The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.
The document discusses the classification of odontogenic cysts. It describes several classification systems proposed over time, including Robinson's classification from 1945, Thoma-Robinson-Bernier classification from 1960, Pindborg and Kramer's classification from 1971, and the WHO classification from 1971 and its update in 1992. The WHO classifications categorize cysts as developmental or inflammatory, and further divide developmental cysts into odontogenic and non-odontogenic types. Shafer's classification also categorizes cysts based on etiology as developmental or inflammatory, and further divides them based on the tissue of origin.
The document provides information about Pindborg tumor, also known as calcifying epithelial odontogenic tumor (CEOT). It defines CEOT as a locally invasive epithelial odontogenic neoplasm characterized by the presence of amyloid material that may become calcified. The document discusses the pathogenesis, histopathological features including epithelial cells, amyloid-like material and calcific deposits, immunohistochemical findings, differential diagnosis and treatment of CEOT. It also mentions the recurrence rate of CEOT is typically 10-15% but can be higher in certain variants.
Dr. Gaurav S. Salunkhe presented on oral and maxillofacial pathology. The document discussed the classification, development, diseases and conditions that affect bone, including the alveolar bone. Specific conditions discussed in detail include cherubism, osteogenesis imperfecta, osteopetrosis, and cleidocranial dysplasia. Cherubism is a rare hereditary condition causing bilateral jaw swelling in children that typically resolves after puberty.
This document discusses various non-odontogenic tumors classified according to their origin. It covers giant cell lesions including central giant cell granuloma and brown tumor of hyperparathyroidism. Vascular lesions such as hemangiomas, vascular malformations, and neurogenic tumors including neurofibromas are also discussed. For each type of lesion, the document provides information on classification, clinical features, radiographic findings, diagnosis, and treatment options.
This document discusses mixed odontogenic tumors and odontogenic sarcomas according to the 2005 WHO classification. It provides details on ameloblastic fibroma, its clinical features, histopathology, radiographic features and differential diagnosis. It also discusses ameloblastic fibro-odontoma and fibro-dentinoma, calcifying cystic odontogenic tumor, complex and compound odontomas, and odontoameloblastoma. The key information provided includes the definitions, epidemiology, clinical and radiographic presentation, histopathology, and differential diagnosis of these odontogenic lesions.
Calcifying epithelial odontogenic tumor is a rare, aggressive but benign odontogenic tumor accounting for 1% of all odontogenic tumors. It was first recognized by Pindborg and is of epithelial origin. Intraosseous tumors are more common than extraosseous tumors. Radiographically, it appears as a radiolucency that may contain small radiopacities. Surgical removal by enucleation is the treatment of choice, with recurrence rates of 10-15%.
This document discusses different types of odontogenic tumors. It classifies them into three categories: tumors of odontogenic epithelium, mixed odontogenic tumors, and tumors of odontogenic ectomesenchyme. Key tumors discussed include ameloblastoma, adenomatoid odontogenic tumor (AOT), and calcifying epithelial odontogenic tumor (CEOT). Ameloblastoma is the most common odontogenic tumor and can be solid/multicystic, unicystic, or peripheral. AOT typically occurs in younger females in the anterior maxilla. CEOT accounts for less than 1% of odontogenic tumors and resembles cells of the enamel organ or dental lamina.
Necrotizing gingivostomatitis is an inflammatory condition affecting the gingiva and mouth that is caused by fusiform bacteria and spirochetes. It is characterized by painful, red gingiva with crater-like sores between teeth, bleeding gums, bad breath, and fever. Examination of affected tissue shows many bacteria and white blood cells. Histologically, there is gingival ulceration and necrosis with an inflammatory cell infiltrate. Treatment involves cleaning the mouth with antiseptics followed by scaling and antibiotics to resolve the infection.
The document discusses the classification of odontogenic cysts. It describes several classification systems proposed over time, including Robinson's classification from 1945, Thoma-Robinson-Bernier classification from 1960, Pindborg and Kramer's classification from 1971, and the WHO classification from 1971 and its update in 1992. The WHO classifications categorize cysts as developmental or inflammatory, and further divide developmental cysts into odontogenic and non-odontogenic types. Shafer's classification also categorizes cysts based on etiology as developmental or inflammatory, and further divides them based on the tissue of origin.
The document provides information about Pindborg tumor, also known as calcifying epithelial odontogenic tumor (CEOT). It defines CEOT as a locally invasive epithelial odontogenic neoplasm characterized by the presence of amyloid material that may become calcified. The document discusses the pathogenesis, histopathological features including epithelial cells, amyloid-like material and calcific deposits, immunohistochemical findings, differential diagnosis and treatment of CEOT. It also mentions the recurrence rate of CEOT is typically 10-15% but can be higher in certain variants.
Dr. Gaurav S. Salunkhe presented on oral and maxillofacial pathology. The document discussed the classification, development, diseases and conditions that affect bone, including the alveolar bone. Specific conditions discussed in detail include cherubism, osteogenesis imperfecta, osteopetrosis, and cleidocranial dysplasia. Cherubism is a rare hereditary condition causing bilateral jaw swelling in children that typically resolves after puberty.
This document discusses various non-odontogenic tumors classified according to their origin. It covers giant cell lesions including central giant cell granuloma and brown tumor of hyperparathyroidism. Vascular lesions such as hemangiomas, vascular malformations, and neurogenic tumors including neurofibromas are also discussed. For each type of lesion, the document provides information on classification, clinical features, radiographic findings, diagnosis, and treatment options.
This document discusses mixed odontogenic tumors and odontogenic sarcomas according to the 2005 WHO classification. It provides details on ameloblastic fibroma, its clinical features, histopathology, radiographic features and differential diagnosis. It also discusses ameloblastic fibro-odontoma and fibro-dentinoma, calcifying cystic odontogenic tumor, complex and compound odontomas, and odontoameloblastoma. The key information provided includes the definitions, epidemiology, clinical and radiographic presentation, histopathology, and differential diagnosis of these odontogenic lesions.
Calcifying epithelial odontogenic tumor is a rare, aggressive but benign odontogenic tumor accounting for 1% of all odontogenic tumors. It was first recognized by Pindborg and is of epithelial origin. Intraosseous tumors are more common than extraosseous tumors. Radiographically, it appears as a radiolucency that may contain small radiopacities. Surgical removal by enucleation is the treatment of choice, with recurrence rates of 10-15%.
This document discusses different types of odontogenic tumors. It classifies them into three categories: tumors of odontogenic epithelium, mixed odontogenic tumors, and tumors of odontogenic ectomesenchyme. Key tumors discussed include ameloblastoma, adenomatoid odontogenic tumor (AOT), and calcifying epithelial odontogenic tumor (CEOT). Ameloblastoma is the most common odontogenic tumor and can be solid/multicystic, unicystic, or peripheral. AOT typically occurs in younger females in the anterior maxilla. CEOT accounts for less than 1% of odontogenic tumors and resembles cells of the enamel organ or dental lamina.
Necrotizing gingivostomatitis is an inflammatory condition affecting the gingiva and mouth that is caused by fusiform bacteria and spirochetes. It is characterized by painful, red gingiva with crater-like sores between teeth, bleeding gums, bad breath, and fever. Examination of affected tissue shows many bacteria and white blood cells. Histologically, there is gingival ulceration and necrosis with an inflammatory cell infiltrate. Treatment involves cleaning the mouth with antiseptics followed by scaling and antibiotics to resolve the infection.
Cysts in orofacial regions were discussed. Key points include:
1. Cysts are pathological cavities lined by epithelium and filled with fluid/semi-solid material. Common types are odontogenic cysts like dentigerous and keratocysts.
2. Dentigerous cysts form between reduced enamel epithelium and tooth crown, associated with unerupted teeth. Keratocysts have high recurrence rates due to thin fragile lining.
3. Treatment options are marsupialization to shrink large cysts, and enucleation to remove the cyst lining along with the associated tooth/teeth.
This document provides information about calcifying odontogenic cysts (COCs). It defines COCs and classifies them according to the WHO. COCs are rare jaw lesions characterized by ghost cells and calcifications. They are thought to arise from odontogenic epithelial remnants. Clinically, they typically present in the second decade of life with lesions more common in the maxilla than mandible. Radiographically, COCs appear well-defined with variable calcifications. Histologically, they contain ghost cells and basal cell layer with hyperchromatic nuclei. Prognosis is generally good when treated with surgical removal.
1. The document discusses various types of orofacial pain including somatic, neurogenic, and psychogenic pain. It describes trigeminal neuralgia as a common type of neurogenic pain characterized by sharp, electric shock-like pain in the face that is triggered by stimulation of specific trigger zones. 2. Evaluation of orofacial pain involves taking a thorough medical history and performing physical and neurological examinations. Differential diagnosis considers local causes as well as neurological disorders. 3. Treatment depends on the underlying cause but may include medications, nerve blocks, surgery, or a combination. Carbamazepine is first-line treatment for trigeminal neuralgia.
This document discusses fibro-osseous lesions, which replace normal bone with fibrous tissue containing newly formed mineralized structures. It describes several types of fibro-osseous lesions including fibrous dysplasia, cemento-osseous dysplasias like periapical cemental dysplasia, and fibro-osseous neoplasms like ossifying fibroma. For each type, it covers definitions, clinical features, radiographic appearances, differential diagnosis, and treatment approaches.
Periapical radiolucencies can have many causes, both benign and malignant. They are often classified as either anatomical pseudoperiapical radiolucencies, which do not contact the tooth apex, or true periapical radiolucent lesions, which do. Common true lesions include periapical granulomas, radicular cysts, and periapical abscesses. Periapical granulomas appear as well-defined radiolucencies, while radicular cysts can cause tooth displacement if left untreated. Management depends on the diagnosis and may involve root canal treatment, extraction, or surgery. Differential diagnosis considers conditions like osteomyelitis, dentigerous cysts,
The document discusses osteomyelitis, which is an inflammatory condition of bone that begins as an infection of the medullary cavity and spreads to involve the periosteum. It can be acute or chronic, and is caused by bacteria or fungi entering via trauma or a blood-borne route. Symptoms include pain, swelling, and pus drainage. Diagnosis involves medical imaging and biopsy. Treatment involves antibiotics, drainage of pus, debridement of infected tissue, and sometimes surgery. Chronic osteomyelitis can be difficult to treat and may require repeated surgeries. Risk factors include reduced blood supply to bone from conditions like diabetes.
MPDS, or myofascial pain disorder syndrome, is a pain disorder characterized by unilateral pain referred from trigger points in muscles of the head and neck. These trigger points are localized tender areas within taut muscle bands caused by micro- or macro-trauma to the musculoskeletal system. Accumulation of chemicals like lactic acid and prostaglandins in the muscles lowers the pain threshold, leading to MPDS symptoms like pain, limited jaw motion, and joint noises. Diagnosis involves assessing range of motion, palpating muscles for tenderness, and grading joint clicks. Treatment aims to inactivate trigger points, prevent recurrence, and correct perpetuating factors through therapies like physical modalities, anesthesia, pharmacotherapy, and occasionally
The document discusses different types of cysts that can occur in the oral and maxillofacial region. It defines cysts and classifies them based on their origin and location. It provides details on the pathogenesis, clinical features, radiographic appearance and histology of specific cysts such as dentigerous cysts and odontogenic keratocysts. Dentigerous cysts are defined as cysts originating from the separation of the dental follicle from around the crown of an unerupted tooth. Odontogenic keratocysts are distinctive cysts that arise from cell rests of the dental lamina and have more aggressive behavior than other cysts. Complications of cysts include recurrence, development of
Dr. Abdelhady provides a lecture on odontogenic tumors. The lecture aims to help students classify and diagnose odontogenic tumors, examine patients presenting with facial swellings, and determine differential diagnoses and management techniques for mandibular and maxillary swellings. Specific odontogenic tumors discussed include cementoblastoma, odontogenic fibroma, central giant cell granuloma, cherubism, fibrous dysplasia, and ossifying fibroma. Radiographic features, histology, treatment options and prognosis are described for each tumor type.
This document discusses Necrotizing Ulcerative Gingivitis (NUG), also known as trench mouth. It defines NUG as a microbial disease of the gingiva caused by an impaired host response. Key clinical features include necrosis of gingival tissue and pain. Diagnosis is based on these clinical findings and microscopic examination. Management involves reducing the microbial load, removing necrotic tissue, treating any systemic conditions, and supportive periodontal therapy. Prognosis is generally good with treatment but recurrence is possible without ongoing maintenance of oral hygiene.
Dentinogenesis imperfecta is a hereditary condition that affects the formation of dentin in both primary and permanent teeth. It is classified into two main types - dentinogenesis imperfecta type 1 and type 2. Type 1 is caused by mutations in the DSPP gene and affects only the teeth. Type 2 may be caused by mutations in two tightly linked genes and is characterized by multiple pulp exposures and shell-like teeth. Treatment aims to prevent wear of enamel and dentin through full coverage restorations.
This document provides an overview of cysts of the oral and maxillofacial region. It defines cysts and discusses their classification, parts, pathogenesis and mechanisms of enlargement. It also describes key cysts such as dentigerous cysts, odontogenic keratocysts and eruption cysts in detail, covering their definitions, locations, clinical and radiographic features, histology, differential diagnosis and complications. Dentigerous and odontogenic keratocysts are the most common epithelial cysts of the jaws.
- Trauma from occlusion occurs when occlusal forces exceed the adaptive capacity of the periodontium, causing injury. It can be acute or chronic.
- The magnitude, direction, duration, and frequency of forces impact the periodontium's ability to adapt. Excessive pressure or tension can damage tissues.
- Primary trauma from occlusion is caused by changes in occlusal forces, while secondary trauma occurs when reduced bone support impairs the tissues' resistance to normal forces.
- The periodontium responds to trauma in three stages - injury, repair through new tissue formation, and adaptive remodeling to better withstand forces. Trauma can cause reversible damage if forces are reduced, or lead to irreversible injury if
Fibro-osseous lesions of the jaws
Fibrous dysplasia
Cemento-osseous dysplasia
Focal cemento-osseous dysplasia
Periapical cemento-osseous dysplasia
Florid cemento-osseous dysplasia
Ossifying fibroma
Juvenile aggressive ossifying fibroma
Cherubism
Fibro-osseous lesions (FOL) are characterized by replacement of normal bone architecture by collagen fibers and fibroblasts containing calcified tissue.
They include a wide variety of lesions of developmental, dysplastic and neoplastic origins with clinical and radiographic presentation and behavior.
Because of the histological similarities between diverse diseases, proper diagnosis requires correlation of history, clinical and radiographic findings.Fibrous Dysplasia
2. Reactive (dysplastic lesions arising in the tooth-bearing area (presumably of periodontal origin).
a. Periapical cemento-osseous dysplasia
b. Focal cemento-osseous dysplasia
c. Florid cemento-osseous dysplasia
3. Fibro-osseous neoplasms (widely designated as cementifying fibroma, ossifying fibroma or cemento-ossifying fibroma.Bone dysplasias
a. Fibrous dyspla i. Monostoticii. Polyostotic
iii. Polyostotic with endocrinopathy (McCune-Albright)
iv Osteofibrous dysplasia
b. Osteitis deformansc. Pagetoid heritable bone dysplasias of childhood
d. Segmental odontomaxillary dysplasia
2. Cemento-osseous dysplasias
a. Focal cemento-osseous dysplasia b. Florid cemento-osseous dysplasia
3.Inflammatory/reactive processes
a. Focal sclerosing osteomyelitisb. Diffuse sclerosing osteomyelitis
c. Proliferative periostitis
4. Metabolic Disease: hyperparathyroidism
5. Neoplastic lesions (Ossifying fibromas)
a. Ossifying fibromab. Hyperparathyroidism jaw lesion syndrome
c. Juvenile ossifying fibroma i. Trabecular typeii. Psammomatoid type
d. Gigantiform cementomas
This document discusses various types of pseudocysts and true cysts found in the body. It begins by defining the key differences between a true cyst and a pseudocyst. Pseudocysts are lined by granulation and/or fibrous tissue rather than an epithelial cell layer. The document then classifies and describes different types of pseudocysts including traumatic bone cysts, aneurysmal bone cysts, and developmental cysts. It provides details on the etiology, location, patient demographics, clinical presentation, radiographic features, histopathology and treatment for each type.
This document discusses the anatomy and histopathology of the periodontium, which consists of cementum, periodontal ligament, and alveolar bone. It describes the different types of cementum and cells found in the periodontal ligament. Chronic periapical lesions are discussed, including their etiology, clinical features, classifications, and examples such as chronic apical periodontitis and periapical granuloma. Treatment options are mentioned for various pathological conditions like symptomatic apical periodontitis.
Odontogenic keratocyst (OKC) is the cyst arising from the cell rests of dental lamina. It can occur anywhere in the jaw, but commonly seen in the posterior part of the mandible. Radiographically, most OKCs are unilocular when presented at the periapex and can be mistaken for radicular or lateral periodontal cyst.
The document discusses the classification and stages of gingivitis. It describes gingivitis as being classified based on duration into acute or chronic, and based on distribution into localized, generalized, marginal or papillary. It then outlines the four stages of gingivitis - initial lesion, early lesion, established lesion, and advanced lesion - providing details on the clinical and microscopic features of each stage. It also discusses changes in the position of the gingiva that can occur, such as coronal migration (pseudopockets) or apical migration (recession).
This document provides an overview of fibrous dysplasia. It begins with an introduction discussing bone composition and fibro-osseous lesions. It then covers the classification, definition, etiology, clinical features including monostotic and polyostotic forms, histologic features, radiographic features, treatment and prognosis. Special forms of fibrous dysplasia are also mentioned. In conclusion, it states that asymptomatic cases are managed conservatively while symptomatic cases can now be treated reliably to restore function and improve aesthetics.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
This document discusses pemphigus vulgaris, an autoimmune disease characterized by the formation of blisters within the epidermis caused by autoantibodies against desmoglein proteins. It most commonly affects individuals in their 40s and 50s and is more prevalent in Ashkenazi Jews and those of Mediterranean descent. Clinical features include painful oral and skin ulcers. Diagnosis is confirmed through direct immunofluorescence detecting autoantibodies at the dermoepidermal junction. Treatment ranges from topical corticosteroids for mild cases to systemic immunosuppressants like prednisone and azathioprine for moderate to severe disease.
Cysts in orofacial regions were discussed. Key points include:
1. Cysts are pathological cavities lined by epithelium and filled with fluid/semi-solid material. Common types are odontogenic cysts like dentigerous and keratocysts.
2. Dentigerous cysts form between reduced enamel epithelium and tooth crown, associated with unerupted teeth. Keratocysts have high recurrence rates due to thin fragile lining.
3. Treatment options are marsupialization to shrink large cysts, and enucleation to remove the cyst lining along with the associated tooth/teeth.
This document provides information about calcifying odontogenic cysts (COCs). It defines COCs and classifies them according to the WHO. COCs are rare jaw lesions characterized by ghost cells and calcifications. They are thought to arise from odontogenic epithelial remnants. Clinically, they typically present in the second decade of life with lesions more common in the maxilla than mandible. Radiographically, COCs appear well-defined with variable calcifications. Histologically, they contain ghost cells and basal cell layer with hyperchromatic nuclei. Prognosis is generally good when treated with surgical removal.
1. The document discusses various types of orofacial pain including somatic, neurogenic, and psychogenic pain. It describes trigeminal neuralgia as a common type of neurogenic pain characterized by sharp, electric shock-like pain in the face that is triggered by stimulation of specific trigger zones. 2. Evaluation of orofacial pain involves taking a thorough medical history and performing physical and neurological examinations. Differential diagnosis considers local causes as well as neurological disorders. 3. Treatment depends on the underlying cause but may include medications, nerve blocks, surgery, or a combination. Carbamazepine is first-line treatment for trigeminal neuralgia.
This document discusses fibro-osseous lesions, which replace normal bone with fibrous tissue containing newly formed mineralized structures. It describes several types of fibro-osseous lesions including fibrous dysplasia, cemento-osseous dysplasias like periapical cemental dysplasia, and fibro-osseous neoplasms like ossifying fibroma. For each type, it covers definitions, clinical features, radiographic appearances, differential diagnosis, and treatment approaches.
Periapical radiolucencies can have many causes, both benign and malignant. They are often classified as either anatomical pseudoperiapical radiolucencies, which do not contact the tooth apex, or true periapical radiolucent lesions, which do. Common true lesions include periapical granulomas, radicular cysts, and periapical abscesses. Periapical granulomas appear as well-defined radiolucencies, while radicular cysts can cause tooth displacement if left untreated. Management depends on the diagnosis and may involve root canal treatment, extraction, or surgery. Differential diagnosis considers conditions like osteomyelitis, dentigerous cysts,
The document discusses osteomyelitis, which is an inflammatory condition of bone that begins as an infection of the medullary cavity and spreads to involve the periosteum. It can be acute or chronic, and is caused by bacteria or fungi entering via trauma or a blood-borne route. Symptoms include pain, swelling, and pus drainage. Diagnosis involves medical imaging and biopsy. Treatment involves antibiotics, drainage of pus, debridement of infected tissue, and sometimes surgery. Chronic osteomyelitis can be difficult to treat and may require repeated surgeries. Risk factors include reduced blood supply to bone from conditions like diabetes.
MPDS, or myofascial pain disorder syndrome, is a pain disorder characterized by unilateral pain referred from trigger points in muscles of the head and neck. These trigger points are localized tender areas within taut muscle bands caused by micro- or macro-trauma to the musculoskeletal system. Accumulation of chemicals like lactic acid and prostaglandins in the muscles lowers the pain threshold, leading to MPDS symptoms like pain, limited jaw motion, and joint noises. Diagnosis involves assessing range of motion, palpating muscles for tenderness, and grading joint clicks. Treatment aims to inactivate trigger points, prevent recurrence, and correct perpetuating factors through therapies like physical modalities, anesthesia, pharmacotherapy, and occasionally
The document discusses different types of cysts that can occur in the oral and maxillofacial region. It defines cysts and classifies them based on their origin and location. It provides details on the pathogenesis, clinical features, radiographic appearance and histology of specific cysts such as dentigerous cysts and odontogenic keratocysts. Dentigerous cysts are defined as cysts originating from the separation of the dental follicle from around the crown of an unerupted tooth. Odontogenic keratocysts are distinctive cysts that arise from cell rests of the dental lamina and have more aggressive behavior than other cysts. Complications of cysts include recurrence, development of
Dr. Abdelhady provides a lecture on odontogenic tumors. The lecture aims to help students classify and diagnose odontogenic tumors, examine patients presenting with facial swellings, and determine differential diagnoses and management techniques for mandibular and maxillary swellings. Specific odontogenic tumors discussed include cementoblastoma, odontogenic fibroma, central giant cell granuloma, cherubism, fibrous dysplasia, and ossifying fibroma. Radiographic features, histology, treatment options and prognosis are described for each tumor type.
This document discusses Necrotizing Ulcerative Gingivitis (NUG), also known as trench mouth. It defines NUG as a microbial disease of the gingiva caused by an impaired host response. Key clinical features include necrosis of gingival tissue and pain. Diagnosis is based on these clinical findings and microscopic examination. Management involves reducing the microbial load, removing necrotic tissue, treating any systemic conditions, and supportive periodontal therapy. Prognosis is generally good with treatment but recurrence is possible without ongoing maintenance of oral hygiene.
Dentinogenesis imperfecta is a hereditary condition that affects the formation of dentin in both primary and permanent teeth. It is classified into two main types - dentinogenesis imperfecta type 1 and type 2. Type 1 is caused by mutations in the DSPP gene and affects only the teeth. Type 2 may be caused by mutations in two tightly linked genes and is characterized by multiple pulp exposures and shell-like teeth. Treatment aims to prevent wear of enamel and dentin through full coverage restorations.
This document provides an overview of cysts of the oral and maxillofacial region. It defines cysts and discusses their classification, parts, pathogenesis and mechanisms of enlargement. It also describes key cysts such as dentigerous cysts, odontogenic keratocysts and eruption cysts in detail, covering their definitions, locations, clinical and radiographic features, histology, differential diagnosis and complications. Dentigerous and odontogenic keratocysts are the most common epithelial cysts of the jaws.
- Trauma from occlusion occurs when occlusal forces exceed the adaptive capacity of the periodontium, causing injury. It can be acute or chronic.
- The magnitude, direction, duration, and frequency of forces impact the periodontium's ability to adapt. Excessive pressure or tension can damage tissues.
- Primary trauma from occlusion is caused by changes in occlusal forces, while secondary trauma occurs when reduced bone support impairs the tissues' resistance to normal forces.
- The periodontium responds to trauma in three stages - injury, repair through new tissue formation, and adaptive remodeling to better withstand forces. Trauma can cause reversible damage if forces are reduced, or lead to irreversible injury if
Fibro-osseous lesions of the jaws
Fibrous dysplasia
Cemento-osseous dysplasia
Focal cemento-osseous dysplasia
Periapical cemento-osseous dysplasia
Florid cemento-osseous dysplasia
Ossifying fibroma
Juvenile aggressive ossifying fibroma
Cherubism
Fibro-osseous lesions (FOL) are characterized by replacement of normal bone architecture by collagen fibers and fibroblasts containing calcified tissue.
They include a wide variety of lesions of developmental, dysplastic and neoplastic origins with clinical and radiographic presentation and behavior.
Because of the histological similarities between diverse diseases, proper diagnosis requires correlation of history, clinical and radiographic findings.Fibrous Dysplasia
2. Reactive (dysplastic lesions arising in the tooth-bearing area (presumably of periodontal origin).
a. Periapical cemento-osseous dysplasia
b. Focal cemento-osseous dysplasia
c. Florid cemento-osseous dysplasia
3. Fibro-osseous neoplasms (widely designated as cementifying fibroma, ossifying fibroma or cemento-ossifying fibroma.Bone dysplasias
a. Fibrous dyspla i. Monostoticii. Polyostotic
iii. Polyostotic with endocrinopathy (McCune-Albright)
iv Osteofibrous dysplasia
b. Osteitis deformansc. Pagetoid heritable bone dysplasias of childhood
d. Segmental odontomaxillary dysplasia
2. Cemento-osseous dysplasias
a. Focal cemento-osseous dysplasia b. Florid cemento-osseous dysplasia
3.Inflammatory/reactive processes
a. Focal sclerosing osteomyelitisb. Diffuse sclerosing osteomyelitis
c. Proliferative periostitis
4. Metabolic Disease: hyperparathyroidism
5. Neoplastic lesions (Ossifying fibromas)
a. Ossifying fibromab. Hyperparathyroidism jaw lesion syndrome
c. Juvenile ossifying fibroma i. Trabecular typeii. Psammomatoid type
d. Gigantiform cementomas
This document discusses various types of pseudocysts and true cysts found in the body. It begins by defining the key differences between a true cyst and a pseudocyst. Pseudocysts are lined by granulation and/or fibrous tissue rather than an epithelial cell layer. The document then classifies and describes different types of pseudocysts including traumatic bone cysts, aneurysmal bone cysts, and developmental cysts. It provides details on the etiology, location, patient demographics, clinical presentation, radiographic features, histopathology and treatment for each type.
This document discusses the anatomy and histopathology of the periodontium, which consists of cementum, periodontal ligament, and alveolar bone. It describes the different types of cementum and cells found in the periodontal ligament. Chronic periapical lesions are discussed, including their etiology, clinical features, classifications, and examples such as chronic apical periodontitis and periapical granuloma. Treatment options are mentioned for various pathological conditions like symptomatic apical periodontitis.
Odontogenic keratocyst (OKC) is the cyst arising from the cell rests of dental lamina. It can occur anywhere in the jaw, but commonly seen in the posterior part of the mandible. Radiographically, most OKCs are unilocular when presented at the periapex and can be mistaken for radicular or lateral periodontal cyst.
The document discusses the classification and stages of gingivitis. It describes gingivitis as being classified based on duration into acute or chronic, and based on distribution into localized, generalized, marginal or papillary. It then outlines the four stages of gingivitis - initial lesion, early lesion, established lesion, and advanced lesion - providing details on the clinical and microscopic features of each stage. It also discusses changes in the position of the gingiva that can occur, such as coronal migration (pseudopockets) or apical migration (recession).
This document provides an overview of fibrous dysplasia. It begins with an introduction discussing bone composition and fibro-osseous lesions. It then covers the classification, definition, etiology, clinical features including monostotic and polyostotic forms, histologic features, radiographic features, treatment and prognosis. Special forms of fibrous dysplasia are also mentioned. In conclusion, it states that asymptomatic cases are managed conservatively while symptomatic cases can now be treated reliably to restore function and improve aesthetics.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
This document discusses pemphigus vulgaris, an autoimmune disease characterized by the formation of blisters within the epidermis caused by autoantibodies against desmoglein proteins. It most commonly affects individuals in their 40s and 50s and is more prevalent in Ashkenazi Jews and those of Mediterranean descent. Clinical features include painful oral and skin ulcers. Diagnosis is confirmed through direct immunofluorescence detecting autoantibodies at the dermoepidermal junction. Treatment ranges from topical corticosteroids for mild cases to systemic immunosuppressants like prednisone and azathioprine for moderate to severe disease.
This document discusses various odontogenic tumors, which are lesions derived from cellular elements that form tooth structures. It defines key tumor types such as ameloblastoma, unicystic ameloblastoma, peripheral ameloblastoma, adenomatoid odontogenic tumor, calcifying epithelial odontogenic tumor, and odontoma. It provides details on the classification, clinical features, histopathology, treatment options and prognosis for each tumor type. Radiographic features are also described to aid in diagnosis. The goal of treatment is complete surgical removal while preserving function and appearance.
This document provides information on oral submucous fibrosis (OSMF), including its definition, history, pathogenesis, clinical features, and staging classifications. OSMF is a chronic disease affecting the oral cavity and sometimes pharynx, characterized by juxtaepithelial inflammatory reaction and fibrosis of the lamina propria. Chewing betel quid and areca nut releases alkaloids and tannins that cause irritation and inflammation, activating fibroblasts and increasing production and cross-linking of collagen over time, resulting in stiffness and inability to open the mouth. OSMF is considered a precancerous condition due to its association with oral cancer. Staging systems describe progression from initial symptoms to trismus and possible malignancy
Oral lichen planus is a chronic inflammatory disease that affects the oral mucosa. It is characterized by white striations (Wickham's striae) and varies in appearance from reticular to erythematous or ulcerative lesions. The cause is unknown but involves a cell-mediated immune response. Treatment focuses on reducing symptoms and includes topical corticosteroids, immunosuppressants, or retinoids. Malignant transformation may rarely occur so follow-up is important.
Final practical (( oralpatho )) for 3rd students 1-7-2013TUDSU
This document provides an overview of various oral pathology topics including developmental disorders of teeth, dental caries, pulp diseases, cysts of oral tissues, bacterial and fungal diseases of the oral mucosa, white lesions, infectious diseases, tumor-like lesions, bone diseases, salivary gland diseases, and oral ulcerations. Specific conditions mentioned include hypodontia, mesiodens, cleidocranial dysplasia, amelogenesis imperfecta, dentinogenesis imperfecta, dental caries, pulpitis, periapical lesions, dentigerous cyst, odontogenic keratocyst, tongue anomalies, ameloblastoma, tuberculosis, syphilis, lichen planus, osteogenesis imperfect
For more free medical powerpoints, visit www. medicaldump.com, Free updates everyday on all specialties including cardiology, nephrology, neurology, pulmonology, etc.
Odontogenic cysts and tumors originate from tooth-forming tissues. The most common cyst is the radicular cyst, which develops from inflammation around the root apex of a nonvital tooth. Dentigerous cysts encase the crown of an unerupted tooth. Odontogenic keratocyst is lined by keratinizing epithelium and has a high recurrence rate. Odontomas are benign odontogenic tumors composed of enamel and dentin. The most common odontogenic tumor is the ameloblastoma, which is locally invasive and has a high recurrence rate if not completely excised. Calcifying odontogenic tumor demonstrates epithelial cells and calcified material.
A cyst is an epithelium-lined sac containing fluid or semisolid material. In the formation of a cyst, the epithelial cells first proliferate and later undergo degeneration and liquefaction. The liquefied material exerts equal pressure on the walls of the cyst from within. Cysts grow by expansion and thus displace the adjacent teeth by pressure. May can produce expansion of the cortical bone. On a radiograph, the radiolucency of a cyst is usually bordered by a radiopaque periphery of dense sclerotic bone. The radiolucency may be unilocular or multilocular. Odontogenic cysts are those which arise from the epithelium associated with the development of teeth. The source of epithelium is from the enamel organ, the reduced enamel epithelium, the cell rests of Malassez or the remnants of the dental lamina.
The document discusses several types of odontogenic tumors that can occur in the jaws. It focuses on describing ameloblastoma, adenomatoid tumor, and calcifying epithelial odontogenic tumor. Ameloblastoma is a benign but locally aggressive tumor arising from odontogenic epithelium. It commonly presents as a painless swelling in the mandible and radiographs show multilocular radiolucency. Histologically there are follicular or plexiform patterns. Adenomatoid tumor is a rare benign tumor associated with impacted teeth. Calcifying epithelial odontogenic tumor is a rare, locally aggressive tumor that can be mistaken for carcinoma, presenting with calcified masses visible on radiographs.
This document defines and describes odontogenic tumors, which are neoplasms or tumor-like malformations arising from odontogenic tissues. It classifies various odontogenic tumors based on their origin from odontogenic epithelium, mesenchyme, or mixed tissues. Key tumors discussed include ameloblastoma, adenomatoid odontogenic tumor (AOT), calcifying epithelial odontogenic tumor (CEOT), and ameloblastic fibroma. Clinical features, imaging findings, and histopathological characteristics are provided for each tumor type.
The document discusses different types of cysts that can occur in the oral region, dividing them into odontogenic cysts and non-odontogenic cysts. Odontogenic cysts include radicular, dentigerous, primordial, odontogenic keratocyst, and lateral periodontal cysts. Non-odontogenic cysts include globulomaxillary, nasolabial, median palatal, and nasopalatine canal cysts. Each cyst type is described in terms of etiology, clinical features, radiographic appearance, histology, and treatment.
This document discusses various types of odontogenic cysts. It begins with introducing cysts in general and then classifies odontogenic cysts based on etiology and tissue of origin. Several specific types of odontogenic cysts are then described in more detail, including their clinical features, radiographic features, and differential diagnosis. These include dentigerous cysts, eruption cysts, odontogenic keratocysts, gingival cysts of newborn and adult, lateral periodontal cysts, calcifying odontogenic cysts, periapical cysts, residual cysts, and paradental cysts.
DIAGNOSIS AND TREATMENT PLANNING FOR COMPLETE DENTURES .pdfHimanshu Tiwari
The document discusses diagnosis and treatment planning for complete dentures. It covers:
1. Successful complete denture therapy requires a thorough assessment of the patient's physical and psychological condition to deliver a functional denture that meets their expectations.
2. Treatment planning involves developing a course of action based on the diagnosis to serve the patient's needs, and includes examination of medical and dental history, clinical examination, and radiographs.
3. The first appointment is critical for developing trust and understanding the patient's chief complaint and expectations.
Case history & diagnosis in periodontics /certified fixed orthodontic course...Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
Case history & diagnosis in periodontics /certified fixed orthodontic course...Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
This document provides an overview of how to take a case history for a dental patient. It discusses the importance of gathering demographic data, chief complaints, medical history, dental history and conducting examinations. The key components of a case history are outlined, including the steps of taking a history, examining the patient, making a provisional diagnosis, conducting investigations, reaching a final diagnosis and developing a treatment plan. Taking a thorough case history is important for understanding the patient's condition, making an accurate diagnosis and determining an effective treatment approach.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.
This document discusses the examination and diagnosis of complete denture patients. It emphasizes the importance of a thorough case history and physical examination. The case history should explore the patient's dental history, medical history, habits, expectations and mental attitude. The physical examination involves both extraoral and intraoral assessment including facial form, profile, symmetry, complexion and lip support. A systematic examination allows for an accurate diagnosis, prognosis, and treatment plan.
This document provides an overview of periodontal disease, including the periodontal team, causes, symptoms, classifications, diagnostic procedures, and treatments. The main points are:
- Periodontal disease involves inflammation and infection of the gums and bone around the teeth. It is common and can be caused by plaque, calculus, poor oral hygiene, and certain medical conditions.
- The periodontal team includes the periodontist, dental assistants, hygienists, and office staff who work together to treat and manage periodontal disease.
- Diagnostic procedures include a medical history, examination, x-rays, and periodontal screening to assess the patient's condition and develop a treatment plan.
Oral cancer screening involves examination of the mouth by a dentist or doctor to identify signs of cancer early. Screening methods include visual examination with tools like a mouth mirror [1], as well as adjunct tests to identify abnormal cells, like oral cytology or brush biopsy [2]. Additional aids like vital stains that detect high nucleic acid areas can specify sites for biopsy [3]. Early detection through regular screening is important as 5-year survival rates for oral cancer remain below 50% [1].
This document provides information on diagnosis and treatment planning for completely edentulous patients. It defines diagnosis as determining the nature of a disease and treatment planning as the sequence of procedures planned after diagnosis. The document outlines the process of evaluating a patient, including obtaining their medical and dental history, performing extraoral and intraoral examinations, and taking radiographs and impressions. It emphasizes the importance of understanding a patient's physical and mental health, needs, and expectations in order to develop an appropriate treatment plan.
Cancer is a disease of the cells in the body. The body is made up from millions of tiny cells. There are several types of oral cancers, but around 90% are squamous cell carcinomas originating in the tissues that line the mouth and lips.
Oral or mouth cancer most commonly involves the tongue.
This document provides an overview of periodontal examination and diagnosis. It discusses taking a medical and dental history, performing a radiographic survey, examining casts, photographs, oral hygiene, the teeth, periodontium, and gingiva. Scoring indices like the Gingival Index, Plaque Index, Bleeding on Probing, and Calculus Index are also described to evaluate inflammation levels. The goal of periodontal diagnosis is to determine disease presence, type, extent, severity and develop a treatment plan.
Introduction: Oral cancer is one of the most prevalent diseases worldwide, accounting for 30-40% of the head and neck cancer. It is fairly common and very curable if found and treated at an early stage.
Definition: Oral cancer is also known as mouth cancer, is cancer of the lining of the lips, mouth or upper throat. It belongs to a large group of cancers called head and neck cancers.
Classification: The TNM classification stages different types of cancer based on certain standard criteria:
T describes the size of the primary tumor
N describe the lymph nodes
M describes whether the cancer has metastasized.
This document discusses the process of periodontal diagnosis. It begins by defining diagnosis and explaining its importance. There are several types of diagnoses discussed - provisional, differential, and final. The key stages of diagnosis are then outlined, including history taking, clinical examination, investigations, and arriving at a diagnosis. Specific aspects examined clinically are also described, such as tooth mobility, fremitus, trauma from occlusion, and pathologic migration. Periodontal charts are mentioned as an important tool for recording examination findings.
This document discusses gingival diseases that can affect children. It begins by describing normal pediatric periodontium and then classifies and describes various gingival diseases including eruption gingivitis, dental plaque-induced gingivitis, acute conditions like herpes gingivostomatitis and recurrent aphthous ulcers, and gingival diseases modified by systemic factors. Treatment options are provided for each condition with an emphasis on prevention, improved oral hygiene and dental care, and management of predisposing factors.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.
Fixed prosthodontic treatment can offer exceptional satisfaction for both patient and the dentist. Fixed Prosthodontics can transform an unhealthy, unattractive dentition with poor function into a comfortable, healthy occlusion capable of giving years of further service while greatly enhancing esthetics.
Nothing is more important in the construction of fixed partial dentures than an adequate diagnosis and a well-devised treatment plan. Although these two subjects are usually considered together. Astute dentists must recognize their subtle differences. Diagnosis is an evaluation of the condition of the patient when he presents for treatment. Treatment planning concerns the treatment procedures by which the dentist will restore the patient to an optimum state of dental health.
A comprehensive presentation of how oral health is related to overall systemic health, The Perio Protect Program is an easy and effective way to treat and manage periodontal disease.
oral cancer is the common melignancy in male and can leads to death of patient and social isolation among patient this ppt help in knowing the condition and refers by nurses for their knowledge and application in their clinical practice
Similar to Copy of dentinogenic ghost cell tumor ready / dental implant courses (20)
Opportunity for Dentists (BDS/MDS )to relocate to United kingdom -Register as a DENTAL HYGIENIST/ DENTAL THERAPIST without Board exams and after approval you can register in GDC as a DH/DT and start working as a DH/DT Immediately and get paid.
You can complete the whole process in 3-4 months.Salary range for DH/DT is around 2500-3500 Pounds per month.
Eligibility / requirements-
1. An International English Language Testing System (IELTS) certificate
at the appropriate level.(Within 2 yrs of application date )
2: A recent primary dental qualification that has been taught and examined in English..(Within 2 yrs of application date )
3: A recent pass in a language test for registration with a regulatory authority in a country where the first language is English.
If you are interested Please contact us for more details.
1ST, 2ND AND 3RD ORDER BENDS IN STANDARD EDGEWISE APPLIANCE SYSTEM /Fixed ort...Indian dental academy
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals
who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry,
Periodontics and General Dentistry.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
I –Aligners are made with FDA approved transparent thermoplastic materials using 3D scanning, 3D Printing and finally Trays with Pressure vacuum formers.
Dear Doctor,
Indian Dental Academy Now offers comprehensive online Orthodontics course.
Course includes:
1.whiteboard lecture presentations
2.Case Discussions
3.with hundreds of pictures.
4.Demo on Models
5.Demo on Patients
6. subtitles in your own language
12 months unlimited access and support @350 USD only.
For Demo please visit :www.idalectures.com/preview/
For more details visit: www.idalectures.com
Please contact us for any clarifications:
idalectures@gmail.com
indiandentalacademy@gmail.com
Thanks & Regards
Indian Dental Academy
--
Indian Dental Academy
Leader in continuing dental education
www.indiandentalacademy.com
skype:indiandentalacademy
+919248678078
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Cytotoxicity of silicone materials used in maxillofacial prosthesis / dental ...Indian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Diagnosis and treatment planning in completely endntulous arches/dental coursesIndian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Properties of Denture base materials /rotary endodontic coursesIndian dental academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
Use of modified tooth forms in complete denture occlusion / dental implant...Indian dental academy
This document discusses dental occlusion concepts and philosophies for complete dentures. It introduces key terms like physiologic occlusion and defines different occlusion schemes like balanced articulation and monoplane articulation. The document discusses advantages and disadvantages of using anatomic versus non-anatomic teeth for complete dentures. It also outlines requirements for maintaining denture stability, such as balanced occlusal contacts and control of horizontal forces. The goal of occlusion for complete dentures is to re-establish the homeostasis of the masticatory system disrupted by edentulism.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
This document discusses dental casting investment materials. It describes the three main types of investments - gypsum bonded, phosphate bonded, and ethyl silicate bonded investments. For gypsum bonded investments specifically, it details their classification, composition including the roles of gypsum, silica, and modifiers, setting time, normal and hygroscopic setting expansion, and thermal expansion. It provides information on how the properties of gypsum bonded investments are affected by their composition. The document serves as a comprehensive overview of dental casting investment materials.
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.for more details please visit
www.indiandentalacademy.com
ISO/IEC 27001, ISO/IEC 42001, and GDPR: Best Practices for Implementation and...PECB
Denis is a dynamic and results-driven Chief Information Officer (CIO) with a distinguished career spanning information systems analysis and technical project management. With a proven track record of spearheading the design and delivery of cutting-edge Information Management solutions, he has consistently elevated business operations, streamlined reporting functions, and maximized process efficiency.
Certified as an ISO/IEC 27001: Information Security Management Systems (ISMS) Lead Implementer, Data Protection Officer, and Cyber Risks Analyst, Denis brings a heightened focus on data security, privacy, and cyber resilience to every endeavor.
His expertise extends across a diverse spectrum of reporting, database, and web development applications, underpinned by an exceptional grasp of data storage and virtualization technologies. His proficiency in application testing, database administration, and data cleansing ensures seamless execution of complex projects.
What sets Denis apart is his comprehensive understanding of Business and Systems Analysis technologies, honed through involvement in all phases of the Software Development Lifecycle (SDLC). From meticulous requirements gathering to precise analysis, innovative design, rigorous development, thorough testing, and successful implementation, he has consistently delivered exceptional results.
Throughout his career, he has taken on multifaceted roles, from leading technical project management teams to owning solutions that drive operational excellence. His conscientious and proactive approach is unwavering, whether he is working independently or collaboratively within a team. His ability to connect with colleagues on a personal level underscores his commitment to fostering a harmonious and productive workplace environment.
Date: May 29, 2024
Tags: Information Security, ISO/IEC 27001, ISO/IEC 42001, Artificial Intelligence, GDPR
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Find out more about ISO training and certification services
Training: ISO/IEC 27001 Information Security Management System - EN | PECB
ISO/IEC 42001 Artificial Intelligence Management System - EN | PECB
General Data Protection Regulation (GDPR) - Training Courses - EN | PECB
Webinars: https://pecb.com/webinars
Article: https://pecb.com/article
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For more information about PECB:
Website: https://pecb.com/
LinkedIn: https://www.linkedin.com/company/pecb/
Facebook: https://www.facebook.com/PECBInternational/
Slideshare: http://www.slideshare.net/PECBCERTIFICATION
How to Make a Field Mandatory in Odoo 17Celine George
In Odoo, making a field required can be done through both Python code and XML views. When you set the required attribute to True in Python code, it makes the field required across all views where it's used. Conversely, when you set the required attribute in XML views, it makes the field required only in the context of that particular view.
Leveraging Generative AI to Drive Nonprofit InnovationTechSoup
In this webinar, participants learned how to utilize Generative AI to streamline operations and elevate member engagement. Amazon Web Service experts provided a customer specific use cases and dived into low/no-code tools that are quick and easy to deploy through Amazon Web Service (AWS.)
Chapter wise All Notes of First year Basic Civil Engineering.pptxDenish Jangid
Chapter wise All Notes of First year Basic Civil Engineering
Syllabus
Chapter-1
Introduction to objective, scope and outcome the subject
Chapter 2
Introduction: Scope and Specialization of Civil Engineering, Role of civil Engineer in Society, Impact of infrastructural development on economy of country.
Chapter 3
Surveying: Object Principles & Types of Surveying; Site Plans, Plans & Maps; Scales & Unit of different Measurements.
Linear Measurements: Instruments used. Linear Measurement by Tape, Ranging out Survey Lines and overcoming Obstructions; Measurements on sloping ground; Tape corrections, conventional symbols. Angular Measurements: Instruments used; Introduction to Compass Surveying, Bearings and Longitude & Latitude of a Line, Introduction to total station.
Levelling: Instrument used Object of levelling, Methods of levelling in brief, and Contour maps.
Chapter 4
Buildings: Selection of site for Buildings, Layout of Building Plan, Types of buildings, Plinth area, carpet area, floor space index, Introduction to building byelaws, concept of sun light & ventilation. Components of Buildings & their functions, Basic concept of R.C.C., Introduction to types of foundation
Chapter 5
Transportation: Introduction to Transportation Engineering; Traffic and Road Safety: Types and Characteristics of Various Modes of Transportation; Various Road Traffic Signs, Causes of Accidents and Road Safety Measures.
Chapter 6
Environmental Engineering: Environmental Pollution, Environmental Acts and Regulations, Functional Concepts of Ecology, Basics of Species, Biodiversity, Ecosystem, Hydrological Cycle; Chemical Cycles: Carbon, Nitrogen & Phosphorus; Energy Flow in Ecosystems.
Water Pollution: Water Quality standards, Introduction to Treatment & Disposal of Waste Water. Reuse and Saving of Water, Rain Water Harvesting. Solid Waste Management: Classification of Solid Waste, Collection, Transportation and Disposal of Solid. Recycling of Solid Waste: Energy Recovery, Sanitary Landfill, On-Site Sanitation. Air & Noise Pollution: Primary and Secondary air pollutants, Harmful effects of Air Pollution, Control of Air Pollution. . Noise Pollution Harmful Effects of noise pollution, control of noise pollution, Global warming & Climate Change, Ozone depletion, Greenhouse effect
Text Books:
1. Palancharmy, Basic Civil Engineering, McGraw Hill publishers.
2. Satheesh Gopi, Basic Civil Engineering, Pearson Publishers.
3. Ketki Rangwala Dalal, Essentials of Civil Engineering, Charotar Publishing House.
4. BCP, Surveying volume 1
The chapter Lifelines of National Economy in Class 10 Geography focuses on the various modes of transportation and communication that play a vital role in the economic development of a country. These lifelines are crucial for the movement of goods, services, and people, thereby connecting different regions and promoting economic activities.
This presentation was provided by Rebecca Benner, Ph.D., of the American Society of Anesthesiologists, for the second session of NISO's 2024 Training Series "DEIA in the Scholarly Landscape." Session Two: 'Expanding Pathways to Publishing Careers,' was held June 13, 2024.
Beyond Degrees - Empowering the Workforce in the Context of Skills-First.pptxEduSkills OECD
Iván Bornacelly, Policy Analyst at the OECD Centre for Skills, OECD, presents at the webinar 'Tackling job market gaps with a skills-first approach' on 12 June 2024
A Visual Guide to 1 Samuel | A Tale of Two HeartsSteve Thomason
These slides walk through the story of 1 Samuel. Samuel is the last judge of Israel. The people reject God and want a king. Saul is anointed as the first king, but he is not a good king. David, the shepherd boy is anointed and Saul is envious of him. David shows honor while Saul continues to self destruct.
2. CASE HISTORYCASE HISTORY
Chief Complaint:
Swelling – One month
Pain – 15 days
History of Present Illness:
Swelling – insidious in onset, small size
Associated with pain, insidious in onset, continuous,
dull aching, localised, aggravates on chewing food.
www.indiandentalacademy.com
3. CASE HISTORYCASE HISTORY
Past Medical History:
Past Dental History:
Got teeth extracted 4yrs back due to decay and pain
and is a complete denture wearer since four months.
Personal history:
Habits: Patient smokes 25 beedies per day since 40yrs.
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5. CASE HISTORYCASE HISTORY
General Physical Examination:
Extra Oral Examination:
Lymph node Examination:
Left Submandibular lymphadenopathy.
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18. Dentinogenic Ghost Cell Tumor
First proposed by Praetorius in 1981
Synonyms:
Odontogenic Ghost Cell Tumor(Colmero et al,1986)
Calcifying Ghost Cell Odontogenic Tumor
(Fejerskov & Krogh, 1992)
Epithelial Odontogenic Ghost Cell Tumor (Gunhan
et al, 2001)
Dentinoameloblastoma (Shear)www.indiandentalacademy.com
19. Dentinogenic Ghost Cell
TumorDentinogenic ghost cell tumor is defined as solid
neoplastic growth formed by groups and islands of
epithelial cells showing an ameloblastoma-like basal
cell layer that sometime shows nuclear polarization.
Quintessance International, 1997;28(1): 45-47
www.indiandentalacademy.com
20. Dentinogenic Ghost Cell
Tumor
Hong et al classified into Two Types,
1.Central or Intraosseous type
2.Peripheral or Extraosseous type
American Journal of Neuroradiology, 2001;22:175-179
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21. Dentinogenic Ghost Cell
Tumor
Peripheral Type:
Remain localized.
Age –50-72yrs, mean age – 53.8yrs
Sex – Males
Occur on the gingiva and edentulous alveolar
ridge on alveolar mucosa.
Peripheral presentation is related to their
infiltrative behaviour and appears to be
common.
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22. Peripheral DGCT
Size varies from 1.5cm to 2cm.
Present as sessile or pedunculated exophytic nodules
of gingiva.
May be tender or non tender, hard or soft and friable.
Journal of Clinical Periodontology,2007;78(8):1635-38
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24. Histopathologic Features
Ameloblastoma like epithelial elements are seen
in association with grouped ghost cells.
Ghost cells exhibit abundant, granular
eosinophilic cytoplasm and faint nuclear outline.
Ghost cells form stratified masses with several
portions entrapped within areas of irregular
osteoid or dentinoid material.
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25. Dentinogenic Ghost Cell
Tumor
There is proliferative epithelium and ghost cells
interspersed with abundant dentinoid like
material, hence the lesion is called dentinogenic
ghost cell tumor.
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26. Immunohistochemistry
Bcl – 2 and MIB-1 - positivity for cells of
odontogenic epithelium
The ghost cells and dentinoid material are
completely negative .
Journal of Periodontology, 2007; 78(8):1635-38
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27. TREATMENT
Peripheral lesion remain localised and can be
treated with simple excision.
• Do not recur.
BJOMS, 2004; 42(2):173-75
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