1) The triangular fibrocartilage complex (TFC) provides stability to the distal radioulnar joint (DRUJ) and allows for forearm pronation and supination. Injuries to the TFC can cause ulnar-sided wrist pain and DRUJ instability.
2) Clinical examination of TFC injuries may reveal DRUJ instability on tests like the ulnar fovea sign and distal ulna ballottment test. MRI or arthroscopy can help diagnose the specific type of TFC tear.
3) Surgical treatment depends on the type and location of the TFC tear. Debridement is used for central perforations while suture repair or foveal
Hoffa's Fracture: Diagnosis, management & New Classification System by BAGARI...Vaibhav Bagaria
Hoffa's Fracture - coronal split fracture of distal femur, its diagnosis, management strategy, a new classification and tips and tricks of management. First described Hoffa, a new classification system by Bagaria et al helps plan the surgery for these tricky fracture. The most crucial step is not to miss these fractures in ER.
a summary of the pertinent elbow anatomy, mechanism of injury, primary and secondary stabilizers of the elbow, and treatment options of elbow terrible triad
Tendoachilles rupture and its managementRohan Vakta
Achilles tendon is the strongest tendon of body. There are many causes of its rupture. It can be acute or chronic rupture. Management of chronic rupture by semitendinosus tendon is mentioned here.
TFCC Repair in 2014: from hammoc to icebergNikos Darlis
State of the art in Triangular FibroCartilage Complex lesion management. Current concepts in anatomy biomechanics and treatment with special focus in arthroscopic techniques. Detailed step by step description of the surgical technique with animations and video. See also https://www.youtube.com/watch?v=rgbemvKbtFk. Visit www.orthoinfo.gr
Συγχρονες τεχνικές αντιμετώπισης των βλαβών του Τρίφωνου Ινοχόνδρινου Συμπλέγματος στον Καρπό
Hoffa's Fracture: Diagnosis, management & New Classification System by BAGARI...Vaibhav Bagaria
Hoffa's Fracture - coronal split fracture of distal femur, its diagnosis, management strategy, a new classification and tips and tricks of management. First described Hoffa, a new classification system by Bagaria et al helps plan the surgery for these tricky fracture. The most crucial step is not to miss these fractures in ER.
a summary of the pertinent elbow anatomy, mechanism of injury, primary and secondary stabilizers of the elbow, and treatment options of elbow terrible triad
Tendoachilles rupture and its managementRohan Vakta
Achilles tendon is the strongest tendon of body. There are many causes of its rupture. It can be acute or chronic rupture. Management of chronic rupture by semitendinosus tendon is mentioned here.
TFCC Repair in 2014: from hammoc to icebergNikos Darlis
State of the art in Triangular FibroCartilage Complex lesion management. Current concepts in anatomy biomechanics and treatment with special focus in arthroscopic techniques. Detailed step by step description of the surgical technique with animations and video. See also https://www.youtube.com/watch?v=rgbemvKbtFk. Visit www.orthoinfo.gr
Συγχρονες τεχνικές αντιμετώπισης των βλαβών του Τρίφωνου Ινοχόνδρινου Συμπλέγματος στον Καρπό
Διάγνωση και αντιμετώπιση της οξείας ασταθειας της απω κερκιδωλενικής. Acute distal radioulnar joint Instability, isolated and with concommitan fracture, diagnosis and treatment
Triangular Fibrocartilage Complex - A Sprain in the WristJeffBudoff
Triangular Fibrocartilage Complex (TFCC) is a complicate structure and is a chief provider to the strength and stability of the wrist. It is also a common source of pain for the wrist by the side of the ulna (small finger).
3 common clinical scenarios leading to wrist arthroscopy. Alexandropolis 2014Νίκος Δαρλής
Overview of the 3 most common clinical scenarios leading to wrist arthroscopy. Invited lecture at the 20th Congress of the Hellenic Hand Surgery Society Meeting, Sep 4-6 2014.
Ανασκόπηση των 3 κυριοτέρων κλινικών Σεναρίων που οδηγούν σε αρθροσκόπηση του καρπού. Προσκεκλημένη Ομιλία στο 20ο Συνέδριο της Ελληνικής Εταιρείας Χειρουργικής του Χεριού, 4-6 Σεπ, Αλεξανδρούπολη,
This video explains Lumbar Disc Replacement in Detail. When degenerative disc disease begins to affect the spine this is called degenerative disc disease. This video highlights the history, epidemiology, and treatment options both conservative and surgical. If you or someone you know needs to be seen in regards to Lumbar Disc Replacement feel free to look us up online www.beverlyspine.com or www.santamonicaspine.com OR call toll free 1-8SPINECAL-1
Αρθροσκόπηση του Καρπού, απο την Κλινική Εξέταση στις Σύνθετες Επεμβάσεις- Wr...Nikos Darlis
Review of the clinical exam, radiologic findings and operative treatment of common wrist conditions treated with wrist arthroscopy
Ομιλία στο Σεμινάριο Χειρουργικής του Χεριού, Ιωάννινα 30 Οκτ- 1 Νοε, 2014. "Ανασκόπηση της Αρθροσκόπησης στο Χέρι".
the slide describes femoral fracture with case presentations as well as rediological diagnosis ,when opened and closed .the management from emergency period and through to stabilization
Τρία κοινά κλινικά σενάρια που οδηγούν σε αρθροσκόπηση του καρπού- Three comm...Nikos Darlis
Three common clinical scenarios leading to wrist arthroscopy
Ομιλία στο 20ο Συνέδριο της Ελληνικής Εταιρείας Χειρουργικής του Χεριού, 4-6 Σεπ, Αλεξανδρούπολη,
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
10. Anatomy
• DRUJ congruity
– 60% in neutral position
– 10% during maximal P/S
Arthroscopic Management of
Ulnar Pain, F. del Piñal et al.
11. Anatomy
• The dorsal capsule
– extended in P
– folded in S
• The palmar capsule
– extended in S
– folded in P
Arthroscopic Management of
Ulnar Pain, F. del Piñal et al.
12. Anatomy
• TFCComplex
– triangular fibrocartilage
– meniscus homologue
– RU ligaments
– UL and UT ligaments
– sheat floor of ECU
– ulnar joint capsule
Arthroscopic Management of
Ulnar Pain, F. del Piñal et al.
14. Clinical Examination
• DRUJ instability
– ulnar fovea sign
• DRUJ instability
– ulnar fovea sign
Atzei A et al. Foveal TFCC Tear Classification
and Treatment. Hand Clin 2011
15. Clinical Examination
• DRUJ instability
– ulnar fovea sign
Arthroscopic Management of
Ulnar Pain, F. del Piñal et al.
• DRUJ instability
– ulnar fovea sign
– distal ulna ballottment test
16. Clinical Examination
• DRUJ instability
– ulnar fovea sign
– distal ulna ballottment test
Functional Evaluation of the
Distal Radioulnar Joint, N.
Badur and M. Garcia-Elias
17. Clinical Examination
• DRUJ instability
– ulnar foveal sign
– distal ulna ballottment test
– piano key sign
Functional Evaluation of the
Distal Radioulnar Joint, N.
Badur and M. Garcia-Elias
25. Diagnosis: arthroscopy
• Hook test
– positive TFCC tear
– negative No tear
Arthroscopic Management of Ulnar
Pain. F. del Piñal et al.
26. Diagnosis: arthroscopy
• Hook test1, 2
– positive TFCC tear
– negative No tear
1
Atzei A et al. New trends in
arthroscopic management of
type 1-B TFCC injuries with
DRUJ instability. JHS Eur
2009
2
Atzei A et al. Foveal TFCC
tear classification and
treatment. Hand Clin 2011
Video: https://www.youtube.com/watch?v=EO8VR5XUF2g
28. Diagnosis: arthroscopy
• Trampoline test1
– positive TFCC tear
– negative No tear
1
Hermansdorfer JD et al.
Management of chronic
peripheral tears of the TFCC.
JHS Am 1991
Video: https://www.youtube.com/watch?v=u2zC5DgFUFA
29. Palmer Classification of TFCC Lesions1
• I Traumatic injury
– A: central perforation
– B: ulnar avulsion
– C: distal avulsion
– D: radial avulsion
• II Degenerative injury
1
Palmer AK. Triangular fibrocartilage complex
lesions: a classification. JHS Am 1989
30. Central perforation
Oneson SR., Chamoy L. et al. MR
Interpretation of the Palmer Classification of
TFCC Lesions. RadioGraphic 1996
• Palmer Class IA Lesions
– perforation of TFC proper
– sagittally oriented
– avascular portion
– debridement
Nakamura T. et al. Repair of foveal
detachment of the triangular fibrocartilage
complex: open and arthroscopic
transosseous techniques. Hand Clin 2011
32. Distal (Volar) avulsions
• Palmer Class IC Lesions
– avulsion volar attachments
– sagittally oriented
– ulnocarpal instability
– surgical repair
– debridement
– ulnar shortening
Oneson SR., Chamoy L. et al. MR
Interpretation of the Palmer Classification of
TFCC Lesions. RadioGraphic 1996
33. Radial avulsions
• Palmer Class ID Lesions
– radial avulsion
– less common than IA/B
– by distal radius fractures
– avascular
– surgical repair
– debridement
Oneson SR., Chamoy L. et al. MR
Interpretation of the Palmer Classification of
TFCC Lesions. RadioGraphic 1996
37. Ulnar avulsions
• Palmer Class IB Lesions
– well-vascularized
– ulnar lesions
– capsule tear
– foveal tear
– surgical repair by instability
Oneson SR., Chamoy L. et al. MR
Interpretation of the Palmer Classification of
TFCC Lesions. RadioGraphic 1996
38. Ulnar avulsions
• Palmer Class IB Lesions
– well-vascularized
– ulnar lesions
– capsule or distal tear
– foveal or proximal tear
– complete tear
– surgical repair by instability
Nakamura T. et al. Functional anatomy of the
triangular fibrocartilage complex. JHS Br 1996
39. Ulnar avulsions
• Palmer Class IB Lesions
– well-vascularized
– ulnar lesions
– capsule or distal tear
– foveal or proximal tear
– complete tear
– surgical repair by instability
Nakamura T. et al. Functional anatomy of the
triangular fibrocartilage complex. JHS Br 1996
40. Atzei-EWAS Classification of TFCC1
1
Atzei A. New Trends in arthroscopic
management of 1-B TFCC injuries with
DRUJ instability. JHS Eur 2009
41. Atzei-EWAS Classification of TFCC1
Palmer Class IB
1
Atzei A. New Trends in arthroscopic
management of 1-B TFCC injuries with
DRUJ instability. JHS Eur 2009
42. Atzei-EWAS Classification of TFCC1
Palmer Class II
1
Atzei A. New Trends in arthroscopic
management of 1-B TFCC injuries with
DRUJ instability. JHS Eur 2009
48. Surgical Treatment: foveal refixation
• Repair: foveal refixation
Nakamura T. et al. Repair of foveal
detachment of the triangular fibrocartilage
complex: open and arthroscopic
transosseous techniques. Hand Clin 2011
51. Open versus Arthroscopic Repair
Lucchetti R. Comparison between open and arthroscopic-
assisted foveal triangular fibrocartilage complex repair for post-
traumatic distal radio-ulnar joint instability. JHS Eur 2014
52. Rehabilitation
• 1-3° weeks
– long-arm cast
– neutral rotation
– elbow F/E
• 4-6° weeks
– short cast
– start wrist F/E
– assisted forearm rotation
• 7-10° weeks
– short cast at night
– resume daily activities
Sport and heavy works tasks > 3 months
53. • 1-3° weeks
– short cast
– start wrist F/E
– assisted forearm rotation
• 4-6° weeks
– wrist widget during the day
– short cast at night
– active E/F and P/S
• 7-10° weeks
– resume daily activities
– progressive forceful loading
Partial lesion: treatment
54. The author declares that the research for and communication
of this independent body of work does not constitute any
financial or other conflict of interest.
Take home message
• Clinical assessment
• MRI +/- arthrography
• Arthroscopic repair
• 3-6 months rehabilitation