Domina Petric, MD
ECG L
Left atrial enlargement
Burns E. Left Atrial Enlargement (April 16, 2017). Retrieved from
https://lifeinthefastlane.com/ecg-library/basics/left-atrial-enlargement/
Left atrial enlargement
Left atrial enlargement (LAE) is due to
pressure or volume overload of the
left atrium.
It is often a precursor to atrial
fibrillation!!!
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ECG features
DII
Bifid P wave with
>40 ms between the
two peaks!
Total P wave
duration >110 ms!
V1
Biphasic P wave with
terminal negative
portion >40 ms
duration!
Biphasic P wave with
terminal negative
portion >1 mm deep!
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ECG features
DII V1
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Bifid P wave with duration of 120 ms
and more than 40 ms between two peaks!
Biphasic P wave with negative portion
>40 ms duration (or >1 mm deep)!
Causes
Mitral stenosis
Systemic
hypertension
Aortic stenosis
Mitral
incompetence
Hypertrophic
cardiomyopathy
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Left Anterior Fascicular Block
Burns E. Left Anterior Fascicular Block (April 16, 2017). Retrieved from
https://lifeinthefastlane.com/ecg-library/basics/left-anterior-fascicular-block/
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LAFB
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ECG features
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LAFB
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Left axis deviation!
qR in DI
rS in DII
rS in DIII
qR in aVL
rS in aVF
Left Bundle Branch Block
Burns E. Left Bundle Branch Block (April 16, 2017). Retrieved from
https://lifeinthefastlane.com/ecg-library/basics/left-bundle-branch-block/
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LBBB
•In LBBB, the normal direction of
septal depolarisation is reversed
(becomes right to left), as the
impulse spreads first to the right
ventricle via the right bundle
branch and then to the left ventricle
via the septum.
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ECG criteria
QRS duration of >120 ms.
Dominant S wave in V1.
Broad monophasic R wave in lateral leads (DI, aVL, V5-V6).
Absence of Q waves in lateral leads (DI, V5-V6).
Prolonged R wave peak time >60 ms in left precordial leads (V5-V6).
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ECG criteria
• Appropriate discordance: the ST segments
and T waves always go in the opposite
direction to the main vector of the QRS
complex.
• Poor R wave progression in the chest leads
and left axis deviation.
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rS
ST elevation and upright T wave
QRS morphology
The R wave in the lateral leads may be either:
•M-shaped
•Notched
•Monophasic
•RS complex
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Causes
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LMCA occlusion
Burns E. ST Elevation in aVR-LMCA occlusion (April 3, 2017).
Retrieved from https://lifeinthefastlane.com/ecg-library/lmca/
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LMCA occlusion
Widespread horizontal ST depression,
most prominent in leads DI, DII and V4-V6!
ST elevation in aVR ≥1mm!
ST elevation in aVR ≥V1!
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ST-elevation in aVR may also be seen with:
proximal left anterior descending artery
(LAD) occlusion
severe triple-vessel disease (3VD)
diffuse subendocardial ischaemia
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ECG example
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ST-elevation in aVR>V1
Left Posterior Fascicular Block (LPFB)
Burns E. Left Posterior Fascicular Block (April 16, 2017). Retrieved from
https://lifeinthefastlane.com/ecg-library/basics/left-posterior-fascicular-block/
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LPFB
•In left posterior fascicular block (aka left
posterior hemiblock), impulses are
conducted to the left ventricle via the left
anterior fascicle, which inserts into the
upper, lateral wall of the left ventricle
along its endocardial surface.
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ECG features
Right axis deviation (> +90 degrees)
Small R waves with deep S waves (rS complexes) in leads DI and aVL
Small Q waves with tall R waves (qR complexes) in leads DII, DIII and aVF
QRS duration normal or slightly prolonged (80-110ms)
Prolonged R wave peak time in aVF
Increased QRS voltage in the limb leads
No evidence of right ventricular hypertrophy
No evidence of any other cause for right axis deviation
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LPHB
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rS
rSqR
qR qR
Right axis deviation
Left ventricular aneurysm
Burns E. Left Ventricular Aneursym (April 3, 2017). Retrieved from
https://lifeinthefastlane.com/ecg-library/left-ventricular-aneursym/
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ECG features
ST elevation seen after 2 weeks following an acute
myocardial infarction:
• most commonly seen in the precordial leads
• may exhibit concave or convex morphology
• usually associated with well-formed Q or QS
waves
• T-waves have a relatively small amplitude in
comparison to the QRS complex (unlike the
hyperacute T-waves of acute STEMI)
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ECG features
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Persistent ST elevation
qS waves
T-waves have smaller
amplitude than QRS complexes
Left ventricular hypertrophy
Burns E. Left Ventricular Hypertrophy (March 18, 2017). Retrieved from
https://lifeinthefastlane.com/ecg-library/basics/left-ventricular-hypertrophy/
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ECG features
The most commonly used
are the Sokolov-
Lyon criteria: S wave depth
in V1 + tallest R wave
height in V5-V6 >35 mm.
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ECG features
R wave in lead DI + S wave in
lead DIII >25 mm
R wave in aVL >11 mm
R wave in aVF >20 mm
S wave in aVR >14 mm
R wave in V4, V5
or V6 >26 mm
R wave in V5 or V6
plus S wave in V1
>35 mm
Largest R wave
plus largest S wave
in precordial leads
>45 mm
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ECG features
Left atrial enlargement
Left axis deviation
ST elevation in the right precordial leads
V1-V3 (discordant to the deep S waves)
Prominent U waves (proportional to
increased QRS amplitude)
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Lown-Ganong-Levine (LGL) Syndrome
Lifeinthefastlane.com
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LGL
•Accessory pathway composed
of James fibres.
•ECG features are:
•PR interval <120ms
•Normal QRS morphology
•No delta wave
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LOW QRS VOLTAGE
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Low QRS voltage
•The amplitudes of all the QRS complexes
in the limb leads are <5 mm.
•The amplitudes of all the QRS complexes
in the precordial leads are <10 mm.
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ECG L

  • 1.
  • 2.
    Left atrial enlargement BurnsE. Left Atrial Enlargement (April 16, 2017). Retrieved from https://lifeinthefastlane.com/ecg-library/basics/left-atrial-enlargement/
  • 3.
    Left atrial enlargement Leftatrial enlargement (LAE) is due to pressure or volume overload of the left atrium. It is often a precursor to atrial fibrillation!!! PowerPlugs Templates for PowerPoint Preview 3
  • 4.
    ECG features DII Bifid Pwave with >40 ms between the two peaks! Total P wave duration >110 ms! V1 Biphasic P wave with terminal negative portion >40 ms duration! Biphasic P wave with terminal negative portion >1 mm deep! PowerPlugs Templates for PowerPoint Preview 4
  • 5.
    ECG features DII V1 PowerPlugsTemplates for PowerPoint Preview 5 Bifid P wave with duration of 120 ms and more than 40 ms between two peaks! Biphasic P wave with negative portion >40 ms duration (or >1 mm deep)!
  • 6.
  • 7.
    Left Anterior FascicularBlock Burns E. Left Anterior Fascicular Block (April 16, 2017). Retrieved from https://lifeinthefastlane.com/ecg-library/basics/left-anterior-fascicular-block/ PowerPlugs Templates for PowerPoint Preview 7
  • 8.
    LAFB PowerPlugs Templates forPowerPoint Preview 8
  • 9.
    ECG features PowerPlugs Templatesfor PowerPoint Preview 9
  • 10.
    LAFB PowerPlugs Templates forPowerPoint Preview 10 Left axis deviation! qR in DI rS in DII rS in DIII qR in aVL rS in aVF
  • 11.
    Left Bundle BranchBlock Burns E. Left Bundle Branch Block (April 16, 2017). Retrieved from https://lifeinthefastlane.com/ecg-library/basics/left-bundle-branch-block/ PowerPlugs Templates for PowerPoint Preview 11
  • 12.
    LBBB •In LBBB, thenormal direction of septal depolarisation is reversed (becomes right to left), as the impulse spreads first to the right ventricle via the right bundle branch and then to the left ventricle via the septum. PowerPlugs Templates for PowerPoint Preview 12
  • 13.
    ECG criteria QRS durationof >120 ms. Dominant S wave in V1. Broad monophasic R wave in lateral leads (DI, aVL, V5-V6). Absence of Q waves in lateral leads (DI, V5-V6). Prolonged R wave peak time >60 ms in left precordial leads (V5-V6). PowerPlugs Templates for PowerPoint Preview 13
  • 14.
    ECG criteria • Appropriatediscordance: the ST segments and T waves always go in the opposite direction to the main vector of the QRS complex. • Poor R wave progression in the chest leads and left axis deviation. PowerPlugs Templates for PowerPoint Preview 14 rS ST elevation and upright T wave
  • 15.
    QRS morphology The Rwave in the lateral leads may be either: •M-shaped •Notched •Monophasic •RS complex PowerPlugs Templates for PowerPoint Preview 15
  • 16.
    Causes PowerPlugs Templates forPowerPoint Preview 16
  • 17.
    LMCA occlusion Burns E.ST Elevation in aVR-LMCA occlusion (April 3, 2017). Retrieved from https://lifeinthefastlane.com/ecg-library/lmca/ PowerPlugs Templates for PowerPoint Preview 17
  • 18.
    LMCA occlusion Widespread horizontalST depression, most prominent in leads DI, DII and V4-V6! ST elevation in aVR ≥1mm! ST elevation in aVR ≥V1! PowerPlugs Templates for PowerPoint Preview 18
  • 19.
    ST-elevation in aVRmay also be seen with: proximal left anterior descending artery (LAD) occlusion severe triple-vessel disease (3VD) diffuse subendocardial ischaemia PowerPlugs Templates for PowerPoint Preview 19
  • 20.
    ECG example PowerPlugs Templatesfor PowerPoint Preview 20 ST-elevation in aVR>V1
  • 21.
    Left Posterior FascicularBlock (LPFB) Burns E. Left Posterior Fascicular Block (April 16, 2017). Retrieved from https://lifeinthefastlane.com/ecg-library/basics/left-posterior-fascicular-block/ PowerPlugs Templates for PowerPoint Preview 21
  • 22.
    LPFB •In left posteriorfascicular block (aka left posterior hemiblock), impulses are conducted to the left ventricle via the left anterior fascicle, which inserts into the upper, lateral wall of the left ventricle along its endocardial surface. PowerPlugs Templates for PowerPoint Preview 22
  • 23.
    ECG features Right axisdeviation (> +90 degrees) Small R waves with deep S waves (rS complexes) in leads DI and aVL Small Q waves with tall R waves (qR complexes) in leads DII, DIII and aVF QRS duration normal or slightly prolonged (80-110ms) Prolonged R wave peak time in aVF Increased QRS voltage in the limb leads No evidence of right ventricular hypertrophy No evidence of any other cause for right axis deviation PowerPlugs Templates for PowerPoint Preview 23
  • 24.
    LPHB PowerPlugs Templates forPowerPoint Preview 24 rS rSqR qR qR Right axis deviation
  • 25.
    Left ventricular aneurysm BurnsE. Left Ventricular Aneursym (April 3, 2017). Retrieved from https://lifeinthefastlane.com/ecg-library/left-ventricular-aneursym/ PowerPlugs Templates for PowerPoint Preview 25
  • 26.
    ECG features ST elevationseen after 2 weeks following an acute myocardial infarction: • most commonly seen in the precordial leads • may exhibit concave or convex morphology • usually associated with well-formed Q or QS waves • T-waves have a relatively small amplitude in comparison to the QRS complex (unlike the hyperacute T-waves of acute STEMI) PowerPlugs Templates for PowerPoint Preview 26
  • 27.
    ECG features PowerPlugs Templatesfor PowerPoint Preview 27 Persistent ST elevation qS waves T-waves have smaller amplitude than QRS complexes
  • 28.
    Left ventricular hypertrophy BurnsE. Left Ventricular Hypertrophy (March 18, 2017). Retrieved from https://lifeinthefastlane.com/ecg-library/basics/left-ventricular-hypertrophy/ PowerPlugs Templates for PowerPoint Preview 28
  • 29.
    ECG features The mostcommonly used are the Sokolov- Lyon criteria: S wave depth in V1 + tallest R wave height in V5-V6 >35 mm. PowerPlugs Templates for PowerPoint Preview 29
  • 30.
    ECG features R wavein lead DI + S wave in lead DIII >25 mm R wave in aVL >11 mm R wave in aVF >20 mm S wave in aVR >14 mm R wave in V4, V5 or V6 >26 mm R wave in V5 or V6 plus S wave in V1 >35 mm Largest R wave plus largest S wave in precordial leads >45 mm PowerPlugs Templates for PowerPoint Preview 30
  • 31.
    ECG features Left atrialenlargement Left axis deviation ST elevation in the right precordial leads V1-V3 (discordant to the deep S waves) Prominent U waves (proportional to increased QRS amplitude) PowerPlugs Templates for PowerPoint Preview 31
  • 32.
  • 33.
    LGL •Accessory pathway composed ofJames fibres. •ECG features are: •PR interval <120ms •Normal QRS morphology •No delta wave PowerPlugs Templates for PowerPoint Preview 33
  • 34.
    LOW QRS VOLTAGE PowerPlugsTemplates for PowerPoint Preview 34
  • 35.
    Low QRS voltage •Theamplitudes of all the QRS complexes in the limb leads are <5 mm. •The amplitudes of all the QRS complexes in the precordial leads are <10 mm. PowerPlugs Templates for PowerPoint Preview 35
  • 36.
    PowerPlugs Templates forPowerPoint Preview 36