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ENDOCARDITIS




     Group three presentation
Group members


   Shida mbeye
   Taonga kanyenda
   Panesi white
   Chigomezgo munthali
   Maria kamanga
   Gilly banda
BROAD OBJECTIVE

   By the end of the presentation the audience must acquire knowledge
    and skills on the condition of endocarditis
SPECIFIC OBJECTIVES

   Definition of endocarditis
   Etiology
   Risk factors
   Pathophysiology
   Clinical manifestation
   Diagnostic evaluation
   Nursing assessment
   Nursing diagnosis
   Nursing management
   Medical management
   complications
   Introduction
       The heart is a vital organ that pumps blood and it has three layers
        which are endocardium, myocardium and pericadium.
definition

•   Endocarditis is an inflammation of the inner layer of
    the heart, the endocardium. it usually involves the
    valves and other structures like the intraventricular
    and septum.
etiology


   Fungus e.g. candida, aspagellus
   Gram negative organisms e.g. pseudomonas
   Bacterias e.g. staphylococci
   Acute rheumatic fever which cause enlarged and
    tender lymph nodes, damages the valves
   Congenital heart disease
Risk factors

   Previous heart damage
   Dental procedures which lead into the introduction of
    bacterias
   Heart surgery
   Intubations
   Procedures involving gastrol intestinal and genitourinaly
    tracts e.g.
    barium, enemas, sigmoidoscopy, catheterisation and
    cytoscopy
   Reproductive conditions like delivery of new
    babies, abortions and pelvic inflammatory disease
pathophysiology

   Usually in this case the bacterias or any other causing agents
    enter the blood stream through invasive procedures like dental
    procedures, surgery , urinary catherisation.
   Then they accumulate on the valves of the heart or
    endocardium
   Finally they form vegetations or crusters
   These vegetation they lead into damage heart valves by
    perforating and deforming the valves leaflets
   This at the end leads to tearing which means there is poor flow
    of blood and lead into accumulation of blood in chambers of the
    heart hence endocarditis
Clinical manifestations

   Loss of vision
   Ischemia
   Fever
   Shortness of breath
   Shock
   Finger clubbing
   Myocardial infarction
   Weight loss
   Malaise
   Chills
   Night sweats
   Chest and abdominal pain
   anorexia
Diagnostic evaluation

   Blood culture; helps in testing the presence of microorganisms
   Urinalysis to see microscopic hematuria.
   Doppler echocardiography assist in the diagnosis by
    demonstrating a muscle on the valve.
   Computerized tomography rule out heart damage
   Review bun and creatinine levels to evaluate renal function
   Review endocardiography finding if available for valvuler and
    ventricular function and presence of vegetation
   Review white blood cell count to evaluate course if infection
Nursing assessment



   It includes history taking like;
     – Subjective data:
     – past medical history: patient asked of signs of the
         disease and the onset of the disease and review
         with patient history of risk factors like cardiac
         failure, shock
     – Medication history: has the pt ever taken any
         medication, what happened afterwards
     – Family history:asked of any case at home of the
         similar conditions
–   Social history: social behaviours that can trigger
    the problem
–   Surgical history: if ever operated on
–   Objective data: assess for temperature
    elevations, heart mummer, evidence of cough ,
    peripheral edema and embolism, alscultate for
    heart sound, monitor arterial blood gas, rapid
    purse rate,dyspnea, restlessness and
    manifestation of heart failure
Nursing diagnoses


   Infective breathing pattern related to inflammation of
    heart muscle as evidenced by use of accessory
    muscle, dyspnea.
   Impaired gaseous exchange related to fluid
    accumulation in the lungs as evidenced by shortness
    of breath
   Decreased cardiac output related to valvular
    dysfunction as evidenced by poor tissue perfusion
   Imbalanced nutrition less than body requirement
    related to anorexia as evidenced by loss of weight.
   Altered thermoregulation related to infection as
    evidenced by increased body temperature (fever).
   impaired physical mobility related to fatigue
   Ineffective tissue perfusion related to embolisation
   Anxiety related to hypoxia or life threatening situation
    as evidenced by patient verbalization
   Altered comfort pain related to use of accessory
    muscle as evidenced by patient verbalization
Nursing management

   Position the patient at semi fowlers position to help in infective
    breathing through providing enough room for lung expansion as
    abdominal contents goes down
   Administer oxygen therapy 4-6 l/min to help pt in breathing
    effectively through supplementing oxygen
   Monitor arterial blood gas , carbon dioxide, oxygen saturation
    hourly and document to monitor signs of respiratory acidosis
   Encourage and provide small frequent meals reach in proteins
    helping in repairing worn-out tissues
   Monitor vital signs , heart and lung sound, level of consciousness
    to evaluate how effectively the organs like the heart and the lungs
    are working
   Schedule nursing activities to allow rest
   Encourage and assist pt to cough and deep breath to promote
    chest expansion
    provide tepid sponging to reduce raised body temperature by
    evaporation and conduction
   Encourage patient on exercises in order to improve patients
    mobility through making the body physically fit
   Make yourself available to the patient and nurse with love and
    respond well to his/her questions to array pain and anxiety
   Educate the patient on disease process to make pt cope up
    with therapy and the condition
Medical management


   if the patient is having poor intake of oxygen administer oxygen
    therapy 4-6 l/m in helping the patient get supplemented with
    enough oxygen
   Administer antibiotic treatment like ampicillin + flucloxacilin
    +gentamicin
   Administer pcm 1g tds p.o as analgesic that help in relieving
    pain
   Administer inotropic drugs like digoxin that help in increasing
    contractility of the heart
   The patient can also be administered with morphine which help
    to decrease anxiety
complications


   Chronic heart failure
   Shock
   Stroke
   embolism
In conclusion


   Endocarditis as explained above it is the inflammation of
    endocardium and it brings a lot complications



                        Therefore:
references


    PAUL ONEILL (2012), MASTER MEDICINE. SECOND
     EDITION
    LINTON (2007). INTRO. TO MEDI-SURGE NURSING,4TH
     EDITION
    •S. Lewis, M. Heitkemper, S. Dirksen, P. Obrien and L.
    Bucher (2007) Medical Surgical Nursing; Assessment and
    Management of Clinical problems (7th edition)St
    Louisi, Mosby.

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Endocarditis

  • 1. ENDOCARDITIS Group three presentation
  • 2. Group members  Shida mbeye  Taonga kanyenda  Panesi white  Chigomezgo munthali  Maria kamanga  Gilly banda
  • 3. BROAD OBJECTIVE  By the end of the presentation the audience must acquire knowledge and skills on the condition of endocarditis
  • 4. SPECIFIC OBJECTIVES  Definition of endocarditis  Etiology  Risk factors  Pathophysiology  Clinical manifestation  Diagnostic evaluation  Nursing assessment  Nursing diagnosis  Nursing management  Medical management  complications
  • 5. Introduction  The heart is a vital organ that pumps blood and it has three layers which are endocardium, myocardium and pericadium.
  • 6. definition • Endocarditis is an inflammation of the inner layer of the heart, the endocardium. it usually involves the valves and other structures like the intraventricular and septum.
  • 7. etiology  Fungus e.g. candida, aspagellus  Gram negative organisms e.g. pseudomonas  Bacterias e.g. staphylococci  Acute rheumatic fever which cause enlarged and tender lymph nodes, damages the valves  Congenital heart disease
  • 8. Risk factors  Previous heart damage  Dental procedures which lead into the introduction of bacterias  Heart surgery  Intubations  Procedures involving gastrol intestinal and genitourinaly tracts e.g. barium, enemas, sigmoidoscopy, catheterisation and cytoscopy  Reproductive conditions like delivery of new babies, abortions and pelvic inflammatory disease
  • 9. pathophysiology  Usually in this case the bacterias or any other causing agents enter the blood stream through invasive procedures like dental procedures, surgery , urinary catherisation.  Then they accumulate on the valves of the heart or endocardium  Finally they form vegetations or crusters  These vegetation they lead into damage heart valves by perforating and deforming the valves leaflets  This at the end leads to tearing which means there is poor flow of blood and lead into accumulation of blood in chambers of the heart hence endocarditis
  • 10. Clinical manifestations  Loss of vision  Ischemia  Fever  Shortness of breath  Shock  Finger clubbing  Myocardial infarction  Weight loss  Malaise  Chills  Night sweats  Chest and abdominal pain  anorexia
  • 11. Diagnostic evaluation  Blood culture; helps in testing the presence of microorganisms  Urinalysis to see microscopic hematuria.  Doppler echocardiography assist in the diagnosis by demonstrating a muscle on the valve.  Computerized tomography rule out heart damage  Review bun and creatinine levels to evaluate renal function  Review endocardiography finding if available for valvuler and ventricular function and presence of vegetation  Review white blood cell count to evaluate course if infection
  • 12. Nursing assessment  It includes history taking like; – Subjective data: – past medical history: patient asked of signs of the disease and the onset of the disease and review with patient history of risk factors like cardiac failure, shock – Medication history: has the pt ever taken any medication, what happened afterwards – Family history:asked of any case at home of the similar conditions
  • 13. Social history: social behaviours that can trigger the problem – Surgical history: if ever operated on – Objective data: assess for temperature elevations, heart mummer, evidence of cough , peripheral edema and embolism, alscultate for heart sound, monitor arterial blood gas, rapid purse rate,dyspnea, restlessness and manifestation of heart failure
  • 14. Nursing diagnoses  Infective breathing pattern related to inflammation of heart muscle as evidenced by use of accessory muscle, dyspnea.  Impaired gaseous exchange related to fluid accumulation in the lungs as evidenced by shortness of breath  Decreased cardiac output related to valvular dysfunction as evidenced by poor tissue perfusion  Imbalanced nutrition less than body requirement related to anorexia as evidenced by loss of weight.
  • 15. Altered thermoregulation related to infection as evidenced by increased body temperature (fever).  impaired physical mobility related to fatigue  Ineffective tissue perfusion related to embolisation  Anxiety related to hypoxia or life threatening situation as evidenced by patient verbalization  Altered comfort pain related to use of accessory muscle as evidenced by patient verbalization
  • 16. Nursing management  Position the patient at semi fowlers position to help in infective breathing through providing enough room for lung expansion as abdominal contents goes down  Administer oxygen therapy 4-6 l/min to help pt in breathing effectively through supplementing oxygen  Monitor arterial blood gas , carbon dioxide, oxygen saturation hourly and document to monitor signs of respiratory acidosis  Encourage and provide small frequent meals reach in proteins helping in repairing worn-out tissues  Monitor vital signs , heart and lung sound, level of consciousness to evaluate how effectively the organs like the heart and the lungs are working
  • 17. Schedule nursing activities to allow rest  Encourage and assist pt to cough and deep breath to promote chest expansion  provide tepid sponging to reduce raised body temperature by evaporation and conduction  Encourage patient on exercises in order to improve patients mobility through making the body physically fit  Make yourself available to the patient and nurse with love and respond well to his/her questions to array pain and anxiety  Educate the patient on disease process to make pt cope up with therapy and the condition
  • 18. Medical management  if the patient is having poor intake of oxygen administer oxygen therapy 4-6 l/m in helping the patient get supplemented with enough oxygen  Administer antibiotic treatment like ampicillin + flucloxacilin +gentamicin  Administer pcm 1g tds p.o as analgesic that help in relieving pain  Administer inotropic drugs like digoxin that help in increasing contractility of the heart  The patient can also be administered with morphine which help to decrease anxiety
  • 19. complications  Chronic heart failure  Shock  Stroke  embolism
  • 20. In conclusion  Endocarditis as explained above it is the inflammation of endocardium and it brings a lot complications  Therefore:
  • 21. references  PAUL ONEILL (2012), MASTER MEDICINE. SECOND EDITION  LINTON (2007). INTRO. TO MEDI-SURGE NURSING,4TH EDITION •S. Lewis, M. Heitkemper, S. Dirksen, P. Obrien and L. Bucher (2007) Medical Surgical Nursing; Assessment and Management of Clinical problems (7th edition)St Louisi, Mosby.