What Is Fetal Alcohol Syndrome?
1) When a pregnant person drinks alcohol, some of that alcohol easily passes across the placenta to the fetus.
2) People with this condition may have problems with their vision, hearing, memory, attention span, and abilities to learn and communicate.
3) You can prevent fetal alcohol syndrome by avoiding alcohol during pregnancy.
What Is Fetal Alcohol Syndrome?
1) When a pregnant person drinks alcohol, some of that alcohol easily passes across the placenta to the fetus.
2) People with this condition may have problems with their vision, hearing, memory, attention span, and abilities to learn and communicate.
3) You can prevent fetal alcohol syndrome by avoiding alcohol during pregnancy.
FETAL ALCOHOL SYNDROME (FAS) is the leading known cause of mental retardation
and birth defects in the world. Fetal alcohol syndrome is a pattern of physical, behavioral and
cognitive abnormalities seen in individuals exposed to alcohol in uterus. Because alcohol is a
known teratogen and the damage done to a fetus by alcohol exposure is permanent, public
education about the dangers of prenatal alcohol exposure has been extensive. Many of the
features of the fetal alcohol syndrome are secondary to the effect of alcohol on brain
development. These include microcephaly, short palpebral fissures, the long smooth philtrum
and thin vermilion of the upper lip, joint anomalies, altered palmar crease pattern, and mental
retardation. Animal studies as well as a limited amount of human data also show that
maternal genotype is a key player : Advances in the development of novel antioxidant
therapies as an approach for fetal alcohol syndrome prevention. Peptides NAPVSIPQ (NAP)
and SALLRSIPA (SAL), related to activity-dependent neuroprotective protein (ADNP),
prevent alcohol-induced damage in a mouse model of FAS. In a recent study, the thyroxin
reversed the deficit both in the level of their genes and their social behaviour, research is
going on how the prenatal thyroid hormone supplementation reverses the behavioural deficits
in the fetal alcohol spectrum disorder model. Another study used a priori approach to assess
molecular phenotype in the cranial neural folds (head fold) of early mouse embryos soon
after maternal alcohol treatment. FAS can be modulated pharmacologically with PK11195, a
potent ligand with specific binding to the mitochondrial peripheral-type benzodiazepine
receptor recognition site. PK11195 has been shown to protect early mouse embryos from eye
and brain defects induced with diverse teratogens and to protect adult tissues from some
inflammatory lesions. Microarray transcript profiling of the embryonic head fold at 3.0 hr
after alcohol exposure or PK11195 counter-exposure enabled prioritization of candidate
pathways that integrate the genomic response with genetic susceptibility of the system. These
findings are consistent with the growing view that developmental exposure to alcohol alters
common signalling pathways linking receptor activation to cytoskeletal reorganization. The
programmatic shift in cell motility and metabolic capacity further implies cell signals and
responses that are integrated by the mitochondrial recognition site for PK11195.
CONCLUSIONS: Until the advent of effective prevention measures, it will remain
necessary to seek ways to treat the life-long neurobehavioral consequences of prenatal
alcohol exposure. Nevertheless, alcohol is a widely accepted and legal social drug, and many
pregnant mothers continue to drink it while pregnant. Whereas, FAS is totally preventable by
avoiding alco
Congenital Adr Hyperplasia (CAH) can appear at any age from birth to puberty where it can lead to ambiguous genitalia. It is due to absolute or relative deficiency of 17 Hydroxylase or 21 Hydroxylase enzyme.
Pediatrics. trisomy 21. Meiotic non-disjunction of chromosome 21. clinical features and associated abnormalities of down syndrome. screening test for down syndrome. counseling for parents in down syndrome.
FETAL ALCOHOL SYNDROME (FAS) is the leading known cause of mental retardation
and birth defects in the world. Fetal alcohol syndrome is a pattern of physical, behavioral and
cognitive abnormalities seen in individuals exposed to alcohol in uterus. Because alcohol is a
known teratogen and the damage done to a fetus by alcohol exposure is permanent, public
education about the dangers of prenatal alcohol exposure has been extensive. Many of the
features of the fetal alcohol syndrome are secondary to the effect of alcohol on brain
development. These include microcephaly, short palpebral fissures, the long smooth philtrum
and thin vermilion of the upper lip, joint anomalies, altered palmar crease pattern, and mental
retardation. Animal studies as well as a limited amount of human data also show that
maternal genotype is a key player : Advances in the development of novel antioxidant
therapies as an approach for fetal alcohol syndrome prevention. Peptides NAPVSIPQ (NAP)
and SALLRSIPA (SAL), related to activity-dependent neuroprotective protein (ADNP),
prevent alcohol-induced damage in a mouse model of FAS. In a recent study, the thyroxin
reversed the deficit both in the level of their genes and their social behaviour, research is
going on how the prenatal thyroid hormone supplementation reverses the behavioural deficits
in the fetal alcohol spectrum disorder model. Another study used a priori approach to assess
molecular phenotype in the cranial neural folds (head fold) of early mouse embryos soon
after maternal alcohol treatment. FAS can be modulated pharmacologically with PK11195, a
potent ligand with specific binding to the mitochondrial peripheral-type benzodiazepine
receptor recognition site. PK11195 has been shown to protect early mouse embryos from eye
and brain defects induced with diverse teratogens and to protect adult tissues from some
inflammatory lesions. Microarray transcript profiling of the embryonic head fold at 3.0 hr
after alcohol exposure or PK11195 counter-exposure enabled prioritization of candidate
pathways that integrate the genomic response with genetic susceptibility of the system. These
findings are consistent with the growing view that developmental exposure to alcohol alters
common signalling pathways linking receptor activation to cytoskeletal reorganization. The
programmatic shift in cell motility and metabolic capacity further implies cell signals and
responses that are integrated by the mitochondrial recognition site for PK11195.
CONCLUSIONS: Until the advent of effective prevention measures, it will remain
necessary to seek ways to treat the life-long neurobehavioral consequences of prenatal
alcohol exposure. Nevertheless, alcohol is a widely accepted and legal social drug, and many
pregnant mothers continue to drink it while pregnant. Whereas, FAS is totally preventable by
avoiding alco
Congenital Adr Hyperplasia (CAH) can appear at any age from birth to puberty where it can lead to ambiguous genitalia. It is due to absolute or relative deficiency of 17 Hydroxylase or 21 Hydroxylase enzyme.
Pediatrics. trisomy 21. Meiotic non-disjunction of chromosome 21. clinical features and associated abnormalities of down syndrome. screening test for down syndrome. counseling for parents in down syndrome.
Neurotransmitter and neuroendocrinologyPooja Saharan
neurotransmitter description and neuroendocrinology.How alteration in the hormones secreted by pituitary and thyroid can results into emotional and behavioral problems.
INTRODUCTION TO OCCUPATIONAL THERAPY, GOALS OF OCCUPATIONAL THERAPY, THE VARIOUS CLASSIFICATION OF OCCUPATIONAL THERAPY, DIFFERENT OCCUPATIONAL THERAPIES FOR DIFFERENT MENTA DISORDERS TO TREAT THE PATIENTS EFFECTIVELY.
PowerPoint presentation regarding many facets of Fetal Alcohol Spectrum Disorder. Feel free to share this with anyone who would benefit from this information. Some of the information is quite shocking. Included parenting and teaching strategies for those responsible for caring for a child with FASD.
Abstract: Fetal alcohol spectrum disorder (FASD) is a significant public health issue in Australia that is poorly diagnosed, chronic and costly.
FASD is a diffuse acquired brain injury secondary to prenatal alcohol exposure. The prevalence rate of FASD among the general population in Australia is currently unknown; however, an Australian study in a selected high-risk population reported some of the highest rates of FASD in the world. A common misconception among clinicians is that a child must have ‘the face’ of FASD to have the disorder. This is incorrect. The three
sentinel facial features only occur in the minority of individuals with FASD. FASD should be considered as a ‘whole body’ disorder as increased susceptibility to chronic health problems suggests suboptimal in utero environments places the individual at risk of later disease. Clinicians are reluctant to consider FASD as a possible diagnosis because of the concern of inducing stigma; however, this concern is neither supported by the
evidence nor patient stories. The Australian Guide to the Diagnosis of FASD is now available to assist health professionals in providing timely and accurate diagnoses, which can lead to improved outcomes via evidence-based intervention and is an important first step in future prevention.
Of the 415 children in the study (195 girls and 220 boys; mean [SD] age, 363.0 [8.3] days), a consistent association between craniofacial shape and prenatal alcohol exposure was observed at almost any level regardless of whether exposure occurred only in the first trimester or throughout pregnancy. Regions of difference were concentrated around the midface, nose, lips, and eyes. Directional visualization showed that these differences corresponded to general recession of the midface and superior displacement of the nose, especially the tip of the nose, indicating shortening of the nose and upturning of the nose tip.
Differences were most pronounced between groups with no exposure and groups with low exposure in the first trimester (forehead), moderate to high exposure in the first trimester (eyes, midface, chin, and parietal region), and binge-level exposure in the first trimester (chin).
CONCLUSIONS AND RELEVANCE Prenatal alcohol exposure, even at low levels, can influence craniofacial development. Although the clinical significance of these findings is yet to be determined, they support the conclusion that for women who are or may become pregnant, avoiding alcohol is the safest option.
Response to the five Danish papers. Submitted but not accepted for publicationBARRY STANLEY 2 fasd
In 2012 the British Journal of Obstetrics and Gynaecology publish five papers from the Danish Lifestyle During Pregnancy Study Group. These publications received a great deal or world wide media coverage. The emphasis of most of this coverage was in support of drinking low to moderate amounts of alcohol during pregnancy.
Over a period of approximately six months I corresponded with the editor of the BJOG to have a letter of response published in the journal. Although I modified my letter as was requested it was not published.
Barry Stanley
A systematic review of prevention interventions to reduce prenatal alcohol ex...BARRY STANLEY 2 fasd
Fetal alcohol spectrum disorder (FASD) is a preventable, lifelong neurodevelopmental disorder caused by prenatal alcohol
exposure. FASD negatively impacts individual Indigenous communities around the world. Although many prevention
interventions have been developed and implemented, they have not been adequately evaluated. This systematic review updates
the evidence for the effectiveness of FASD prevention interventions in Indigenous/Aboriginal populations internationally, and in specific populations in North America and New Zealand, and offers recommendations for future work.
The range of consequences from drinking alcohol during pregnancy are collectively called fetal alcohol spectrum disorders, as not all signs and symptoms are present in all children with the disorder. This range includes:
Alcohol-related neurodevelopmental disorder — intellectual disabilities or behavioral and learning problems caused by drinking alcohol during pregnancy
Alcohol-related birth defects — physical birth defects caused by drinking alcohol during pregnancy
Fetal alcohol syndrome — the severe end of the fetal alcohol spectrum disorders, which includes both neurodevelopmental disorder and birth defects caused by drinking alcohol during pregnancy
Partial fetal alcohol syndrome — presence of some signs and symptoms of fetal alcohol syndrome caused by drinking alcohol during pregnancy, but the criteria for the diagnosis are not met
Irit Bar Netzer: Children with Fetal Alcohol Syndrome in Adoptive and Foster ...Beitissie1
The lecture focuses on the great importance of clinical and therapeutic intervention in improving behaviors, parent-child relationships and more, in Fetal Alcohol Syndrome cases.
Irit Bar Netzer: Children with Fetal Alcohol Syndrome in Adoptive and Foster ...Beitissie1
The lecture focuses on the great importance of clinical and therapeutic intervention in improving behaviors, parent-child relationships and more, in Fetal Alcohol Syndrome cases.
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
263778731218 Abortion Clinic /Pills In Harare ,sisternakatoto
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New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
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micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
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Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
2. Fetal Alcohol Spectrum Disorders
(FASD)
• Prenatal exposures to alcohol cause wide
range of disorders
• One of the most severe effects of drinking is
Fetal Alcohol Syndrome (FAS)
3.
4. Fetal Alcohol Syndrome
• Fetal Alcohol Syndrome (FAS) is a birth defect
caused by drinking alcohol during pregnancy
• One of the most severe defects for a fetus
• Leading known preventable cause of mental
retardation/other birth defects
5. • Int J Environ Res Public Health. 2013 Apr 16;10(4):1547-61. doi: 10.3390/ijerph10041547.
• Prevalence of fetal alcohol syndrome and maternal characteristics in a sample of schoolchildren from a rural
province of croatia.
• hildren's Hospital Srebrnjak, Srebrnjak 100, Zagreb 10000, Croatia. gigi.petkovic@
Abstract
• Fetal alcohol syndrome (FAS) is a congenital syndrome caused by maternal alcohol consumption
during pregnancy and is entirely preventable by abstinence from alcohol drinking during this time.
Little is known about the prevalence of FAS and maternal alcohol consumption during pregnancy in
Western countries. We present the results of FAS/partial fetal alcohol syndrome (PFAS) prevalence
study and maternal characteristics in a sample of schoolchildren from a rural province of Croatia.
This study involved seven elementary schools with 1,110 enrolled children attending 1st to 4th
grade and their mothers. We used an active case ascertainment method with passive parental
consent and Clarified IOM criteria. The investigation protocol involved maternal data collection and
clinical examination of children. Out of 1,110 mothers, 917 (82.6%) answered the
questionnaire. Alcoholexposure during pregnancy was admitted by 11.5%, regular drinking by 4.0%
and binge drinking by 1.4% of questioned mothers. Clinical examination involved 824 (74.2%)
schoolchildren and disclosed 14 (1.7%) with clinical signs of FAS and 41 (5.0%) of PFAS. The
observed FAS prevalence, based on 74.2% participation rate, was 16.9, PFAS 49.7 and combined
prevalence was 66.7/1,000 examined schoolchildren. This is the first FAS prevalence study based
on active ascertainment among schoolchildren and pregnancy alcohol drinking analysis performed
in a rural community of Croatia and Europe. High prevalence of FAS/PFAS and
pregnancy alcohol consumption observed in this study revealed that FAS is serious health
problem in rural regions as well as a need to develop future studies and preventive measures for
pregnancy alcohol drinking and FASD.
9. Facial Characteristics
• Low nasal bridge
• Short nose
• Indistinct philtrum
(groove between nose
and upper lip)
• Small head circumference
• Small eye opening
• Small midface
• Thin upper lip.
10. Growth Deficiency Characteristics
• Small size for gestational
age or small stature in
relation to peers
• Small head or
facial/dental anomalies
• Heart defects or other
organ dysfunction
• Deformities of joints,
limbs, and fingers
• Vision or hearing
problems.
11. Central Nervous System
Characteristics
• Poor coordination
• Hyperactive behavior
• Learning disabilities
• Developmental disabilities
(for example, speech and
language delays)
• Mental retardation or low IQ
• Problems with daily living
• Poor reasoning and judgment
skills
• Sleep and sucking
disturbances in infancy.
12. DIAGNOSTIC CRITERIA
• The following criteria must be fully met for an FAS
diagnosis
• Growth deficiency — Prenatal or postnatal height or
weight (or both) at or below the 10th percentile
• FAS facial features — All three FAS facial features
present
• Central nervous system damage — Clinically significant
structural, neurological, or functional impairment
• Prenatal alcohol exposure — Confirmed or Unknown
prenatal alcohol exposure
13. PREVENTION
• The only certain way to prevent FAS is to
simply avoid drinking alcohol during
pregnancy
15. MEDICAL CARE
• Pediatrician
• Primary care provider
• Otolaryngologist
• Audiologist
• Immunologist
• Neurologist
• Mental health professionals
(child psychiatrist and
psychologist, school
psychologist, behavior
management specialist)
• Opthalmologist
• Plastic surgeon
• Endocrinologist
• Gastroenterologist
• Nutritionist
• Geneticist
• Speech-language
pathologist
• Occupational therapist
• Physical therapist
16. MEDICATIONS
• No medications have been approved specifically to
treat FASDs. But, several medications can help
improve some of the symptoms of FASDs.
• Stimulants
Hyperactivity, problems paying attention, and poor
impulse control, as well as other behavior issues.
• Antidepressants
sad mood, loss of interest, sleep problems, school
disruption, negativity, irritability, aggression, and
anti-social behaviors.
20. PARENT TRAINING
• Concentrate on your child's
strengths and talents
• Accept your child's
limitations
• Be consistent with
everything (discipline,
school, behaviors)
• Use concrete language and
examples
• Use stable routines that do
not change daily
21. • Keep it simple
• Be specific-say exactly what you mean
• Structure your child's world to provide a
foundation for daily living
• Use visual aides, music, and hands-on activities to
help your child learn
• Use positive reinforcement often (praise,
incentives)
• Supervise: friends, visits, routines
• Repeat, repeat, repeat