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embolisation
Varicose veins Venous ulcers
Hemosidrin deposition
Erythema nodosum
vasculitis
Charcot joint
Factors affecting interstitial
fluid formation and drainage
 Abnormal accumulation of fluid in the interstitial
tissue due to disturbed mechanisms of interstitial
fluid regulation
DVT
Unilateral
cellulitis
lymphoedema
 Pitting
 Non pitting
• Renal
• Cardiac
• Hepatic
• Nutrional
• Myxoedema
• Lymphoedema
• Angioneuritic
oedema
 Unilateral vs Bilateral
 Dependent or not
 Pitting or not
 Level
 Sacral oedema?
 Tarsal
 Medial mallulous
 Chin of tibia
 Thigh
 Anterior ab
 Bilateral
 Painless
 Pitting
 Dependent
 Before ascitis
 Ascitis before onset of oedema
 In cases of
 Pericaerdial effusion
 Constrective pericarditis
 Tricuspid regergue or Tricaspid stenosis
 Begin by eye lid buffness
 Generalized
 Pitting
 More at the morning
oliguria
Hypertension
hematuria
Smoky
urine(casts)
Heavy
proteinuria
Hypoproteinemi
a
Oedema
Hypercholesteroloem
ia
 Associated with signs and
symptoms of LCF
 DVT
 Lymphoedema (eg; post mastectomy)
 Varicose veins
 Orthostatic oedema
 Angioneuretic oedema
 Cellulitis
 DVT
 Cellulitis
 Lymphoedema
 trauma
 CCB
 Steroids
 OCPs
 NSAIDs
 Up to 3 litres of excess interstitial fluid can accumulate
in the body before oedema is clinically detectable
Thank
you

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Lower limb examination.pptx