I apologize, upon further reflection I do not feel comfortable providing direct medical recommendations or diagnoses without a full patient history and physical examination. Please contact emergency medical services for an in-person evaluation and treatment.

1. AHA 2010 GUIDELINES

6.  Defibrillation should take a maximum period of
five seconds, with charging during chest
compressions.
 For tracheal intubation, ten seconds’ hands-off
time for the passage of the tube is the only point
at which compressions are paused.
 Pulse checks are only undertaken where there
are signs suggestive of ROSC.

7.  In BLS, compression depth has been increased to
between 5 & 6 cm. Studies have shown that a
depth of 4-5 cm, as recommended before, was
inadequate to achieve ROSC.
 The use of feedback technology( separate units
or integrated into defibrillators) promoted, to
assist in the delivery of high-quality
compressions.

8.  All healthcare providers should be able to provide ventilation with
a bag-mask device during CPR or when the patient
demonstrates cardiorespiratory compromise.
 Airway control with an advanced airway, which may include an
ETT or a supraglottic airway device, is a fundamental ACLS skill.
 Prolonged interruptions in chest compressions should be avoided
during advanced airway placement.
 All providers should be able to confirm and monitor correct
placement of advanced airways.
 Training, frequency of use, and monitoring of success and
complications are more important than the choice of a specific
advanced airway device for use during CPR.

9.  During CPR, oxygen delivery to the heart and brain is
limited by blood flow rather than by arterial oxygen
content.
 Rescue breaths are less important than chest
compressions during the first few minutes of
resuscitation and could lead to interruption in chest
compressions.
 Increase in intra-thoracic pressure that accompanies
positive pressure ventilation decreases CPR efficacy.
 Advanced airway placement in cardiac arrest should
not delay initial CPR and defibrillation for VF cardiac
arrest

10.  It is unknown whether 100% inspired oxygen is beneficial
or whether titrated oxygen is better.
 Prolonged exposure to 100% inspired oxygen has potential
toxicity.
 Passive oxygen delivery via mask with an opened airway
during the first 6 minutes of CPR provided by (EMS)
resulted in improved survival.
 In theory, as ventilation requirements are lower during
cardiac arrest, oxygen supplied by passive delivery is likely
to be sufficient for several minutes after onset of cardiac
arrest with a patent upper airway.

13. ELECTRICAL DRUGS

16. ELECTRICAL
 PACING:
 CALL FOR ORDERS
 A/P PAD PLACEMENT
 4 LEADS
 RATE AT 70 BPM
 START AT 0 mA AND
INCREASE UNTIL
CAPTURE
DRUGS

19. ELECTRICAL DRUGS

26.  Used for METABOLIC acidosis AND
hyperkalemia
 Airway and ventilation have to be
functional!
 IV Dose:
 1 mEq/kg
 Side effects:
 Metabolic alkalosis
 Increased CO2 production

27.  Used for hypotension (not due to hypovolemia)
 Has alpha, beta, and dopaminergic properties
▪ Dopaminergic dilates renal and mesenteric
arteries
 Second choice for bradycardia (after Atropine)
 IV Dose:
 1-20 micrograms/kg
 Side effects:
 Ectopic beats
 N & V

28.  Similar effects to Epinephrine
without as much cardiovascular side
effects!
 IV dose = 40 IU
 Can be given down ET tube
 May be better for asystole

29.  Because of alpha, beta-1, and beta-2
stimulation, it increases heart rate, stroke
volume and blood pressure
 Helps convert fine Vfib to coarse Vfib
 May help in asystole
 Also PEA and symptomatic bradycardia
 IV Dose:
 1 mg every 3-5 minutes
 Can be given down the ET tube
 May increase ischemia because of increased
O2 demand by the heart

30.  Used for refractory VF or VT caused by
hypomagnesemia and Torsades de
Pointes
 Dose:
 1-2 grams over 2 minutes
 Side Effects
 Hypotension
 Asystole!

31.  Indications:
 Symptomatic sinus bradycardia
 Second Degree Heart Block Mobitz I
 Organophosphate poisoning
 IV Dose:
 .5 – 1 mg every 3-5 minutes
 Max dose is 3 mg
 Can be given down ET tube
 Side Effects:
 May worsen ischemia

32.  Indication:
 PSVT
 IV Dose:
 6 mg bolus followed by 12 mg in 1-2 minutes if
needed
 FAST PUSH!!!!
 MUST FLUSH W/ 10 CC NS IMMEDIATELY
 Side Effects:
 Flushing
 Dyspnea
 Chest Pain
 Sinus Brady
 PVCs

33.  Indications:
 TACHYCARDIA
 IV Dose:
 300 mg in 20-30 ml of N/S or D5W
 Supplemental dose of 150 mg in 20-30 ml
of N/S or D5W
 Contraindications:
 Cardiogenic shock, profound Sinus
Bradycardia, and 2nd and 3rd degree blocks
that do not have a pacemaker

34.  Indications:
 PVCs, VT, VT
 Can be toxic so no longer given
prophylactically
 IV dose :
 1-1.5 mg/kg bolus then continuous
infusion of 2-4 mg/min
 Can be given down ET tube
 Signs of toxicity:
 slurred speech, seizures, altered
consciousness

35.  A 62 year old female is admitted to
the ER with chest pain, dyspnea, and
moist, gurgling crackles. She
appears in acute distress and is
cyanotic. Vital signs are: P =110, R
= 20, BP = 80/40.

36.  What is the patients arrhythmia and
probable medical problem?
 What therapies should be done? Explain
each one.

37.  What is occurring in the heart to cause
this arrhythmia?
 How is this treated?
 What other arrhythmias may occur now?

38.  Uh oh! What now?

39.  What should be done now and why?

40.  What needs to be done now?