The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
offering a wide range of dental certified courses in different formats.
The trigeminal nerve is the 5th cranial nerve and is a mixed nerve responsible for sensation in the face and motor function of muscles of mastication. It has 3 main divisions - the ophthalmic, maxillary, and mandibular nerves. The mandibular nerve is the largest division and supplies motor innervation to the muscles of mastication as well as sensory innervation to parts of the face and scalp.
Maxillary sinus is the largest of the paranasal sinuses. It develops from a shallow groove in the maxilla and reaches its maximum size by age 18. It has multiple walls and communicates with the nasal cavity via the osteum. Maxillary sinusitis can result from dental issues like periapical abscesses, cysts, foreign bodies or trauma. Odontogenic tumors and cysts can also involve the maxillary sinus. Care must be taken during dental procedures near the maxillary sinus to prevent oroantral communications.
This document provides an overview of the trigeminal nerve (CN V), including its nuclei, functional components, course and distribution, the trigeminal ganglion, and the three divisions of the trigeminal nerve - ophthalmic, maxillary, and mandibular nerves. It describes the sensory and motor nuclei of the trigeminal nerve in the brainstem and discusses the sensory and motor roots. It also outlines the anatomy and branches of the three divisions of the trigeminal nerve.
Rene LeFort in 1901 classified midface fractures based on the level of injury into 3 types: Lefort I, II, and III fractures. Since then there have been modifications to the classification system. Lefort I fractures involve the maxilla above the teeth and nasal floor. Treatment involves either closed or open reduction with fixation methods like miniplates or wires. Potential complications include nerve damage, infection, malocclusion, and sinus issues. Contemporary approaches emphasize early open reduction and anatomical fixation with miniplates to achieve accurate reconstruction of the midface structural pillars.
This document discusses mandibular fractures, including:
- The anatomy and common sites of fracture in the mandible.
- Various classification systems used to describe fracture location and complexity.
- Clinical signs seen with mandibular fractures like swelling, step deformities, and malocclusion.
- Radiographic tools like panoramic x-rays, CT scans, and occlusal views used to diagnose and characterize fractures.
- Principles of managing mandibular fractures through techniques like open reduction and internal fixation.
Local Anesthesia in Oral and Maxillofacial SurgerySapna Vadera
Local anaesthesia is a loss of sensation in a circumscribed area without loss of consciousness. The document discusses the history of local anaesthesia from ancient times to modern developments. It also covers the desirable properties, mechanisms of action, classifications, pharmacology and clinical aspects of local anaesthetics. The summary provides a high-level overview of the key topics covered in the document relating to the definition, history, properties and mechanisms of local anaesthetics.
The gingiva is a masticatory mucosa that covers the alveolar process of the jaw and surrounds the neck of the teeth. It is made up of epithelium and connective tissue. The gingiva can be divided into three types - free gingiva, gingival sulcus, and attached gingiva. Microscopically, the gingival epithelium consists of outer oral epithelium, sulcular epithelium, and junctional epithelium. The gingiva also contains dense collagen fibers called the gingival ligament. Blood supply to the gingiva is provided by the alveolar artery and it receives nerve innervation from various nerves depending on location.
This document provides an overview of surveying and surveying tools used in the process of designing removable partial dentures (RPDs). It discusses the history and development of surveying, types of surveyors, principles of surveying including survey lines and path of insertion. The document outlines the step-by-step survey process including orienting the cast, tilting, marking survey lines, measuring undercuts, identifying interferences, and tripoding the cast for future reference. Various surveying tools such as the analyzing rod, carbon marker, undercut gauges, and wax trimmers are also described.
The trigeminal nerve is the 5th cranial nerve and is a mixed nerve responsible for sensation in the face and motor function of muscles of mastication. It has 3 main divisions - the ophthalmic, maxillary, and mandibular nerves. The mandibular nerve is the largest division and supplies motor innervation to the muscles of mastication as well as sensory innervation to parts of the face and scalp.
Maxillary sinus is the largest of the paranasal sinuses. It develops from a shallow groove in the maxilla and reaches its maximum size by age 18. It has multiple walls and communicates with the nasal cavity via the osteum. Maxillary sinusitis can result from dental issues like periapical abscesses, cysts, foreign bodies or trauma. Odontogenic tumors and cysts can also involve the maxillary sinus. Care must be taken during dental procedures near the maxillary sinus to prevent oroantral communications.
This document provides an overview of the trigeminal nerve (CN V), including its nuclei, functional components, course and distribution, the trigeminal ganglion, and the three divisions of the trigeminal nerve - ophthalmic, maxillary, and mandibular nerves. It describes the sensory and motor nuclei of the trigeminal nerve in the brainstem and discusses the sensory and motor roots. It also outlines the anatomy and branches of the three divisions of the trigeminal nerve.
Rene LeFort in 1901 classified midface fractures based on the level of injury into 3 types: Lefort I, II, and III fractures. Since then there have been modifications to the classification system. Lefort I fractures involve the maxilla above the teeth and nasal floor. Treatment involves either closed or open reduction with fixation methods like miniplates or wires. Potential complications include nerve damage, infection, malocclusion, and sinus issues. Contemporary approaches emphasize early open reduction and anatomical fixation with miniplates to achieve accurate reconstruction of the midface structural pillars.
This document discusses mandibular fractures, including:
- The anatomy and common sites of fracture in the mandible.
- Various classification systems used to describe fracture location and complexity.
- Clinical signs seen with mandibular fractures like swelling, step deformities, and malocclusion.
- Radiographic tools like panoramic x-rays, CT scans, and occlusal views used to diagnose and characterize fractures.
- Principles of managing mandibular fractures through techniques like open reduction and internal fixation.
Local Anesthesia in Oral and Maxillofacial SurgerySapna Vadera
Local anaesthesia is a loss of sensation in a circumscribed area without loss of consciousness. The document discusses the history of local anaesthesia from ancient times to modern developments. It also covers the desirable properties, mechanisms of action, classifications, pharmacology and clinical aspects of local anaesthetics. The summary provides a high-level overview of the key topics covered in the document relating to the definition, history, properties and mechanisms of local anaesthetics.
The gingiva is a masticatory mucosa that covers the alveolar process of the jaw and surrounds the neck of the teeth. It is made up of epithelium and connective tissue. The gingiva can be divided into three types - free gingiva, gingival sulcus, and attached gingiva. Microscopically, the gingival epithelium consists of outer oral epithelium, sulcular epithelium, and junctional epithelium. The gingiva also contains dense collagen fibers called the gingival ligament. Blood supply to the gingiva is provided by the alveolar artery and it receives nerve innervation from various nerves depending on location.
This document provides an overview of surveying and surveying tools used in the process of designing removable partial dentures (RPDs). It discusses the history and development of surveying, types of surveyors, principles of surveying including survey lines and path of insertion. The document outlines the step-by-step survey process including orienting the cast, tilting, marking survey lines, measuring undercuts, identifying interferences, and tripoding the cast for future reference. Various surveying tools such as the analyzing rod, carbon marker, undercut gauges, and wax trimmers are also described.
Gow gates & vazirani akinosi technique of nervePOOJAKUMARI277
The document summarizes two techniques for mandibular nerve blocks - the Gow-Gates technique and the Vazirani-Akinosi closed mouth technique.
The Gow-Gates technique involves injecting the anesthetic at the neck of the condyle using intraoral and extraoral landmarks to block the mandibular nerve. It provides anesthesia of the mandibular teeth and surrounding soft tissues with a single injection. The Vazirani-Akinosi technique is done with the patient's mouth closed by inserting the needle through the mucosa at the level of the maxillary molar junction to block the mandibular nerve. Both techniques effectively anesthetize the mandibular region for dental
The gingiva is divided anatomically into the marginal, attached, and interdental gingiva. The marginal gingiva forms the soft tissue wall around the teeth. The attached gingiva is firmly bound to the underlying bone. The interdental gingiva occupies the spaces between teeth. Microscopically, the gingiva contains an epithelial layer and underlying connective tissue. The epithelial layer includes the sulcular, junctional, and oral epithelium. The connective tissue contains collagen, fibroblasts, and ground substance.
This document provides an overview of fibro-osseous lesions of the jaws. It discusses the classification of these lesions, which include fibrous dysplasia, ossifying fibroma, cemento-osseous dysplasia, central giant cell granuloma, cherubism, aneurysmal bone cyst, and solitary bone cyst. It focuses on the etiology, pathophysiology, clinical features, and oral manifestations of fibrous dysplasia, including monostotic fibrous dysplasia, polyostotic fibrous dysplasia, Jaffe's lichtenstein syndrome, McCune-Albright syndrome, and craniofacial fibrous dysplasia.
This document discusses odontogenic keratocysts (OKCs), a type of jaw cyst. It covers the classification, causes, histopathology, clinical features, radiographic features, differential diagnosis, treatment principles, and surgical treatment options for OKCs. OKCs most commonly occur in the mandibular molar and ramus areas and are often radiolucent and multilocular in appearance on radiographs. Treatment options include wide surgical excision or marsupialization to prevent recurrence of these cysts which have a high rate of recurrence compared to other jaw cysts.
Mandibular Anesthesia : Inferior alveolar nerve blockد.عبد الله الناصر
This document provides information on the inferior alveolar nerve block (IANB) dental anesthesia technique. It summarizes that the IANB anesthetizes the inferior alveolar nerve and its branches, anesthetizing the mandibular teeth and surrounding soft tissues. The technique involves locating the coronoid notch and pterygomandibular raphe landmarks and inserting the needle at the intersection of lines based on these landmarks, advancing the needle until bone contact is made at a depth of 20-25mm. Proper administration results in numbness of the lower lip and tongue, indicating successful anesthesia of the mental and lingual nerves. Precautions include avoiding deposition without bone contact to prevent facial nerve injury.
This document provides an overview of oral submucous fibrosis (OSF), including its definition, epidemiology, classification, etiology, pathogenesis, clinical features, and histopathology. OSF is a chronic disease characterized by inflammation and fibrosis of the submucosal tissues caused by chewing areca nut. It predominantly affects people from South Asia and is associated with significantly increased risk of oral cancer. The areca nut alkaloid arecoline is the main causative agent, inducing fibrosis through oxidative damage, upregulation of growth factors and cytokines, and inhibition of collagen degradation. Clinically, OSF presents with burning sensation and scarring that results in restricted mouth opening and tongue movement.
Indian Dental Academy is a leader in continuing dental education, providing both online and offline courses. The document discusses ameloblastoma, a type of odontogenic tumor. It defines ameloblastoma, provides its history and classifications including clinical, radiological, and histopathological. Treatment options discussed include medical therapy, radiotherapy, and various surgical treatments such as enucleation, marsupialization, and curettage. Radiographs, biopsy, CT, and MRI are investigated for ameloblastoma.
This document provides an overview of internal derangements of the temporomandibular joint (TMJ). It defines internal derangement as an abnormal relationship between the articular disc and condyle. The most common type is anterior disc displacement, which can be with or without reduction. Causes include trauma, functional overloading, joint laxity, and muscle spasms. Symptoms vary depending on the type but may include clicking, limited opening, and pain. Diagnosis involves clinical exams and MRI imaging. Treatment ranges from splint therapy to arthrocentesis or arthroscopy for lavage and relief of adhesions. Arthrocentesis is shown to improve opening and reduce pain by removing inflammatory factors from the
The document discusses the facial nerve (cranial nerve VII) in three paragraphs or less:
The facial nerve controls muscles of the face and allows for facial expressions. It has both motor and sensory components that originate from different embryonic structures and nuclei. The nerve exits the skull through the stylomastoid foramen and gives off five terminal branches innervating various facial muscles. Facial nerve palsy can result from lesions along the nerve's course and have varying clinical presentations depending on the location of injury. Common causes include Bell's palsy, tumors, trauma, and infections. Differential diagnosis and management involve identifying the underlying etiology.
Orthodontic Case History and ExaminationAhmed Gamil
The document discusses essential and supplemental diagnostic aids used in orthodontic diagnosis. Essential aids include case history, clinical examination, study models, and certain radiographs. Supplemental aids provide additional information and include specialized radiographs, electromyography, and biopsy. A thorough case history covers chief complaint, medical/dental history, and family history. Clinical examination assesses facial symmetry, profile, jaw relationships, and soft tissues. Essential radiographs aid in case evaluation and treatment planning.
TMJ is a ginglymo-diarthroidal joint that is freely mobile with superior and inferior joint spaces separated by articular disc.
The type of imaging technique depends upon the clinical problems associated, so either imaging of hard tissue (OSSEOUS) or soft tissue is desired.
Certain protocols are to be taken care before the imaging procedure:
the amount of diagnostic information available from particular imaging modality.
The cost of examination
The radiation dose
Muscles of mastication are the group of muscles that help in movement of the mandible as during chewing and speech. We need to study these muscles as they control the opening & closing the mouth & their role in the equilibrium created within the mouth. They also play a role in the configuration of face.
This document provides an overview of halitosis (bad breath), including its classification, etiology, diagnosis, and management. It discusses the role of volatile sulfur compounds and certain bacteria in causing halitosis. Diagnostic tools include organoleptic measurement, gas chromatography, and volatile sulfide monitoring to detect these compounds. Treatment involves identifying and addressing the underlying causes, such as periodontal disease, dry mouth, dental caries, or systemic conditions. Preventive measures focus on proper oral hygiene and avoiding foods that can cause temporary halitosis.
Cementum is the mineralized tissue covering the roots of teeth that provides attachment for collagen fibers linking the tooth to surrounding bone. It begins at the cementoenamel junction and continues along the root to the apex. Cementum is avascular and less hard than dentin. It contains both inorganic minerals and organic materials including collagen. Cementoblast cells synthesize cementum by laying down an organic matrix that subsequently mineralizes. Cementum thickness varies along the root and increases with age. It provides for functional adaptation and resistance to resorption during orthodontic tooth movement.
This document discusses oroantral communications and fistulas. It defines them as abnormal connections between the oral and maxillary sinus cavities. Causes include tooth extraction, tumors, cysts, and trauma. Signs and symptoms may include unpleasant taste/odor, fluid/food reflux into the nose, and air leakage. Examination involves inspection, suctioning the socket, and radiographs. Management includes immediate closure attempts and antibiotics to prevent sinusitis. Surgical techniques like buccal and palatal flaps are used for larger defects or fistulas based on factors like location, size, and presence of infection.
The maxillary nerve is the second division of the trigeminal nerve. It originates in the lateral wall of the cavernous sinus and passes through the foramen rotundum into the pterygopalatine fossa. It gives off several branches in the pterygopalatine fossa and infratemporal fossa, including the posterior superior alveolar nerve, zygomatic nerve, and anterior and middle superior alveolar nerves. The maxillary nerve terminates by innervating structures in the face, providing sensation to the maxillary region. A maxillary nerve block can provide anesthesia for dental procedures involving the maxillary area, such as when a large canine space abscess is
Odontogenic keratocyst (OKC) is the cyst arising from the cell rests of dental lamina. It can occur anywhere in the jaw, but commonly seen in the posterior part of the mandible. Radiographically, most OKCs are unilocular when presented at the periapex and can be mistaken for radicular or lateral periodontal cyst.
This document provides an overview of the muscles of mastication. It begins by defining muscle and mastication. It then discusses the development, classification, properties and functions of the primary muscles of mastication - the masseter, temporalis, lateral pterygoid, and medial pterygoid muscles. The document also covers the accessory muscles of mastication and their functions. Finally, it discusses some clinical considerations regarding these muscles, including bruxism, tetanus, and myofascial pain disorders.
Vestibuloplasty is a surgical procedure to deepen the oral vestibule by changing the attachments of the soft tissue. There are several types of vestibuloplasty procedures, including mucosal advancement, secondary epithelization, and grafting. Mucosal advancement involves undermining and advancing the oral mucosa, while secondary epithelization uses the oral mucosa to line one side and allows the other side to heal through epithelization. Grafting can use skin, mucosa, or dermis grafts to line the extended vestibule. The document discusses techniques for each type of vestibuloplasty procedure.
This document discusses Ardita Vata (facial palsy) according to Ayurveda and modern medicine. It defines Ardita Vata as a deviation leading to deformity of one side of the face or half of the body. The causes, symptoms and types are described according to Ayurvedic texts like Charaka Samhita. Bell's palsy, the most common type of facial palsy, is also discussed in detail including causes like infection, trauma and tumors. The document compares unilateral lower motor neuron lesions in Bell's palsy versus bilateral upper motor neuron lesions. Various treatment approaches are summarized including nerve grafts, slings and weights to correct facial paralysis.
Bell's palsy is a form of temporary facial paralysis resulting from damage or trauma to the facial nerves. The facial nerve-also called the 7th cranial nerve-travels through a narrow, bony canal (called the Fallopian canal) in the skull, beneath the ear, to the muscles on each side of the face. For most of its journey, the nerve is encased in this bony shell.
Each facial nerve directs the muscles on one side of the face, including those that control eye blinking and closing, and facial expressions such as smiling and frowning. Additionally, the facial nerves carries nerve impulses to the lacrimal or tear glands, the saliva glands, and the muscles of a small bone in the middle of the ear called the stapes. The facial nerve also transmits taste sensations from the tongue.
When Bell's palsy occurs, the function of the facial nerve is disrupted; causing an interruption in the messages the brain sends to the facial muscles. This interruption results in facial weakness or paralysis.
Bell's palsy is named for Sir Charles Bell, a 19th century Scottish surgeon who was the first to describe the condition. The disorder, which is not related to stroke, is the most common cause of facial paralysis. Generally, Bell's palsy affects only one of the paired facial nerves and one side of the face, however, in rare cases, it can affect both sides.
Gow gates & vazirani akinosi technique of nervePOOJAKUMARI277
The document summarizes two techniques for mandibular nerve blocks - the Gow-Gates technique and the Vazirani-Akinosi closed mouth technique.
The Gow-Gates technique involves injecting the anesthetic at the neck of the condyle using intraoral and extraoral landmarks to block the mandibular nerve. It provides anesthesia of the mandibular teeth and surrounding soft tissues with a single injection. The Vazirani-Akinosi technique is done with the patient's mouth closed by inserting the needle through the mucosa at the level of the maxillary molar junction to block the mandibular nerve. Both techniques effectively anesthetize the mandibular region for dental
The gingiva is divided anatomically into the marginal, attached, and interdental gingiva. The marginal gingiva forms the soft tissue wall around the teeth. The attached gingiva is firmly bound to the underlying bone. The interdental gingiva occupies the spaces between teeth. Microscopically, the gingiva contains an epithelial layer and underlying connective tissue. The epithelial layer includes the sulcular, junctional, and oral epithelium. The connective tissue contains collagen, fibroblasts, and ground substance.
This document provides an overview of fibro-osseous lesions of the jaws. It discusses the classification of these lesions, which include fibrous dysplasia, ossifying fibroma, cemento-osseous dysplasia, central giant cell granuloma, cherubism, aneurysmal bone cyst, and solitary bone cyst. It focuses on the etiology, pathophysiology, clinical features, and oral manifestations of fibrous dysplasia, including monostotic fibrous dysplasia, polyostotic fibrous dysplasia, Jaffe's lichtenstein syndrome, McCune-Albright syndrome, and craniofacial fibrous dysplasia.
This document discusses odontogenic keratocysts (OKCs), a type of jaw cyst. It covers the classification, causes, histopathology, clinical features, radiographic features, differential diagnosis, treatment principles, and surgical treatment options for OKCs. OKCs most commonly occur in the mandibular molar and ramus areas and are often radiolucent and multilocular in appearance on radiographs. Treatment options include wide surgical excision or marsupialization to prevent recurrence of these cysts which have a high rate of recurrence compared to other jaw cysts.
Mandibular Anesthesia : Inferior alveolar nerve blockد.عبد الله الناصر
This document provides information on the inferior alveolar nerve block (IANB) dental anesthesia technique. It summarizes that the IANB anesthetizes the inferior alveolar nerve and its branches, anesthetizing the mandibular teeth and surrounding soft tissues. The technique involves locating the coronoid notch and pterygomandibular raphe landmarks and inserting the needle at the intersection of lines based on these landmarks, advancing the needle until bone contact is made at a depth of 20-25mm. Proper administration results in numbness of the lower lip and tongue, indicating successful anesthesia of the mental and lingual nerves. Precautions include avoiding deposition without bone contact to prevent facial nerve injury.
This document provides an overview of oral submucous fibrosis (OSF), including its definition, epidemiology, classification, etiology, pathogenesis, clinical features, and histopathology. OSF is a chronic disease characterized by inflammation and fibrosis of the submucosal tissues caused by chewing areca nut. It predominantly affects people from South Asia and is associated with significantly increased risk of oral cancer. The areca nut alkaloid arecoline is the main causative agent, inducing fibrosis through oxidative damage, upregulation of growth factors and cytokines, and inhibition of collagen degradation. Clinically, OSF presents with burning sensation and scarring that results in restricted mouth opening and tongue movement.
Indian Dental Academy is a leader in continuing dental education, providing both online and offline courses. The document discusses ameloblastoma, a type of odontogenic tumor. It defines ameloblastoma, provides its history and classifications including clinical, radiological, and histopathological. Treatment options discussed include medical therapy, radiotherapy, and various surgical treatments such as enucleation, marsupialization, and curettage. Radiographs, biopsy, CT, and MRI are investigated for ameloblastoma.
This document provides an overview of internal derangements of the temporomandibular joint (TMJ). It defines internal derangement as an abnormal relationship between the articular disc and condyle. The most common type is anterior disc displacement, which can be with or without reduction. Causes include trauma, functional overloading, joint laxity, and muscle spasms. Symptoms vary depending on the type but may include clicking, limited opening, and pain. Diagnosis involves clinical exams and MRI imaging. Treatment ranges from splint therapy to arthrocentesis or arthroscopy for lavage and relief of adhesions. Arthrocentesis is shown to improve opening and reduce pain by removing inflammatory factors from the
The document discusses the facial nerve (cranial nerve VII) in three paragraphs or less:
The facial nerve controls muscles of the face and allows for facial expressions. It has both motor and sensory components that originate from different embryonic structures and nuclei. The nerve exits the skull through the stylomastoid foramen and gives off five terminal branches innervating various facial muscles. Facial nerve palsy can result from lesions along the nerve's course and have varying clinical presentations depending on the location of injury. Common causes include Bell's palsy, tumors, trauma, and infections. Differential diagnosis and management involve identifying the underlying etiology.
Orthodontic Case History and ExaminationAhmed Gamil
The document discusses essential and supplemental diagnostic aids used in orthodontic diagnosis. Essential aids include case history, clinical examination, study models, and certain radiographs. Supplemental aids provide additional information and include specialized radiographs, electromyography, and biopsy. A thorough case history covers chief complaint, medical/dental history, and family history. Clinical examination assesses facial symmetry, profile, jaw relationships, and soft tissues. Essential radiographs aid in case evaluation and treatment planning.
TMJ is a ginglymo-diarthroidal joint that is freely mobile with superior and inferior joint spaces separated by articular disc.
The type of imaging technique depends upon the clinical problems associated, so either imaging of hard tissue (OSSEOUS) or soft tissue is desired.
Certain protocols are to be taken care before the imaging procedure:
the amount of diagnostic information available from particular imaging modality.
The cost of examination
The radiation dose
Muscles of mastication are the group of muscles that help in movement of the mandible as during chewing and speech. We need to study these muscles as they control the opening & closing the mouth & their role in the equilibrium created within the mouth. They also play a role in the configuration of face.
This document provides an overview of halitosis (bad breath), including its classification, etiology, diagnosis, and management. It discusses the role of volatile sulfur compounds and certain bacteria in causing halitosis. Diagnostic tools include organoleptic measurement, gas chromatography, and volatile sulfide monitoring to detect these compounds. Treatment involves identifying and addressing the underlying causes, such as periodontal disease, dry mouth, dental caries, or systemic conditions. Preventive measures focus on proper oral hygiene and avoiding foods that can cause temporary halitosis.
Cementum is the mineralized tissue covering the roots of teeth that provides attachment for collagen fibers linking the tooth to surrounding bone. It begins at the cementoenamel junction and continues along the root to the apex. Cementum is avascular and less hard than dentin. It contains both inorganic minerals and organic materials including collagen. Cementoblast cells synthesize cementum by laying down an organic matrix that subsequently mineralizes. Cementum thickness varies along the root and increases with age. It provides for functional adaptation and resistance to resorption during orthodontic tooth movement.
This document discusses oroantral communications and fistulas. It defines them as abnormal connections between the oral and maxillary sinus cavities. Causes include tooth extraction, tumors, cysts, and trauma. Signs and symptoms may include unpleasant taste/odor, fluid/food reflux into the nose, and air leakage. Examination involves inspection, suctioning the socket, and radiographs. Management includes immediate closure attempts and antibiotics to prevent sinusitis. Surgical techniques like buccal and palatal flaps are used for larger defects or fistulas based on factors like location, size, and presence of infection.
The maxillary nerve is the second division of the trigeminal nerve. It originates in the lateral wall of the cavernous sinus and passes through the foramen rotundum into the pterygopalatine fossa. It gives off several branches in the pterygopalatine fossa and infratemporal fossa, including the posterior superior alveolar nerve, zygomatic nerve, and anterior and middle superior alveolar nerves. The maxillary nerve terminates by innervating structures in the face, providing sensation to the maxillary region. A maxillary nerve block can provide anesthesia for dental procedures involving the maxillary area, such as when a large canine space abscess is
Odontogenic keratocyst (OKC) is the cyst arising from the cell rests of dental lamina. It can occur anywhere in the jaw, but commonly seen in the posterior part of the mandible. Radiographically, most OKCs are unilocular when presented at the periapex and can be mistaken for radicular or lateral periodontal cyst.
This document provides an overview of the muscles of mastication. It begins by defining muscle and mastication. It then discusses the development, classification, properties and functions of the primary muscles of mastication - the masseter, temporalis, lateral pterygoid, and medial pterygoid muscles. The document also covers the accessory muscles of mastication and their functions. Finally, it discusses some clinical considerations regarding these muscles, including bruxism, tetanus, and myofascial pain disorders.
Vestibuloplasty is a surgical procedure to deepen the oral vestibule by changing the attachments of the soft tissue. There are several types of vestibuloplasty procedures, including mucosal advancement, secondary epithelization, and grafting. Mucosal advancement involves undermining and advancing the oral mucosa, while secondary epithelization uses the oral mucosa to line one side and allows the other side to heal through epithelization. Grafting can use skin, mucosa, or dermis grafts to line the extended vestibule. The document discusses techniques for each type of vestibuloplasty procedure.
This document discusses Ardita Vata (facial palsy) according to Ayurveda and modern medicine. It defines Ardita Vata as a deviation leading to deformity of one side of the face or half of the body. The causes, symptoms and types are described according to Ayurvedic texts like Charaka Samhita. Bell's palsy, the most common type of facial palsy, is also discussed in detail including causes like infection, trauma and tumors. The document compares unilateral lower motor neuron lesions in Bell's palsy versus bilateral upper motor neuron lesions. Various treatment approaches are summarized including nerve grafts, slings and weights to correct facial paralysis.
Bell's palsy is a form of temporary facial paralysis resulting from damage or trauma to the facial nerves. The facial nerve-also called the 7th cranial nerve-travels through a narrow, bony canal (called the Fallopian canal) in the skull, beneath the ear, to the muscles on each side of the face. For most of its journey, the nerve is encased in this bony shell.
Each facial nerve directs the muscles on one side of the face, including those that control eye blinking and closing, and facial expressions such as smiling and frowning. Additionally, the facial nerves carries nerve impulses to the lacrimal or tear glands, the saliva glands, and the muscles of a small bone in the middle of the ear called the stapes. The facial nerve also transmits taste sensations from the tongue.
When Bell's palsy occurs, the function of the facial nerve is disrupted; causing an interruption in the messages the brain sends to the facial muscles. This interruption results in facial weakness or paralysis.
Bell's palsy is named for Sir Charles Bell, a 19th century Scottish surgeon who was the first to describe the condition. The disorder, which is not related to stroke, is the most common cause of facial paralysis. Generally, Bell's palsy affects only one of the paired facial nerves and one side of the face, however, in rare cases, it can affect both sides.
This document summarizes a study that evaluated the effectiveness of prednisolone and acyclovir in treating Bell's palsy. The study was a double-blind, randomized controlled trial conducted in Scotland from 2004-2007. Over 500 patients with Bell's palsy were randomly assigned to receive prednisolone, acyclovir, both, or placebo. The primary outcome was complete facial recovery at 3 and 9 months, assessed using a standardized grading scale. Results showed patients receiving prednisolone had significantly higher recovery rates compared to those without prednisolone at both timepoints. There was no significant difference in recovery rates between those receiving acyclovir and those who did not. Secondary outcomes like
Bell's palsy is a temporary paralysis of the facial nerve causing an inability to control facial muscles on one side of the face. It is the most common cause of acute facial nerve paralysis and may develop over several days and last several months, with most cases recovering spontaneously. Treatment includes corticosteroids and protecting the affected eye. The condition results from swelling of the facial nerve as it travels through the skull, inhibiting nerve function.
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Bell's palsy is a facial paralysis caused by inflammation of the facial nerve as it passes through the stylomastoid foramen. It affects the muscles of facial expression on one side of the face. Causes include trauma, infection, tumors, and idiopathic. Symptoms include pain behind the ear, inability to close the eye, and drooping of the mouth corner on the affected side. Treatment involves corticosteroids to reduce swelling, vitamin B, physical therapy like massage and electrical stimulation to prevent muscle atrophy, and heat/cold for increased blood flow and muscle contraction.
The facial nerve emerges from the brainstem and controls facial muscle expression. It has motor, sensory, and parasympathetic components. The nerve passes through the internal auditory canal into the middle ear. It can be injured through temporal bone fractures, surgery, Bell's palsy, or trauma. Facial nerve injuries are classified using the Sunderland or House-Brackmann system to describe the severity and prognosis. Physical exam involves testing facial muscle function to localize the site of injury.
1. Recent advances in grading facial nerve function have led to the development of several new grading systems to improve on existing scales like the House-Brackmann Grade Scale (HBGS).
2. The Movement, Rest, Secondary defects, and Subjective scoring (MoReSS) system aims to improve reproducibility over HBGS by separately assessing dynamic and static components as well as secondary defects.
3. The Facial Nerve Grading System 2.0 (FNGS 2.0) incorporates regional scoring of facial movement to provide additional information while maintaining agreement with the original HBGS. It also addresses ambiguities in use.
4. The Gordon Facial Muscle Weakness Assessment
The facial nerve is the 7th cranial nerve that has both motor and sensory functions. It has a complex anatomical course through the skull and face. Facial paralysis can result from lesions anywhere along this course. Bell's palsy is the most common cause of acute facial paralysis, believed to be due to a viral infection causing inflammation where the nerve exits the skull. Other potential causes include trauma, tumors, infections, and systemic diseases. Treatment depends on the underlying cause but often includes corticosteroids for Bell's palsy and surgery for decompression or repair of severed nerve segments.
The document discusses facial nerve anatomy and Bell's palsy. It notes that the facial nerve controls blinking, eye closing, smiling, frowning, and eyebrow raising. Bell's palsy is a type of facial paralysis caused by dysfunction of the 7th cranial nerve, resulting in an inability to control facial muscles on one side of the face. While the cause is often unknown, it may be due to viral infections like herpes or Lyme disease. Common treatments include corticosteroids to reduce inflammation and antiviral drugs to limit viral spread. Physical therapy can help prevent paralyzed muscles from shrinking.
The document discusses Bell's palsy, which causes sudden weakness or paralysis of the muscles on one side of the face. It is caused by inflammation and swelling of the 7th cranial nerve, which controls facial muscle movement. Symptoms include an inability to smile or close one eye fully. Treatment involves corticosteroids to reduce swelling along with antiviral medications, as herpes simplex virus is a common cause. Most people fully recover facial function within a few months, though symptoms may persist in rare cases.
This document provides information on facial paralysis (palsy) including its causes, types, treatments, and more. It begins with an introduction to facial function and paralysis. It then covers nerve anatomy and classifications of nerve injuries. Specific topics include facial nerve anatomy, types of facial paralysis (central vs peripheral), common causes like Bell's palsy, and surgical treatment options depending on when paralysis occurred (acute, intermediate, or chronic stages). Evaluation methods and the House-Brackmann grading scale for facial function are also summarized.
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BELL'S PALSY IS AN IDIOPATHIC LMN TYPE FACIAL PALSY..THE SEMINAR TELLS YOU OF COURSE OF NERVE..FACIAL MUSCLES THEIR ACTION..HOW TO EXAMINE..THE SEQUELAE OF FACIAL PALSY...LOOK AT IT..
The document summarizes the anatomy and clinical applications of the facial nerve. It begins with the nuclear origin and functional components of the facial nerve. It then describes the intra cranial and extra cranial course of the nerve, its branches including the greater petrosal, chorda tympani, and terminal branches. Applications including facial nerve palsy, Bell's palsy, and preventing injury during dental procedures are discussed. Clinical testing and special tests of facial nerve function are also outlined.
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This document discusses techniques for reanimating facial paralysis. It begins by outlining general principles, including reinnervating muscles early, separately reanimating the upper and lower face, and tailoring the procedure to the patient's needs and assessment. Surgical techniques are then described, including neural methods like nerve grafting and transfers, musculofacial transpositions, and static procedures. The timing of different techniques depends on whether the paralysis is acute (<3 weeks), intermediate (3 weeks to 2 years), or chronic (>2 years). Assessment involves evaluating the cause and extent of paralysis along with patient factors. The goal is to restore facial symmetry, competence, protection, and dynamic smile.
The document provides information on the facial nerve (CN VII) including its anatomy, branches, segments, development and disorders. Some key points:
- The facial nerve has motor and sensory components and contains fibers for facial expression, taste, and lacrimal/salivary glands.
- It exits the skull via the stylomastoid foramen and has segments within the skull, internal auditory canal, middle ear and mastoid.
- Injuries are classified using Seddon or Sunderland systems based on the level of nerve disruption.
- Evaluation includes tests of lacrimal, stapedius, taste and salivary functions to localize the lesion. Electrodiagnostic tests assess prognosis
The facial nerve can be divided into six segments as it travels from the brainstem to the face. The intrameatal segment, as it travels through the internal auditory canal, is the narrowest point where the nerve is most susceptible to entrapment from inflammation. Bell's palsy, the most common form of facial paralysis, presents with retroauricular pain followed by unilateral peripheral facial paralysis affecting the frontal branch. It is diagnosed based on symptoms in the absence of an identifiable cause. Treatment involves corticosteroids while protecting the cornea from issues like lagophthalmos.
Bell's palsy is a condition that causes temporary weakness or paralysis of the muscles on one side of the face. It is caused by inflammation and swelling of the facial nerve as it passes through the facial canal. The symptoms include an inability to smile or close one eye, weakness of the mouth muscles on the affected side, and a loss of taste on the tongue. Most people recover fully within a few weeks or months, though some may experience long-term issues like facial spasms. Treatment focuses on the use of corticosteroids and antiviral drugs to reduce inflammation and speed recovery.
The document summarizes the anatomy and pathophysiology of Bell's palsy, which is a sudden, unilateral facial paralysis of unknown cause. It describes the facial nerve's course from the brainstem nuclei through the facial canal. Bell's palsy is most common in ages 15-45 and of slightly higher incidence in Japanese individuals. The main cause is believed to be reactivation of latent herpes viruses in the facial nerve ganglia. Patients experience rapid onset of maximal facial weakness within two days and associated symptoms of tearing and taste abnormalities. Physical exam reveals impairment of facial muscle movement on one side. Management focuses on eye care to protect the cornea from drying due to impaired lid closure and tearing.
This document discusses facial nerve paralysis, including:
- The anatomy of the facial nerve and branches that innervate facial muscles.
- Common causes of facial nerve paralysis like Bell's palsy.
- Evaluating facial nerve paralysis through examining facial muscles, taste sensation, lacrimation, and nerve conduction velocity.
- Treating facial nerve paralysis with physical therapy including heat, electrotherapy, exercises and occasionally splinting.
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The facial nerve controls facial muscles and allows for facial expressions. Injury can cause devastating physical and psychological effects. Bell's palsy is the most common cause of facial paralysis, resulting from swelling of the facial nerve from a viral infection. It usually resolves spontaneously within 6 weeks. Treatment involves steroids and antivirals to reduce inflammation and swelling of the nerve. For severe long-term paralysis, surgical procedures like nerve grafts or muscle transfers can help restore function. Prognosis depends on the underlying cause but most cases of Bell's palsy fully recover with early diagnosis and treatment.
Bell's palsy is the most common cause of acute facial paralysis. It results in weakness or paralysis of muscles on one side of the face. While the exact cause is unclear, it is thought to be due to viral infection of the facial nerve. Diagnosis is clinical and treatment involves corticosteroids to reduce inflammation. Most patients recover fully within 6 months. Surgical options are considered for incomplete or delayed recovery and include nerve grafts and transfers to restore muscle function. Static procedures correct asymmetry at rest while dynamic reanimation aims to restore symmetry during smiling.
Prognostic test in facial nerve palsy in( ENT )haneen ayad
The document summarizes key information about the facial nerve (cranial nerve VII), including its anatomy, branches, paralysis, causes, signs, and prognostic tests. It notes that the facial nerve has motor, sensory and parasympathetic fibers. It can be divided into intracranial and extracranial parts. Causes of facial nerve paralysis include Bell's palsy, infection, trauma, tumors and stroke. Signs include facial asymmetry and inability to close the eye. Prognostic tests discussed include Schirmer test, stapedial reflex testing, electrogustometry, and electromyography.
This document discusses the facial nerve, which originates in the brainstem and supplies motor and sensory functions to parts of the face and ear. It describes structures innervated by the facial nerve such as the lacrimal gland and muscles of facial expression. Causes of facial nerve palsy include Bell's palsy, strokes, trauma, herpes zoster infection, and tumors. Bell's palsy specifically is thought to be caused by inflammation of the facial nerve as it travels through skull bones. Treatment depends on the underlying cause but may include corticosteroids, antiviral drugs, eye protection, and facial exercises.
This document provides information on the surgical anatomy of the facial nerve. It begins with an introduction to the facial nerve and its functional components and nuclei. It then describes the different parts of the facial nerve from its intracranial portion to its extra-temporal portion in the neck. Several clinical considerations are discussed, including Bell's palsy, Ramsay Hunt syndrome, and Guillain-Barre syndrome. Surgical techniques for facial nerve repair are outlined, including nerve grafting and substitution techniques like hypoglossal-facial nerve crossover. In summary, this document details the anatomy and clinical implications of the facial nerve as well as surgical strategies for repairing injuries to this nerve.
This document provides information on the facial nerve (cranial nerve VII) including its embryology, anatomy, functions, and various disorders. It discusses the facial nerve's motor and sensory roles. Disorders covered include Bell's palsy, Ramsay Hunt syndrome, Moebius syndrome, and Guillain-Barré syndrome. Classification systems for facial nerve paralysis and nerve injuries are also summarized. The document provides detailed information on evaluating facial nerve disorders.
Facial nerve (VII):
Involved in facial expressions, taste sensation, and control of the lacrimal and salivary glands. The facial nerve emerges from the pons.
It has two roots
Medial Motor root
Sensory (Nervous intermedius) root
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The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and
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This document discusses dental casting investment materials. It describes the three main types of investments - gypsum bonded, phosphate bonded, and ethyl silicate bonded investments. For gypsum bonded investments specifically, it details their classification, composition including the roles of gypsum, silica, and modifiers, setting time, normal and hygroscopic setting expansion, and thermal expansion. It provides information on how the properties of gypsum bonded investments are affected by their composition. The document serves as a comprehensive overview of dental casting investment materials.
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it describes the bony anatomy including the femoral head , acetabulum, labrum . also discusses the capsule , ligaments . muscle that act on the hip joint and the range of motion are outlined. factors affecting hip joint stability and weight transmission through the joint are summarized.
This presentation includes basic of PCOS their pathology and treatment and also Ayurveda correlation of PCOS and Ayurvedic line of treatment mentioned in classics.
A review of the growth of the Israel Genealogy Research Association Database Collection for the last 12 months. Our collection is now passed the 3 million mark and still growing. See which archives have contributed the most. See the different types of records we have, and which years have had records added. You can also see what we have for the future.
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This slide is special for master students (MIBS & MIFB) in UUM. Also useful for readers who are interested in the topic of contemporary Islamic banking.
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Iván Bornacelly, Policy Analyst at the OECD Centre for Skills, OECD, presents at the webinar 'Tackling job market gaps with a skills-first approach' on 12 June 2024
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Tags: Information Security, ISO/IEC 27001, ISO/IEC 42001, Artificial Intelligence, GDPR
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A workshop hosted by the South African Journal of Science aimed at postgraduate students and early career researchers with little or no experience in writing and publishing journal articles.
5. Nuclei of facial nerve
Main motor
Chief sensory
(tractus solitarius)
Lacrimatory
Superior salivatory
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6. Branchial motor (special visceral efferent)
Supplies muscles of facial expression, posterior belly ofSupplies muscles of facial expression, posterior belly of
digastric, stylohyoid and stapediusdigastric, stylohyoid and stapedius..
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7. Visceral motor (general visceral efferent)
Parasympathetic of the lacrimal, submandibular, and sublingualParasympathetic of the lacrimal, submandibular, and sublingual
glands, mucous membranes of nasopharynx, hard and soft palate.glands, mucous membranes of nasopharynx, hard and soft palate.
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8. Taste from the anterior 2/3 of tongueTaste from the anterior 2/3 of tongue
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9. General sensory (general somatic afferent)
General sensation from skin of concha of auricle andGeneral sensation from skin of concha of auricle and
small area behind earsmall area behind ear..
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10. Main motor nucleus
Part of nucleus that supplies
upper part of face receives
corticonuclear fibres from
both cerebral hemispheres.
Part of nucleus that supplies
lower part receives fibres from
opposite cerebral hemisphere
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12. Upper motor nerve lesion
Results from the damage to the neuronal cell bodies in the
cortex or their axons that project via the corticobulbar tract
Voluntary control of only the lower muscles of facial
expression on the side contralateral to the lesion will be lost
Voluntary control of muscles of the forehead will be spared
due to the bilateral innervation of the portion of the motor
nucleus of the cranial nerve VII that innervates the upper
muscles of facial expression
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13. Characteristic of UMN lesion of the facial nerve
Facial asymmetry
Atrophy of muscles of lower portion of the face on
affected side
No eyebrow droop
Smoothing of nasolabial folds on affected side
Lips cannot be held tightly together or pursed
Difficulty keeping food in mouth while chewing on
affected side
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15. Lower motor neuron lesion
Results from damage to the motor nucleus of
CN VII or its axons
Results in paralysis of all muscles of facial
expression (including those of the forehead)
ipsilateral to the lesion
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16. Clinical correlation
A LMN lesion of CN VII
which occurs at or
beyond the
stylomastoid foramen is
commonly referred to
as a bell’s palsy
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17. Bell’s palsy
Defined as idiopathic paresis or paralysis of
the facial nerve of sudden onset.
Unilateral lower motor neuron paralysis of
sudden onset, not related to any other
disease elsewhere in the body
Name ascribed to sir Charles bell, who in
1821, demonstrated the separation of the
motor & sensory innervations
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18. Incidence : 15 -40 cases per 1,00,000
Women affected more
3.3 times more common in pregnant woman
Prevalent in 3rd
trimester or 1st
week of post
partum
More in diabetics
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19. Can occur at any age, more common in
middle age
Both sides equally affected
1% bilateral
8% cases positive family history
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20. Etiology
Rheumatic hypothesis: Berard in 1936.
rheumatic swelling may press the nerve
against facial canal
Cold hypothesis: Bell. Exposure to extreme
cold or cold draught
Ischaemic hypothesis: ischemia from
disturbed circulation
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22. Immunological hypoyhesis: in vivo
sensitizatiion of lymphocytes to peripheral
nerve myelin
Viral hypothesis: HSV. Elevated viral antibody
titre & interferons
Facial palsy + herpes →→ Ramsay Hunt
syndrome
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23. Histology
Thickened, edematous perineurium &
associated blood vessels
Diffuse infiltrate of inflammatory cells
between nerve bundles
Degeneration of myelin sheath
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24. Clinical features
Sudden onset, usually in morning after
awakening
Unilateral involvement of entire face
Corner of mouth droops down, causing
drooling of saliva
Whistling is impossible
Excessive tearing
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25. Inability to close the affected eye or wink or
raise the eye brow
Bell’s sign: In an attempt to close the eye lid,
the eye balls rolls upward, so that pupil is
covered and white sclera is seen
Inability to wrinkle the forehead
Inability to elevate upper or lower lip
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26. Widening of palpebral fissure
Loss of blink reflex
Obliteration of nasolabial fold
Mask like face
Slurred speech
Loss/ alteration of taste occasionally
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27. Prognosis
Initial weakness of facial musculature →
worsens over 2 to 3 days → maximum by 2
weeks → remission → Spontaneous
recovery
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29. House-Brackman
classification of facial palsy
Grade I – Normal function without weakness
Grade II- Slight facial asymmetry with minor synkinesis
Grade III-Obvious asymmetry with residual forehead movement
Grade IV-Obvious disfiguring asymmetry with lack of forehead motion and
incomplete eyeclosure
Grade V- Asymmetry at rest and only slight facial movement
Grade VI – Complete absence of tone or motion
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30. TREATMENT
MEDICAL
Steroids
Physiotherapy
Eye care
Reassurance
SURGICAL
Facial Nerve Decompression
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31. Nerve decompression-
Can be carried out internally or externally
Internal decompression-the nerve is exposed in the
fallopian canal and pressure in the canal is relieved
by exposing the nerve and the epineural sheath is
opened to visualise the nerve fibres and release
adhesions or reestablish continuity
External decompression is done by releasing the
epineural sheath from surrounding scar tissue ,bone
or foreign body
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32. Paralyzed eye lid
Non surgical correction
Surgical correction
Gold weight implants
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33. Nerve anastomosis-reanimation-
anastomosis of the
central end of the
hypoglossal or
spinal accessory
nerve with the distal
end of the facial
nerve is done
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34. Nerve grafting-
whenever there is
evidence of neuroma or
loss of portion of the
nerve, nerve grafting
can be considered
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