This document discusses the management of behavioral emergencies in psychiatry. It covers creating a safe environment, assessing agitation levels, pharmacological interventions including benzodiazepines and antipsychotics, and physical restraint as a last resort with proper training and personnel. The goal is rapid control of agitation while prioritizing patient and staff safety.
The world’s population is ageing rapidly, and with it is coming to a significant increase in the number of
older people with dementia. This increase presents major challenges for the provision of healthcare
generally and for dementia care in particular, for as more people have dementia, there will be more
people exhibiting behavioural and psychological symptoms of dementia (BPSD).
BPSD exact a high price from both the patient and the caregiver in terms of the distress and disability
they cause if left untreated. BPSD is recognisable, understandable and treatable. The recognition and
appropriate management of BPSD are important factors in improving our care of dementia patients
and their caregivers,
The term “psychosomatic disorder” is mainly used to mean “a physical disease that is thought to be caused, or made worse, by mental factors.” ... For example, chest pain may be caused by stress and no physical disease can be found.
Module: Pharmacotherapy III
Module Coordinator: Dr. Arwa M. Amin Mostafa
Academic Level: Postgraduate, Master of Pharmacy in Clinical Pharmacy
School: Dubai Pharmacy College
Year of first presented in Class: 2018
This presentation is for Educational Purpose. It has no commercial value associated with it.
The world’s population is ageing rapidly, and with it is coming to a significant increase in the number of
older people with dementia. This increase presents major challenges for the provision of healthcare
generally and for dementia care in particular, for as more people have dementia, there will be more
people exhibiting behavioural and psychological symptoms of dementia (BPSD).
BPSD exact a high price from both the patient and the caregiver in terms of the distress and disability
they cause if left untreated. BPSD is recognisable, understandable and treatable. The recognition and
appropriate management of BPSD are important factors in improving our care of dementia patients
and their caregivers,
The term “psychosomatic disorder” is mainly used to mean “a physical disease that is thought to be caused, or made worse, by mental factors.” ... For example, chest pain may be caused by stress and no physical disease can be found.
Module: Pharmacotherapy III
Module Coordinator: Dr. Arwa M. Amin Mostafa
Academic Level: Postgraduate, Master of Pharmacy in Clinical Pharmacy
School: Dubai Pharmacy College
Year of first presented in Class: 2018
This presentation is for Educational Purpose. It has no commercial value associated with it.
Approach oriented presentation for some of the emergencies of clinical gastroenterology, including upper GI bleeding, hepatic encephalopathy, acute severe attack of IBD and acute pancreatitis.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
2. EMERGENCY PSYCHIATRY
THE PHYSICIAN DEALS WITH SITUATIONS FOR WHICH
IMMEDIATE THERAPEUTIC INTERVENTION FREQUENTLY
NECESSARY.
3. PSYCOMOTOR AGITATION
PSYCHOMOTOR AGITATION IS DEFINED AS A STATE OF MARKED
MENTAL EXCITATION ACCOMPANIED BY PURPOSELESS MOTOR
ACTIVITY, WHICH MAY VARY FROM SLIGHT RESTLESSNESS TO
VIGOROUS UNCOORDINATED MOVEMENTS.
6. IMPORTANT EVALUATIONS
LETHALITY
SUICIDAL AND HOMICIDAL IDEATION,INTENT,ATTEMT
LEGAL RIGHT(CONSEQUENCES OF CIVIL COMMITMENTS)
FACTORS TRIGGERING HOSPITALISATION
NEED FOR CHEMICAL/PHYSICAL RESTRAINS
7. DESIRED FORTITUDE FOR
CLINICIANS
INTINCTS FOR DANGER
TOLERANCE
EMPATHY NOT SYMPATHY
SELF ASSERTION
HONESTY
RESOURSEFULLNESS AND NETWORKING
CREATIVITY
ENDURANCE
HUMOR
PRAGMATISM
8. SAFE ENVIRONMENT
SPECIAL INTERVIEW ROOM: TWO EXIT DOORS,NO
WIRES/TUBES,CEILING NOT
REACHABLE,NONREMOVABLE MATERIALS,FIVE
STAFFS,VISUAL MONITORING,BOLTED DOWN
FURNITURE,ISOLATION ROOM,RESTRAINS MATERIALS.
SCRENING WEAPONS
PANIC BUTTONS
CODING SYSTEM
SECURITY STAFFS NEAR ENTRANCE
9. INTERVIEW
ASSESS THE SCENE
BE PREPARE TO SPEND EXTRA TIME
SAFE DISTANCE,SITTING ON 45 DEG ANGLE
CALM ,HONEST
METHODICAL(SHOW INTEREST IN PT’S STORY)
NONJUDGEMENTAL
RAPPORT
10. OPEN ENDED QUESTIONS
AVOIDING DIRECT EYE CONTACT,AVOID FRIGHTENING
EXCLUDE DISRUPTIVE PEOPLE
ENCOURAGE PURPOSEFUL MOVEMENTS
AVOIDING CHALLEGING
DEVELOP A PLAN OF ACTION.
ONCE THE PLAN IS SET, ALLOW THE PATIENT TO EXERCISE
SOME CONTROL
11. INTERVIEW COURSE
GENERAL QUESTIONS
ASSESSMENT OF DELIRIUM
SUICIDAL IDEATIONS
MEDICAL ILLNESS
PRESENT MEDICATIONS
H/O POISONING
PAST/FAMILY HISTORY/SOCIAL SUPPORT/COLLATERAL
INFORMATIONS
MSE
13. MSE
THOUGHT
IS THE PATIENT MAKING
SENSE?
MEMORY
RECENT
REMOTE
IMMEDIATE
AFFECT AND MOOD
DO THE INNER FEELINGS
SEEM APPROPRIATE?
PERCEPTION
“DO YOU HEAR THINGS
OTHERS CAN’T?”
14. SECONDARY ASSESSMENT
In examining the
extremities, check for:
Needle tracks
Tremors
Unilateral weakness or
loss of sensation
OBTAIN VITAL SIGNS.
EXAMINE SKIN
TEMPERATURE AND
MOISTURE.
INSPECT THE HEAD AND
PUPILS.
NOTE UNUSUAL ODORS ON
THE BREATH.
15. ASSESSING AGITATION
SHOULDN’T BE RESTRAINED OR MEDICATED IMMEDIATELY.
DETERMINE THE PT’S “RISK OF ESCALATION.”
FOUR STAGES OF AGITATION
STAGE 1: THE AGITATION IS MODIFIED BY VERBAL CUES, WITHOUT
LIMITS OR BOUNDARIES BEING INVOKED.
STAGE 2: THE AGITATION IS CONTAINED VERBALLY THROUGH LIMIT-
SETTING, BUT IT PERSISTS NONETHELESS.
STAGE 3: THE AGITATION SUBSIDES DURING TRANSIENT PHYSICAL
RESTRAINT.
STAGE 4: THE AGITATION REQUIRES PHARMACOTHERAPY. IT IS
OTHERWISE INTRACTABLE.
OFTEN STAGES 3 AND 4 ARE CONFLATED.
16. ALWAYS HAVE AN EXIT STRATEGY, AND ENSURE THAT
OTHERS CAN QUICKLY COME TO YOUR ASSISTANCE,
IN CASE THAT’S REQUIRED.
NEVER PLAY HERO(INE) AND TAKE THINGS INTO YOUR
OWN HANDS!
17. CRITERIA FOR HOSPITALISATION
DANGER TO SELF/OTHER
POOR SELF CARE/BREAKDOWN OF SUPPORT SYSTEM
EXTREME DISTRESS OR CRISIS
EXACERBATION OF PSYCHIAYTRIC ILLNESS
CLARIFICATION OF DIAGNOSIS
POOR INSIGHT/JUDGEMENT
INTOXICATION
REPEATED TREATMENT FAILURE
FOR ECT
18. IT IS IMPORTANT TO FORMULATE A TENTATIVE
DIAGNOSIS AND DIFFERENTIAL DIAGNOSIS TO GUIDE
TREATMENT.
19. MEDICAL CONDITIONS
ACUTE ONSET
FIRST EPISODE
GERIATRIC AGE
CURRENT MEDICAL ILLNESS OR INJURY
SIGNIFICANT SUBSTANCE ABUSE
NON-AUDITORY DISTURBANCES OF PERCEPTION
NEUROLOGICAL SYMPTOMS
COGNITIVE DYSFUNCTION
CONSTRUCTIONAL APRAXIA
22. IMPORTANT CONSIDERATIONS IN
DIAGNOSIS OF A VIOLENT BEHAVIOR IN
PATIENT
1. PATIENT’S PREMORBID PERSONALITY
2. PAST HISTORY
3. THE UNDERLYING DISORDER
4. THE SOCIAL SETTING
23. PHYSICAL AND PSYCHOLOGICAL CONDITIONS
THAT PRESENT WITH ALTERED MOOD.
PHYSICAL
ALCOHOL INTOXICATION
ANTIHYPERTENSIVE
MEDICATION (E.G.,
METHYLDOPA, PROPRANOLOL,
RESERPINE TOXICITY)
ANTIDEPRESSANT
MEDICATION
BENZODIAZEPINE
INTOXICATION
CARCINOMA OF PANCREAS
CEREBRAL TUBERCULOSIS
CEREBROVASCULAR SYPHILIS
CESSATION OF AMPHETAMINE OR
COCAINE USE
CIRRHOSIS OF THE LIVER
CORTICOSTEROID TOXICITY
DEGENERATIVE DISEASES OF THE
CENTRAL NERVOUS SYSTEM (E.G.,
ALZHEIMER’S DISEASE,
HUNTINGTON’S CHOREA, PICK’S
DISEASE)
DIABETES
ENCEPHALITIS
36. DRUGS
BENZODIAZEPINES CATIONS
LORAZEPAM 1 OR 2
MG,PO/IM/IV
FAST ACTING,
EASY,DOSING,ABSORPTIO
N
NO ACTIVE METABOLITES
NO GLUCURONIDATION
COPD
SLEEP APNEA
37. ANTIPSYCHOTICS
HALOPERIDOL DROPERIDOL
TYPICAL
HIGH POTENCY
CAN BE COMBINED
LORAZEPAM
TO AVOID S/E:COMBINE
WITH
TRIHEXYPHENIDRYL/BENZ
TROPINE/DIPHENHYDRAM
INE
SEDATION MORE RAPID
PROLONG QT INTERVAL
40. PHYSICAL RESTRAINT
IMPROVISED OR COMMERCIALLY MADE DEVICES
BE FAMILIAR WITH RESTRAINTS USED BY YOUR AGENCY
MAKE SURE YOU HAVE SUFFICIENT PERSONNEL
MINIMUM OF FIVE TRAINED, ABLE-BODIED PEOPLE
DISCUSS THE PLAN OF ACTION BEFORE YOU BEGIN
INCLUDE LAW ENFORCEMENT
USE THE MINIMUM FORCE NECESSARY
DON’T IMMEDIATELY MOVE TOWARD THE PATIENT
41. DO NOT
TIE ANKLES AND WRISTS TOGETHER
HOBBLE TIE
PLACE A PATIENT FACEDOWN IN A REEVES STRETCHER
42. ONCE IN PLACE
DON’T REMOVE RESTRAINTS.
DON’T NEGOTIATE OR MAKE DEALS.
PLACE A MASK OVER THE FACE OF A SPITTING PATIENT.
43. CONTINUOUSLY MONITOR
THE PATIENT.
NEVER PLACE YOUR PATIENT
FACE DOWN.
CHECK PERIPHERAL
CIRCULATION EVERY FEW
MINUTES.
44. ADVERSE EFFECT CAN PRESENT
WITH AGITATION
SEROTONIN SYNDROME NEUROLEPTIC MALIGNANT
SYNDROME
CAUSES-SSRI,SNRI,COMBINATIONS
OF MULTIDRUGS
DIARRHEA/CONFUSION/DELIRIUM
/COMA/INSTABLE
ANS/TREMOR/RIGITY/MYOCLONU
S/AKI/DIC/ARDS/SEIZURE
RFT/LFT/CPK/ECG/CBC/INR
RX:DISCONTINUE
DRUGS/IVF/BDZ/ICU
ANTIPSYCHOTICS
HTN/DIAPHORESIS/TACHYCARDIA/
LIVER
FAILURE/AKI/MYOCLONUS/CONFU
SION/TREMOR/RIGIDITY/ATAXIA
SEROLOGIC MARKERS INCLUDE
ELEVATED CK, DEMONSTRATING
RHABDOMYOLYSIS; METABOLIC
ACIDOSIS; AND LEUKOCYTOSIS
RX:DISCONTINUATION/IVF/DANTR
OLENE/BROMOCRIPTINE/AMANTI
DINE/LEVODOPA/BENZTROPINE/C
LONAZEPAM/ECT
45. LITHIUM
TOXICITY
ASSOCIATED WITH NAUSEA, VOMITING, DIARRHEA,
WEAKNESS, FATIGUE, LETHARGY, CONFUSION, SEIZURE,
AND POTENTIALLY COMA
TOXICITY NOT ENTIRELY CORRELATED WITH SERUM
LITHIUM LEVEL; TOXICITY MAY DEVELOP AT DIFFERENT
LEVELS FOR DIFFERENT PEOPLE
OBTAIN SERUM LITHIUM LEVEL, AND EKG
ENCOURAGE HYDRATION; CONSIDER HEMODIALYSIS IN
EXTREME CASES
47. Suicide Risk Factors
PREVIOUS ATTEMPTS
DEPRESSION
AGE
15–24 OR OVER 40
ALCOHOL OR DRUG ABUSE
DIVORCED OR WIDOWED
GIVING AWAY BELONGINGS
LIVING ALONE OR IN
ISOLATION
PRESENCE OF PSYCHOSIS WITH
DEPRESSION
MANIA
F20
HOMOSEXUALITY
HIV STATUS
MAJOR SEPARATION
TRAUMA
MAJOR PHYSICAL STRESSES
LOSS OF INDEPENDENCE
LACK OF GOALS AND PLAN
FOR THE FUTURE
SUICIDE OF SAME-SEXED
PARENT
EXPRESSION OF A PLAN
FOR SUICIDE
POSSESSION OF THE
MECHANISM FOR SUICIDE
48. SUICIDAL IDEATION
ASSESSMENT
EVERY DEPRESSED PATIENT MUST BE EVALUATED FOR
SUICIDE RISK.
MOST PATIENTS ARE RELIEVED WHEN THE TOPIC IS
BROUGHT UP.
BROACH THE SUBJECT IN A STEPWISE FASHION.
DIAGNOSE HIGHER-RISK PATIENTS
49.
50. DON’TS
DON’T LECTURE, BLAME OR PREACH
DON’T CRITICIZE CLIENT
DON’T DEBATE THE PROS AND CONS OF SUICIDE
DON’T BE MISLED BY CLIENT’S TELLING YOU THE CRISIS HAS
PASSED
DON’T DENY THE CLIENT’S SUICIDAL IDEAS
DON’T TRY TO CHALLENGE
51. DON’T LEAVE CLIENT ISOLATED, UNOBSERVED OR DISCONNECTED
DON’T DIAGNOSE AND ANALYZE BEHAVIOR OR CONFRONT PERSON
WITH INTERPRETATIONS DURING ACUTE PHASE
DON’T BE PASSIVE
DON’T OVER REACT
DON’T KEEP CLIENT’S SUICIDAL RISK A SECRET
DON’T GET SIDE TRACKED ON EXTERNAL ISSUES OR PERSONS
DON’T GLAMORIZE, MARTYRIZE, GLORIFY OR DEFY SUICIDAL
BEHAVIOR IN OTHERS, PAST OR PRESENT
DON’T FORGET TO TREAT THE PSYCHIATRIC ILLNESS
DON’T FORGET TO FOLLOW UP
52. HOMICIDALITY
RISK FACTORS:
HISTORY OF VIOLENCE;
AGGRESSION
IMPULSIVITY; INTOXICATION
SINCERE PLAN
COMMON ETIOLOGIES
INCLUDE:
PSYCHOSIS (COMMAND
AHS); AFFECTIVE
DISORDERS; PERSONALITY
VULNERABILITIES;
SUBSTANCE INTOXICATION
OR WITHDRAWAL
MANAGEMENTS
CLARIFY THREAT TO OTHER(S)
IF THREAT IS DEEMED SERIOUS
NOTIFY POLICE
MAKE EFFORTS TO WARN
INDIVIDUAL(S) (TARASOFF
RULING)
ADMIT PT UNTIL THREAT
SUBSIDES
DON’T HESITATE TO ADMIT
INVOLUNTARILY EVEN IF PRECISE
PSYCHIATRIC DIAGNOSIS REMAINS
ELUSIVE IN THE END
53. AGITATED DELIRIUM
FLUCTUATING SENSORIUM
SUICIDAL AND HOMICIDAL
RISK
COGNITIVE CLOUDING
VISUAL, TACTILE, AND
AUDITORY HALLUCINATIONS
PARANOIA
EVALUATE ALL POTENTIAL
CONTRIBUTING FACTORS AND
TREAT EACH ACCORDINGLY
REASSURANCE, STRUCTURE,
CLUES TO ORIENTATION
BENZODIAZEPINES
HIGH-POTENCY
ANTIPSYCHOTICS MUST BE
USED WITH EXTREME CARE
BECAUSE OF THEIR POTENTIAL
TO ACT PARADOXICALLY AND
INCREASE AGITATION
54. ALCOHOL DEPENDENCE AND
DELIRIUM
CONFUSION,
DISORIENTATION,
FLUCTUATING
CONSCIOUSNESS AND
PERCEPTION, AUTONOMIC
HYPERACTIVITY; MAY BE
FATAL
BDZ
THIAMINE
MET
ANTICRAVING
55. GERIATRIC BEHAVIORAL PROBLEMS
DISTRESS AND PAIN MAY BE
CAUSED BY:
EXPOSURE TO NEW
EXPERIENCES
ALTERATIONS TO
ROUTINES
ANXIETY AND DEPRESSION
ARE TOO OFTEN
CONSIDERED A “NORMAL
PART OF AGING.”
AGEISM:
DISCRIMINATION
AGAINST OLDER PEOPLE
56. PEDIATRIC BEHAVIORAL PROBLEMS
50% OF CHILDHOOD
MENTAL ILLNESSES WILL
PRESENT BY AGE 14
YEARS.
MORE LIKELY TO HAVE
COEXISTING PROBLEMS
DIFFICULT TO DIAGNOSE
MENTAL STATUS
ASSESSMENT IS SIMILAR TO
THAT OF AN ADULT.
EXCEPTION: CONSIDER
DEVELOPMENTAL LEVEL.
ABNORMAL FINDINGS ARE
OFTEN RELATED TO
ADJUSTMENT DISORDERS
AND STRESS.
57. MANAGEMENT
AVOID SEPARATING YOUNG
CHILDREN FROM THEIR
PARENT.
PREVENT CHILDREN FROM
SEEING THINGS THAT WILL
INCREASE THEIR DISTRESS.
MAKE ALL EXPLANATIONS
BRIEF AND SIMPLE.
BE CALM AND SPEAK SLOWLY.
IDENTIFY YOURSELF.
BE TRUTHFUL WITH CHILDREN.
ENCOURAGE CHILDREN TO
HELP WITH THEIR CARE
REASSURE CHILDREN BY
CARRYING OUT ALL
INTERVENTIONS GENTLY.
DO NOT DISCOURAGE
CHILDREN FROM CRYING OR
SHOWING EMOTIONS.
IF YOU WILL BE SEPARATED
FROM CHILDREN, INTRODUCE
THE NEXT PERSON WHO WILL
ASSUME THEIR CARE.
ALLOW CHILDREN TO KEEP A
FAVORITE BLANKET OR TOY.
DO NOT LEAVE CHILDREN
ALONE.
58. ABUSE OF CHILD OR ADULT
SIGNS OF PHYSICAL
TRAUMA
MANAGEMENT OF
MEDICAL PROBLEMS
59. ADOLESCENT CRISES
SUICIDAL ATTEMPTS AND
IDEATION
SUBSTANCE ABUSE
TRUANCY, TROUBLE WITH
LAW
PREGNANCY
RUNNING AWAY
EATING DISORDERS
PSYCHOSIS
EVALUATION OF SUICIDAL
POTENTIAL
EXTENT OF SUBSTANCE
ABUSE
FAMILY DYNAMICS
CRISIS-ORIENTED
FAMILY AND INDIVIDUAL
THERAPY
HOSPITALIZATION IF
NECESSARY
CONSULTATION WITH
APPROPRIATE
EXTRAFAMILIAL
AUTHORITIES
60. BORDERLINE PERSONALITY
DISORDER
SUICIDAL IDEATION AND
GESTURES
HOMICIDAL IDEATIONS
AND GESTURES
SUBSTANCE ABUSE
MICRO PSYCHOTIC
EPISODES
BURNS, CUT MARKS ON
BODY
SUICIDAL AND HOMICIDAL
EVALUATION (IF GREAT,
HOSPITALIZATION)
SMALL DOSAGES OF
ANTIPSYCHOTICS
CLEAR FOLLOW-UP PLAN
61. BRIEF PSYCHOTIC DISORDER
EMOTIONAL TURMOIL
EXTREME LABILITY
ACUTELY IMPAIRED
REALITY TESTING AFTER
OBVIOUS PSYCHOSOCIAL
STRESS
HOSPITALIZATION OFTEN
NECESSARY
LOW DOSAGE OF
ANTIPSYCHOTICS MAY BE
NECESSARY BUT OFTEN
RESOLVES SPONTANEOUSLY
62. CATATONIC SCHIZOPHRENIA
MARKED PSYCHOMOTOR
DISTURBANCE (EITHER
EXCITEMENT OR STUPOR)
EXHAUSTION
CAN BE FATAL
RAPID TRANQUILIZATION
WITH ANTIPSYCHOTICS
MONITOR VITAL SIGNS
AMOBARBITAL MAY
RELEASE PATIENT FROM
CATATONIC MUTISM
STUPOR BUT CAN
PRECIPITATE VIOLENT
BEHAVIOR
LORAZEPAM CAN BE USED
63. DELUSIONAL DISORDER
MOST OFTEN BROUGHT IN
TO EMERGENCY ROOM
INVOLUNTARILY; THREATS
DIRECTED TOWARD OTHERS
ANTIPSYCHOTICS IF PATIENT
WILL COMPLY (IM IF
NECESSARY)
INTENSIVE FAMILY
INTERVENTION
HOSPITALIZATION IF
NECESSARY
64. DEMENTIA
UNABLE TO CARE FOR SELF
VIOLENT OUTBURSTS
PSYCHOSIS
DEPRESSION AND SUICIDAL
IDEATION
CONFUSION
SMALL DOSAGES OF HIGH-
POTENCY ANTIPSYCHOTICS
CLUES TO ORIENTATION
ORGANIC EVALUATION,
INCLUDING MEDICATION
USE
FAMILY INTERVENTION
65. DEPRESSIVE DISORDERS
SUICIDAL IDEATION AND
ATTEMPTS
SELF-NEGLECT
SUBSTANCE ABUSE
ASSESSMENT OF DANGER
TO SELF
HOSPITALIZATION IF
NECESSARY
NONPSYCHIATRIC CAUSES
OF DEPRESSION MUST BE
EVALUATED
66. Panic disorder
PANIC, TERROR; ACUTE
ONSET
MUST DIFFERENTIATE
FROM OTHER ANXIETY-
PRODUCING DISORDERS,
BOTH MEDICAL AND
PSYCHIATRIC; ECG TO RULE
OUT MITRAL VALVE
PROLAPSE
ALPRAZOLAM (0.25 TO 2.0
MG); LONG-TERM
MANAGEMENT MAY
INCLUDE AN
ANTIDEPRESSANT
67. HOMOSEXUAL PANIC
ADAMANTLY DENY
HAVING ANY
HOMOEROTIC IMPULSES
AROUSED BY TALK, A
PHYSICAL OVERTURE
PLAY AMONG SAME-SEX
FRIENDS
PANICKED PERSON SEES
OTHERS AS SEXUALLY
INTERESTED IN HIM
VENTILATION,
ENVIRONMENTAL
STRUCTURING
BDZ/ ANTIPSYCHOTICS MAY
BE REQUIRED
OPPOSITE-SEX CLINICIAN
SHOULD EVALUATE THE
PATIENT WHENEVER POSSIBLE
69. RAPE AND SEXUAL ASSAULT
AN UNEXPECTED AND VIOLENT THREAT ON ONE’S LIFE.
IT IS A LOSS, VIOLATION AND INSTANT DEMORALIZATION.
TYPICAL REACTIONS INCLUDE SHAME, HUMILIATION,
ANXIETY, CONFUSION AND OUT RAGE.
70. MANAGEMENT
1. STAY WITH THE PATIENT THE ENTIRE TIME IN THE E.R.
2.EXPLANATIONS FOR SPECIFIC DATA THAT IS NEEDED.
3. CONSENT FOR EXAMINATION AND SPECIMEN COLLECTION
4. PATIENT AND CONSIDERATE. NEVER PRESS OR HARASS THE
PATIENT FOR ANSWERS.
5.ANSWER THE PATIENT’S QUESTIONS AND FREQUENT
REASSURANCE THAT THE PATIENT IS IN A SAFE PLACE.
6.THE PATIENT MUST BE GIVEN TIME AND DATE TO MAKE HER
OWN DECISION ABOUT THE LEGAL PROCESS.
71. 7. EDUCATE THE PATIENT ABOUT THE RAPE TRAUMA
SYNDROME.
8. CALL THE PATIENT 48HOURS LATER AND THEN WEEKLY FOR
FOLLOW UP.
9. ON LATER STAGES, PROVIDE COUNSELING WITH REALISTIC
ISSUES SUCH AS WORK, HOME, LEGAL DIFFICULTIES, SHARING
OF EMOTION, FUTURE REHABILITATION.
72. CRISIS INTERVENTION
UNEXPECTED SERIES OF EVENT
DANGER OR OPPORTUNITY
PHYSICAL,PSYCHOLOGICAL,INTERPERSONAL
DEVELOPMENTAL,SITUATIONAL
74. DEATH AND DYING
DENIAL AND ISOLATION
ANGER
BARGAINING
DEPRESSION
ACCEPTANCE
75. LEGAL ISSUES IN EMERGENCY
PSYCHIATRY
CONFIDENTIALITY
DUTY TO WARN
COMPETENCY
INFORMED CONSENT
INVOLUNTARY COMMITMENT
76. BEHAVIORAL EMERGENCIES CAN PRESENT UNIQUE
CHALLENGES IN PATIENT MANAGEMENT. FOCUS ON
REDUCING THE PATIENT’S STRESS WITHOUT
EXPOSING OWNSELF TO UNNECESSARY RISKS.
77. OUR GREATEST WEAKNESS LIES IN GIVING UP. THE
MOST CERTAIN WAY TO SUCCEED IS ALWAYS TO TRY
JUST
ONE MORE TIME.
-THOMAS EDISON
78. REFERENCES
Kaplan & sadock's comprehensive textbook of
psychiatry, 9th edition
Emergency psychiatry by Hani raoul khouzam,Doris
tiu tan,Tirath sing gill