This document discusses various age-related changes that can occur in the eye and vision. It begins by defining what is considered "normal" aging vision in those over 65. It then discusses general physiological changes that commonly occur, such as a thinning lens and decreased pupil size. Specific diseases that are more prevalent with age, like cataracts, glaucoma, macular degeneration, and diabetic retinopathy, are also outlined. Treatment options and risk factors for these conditions are provided. Other topics covered include dry eye, low vision aids, and general visual performance changes associated with the aging process.
Presbyopia/ Methods of Presbyopic Addition Determination (healthkura.com)Bikash Sapkota
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Presbyopia and techniques of measurement
A fantastic presentation in the topic "Presbyopia and techniques of measurement"
A detailed information about presbyopia, techniques of presbyopic add determination and different correction methods.
Informative slide presentation on presbyopia for ophthalmology residents, ophthalmologists, optometrists, ophthalmic assistants, ophthalmic technicians, ophthalmic nurses, medical students, medical professors, teaching guides.
Presentation Contents:
--Introduction to presbyopia
-Types of presbyopia
-Risk factors
-Symptoms and signs
-Refractive error and presbyopia
-Methods of determining near add.
-Management of presbyopia
In a nutshell..
- The evaluation and management of presbyopia are important because significant functional deficits can occur when the condition is left untreated
- Undercorrected or uncorrected presbyopia can cause significant visual disability and have a negative impact on the pt.'s quality of life
- Finally, every tentative addition should be adjusted according to the particular needs of the patient
For Further Reading:
-Clinical Procedures in Optometry by J.D. Bartlett, J.B. Eskridge, J.F. Amos
-Primary Care Optometry by Theodere Grosvenor
-Borish’s Clinical Refraction by W.J. Benjamin
-Clinical Procedures for Ocular examination by Carlson et al
-American Academy of Ophthalmology
-Optometric Clinical Practice Guideline by American Optometric Association
-Internet
Follow me to get in touch with optometric and ophthalmic updates.
Management of visual problems of Aging by Ashith Tripathi Ashith Tripathi
This presentation contains headings - Visual performance in the ageing eye
Routine optometric and ocular examination of an older adult:
History
Ocular health examination
Visual acuity measurement
Refraction
Binocular vision
Visual field measurement
Colour vision
Management of vision problems in older adults
Frame requirement
Lens requirements
And special instructions etc.
Presbyopia/ Methods of Presbyopic Addition Determination (healthkura.com)Bikash Sapkota
DIRECT DOWNLOAD LINK ❤❤https://healthkura.com/presbyopia-near-addition/❤❤
Dear viewers Check Out my other piece of works at ❤❤❤ https://healthkura.com ❤❤❤
Presbyopia and techniques of measurement
A fantastic presentation in the topic "Presbyopia and techniques of measurement"
A detailed information about presbyopia, techniques of presbyopic add determination and different correction methods.
Informative slide presentation on presbyopia for ophthalmology residents, ophthalmologists, optometrists, ophthalmic assistants, ophthalmic technicians, ophthalmic nurses, medical students, medical professors, teaching guides.
Presentation Contents:
--Introduction to presbyopia
-Types of presbyopia
-Risk factors
-Symptoms and signs
-Refractive error and presbyopia
-Methods of determining near add.
-Management of presbyopia
In a nutshell..
- The evaluation and management of presbyopia are important because significant functional deficits can occur when the condition is left untreated
- Undercorrected or uncorrected presbyopia can cause significant visual disability and have a negative impact on the pt.'s quality of life
- Finally, every tentative addition should be adjusted according to the particular needs of the patient
For Further Reading:
-Clinical Procedures in Optometry by J.D. Bartlett, J.B. Eskridge, J.F. Amos
-Primary Care Optometry by Theodere Grosvenor
-Borish’s Clinical Refraction by W.J. Benjamin
-Clinical Procedures for Ocular examination by Carlson et al
-American Academy of Ophthalmology
-Optometric Clinical Practice Guideline by American Optometric Association
-Internet
Follow me to get in touch with optometric and ophthalmic updates.
Management of visual problems of Aging by Ashith Tripathi Ashith Tripathi
This presentation contains headings - Visual performance in the ageing eye
Routine optometric and ocular examination of an older adult:
History
Ocular health examination
Visual acuity measurement
Refraction
Binocular vision
Visual field measurement
Colour vision
Management of vision problems in older adults
Frame requirement
Lens requirements
And special instructions etc.
This is a guide for Visual function assessment in low vision. Useful for Optometrists in providing better care to Low vision Patients by assessing the conditions better.
This is a guide for Visual function assessment in low vision. Useful for Optometrists in providing better care to Low vision Patients by assessing the conditions better.
Optometric examination and management of geriatric problems.pptxAnisha Heka
Normal age related changes
Common pathological changes with age
Optometric examination of geriatric population
Complications in examination of older patient
Vision Corrections in older patient
This was a lecture in the course "Significant Medical Conditions in Seniors" presented at Peer Learning in Chapel Hill, NC, USA in 2016 by Michael C. Joseph, MD, MPH.
It extends from the etiology to the management measures. A little effort to make people understand LOW VISION. Vivek Chaudhary, Christian College BSc Optometry, Bangalore. Frm NEPAL, LAHAN
285 milion people around the world are blind or visually impaired.
About 80% of these cases are avoidable.
Poorer population are more affected.
90% of these patients lives in developing countries.
Macular degeneration is an eye disease and is the most common type of macular damage in adults. Because the disease develops as a person ages, it is often known as age-related macular degeneration (AMD).
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
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Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
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Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...
Aging and The Eye
1.
2. AGING AND THE EYE
By:
RABIA AMMER
OPTOMETRIST &ORTHOPTIST
GOLD MEDALIST
3. NORMAL AGING VISION
What exactly is “normal” in people over the
age of 65?
Definition: Having no immediately apparent
structural or pathological defect that could
account for reduced function
Conditions that occur in most older people
(loss of accommodation, miosis) are
considered normal
4. WE KNOW THIS ALREADY
Most older people
have “excellent”
vision
Some eye problems
associated with age
are annoying but
do not cause visual
loss
5. The Challenges of Aging
How older patients are different from all other
patients
As people age, some physiologic changes are
inevitable
Other changes, while not universal, are far
more common than among younger people
Older people also face unique psycho-social
challenges
These changes and challenges can lead to a
variety of geriatric syndromes and issues
These in turn can lead to poor health
outcomes, functional decline, frailty, disability
and dependence
10. More General Aging Changes
Retina- dulls, blood vessel changes
Optic Nerve- boundaries less defined, fewer
capillaries
Macula- little or no foveal reflex, drusen and
lipofuscin deposits, pigmentation
Lids- orbicularis oculi muscle weakens
Lacrimal Glands/Tears- production
Orbit- fat loss, enophthalmos
11. Age Changes in Performance
Refraction- lens and ciliary muscles
Results in Presbyopia
Age 40+
Acuity and Contrast
Decreases after age 50
Due to Brain
Glare
Due to lens and vitreous humor
Dark
Pupil and Lens
12. More Performance Changes
Vitreous Humor
Haziness
Flashing Lights
Moving Spots
Color
Discrimination as cones
Dark
Pupil and Lens
Visual Field
Size 1 to 3 degrees per decade
14. Pupillary Miosis & Light
Diameter of pupil in
the dark minus
diameter of pupil in
bright light
becomes less & less
with age
Consequence:
reduction in retinal
illuminance
15. Visual Difficulties
< 50% of people
under the age of 40
wear refractive
correction
Approximately 90%
of people in their
40s or beyond need
lens correction
16. Visual Function & Age
Up to age 70, loss in static corrected
acuity can be explained by miosis & lens
changes
As age increases, the # of people who
achieve normal corrected acuity
decreases
VERY IMPORTANT: These people show no
signs of ocular disease
17. CORNEA
Corneal sensitivity decreases with age
Touch threshold almost doubles
between the ages of 10 and 80,
especially after 40
Consider entropion & ectropion
Advantages and disadvantages
regarding contact lenses
Corneal curvature changes with age
Astigmatism increases throughout life
18. ANTERIOR CHAMBER
Depth of anterior chamber decreases
At age 15-20, average depth = 3.6 mm
At age 70, average depth = 3.0 mm
This decrease in chamber depth could cause interference
with aqueous flow
20. LENS
Yellow pigment of the lens absorbs
short wavelengths more than long
ones
Older people have decreased
sensitivity in violet/blue end of the
spectrum
White objects may appear yellow &
distinction between blues & greens is
decreased
21. LENS
Older people need significantly more light to
achieve the same level of retinal illuminance
Visual performance of an older individual will be
especially impaired at twilight
Driving should be given much more thought
(testing conditions vs. reality)
22. RETINA & BEYOND
In the absence of pathology, changes in the retina & further
upstream are not directly observable
Inferences must be made on the basis of visual function
There’s the rub! Which function(s)?
23. LIGHT, DARK & GLARE
Older people
Cannot reach the
levels of dark
adaptation that
younger people can
24. LIGHT, DARK & GLARE
Older people - more sensitive to glare & take longer to
recover
25. VISUAL FUNCTION & AGE
Inability to achieve at least 20/25 acuity cannot be explained
for about 10% of people between the ages of 75 & 85
Visual acuity has long been understood (at least by the
rehabilitation community) to be an inadequate measure of
visual function
27. CONTRAST SENSITIVITY
High-frequency cut-off
can be mimicked by
artificial pupils & neutral
density filters
This means that the lens
and pupil changes are
responsible for upper
frequency loss.
28. VERNIER ACUITY
Observer is asked to align objects, not recognize
them (hyperacuity task)
Segments of a line, points of light, etc. are aligned
vertically or horizontally
This ability remains intact throughout life
30. STILES-CRAWFORD EFFECT
Tests the directional sensitivity of the retina
Relies on proper alignment of the retinal
receptors
Light entering different points of the pupil is
differently absorbed by receptors
33. COLOUR PERCEPTION
There has always been a debate
concerning colour vision changes
in older people
Question:
To what degree are changes in colour
perception due to optical media
(especially the lens) & to what extent
are they due to retinal changes?
Are the short-wavelength (blue)
cones playing a big role?
35. SYMMETRY PERCEPTION
Detection of
symmetry is an
important visual
ability
Consider how many
objects in our
environment are
approximately
symmetrical (faces,
butterflies etc)
36. SYMMETRY PERCEPTION
Our data show a
decline in symmetry
perception in
people over 65 …
but it’s an
organized change
50. What is it?
A group of eye diseases that can
damage the optic nerve in the
eye.
Glaucoma can develop in one or
both eyes.
Primary open-angle glaucoma is
the most common form.
Glaucoma
51. Who is at higher risk?
African Americans over age 40.
Everyone over the age of 60, especially Mexican
Americans.
People with a family history of glaucoma.
Glaucoma
52. Same scene as viewed by a
person with glaucoma
Normal vision
Glaucoma
53. Symptoms
No early warning signs or
symptoms
No pain
Loss of side vision
Treatment options
Medications, usually eye drops
Laser or conventional surgery
Glaucoma
54. What can you do?
People at higher risk should get a
comprehensive dilated eye exam
every one to two years or as
instructed by your eye care
professional.
Glaucoma
56. Age-Related Macular
Degeneration (AMD)
What is it?
Common among people aged 60 or older.
Can damage the macula, which is needed for
sharp, detailed central vision.
57. Who is at higher risk?
The greatest risk factor is age.
Other risk factors
Smoking.
Family history.
Obesity.
Race. Caucasians are more likely to lose vision from
AMD.
Age-Related Macular
Degeneration (AMD)
58. Same scene as viewed by a
person with AMD
Normal vision
Age-Related Macular
Degeneration (AMD)
59. Symptoms
No pain.
Blurred vision.
Drusen (can only be seen by an
eye care professional).
Age-Related Macular
Degeneration (AMD)
63. What can you do?
Eat a healthy diet
Don’t smoke, or stop smoking
Maintain normal blood pressure
Maintain a healthy weight
Exercise
Age-Related Macular
Degeneration (AMD)
65. What is it?
A group of eye problems
associated with diabetes.
Diabetic retinopathy is a leading
cause of vision loss and blindness.
Diabetic Retinopthy
66. Who is at higher risk?
People with diabetes.
The longer someone has
diabetes, the more likely it is he
or she will get diabetic
retinopathy.
Diabetic Retinopthy
67. Same scene as viewed by a
person with diabetic retinopathy
Normal vision
Diabetic Retinopthy
68. Diabetic Eye Disease
Symptoms
No early warning signs or symptoms
Early detection and timely
treatment can reduce the risk
of vision loss.
Treatment options
Laser treatment
Surgery
69. Diabetic Eye Disease
What can you do?
Control your ABCs - A1C, blood
pressure, and cholesterol.
Take your medications as directed.
Maintain a healthy weight.
Exercise.
Don’t smoke.
Have a dilated eye exam at least
once a year.
70. Dry Eye
What is it?
The eye does not produce tears
properly.
Tears evaporate too quickly.
Inflammation of the surface of the
eye may occur along with dry
eye.
71. Who is at higher risk?
Women often experience dry eye more than men.
Dry eye can occur at any age.
Older adults frequently experience dryness of the
eyes.
Dry Eye
72. Symptoms
Stinging or burning of the eye.
Feeling as if sand or grit is in the eye.
Episodes of excess tears following dry
eye periods.
A stringy discharge from the eye.
Pain and redness of the eye.
Episodes of blurred vision.
Dry Eye
73. Symptoms
Heavy eyelids.
Decreased tearing or inability to shed tears when
crying.
Uncomfortable contact lenses.
Decreased tolerance to any activity that requires
prolonged visual attention.
Eye fatigue.
Dry Eye
74. Treatment options
Using artificial tears, prescription eye drops, gels, gel
inserts, and ointments.
Wearing glasses or sunglasses.
Getting punctal plugs.
Dry Eye
75. What can you do?
Use an air cleaner to filter dust
Avoid dry conditions
Use lubricating eye drops
Visit an eye care professional
Dry Eye
78. What is it?
A visual impairment that is not
corrected by standard
eyeglasses, contact lenses,
medication, or surgery.
It interferes with the ability to
perform everyday activities.
Low Vision
79. Who is at higher risk?
People with eye disease.
Old aged people.
Some people develop vision loss
after eye injuries or from birth
defects.
Low Vision
80. Treatment options
Vision rehabilitation.
What can you do?
See a specialist in low vision.
Talk to your eye care professional
about vision rehabilitation.
Use low vision devices.
Low Vision