By: Dr. Rekha Marbate
 History [ sign and symptoms]
 Physical Examination
1. General examination
2. Examination [ inspection, palpation, percussion and
auscultation]
 Investigations for confirmatory diagnosis
 Cardinal symptoms
1. Dyspnea or Breathlessness on exertion:
2. Chest pain
3. Cough
4. Expectoration
5. Hemoptysis
6. Palpitation and
7. Syncopal attack
8. Fever with chills
9. Other
 Def:- Dyspnoea is defined as an abnormally uncomfortable
awareness of breathing.
 Causes-
1. Cardiac origin-
a. Left heart failure
b. Congenital heart diseases (Shunts and valvular lesions)
c. Acquired valvular heart diseases
d. Coronary heart disease
e. Hypertensive heart disease
f. Cardiomyopathies.
2. Dyspnea is the major symptom of left heart failure -
Respiratory
A. Bronchial asthma
B. Chronic obstructive lung disease—Chronic
bronchitis, emphysema
C. Chronic restrictive lung disease
1. Parenchymal Pneumoconiosis
2. Interstitial lung disease
3. Extra parenchymal- Myasthenia gravis, Guillain-
Barré syndrome, Ankylosing spondylitis
Kyphoscoliosis, Obesity
D. Pneumonias
E. Pulmonary neoplasm
F. Pulmonary embolism
G. Laryngeal or tracheal obstruction Inhalation of toxic
gases and fumes.
H. Haematological: Severe anaemia.
I. Miscellaneous: Anxiety,
NYHA
grade
Description
Class I No symptoms with ordinary pysical activity.
Class II Symptoms with ordinary activity, slight
limitation of physical activity
Class III Symptoms with less than ordinary activity.
Marked limitation of activity
Class IV Symptoms with any physical activity or even at
rest
Grading for dyspnoea, palpitation, fatigue and angina in
patients with cardiovascular disease.
Class I
 Dyspnoea that develops in recumbent position and is
relieved by sitting up or elevation of the head with
pillows.
 Orthopnoea occurs within 1-2 minutes after assuming
recumbency and recovery on sitting up is also
immediate.
 Causes -
1. Acute left heart failure
2. Extreme degree of CCF.
 Attacks of dyspnoea which occur at night and awaken the
patient from sleep.
 It occurs 2–5 hours after the onset of sleep and takes 10–30
minutes for recovery after assuming the upright posture.
 Causes-
1. Ischaemic heart disease
2. Aortic valve disease
3. Hypertension
4. Cardiomyopathy
5. Atrial fibrillation
6. Rarely in mitral disease or atrial tumours.
7. PND is the earliest symptom of left heart failure.
 Platypnoea is dyspnoea which occurs only in the
upright position.
 Causes-
1. Left atrial thrombus
2. Left atrial tumour
3. Pulmonary arteriovenous fistula.
 A discomfort in the chest and adjacent area due to
myocardial ischaemia.
 It is due to a discrepancy between myocardial oxygen
demand and supply.
Anginal grading Description
Grade I Angina on severe exertion
Grade II Angina on walking uphill or
climbing more than one
flight of ordinary stairs
Grade III Angina on walking on level
ground or climbing one flight
of ordinary stairs
Grade IV Angina at rest
.
Site Substernal
Nature Pressing, squeezing, strangling, constricting, ‘a
band across the chest’, ‘a weight in the centre of
the chest’. The patient cannot pinpoint the site of
pain
Radiation . To both the shoulders, epigastrium, back, neck,
jaw, teeth. Anginal pain can radiate in all
directions, as mentioned above, but more
commonly radiates to the left shoulder and ulnar
aspect of the left arm.
Duration 5 to 15 minutes
Aggravating
Factor
Exertion, emotion, after a heavy meal, or exposure
to cold
Reliving factor Rest, nitrates.
1. Coronary artery disease: Due to narrowing or spasm
of coronary artery.
2. Aortic stenosis: Due to decreased stroke volume,
reducing coronary perfusion and compression of
coronaries by hypertrophied myocardium,
decreasing O2 supply and increased O2 demand by
the hypertrophied myocardium. Decreased capillary
density and co-existent atherosclerosis increase the
ischaemic burden.
3. Aortic regurgitation: Due to decreased coronary
perfusion as a result of run-off of blood back into the
LV and periphery during diastole; in syphilitic AR, in
addition there is coronary ostial stenosis.
4. Systemic hypertension: Due to decreased diastolic
coronary perfusion as a result of left ventricular
hypertrophy.
5. Severe anaemia
6. Connective tissue disorders (due to arteritis)
7. Extreme tachyarrhythmias.
Def:- Palpitation is defined as an unpleasant awareness
of forceful, arrhythmic or rapid beating of the heart.
 Causes-
1. Extrasystoles - Atrial, ventricular
2. Tachyarrhythmias - Atrial, ventricular
3. Endocrine – Thyrotoxicosis,Hypoglycaemia
4. High output states- Anaemia, pyrexia,Aortic
regurgitation, Patent ductus arteriosus.
5. Drugs- Atropine, adrenaline, aminophylline,
thyroxine, coffee, tea, alcohol
 Syncope may be defined as a transient loss of
consciousness due to inadequate cerebral blood flow
secondary to abrupt decrease in cardiac output.
 Depending on the duration of syncope, the symptoms
experienced by the patient may vary.
 If syncope lasts for 5 seconds- patient experiences
dizziness
 10 seconds- patient may become unconscious
 Causes-
1. Cardiac
i. Electrical abnormalities - Extreme bradycardia, Heart
block , tachyarrhythmias.
ii. Mechanical causes - Aortic stenosis, Hypertrophic
obstructive cardiomyopathy , Left atrial tumours and
thrombus , Pulmonary stenosis, Pulmonary
hypertension , Pulmonary embolism , Tetralogy of
Fallot.
2. Drugs
i. a. Antihypertensives
ii. b. Beta blockers
iii. c. Vasodilators—nitrates, ACE inhibitors.
3. Hypovolaemia: - Haemorrhage, fluid loss, diabetic
precoma
 Forceful expelling strategies of human body to remove
foreign particle outside the respiratory tract.
 Severity – On VAS
 Type-Dry/productive
 Frequency: occasional/ continous or
constant/intermittent
 Type: (As per miller’s Classification)mucoid, purulent,
mucopurulent
 Colour- Yellow, white, greenish, brownishng, with
bright red blood
 Consistency: thin/thick/viscous/tenacious /frothy
 Quantity/volume: scanty- few teaspoon,
 moderate- small cup
 copious- a pint
 Odour-
 Hemoptysis, expectoration of sputum containing
blood, varies in severity from slight streaking to frank
bleeding.
 Cyanosis is a bluish discolouration of the skin and
mucous membrane due to an increased quantity of
reduced haemoglobin > 4 g per dl or > 30% of total
Hb, and PaO2 < 85%, or due to the presence of
abnormal haemoglobin pigments in the blood
perfusing these areas.
Types
Central Peripheral
Features Central cyanosis Peripheral
cyanosis
Mechanism Right to left shunts
Peripheral stasis or
lung disorders
Peripheral stasis
Site Whole body Nail bed, nose tip,
earlobe, extremities
Associations Clubbing -
Extremities warm cold
On warming the
extremities
No change Disappears
O2 inhalation Slight improvement No change
Arterial blood Low < 85% Normal 85–100%
gas-PaO2
A. Build and nutrition
B. Nails and conjunctiva for pallor [mucus membrane of
lower eye lid],
icterus, clubbing, cyanosis.
C. Lymphadenopathy and thyroid swelling
D. Edema
E. Skin - for petechial hemorrhages, Osler nodes,
rheumatic nodules
 Rheumatic nodules
F. Skeletal system - Kyphoscoliosis, chest shape
 G. TPR, BP
 T- temperature [36.5-37.5 degree Celsius or 97.7 – 99.5
degree frenite]
 P- pulse
 R- respiratory Rate
 BP- Blood Pressure
 JVP
 Examination of the neck veins has a two purpose.
1. To assess approximately the mean right atrial
pressure.
2. To study the waveforms.
 Jugular Venous Pressure Jugular venous pressure (JVP)
is expressed as the vertical height from the sternal
angle to the zone of transition of distended and
collapsed internal jugular veins.
 When measured with the patient reclining at 45° is
normally about 4-5 cm.
 The right internal jugular vein is selected because it is
larger, straighter and has no valves.
 It is situated between two heads of sternomastoid.
1. Precordium- is the anterior aspect of the chest
 Normally the precordium has a smooth contour,
slightly convex and symmetrical with part of the
chest wall on the right side.
A. Bulging: Precordium may be bulging in:
1. Enlarged heart
2. Pericardial effusion
3. Mediastinal tumor
4. Pleural effusion
5. Scoliosis
 B. Flattened: Precordium may be flattened in the
following conditions:
 1. Fibrosis of lung
 2. Old pleural or pericardial effusions
 3.Congenital deformity
2. Apex Impulse:- is the lowermost and outermost part
of cardiac impulse seen.
 Normally, it is in the fifth left intercostal space just
inside the mid clavicular line.
 The impulse may not be visible if it is lying just
behind a rib.
1. Apex Beat
 Apex beat is the lower most and outer most point
where maximum cardiac impulse is felt.
 It gives a gentle thrust to the palpating finger.
 Normally, it is located in the fifth left intercostal
space within the mid-clavicular line.
2. Parasternal heave
 Systolic impulse in the left parasternal region commonly
felt in right ventricular enlargement is parasternal heave.
3. Thrills
 Thrills are palpable vibrations (like the purring of a cat that
is felt by the hand) associated with heart murmurs.
 felt with the palm of the hand.
 It is intensified if the chest wall is thin, site of production
is near the surface of the chest wall and the blood flow is
rapid.
 Presence of a thrill is a definite evidence of the presence of
an organic disease of the heart. They may be systolic (AS,
PS, MR, TR, ASD, VSD, PDA)
 Percussion is mainly done to determine the
boundaries of the heart.
 I. Left Border: The patient must be percussed in the
fourth and fifth space in the mid-axillary region and
then medially towards the left border of the heart. The
resonant note of the lung becomes dull. Normally the
left border is along the apex beat.
 If it is outside apex beat suggest pericardial effusion.
 11. Upper border: The patient must be percussed in the
second and third left intercostal spaces in the
parasternal line, which is the line between the mid-
clavicular and the lateral sternal line. Normally there is
resonant note in the second space and dull note in the
third space.
 If there is a dull note in the second space it suggests: 1.
Pericardia! effusion 2. Aneurysm of aorta 3. Pulmonary
hypertension 4. Left atrial enlargement 5. Mediastinal
mass rder:
 Right border: patient must be percussed anteriorly in
the mid-clavicular line on the right side until the liver
dullness is percussed.
 Then the percussion is done one space higher from
the mid-clavicular line medially to the sternal border.
Normally the right border of the heart is retrosternal.
 If the dullness is parasternal it suggests: l. Pericardia!
effusion 2. Aneurysm of ascending aorta 3. Right atrial
enlargement 4. Dextrocardia
 Auscultation is done to describe
 I. Heart Sounds
 II. Murmurs
 III. Other sounds
Heart sound Produced by Characterstics
S1 closure of atrioventricular
valves, Mitral (M1) and
Tricuspid (T1).
It is a high frequency sound
heard best with the diaphragm
of the stethoscope.
S2 closure of the aortic (A2) and
pulmonary (P2) valves
it is a high frequency sound,
heard better in the aortic and
pulmonary area
S3 S3 is produced by initial passive
filling of the ventricles
Low frequency sound,
normally inaudible
S4 S4 is produced by a rapid
emptying of the atrium into
non-compliant ventricle.
Low frequency sound,
normally inaudible
Murmur Murmurs are abnormal heart
sounds caused by vibration of
the valves or the wall of the
heart or great vessels.
Murmurs may be systolic (ifit is
between first and second heart
sound) or diastolic (if between
the second and first heart
·sound) or continuous
 Chest X-Ray
 ECG
 2 D ECHO
 Angiography
 Biomedical markers
 1. PJ Mehta’s Practical Medicine 20th edition
 2. R. Algappan Manual of Practical medicine 4th
edition.
Assessment of cardiovascular system

Assessment of cardiovascular system

  • 1.
  • 2.
     History [sign and symptoms]  Physical Examination 1. General examination 2. Examination [ inspection, palpation, percussion and auscultation]  Investigations for confirmatory diagnosis
  • 3.
     Cardinal symptoms 1.Dyspnea or Breathlessness on exertion: 2. Chest pain 3. Cough 4. Expectoration 5. Hemoptysis 6. Palpitation and 7. Syncopal attack 8. Fever with chills 9. Other
  • 4.
     Def:- Dyspnoeais defined as an abnormally uncomfortable awareness of breathing.  Causes- 1. Cardiac origin- a. Left heart failure b. Congenital heart diseases (Shunts and valvular lesions) c. Acquired valvular heart diseases d. Coronary heart disease e. Hypertensive heart disease f. Cardiomyopathies.
  • 5.
    2. Dyspnea isthe major symptom of left heart failure - Respiratory A. Bronchial asthma B. Chronic obstructive lung disease—Chronic bronchitis, emphysema C. Chronic restrictive lung disease 1. Parenchymal Pneumoconiosis 2. Interstitial lung disease 3. Extra parenchymal- Myasthenia gravis, Guillain- Barré syndrome, Ankylosing spondylitis Kyphoscoliosis, Obesity D. Pneumonias
  • 6.
    E. Pulmonary neoplasm F.Pulmonary embolism G. Laryngeal or tracheal obstruction Inhalation of toxic gases and fumes. H. Haematological: Severe anaemia. I. Miscellaneous: Anxiety,
  • 7.
    NYHA grade Description Class I Nosymptoms with ordinary pysical activity. Class II Symptoms with ordinary activity, slight limitation of physical activity Class III Symptoms with less than ordinary activity. Marked limitation of activity Class IV Symptoms with any physical activity or even at rest Grading for dyspnoea, palpitation, fatigue and angina in patients with cardiovascular disease. Class I
  • 8.
     Dyspnoea thatdevelops in recumbent position and is relieved by sitting up or elevation of the head with pillows.  Orthopnoea occurs within 1-2 minutes after assuming recumbency and recovery on sitting up is also immediate.  Causes - 1. Acute left heart failure 2. Extreme degree of CCF.
  • 9.
     Attacks ofdyspnoea which occur at night and awaken the patient from sleep.  It occurs 2–5 hours after the onset of sleep and takes 10–30 minutes for recovery after assuming the upright posture.  Causes- 1. Ischaemic heart disease 2. Aortic valve disease 3. Hypertension 4. Cardiomyopathy 5. Atrial fibrillation 6. Rarely in mitral disease or atrial tumours. 7. PND is the earliest symptom of left heart failure.
  • 10.
     Platypnoea isdyspnoea which occurs only in the upright position.  Causes- 1. Left atrial thrombus 2. Left atrial tumour 3. Pulmonary arteriovenous fistula.
  • 11.
     A discomfortin the chest and adjacent area due to myocardial ischaemia.  It is due to a discrepancy between myocardial oxygen demand and supply.
  • 12.
    Anginal grading Description GradeI Angina on severe exertion Grade II Angina on walking uphill or climbing more than one flight of ordinary stairs Grade III Angina on walking on level ground or climbing one flight of ordinary stairs Grade IV Angina at rest .
  • 13.
    Site Substernal Nature Pressing,squeezing, strangling, constricting, ‘a band across the chest’, ‘a weight in the centre of the chest’. The patient cannot pinpoint the site of pain Radiation . To both the shoulders, epigastrium, back, neck, jaw, teeth. Anginal pain can radiate in all directions, as mentioned above, but more commonly radiates to the left shoulder and ulnar aspect of the left arm. Duration 5 to 15 minutes Aggravating Factor Exertion, emotion, after a heavy meal, or exposure to cold Reliving factor Rest, nitrates.
  • 14.
    1. Coronary arterydisease: Due to narrowing or spasm of coronary artery. 2. Aortic stenosis: Due to decreased stroke volume, reducing coronary perfusion and compression of coronaries by hypertrophied myocardium, decreasing O2 supply and increased O2 demand by the hypertrophied myocardium. Decreased capillary density and co-existent atherosclerosis increase the ischaemic burden.
  • 15.
    3. Aortic regurgitation:Due to decreased coronary perfusion as a result of run-off of blood back into the LV and periphery during diastole; in syphilitic AR, in addition there is coronary ostial stenosis. 4. Systemic hypertension: Due to decreased diastolic coronary perfusion as a result of left ventricular hypertrophy. 5. Severe anaemia 6. Connective tissue disorders (due to arteritis) 7. Extreme tachyarrhythmias.
  • 16.
    Def:- Palpitation isdefined as an unpleasant awareness of forceful, arrhythmic or rapid beating of the heart.  Causes- 1. Extrasystoles - Atrial, ventricular 2. Tachyarrhythmias - Atrial, ventricular 3. Endocrine – Thyrotoxicosis,Hypoglycaemia 4. High output states- Anaemia, pyrexia,Aortic regurgitation, Patent ductus arteriosus. 5. Drugs- Atropine, adrenaline, aminophylline, thyroxine, coffee, tea, alcohol
  • 17.
     Syncope maybe defined as a transient loss of consciousness due to inadequate cerebral blood flow secondary to abrupt decrease in cardiac output.  Depending on the duration of syncope, the symptoms experienced by the patient may vary.  If syncope lasts for 5 seconds- patient experiences dizziness  10 seconds- patient may become unconscious
  • 18.
     Causes- 1. Cardiac i.Electrical abnormalities - Extreme bradycardia, Heart block , tachyarrhythmias. ii. Mechanical causes - Aortic stenosis, Hypertrophic obstructive cardiomyopathy , Left atrial tumours and thrombus , Pulmonary stenosis, Pulmonary hypertension , Pulmonary embolism , Tetralogy of Fallot. 2. Drugs i. a. Antihypertensives ii. b. Beta blockers iii. c. Vasodilators—nitrates, ACE inhibitors. 3. Hypovolaemia: - Haemorrhage, fluid loss, diabetic precoma
  • 19.
     Forceful expellingstrategies of human body to remove foreign particle outside the respiratory tract.  Severity – On VAS  Type-Dry/productive  Frequency: occasional/ continous or constant/intermittent
  • 21.
     Type: (Asper miller’s Classification)mucoid, purulent, mucopurulent  Colour- Yellow, white, greenish, brownishng, with bright red blood  Consistency: thin/thick/viscous/tenacious /frothy  Quantity/volume: scanty- few teaspoon,  moderate- small cup  copious- a pint  Odour-
  • 23.
     Hemoptysis, expectorationof sputum containing blood, varies in severity from slight streaking to frank bleeding.
  • 24.
     Cyanosis isa bluish discolouration of the skin and mucous membrane due to an increased quantity of reduced haemoglobin > 4 g per dl or > 30% of total Hb, and PaO2 < 85%, or due to the presence of abnormal haemoglobin pigments in the blood perfusing these areas. Types Central Peripheral
  • 25.
    Features Central cyanosisPeripheral cyanosis Mechanism Right to left shunts Peripheral stasis or lung disorders Peripheral stasis Site Whole body Nail bed, nose tip, earlobe, extremities Associations Clubbing - Extremities warm cold On warming the extremities No change Disappears O2 inhalation Slight improvement No change Arterial blood Low < 85% Normal 85–100% gas-PaO2
  • 27.
    A. Build andnutrition
  • 28.
    B. Nails andconjunctiva for pallor [mucus membrane of lower eye lid], icterus, clubbing, cyanosis.
  • 29.
    C. Lymphadenopathy andthyroid swelling
  • 31.
  • 32.
    E. Skin -for petechial hemorrhages, Osler nodes, rheumatic nodules
  • 33.
  • 34.
    F. Skeletal system- Kyphoscoliosis, chest shape
  • 35.
     G. TPR,BP  T- temperature [36.5-37.5 degree Celsius or 97.7 – 99.5 degree frenite]  P- pulse  R- respiratory Rate  BP- Blood Pressure
  • 36.
  • 37.
     Examination ofthe neck veins has a two purpose. 1. To assess approximately the mean right atrial pressure. 2. To study the waveforms.  Jugular Venous Pressure Jugular venous pressure (JVP) is expressed as the vertical height from the sternal angle to the zone of transition of distended and collapsed internal jugular veins.  When measured with the patient reclining at 45° is normally about 4-5 cm.  The right internal jugular vein is selected because it is larger, straighter and has no valves.  It is situated between two heads of sternomastoid.
  • 38.
    1. Precordium- isthe anterior aspect of the chest  Normally the precordium has a smooth contour, slightly convex and symmetrical with part of the chest wall on the right side. A. Bulging: Precordium may be bulging in: 1. Enlarged heart 2. Pericardial effusion 3. Mediastinal tumor 4. Pleural effusion 5. Scoliosis
  • 39.
     B. Flattened:Precordium may be flattened in the following conditions:  1. Fibrosis of lung  2. Old pleural or pericardial effusions  3.Congenital deformity 2. Apex Impulse:- is the lowermost and outermost part of cardiac impulse seen.  Normally, it is in the fifth left intercostal space just inside the mid clavicular line.  The impulse may not be visible if it is lying just behind a rib.
  • 40.
    1. Apex Beat Apex beat is the lower most and outer most point where maximum cardiac impulse is felt.  It gives a gentle thrust to the palpating finger.  Normally, it is located in the fifth left intercostal space within the mid-clavicular line.
  • 41.
    2. Parasternal heave Systolic impulse in the left parasternal region commonly felt in right ventricular enlargement is parasternal heave. 3. Thrills  Thrills are palpable vibrations (like the purring of a cat that is felt by the hand) associated with heart murmurs.  felt with the palm of the hand.  It is intensified if the chest wall is thin, site of production is near the surface of the chest wall and the blood flow is rapid.  Presence of a thrill is a definite evidence of the presence of an organic disease of the heart. They may be systolic (AS, PS, MR, TR, ASD, VSD, PDA)
  • 42.
     Percussion ismainly done to determine the boundaries of the heart.  I. Left Border: The patient must be percussed in the fourth and fifth space in the mid-axillary region and then medially towards the left border of the heart. The resonant note of the lung becomes dull. Normally the left border is along the apex beat.  If it is outside apex beat suggest pericardial effusion.
  • 44.
     11. Upperborder: The patient must be percussed in the second and third left intercostal spaces in the parasternal line, which is the line between the mid- clavicular and the lateral sternal line. Normally there is resonant note in the second space and dull note in the third space.  If there is a dull note in the second space it suggests: 1. Pericardia! effusion 2. Aneurysm of aorta 3. Pulmonary hypertension 4. Left atrial enlargement 5. Mediastinal mass rder:
  • 45.
     Right border:patient must be percussed anteriorly in the mid-clavicular line on the right side until the liver dullness is percussed.  Then the percussion is done one space higher from the mid-clavicular line medially to the sternal border. Normally the right border of the heart is retrosternal.  If the dullness is parasternal it suggests: l. Pericardia! effusion 2. Aneurysm of ascending aorta 3. Right atrial enlargement 4. Dextrocardia
  • 46.
     Auscultation isdone to describe  I. Heart Sounds  II. Murmurs  III. Other sounds
  • 48.
    Heart sound Producedby Characterstics S1 closure of atrioventricular valves, Mitral (M1) and Tricuspid (T1). It is a high frequency sound heard best with the diaphragm of the stethoscope. S2 closure of the aortic (A2) and pulmonary (P2) valves it is a high frequency sound, heard better in the aortic and pulmonary area S3 S3 is produced by initial passive filling of the ventricles Low frequency sound, normally inaudible S4 S4 is produced by a rapid emptying of the atrium into non-compliant ventricle. Low frequency sound, normally inaudible Murmur Murmurs are abnormal heart sounds caused by vibration of the valves or the wall of the heart or great vessels. Murmurs may be systolic (ifit is between first and second heart sound) or diastolic (if between the second and first heart ·sound) or continuous
  • 49.
     Chest X-Ray ECG  2 D ECHO  Angiography  Biomedical markers
  • 50.
     1. PJMehta’s Practical Medicine 20th edition  2. R. Algappan Manual of Practical medicine 4th edition.