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INTERESTING
ECGS
Dr. Nagula Praveen MD, DM
Assistant Professor of Cardiology
Osmania General Hospital
Hyderabad
drpraveennagula@gmail.com
@kizashipraveen
Case 1
 A 45 year male, smoker, alcoholic came to ER with chest pain.
 ECG at ER S/O STEMI
 Thrombolysed as lab is occupied
 He had AIVR post thrombolysis
 Shifted to ICCU
 ECG at ICCU…
• When reviewed, the ECGs in ICCU and ER are different…
At ER
At ICCU
What might have happened?
A. Lateral wall reinfarction
B. Change in focus of atrial origin from right atrial to left atrial focus after
transient AF
C. Lead reversal RA and LL
D. Lead reversal LA and LL
•Lead III becomes inverted.
•Leads I and II switch places.
•Leads aVL and aVF switch
places.
•Lead aVR remains
unchanged.
Lead II
Lead I
Lead -III
Lead AVF
Lead AVL
Case 2
 An elderly male 75 years, came with fatigue.
 He is a known hypertensive and Valvular heart disease, has been
recently changed the medications.
 Lab profile was sent which was normal
 His ECG on evaluation showed…
A.Digoxin toxicity
B.2:1 AV block
C.Hypokalemia
D.Sinus bradycardia
Case 3
 A 65 year male, diabetic, history of CAD bought to ER in
unresponsive state.
 ECG at ER…
What is the Immediate action required..?
A. Cardioversion
B. Defibrillation
C. Calcium gluconate bolus followed by insulin
D. Activate cathlab and do Primary PCI
ECG9
Case 4
21 year female with cyanosis
A. Tricuspid atresia
B. D TGA
C. L TGA
D. DORV
Case 5
• A 50 year male hypertensive, diabetic with chest pain.
• He was placed TPI through femoral route for CHB.
• Admission sugars were 200mg/dl
• Immediate profile of electrolytes was normal.
• Post TPI, his ECG
What is the likely cause of tall T waves in precordial leads ?
A. Hyperkalemia
B. Ischemia
C. Cerebral T waves
D. None
What is the lead position on this ECG?
A. RV Apex
B. RV Mid septum
C. RV Anterior wall or free wall
Lead position based on ECG
 Early transition of QRS is mid
septum
 Late transition of QRS in the
precordial leads but before V5,V6
– Free wall (Anterior wall)
 All QRS complexes negatively
concordant , R wave in aVL - RV
Apex
https://doi.org/10.1093/eurheartj/suaa094
What would be the probable cause of undersensing asynchronous pacing in this ECG ?
A. Lead fracture B. Infarction C. Electrolyte imbalance D. Magnet applied
Case 6
 A 56 year male diabetic, smoker, previous history of CAD came
with chest pain
 ECG at ER
What is unusual finding in this ECG?
A. ST elevations and depression not classical of STEMI B. First degree AV block mimicking CHB
C. RBBB D. Lateral extension of infarct
ECG6
Case 7
A 35 year male, with congenital heart defect (no intracardiac
lesions) complains of SOB
• A.
A. LA RA Lead reversal, normal precordial leads, patient is having levocardia
B. LA RA Lead reversal, normal precordial leads, patient is having dextrocardia
C. Normal limb, normal precordial leads, patient is having dextrocardia
D. LA RA lead reversal , right sided precordial leads with dextrocardia
If you don’t reverse limb leads LA RA
AVL will behave as AVR
Axis will be towards Lead III (+120)
“which is same as towards lead II in normal heart”
If you totally reverse the limb leads (mirror image) then the ECG will be as normal
Case 8
A. 2 :1 AV block
B. Complete Heart block
C. Mobitz type I AV block
D. Sinus pause
Case 9
ECG diagnosis is
A. Anterolateral STEMI with Precordial leads misplacement
B. Extensive Anterior wall MI
C. Extensive Anterior wall STEMI with precordial leads misplacement
Case 10
Case 11
What is the unsual in this ECG of this patient with no comorbidities, presenting with chest
pain considering a single diagnosis ?
A. Pericarditis
B. Bradycardia
C. It can be STEMI
D. Early repolarization variant
Case 12
A 65 year female with seizure disorder had TLOC and presented to ER,
ECG showed
A. Cerebral T waves
B. CHB with T waves secondary
to stoke adams episode
C. Ischemia
D. HCM
• Giant T wave inversions indicate VF as cause of loss of
consciousness than standstill.
Case 13
Elderly hypertensive with giddiness
A. Sinus bradycardia with junctional escape rhythm, isorhythmic dissociation
B. Type II Sino atrial exit block
C. Complete heart block
D. Type II Mobitz AV block
Case 14 Young female
A. Pericarditis
B. Early repolarization variant
C. STEMI
D. Brugada syndrome
Case 15
A 28 year male, recent COVID, came for
evaluation of cardiac function post recovery
A. Pericarditis
B. Early repolarization variant
C. STEMI
D. Hyperkalemia
Take Home Message
1. Lead reversal can change the culprit artery in STEMI.
2. Sinus bradycardia can result in u waves, not be mistaken for hypokalemia unless the
amplitude of U wave being more than T wave and having ST depression.
3. It is important to give membrane stabilizer calcium in a patient with hyperkalemia
followed by agents causing influx of potassium.
4. Cyanotic CHD with LAD,LV volume overload – tricuspid atresia is the most common.
5. Loss of capture, undersensing can be encountered in STEMI patient being kept on
TPI.
6. Inferior wall MI with RBBB,RAD – suspect distant ischemia in LAD.
7. Change of limb leads, precordial leads ( mirror image) – would result in normal ECG
in a patient with dextrocardia.
8. When P marches through QRS and not married – suspect CHB.
9. Stoke Adams syndrome is an important cause of deep T wave inversions
10. Early repolarization variant, is not a benign condition, it has considerable risk of SCD
.
THANK YOU

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INTERESTING ECGS

  • 1. INTERESTING ECGS Dr. Nagula Praveen MD, DM Assistant Professor of Cardiology Osmania General Hospital Hyderabad drpraveennagula@gmail.com @kizashipraveen
  • 2. Case 1  A 45 year male, smoker, alcoholic came to ER with chest pain.  ECG at ER S/O STEMI  Thrombolysed as lab is occupied  He had AIVR post thrombolysis  Shifted to ICCU  ECG at ICCU…
  • 3.
  • 4. • When reviewed, the ECGs in ICCU and ER are different…
  • 7.
  • 8. What might have happened? A. Lateral wall reinfarction B. Change in focus of atrial origin from right atrial to left atrial focus after transient AF C. Lead reversal RA and LL D. Lead reversal LA and LL
  • 9. •Lead III becomes inverted. •Leads I and II switch places. •Leads aVL and aVF switch places. •Lead aVR remains unchanged. Lead II Lead I Lead -III Lead AVF Lead AVL
  • 10.
  • 11. Case 2  An elderly male 75 years, came with fatigue.  He is a known hypertensive and Valvular heart disease, has been recently changed the medications.  Lab profile was sent which was normal  His ECG on evaluation showed…
  • 12. A.Digoxin toxicity B.2:1 AV block C.Hypokalemia D.Sinus bradycardia
  • 13. Case 3  A 65 year male, diabetic, history of CAD bought to ER in unresponsive state.  ECG at ER…
  • 14. What is the Immediate action required..? A. Cardioversion B. Defibrillation C. Calcium gluconate bolus followed by insulin D. Activate cathlab and do Primary PCI
  • 15. ECG9
  • 16.
  • 17. Case 4 21 year female with cyanosis A. Tricuspid atresia B. D TGA C. L TGA D. DORV
  • 18.
  • 19.
  • 20. Case 5 • A 50 year male hypertensive, diabetic with chest pain. • He was placed TPI through femoral route for CHB. • Admission sugars were 200mg/dl • Immediate profile of electrolytes was normal. • Post TPI, his ECG
  • 21. What is the likely cause of tall T waves in precordial leads ? A. Hyperkalemia B. Ischemia C. Cerebral T waves D. None
  • 22. What is the lead position on this ECG? A. RV Apex B. RV Mid septum C. RV Anterior wall or free wall
  • 23. Lead position based on ECG  Early transition of QRS is mid septum  Late transition of QRS in the precordial leads but before V5,V6 – Free wall (Anterior wall)  All QRS complexes negatively concordant , R wave in aVL - RV Apex https://doi.org/10.1093/eurheartj/suaa094
  • 24.
  • 25. What would be the probable cause of undersensing asynchronous pacing in this ECG ? A. Lead fracture B. Infarction C. Electrolyte imbalance D. Magnet applied
  • 26. Case 6  A 56 year male diabetic, smoker, previous history of CAD came with chest pain  ECG at ER
  • 27. What is unusual finding in this ECG? A. ST elevations and depression not classical of STEMI B. First degree AV block mimicking CHB C. RBBB D. Lateral extension of infarct
  • 28. ECG6
  • 29.
  • 30.
  • 31. Case 7 A 35 year male, with congenital heart defect (no intracardiac lesions) complains of SOB • A. A. LA RA Lead reversal, normal precordial leads, patient is having levocardia B. LA RA Lead reversal, normal precordial leads, patient is having dextrocardia C. Normal limb, normal precordial leads, patient is having dextrocardia D. LA RA lead reversal , right sided precordial leads with dextrocardia
  • 32. If you don’t reverse limb leads LA RA AVL will behave as AVR Axis will be towards Lead III (+120) “which is same as towards lead II in normal heart” If you totally reverse the limb leads (mirror image) then the ECG will be as normal
  • 33.
  • 34.
  • 35. Case 8 A. 2 :1 AV block B. Complete Heart block C. Mobitz type I AV block D. Sinus pause
  • 36.
  • 37. Case 9 ECG diagnosis is A. Anterolateral STEMI with Precordial leads misplacement B. Extensive Anterior wall MI C. Extensive Anterior wall STEMI with precordial leads misplacement
  • 39. Case 11 What is the unsual in this ECG of this patient with no comorbidities, presenting with chest pain considering a single diagnosis ? A. Pericarditis B. Bradycardia C. It can be STEMI D. Early repolarization variant
  • 40. Case 12 A 65 year female with seizure disorder had TLOC and presented to ER, ECG showed A. Cerebral T waves B. CHB with T waves secondary to stoke adams episode C. Ischemia D. HCM
  • 41.
  • 42. • Giant T wave inversions indicate VF as cause of loss of consciousness than standstill.
  • 43.
  • 44. Case 13 Elderly hypertensive with giddiness A. Sinus bradycardia with junctional escape rhythm, isorhythmic dissociation B. Type II Sino atrial exit block C. Complete heart block D. Type II Mobitz AV block
  • 45.
  • 46. Case 14 Young female A. Pericarditis B. Early repolarization variant C. STEMI D. Brugada syndrome
  • 47. Case 15 A 28 year male, recent COVID, came for evaluation of cardiac function post recovery A. Pericarditis B. Early repolarization variant C. STEMI D. Hyperkalemia
  • 48.
  • 49. Take Home Message 1. Lead reversal can change the culprit artery in STEMI. 2. Sinus bradycardia can result in u waves, not be mistaken for hypokalemia unless the amplitude of U wave being more than T wave and having ST depression. 3. It is important to give membrane stabilizer calcium in a patient with hyperkalemia followed by agents causing influx of potassium. 4. Cyanotic CHD with LAD,LV volume overload – tricuspid atresia is the most common. 5. Loss of capture, undersensing can be encountered in STEMI patient being kept on TPI. 6. Inferior wall MI with RBBB,RAD – suspect distant ischemia in LAD. 7. Change of limb leads, precordial leads ( mirror image) – would result in normal ECG in a patient with dextrocardia. 8. When P marches through QRS and not married – suspect CHB. 9. Stoke Adams syndrome is an important cause of deep T wave inversions 10. Early repolarization variant, is not a benign condition, it has considerable risk of SCD .