Adjustment disorders and post-traumatic stress disorder (PTSD) are both mental health conditions that can develop after experiencing stressful or traumatic events. Adjustment disorders involve difficulty coping with stressors and result in emotional or behavioral symptoms that impair functioning. PTSD involves experiencing trauma symptoms like flashbacks, nightmares, and avoidance behaviors in response to a traumatic event. While adjustment disorders are usually short-lived and respond well to therapy, PTSD symptoms are more severe and long-lasting without treatment. The documents provide details on the diagnostic criteria, symptoms, prevalence, course, and treatment options for each disorder.
Course Description (From www.PESI.com):
Attend this day of training and leave with a brand new toolkit of skills, interventions, and principles for rapid success with traumatized clients. Join Jamie Marich and learn the standard of care for treatment in the field of traumatic stress – and its key ingredients. Implement evidence-based treatment protocols and interventions for establishing safety, desensitizing and reprocessing trauma memories, metabolizing and resolving grief/loss and finally, assisting clients in reconnecting to lives full of hope, connection, and achievement.
Jamie is a certified EMDR Therapist and approved consultant through the EMDR International Association (EMDR). She is additionally a member of the American Academy of Experts in Traumatic Stress, the International Association of Trauma Professionals (IATP), and has earned Certification in Disaster Thanatology.
Jamie began her career in social services as a humanitarian aid worker in post-war Bosnia-Herzegovina opening her eyes to the widespread, horrific impact of traumatic stress and grief.
Objectives:
Describe the etiology and impact of traumatic stress on the client utilizing multiple assessment strategies.
Assess a client’s reaction to a traumatic event and make an appropriate diagnosis.
Explain how grief, bereavement, and mourning are accounted for in the new DSM-5®.
Implement interventions to assist a client in dealing with the biopsychosocial manifestations of trauma, PTSD, and traumatic grief/complicated mourning.
Utilize appropriate evidence-based interventions to assist a client in dealing with the biopsychosocial-spiritual manifestations of trauma.
Explain the effects of trauma on the structure and function of the brain.
Review of the latest research in the field on grief therapy and practice tips for practitioners. Topics include:
• The difference between normal grief and complicated or prolonged grief
• Research and issues involved in the inclusion of “Prolonged Grief Disorder” in DSM-V
• Cognitive behavioral techniques to treat prolonged grief
• The importance of self-awareness and the necessity of self-care when providing grief counseling
• Different cultural views of death
Presented by Susan Stuber, Ph.D. at the Philadelphia Society of Clinical Psychologists continuing education conference at the Philadelphia College of Osteopathic Medicine, March 22, 2013. A copy of the full presentation notes accompanying these slides may be obtained by contacting Dr. Stuber at sstuber@susanstuberphd.com.
Course Description (From www.PESI.com):
Attend this day of training and leave with a brand new toolkit of skills, interventions, and principles for rapid success with traumatized clients. Join Jamie Marich and learn the standard of care for treatment in the field of traumatic stress – and its key ingredients. Implement evidence-based treatment protocols and interventions for establishing safety, desensitizing and reprocessing trauma memories, metabolizing and resolving grief/loss and finally, assisting clients in reconnecting to lives full of hope, connection, and achievement.
Jamie is a certified EMDR Therapist and approved consultant through the EMDR International Association (EMDR). She is additionally a member of the American Academy of Experts in Traumatic Stress, the International Association of Trauma Professionals (IATP), and has earned Certification in Disaster Thanatology.
Jamie began her career in social services as a humanitarian aid worker in post-war Bosnia-Herzegovina opening her eyes to the widespread, horrific impact of traumatic stress and grief.
Objectives:
Describe the etiology and impact of traumatic stress on the client utilizing multiple assessment strategies.
Assess a client’s reaction to a traumatic event and make an appropriate diagnosis.
Explain how grief, bereavement, and mourning are accounted for in the new DSM-5®.
Implement interventions to assist a client in dealing with the biopsychosocial manifestations of trauma, PTSD, and traumatic grief/complicated mourning.
Utilize appropriate evidence-based interventions to assist a client in dealing with the biopsychosocial-spiritual manifestations of trauma.
Explain the effects of trauma on the structure and function of the brain.
Review of the latest research in the field on grief therapy and practice tips for practitioners. Topics include:
• The difference between normal grief and complicated or prolonged grief
• Research and issues involved in the inclusion of “Prolonged Grief Disorder” in DSM-V
• Cognitive behavioral techniques to treat prolonged grief
• The importance of self-awareness and the necessity of self-care when providing grief counseling
• Different cultural views of death
Presented by Susan Stuber, Ph.D. at the Philadelphia Society of Clinical Psychologists continuing education conference at the Philadelphia College of Osteopathic Medicine, March 22, 2013. A copy of the full presentation notes accompanying these slides may be obtained by contacting Dr. Stuber at sstuber@susanstuberphd.com.
Adjustment disorders are commonly seen in primary care settings in which the 1-year prevalence varies from 11% to 18% of those with any clinical psychiatric disorder. [Casey PR et al., 1984]
A recent study [Maercker A et al., 2012] in the general population found the prevalence of adjustment disorder to be 0.9%,
Diagnostic Criteria:
exposure to actual or threatened death, serious, or sexual violence in one( or more) of the following ways:
1) Directly experiencing the traumatic events.
2) Witnessing in person
3) Learning that the traumatic event occur to close family member or friend.
4) Experiencing repeated or extreme exposure to aversive details of the traumatic events.
Posttraumatic stress disorder (PTSD) is an anxiety disorder that a person may develop after experiencing or witnessing an extreme, overwhelming traumatic event during which they felt intense fear, helplessness, or horror.
This ppt will provide a complete information on the topic Depression. It Will also provide the types of depression, pathophysiology involved, causes, drugs used in Depression and its management.
Adjustment disorders are commonly seen in primary care settings in which the 1-year prevalence varies from 11% to 18% of those with any clinical psychiatric disorder. [Casey PR et al., 1984]
A recent study [Maercker A et al., 2012] in the general population found the prevalence of adjustment disorder to be 0.9%,
Diagnostic Criteria:
exposure to actual or threatened death, serious, or sexual violence in one( or more) of the following ways:
1) Directly experiencing the traumatic events.
2) Witnessing in person
3) Learning that the traumatic event occur to close family member or friend.
4) Experiencing repeated or extreme exposure to aversive details of the traumatic events.
Posttraumatic stress disorder (PTSD) is an anxiety disorder that a person may develop after experiencing or witnessing an extreme, overwhelming traumatic event during which they felt intense fear, helplessness, or horror.
This ppt will provide a complete information on the topic Depression. It Will also provide the types of depression, pathophysiology involved, causes, drugs used in Depression and its management.
Mental Health in Fact-checking and Journalism: Dealing with Stress and Trauma211 Check
Mental Health in Fact-checking and Journalism: Dealing with Stress and Trauma is a presentation by Jibi Moses Daniel Associate Editor and Associate Coordinator for the 211 Check Project at Defyhatenow. He made the presentation for an audience of about 25 attendees via Zoom and YouTube Livestream with support from the International Fact-checking Netwotk (IFCN) through the BUILD Grant
What is Generalized anxiety disorder (GAD), Definition of Generalized anxiety disorder (GAD), Classification of Generalized anxiety disorder (GAD), Clinical manifestation of Generalized anxiety disorder (GAD), Risk factors and investigations of Generalized anxiety disorder (GAD), Medications and therapies for Generalized anxiety disorder (GAD),
adjustment disorders and distress in Palliative careruparnakhurana
Psychosocial disorders are very common in patients with advanced malignancies with the commonest being anxiety and depression. Early identification and treatment will help in improving the quality of life of patients and their families and increasing compliance towards treatment and self care,
Definition of ethics, Ethics and counselling,
Professional codes of ethics and standards,
the Development of Code of Ethics of
Counsellors, Ethical counselling
Seminar of U.V. Spectroscopy by SAMIR PANDASAMIR PANDA
Spectroscopy is a branch of science dealing the study of interaction of electromagnetic radiation with matter.
Ultraviolet-visible spectroscopy refers to absorption spectroscopy or reflect spectroscopy in the UV-VIS spectral region.
Ultraviolet-visible spectroscopy is an analytical method that can measure the amount of light received by the analyte.
This pdf is about the Schizophrenia.
For more details visit on YouTube; @SELF-EXPLANATORY;
https://www.youtube.com/channel/UCAiarMZDNhe1A3Rnpr_WkzA/videos
Thanks...!
A brief information about the SCOP protein database used in bioinformatics.
The Structural Classification of Proteins (SCOP) database is a comprehensive and authoritative resource for the structural and evolutionary relationships of proteins. It provides a detailed and curated classification of protein structures, grouping them into families, superfamilies, and folds based on their structural and sequence similarities.
Deep Behavioral Phenotyping in Systems Neuroscience for Functional Atlasing a...Ana Luísa Pinho
Functional Magnetic Resonance Imaging (fMRI) provides means to characterize brain activations in response to behavior. However, cognitive neuroscience has been limited to group-level effects referring to the performance of specific tasks. To obtain the functional profile of elementary cognitive mechanisms, the combination of brain responses to many tasks is required. Yet, to date, both structural atlases and parcellation-based activations do not fully account for cognitive function and still present several limitations. Further, they do not adapt overall to individual characteristics. In this talk, I will give an account of deep-behavioral phenotyping strategies, namely data-driven methods in large task-fMRI datasets, to optimize functional brain-data collection and improve inference of effects-of-interest related to mental processes. Key to this approach is the employment of fast multi-functional paradigms rich on features that can be well parametrized and, consequently, facilitate the creation of psycho-physiological constructs to be modelled with imaging data. Particular emphasis will be given to music stimuli when studying high-order cognitive mechanisms, due to their ecological nature and quality to enable complex behavior compounded by discrete entities. I will also discuss how deep-behavioral phenotyping and individualized models applied to neuroimaging data can better account for the subject-specific organization of domain-general cognitive systems in the human brain. Finally, the accumulation of functional brain signatures brings the possibility to clarify relationships among tasks and create a univocal link between brain systems and mental functions through: (1) the development of ontologies proposing an organization of cognitive processes; and (2) brain-network taxonomies describing functional specialization. To this end, tools to improve commensurability in cognitive science are necessary, such as public repositories, ontology-based platforms and automated meta-analysis tools. I will thus discuss some brain-atlasing resources currently under development, and their applicability in cognitive as well as clinical neuroscience.
Richard's entangled aventures in wonderlandRichard Gill
Since the loophole-free Bell experiments of 2020 and the Nobel prizes in physics of 2022, critics of Bell's work have retreated to the fortress of super-determinism. Now, super-determinism is a derogatory word - it just means "determinism". Palmer, Hance and Hossenfelder argue that quantum mechanics and determinism are not incompatible, using a sophisticated mathematical construction based on a subtle thinning of allowed states and measurements in quantum mechanics, such that what is left appears to make Bell's argument fail, without altering the empirical predictions of quantum mechanics. I think however that it is a smoke screen, and the slogan "lost in math" comes to my mind. I will discuss some other recent disproofs of Bell's theorem using the language of causality based on causal graphs. Causal thinking is also central to law and justice. I will mention surprising connections to my work on serial killer nurse cases, in particular the Dutch case of Lucia de Berk and the current UK case of Lucy Letby.
Slide 1: Title Slide
Extrachromosomal Inheritance
Slide 2: Introduction to Extrachromosomal Inheritance
Definition: Extrachromosomal inheritance refers to the transmission of genetic material that is not found within the nucleus.
Key Components: Involves genes located in mitochondria, chloroplasts, and plasmids.
Slide 3: Mitochondrial Inheritance
Mitochondria: Organelles responsible for energy production.
Mitochondrial DNA (mtDNA): Circular DNA molecule found in mitochondria.
Inheritance Pattern: Maternally inherited, meaning it is passed from mothers to all their offspring.
Diseases: Examples include Leber’s hereditary optic neuropathy (LHON) and mitochondrial myopathy.
Slide 4: Chloroplast Inheritance
Chloroplasts: Organelles responsible for photosynthesis in plants.
Chloroplast DNA (cpDNA): Circular DNA molecule found in chloroplasts.
Inheritance Pattern: Often maternally inherited in most plants, but can vary in some species.
Examples: Variegation in plants, where leaf color patterns are determined by chloroplast DNA.
Slide 5: Plasmid Inheritance
Plasmids: Small, circular DNA molecules found in bacteria and some eukaryotes.
Features: Can carry antibiotic resistance genes and can be transferred between cells through processes like conjugation.
Significance: Important in biotechnology for gene cloning and genetic engineering.
Slide 6: Mechanisms of Extrachromosomal Inheritance
Non-Mendelian Patterns: Do not follow Mendel’s laws of inheritance.
Cytoplasmic Segregation: During cell division, organelles like mitochondria and chloroplasts are randomly distributed to daughter cells.
Heteroplasmy: Presence of more than one type of organellar genome within a cell, leading to variation in expression.
Slide 7: Examples of Extrachromosomal Inheritance
Four O’clock Plant (Mirabilis jalapa): Shows variegated leaves due to different cpDNA in leaf cells.
Petite Mutants in Yeast: Result from mutations in mitochondrial DNA affecting respiration.
Slide 8: Importance of Extrachromosomal Inheritance
Evolution: Provides insight into the evolution of eukaryotic cells.
Medicine: Understanding mitochondrial inheritance helps in diagnosing and treating mitochondrial diseases.
Agriculture: Chloroplast inheritance can be used in plant breeding and genetic modification.
Slide 9: Recent Research and Advances
Gene Editing: Techniques like CRISPR-Cas9 are being used to edit mitochondrial and chloroplast DNA.
Therapies: Development of mitochondrial replacement therapy (MRT) for preventing mitochondrial diseases.
Slide 10: Conclusion
Summary: Extrachromosomal inheritance involves the transmission of genetic material outside the nucleus and plays a crucial role in genetics, medicine, and biotechnology.
Future Directions: Continued research and technological advancements hold promise for new treatments and applications.
Slide 11: Questions and Discussion
Invite Audience: Open the floor for any questions or further discussion on the topic.
This presentation explores a brief idea about the structural and functional attributes of nucleotides, the structure and function of genetic materials along with the impact of UV rays and pH upon them.
Multi-source connectivity as the driver of solar wind variability in the heli...Sérgio Sacani
The ambient solar wind that flls the heliosphere originates from multiple
sources in the solar corona and is highly structured. It is often described
as high-speed, relatively homogeneous, plasma streams from coronal
holes and slow-speed, highly variable, streams whose source regions are
under debate. A key goal of ESA/NASA’s Solar Orbiter mission is to identify
solar wind sources and understand what drives the complexity seen in the
heliosphere. By combining magnetic feld modelling and spectroscopic
techniques with high-resolution observations and measurements, we show
that the solar wind variability detected in situ by Solar Orbiter in March
2022 is driven by spatio-temporal changes in the magnetic connectivity to
multiple sources in the solar atmosphere. The magnetic feld footpoints
connected to the spacecraft moved from the boundaries of a coronal hole
to one active region (12961) and then across to another region (12957). This
is refected in the in situ measurements, which show the transition from fast
to highly Alfvénic then to slow solar wind that is disrupted by the arrival of
a coronal mass ejection. Our results describe solar wind variability at 0.5 au
but are applicable to near-Earth observatories.
Richard's aventures in two entangled wonderlandsRichard Gill
Since the loophole-free Bell experiments of 2020 and the Nobel prizes in physics of 2022, critics of Bell's work have retreated to the fortress of super-determinism. Now, super-determinism is a derogatory word - it just means "determinism". Palmer, Hance and Hossenfelder argue that quantum mechanics and determinism are not incompatible, using a sophisticated mathematical construction based on a subtle thinning of allowed states and measurements in quantum mechanics, such that what is left appears to make Bell's argument fail, without altering the empirical predictions of quantum mechanics. I think however that it is a smoke screen, and the slogan "lost in math" comes to my mind. I will discuss some other recent disproofs of Bell's theorem using the language of causality based on causal graphs. Causal thinking is also central to law and justice. I will mention surprising connections to my work on serial killer nurse cases, in particular the Dutch case of Lucia de Berk and the current UK case of Lucy Letby.
2. Adjustment Disorders
• 309.9 (F43.20)
• Group of conditions that can occur when one have
difficulty coping with a stressful life event
• These can include the leaving house, relationship
issues, or being fired from work
• The inability to adjust to the stressful event can cause
one or more severe psychological symptoms and
sometimes even physical symptoms
3. • There are six types of adjustment disorders,
each type with distinct symptoms and signs
• Adjustment disorders can affect both adults
and children
• These disorders are treated with therapy,
medication, or a combination of both
4. Recognizing the symptoms of
adjustment disorder
• The symptoms usually occur during or
immediately after you experience a stressful
event
• Some people have just one symptom
• Others may experience many symptoms
5. Mental symptoms
• Rebellious or impulsive actions
• Anxiousness
• Feelings of sadness, hopelessness, or being
trapped
• Crying
• Withdrawn attitude
• Lack of concentration
• Loss of self-esteem
• Suicidal thoughts
7. Types of adjustment disorder
• Adjustment disorder with depressed mood
• Adjustment disorder with anxiety
• Adjustment disorder with mixed anxiety and
depressed mood
• Adjustment disorder with disturbance of conduct
• Adjustment disorder with mixed disturbance of
emotions and conduct
• Adjustment disorder unspecified
8. Adjustment disorder with depressed
mood
People diagnosed with this type of adjustment
disorder tend to experience
Feelings of sadness and hopelessness
It’s also associated with crying
One no longer enjoy activities that they did formerly.
9. Adjustment disorder with anxiety
Symptoms include
• Feeling overwhelmed, anxious, and worried
• Problems with concentration and memory
For children,
this diagnosis is usually associated with separation
anxiety from parents and loved ones.
10. Adjustment disorder with mixed
anxiety and depressed mood
• People with this disorder experience both
depression and anxiety
• Feel hopeless and sadder than would be
expected after a stressful event
11. Adjustment disorder with disturbance
of conduct
• Symptoms involve behavioral issues like
driving recklessly or starting fights.
• Teens with this disorder may steal or vandalize
property. They might also start missing school
12. Adjustment disorder with mixed
disturbance of emotions and conduct
• Symptoms include depression, anxiety, and
behavioral problems
13. Adjustment disorder unspecified
• Those diagnosed with this have symptoms that
aren’t associated with the other types of
adjustment disorder.
• These often include physical symptoms or
problems with friends, family, work, or school.
14. What causes adjustment disorders?
• death of a family member or friend
• relationship issues or divorce
• major life changes
• moving to a new house or place
• sudden disasters
• money troubles or fears
Some common causes in adults include:
15. Con..
• Family fights or problems
• Problems in school
• Anxiety over sexuality
Typical causes in children and teenagers include:
16. Who is at risk of developing
adjustment disorder?
• Anyone can develop an adjustment disorder.
• There isn’t any way to tell who out of a group of
people experiencing the same stressor will
develop one.
• A person’s social skills and methods for coping
with other stressors may determine whether or
not they develop an adjustment disorder.
17. How is adjustment disorder diagnosed?
In order to be diagnosed with an adjustment disorder,
a person must meet the following criteria:
Criteria-A Experiencing psychological or
behavioral symptoms within three months of an
identifiable stressor or stressors occurring in
life
18. Criteria-B
These symptoms or behaviors are clinically
significant, as evidenced by one or both of the
following
Having more stress than would be ordinary in
response to a specific stressor
Significant impairment in social,
occupational, or other important areas of
functioning.
19. • Criteria-C. The stress-related
disturbance does not meet the criteria for
another mental disorder and is not
merely an exacerbation of a preexisting
mental disorder
• Criteria-D. The symptoms do not
represent normal bereavement.
20. • Criteria-E. Once the stressor or its
consequences have terminated, the symptoms
do not persist for more than an additional 6
months.
21. Diagnostic Features
• a single event (e.g., a termination of a
romantic relationship)
• multiple stressors (e.g., marked business
difficulties and marital problems)
Presence of emotional or behavioral symptoms in response to
an identifiable stressor is the essential feature of adjustment
disorders (Criterion A)
The stressor may be
22. • Recurrent stressors (e.g., associated with
seasonal business crises, unfulfilling sexual
relationships)
• continuous stressors (e.g., a persistent painful
illness with increasing disability, living in a
crime-ridden neighborhood)
• Stressors may affect a single individual, an
entire family, or a larger group or community
(e.g., a natural disaster)
23. • stressors may accompany specific
developmental events
e.g.,
Going to school,
Leaving a parental home,
Reentering a parental home,
Getting married,
25. • Adjustment disorders are associated with an
increased risk of suicide attempts and completed
suicide
26. Prevalence
• The percentage of individuals in outpatient
mental health treatment with a principal
diagnosis of an adjustment disorder ranges
from approximately 5% to 20%
27. Development and Course
• The disturbance begins within 3 months of
onset of a stressor and lasts no longer than 6
months after the stressor or its consequences
have ceased.
• If the stressor is an acute event (e.g., being
fired from a job), the onset of the disturbance
is usually immediate (i.e., within a few days)
and the duration is relatively brief
28. • If the stressor or its consequences persist, the
adjustment disorder may also continue to be
present and become the persistent form.
29. Risk and Prognostic Factors
• Individuals from disadvantaged life
circumstances experience a high rate of
stressors and may be at increased risk for
adjustment disorders.
30. Differential Diagnosis
• Major depressive disorder. If an individual has
symptoms that meet criteria for a major
depressive disorder in response to a stressor,
the diagnosis of an adjustment disorder is not
applicable.
• The symptom profile of major depressive
disorder differentiates it from adjustment
disorders.
31. Posttraumatic stress disorder and acute
stress disorder.
• In adjustment disorders, the stressor can be of
any severity rather than of the severity and
type required by Criterion A of acute stress
disorder and posttraumatic stress disorder
(PTSD).
• To distinguish adjustment disorders from these
two posttraumatic diagnoses, there are both
timing and symptom profile considerations.
32. • Adjustment disorders can be diagnosed
immediately and persist up to 6 months after
exposure to the traumatic event
• Acute stress disorder can only occur between
3 days and 1 month of exposure to the stressor
• PTSD cannot be diagnosed until at least 1
month has passed since the occurrence of the
traumatic stressor
33. • With regard to symptom profiles, an
adjustment disorder may be diagnosed when an
individual exhibits symptoms of either acute
stress disorder or PTSD that do not meet or
exceed the diagnostic threshold for either
disorder.
• adjustment disorder should also be diagnosed
for individuals who have not been exposed to a
traumatic event but who otherwise exhibit the
full symptom profile of either acute stress
disorder or PTSD.
34. Personality disorders
• Some personality features may be associated
with a vulnerability to situational distress that
may resemble an adjustment disorder
• The lifetime history of personality
functioning will help inform the interpretation
of distressed behaviors to aid in distinguishing
a long-standing personality disorder from an
adjustment disorder.
35. • In the presence of a personality disorder, if
the symptom criteria for an adjustment
disorder are met, and the stress-related
disturbance exceeds what may be attributable
to maladaptive personality disorder symptoms
(i.e.. Criterion C is met), then the diagnosis of
an adjustment disorder should be made.
36. Psychological factors affecting other
medical conditions
• specific psychological entities (e.g., psychological
symptoms, behaviors, other factors) exacerbate a
medical condition
• These psychological factors can precipitate,
exacerbate, or put an individual at risk for
medical illness, or they can worsen an existing
condition.
• In contrast, an adjustment disorder is a reaction to
the stressor (e.g., having a medical illness)
37. Normative stress reactions
• When bad things happen, most people get
upset. This is not an adjustment disorder.
• The diagnosis should only be made when the
magnitude of the distress (e.g., alterations in
mood, anxiety, or conduct) exceeds what
would normally be expected (which may vary
in different cultures) or when the adverse event
precipitates functional impairment
38. Comorbidity
• Adjustment disorders can be diagnosed in
addition to another mental disorder only if the
latter does not explain the particular symptoms
that occur in reaction to the stressor
• For example,
• an individual may develop an adjustment
disorder, with depressed mood, after losing a
job and at the same time have a diagnosis of
obsessive-compulsive disorder.
39. • An individual may have a depressive or bipolar
disorder and an adjustment disorder as long as
the criteria for both are met.
• Adjustment disorders are common
accompaniments of mental illness and may be
the major psychological response to a mental
disorder.
41. How is adjustment disorder treated?
• Adjustment disorder is typically treated
with therapy, medications, or a
combination of both.
• Therapy
• Psychotherapy
• Crisis intervention (emergency
psychological care)
42. • Family and group therapies-support
groups specific to the cause of the
adjustment disorder
• Cognitive behavioral therapy, or CBT
(which focuses on solving problems by
changing unproductive thinking and
behaviors)
• Interpersonal psychotherapy, or IPT
(short-term psychotherapy treatment)
43. Medication
• Medications are used to lessen some of the
symptoms of adjustment disorders, such as
insomnia, depression, and anxiety
• These medications include:
• Benzodiazepines, such as lorazepam (Ativan) and
alprazolam (Xanax)
• Nonbenzodiazepine anxiolytics, such as
gabapentin (Neurontin)
• SSRIs or SNRIs, such as sertraline (Zoloft) or
venlafaxine (Effexor XR)
45. Post Traumatic Stress Disorder
• DSM-5 code 309.81,
• ICD-10 code F43.10
• Post Traumatic Stress Disorder (PTSD) is a
common, treatable, but often misunderstood
behavioral health condition that can occur after
someone experiences a traumatic event.
46. Trauma
• Trauma is extreme stress that overwhelms
the person’s ability to cope
• Threat to life
• Threat of bodily harm
• Threat of sanity
• A person may feel overwhelmed
physically, emotionally and/or mentally.
47. Sources of Significant Trauma
• Violent personal assault
• Childhood physical or sexual abuse
• Being kidnapped
• Being taken hostage
• Terrorist attacks
48. • Severe natural or manmade disasters
• Severe accidents
• Being diagnosed with a life-threatening illness
One person’s trauma is not another’s
49. Diagnostic Criteria
A. Exposure to actual or threatened death, serious
injury, or sexual violence in one (or more) of the
following ways:
• Directly experiencing the traumatic event(s).
• Witnessing, in person, the event(s) as it occurred
to others.
• Learning that the traumatic event(s) occurred to
a close family member or close friend. In cases of
actual or threatened death of a family member or
friend, the event(s) must have been violent or
accidental.
50. • Experiencing repeated or extreme
exposure to aversive details of the
traumatic event(s) (e.g., first responders
collecting human remains: police officers
repeatedly exposed to details of child
abuse).
Note: Criterion A does not apply to exposure
through electronic media, television,
movies, or pictures, unless this exposure is
work related.
51. B. Presence of one (or more) of the
following intrusion symptoms associated
with the traumatic event(s), beginning
after the traumatic event(s) occurred:
Recurrent, involuntary, and
intrusive distressing memories of
the traumatic event(s).
Note: In children older than 6 years, repetitive play
may occur in which themes or aspects of the
traumatic event(s) are expressed.
52. • Recurrent distressing dreams in which the
content and/or affect of the dream are related
to the traumatic event(s).
• Dissociative reactions (e.g., flashbacks)
in which the individual feels or acts as if the
traumatic event(s) were recurring. (Such
reactions may occur on a continuum, with
the most extreme expression being a complete
loss of awareness of present
surroundings.)
Note: In children, there may be frightening dreams
without recognizable content.
53. • Note: In children, trauma-specific re-
enactment may occur in play.
• Intense or prolonged psychological
distress at exposure to internal or external
cues that symbolize or resemble an
aspect of the traumatic event(s).
• Marked physiological reactions to internal
or external cues that symbolize or
resemble an aspect of the traumatic
event(s).
54. C. Persistent avoidance of stimuli associated
with the traumatic event(s), beginning after
the traumatic event(s) occurred, as
evidenced by one or both of the following:
• Avoidance of or efforts to avoid distressing
memories, thoughts, or feelings about or
closely associated with the traumatic
event(s).
55. • Avoidance of or efforts to avoid external
reminders (people, places, conversations,
activities, objects, situations) that arouse
distressing memories, thoughts, or
feelings about or closely associated with
the traumatic event(s).
56. D. Negative alterations in cognitions and
mood associated with the traumatic event(s),
beginning or worsening after the traumatic
event(s) occurred, as evidenced by two (or
more) of the following:
• Inability to remember an important aspect
of the traumatic event(s) (typically due to
dissociative amnesia and not to other
factors such as head injury, alcohol, or
drugs).
57. • Persistent and exaggerated negative beliefs
or expectations about oneself, others, or the
world (e.g., “I am bad,” “No one can be
trusted,” ‘The world is completely
dangerous,” “My whole nervous system is
permanently ruined”).
• Persistent, distorted cognitions about the
cause or consequences of the traumatic
event(s) that lead the individual to blame
himself/herself or others.
58. • Persistent negative emotional state (e.g., fear,
horror, anger, guilt, or shame).
• Markedly diminished interest or participation
in significant activities.
• Feelings of detachment or estrangement from
others.
• Persistent inability to experience positive
emotions (e.g., inability to experience
happiness, satisfaction, or loving feelings).
59. E. Marked alterations in arousal and
reactivity associated with the traumatic
event(s), beginning or worsening after the
traumatic event(s) occurred, as evidenced by
two (or more) of the following:
• Irritable behavior and angry outbursts
(with little or no provocation) typically
expressed as verbal or physical aggression
toward people or objects.
60. • Reckless or self-destructive behavior.
• Hypervigilance.
• Exaggerated startle response.
• Problems with concentration.
• Sleep disturbance (e.g., difficulty falling or
staying asleep or restless sleep).
61. F. Duration of the disturbance (Criteria B, C, D,
and E) is more than 1 month.
G. The disturbance causes clinically significant
distress or impairment in social, occupational,
or other important areas of functioning.
H. The disturbance is not attributable to the
physiological effects of a substance (e.g.,
medication, alcohol) or another medical
condition.
62. • Specify whether: With dissociative symptoms:
The individual’s symptoms meet the criteria for
posttraumatic stress disorder, and in addition, in
response to the stressor, the individual
experiences persistent or recurrent symptoms of
either of the following:
1. Depersonalization: Persistent or recurrent
experiences of feeling detached from, and as if
one were an outside observer of, one’s mental
processes or body (e.g., feeling as though one
were in a dream; feeling a sense of unreality of
self or body or of time moving slowly).
63. 2.Dereaiization: Persistent or recurrent
experiences of unreality of surroundings (e.g., the
world around the individual is experienced as
unreal, dreamlike, distant, or distorted).
Specify if: With delayed expression: If the full
diagnostic criteria are not met until at least 6
months after the event (although the onset and
expression of some symptoms may be immediate).
Note: To use this subtype, the dissociative symptoms must not be
attributable to the physiological effects of a substance (e.g.,
blackouts, behavior during alcohol intoxication) or another
medical condition (e.g., complex partial seizures).
64. Features and Symptoms of PTSD
o Reliving the event
• Bad memories or thoughts, nightmares,
flashbacks
o Avoiding situations that are reminders of the
event
• Avoiding people or situations
• Avoiding talking about the event
65. o Negative changes in beliefs and feelings
• Feeling fear, guilt, shame or impending doom
• Lost of interest in activities
o Feeling keyed up
• Jittery, on alert, easily startled
• Difficulty concentrating or sleeping
66. PTSD for Children 6 Years and
Younger
o exposure to actual or threatened death, serious
injury, or sexual violence
• Directly experiencing the traumatic event(s).
• Witnessing, in person, the event(s) as it
occurred to others, especially primary
caregivers.
67. • Note: Witnessing does not include events that
are witnessed only in electronic media,
television, movies, or pictures.
• Learning that the traumatic event(s) occurred
to a parent or caregiving figure.
68. o Presence of one (or more) of the intrusion
symptoms associated with the traumatic
event(s), beginning after the traumatic event(s)
occurred:
• Recurrent, involuntary, and intrusive
distressing memories of the traumatic event(s).
• Recurrent distressing dreams in which the
content and/or affect of the dream are related to
the traumatic event(s).
69. • Dissociative reactions
In which the child feels or acts as if the
traumatic event(s) were recurring.
• Intense or prolonged psychological distress at
exposure to internal or external cues that
symbolize or resemble an aspect of the
traumatic event(s).
• Marked physiological reactions to reminders of
the traumatic event(s).
70. o One (or more) of the following symptoms,
• representing either persistent avoidance of
stimuli associated with the traumatic
event(s)
• or negative alterations in cognitions and
mood associated with the traumatic
event(s),
• must be present, beginning after the
event(s) or worsening after the event(s):
71. Persistent Avoidance of Stimuli
Avoidance of or efforts to avoid activities,
places, or physical reminders that arouse
recollections of the traumatic event(s).
Avoidance of or efforts to avoid people,
conversations, or interpersonal situations that
arouse recollections of the traumatic event(s).
72. Negative Alterations in Cognitions
Substantially increased frequency of negative
emotional states (e.g., fear, guilt, sadness, shame,
confusion).
Markedly diminished interest or participation in
significant activities, including constriction of
play.
Socially withdrawn behavior. 6. Persistent
reduction in expression of positive emotions.
73. o Alterations in arousal and reactivity associated
with the traumatic event(s), beginning or
worsening after the traumatic event(s)
occurred, as evidenced by two (or more) of the
following:
• Irritable behavior and angry outbursts (with
little or no provocation) typically expressed as
verbal or physical aggression toward people or
objects (including extreme temper tantrums).
74. • Hypervigilance.
• Exaggerated startle response.
• Problems with concentration.
• Sleep disturbance (e.g., difficulty falling or
staying asleep or restless sleep).
75. o The duration of the disturbance is more than 1
month
o The disturbance causes clinically significant
distress or impairment in relationships with
parents, siblings, peers, or other caregivers or
with school behavior.
o The disturbance is not attributable to the
physiological effects of a substance (e.g.,
medication or alcohol) or another medical
condition.
76. • Specify whether: With dissociative symptoms:
The individual’s symptoms meet the criteria
for PTSD, and the individual experiences
persistent or recurrent symptoms of either of
the following:
Depersonalization: Persistent or recurrent
experiences of feeling detached from, and as if
one were an outside observer of, one’s mental
processes or body (e.g., feeling as though one
were in a dream; feeling a sense of unreality
of self or body or of time moving slowly).
77. Derealization: Persistent or recurrent
experiences of unreality of surroundings (e.g.,
the world around the individual is experienced
as unreal, dreamlike, distant, or distorted).
To use this subtype, the dissociative symptoms
must not be attributable to the physiological
effects of a substance (e.g., blackouts) or
another medical condition
78. Other Issues Associated with PTSD
oDepression, anxiety and substance abuse
oIncreased rates of unemployment,
divorce, separation, and spousal abuse
oPhysical symptoms and possible changes
in brain structure and activity
79. The Course of PTSD
o Longer than 1 month and may last for
months or years
o Symptoms may develop immediately or they
may emerge months or years after the trauma
o Symptoms may arise suddenly or gradually
over time
80. Risk and Resiliency Factors
Risk Factors
• Being injured during
the event
• Seeing others hurt or
killed
• Feelings of horror,
helplessness or
extreme fear
Resiliency Factors
• Having a good
support network
before the event
• Seeking out support
from family and
friends
• Finding a support
group after the event
81. Con..
Risk Factors
• Having little or no
social support after the
event
• Presence of extra stress
after the event, (loss of a
loved one, pain, injury,
loss of job or home)
• History of mental illness
Resiliency Factors
• Feeling good about
one’s own actions in the
face of danger
• Having a coping
strategy
• Being able to act and
respond effectively
despite feeling fear
82. Prevalence
• The prevalence of PTSD may vary across
development; children and adolescents,
including preschool children, generally have
displayed lower prevalence following exposure
to serious traumatic events;
• The prevalence of full-threshold PTSD also
appears to be lower among older adults
compared with the general population
83. • Compared with U.S. non-Latino whites, higher
rates of PTSD have been reported among U.S.
Latinos, African Americans, and American
Indians, and lower rates have been reported
among Asian Americans, after adjustment for
traumatic exposure and demographic variables.
84. Risk and Prognostic Factors
Risk (and protective) factors are generally
divided into
• Pretraumatic factors
• Peritraumatic factors
• Posttraumatic factors
85. Pretraumatic factors
• Temperamental. These include childhood
emotional problems by age 6 years (e.g., prior
traumatic exposure, externalizing or anxiety
problems) and prior mental disorders (e.g.,
panic disorder, depressive disorder, PTSD, or
obsessive-compulsive disorder [OCD]).
86. • Environmental. These include lower
socioeconomic status; lower education;
exposure to prior trauma (especially during
childhood); childhood adversity (e.g.,
economic deprivation, family dysRinction,
parental separation or death); cultural
characteristics (e.g., fatalistic or self-blaming
coping strategies); lower intelligence; minority
racial/ethnic status; and a family psychiatric
history. Social support prior to event exposure
is protective.
87. • Genetic and physiological. These include
female gender and younger age at the time of
trauma exposure (for adults). Certain
genotypes may either be protective or increase
risk of PTSD after exposure to traumatic
events.
88. Peritraumatic factors
• Environmental. These include severity (dose) of
the trauma (the greater the magnitude of trauma,
the greater the likelihood of PTSD), perceived life
threat, personal injury, interpersonal violence
(particularly trauma involving a witnessed threat
to a caregiver in children), and, for military
personnel, being a perpetrator, witnessing
atrocities, or killing the enemy. Finally,
dissociation that occurs during the trauma and
persists afterward is a risk factor.
89. Posttraumatic factors
• Temperamental. These include negative
appraisals, inappropriate coping strategies, and
development of acute stress disorder.
• Environmental. These include subsequent
exposure to repeated upsetting reminders,
subsequent adverse life events, and financial or
other trauma-related losses. Social support
(including family stability, for children) is a
protective factor that moderates outcome after
trauma.
90. Suicide Risk
• Traumatic events such as childhood abuse
increase a person's suicide risk.
• PTSD is associated with suicidal ideation and
suicide attempts, and presence of the disorder
may indicate which individuals with ideation
eventually make a suicide plan or actually
attempt suicide.
91. Differential Diagnosis
Adjustment disorders.
• In adjustment disorders, the stressor can be of any
severity or type rather than that required by PTSD
Criterion A
• The diagnosis of an adjustment disorder is used
when the response to a stressor that meets PTSD
Criterion A does not meet all other PTSD criteria
(or criteria for another mental disorder)
92. • An adjustment disorder is also diagnosed when
the symptom pattern of PTSD occurs in
response to a stressor that does not meet PTSD
Criterion A (e.g., spouse leaving, being fired).
93. Other posttraumatic disorders and
conditions.
• Not all psychopathology that occurs in individuals
exposed to an extreme stressor should necessarily be
attributed to PTSD
• The diagnosis requires that trauma exposure precede
the onset or exacerbation of pertinent symptoms.
• if the symptom response pattern to the extreme
stressor meets criteria for another mental disorder,
these diagnoses should be given instead of, or in
addition to, PTSD
94. • Other diagnoses and conditions are excluded if
they are better explained by PTSD (e.g.,
symptoms of panic disorder that occur only
after exposure to traumatic reminders)
• If severe, symptom response patterns to the
extreme stressor may warrant a separate
diagnosis (e.g., dissociative amnesia).
95. Acute stress disorder
• Acute stress disorder is distinguished from
PTSD because the symptom pattern in acute
stress disorder is restricted to a duration of 3
days to 1 month following exposure to the
traumatic event.
96. Anxiety disorders and obsessive-
compulsive disorder
• In OCD, there are recurrent intrusive thoughts,
but these meet the definition of an obsession.
• In addition, the intrusive thoughts are not
related to an experienced traumatic event,
compulsions are usually present, and other
symptoms of PTSD or acute stress disorder are
typically absent
97. • Neither the arousal and dissociative symptoms
of panic disorder nor the avoidance, irritability,
and anxiety of generalized anxiety disorder are
associated with a specific traumatic event
• The symptoms of separation anxiety disorder
are clearly related to separation from home or
family, rather than to a traumatic event
98. Major depressive disorder
• Major depression may or may not be preceded
by a traumatic event and should be diagnosed
if other PTSD symptoms are absent
• Specifically, major depressive disorder does
not include any PTSD Criterion B or C
symptoms.
• Or does it include a number of symptoms from
PTSD Criterion D or E.
99. Personality disorders
• Interpersonal difficulties that had their onset,
or were greatly exacerbated, after exposure to
a traumatic event may be an indication of
PTSD, rather than a personality disorder, in
which such difficulties would be expected
independently of any traumatic exposure.
100. Dissociative disorders
• Dissociative amnesia, dissociative identity
disorder, and de- personalization,de-realization
disorder may or may not be preceded by
exposure to a traumatic event or may or may
not have co-occurring PTSD symptoms. When
full PTSD criteria are also met, however, the
PTSD ''with dissociative symptoms" subtype
should be considered.
101. Conversion disorder (functional
neurological symptom disorder)
• New onset of somatic symptoms within the
context of posttraumatic distress might be an
indication of PTSD rather than conversion
disorder (functional neurological symptom
disorder).
102. Psychotic disorders.
• Flashbacks in PTSD must be distinguished
from illusions, hallucinations, and other
perceptual disturbances that may occur in
schizophrenia, brief psychotic disorder, and
other psychotic disorders; depressive and
bipolar disorders with psychotic features;
delirium; substance/medication-induced
disorders; and psychotic disorders due to
another medical condition.
103. Traumatic brain injury
• When a brain injury occurs in the context of a
traumatic event (e.g., traumatic accident, bomb
blast, acceleration/deceleration trauma),
symptoms of PTSD may appear.
104. • An event causing head trauma may also
constitute a psychological traumatic event, and
traumatic brain injury (TBI)-related
neurocognitive symptoms are not mutually
exclusive and may occur concurrently.
• Symptoms previously termed postconcussive
(e.g., headaches, dizziness, sensitivity to light or
sound, irritability, concentration deficits) can
occur in brain- injured and non-brain-injured
populations, including individuals with PTSD
105. • Because symptoms of PTSD and TBI-related
neurocognitive symptoms can overlap, a
differential diagnosis between PTSD and
neurocognitive disorder symptoms attributable
to TBI may be possible based on the presence
of symptoms that are distinctive to each
presentation.
106. • Whereas re-experiencing and avoidance are
characteristic of PTSD and not the effects of
TBI, persistent disorientation and confusion
are more specific to TBI (neurocognitive
effects) than to PTSD.
107. Comorbidity
• Individuals with PTSD are 80% more likely
than those without PTSD to have symptoms
that meet diagnostic criteria for at least one
other mental disorder (e.g., depressive, bipolar,
anxiety, or substance use disorders).
108. • Comorbid substance use disorder and conduct
disorder are more common among males than
among females.
• Among U.S. military personnel and combat
veterans who have been deployed to recent
wars in Afghanistan and Iraq, co-occurrence of
PTSD and mild TBI is 48%
109. • Although most young children with PTSD also
have at least one other diagnosis, the patterns
of comorbidity are different than in adults,
with oppositional defiant disorder and
separation anxiety disorder predominating.
• Finally, there is considerable comorbidity
between PTSD and major neurocognitive
disorder and some overlapping symptoms
between these disorders.
111. Treatment Options
o Psychotherapy
o Medication
• Helps control symptoms like sadness, worry,
anger and feeling numb
• Some people may experience side effects
• Does not have to be permanent
112. Myths about PTSD
“People with PTSD are violent and
unpredictable”
FACT: The presence of PTSD does not make
someone more prone to violence. Factors to
consider are:
Alcohol/drug misuse, Past criminal history,
Having witnessed family violence, High
anger/irritability
113. Myths about PTSD
People with PTSD will never recover or if they
do, they will never be “right.”
FACT: Most people with PTSD recover and
many recover completely and live happy and
productive lives.