotional condition and who also require health and related services of a type or amount beyond that required by children generally.” (McPherson,1998)
this ppt is used for presentation in public flora as well as for doctors.this is not for commercial purpose. it is only for educating.if any unwarranted mistakes are present please forgive me
mental retardation power point presentationjagan _jaggi
Intellectual disability (ID), once called mental retardation, is characterized by below-average intelligence or mental ability and a lack of skills necessary for day-to-day living. People with intellectual disabilities can and do learn new skills, but they learn them more slowly.
The DSM-IV definition utilizes four degrees of severity that reflect the level of intellectual impairment: IQ levels between 50–55 to approximately 70 characterize mild mental retardation, 35—40 to 50–55 characterize moderate mental retardation, 20–25 to 35–40 characterize severe mental retardation,
Challenged children are one who deviated from normal health status either physically, mentally or socially and requires special care, treatment and education.
Definition
Children with special health care needs are those who have or are at increased risk for a chronic physical, developmental, behavioral, or emotional condition and who also require health and related services of a type or amount beyond that required by children generally.” (McPherson,1998)
this ppt is used for presentation in public flora as well as for doctors.this is not for commercial purpose. it is only for educating.if any unwarranted mistakes are present please forgive me
Mental retardation{intellectual disability} is a condition of arrested or incomplete development of mind, which is specially characterized by impairment of skills manifested during the developmental period, which contribute to the overall level of intelligence, i.e cognitive, language, motor and social abilities.
Prevalence of mental disorders is 4-5 times higher in person with intellectual disability
CAUSES-
GENETIC
ENVIORNMENTAL/SOCIO-CULTURAL
PRENATAL,PERINATAL AND POSTNATAL FACTOR
COMBINED Behavior management
Monitoring the child’s development needs & problems.
Programs that maximize speech, language, cognitive, psychomotor, social, self-care, & occupational skills.
Ongoing evaluation for overlapping psychiatric disorders, such as depression, bipolar disorder, & ADHD.
Family therapy to help parents develop coping skills & deal with guilt or anger.
Provide day schools to train the child in basic skills, such as bathing & feeding.
Topic: Learning Disabilities & Disorder
Student Name: Anila Parveen
Class: M.Ed.
Project Name: “Young Teachers' Professional Development (TPD)"
"Project Founder: Prof. Dr. Amjad Ali Arain
Faculty of Education, University of Sindh, Pakistan
Developmental delay is the spectrum of problems encompassing delay in the cognitive, social, emotional, sexual and physical developmental skills. This presentation briefs the Cognitive developmental delay
Nutrition in New born and Kids
Calorie requirement of newborn and growing kids
Protein energy malnutrition
Vitamin deficiency disorders in kids
Ricketts
Scurvy
Kwashiorkor
Marasmus
The limbic system, also known as the paleomammalian cortex, is a set of brain structures located on both sides of the thalamus, immediately beneath the medial temporal lobe of the cerebrum primarily in the forebrain.[1]
It supports a variety of functions including emotion, behavior, motivation, long-term memory, and olfaction.[2] Emotional life is largely housed in the limbic system, and it critically aids the formation of memories.
With a primordial structure, the limbic system is involved in lower order emotional processing of input from sensory systems
This PPT describes neurological gait deviations.
It describes Hemiplegic/circumductory gait, Spastic Diplegic gait, Parkinson gait, Myopathic & Ataxic gait in detail along with its causes and management in with Physiotherapy treatment. detail
This PPT is made to explain basic techniques of therapeutic massage in detail.
It includes : Stroking, Pressure Manipulation, Tapotement & Vibration
Physiological effects, Therapeutic uses, Caution & Contraindications.
Description : Osteogenesis Imperfecta/
Brittle bone disease :
It is disorder of type I collagen synthesis that affects all connective tissue in the body.
Musculoskeletal involvement is diffuse and includes osteoporosis with excessive fracture even at birth, bowing of long bone, spinal deformities, muscle weakness and ligamentous laxity.
Key words :
Osteogenesis Imperfecta, Brittle bone disease, Genetic disorder, Pathophysiology, Types of OI, Denetinogenesis Imperfecta, Bluish sclera, Frequent fractures, fractures, Hearing loss, Management, orthopedic, Rehabilitation
Physiotherapy, pediatrics, physiotherapist, pediatric orthopedic surgery.
Arthrogryposis multiplex congenita is a disorder that affects the early development of body joints in a fetus, most commonly the large joints in the arms and legs. An infant who is born with the condition typically has limited mobility and obvious physical deformities in one or more joints.
1. Scar Tissue
2.What makes a scar
3. Characteristics of Scar
4. Scar & Adhesion
5. Classification as External and Internal scars
6 Detriments/Disadvantages
7. How scar tissue forms in muscle
8. Phases of scar healing
9.Phases of wound healing
10. Release of scar and adhesion
11. Massage
12. Massage techniques
13. Conditions : Burns & Skin grafting
14. Cautions
15. Procedure
16. When to start massage after surgery
17. How to massage scar tissue
18. Effleurage, Kneading, Skin rolling and Friction
19. Caution
20. Keloid
21. Warning.
What is oedema?
types of oedema
1. soft and mobile
2. consolidated/endurated
Conditions in which oedema is treated with massage
a) Radical mastectomy
b) Venous ulcer
c) gravitational/paralytic oedema
Technique of massage to reduce oedema
Kneading
Effleurage
Picking Up &
Friction
Instructions to patient
Position of Patient
Aim
Procedure &
Steps of Massage Application
Diabetes Mellitus
Introduction
Pathophysiology
Types of Diabetes Mellitus
Type 1, 2 and
gestational diabetes
rescent research in Type 1 diabetes
Risk factors and causes
Complications short term and long term of diabetes
Management
Treatment with Insulin
Diabetic drugs
Healthy Diet
Exercises prescription
aerobic exercises,
resistance exercises and
flexibility
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
2. TERMINOLOGY AND DEFINITIONS
Mental Retardation:
• Significantly sub-average intellectual functioning and concurrent
deficits in adaptive functioning.
• Severity varies considerably from individual to individual.
Developmental Disability or Delay:
• A more general term that refers to significant deficits or delays
in various areas of development.
• Areas of development include: cognitive, motor, social, physical,
emotional.
3. DEFICITS IN INTELLECTUAL
FUNCTIONING
• Intellectual functioning is assessed using IQ
tests.
• IQ scores are distributed normally, have a
mean of 100 and a SD of 15.
• Child’s performance is compared to that of
other children his/her age, and an IQ score is
given.
• How do we know when a score is below
average?
4.
5. DEFICITS IN ADAPTIVE
FUNCTIONING
• What is meant by adaptive functioning?
• DSM-IV diagnostic criteria require deficits in
at least 2 of the following areas:
• communication, self-care, home living,
social/interpersonal skills, use of community
resources, self-direction, functional academic skills,
work, leisure, health, and safety.
6. MR - MILD SEVERITY
• IQs ranging from about 70 to 55
• The largest segment of those with the disorder (about 85%)
• Social and communication skills develop during preschool,
minimal impairment in sensory and motor skills.
• Hard to diagnose during the preschool years.
• General achievement ranges from second to fifth grade.
• Can live successfully in the community with appropriate support.
7. MR - MODERATE SEVERITY
• IQs ranging from about 55 to 40.
• About 10% of the entire population of MR.
• Communication skills develop during early childhood.
• Can benefit from vocational training and can attend to
personal care with some supervision.
• Unlikely to progress beyond second grade level in
academic subjects.
• Can live successfully in the community, completing
unskilled or semiskilled work.
8. MR - SEVERE SEVERITY
• IQs from about 40 to 25.
• Constitutes 3-4% of the entire population of MR.
• Can learn basic personal care skills.
• academically, may achieve basic familiarity with the
alphabet, simple counting, and may learn to sight read
some basic words.
• can live successfully in the community under supervision,
with their families or in group homes.
9. MR - PROFOUND SEVERITY
• IQs less than about 25.
• Only 1-2% of persons with MR.
• Most have an identified neurological condition that
accounts for MR.
• Demonstrate considerable impairments in sensory and
motor development early on.
• Require constant support and supervision.
• With intensive support, may demonstrate improvements
in motor, self-care and communication.
10. MR - PREVALENCE AND ETIOLOGY
Prevalence:
• Theoretical = 2.28%
• Actual prevalence = as many as 3%
Causes:
• Can only be specified in 25% of children with mild MR.
• Cultural-familial
• Organic
• Prenatal, perinatal, postnatal
11. L’ARCHE MODEL OF “TREATMENT”
• L’arche provides homes for individuals with MR and the people
who assist them.
• Founded by Jean Vanier in France in the 1960s.
Philosophy:
• All people have basic needs for relationship, agency, and for
being needed by others.
• Individuals with MR have unique gifts to share with others.
• Well-being results when the needs stated above are met.
13. LEARNING DISABILITIES
“Learning disabilities are disorders in one or more of the processes
involved in understanding or using symbols of spoken language. The
disorders result in a significant discrepancy between academic
performance and assessed intellectual ability, with deficits in at least
one of the following areas: receptive language — listening, reading;
language processing — thinking, conceptualizing, integrating;
expressive language — talking, spelling, writing, and mathematical
computations. Such deficits become evident in both academic and
social situations. The definition does not include children who have
learning problems that are primarily the result of hearing, motor
handicaps, mental retardation, primary emotional disturbance, or
environmental, cultural, or economic disadvantage”. (Ontario, Ministry
of Education, 1980)
14. SEVEN COMMON ELEMENTS OF
LD DEFINITIONS
• difficulties in academic and learning tasks
• discrepancy between potential and performance
• assumption of at least average intelligence
• exclusion of other causes
• uneven growth pattern
• disorder in one or more basic cognitive processes
(memory, auditory, visual)
• soft-signs of neurological dysfunction (clumsiness)
16. IMPACT OF LD ON SCHOOL/LIFE
FUNCTIONING
• Often not identified until around Grade 3
• Effect of LD on work habits
• Communication difficulties associated with LD
• Social difficulties associated with LD
17. DISORDERS IN COGNITIVE PROCESSES
ASSOCIATED WITH LD
Basic cognitive processes/skills:
• allow learning to take place
• 4 general ‘steps’: input, integration, memory, output
Input difficulties:
• visual
• auditory
18. DISORDERS IN COGNITIVE PROCESSES
ASSOCIATED WITH LD
Integration difficulties:
• sequencing
• abstraction
• organization
Memory difficulties:
Output difficulties:
• language
• motor