This document provides information about assessing inherited color vision defects in clinical practice. It discusses the purpose of the document, introduces color vision deficiency (CVD), describes different types of CVD including their mechanisms and characteristics. Diagnostic tests for CVD are explained, including the Ishihara test, Farnsworth Panel D-15, and Richmond HRR test. Interpretation of results and care/use of the tests are covered. Occupations that may be affected by CVD and management of patients with CVD are also mentioned.
This document provides information about colour vision examination and testing. It discusses:
- The three types of cone cells in the eye responsible for colour vision and their peak wavelength sensitivities.
- Common types of colour vision deficiencies including dichromacy (missing one cone type) and anomalous trichromacy (shifted sensitivity of one cone type).
- Tests used to screen for and diagnose colour vision deficiencies, including pseudoisochromatic plates like Ishihara plates, hue arrangement tests like the Farnsworth D-15, and colour matching tests using an anomaloscope.
- Guidelines for administering common colour vision tests and interpreting their results to identify type and severity of any deficiency.
Color vision : Physiology ,Defects, Detection, Diagnosis & ManagementAayush Chandan
This document discusses clinical examination of color vision including a presentation outline covering introduction to color vision, physiology of color vision, theories of color vision, after images, color vision defects, inheritance of color vision, and color vision tests. It provides details on the trichromatic theory, opponent color theory, physiology of the eye and brain in perceiving color, types of color vision defects, and color vision tests. The document seeks to provide an overview of color vision for clinical examination, diagnosis, and management of color vision defects.
This document discusses exodeviations, or divergent strabismus, which is when the visual axis is deviated laterally and the fovea is rotated nasally. It describes the different types of exodeviations including comitant deviations like infantile exotropia and incomitant deviations caused by paralysis or muscle restrictions. Symptoms, causes, and treatments for various exodeviations are outlined, with non-surgical treatments including prism therapy and orthoptic exercises, and surgical treatments involving lateral rectus recession and medial rectus resection.
Contact lenses require proper cleaning, disinfecting, and storage to maintain eye health. There are various types of contact lens solutions designed for different purposes, such as multipurpose solutions for cleaning, rinsing, disinfecting, and storing lenses, and hydrogen peroxide-based solutions for cleaning and disinfecting. Follow-up with an eye doctor and proper hygiene are important for safe contact lens wear.
Here are 3 options for very difficult post-graft cases when standard lenses are not working:
1. Try a scleral lens with a large optic zone to vault over the irregularity. This can improve comfort and vision.
2. Consider corneal collagen cross-linking (CXL) to strengthen the cornea followed by intracorneal ring segments (Intacs) to help regularize the shape.
3. As a last resort, a second corneal transplant may be needed to obtain an adequate surface for contact lens wear or improve vision. However, the success rate decreases with subsequent grafts.
The key is finding the best option to improve vision and comfort while protecting the ocular surface long-term. A
Amblyopia is a condition of reduced vision in one or both eyes that is not caused by structural eye problems. It occurs during early childhood development when there is inadequate visual stimulation to one or both eyes. Common causes include strabismus, significant refractive error differences between the eyes, form deprivation, and abnormal binocular interaction. Treatment involves correcting any refractive errors and using occlusion therapy or drugs to blur vision in the non-amblyopic eye, forcing use of the amblyopic eye. Occlusion therapy is the most common treatment but requires compliance to achieve results. Other options include penalization, visual stimulation, and drugs, but occlusion remains the standard first approach. Success depends on early diagnosis and treatment before age 7.
To know Humphrey visual field analyser
To know about various types of perimetry
To identify field defect
To recognize that field defect is due to glaucoma or neurological lesion
To know that field defect is progressive or not
Interpretation of HVFA
This document discusses the evaluation of ptosis, or drooping of the eyelids. It begins by defining ptosis and distinguishing between true and pseudo ptosis. True ptosis is classified as acquired or congenital, with acquired further divided into neurogenic, myogenic, aponeurotic, and mechanical types. The evaluation of ptosis involves a thorough history, measurement of margin-reflex distance, palpebral fissure height, levator function, and upper lid crease. Additional tests include assessing for fatigability, Cogan's twitch sign, and jaw winking phenomenon. Confirmatory tests include the ice test and edrophonium (Tensilon) test. Treatment options mentioned include eyelid
This document provides information about colour vision examination and testing. It discusses:
- The three types of cone cells in the eye responsible for colour vision and their peak wavelength sensitivities.
- Common types of colour vision deficiencies including dichromacy (missing one cone type) and anomalous trichromacy (shifted sensitivity of one cone type).
- Tests used to screen for and diagnose colour vision deficiencies, including pseudoisochromatic plates like Ishihara plates, hue arrangement tests like the Farnsworth D-15, and colour matching tests using an anomaloscope.
- Guidelines for administering common colour vision tests and interpreting their results to identify type and severity of any deficiency.
Color vision : Physiology ,Defects, Detection, Diagnosis & ManagementAayush Chandan
This document discusses clinical examination of color vision including a presentation outline covering introduction to color vision, physiology of color vision, theories of color vision, after images, color vision defects, inheritance of color vision, and color vision tests. It provides details on the trichromatic theory, opponent color theory, physiology of the eye and brain in perceiving color, types of color vision defects, and color vision tests. The document seeks to provide an overview of color vision for clinical examination, diagnosis, and management of color vision defects.
This document discusses exodeviations, or divergent strabismus, which is when the visual axis is deviated laterally and the fovea is rotated nasally. It describes the different types of exodeviations including comitant deviations like infantile exotropia and incomitant deviations caused by paralysis or muscle restrictions. Symptoms, causes, and treatments for various exodeviations are outlined, with non-surgical treatments including prism therapy and orthoptic exercises, and surgical treatments involving lateral rectus recession and medial rectus resection.
Contact lenses require proper cleaning, disinfecting, and storage to maintain eye health. There are various types of contact lens solutions designed for different purposes, such as multipurpose solutions for cleaning, rinsing, disinfecting, and storing lenses, and hydrogen peroxide-based solutions for cleaning and disinfecting. Follow-up with an eye doctor and proper hygiene are important for safe contact lens wear.
Here are 3 options for very difficult post-graft cases when standard lenses are not working:
1. Try a scleral lens with a large optic zone to vault over the irregularity. This can improve comfort and vision.
2. Consider corneal collagen cross-linking (CXL) to strengthen the cornea followed by intracorneal ring segments (Intacs) to help regularize the shape.
3. As a last resort, a second corneal transplant may be needed to obtain an adequate surface for contact lens wear or improve vision. However, the success rate decreases with subsequent grafts.
The key is finding the best option to improve vision and comfort while protecting the ocular surface long-term. A
Amblyopia is a condition of reduced vision in one or both eyes that is not caused by structural eye problems. It occurs during early childhood development when there is inadequate visual stimulation to one or both eyes. Common causes include strabismus, significant refractive error differences between the eyes, form deprivation, and abnormal binocular interaction. Treatment involves correcting any refractive errors and using occlusion therapy or drugs to blur vision in the non-amblyopic eye, forcing use of the amblyopic eye. Occlusion therapy is the most common treatment but requires compliance to achieve results. Other options include penalization, visual stimulation, and drugs, but occlusion remains the standard first approach. Success depends on early diagnosis and treatment before age 7.
To know Humphrey visual field analyser
To know about various types of perimetry
To identify field defect
To recognize that field defect is due to glaucoma or neurological lesion
To know that field defect is progressive or not
Interpretation of HVFA
This document discusses the evaluation of ptosis, or drooping of the eyelids. It begins by defining ptosis and distinguishing between true and pseudo ptosis. True ptosis is classified as acquired or congenital, with acquired further divided into neurogenic, myogenic, aponeurotic, and mechanical types. The evaluation of ptosis involves a thorough history, measurement of margin-reflex distance, palpebral fissure height, levator function, and upper lid crease. Additional tests include assessing for fatigability, Cogan's twitch sign, and jaw winking phenomenon. Confirmatory tests include the ice test and edrophonium (Tensilon) test. Treatment options mentioned include eyelid
Retinoscopy is the primary objective method for determining a patient's refractive error. It involves using a retinoscope to illuminate the retina and observe the movement of the reflected light. For myopic patients, the light moves in the opposite direction of the retinoscope's movement, while for hyperopic patients it moves in the same direction. The goal is to find the neutralization point where no movement is seen, indicating the proper refractive correction. Factors like the working distance, type of mirror used, and patient's fixation can impact results. Retinoscopy is useful for initial refractive estimates and screening for ocular conditions.
The document discusses color vision testing techniques and color deficiencies. It describes various color vision tests including pseudoisochromatic plates, lantern tests, arrangement tests, and anomaloscopes. It explains what each test screens for and its purpose. It also discusses the different types of color deficiencies including red, green, blue deficiencies and how color may appear to those with deficiencies.
This document discusses colour vision and colour blindness. It begins by explaining how colour vision works through the visual system's cone cells and their sensitivity to different wavelengths of light. It then describes the two main theories of colour vision: Young-Helmholtz trichromatic theory involving red, green and blue cones, and Hering's opponent process theory. The rest of the document details different types of colour blindness, methods for testing colour vision, and explains that while colour blindness cannot be cured, special lenses can help some colour blind individuals distinguish some colours.
The document discusses color vision and color blindness. It begins by describing the structure of the retina and how rods and cones detect light and enable vision. It then discusses theories of color vision from Aristotle to modern trichromatic and opponent-process theories. The document also covers tests for color vision deficiency and different types of color blindness caused by genetic mutations. It notes some challenges faced by those with color vision deficiencies and potential implications in medicine. It concludes by outlining an experimental device called the eyeborg that aims to help treat color blindness by converting color wavelengths into sounds.
B-scan ultrasonography provides two-dimensional images of the eye that can reveal information about the shape, location, extension, mobility, and thickness of tissues. It uses high frequency sound waves reflected off structures in the eye. The transducer sends pulses and receives echoes to build an image. B-scan is useful when the ocular media is opaque and for evaluating conditions like tumors, detachments, inflammation and measuring the eye's dimensions. Pathological features seen on B-scan include vitreous hemorrhage, asteroid hyalosis, retinoschisis, choroidal detachment, retinal detachment in various configurations, cysticercosis, choroidal melanoma and more.
Binocular single vision refers to simultaneous vision with two eyes that occurs when an individual fixates on an object. There are three grades of binocular vision: simultaneous perception, fusion, and stereopsis. Fusion is the ability to see a composite picture from two similar images, while stereopsis provides the impression of depth by superimposing images from slightly different angles. Tests for binocular vision include those for simultaneous perception, fusion, and stereopsis using instruments like the synaptophore. Binocular vision develops through infancy and childhood as the visual axes become coordinated to direct each fovea at the object of regard.
Corneal physiology in relation to contact lens wearHira Dahal
This document discusses corneal physiology in relation to contact lens wear. It describes the layers of the cornea and its blood, nerve and oxygen supply. Maintaining corneal transparency requires adequate oxygen and metabolism. Contact lenses reduce oxygen levels, which can cause swelling, hypoesthesia, and structural changes if levels fall below what the cornea requires. The minimum oxygen needed varies from 5-17.9% depending on the activity. Soft lenses induce more swelling than RGP lenses. Hypoxia affects epithelial healing, sensitivity and metabolism.
Contrast sensitivity is a measure of the ability to detect slight differences in luminance or color. It is tested using sine wave or square wave gratings that vary in spatial frequency and contrast level. Contrast sensitivity is a better predictor of visual function than visual acuity alone, as it can detect losses from conditions like cataracts, glaucoma, and AMD even before acuity is affected. Contrast sensitivity is measured using charts like Pelli-Robson, FACT, and Arden plates that test sensitivity across spatial frequencies. Many ocular and systemic factors can influence contrast sensitivity, including refractive error, age, cataracts, diabetes, glaucoma, and macular diseases. Contrast sensitivity testing provides additional information about visual
The document discusses paralytic strabismus, including:
1) Hering's law of equal innervation and Sherington's law of reciprocal innervation which are important in diagnosing paralytic strabismus.
2) The sequelae of ocular muscle palsy including overactions and underactions of muscles.
3) Methods for investigating incomitant strabismus including cover tests, motility examination, and Hess screen plots to identify the affected muscle.
The Maddox rod test is used to detect heterophoria or heterotropia. It consists of a series of cylindrical lenses mounted in a trial frame that produces an elongated streak of light. When viewed through the Maddox rod, a spot of light appears as a streak. The orientation of the streak indicates whether the eye is deviated vertically or horizontally. The test is easy for patients to understand and perform, and useful for detecting vertical deviations.
Real subjective refraction in astigmatismBipin Koirala
1) The document discusses subjective refraction techniques for astigmatism, including determining the spherical and cylindrical corrections.
2) Key steps include controlling accommodation, finding the monocular best sphere using VA or bichrome tests, and determining the cylindrical component using fogging with targets like clock dials or Jackson cross cylinders.
3) The axis of the cylindrical correction must match the axis of the patient's astigmatism to fully correct their refractive error.
The document summarizes a case study of a 20-year-old male patient with left eye vision loss since childhood due to corneal scarring who was fitted for a prosthetic soft contact lens. Details are provided on the patient's history and examination, differential diagnosis, types and fitting criteria of prosthetic contact lenses, fitting of a medium brown type D prosthetic lens, and fitting assessment showing good coverage, centration, and movement. The plan is for the patient to be fitted with a single purecon prosthetic soft contact lens.
The Worth Four Dot test is used to assess binocular vision. It presents four lights - red, green, green, white - through red-green glasses. The test checks for suppression or diplopia by asking the patient to report the number, color, and position of lights seen. Abnormal responses can indicate conditions like strabismus, suppression in one eye, or vertical/horizontal diplopia from deviations. The test is inexpensive and easy to perform but relies on subjective patient responses.
Fitting soft contact lenses requires considering many patient-specific factors to achieve excellent vision and ocular health. A proper fit involves selecting the correct total diameter, base curve, thickness, and material based on the patient's prescription, corneal shape, lifestyle, and health. Trial lenses are used to evaluate fit parameters like coverage, centration, movement, comfort, and vision to optimize on-eye performance while avoiding issues like tightness or looseness that could impact ocular health or vision. The goal is to find a lens that provides optimum vision and good comfort without causing any ocular insult.
This document discusses the AC/A ratio, which is the ratio of accommodative convergence to accommodation. It defines the AC/A ratio and notes the normal range is 3-5 prism diopters per diopter of accommodation. Abnormal AC/A ratios can cause strabismus. The document outlines methods to measure the AC/A ratio clinically and discusses its uses in diagnosing different types of strabismus and their management approaches.
- Squint, or strabismus, is a misalignment of the visual axes that leads to loss of binocular single vision. It can be caused by issues in the orbit, eye muscles, motor nerves, or brainstem.
- Strabismus is classified as apparent, latent, or manifest. Manifest strabismus is further divided into concomitant, where the deviation is the same in all gazes, and incomitant, where the deviation varies with gaze.
- Evaluation of strabismus involves assessing history, visual acuity, refractive error, eye alignment tests, and binocular vision. Accurately measuring any refractive errors and prescribing corrections as needed is important for diagnosis and treatment of
This document discusses progressive addition lenses (PALs) and how to properly fit them. PALs provide uninterrupted vision at all distances without visible lines but require careful fitting. The fitting process involves identifying patient needs, selecting an appropriate frame, adjusting the frame measurements, determining pupil distance, marking the lens insert position, checking the fit, and delivering instructions. Common fitting issues include incorrect pupil distance, lens heights, or prescriptions. Proper troubleshooting requires identifying potential causes and remedies through refitting adjustments or remaking lenses. Attention to detail in fitting and addressing patient complaints is important for successful PAL adaptation.
This document discusses methods for assessing visual acuity in pediatric patients. It begins by defining visual acuity and describing its normal development from birth through age 6. It then outlines different techniques for measuring various types of visual acuity, including detection, resolution, and recognition acuity. These techniques include methods that elicit voluntary responses like candy beads, as well as involuntary responses like optokinetic nystagmus drums and visual evoked potentials. Preferential looking tests using cards with different grating frequencies are described as a way to measure resolution acuity in nonverbal children.
250+ High Frequency MCQs in Optometry and OphthalmologyRabindraAdhikary
This document provides a collection of 250+ multiple choice questions (MCQs) in optometry and ophthalmology. It aims to help students study for entrance, licensing, and job exams in these fields. The questions cover a range of difficulty levels and come from past exams. Authentic answers are provided from experienced professionals. More similar questions and answers are available at the provided link. The questions are regularly updated to help eye health professionals prepare for various exams.
Dvorine Color Test offered by Operture.comTim Burns
Dvorine pseudo-isochromatic plates is an approved test for color deficiency. Only available at http://www.operture.com/shopping/product/2253-dvorine-pseudoisochromatic-plates-color-vision-test?search=dvorine
This document summarizes a presentation on color vision and its clinical aspects. It begins with an introduction to color vision and discusses the trichromatic and opponent process theories of color vision. It then covers the neurophysiology of color vision including processing in the retina, lateral geniculate body, and visual cortex. It discusses normal color attributes and various types of color blindness. Finally, it reviews common color vision tests including Ishihara plates and the American Optic Hardy-Rand-Ritter plates.
Retinoscopy is the primary objective method for determining a patient's refractive error. It involves using a retinoscope to illuminate the retina and observe the movement of the reflected light. For myopic patients, the light moves in the opposite direction of the retinoscope's movement, while for hyperopic patients it moves in the same direction. The goal is to find the neutralization point where no movement is seen, indicating the proper refractive correction. Factors like the working distance, type of mirror used, and patient's fixation can impact results. Retinoscopy is useful for initial refractive estimates and screening for ocular conditions.
The document discusses color vision testing techniques and color deficiencies. It describes various color vision tests including pseudoisochromatic plates, lantern tests, arrangement tests, and anomaloscopes. It explains what each test screens for and its purpose. It also discusses the different types of color deficiencies including red, green, blue deficiencies and how color may appear to those with deficiencies.
This document discusses colour vision and colour blindness. It begins by explaining how colour vision works through the visual system's cone cells and their sensitivity to different wavelengths of light. It then describes the two main theories of colour vision: Young-Helmholtz trichromatic theory involving red, green and blue cones, and Hering's opponent process theory. The rest of the document details different types of colour blindness, methods for testing colour vision, and explains that while colour blindness cannot be cured, special lenses can help some colour blind individuals distinguish some colours.
The document discusses color vision and color blindness. It begins by describing the structure of the retina and how rods and cones detect light and enable vision. It then discusses theories of color vision from Aristotle to modern trichromatic and opponent-process theories. The document also covers tests for color vision deficiency and different types of color blindness caused by genetic mutations. It notes some challenges faced by those with color vision deficiencies and potential implications in medicine. It concludes by outlining an experimental device called the eyeborg that aims to help treat color blindness by converting color wavelengths into sounds.
B-scan ultrasonography provides two-dimensional images of the eye that can reveal information about the shape, location, extension, mobility, and thickness of tissues. It uses high frequency sound waves reflected off structures in the eye. The transducer sends pulses and receives echoes to build an image. B-scan is useful when the ocular media is opaque and for evaluating conditions like tumors, detachments, inflammation and measuring the eye's dimensions. Pathological features seen on B-scan include vitreous hemorrhage, asteroid hyalosis, retinoschisis, choroidal detachment, retinal detachment in various configurations, cysticercosis, choroidal melanoma and more.
Binocular single vision refers to simultaneous vision with two eyes that occurs when an individual fixates on an object. There are three grades of binocular vision: simultaneous perception, fusion, and stereopsis. Fusion is the ability to see a composite picture from two similar images, while stereopsis provides the impression of depth by superimposing images from slightly different angles. Tests for binocular vision include those for simultaneous perception, fusion, and stereopsis using instruments like the synaptophore. Binocular vision develops through infancy and childhood as the visual axes become coordinated to direct each fovea at the object of regard.
Corneal physiology in relation to contact lens wearHira Dahal
This document discusses corneal physiology in relation to contact lens wear. It describes the layers of the cornea and its blood, nerve and oxygen supply. Maintaining corneal transparency requires adequate oxygen and metabolism. Contact lenses reduce oxygen levels, which can cause swelling, hypoesthesia, and structural changes if levels fall below what the cornea requires. The minimum oxygen needed varies from 5-17.9% depending on the activity. Soft lenses induce more swelling than RGP lenses. Hypoxia affects epithelial healing, sensitivity and metabolism.
Contrast sensitivity is a measure of the ability to detect slight differences in luminance or color. It is tested using sine wave or square wave gratings that vary in spatial frequency and contrast level. Contrast sensitivity is a better predictor of visual function than visual acuity alone, as it can detect losses from conditions like cataracts, glaucoma, and AMD even before acuity is affected. Contrast sensitivity is measured using charts like Pelli-Robson, FACT, and Arden plates that test sensitivity across spatial frequencies. Many ocular and systemic factors can influence contrast sensitivity, including refractive error, age, cataracts, diabetes, glaucoma, and macular diseases. Contrast sensitivity testing provides additional information about visual
The document discusses paralytic strabismus, including:
1) Hering's law of equal innervation and Sherington's law of reciprocal innervation which are important in diagnosing paralytic strabismus.
2) The sequelae of ocular muscle palsy including overactions and underactions of muscles.
3) Methods for investigating incomitant strabismus including cover tests, motility examination, and Hess screen plots to identify the affected muscle.
The Maddox rod test is used to detect heterophoria or heterotropia. It consists of a series of cylindrical lenses mounted in a trial frame that produces an elongated streak of light. When viewed through the Maddox rod, a spot of light appears as a streak. The orientation of the streak indicates whether the eye is deviated vertically or horizontally. The test is easy for patients to understand and perform, and useful for detecting vertical deviations.
Real subjective refraction in astigmatismBipin Koirala
1) The document discusses subjective refraction techniques for astigmatism, including determining the spherical and cylindrical corrections.
2) Key steps include controlling accommodation, finding the monocular best sphere using VA or bichrome tests, and determining the cylindrical component using fogging with targets like clock dials or Jackson cross cylinders.
3) The axis of the cylindrical correction must match the axis of the patient's astigmatism to fully correct their refractive error.
The document summarizes a case study of a 20-year-old male patient with left eye vision loss since childhood due to corneal scarring who was fitted for a prosthetic soft contact lens. Details are provided on the patient's history and examination, differential diagnosis, types and fitting criteria of prosthetic contact lenses, fitting of a medium brown type D prosthetic lens, and fitting assessment showing good coverage, centration, and movement. The plan is for the patient to be fitted with a single purecon prosthetic soft contact lens.
The Worth Four Dot test is used to assess binocular vision. It presents four lights - red, green, green, white - through red-green glasses. The test checks for suppression or diplopia by asking the patient to report the number, color, and position of lights seen. Abnormal responses can indicate conditions like strabismus, suppression in one eye, or vertical/horizontal diplopia from deviations. The test is inexpensive and easy to perform but relies on subjective patient responses.
Fitting soft contact lenses requires considering many patient-specific factors to achieve excellent vision and ocular health. A proper fit involves selecting the correct total diameter, base curve, thickness, and material based on the patient's prescription, corneal shape, lifestyle, and health. Trial lenses are used to evaluate fit parameters like coverage, centration, movement, comfort, and vision to optimize on-eye performance while avoiding issues like tightness or looseness that could impact ocular health or vision. The goal is to find a lens that provides optimum vision and good comfort without causing any ocular insult.
This document discusses the AC/A ratio, which is the ratio of accommodative convergence to accommodation. It defines the AC/A ratio and notes the normal range is 3-5 prism diopters per diopter of accommodation. Abnormal AC/A ratios can cause strabismus. The document outlines methods to measure the AC/A ratio clinically and discusses its uses in diagnosing different types of strabismus and their management approaches.
- Squint, or strabismus, is a misalignment of the visual axes that leads to loss of binocular single vision. It can be caused by issues in the orbit, eye muscles, motor nerves, or brainstem.
- Strabismus is classified as apparent, latent, or manifest. Manifest strabismus is further divided into concomitant, where the deviation is the same in all gazes, and incomitant, where the deviation varies with gaze.
- Evaluation of strabismus involves assessing history, visual acuity, refractive error, eye alignment tests, and binocular vision. Accurately measuring any refractive errors and prescribing corrections as needed is important for diagnosis and treatment of
This document discusses progressive addition lenses (PALs) and how to properly fit them. PALs provide uninterrupted vision at all distances without visible lines but require careful fitting. The fitting process involves identifying patient needs, selecting an appropriate frame, adjusting the frame measurements, determining pupil distance, marking the lens insert position, checking the fit, and delivering instructions. Common fitting issues include incorrect pupil distance, lens heights, or prescriptions. Proper troubleshooting requires identifying potential causes and remedies through refitting adjustments or remaking lenses. Attention to detail in fitting and addressing patient complaints is important for successful PAL adaptation.
This document discusses methods for assessing visual acuity in pediatric patients. It begins by defining visual acuity and describing its normal development from birth through age 6. It then outlines different techniques for measuring various types of visual acuity, including detection, resolution, and recognition acuity. These techniques include methods that elicit voluntary responses like candy beads, as well as involuntary responses like optokinetic nystagmus drums and visual evoked potentials. Preferential looking tests using cards with different grating frequencies are described as a way to measure resolution acuity in nonverbal children.
250+ High Frequency MCQs in Optometry and OphthalmologyRabindraAdhikary
This document provides a collection of 250+ multiple choice questions (MCQs) in optometry and ophthalmology. It aims to help students study for entrance, licensing, and job exams in these fields. The questions cover a range of difficulty levels and come from past exams. Authentic answers are provided from experienced professionals. More similar questions and answers are available at the provided link. The questions are regularly updated to help eye health professionals prepare for various exams.
Dvorine Color Test offered by Operture.comTim Burns
Dvorine pseudo-isochromatic plates is an approved test for color deficiency. Only available at http://www.operture.com/shopping/product/2253-dvorine-pseudoisochromatic-plates-color-vision-test?search=dvorine
This document summarizes a presentation on color vision and its clinical aspects. It begins with an introduction to color vision and discusses the trichromatic and opponent process theories of color vision. It then covers the neurophysiology of color vision including processing in the retina, lateral geniculate body, and visual cortex. It discusses normal color attributes and various types of color blindness. Finally, it reviews common color vision tests including Ishihara plates and the American Optic Hardy-Rand-Ritter plates.
The document provides context about the English Restoration period and details about the poet Andrew Marvell and his famous poem "To His Coy Mistress." It summarizes the poem, describing how the speaker initially promises to wait forever for his mistress but then shifts to urging her not to delay as time passes quickly. Key themes are the value of time and seizing opportunities before they pass. Figurative language includes metaphors, hyperbole, and a logical argument structure.
This document outlines the workshop "Science of the Eye Professional Development Workshop" presented by Ishara Mills-Henry, Ph.D. from MIT. The workshop covers color vision and the retina, vision and the brain, and includes activities to teach concepts like phototransduction, color blindness, and optical illusions. Feedback from participants will be gathered at the end to improve future workshops. The goal is to teach vision science concepts in an engaging way and address common misconceptions.
This document provides an overview of color vision and its management. It defines color vision and discusses its history. It explains the mechanisms of color vision including the trichromatic and opponent process theories. It describes the neurophysiology of color vision from the retina to the visual cortex. It discusses different types of color vision deficiencies including inherited and acquired defects. It outlines methods to diagnose color vision including Ishihara plates, AO-HRR, and Nagel's anomaloscope. It provides an overview of managing color vision deficiencies through treatments like red filters and prevention strategies.
This document provides information about achromatopsia, a rare genetic disorder characterized by the inability to perceive color. It discusses the signs and symptoms, which include seeing only in shades of gray. The causes are described as either acquired damage to the thalamus or retina or congenital mutations impacting color vision genes like CNGA3, CNGB3, GNAT2, PDE6C and PDE6H. Diagnosis involves color vision testing, electroretinography and genetic testing. Treatments aim to help patients cope rather than cure the condition. The document also summarizes studies on Pakistani families that identified genetic mutations responsible for their achromatopsia.
The document discusses color vision and color blindness. It describes how the normal human retina contains rod and cone cells responsible for vision in low and normal light as well as color perception. Color blindness results from partial or complete loss of cone cell function. There are normally three types of cone cells sensitive to red, green, and blue wavelengths. Color blindness testing assesses for defects and classifies types, with common tests using pseudoisochromatic plates where those with normal vision see figures others cannot. While there is no cure, some filters may help those with color blindness better distinguish colors.
The document discusses color vision and color vision deficiencies. It notes that normal color vision is mediated by red, green, and blue cones in the retina. The most common types of color vision deficiencies are anomalous trichromacy, where one color is seen weakly, and dichromacy, where one cone type is absent. Common forms of deficiencies are protanomaly, deuteranomaly, and tritanomaly. Several tests are described to diagnose color vision deficiencies, including pseudoisochromatic plates, the Farnsworth-Munsell 100 hue test, and the Nagel anomaloscope. The document provides details on administration and analysis of the Ishihara and other color vision tests.
Color blindness, also known as color vision deficiency, is the inability to perceive differences between some colors that others can distinguish. It is often genetic but can also be caused by eye, nerve, or brain damage or chemical exposure. The most common types are red-green deficiencies but it is also possible to be deficient in blue perception or see only in black and white. Color blindness is diagnosed using tests that show numbers or patterns embedded in color plates that those with normal color vision can see but those who are color deficient may not be able to distinguish.
The document discusses different types of color vision deficiencies including monochromacy where only one pigment is present, dichromacy where two pigments are present, and trichromacy where all three pigments are present. It provides details on the prevalence, characteristics, inheritance patterns, and distinguishing features of various congenital and acquired color vision defects. Kollner's Rule is also summarized as relating blue-yellow defects to outer retinal diseases and media changes while red-green defects are associated with inner retinal and pathway conditions.
This document provides instructions for using various features in Microsoft PowerPoint 2007. It describes the PowerPoint window layout, including the ribbon interface and slides/outline pane. It explains how to add and format slides, including choosing slide layouts, backgrounds, themes, fonts, and inserting clip art or pictures. Tips are provided for different views for editing or presenting slides.
The document discusses various tests used to evaluate color vision and the central visual field, including the Amsler grid and color vision charts. The Amsler grid is used to screen for macular diseases by having the patient view a grid and report any distortions, gaps, or unusual areas. Color vision tests include pseudoisochromatic plates like Ishihara plates to screen for red-green deficiencies, as well as more advanced tests like the Farnsworth-Munsell 100 Hue test to grade the severity of defects. Spectral tests like the Nagel anomaloscope are also described, which use light mixtures to accurately diagnose color vision abnormalities.
This document provides an overview of color vision, including:
- The mechanisms of color vision are based on cones in the retina that detect different wavelengths of light.
- Theories of color vision include Young-Helmholtz trichromatic theory involving red, green and blue cones, and Hering's opponent process theory involving blue-yellow and red-green opponent pairs.
- Color blindness is classified according to which cone is deficient, and can be tested using Ishihara plates or wool tests. Normal color vision is important for occupations like piloting that rely on color coding.
Prevalence of red green color vision defectsAmna Jalil
The document discusses color vision deficiency, also known as color blindness. It begins by describing the anatomy and physiology of normal color vision, which involves three types of cone cells in the retina that are sensitive to different wavelengths of light corresponding to red, green, and blue. Color blindness is usually caused by defects in or absence of one of the cone cell types and results in the inability to distinguish certain colors. The most common forms are red-green color blindness. The document then describes methods used to test for color vision deficiency, including the Ishihara color test, in a study of Muslim and Hindu populations in Manipur, India.
Abstract: Color vision deficiency (CVD) is characterized by the inability or decreased ability in perception of some specific colors. CVD is further divisible into several types, of which red-green color vision defects are the most common whereas blue-yellow color vision defect is rarer. These kinds of CVDs interrupt color perception but do not impinge on the sharpness of vision. CVD may be X-linked recessive or an autosomal dominant or very rarely an autosomal recessive trait associated with X chromosome or chromosome 7 or 2 or 8 or 10. CVD in a subject is detected and its severity is analyzed by means of several tests, viz, Ishihara Pseudochromatic Test, Lantern Test and Anomaloscope test. The prevalence of CVD varies worldwide and some parts of the world show considerable prevalence. It is very important to identify CVD affected individuals, mild or severe, at an early age by proper counseling in order to guide them in selection of the suitable career path. Few countries have initiated awareness and counseling program for CVD affected individuals, however it is imperative for all countries to take necessary steps in this regard.
q Colour Vision Deficiency Presented by : Optometrist (intern) Asma Al-Jroudi Saudi Arabia, Riyadh, King Abdulaziz University Hospital 30 Dec 14
2. • What Is Color Vision Deficiency? • Causes Of Color Vision Deficiency • Types Of Color Vision Deficiency • Tretments Of Color Vision Deficiency • Ishihara’s Test • Conclousion
3. What is Colour Blindness? • Color blindness, or color vision deficiency, is the inability or decreased ability to see color, or perceive color differences, under normal lighting conditions. •This condition results from an absence of color- sensitive pigment in the cone cells of the retina, the nerve layer at the back of the eye.
4. What is Colour Blindness? • Cones are the coulored light receptors in back of the eye: Red light receptors, Blue light receptors and Green light receptors. • Colour blindness occurs when one or more of the cone types are defected.
5. Causes of Color Blindness • Genetic: Many more men are affected than women. • Acquired : Chronic illness, Accidents, Medications and Age.
Color Vision Deficiency and Ishihara's TestAsma Al-Jroudi
1) Color vision deficiency is the reduced ability to see color differences due to an absence of color-sensitive pigments in the eye. It is usually genetic and most common in men.
2) There are two main types - red-green deficiency which makes distinguishing red and green difficult, and blue-yellow deficiency which affects blue and green/yellow.
3) The Ishihara test is commonly used to test for red-green deficiency using plates with colored dots that form numbers visible to those with normal color vision but not for those with deficiencies.
This document provides instructions for using the Ishihara Color Vision Test, which consists of a series of plates designed to test for color deficiencies. It summarizes:
- The test uses plates that people with normal color vision see numbers or patterns in, while those with red-green deficiencies may see something different or nothing at all.
- The results are analyzed to determine if the subject's color vision is normal, deficient, or if they have total color weakness or blindness. Most common are red-green deficiencies which are divided into protan and deutan types.
- Plates 1-11 are used initially to separate those with normal versus deficient color vision. The document also provides details on what different subjects should
Presented by our respected teacher
Mohammad Siddique (Optometrist)
thank u sir
Final Year Student Of Optometry at ISRA School Of Optometry
All Rights Reserved
Colorblindness is a vision defect that affects the ability to distinguish colors, especially reds and greens. It is caused by defects in the retina or optic nerve and results from abnormalities in the cone cells of the eye that detect color. While colorblind individuals see normally in other aspects, they have trouble differentiating certain colors. Current treatments are limited to tinted lenses, and colorblindness cannot be cured - it is a lifelong condition.
This document provides information about colour vision and colour vision tests. It discusses:
- The anatomy of colour vision involving three types of cones in the retina sensitive to different wavelengths.
- Theories of colour vision including trichromacy, opponent process, and stage theories.
- Types of colour vision deficiencies including dichromacy, anomalous trichromacy, and monochromacy.
- Common colour vision tests like Ishihara plates, Hardy-Rand-Rittler plates, Farnsworth lantern, and Nagel anomaloscope and how they work.
Colour vision , How to check color vision, About colour vision, Types of colour vision , Color vision tests , Theories of colour vision , Characteristics of colour vision , Causes of colour vision , Colour blindness , Defects of colour vision , Optometry , Causes of colour vision , All about color vision , Colour disability , monochromatism , Determine colour deficiency patients ,
This document provides an overview of color vision and color vision tests. It discusses the anatomy and physiology of color vision, including the three types of cones in the retina and the theories of color vision. It also describes different types of color vision defects including dichromacy, anomalous trichromacy, and monochromacy. The document outlines several types of color vision tests, including pseudoisochromatic plate tests like Ishihara plates, hue discrimination tests like the Farnsworth Munsell 100 Hue Test, spectral anomaloscopes, and lantern tests. The goals, administration, and interpretation of these clinical color vision tests are summarized.
Color vision : introduction, classification, causesAnanta poudel
This document discusses color vision and color blindness. It begins with an introduction to color vision, noting that it is mediated by cone cells in the retina and allows humans to perceive differences in light wavelengths. It then discusses types of color blindness such as red-green and blue-yellow deficiencies. The types of color blindness are classified and the prevalence and inheritance patterns are described. Causes of acquired color vision defects from ocular diseases and medications are also summarized.
- Colour vision allows the eye to discriminate between colours excited by light of different wavelengths. It involves three types of cones in the eye sensitive to short, medium, and long wavelengths of light.
- Colour vision deficiency is the inability to distinguish certain colours and can be congenital or acquired. Congenital types are constant and include red-green deficiencies, while acquired types can progress or regress and often involve blue-yellow deficiencies.
- Colour vision is tested using pseudoisochromatic plates, hue arrangement tests, and other methods to identify, classify, and grade colour vision deficiencies for purposes like screening occupations that require normal colour vision.
Most cases of colour vision deficiency involve a red-green deficiency that can be classified as protan or deutan. Protan deficiencies make red appear grey, while deutan deficiencies make green appear grey. The document examines 24 plates used to test for different types of colour blindness, with instructions on what numbers or lines should be traced by those with normal vision versus those with red-green or total colour deficiencies.
1. The document discusses color vision, including how many colors the human eye can see, the cells responsible for color vision, and the evolution of color vision.
2. It describes the trichromatic and opponent process theories of color vision. The trichromatic theory involves three types of cone cells while the opponent process theory involves color pairs that are processed in the brain.
3. The document discusses defects in color vision including congenital defects like dichromacy and acquired defects, as well as tests used to diagnose color vision deficiencies.
This document discusses color blindness, including what it is, its causes, types, diagnosis, and treatment. Color blindness is the inability to see certain colors or distinguish between colors. It is most often a genetic condition caused by abnormalities in the cones of the eye. There are several types depending on which cones are affected, such as red-green color blindness. It is typically diagnosed using tests like the Ishihara Plate test. While there is no cure, tools like color filters can help some with color vision deficiencies see color more clearly. The document also outlines inheritance patterns and challenges people with color blindness may face.
An evaluation of computer based color vision deficiency test egypt as a stu...Aboul Ella Hassanien
1) The document evaluates a computer-based color vision deficiency test compared to a paper-based Ishihara test.
2) 267 Egyptian university students and staff participated in both tests, with 21 found to have red-green color vision deficiency by both tests.
3) The computer-based test showed 100% sensitivity and 98.78% specificity compared to the paper-based test results.
Color blindness is a condition where one has reduced ability to see colors or differentiate between them. It is often genetic and can make everyday tasks difficult. There are several online tests to check for color blindness, including card tests with colored shapes or objects, color confusion tests with multiple color options, and Ishihara plate tests with numbered plates in different color dots [END SUMMARY]
Color vision is the ability to perceive differences between wavelengths of light using cone cells in the retina that are sensitive to red, green, and blue light. There are two main theories of color vision: Young-Helmholtz theory proposes three types of cone cells each sensitive to a primary color, while Hering's theory proposes that some colors appear mutually exclusive like red-green and yellow-blue. Color blindness is caused by deficiencies in perceiving one or more primary colors and can range from anomalous trichromacy where one color is defective to dichromacy where one color is completely absent to rare monochromacy where only one color is perceived.
Similar to Assessment of Inherited Colour Vision Defects (20)
1. Assessment of Inherited Colour
Vision Defects in Clinical Practice
Yasmine; Maram; Kholoud; Rami30/03/2013
1
2. contents
The purpose of the discussion
Introduction to CVD.
Diagnostic tests for CVD.
Management
Advices to patients
Special Occupations and CVD
Short glossary
30/03/2013
2
Maram Hajir
3. Purposes
30/03/2013
3
To recommend tests for primary
care assessment of colour vision
To Learn you about the methods
and analysis for tests
To give you an info. About
Management for CVD
To provide guidance on the advice
that can be given to patients with
CVD
Maram Hajir
4. 30/03/2013
4
The retina of the human eye contains about 7
million cone cells
and more than 100 million rod cells that enable
normal vision.
5. 30/03/2013
5
Color is light, which is carried as specific
wavelengths that the eye absorbs and the brain
converts into messages so that we ‘see colors.
6. Colour Vision Deficiency ( CVD )
Colour vision deficiency (CVD) is the
inability to distinguish certain shades of color or in
more severe cases, see colours at all.
It is a common functional disorder of vision.
Prevalence of CVD among Caucasian population is
reported as 8% on males and 0.4% on females
30/03/2013
6
Maram Hajir
8. Classifications of CVD
-Typical Monochromasy( rare)
-Blue cone Monochromasy ( rare)
-Protanopia (1% of men 0.01% women)
-Deuteranopia (1% of men 0.01% women)
-Tritanopia (1 in 13,000 both men & women equally)
-Protanomaly (1% of men 0.03% women)
-Deuteranomaly (5% of men 0.35% women)
-Tritanomaly (rare)
Monochromasy
Dichromasy
Anomalous
trichromasy
30/03/2013
8
Maram Hajir
9. MonochromasyTypicalMonochromasy
(rare)
mechanism
mutation of genes encoding the cone-specific alpha
and beta sub-units of the cation channel
Characteristics
Colour blind. No perception of colours. Colours
distinguished by brightness differences only. Very
insensitive to red light.
Nystagmus.
Low visual acuity 6/36 to 6/60
Painless photophobia.
30/03/2013
9
Maram Hajir
10. Cont.
Blueconemonochromasy
mechanism
S (blue) cone pigment only.
Characteristics
Colour blind. Colours distinguished by brightness
differences only.
Rudimentary colour vision in mesopic vision from rod
and blue cone activation.
Very insensitive to red light.
Nystagmus.
Low visual acuity 6/12 to 6/24.
Painless photophobia.
30/03/2013
10
Maram Hajir
11. Dichromasy
Protanopia(1%ofmen0.01%women)
mechanism
Absence of L (red) cone pigment.
Characteristics
Very reduced ability to identify colours.
Confuse red, yellow and green, white and green, and
blue and purple.
Reduced sensitivity to red light.
30/03/2013
11
Maram Hajir
14. Anomalous trichromasy
Protanomaly(1%ofmen0.03%women)
mechanism
L (Red) cone pigment absorption spectrum shifted to
shorter wavelengths of light
Characteristics
May confuse white with green and confuse reds,
yellows and greens but loss of colour discrimination
varies greatly between individuals.
Reduced sensitivity to red light.
30/03/2013
14
Maram Hajir
15. Cont.
Deuteranomaly(5%ofmen0.35%
women)
mechanism
M (Green) cone pigment absorption spectrum shifted
to longer wavelengths of light.
Characteristics
May confuse white with green and confuse reds,
yellows and greens but loss of colour discrimination
varies greatly between individuals.
30/03/2013
15
Maram Hajir
16. Cont.
Tritanomaly(rare)
mechanism
Partial loss of S cone pigment.
Characteristics
Loss of colour discrimination for blues, blue-greens,
and greens.
30/03/2013
16
Maram Hajir
17. Tests that used in CVD detection
CVD
Jasmine R. AbdulRahman
30/03/2013
17
18. Types of tests
Ishihara test
Farnsworth Panel D‐15
Farnsworth‐ Munsell 100‐hue
Medmont C100 test
Richmond HRR test (2002)
30/03/2013
18
Jasmine R. AbdulRahman
19. Isochromatic Vs Pseudoisochromatic!
30/03/2013
19
Pseudoisochromatic plates frequently are used by
eye specialists to get an idea of one’s color
efficiency or deficiency. i.e.:
The Ishihara color test
Richmond HRR 2002
SPP2
Dvorine
Jasmine R. AbdulRahman
21. 30/03/2013
21
“isochromatic”: To a color-deficient person,
all the dots in one or more of the plates will appear
similar or the same.
“Pseudoisochromatic” : To a person without
a color deficiency, some of the dots will appear
dissimilar enough from the other dots to form a
distinct figure (number) on each of the plates
Jasmine R. AbdulRahman
22. Ishihara test
It was created by Dr. Shinobu
Ishihara (1879‐1963).
designed to detect congenital
color deficiencies.
Ishihara contains 38 plates.
Pseudoisochromatic plates
History Ishihara plates
http://www.guldenophthalmics.com/ccp7/media/ecom/prodl
g/Ishihara-Color-Test-Book.jpg
30/03/2013
22
Jasmine R. AbdulRahman
23. Capability
30/03/2013
23
Detects protan and deutan CVD with high sensitivity
and specificity.
Jasmine R. AbdulRahman
Screening Congenital Acquired Ability to
classify
No. of plates
Yes Yes No No 38
24. Methods
Dr. Liana, Please chose one paper from this box :D
Please read the name that you select.
Please …. If you don’t mind, the test will be done on
you.
30/03/2013
24
Jasmine R. AbdulRahman
25. Methods
30/03/2013
25
lit adequately by daylight or under electric light
Electric light should be as far as possible to resemble the effect of the natural
daylight
The plates are held 75 cm from the subject and tilted
the plane of the paper is at right angles to the line of vision
each answer should be given without more than three seconds delay.
If the subject is unable to read numerals, plates 26-38 are used and the
winding lines between the two X’s are traced with the brush. Each
tracing should be completed within ten seconds
Jasmine R. AbdulRahman
26. Methods
30/03/2013
26
In a large scale examination the test may be
simplified to an examination of six plates only
1. No 1
2. one of the Numbers 2, 3, 4, 5
3. one of Numbers 6, 7, 8, 9
4. one of Numbers 10,11, 12, 13
5. one of Numbers 14, 15, 16, 17
6. one of Numbers 18,19,20,21.
It may be necessary to vary the order of the plates if it is
suspected that there is a deliberate deception on the part
of the subject.
Jasmine R. AbdulRahman
28. 30/03/2013
28
Plate number How normal read How red-green Deficiencies read How total blind
read
No. 1 12 12 12
No. 2 8 3 X
No. 4 29 70 X
No. 6 5 2 X
No. 7 3 5 X
No. 8 15 17 X
No. 9 74 21 X
No.11 6 X or read incorrectly X
No. 13 45 X or read incorrectly X
No. 14 5 X or read incorrectly X
No. 15 7 X or read incorrectly X
No. 16 16 X or read incorrectly X
No. 17 73 X or read incorrectly X
No. 18 X 5 X
No. 20 X 45 X
Strong
protan
Mild Protan Strong
Deutan
Mild Deutan
No.22 26 6 2 & 6 (6 clearer) 2 2 & 6 (2 clearer)
No. 23 42 2 4 & 2 (2 clearer) 4 4 & 2 (4 clearer)
29. How to interpret results
30/03/2013
29
Scoring occurs on the first 21 plates
17/21 or more is considered normal
13/21 or less is abnormal.
Jasmine R. AbdulRahman
30. Interpretation
Errors on three or more of the numeral plates indicates red-
green CVD with a small chance (2%) of misdiagnosing
normal colour vision.
Five or more errors indicates certain red-green CVD.
Number of errors is not a useful measure of severity.
Subjects making very few errors will probably have a mild defect
but those who make a large number of errors may be mild or severe.
Failure to see the red numeral indicates protan.
failure to see the red-purple numeral indicates deutan.
30/03/2013
30
Jasmine R. AbdulRahman
31. Care of the book
30/03/2013
31
Book of the test plates should be kept closed.
Except during use :P
Exposure to sunlight causes a faiding of the color of
the plates.
Use sterile cotton swap in tracing plates.
Don’t touch the plate.
Jasmine R. AbdulRahman
32. Farnsworth Panel D‐15
designed by Dean
Farnsworth
30/03/2013
32
History Farnsworth Panel D‐15
http://www.e-mfp.org/Assets/Farnsworth_Panel1.jpg
Kholoud Abu Abdoun
33. 30/03/2013
33
most widely used of the colour sorting tests and
must be part of primary care colour vision
assessment.
Kholoud Abo Abdoun
Screening Congenital Acquired Ability to
classify
No. of plates Time to
Administer
Yes Yes Yes Yes 16 chips Slow
34. Purposes
30/03/2013
34
To reveal the color blindness
To differentiate among subjects affected with
Dyschromatopsia ( little affected abnormal
trichromatic) from those who are
Severely affected ( dichromatic)
Kholoud Abo Abdoun
35. 30/03/2013
35
Description of the test
Cautions
It is recommended:
Not to expose the caps to light.
To avoid touching the colors with fingers.
To avoid damage the caps and their colors
Kholoud Abo Abdoun
36. Methods
30/03/2013
36
Either on Daylight or on front of a wide window
and under a clear sky.
The position for examiner and patient is a cross the
table.
The caps numbered from 1-15 are arranged in
random order.
The subject educated to choose the cap after cap
and arrange them on the case based on the color
degree as he/she see.
patient start from the reference cap P.
Kholoud Abo Abdoun
37. 30/03/2013
37
To eliminate the waste time, examiner told the
patient that the test take only 1-2 minutes to finish.
anyway examiner let the patient to finish normally.
Those who finished quickly, ask him to check their
classifications.
The case is closed and turned over; it is now ready
for future testing.
Starting from the reference cap, the points of
diagram are connected according to the order
presented by the subject.
Kholoud Abo Abdoun
42. Interpretation
30/03/2013
42
The results of the test can be either a:
”success” circular diagram.
or “failure” diagram with parallel lines
http://webvision.med.utah.edu/imageswv/KallColor25.jpg
43. The diagrams of subject
30/03/2013
43
Either normal or slightly deficient follow the circle.
For color blind subject the diagrams from parallel
or crisscrossed lines (with at least two parallel lines
crossing the diagram)
Note: test should be repeated in the event of a
doubtful interpretation.
Kholoud Abo Abdoun
44. some cases of success and failure
30/03/2013
44
Example of success: normal and slightly deficient results. The subjects
with normal vision place the caps in a perfect order or in interchanging one
or two caps .
Example of failure: results of color blind subjects.
Fig. 4 shows the diagram of a red blind patient (dichromatich). The lines f
diagram are parallel to the protan axis.
Fig. 6 shows the diagram of a subject blind to blue or purple with lines
parallel to the tritan axis.
There is a very uncommon case of complete color blindness: the patient
will be totally unable to place the caps in a logical order.
Kholoud Abo Abdoun
45. Richmond HRR
The HRR
pseudoisochromatic
test was developed by
Hardy, Rand and
Rittler.and it is
supports very
efficient color
defeciency screening
30/03/2013
45
History
Maram Hajir
46. Capability
30/03/2013
46
1. Detects protan and deutan CVD with a sensitivity
and specificity only slightly less than that of the
Ishihara test.
2. Ideal confirmation test for the Ishihara test.
3. Detects tritan defects
4. May differentiate protan, deutan and tritan
defects.
5. Classifies severity as mild, medium and strong.
48. 30/03/2013
48
It serves as a confirmation of the result of the
Ishihara test and can guard against the possibility
that the patient has learned the correct answers for
the Ishihara. It has a sensitivity and specificity
almost as good as the Ishihara. It can also detect
tritan defects, which the Ishihara does not.
Maram Hajir
49. 30/03/2013
49
This might be thought to be of little importance as
inherited tritan defects are rare, having a prevalence
of only 1 in 13,00021 and blue and yellow are not
as important in colour codes as are red, green and
yellow, however, tritans can encounter occupational
colour problems.
Maram Hajir
51. 30/03/2013
51
Yes , by popular configuration of the HRR , called
the HRR Combo and provides the full 24 plates
laminated with plastic to protect them against the
acid in fingerprints. The Combo also provides a full
set of Amsler Grids. Amsler testing completes the
prescribed regimen for acquired color testing
Maram Hajir
52. 30/03/2013
52
The last advantage of the HRR test is that it can be
used with very young children because it uses
symbols, a circle, a triangle and a cross, which can
often be named or traced by young children before
they can read numbers.
Maram Hajir
54. 30/03/2013
54
Richmond HRR (24 plates )
first 4 plates
to show how
the test work
the fourth
plate has no
figure
4 plates for
R/G
screening
contain 6
symbols
2 plates for
tritan
screening
contain 4
sbmbols
14 plate for
extent
10 for R/G
4 for blue
CVD
Maram Hajir
55. 30/03/2013
55
Two or more errors with the six symbols on the four red-green screening
plates indicates abnormal colour vision but a few patients (4%) with
normal colour vision will make two errors.
Three or more errors indicates certain red-green CVD. The majority (98%)
of those with a red/green defect make three or more errors on the screening
plates.
No data on detection of tritan defects with the Richmond HRR 2002 test
but the screening plates of the original AO HRR test have been shown to
detect tritan defects, but not all.
56. 30/03/2013
56
If no errors are made on the four symbols on the two tritan screening plates ask
if one of each pair of symbols in the tritan screening and classification plates is
much fainter than the other. If the tritan symbols look fainter, a tritan defect is
probable.
Correct classification as protan or deutan on 86% of occasions, 3% wrongly
classified, remainder ambiguous. Tritan defects clearly differentiated if
detected.
Errors in first five classification plates indicates mild CVD (30% CVDs).
Errors on next three classification plates = medium CVD (45% CVDs) Errors
on last two classification plates = strong CVD (25% CVDs). However,
meaning of ‘medium’ and ‘severe’ is uncertain as some mild CVD are
classified ‘medium’ or ‘strong’ and dichromats may be classified as ‘medium’.
58. Medmont C100 test
The Medmont C-100 owes it
origin to Estvez and
colleagues.
who thought of applying the
principle of flicker
photometry to the assessment
of colour vision.
The first commercially
available instrument using
this principle was the
OSCAR, produced by a
Dutch company Medilog
30/03/2013
58
History Medmont C100 test
http://www.medmont.com.au/medi
a/2722/c100_top.jpg
Jasmine R. AbdulRahman
59. Overview
30/03/2013
59
The Medmont C-100 test is not well known but it
must be a part of the basic battery of colour vision
tests.
It has only one function, which is to differentiate
protans and deutans among those who have red-
green abnormal colour vision
Jasmine R. AbdulRahman
60. Overview
30/03/2013
60
objective screening of colour anomalies and
reductions.
It is an inexpensive test and takes only a minute or
two to administer.
Jasmine R. AbdulRahman
61. Features
30/03/2013
61
1. Rapid colour deficiency indication
2. Unique, easy to read scale (graphic colour bar indicator)
3. Portable, with mains operated power pack (no batteries)
4. 3 Years Warranty
5. Comes with protective case, plug-in mains power pack and User
Guide.
6. Dimensions (mm): 70W x 35H x 113L
Jasmine R. AbdulRahman
62. Procedure
1. Normal room light
conditions are suitable but
any fluorescent lights which
exhibit noticeable flicker
should be switched off.
2. The subject was asked to
hold the instrument at a
distance of about 40 cm,
and look at the small
circular flickering disk. http://www.medmont.com.au/p
roducts/c100-colour-vision-
tester.aspx
30/03/2013
62
Steps Medmont C100
Jasmine R. AbdulRahman
63. Procedure
3. The subject was instructed
to adjust the control knob
located on the top of the
Medmont C100 case
4. Then the subject would
adjust the knob slowly to a
point where the light flicker
disappears or is a minimum.
http://www.medmont.com.au/produc
ts/c100-colour-vision-tester.aspx
30/03/2013
63
Steps Medmont C100
Jasmine R. AbdulRahman
65. Recommendations
30/03/2013
65
The experimenter records the readings as shown on
the indicator at the rear of the instrument.
It is recommended that the patient be given two
practice attempts at obtaining a minimum flicker
point, and the measurements should be repeated at
least four times for statistical averaging.
Jasmine R. AbdulRahman
66. Results Analysis
30/03/2013
66
The settings chosen to achieve no or minimum
flicker are read on an arbitrary scale from -5 to
+ 5.
+2.0 to -2.0 is the extreme range of normal
settings
but typically settings are within ± 1.0
The scale is colour-coded red for protan
settings, green for deutan and yellow for
normal
The colour-coded scale lights correspond to
integers (1, 2, 3 ...)
but can be interpolated to 0.5, when two
adjoining lights are illuminated.
Jasmine R. AbdulRahman
67. 30/03/2013
67
If two adjacent LEDs are equally illuminated, the
reading would be halfway between the two LED
values.
if the two yellow LEDs are equally illuminated, the
reading would be zero.
Similarly, if a yellow LED and its adjacent green LED
are equally illuminated, the reading would be 1.5
68. Results
30/03/2013
68
The Medmont C-100 can also diagnose women
who have normal colour vision but are carriers of
the abnormal gene for protanomaly or protanopia.
The Medmont C-100 colour vision test measures
relative spectral sensitivity using flicker photometry
to differentiate protans and deutans. It should be able
to diagnose Schmidt's sign.
Jasmine R. AbdulRahman
70. Fransworth‐ Munsell 100‐hue
designed by Dean
Farnsworth
30/03/2013
70
History
Rami Danaa
http://www.utsl.co.th/upload/product/Munsell%2
0Color%20FM%20100%20Hue%20Test.jpg
77. Interpretation
30/03/2013
77
• About 68% of the population wit normal
CV has average color discrimination
• The pattern will be characterized by TEC
ranges from 20 to 100
Average
discrimination
• About 16% of the population wit normal
CV has superior color discrimination
• The pattern will be characterized by TEC
ranges from zero to 16
superior
discrimination
• About 16% of the population wit normal
CV has low color discrimination
• The pattern will be characterized by TEC
more than 100
Low
discrimination
Rami Danaa
79. Interpretation
30/03/2013
79
• The mid point is located
between 62 and 70Protans
• The mid point is located
between 56 and 61Duetans
• The mid point is located
between 46 to 52Triatns
Rami Danaa
83. Why color vision test is not always done in
routine clinical practice?
30/03/2013
83
1. there is no single test of colour vision that
provides the clinician with all the information
needed to advise patients.
2. proper assessment of abnormal colour vision
needs several tests, which takes time, and the
clinician has to decide which supplementary
colour vision tests should be used.
Rami Danaa
84. 30/03/2013
84
3. there is no treatment for abnormal colour vision so
there may seem little point in diagnosing it.
4. the classification of CVDs is complex and may
not be easily remembered by practitioners who do
not routinely diagnose abnormal colour vision
with an anomaloscope.
Rami Danaa
85. Treatment, „Compensation‟ & Cure
30/03/2013
85
• Some CVD sufferers can be helped by
color filters which act to increase the
contrast and reportedly make it possible to
distinguish colors close to the confusion
lines
• Some people benefit from the use of an X-
Chrom lens which is available as a contact
lens. Again, these filters may serve to
increase color contrast. Further, spectacles
that reduce glare may also help congenital
CVD sufferers.
‘THERAPEUTIC’
METHODS
• In 2009 the Departments of Ophthalmology
at the University of Washington in Seattle,
the University of Florida in Gainsville and
the Medical College of Wisconsin in
Milwaukee published the results of a
research program aimed at correcting the
red-green vision of squirrel monkeys with
congenital (dichromatic) CVD using gene
therapy. It was shown that after applying
gene therapy the monkeys were able to
distinguish between patterns of gray, green
and red dots.
GENE THERAPY
RESEARCH IN
PRIMATES
Rami Danaa
86. 30/03/2013
86
In the absence of the development of a cure for
congenital CVD, safety remains a key issue. Those
with a strong or medium level of congenital CVD
need to avoid activities where color confusion may
jeopardize others.
Rami Danaa
87. Advice to Patients
30/03/2013
87
The difficulties of giving advice
There is always great potential for misunderstanding
and misremembering.
It should not be assumed that the patient who learns of
his abnormal colour vision for the first time will
receive the news with interest or gratitude
Rami Danaa
88. 30/03/2013
88
Steward and Cole report that of the 18 patients in their survey who were
previously unaware of their abnormal colour vision, half expressed
disbelief and denied they had any problem with colour and half were
accepting and acknowledged that on reflection they did have problems
with colour.
Pickford and Cobb found 44 per cent of a sample of 36 subjects diagnosed
to have CVD for the first time exhibited denial, which they define as ‘a
wide range of attitudes from plain disbelief in the tests to an unwillingness
to agree that the defect, if it does exist, would have any influence on their
daily life’. Only 22 per cent of the sample demonstrated a coping attitude,
that is, acceptance of the defect and an effort to adapt. The rest of the
sample exhibited some form of overcompensation about their defect
Rami Danaa
89. Career advice
30/03/2013
89
Some occupations have a statutory colour vision requirement
but these vary between countries and between states within
countries and are often poorly defined and administered.
There are also several occupations, for which there is no
statutory colour vision requirement but for which abnormal
colour vision is a handicap.
it is not possible to provide a comprehensive list and full
details of all occupational colour vision standards
Kholoud Abo Abdoun
90. Occupational colour requirements broad
categories
30/03/2013
90
1. Normal colour vision
2. Defective colour vision that is sufficiently mild
to enable the colour task to be performed
Kholoud Abo Abdoun
91. 30/03/2013
91
Normalcolour
vision Normal colour vision is required for occupations that involve
precise colour matching
Also for occupations for which it is deemed that recognition
of signal lights and other colour codes is absolutely critical
to safety
Examples: deck officers and seamen, train drivers, air traffic
controllers
(in Australia) and some occupations in the defence forces
All patients with CVD, however mild, can be told that it is
very likely that these careers will not be open to them.
Kholoud Abo Abdoun
92. 30/03/2013
92
Defectivecolour
vision that is sufficiently mild to enable the colour
task to be performed
The two most common occupational colour tasks
that give rise to an occupational colour vision
standard are the recognition of signal lights and
surface colour codes
Lantern test for recognition of signal lights
Farnsworth D15 test for surface colour codes
Kholoud Abo Abdoun
93. Lantern
The present instrument was
developed when the College and
the Association of Optometrists
jointly organised a competition
to meet the need for a new
lantern design for certain British
Aviation & Marine colour vision
tests. In 2002 the CAM
prototype lantern designed by
Prof. R. Fletcher won the
competition.
The Fletcher CAM
Lantern
30/03/2013
93
History
Kholoud Abo Abdoun
94. Overview
30/03/2013
94
Lanterns are valuable practical ways of detecting colour vision
defects, having been used in clinical, aviation and marine
assessments for many years.
Lantern tests can instil confidence perhaps lacking with other
conventional tests.
The task of naming small coloured lights resembles practical signals
& transport situations
This is valuable in Optometric and Ophthalmological practice for
inherited and acquired colour vision defects.
Kholoud Abo Abdoun
95. Typical Uses
1. tests for aircraft pilots, mariners, railway drivers
and other occupations.
2. assessment of inherited anomalies of colour
perception (about 5 – 8 % of the male
population).
3. detection of visual changes sometimes caused by
diabetes, cataract, retinal degeneration, etc
30/03/2013
95
Kholoud Abo Abdoun
96. Methods
30/03/2013
96
Naming the colours, at 6 metres reflected in a
standard mirror, is the essential task.
Aviation and Clinical tests These are presented in
a quiet room with illumination between 80 and 200
lux.
Two manual knobs at the back control the
Aperture sequence & the Colour sequence.
A shutter exposes the coloured lights for 2
seconds, & subjects should respond within 5
seconds.
Kholoud Abo Abdoun
99. How Can Color Deficiencies Limit
Humans?
30/03/2013
99
Jasmine R. AbdulRahman
100. 30/03/2013
100
Careers
Bus Driver, Firefighters, Police Officers, Paint Makers,
Doctors, Chemists, Decorators, Computer Programmers
School
Affects Reading and Math Skills
101. Traffic signs
30/03/2013
101
Jasmine R. AbdulRahman
Normal Deuteranopes protanopes
relying on brightness or location, rather than color, to identify objects or situations
can help.
For example, by learning the order of the three colored lights on a traffic signal and
knowing that if the lowermost light is illuminated, it means that the light is green
103. Colour indicates how well meat is
cooked?
30/03/2013
103
lack of perception of red makes it hard for them to identify
the uncooked piece of meat.
Normal Deuteranopes protanopes
Jasmine R. AbdulRahman
104. Colour indicates ripeness of fruit
30/03/2013
104
Nearly 30 per cent of people with abnormal colour vision
report they have trouble judging the ripeness of fruit.
Normal Deuteranopes protanopes
Jasmine R. AbdulRahman
105. diagnosis of illness!!
30/03/2013
105
Medical practitioners and optometrists who have abnormal colour vision
often report that they have trouble seeing redness of inflammation.
18% of those with abnormal colour vision report that they have difficulty
seeing skin rashes, sunburn and blushing.
Normal Deuteranopes protanopes
Jasmine R. AbdulRahman
106. Colour is often used to distinguish an object from
others that are similar
30/03/2013
106
This is especially the case in police work, where colour is often
used to describe suspects, evidence and motor cars.
They will be able to identify the yellow car and the blue, white and
silver cars but not the red and green cars. Note that the
illuminated brake lights in the red car in the second row of parked
cars are not evident in the dichromatic transformations.
Normal Deuteranopes protanopes
Jasmine R. AbdulRahman
107. Denotative use of colour
30/03/2013
107
Colour is often used as an identifier at school.
An instruction to colour a drawing in a certain colour can be
bewildering for the colour vision deficient school child
Parents should write the names of the colours on the pencils.
Normal Deuteranopes protanopes
Jasmine R. AbdulRahman
108. Colour in search
30/03/2013
108
Colour often marks out objects and facilitates
search for them.
Normal Deuteranopes protanopes
Jasmine R. AbdulRahman
109. Colour and search
30/03/2013
109
Colour coding in maps is used to code the class of feature.
to mark out and differentiate for example, blue for district
names and route numbers.
Jasmine R. AbdulRahman
111. 30/03/2013
111
Achromatism/Achromatopsia
Rare inability to distinguish colors. See also Monochromacy.
Cone
Light-sensitive retinal receptor cell that provides sharp visual acuity and
color discrimination. See also Rod.
Deutan
Refers to a person who has deuteranopia, a type of dichromatism in
which red and green are confused. Also deteranomaly, a type of
anomalous trichromatism in which an abnormally high proportion of
the green is needed when mixing red and green to produce yellow.
Dichromatism
Moderately severe color vision defect in which one of the three basic
color mechanisms is absent or not functioning.
Maram Hajir
112. 30/03/2013
112
Protan
Refers to a person who has protanopia, a type of dichromatism in which
only two hues are seen. Also protanomaly, a type of anomalous
trichromatism in which an abnormally high proportion of the red
primary stimulus is needed when mixing red and green to produce
yellow.
Dyschromatopsia
Any type or degree of defective color vision.
HRR
Hardy-Rand-Rittler pseudoisochromatic plate test of colored dots that
appear as recognizable geometric shapes. Used for identifying color
vision deficiencies.
Ishihara
Pseudoisochromatic plate test similar to the HRR test, but with certain
limitations.
Maram Hajir
113. 30/03/2013
113
Monochromacy/ Achromatism/Achromatopsia
Rare inability to distinguish colors
Munsell Scale
Standardized scale of colored materials having
variations in hue and saturation.
Tetartan
Refers to a person with tetartanopia or tetartanopsia,
theoretical conditions and terms for a type of blue-
yellow blindness in which there are two neutral points.
Maram Hajir
114. 30/03/2013
114
Trichromatic
Requiring the use of three color mixture primaries to match all
perceived hues. Anomalous trichromatic is a form of defective
color vision in which three primary colors are also required for
color matching, but the proportion of primaries in the
mixturematches are significantly different from those required in
normal trichromatism.
Tritan
Refers to a person having tritanomaly or tritanopia. The former is
a rare type of defective color vision in which an abnormally large
proportion of blue must be mixed with green to match a standard
blue-green stimulus. Tritanopia is a form of dichromatism in
which all colors can be matched by suitable mixtures of only a
red primary and a green or blue primary.
Maram Hajir
115. References
Michael N. Wiggins, MD. How we should really be doing and interpreting the Ishihara. Retrieved by
March 16,2013 from www.jomtonline.com/jomt/articles/volumes/5/2/HowWeIshihara.pdf
Alotaibi Z.A et.al. Assessment of the Medmont C100 test for colour vision screening of male Saudi
Arabians. S Afr Optom 2011 70(1) 14-20
Ross W Harris & Barry L Cole. Diagnosing protan heterozygosity using the Medmont C-100 colour
vision test. Clin Exp Optom 2005; 88: 4: 240–247
Cole B.L, Lian K.L & Lakkis C. The new Richmon HRR Pseudoisochromatic test for color vision is better
that ishihara test. Clin Exp Optom 2006; 89: 2: 73–80
Maciej Laskowski. USING CUSTOMIZED PSEUDOISOCHROMATIC PLATES FOR
DETECTING CHOSEN FORMS OF DICHROMACY. Journal of KONES Powertrain and Transport,
Vol. 19, No. 1 2012
SHINOBU ISHIHARA. Ishihara Instructions. Retrieved by March 16,2013 from
white.stanford.edu/newlm/.../Ishihara.14.Plate.Instructions.pdf
30/03/2013
115