This document discusses gastrointestinal disorders in children, beginning with an overview of pediatric differences in GI anatomy and physiology. It then covers signs and symptoms of GI disorders in infants and children, assessment techniques, and common GI disorders including structural defects, disorders of motility, and inflammatory disorders. Specific structural defects discussed in depth include cleft lip and palate, esophageal atresia and tracheoesophageal fistula, with details on diagnosis, management, and nursing care for each.
This document discusses chronic kidney disease (CKD) in pediatrics. It defines CKD as kidney damage lasting at least 3 months as determined by structural abnormalities and/or a glomerular filtration rate below 60 mL/min/1.73m2. The stages of CKD are described based on GFR. Common causes in children include congenital abnormalities and glomerulonephritis. The pathogenesis involves hyperfiltration injury and other factors like proteinuria that accelerate kidney damage. Management aims to address complications through careful monitoring, nutrition, treatment of mineral bone disorders, and controlling blood pressure and electrolyte abnormalities.
Bleeding Per Rectum In Children By Prof. Sushmita N. Bhatnagar MBBS, M.S., M.Ch,M.PHIL(Hospital Management)
HEAD, PEDIATRIC SURGERY
B.J WADIA CHILDREN’S HOSPITAL, MUMBAI
CONSULTANT PEDIATRIC SURGEON
BOMBAY HOSPITAL
JOINT SECRETARY
ASSOCIATION OF MEDICAL CONSULTANTS
For info log on to www.healthlibrary.com.
1. Neonatal seizures are the most common manifestation of neurological dysfunction in newborns and can be caused by hypoxic-ischemic encephalopathy, brain malformations, infections, genetic or metabolic issues.
2. Diagnosis involves a medical history, lab tests of electrolytes and metabolites, imaging like cranial ultrasound, and EEG monitoring.
3. Treatment focuses on correcting any metabolic abnormalities and administering anticonvulsants like phenobarbital while monitoring for side effects. Duration of treatment depends on the underlying cause and resolution of symptoms.
1. Nephrotic syndrome is characterized by massive proteinuria, hypoalbuminemia, edema, and hyperlipidemia. The majority of cases in children are idiopathic or minimal change disease.
2. Secondary causes include systemic diseases, infections, medications, and genetic conditions.
3. Treatment involves corticosteroids as first line, with recurrence being common in minimal change disease. Kidney biopsy may be needed to identify secondary causes or steroid resistance.
Glomerulonephritis refers to kidney diseases that involve inflammation of the glomeruli. There are two main clinical manifestations - the nephritic syndrome characterized by hematuria, edema, hypertension, and reduced kidney function; and the nephrotic syndrome defined by heavy proteinuria, edema, low serum albumin, and hyperlipidemia. Causes include post-infectious glomerulonephritis, IgA disease, and Henoch-Schonlein purpura. Management involves treating symptoms, monitoring fluid balance and output, and administering steroids or immunosuppressants depending on the underlying disease. Complications can arise if the syndromes are not properly managed.
1) A 5-year-old child presented with fever, lethargy, and posturing for 3 days and was brought to the hospital in a comatose state.
2) The document discusses terminology related to altered states of consciousness, various etiologies of coma including infectious, structural, metabolic and toxic causes, and provides details on the pathophysiology of coma and consciousness.
3) It outlines the approach to managing a comatose patient, including rapid assessment, stabilization, detailed history and examination with focus on neurological assessment, appropriate investigations and treatment.
This document defines myocarditis as inflammation of the myocardium, outlines its main etiologies as viral and bacterial infections, and describes its pathogenesis. Signs and symptoms range from asymptomatic to cardiogenic shock. Diagnosis involves ECG, chest X-ray, echocardiogram, and endomyocardial biopsy. Treatment focuses on supportive care and conventional heart failure therapies. Prognosis depends on age, with higher mortality in newborns and potential recovery of function in children and adolescents.
This document discusses gastrointestinal bleeding in children. It notes that GI bleeding accounts for 10-20% of pediatric gastroenterology referrals and around 0.4% of PICU admissions are due to life-threatening GI bleeding. The presentation, classification, causes, diagnostic workup, and treatment of upper and lower GI bleeding in neonates, infants, and children are described in detail over multiple sections. Key points include distinguishing the source and severity of bleeding, identifying specific etiologies, and managing bleeding through supportive care, endoscopic procedures, medications, and surgery as needed.
This document discusses chronic kidney disease (CKD) in pediatrics. It defines CKD as kidney damage lasting at least 3 months as determined by structural abnormalities and/or a glomerular filtration rate below 60 mL/min/1.73m2. The stages of CKD are described based on GFR. Common causes in children include congenital abnormalities and glomerulonephritis. The pathogenesis involves hyperfiltration injury and other factors like proteinuria that accelerate kidney damage. Management aims to address complications through careful monitoring, nutrition, treatment of mineral bone disorders, and controlling blood pressure and electrolyte abnormalities.
Bleeding Per Rectum In Children By Prof. Sushmita N. Bhatnagar MBBS, M.S., M.Ch,M.PHIL(Hospital Management)
HEAD, PEDIATRIC SURGERY
B.J WADIA CHILDREN’S HOSPITAL, MUMBAI
CONSULTANT PEDIATRIC SURGEON
BOMBAY HOSPITAL
JOINT SECRETARY
ASSOCIATION OF MEDICAL CONSULTANTS
For info log on to www.healthlibrary.com.
1. Neonatal seizures are the most common manifestation of neurological dysfunction in newborns and can be caused by hypoxic-ischemic encephalopathy, brain malformations, infections, genetic or metabolic issues.
2. Diagnosis involves a medical history, lab tests of electrolytes and metabolites, imaging like cranial ultrasound, and EEG monitoring.
3. Treatment focuses on correcting any metabolic abnormalities and administering anticonvulsants like phenobarbital while monitoring for side effects. Duration of treatment depends on the underlying cause and resolution of symptoms.
1. Nephrotic syndrome is characterized by massive proteinuria, hypoalbuminemia, edema, and hyperlipidemia. The majority of cases in children are idiopathic or minimal change disease.
2. Secondary causes include systemic diseases, infections, medications, and genetic conditions.
3. Treatment involves corticosteroids as first line, with recurrence being common in minimal change disease. Kidney biopsy may be needed to identify secondary causes or steroid resistance.
Glomerulonephritis refers to kidney diseases that involve inflammation of the glomeruli. There are two main clinical manifestations - the nephritic syndrome characterized by hematuria, edema, hypertension, and reduced kidney function; and the nephrotic syndrome defined by heavy proteinuria, edema, low serum albumin, and hyperlipidemia. Causes include post-infectious glomerulonephritis, IgA disease, and Henoch-Schonlein purpura. Management involves treating symptoms, monitoring fluid balance and output, and administering steroids or immunosuppressants depending on the underlying disease. Complications can arise if the syndromes are not properly managed.
1) A 5-year-old child presented with fever, lethargy, and posturing for 3 days and was brought to the hospital in a comatose state.
2) The document discusses terminology related to altered states of consciousness, various etiologies of coma including infectious, structural, metabolic and toxic causes, and provides details on the pathophysiology of coma and consciousness.
3) It outlines the approach to managing a comatose patient, including rapid assessment, stabilization, detailed history and examination with focus on neurological assessment, appropriate investigations and treatment.
This document defines myocarditis as inflammation of the myocardium, outlines its main etiologies as viral and bacterial infections, and describes its pathogenesis. Signs and symptoms range from asymptomatic to cardiogenic shock. Diagnosis involves ECG, chest X-ray, echocardiogram, and endomyocardial biopsy. Treatment focuses on supportive care and conventional heart failure therapies. Prognosis depends on age, with higher mortality in newborns and potential recovery of function in children and adolescents.
This document discusses gastrointestinal bleeding in children. It notes that GI bleeding accounts for 10-20% of pediatric gastroenterology referrals and around 0.4% of PICU admissions are due to life-threatening GI bleeding. The presentation, classification, causes, diagnostic workup, and treatment of upper and lower GI bleeding in neonates, infants, and children are described in detail over multiple sections. Key points include distinguishing the source and severity of bleeding, identifying specific etiologies, and managing bleeding through supportive care, endoscopic procedures, medications, and surgery as needed.
This document discusses gastrointestinal bleeding in children. It defines different types of GI bleeding based on the location such as melena, hematochezia, and hematemesis. Common causes of upper and lower GI bleeding are listed for newborns, infants, children, and adolescents. Evaluation involves history, physical exam, lab tests, and imaging. Treatment depends on the severity and includes supportive care, medications, endoscopic procedures, and surgery. Portal hypertension is described as a cause of GI bleeding along with its pathophysiology, clinical manifestations, diagnosis, and management.
Basics in Dehydration & it's management in paediatric practice. Prepared by Dr. Viduranga Edirisinghe on request by Prof. Wasantha Karunasekara. [2013 Aug]
This document discusses cleft lip and cleft palate. It begins by defining cleft lip and cleft palate as facial malformations that occur during early fetal development due to failed fusion of tissues. It notes the incidence of each condition and potential risk factors like medications and environmental exposures. The document outlines the types of cleft lip and palate and describes the multi-stage surgical and non-surgical treatments required from infancy through adolescence to correct the conditions.
This document discusses acute diarrhea and fluid therapy. It covers the pathophysiology of osmotic and secretory diarrhea. It also discusses the causes, clinical assessment, types, and management of dehydration caused by acute diarrhea. The management of dehydration involves calculating fluid deficits and administering intravenous or oral rehydration solutions to restore fluid and electrolyte losses. Complications of dehydration include shock, acute renal failure, electrolyte disturbances, and seizures.
This document provides information on the approach and assessment of acyanotic congenital heart diseases in children. It discusses:
1. The typical presenting complaints which include feeding difficulties, respiratory distress, easy fatigability, recurrent infections, and failure to thrive.
2. The physical exam findings to assess including inspection, palpation of pulses, blood pressure, jugular venous pressure, auscultation of heart sounds and murmurs.
3. The classification of acyanotic heart diseases which include left-to-right shunts and outflow obstructions. The most common types are also listed.
This document discusses heart failure in children. It defines heart failure as the heart's inability to pump enough blood to meet the body's needs. The key factors that affect cardiac performance are preload, afterload, and contractility. In children, common causes of heart failure include congenital heart defects, cardiomyopathy, and acquired conditions like myocarditis. Symptoms depend on whether left-sided or right-sided heart failure predominates. Treatment focuses on correcting underlying causes, diet modification, diuretics to reduce preload, digitalis to improve contractility, and dilators to reduce afterload. Imaging studies like echocardiograms are important for diagnosis.
Approach to child with generalized body swellingElhadi Hajow
Edema is characterized by swelling caused by excess fluid in the interstitial tissue. It can be localized or generalized. Common causes include cardiac, renal, or hepatic disease which decrease plasma oncotic pressure allowing fluid shift from vessels into tissue. A thorough history, physical exam, and lab tests are needed to determine the underlying cause and guide treatment such as diuretics, dietary changes, or treating the primary disease.
Indian childhood cirrhosis is a chronic liver disease seen in children in the Indian subcontinent characterized by copper deposition in the liver leading to cirrhosis. It commonly affects children ages 1-3 years old and is more prevalent in males. The disease is caused by genetic and nutritional factors such as vegetarianism. Clinically, patients experience jaundice, abdominal distension, and poor growth. The disease progresses from hepatomegaly to ascites, splenomegaly, and hepatic failure. Diagnosis involves liver function tests and biopsy. Treatment focuses on a copper-chelating drug along with managing symptoms and infections through diet and antibiotics.
Infective endocarditis is inflammation of the heart valves caused by bacterial infection. It is often a complication of congenital or rheumatic heart disease. Common causative organisms include streptococci and staphylococci. Risk factors include prior heart disease, dental/medical procedures, and intravenous drug use. Symptoms include fever, chills, weight loss and heart murmurs. Echocardiography and blood cultures help diagnose. Treatment involves antibiotics for 4-6 weeks. Surgery may be needed for severe valve damage or persistent infection. Prognosis remains serious despite treatment, with 20-25% mortality and high morbidity rates.
This document provides an approach to evaluating a patient presenting with jaundice. It defines jaundice and describes the production and metabolism of bilirubin. The causes of jaundice are divided into unconjugated and conjugated hyperbilirubinemia. A clinical history and physical exam are outlined to determine the underlying etiology. Key laboratory investigations including liver function tests and imaging studies are also reviewed to diagnose the cause of jaundice.
This document provides an overview of acute lymphoblastic leukemia (ALL) in children, including its definition, classification, epidemiology, pathophysiology, diagnosis, and treatment. It discusses how ALL results from mutations that cause immature white blood cells to crowd out the bone marrow. The diagnosis involves blood tests and bone marrow biopsies showing an excess of lymphoblasts. Treatment typically involves chemotherapy in multiple phases over 2 years, including induction, consolidation, and maintenance. Newer targeted drugs and stem cell transplants are used in high-risk cases. Prognostic factors like age, white blood cell count, and specific genetic abnormalities determine treatment approach and predicted outcomes.
Acute kidney injury and chronic kidney disease in childrenSameekshya Pradhan
Acute kidney injury (AKI) and chronic kidney disease can affect children. AKI is defined as a rapid deterioration of renal function over hours to days. It has various etiologies including pre-renal, renal, and post-renal causes. Management involves treating life-threatening complications, maintaining fluid/electrolyte balance, supportive care, and dialysis. Chronic kidney disease develops over months to years and requires long-term management to delay progression.
Dr. Eke Eghosasere Paul gave a presentation on pediatric heart failure to the Nelson Club on September 15, 2014. The presentation covered the epidemiology, etiology, pathophysiology, clinical signs and symptoms, diagnosis, treatment and prognosis of heart failure in children. Heart failure occurs when the heart cannot meet the body's metabolic needs due to reduced cardiac output. Compensatory mechanisms initially help maintain function but eventually become ineffective, leading to worsening clinical symptoms. Proper diagnosis and management of the underlying cause are important for treatment.
Approach to neonatal jaundice - Simplified
references : Cloherty And Stark's Manual Of Neonatal Care
AIIMS Protocol In Neonatology
Care Of The Newborn – Meherban Singh
This document discusses the approach to hematuria in children. It begins by defining hematuria and describing different types. It then outlines the most common etiologies of glomerular and non-glomerular hematuria. The document emphasizes taking a thorough history and physical exam. It recommends investigations including urine analysis, culture and microscopy, blood tests, imaging and potentially renal biopsy. Based on the cause, management may include reassurance, antibiotics, supportive care, monitoring, correcting complications, surgery or dialysis. The document provides a helpful algorithm for evaluating and managing hematuria in children.
This document outlines a presidential action plan for infectious endocarditis in children. It begins with definitions of infective endocarditis and discusses the epidemiology, pathogenesis, clinical features, diagnosis, treatment and prevention. Key points include that infective endocarditis is less common in children than adults but is increasing in those with cardiac surgery or conditions. Common causes are streptococcal and staphylococcal species. Clinical features may include fever, heart murmur and embolic phenomena. Echocardiography is important for diagnosis but blood cultures are also needed under the modified Duke criteria. Surgery may be indicated for complications such as heart failure or abscesses.
The document discusses the importance of the gastrointestinal tract and its functions of digestion and absorption. It provides details on the digestion of carbohydrates, proteins, lipids, and vitamins/minerals in the GI tract. Key enzymes and their sites of action are identified. Malabsorption syndromes are then examined, including causes, classification, epidemiology, clinical presentation, and relevant laboratory studies. Overall, the document emphasizes the critical role of proper GI function for overall health and nutrition.
Mcq in neonatology for medical studentsVarsha Shah
This document contains a multiple choice quiz on neonatal topics for medical students. It includes 6 questions related to newborn examinations, skull anatomy, skin findings, risk factors for developmental dysplasia of the hip, normal birth physiology, and the Apgar score. For each question, the correct answer is identified and feedback is provided to explain the rationale. The feedback often notes limitations or caveats to the incorrect answer choices.
This document provides an overview on approaching and managing a child with jaundice. It begins by defining jaundice as a visible manifestation of increased bilirubin levels. It then discusses the burden of jaundice in newborns, describing how most will experience some jaundice in the first week due to immature bilirubin metabolism. The document outlines how to classify jaundice as physiological or pathological based on clinical signs and bilirubin levels. For pathological jaundice, the main treatment approaches of phototherapy and exchange transfusion are described. The document provides guidance on evaluating the potential causes of jaundice and managing cases based on whether the hyperbilirubinemia is conjugated or
The document discusses coagulation disorders in neonates. It notes that the hemostatic mechanism differs in neonates, with decreased clotting factor activity and platelet function. Causes of bleeding include coagulation defects, platelet defects, fibrinolytic dysfunction, vascular causes, and miscellaneous issues. Evaluation involves history, physical exam, screening tests, and specific coagulation factor tests as needed. Treatment depends on the underlying cause, and may include vitamin K, fresh frozen plasma, whole blood transfusion, or specific clotting factor concentrates. Common disorders discussed include vitamin K deficiency, hemophilia A/B, von Willebrand disease, and disseminated intravascular coagulation.
This document discusses cleft lip and cleft palate, including their anatomy, embryology, definitions, classifications, causes, clinical manifestations, diagnosis, and management. It notes that cleft lip and palate are caused by failures in fusion during facial development between 4-12 weeks of gestation. Signs include openings in the lip and roof of the mouth. Treatment requires a multidisciplinary team and includes surgery to repair the clefts, as well as feeding adaptations, speech therapy, dental care, and psychological support for the child and family.
This document discusses gastrointestinal bleeding in children. It defines different types of GI bleeding based on the location such as melena, hematochezia, and hematemesis. Common causes of upper and lower GI bleeding are listed for newborns, infants, children, and adolescents. Evaluation involves history, physical exam, lab tests, and imaging. Treatment depends on the severity and includes supportive care, medications, endoscopic procedures, and surgery. Portal hypertension is described as a cause of GI bleeding along with its pathophysiology, clinical manifestations, diagnosis, and management.
Basics in Dehydration & it's management in paediatric practice. Prepared by Dr. Viduranga Edirisinghe on request by Prof. Wasantha Karunasekara. [2013 Aug]
This document discusses cleft lip and cleft palate. It begins by defining cleft lip and cleft palate as facial malformations that occur during early fetal development due to failed fusion of tissues. It notes the incidence of each condition and potential risk factors like medications and environmental exposures. The document outlines the types of cleft lip and palate and describes the multi-stage surgical and non-surgical treatments required from infancy through adolescence to correct the conditions.
This document discusses acute diarrhea and fluid therapy. It covers the pathophysiology of osmotic and secretory diarrhea. It also discusses the causes, clinical assessment, types, and management of dehydration caused by acute diarrhea. The management of dehydration involves calculating fluid deficits and administering intravenous or oral rehydration solutions to restore fluid and electrolyte losses. Complications of dehydration include shock, acute renal failure, electrolyte disturbances, and seizures.
This document provides information on the approach and assessment of acyanotic congenital heart diseases in children. It discusses:
1. The typical presenting complaints which include feeding difficulties, respiratory distress, easy fatigability, recurrent infections, and failure to thrive.
2. The physical exam findings to assess including inspection, palpation of pulses, blood pressure, jugular venous pressure, auscultation of heart sounds and murmurs.
3. The classification of acyanotic heart diseases which include left-to-right shunts and outflow obstructions. The most common types are also listed.
This document discusses heart failure in children. It defines heart failure as the heart's inability to pump enough blood to meet the body's needs. The key factors that affect cardiac performance are preload, afterload, and contractility. In children, common causes of heart failure include congenital heart defects, cardiomyopathy, and acquired conditions like myocarditis. Symptoms depend on whether left-sided or right-sided heart failure predominates. Treatment focuses on correcting underlying causes, diet modification, diuretics to reduce preload, digitalis to improve contractility, and dilators to reduce afterload. Imaging studies like echocardiograms are important for diagnosis.
Approach to child with generalized body swellingElhadi Hajow
Edema is characterized by swelling caused by excess fluid in the interstitial tissue. It can be localized or generalized. Common causes include cardiac, renal, or hepatic disease which decrease plasma oncotic pressure allowing fluid shift from vessels into tissue. A thorough history, physical exam, and lab tests are needed to determine the underlying cause and guide treatment such as diuretics, dietary changes, or treating the primary disease.
Indian childhood cirrhosis is a chronic liver disease seen in children in the Indian subcontinent characterized by copper deposition in the liver leading to cirrhosis. It commonly affects children ages 1-3 years old and is more prevalent in males. The disease is caused by genetic and nutritional factors such as vegetarianism. Clinically, patients experience jaundice, abdominal distension, and poor growth. The disease progresses from hepatomegaly to ascites, splenomegaly, and hepatic failure. Diagnosis involves liver function tests and biopsy. Treatment focuses on a copper-chelating drug along with managing symptoms and infections through diet and antibiotics.
Infective endocarditis is inflammation of the heart valves caused by bacterial infection. It is often a complication of congenital or rheumatic heart disease. Common causative organisms include streptococci and staphylococci. Risk factors include prior heart disease, dental/medical procedures, and intravenous drug use. Symptoms include fever, chills, weight loss and heart murmurs. Echocardiography and blood cultures help diagnose. Treatment involves antibiotics for 4-6 weeks. Surgery may be needed for severe valve damage or persistent infection. Prognosis remains serious despite treatment, with 20-25% mortality and high morbidity rates.
This document provides an approach to evaluating a patient presenting with jaundice. It defines jaundice and describes the production and metabolism of bilirubin. The causes of jaundice are divided into unconjugated and conjugated hyperbilirubinemia. A clinical history and physical exam are outlined to determine the underlying etiology. Key laboratory investigations including liver function tests and imaging studies are also reviewed to diagnose the cause of jaundice.
This document provides an overview of acute lymphoblastic leukemia (ALL) in children, including its definition, classification, epidemiology, pathophysiology, diagnosis, and treatment. It discusses how ALL results from mutations that cause immature white blood cells to crowd out the bone marrow. The diagnosis involves blood tests and bone marrow biopsies showing an excess of lymphoblasts. Treatment typically involves chemotherapy in multiple phases over 2 years, including induction, consolidation, and maintenance. Newer targeted drugs and stem cell transplants are used in high-risk cases. Prognostic factors like age, white blood cell count, and specific genetic abnormalities determine treatment approach and predicted outcomes.
Acute kidney injury and chronic kidney disease in childrenSameekshya Pradhan
Acute kidney injury (AKI) and chronic kidney disease can affect children. AKI is defined as a rapid deterioration of renal function over hours to days. It has various etiologies including pre-renal, renal, and post-renal causes. Management involves treating life-threatening complications, maintaining fluid/electrolyte balance, supportive care, and dialysis. Chronic kidney disease develops over months to years and requires long-term management to delay progression.
Dr. Eke Eghosasere Paul gave a presentation on pediatric heart failure to the Nelson Club on September 15, 2014. The presentation covered the epidemiology, etiology, pathophysiology, clinical signs and symptoms, diagnosis, treatment and prognosis of heart failure in children. Heart failure occurs when the heart cannot meet the body's metabolic needs due to reduced cardiac output. Compensatory mechanisms initially help maintain function but eventually become ineffective, leading to worsening clinical symptoms. Proper diagnosis and management of the underlying cause are important for treatment.
Approach to neonatal jaundice - Simplified
references : Cloherty And Stark's Manual Of Neonatal Care
AIIMS Protocol In Neonatology
Care Of The Newborn – Meherban Singh
This document discusses the approach to hematuria in children. It begins by defining hematuria and describing different types. It then outlines the most common etiologies of glomerular and non-glomerular hematuria. The document emphasizes taking a thorough history and physical exam. It recommends investigations including urine analysis, culture and microscopy, blood tests, imaging and potentially renal biopsy. Based on the cause, management may include reassurance, antibiotics, supportive care, monitoring, correcting complications, surgery or dialysis. The document provides a helpful algorithm for evaluating and managing hematuria in children.
This document outlines a presidential action plan for infectious endocarditis in children. It begins with definitions of infective endocarditis and discusses the epidemiology, pathogenesis, clinical features, diagnosis, treatment and prevention. Key points include that infective endocarditis is less common in children than adults but is increasing in those with cardiac surgery or conditions. Common causes are streptococcal and staphylococcal species. Clinical features may include fever, heart murmur and embolic phenomena. Echocardiography is important for diagnosis but blood cultures are also needed under the modified Duke criteria. Surgery may be indicated for complications such as heart failure or abscesses.
The document discusses the importance of the gastrointestinal tract and its functions of digestion and absorption. It provides details on the digestion of carbohydrates, proteins, lipids, and vitamins/minerals in the GI tract. Key enzymes and their sites of action are identified. Malabsorption syndromes are then examined, including causes, classification, epidemiology, clinical presentation, and relevant laboratory studies. Overall, the document emphasizes the critical role of proper GI function for overall health and nutrition.
Mcq in neonatology for medical studentsVarsha Shah
This document contains a multiple choice quiz on neonatal topics for medical students. It includes 6 questions related to newborn examinations, skull anatomy, skin findings, risk factors for developmental dysplasia of the hip, normal birth physiology, and the Apgar score. For each question, the correct answer is identified and feedback is provided to explain the rationale. The feedback often notes limitations or caveats to the incorrect answer choices.
This document provides an overview on approaching and managing a child with jaundice. It begins by defining jaundice as a visible manifestation of increased bilirubin levels. It then discusses the burden of jaundice in newborns, describing how most will experience some jaundice in the first week due to immature bilirubin metabolism. The document outlines how to classify jaundice as physiological or pathological based on clinical signs and bilirubin levels. For pathological jaundice, the main treatment approaches of phototherapy and exchange transfusion are described. The document provides guidance on evaluating the potential causes of jaundice and managing cases based on whether the hyperbilirubinemia is conjugated or
The document discusses coagulation disorders in neonates. It notes that the hemostatic mechanism differs in neonates, with decreased clotting factor activity and platelet function. Causes of bleeding include coagulation defects, platelet defects, fibrinolytic dysfunction, vascular causes, and miscellaneous issues. Evaluation involves history, physical exam, screening tests, and specific coagulation factor tests as needed. Treatment depends on the underlying cause, and may include vitamin K, fresh frozen plasma, whole blood transfusion, or specific clotting factor concentrates. Common disorders discussed include vitamin K deficiency, hemophilia A/B, von Willebrand disease, and disseminated intravascular coagulation.
This document discusses cleft lip and cleft palate, including their anatomy, embryology, definitions, classifications, causes, clinical manifestations, diagnosis, and management. It notes that cleft lip and palate are caused by failures in fusion during facial development between 4-12 weeks of gestation. Signs include openings in the lip and roof of the mouth. Treatment requires a multidisciplinary team and includes surgery to repair the clefts, as well as feeding adaptations, speech therapy, dental care, and psychological support for the child and family.
Cleft lip and cleft palate are birth defects caused by incomplete fusion of the structures that form the lip and roof of the mouth. They can range from a cleft in only the lip or palate to a complete bilateral cleft. Management involves assessment, feeding assistance, prevention of infection, and surgical repair of the cleft. Nurses play an important role in caring for children with clefts through managing feeding, monitoring airway and nutritional status, providing postoperative care and counseling parents.
Cleft palate is a common congenital craniofacial anomaly that requires a multidisciplinary team for treatment. It can involve the lip, palate, or both and has both genetic and environmental causes. Diagnosis is often made prenatally through ultrasound. Treatment involves a coordinated approach including surgery to repair the cleft, orthodontics, speech therapy, and other interventions throughout development. Successful treatment requires technical skill and knowledge of the abnormal anatomy to achieve functional and aesthetic outcomes.
The gastrointestinal tract develops from the endoderm during the 3rd week of gestation. By the 4th week, the foregut, midgut and hindgut regions form and contribute to different parts of the GI system. Notable milestones in GI development include formation of the stomach, esophagus, liver and pancreas by 5-7 weeks and intestinal growth and peristalsis by 8-23 weeks. Tracheo-esophageal fistula and esophageal atresia are congenital disorders where the esophagus fails to develop properly, often requiring surgical correction. Types I-V describe variations in connections between the esophagus and trachea.
Omphalocele/Exomphalos, cleft palate & Oesophageal atresia and Nursing careStephanopoulos Osei
This document discusses several common gastrointestinal defects in children, including cleft lip and palate, exomphalos, and esophageal atresia.
It describes Baby Kunle, a 3-day old with a cleft lip, and provides details on the causes, incidence, pathophysiology, clinical presentation, diagnosis, and treatment of cleft lip and palate. Surgical repair is usually done in the first few months of life. Nursing care involves careful feeding and psychosocial support.
Exomphalos, or omphalocele, is defined as herniation of abdominal contents through the umbilical ring. It has a low incidence. Surgical repair aims to replace organs into the abdomen
The gastrointestinal tract develops from the endoderm during the third week of gestation. By the fourth week, the foregut, midgut, and hindgut have formed and will contribute to different parts of the GI system. Notable milestones in GI development include the formation of the stomach, esophagus, liver and pancreas by 5-7 weeks and intestinal lengthening by 8-12 weeks. Tracheo-esophageal fistula and esophageal atresia are birth defects where the esophagus fails to form properly, sometimes connecting abnormally to the trachea. They require surgical correction, often in stages, to allow for feeding and prevent aspiration. Close nursing observation and care is needed
This document discusses cleft lip and cleft palate, including their causes, signs and symptoms, diagnosis, and management. Cleft lip occurs when tissues of the upper lip don't fuse properly, resulting in an opening. Cleft palate occurs when tissues of the roof of the mouth don't fuse, resulting in openings. Risk factors include family history and exposure to substances like smoking during pregnancy. Treatment may involve surgery to repair the lip and palate, as well as follow up care like speech therapy. Complications can include feeding and ear problems if not treated properly.
Cleft lip and palate is a birth defect caused by failure of fusion of the facial prominences during embryonic development between 4-12 weeks of gestation. It can involve the lip, alveolus, hard and soft palate. The document discusses the anatomy, embryology, classification, clinical manifestations, diagnostic evaluation, management including surgical techniques, and nursing care of infants with cleft lip and palate. Key aspects of nursing management include addressing feeding difficulties, preparing parents for surgery and their infant's needs, and educating on postoperative care to support healing.
Cleft lip and palate beliefs vary globally and are often tied to cultural and religious beliefs about causation. In many areas, clefts are believed to be acts of God, punishment for ancestral sins, or caused by spirits or witchcraft. These beliefs can influence treatment decisions and cause feelings of shame. Understanding local beliefs is important for providing culturally-sensitive care.
This document discusses various classifications and treatments for cleft lip and palate. It describes Davis & Ritchie's 1922 classification system based on the position of the cleft. It also outlines Veau's 1931 and Kernahan's classifications. The document discusses feeding techniques for different cleft types and the role of the pediatric dentist. It provides details on the multi-disciplinary treatment sequence from birth to adulthood.
1. Cleft lip and palate is a common congenital deformity resulting from incomplete fusion of tissues in the face and palate during early embryonic development between 4-8 weeks.
2. Risk factors for cleft lip and palate include family history, certain ethnicities being more susceptible, male sex, environmental exposures like smoking and medications during pregnancy, and advanced parental age.
3. Surgical repair of cleft lip is usually done by 6 months of age, with cleft palate repair by 12 months. Multiple surgeries may be needed. Nursing care focuses on adequate nutrition, positioning, and reducing family anxiety regarding the condition
Cleft lip and cleft palate are congenital defects caused by incomplete fusion of facial structures in utero. Cleft lip involves a split in the upper lip, which can be unilateral or bilateral. Cleft palate is a split in the roof of the mouth. Treatment involves surgery to repair the cleft, with lip repair typically in the first year of life and palate repair by 18 months. Nursing management focuses on pre-operative preparation, careful feeding and positioning, and post-operative monitoring of healing and parental education on home care.
Cleft lip and palate are congenital anomalies resulting from failure of facial structures to fuse during development in utero. A cleft lip involves an opening in the upper lip that may extend into the nose, while a cleft palate occurs when the roof of the mouth contains an opening into the nose. Risk factors include family history and exposure to certain substances during pregnancy. Treatment may involve surgery to repair the cleft, with the timing varying based on the specific type of cleft. Postoperative care focuses on preventing infection, maintaining breathing and tissue integrity, and educating parents on feeding techniques and long term management.
This document discusses common pediatric surgical conditions, including gastrointestinal congenital anomalies present at birth. It identifies anomalies of the mouth (cleft lip and palate), esophagus (esophageal atresia and tracheoesophageal fistula), stomach/duodenum (pyloric stenosis), intestines (imperforate anus, omphalocele, gastroschisis, Hirschsprung's disease, intussusception), and provides descriptions, epidemiology, presentations, diagnoses, and management for each. It also outlines pre and post-operative nursing care for infants with surgical congenital anomalies, focusing on risks like infection, breathing issues, tissue trauma, nutrition and pain management.
This document discusses preventive pedodontics and infant oral health care. It covers levels of prevention including primary, secondary, and tertiary prevention. It defines infant oral health care and discusses the goals of infant oral health programs which include educating parents on risks of dental disease and establishing dental services as part of infant healthcare. The document provides guidance on prenatal counseling, perinatal oral health, colonization of the infant oral cavity, and anticipatory guidance for different age ranges from 6-12 months to 2-6 years.
CLEFT LIP AND CLEFT PALATE.pptx in childrenbeema2434
This document discusses cleft lip and cleft palate, including their definitions, embryology, etiology, classification, clinical features, diagnosis, complications, management, and nursing management. Cleft lip is a gap in the lip or split extending into the nostril due to failed fusion of maxillary and medial nasal processes during development. Cleft palate occurs when the two skull plates forming the hard palate do not completely join due to failed fusion of lateral palatine, nasal septum, and medial palatine processes. Management involves surgical repair of the cleft, as well as modified feeding, dental appliances, speech and hearing treatments, with the goal of addressing complications like feeding and breathing difficulties.
This document provides information about cleft lip and cleft palate including causes, risk factors, diagnosis, treatment, and social aspects. It describes how cleft lip occurs when the tissues of the lip do not fully fuse before birth, and cleft palate occurs when the roof of the mouth does not fully close. Treatment often begins in infancy and may include surgery, dental care, speech therapy, and psychological support. The document also discusses cultural beliefs and stigma around cleft conditions as well as organizations providing cleft care in India.
Similar to Tracheo Esophahgeal Fistula,Cleft lip and Palate (20)
This document defines meningitis and provides information on the incidence, causes, symptoms, diagnosis, and treatment of the disease. It states that meningitis is an inflammation of the protective membranes covering the brain and spinal cord, and can be caused by viral or bacterial infections. The incidence is between 3-5 per 100,000 people in the US, with over 2,000 deaths annually. Symptoms, signs, laboratory tests, and imaging are used to diagnose meningitis. Initial treatment involves empiric use of bactericidal antibiotics such as third generation cephalosporins. Adjunctive treatments including corticosteroids may help reduce complications in some cases.
Meconium aspiration syndrome (MAS) occurs when a baby inhales or aspirates meconium during delivery. Meconium is the dark green intestinal contents produced by fetuses before birth. Factors like post-term delivery or fetal distress can cause a fetus to pass meconium into the amniotic fluid before birth. If this thick, meconium-stained fluid is then inhaled or aspirated during delivery, it can block the baby's airways and cause MAS. Treatment involves immediate suctioning and clearing of the airways after birth, as well as oxygen therapy and antibiotics if needed to treat respiratory distress and prevent infection. More severe cases may require ventilation support or other advanced treatments like
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The document discusses the benefits of exercise for mental health. Regular physical activity can help reduce anxiety and depression and improve mood and cognitive function. Exercise causes chemical changes in the brain that may help protect against mental illness and improve symptoms for those who already suffer from conditions like anxiety and depression.
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This document discusses the growth and development of adolescence across multiple domains. It begins by defining adolescence as the transition period between childhood and adulthood, characterized by rapid physical, cognitive, social, and emotional changes. It then covers the biological changes of puberty, psychosocial development, cognitive development, and the importance of relationships with peers and parents during this stage. The document also addresses nutrition, exercise, sleep, immunization, and other health needs during adolescence.
The document discusses the benefits of exercise for mental health. Regular physical activity can help reduce anxiety and depression and improve mood and cognitive function. Exercise causes chemical changes in the brain that may help protect against developing mental illness and improve symptoms for those who already suffer from conditions like anxiety and depression.
Fine motor skills allow for precise hand and finger movements and involve small muscle groups in the hands, wrists, fingers, and toes. Developing strong fine motor abilities helps with tasks like writing, drawing, buttoning clothes, and using utensils. Children's fine motor skills progress as their hands and fingers grow and develop greater strength, control, and coordination over time.
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This document provides information on growth and development in infants from birth to one year. It discusses patterns of physical development including weight, height, proportions and soft spots in the head. It also covers visual, auditory, motor and language development. Feeding methods such as breastfeeding and bottle feeding are described. The document also discusses gentle handling, bathing, dressing and diapering infants.
Hirschsprung's disease is a disorder of the gut caused by the congenital absence of ganglion cells in the submucosal and myentric plexus of the intestine, also known as megacolon or congenital aganglionic megacolon. It is caused by the congenital absence of autonomic parasympathetic ganglion cells. Surgical management involves removing the aganglionic bowel followed by anastomosis of the remaining portion, done in two stages - a temporary colostomy followed later by a pull through procedure to excise the aganglionic segment and connect the bowel.
The cold chain is a system used to store and transport vaccines at the proper temperatures from manufacturers to recipients. It includes cold chain equipment like walk-in cold rooms, deep freezers, ice-lined refrigerators, cold boxes, vaccine carriers, and day carriers. Proper cold chain storage and transportation is crucial because some vaccines like polio and measles must be kept at very cold temperatures to maintain their efficacy. Nurses play an important role in maintaining the cold chain by arranging and transporting vaccines safely using the appropriate equipment and temperatures.
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More from Prof.Dr.Shali.B.S.Mamata College of Nursing,Khammam,Telangana. (20)
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In addition to infrastructure and capacity constraints, CAR-Ts face a very different risk-benefit dynamic in autoimmune compared to oncology, highlighting the need for tolerable therapies with low adverse event risk. CAR-NK and Treg-based therapies are also being developed in certain autoimmune disorders and may demonstrate favorable safety profiles. Several novel non-cell therapies such as bispecific antibodies, nanobodies, and RNAi drugs, may also offer future alternative competitive solutions with variable value propositions.
Widespread adoption of cell therapies will not only require strong efficacy and safety data, but also adapted pricing and access strategies. At oncology-based price points, CAR-Ts are unlikely to achieve broad market access in autoimmune disorders, with eligible patient populations that are potentially orders of magnitude greater than the number of currently addressable cancer patients. Developers have made strides towards reducing cell therapy COGS while improving manufacturing efficiency, but payors will inevitably restrict access until more sustainable pricing is achieved.
Despite these headwinds, industry leaders and investors remain confident that cell therapies are poised to address significant unmet need in patients suffering from autoimmune disorders. However, the extent of this impact on the treatment landscape remains to be seen, as the industry rapidly approaches an inflection point.
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Osteoporosis - Definition , Evaluation and Management .pdfJim Jacob Roy
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Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Local Advanced Lung Cancer: Artificial Intelligence, Synergetics, Complex Sys...Oleg Kshivets
Overall life span (LS) was 1671.7±1721.6 days and cumulative 5YS reached 62.4%, 10 years – 50.4%, 20 years – 44.6%. 94 LCP lived more than 5 years without cancer (LS=2958.6±1723.6 days), 22 – more than 10 years (LS=5571±1841.8 days). 67 LCP died because of LC (LS=471.9±344 days). AT significantly improved 5YS (68% vs. 53.7%) (P=0.028 by log-rank test). Cox modeling displayed that 5YS of LCP significantly depended on: N0-N12, T3-4, blood cell circuit, cell ratio factors (ratio between cancer cells-CC and blood cells subpopulations), LC cell dynamics, recalcification time, heparin tolerance, prothrombin index, protein, AT, procedure type (P=0.000-0.031). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and N0-12 (rank=1), thrombocytes/CC (rank=2), segmented neutrophils/CC (3), eosinophils/CC (4), erythrocytes/CC (5), healthy cells/CC (6), lymphocytes/CC (7), stick neutrophils/CC (8), leucocytes/CC (9), monocytes/CC (10). Correct prediction of 5YS was 100% by neural networks computing (error=0.000; area under ROC curve=1.0).
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Identification and nursing management of congenital malformations .pptx
Tracheo Esophahgeal Fistula,Cleft lip and Palate
1. COURSE:B.Sc N III YEAR
SUBJECT:CHILD HEALTH NURSING
GUNIT.V
GASTRO INTESTINAL DISORDERS
TOPICS: 1.CLEFT LIP AND PALATE
2.TRACHEOESOPHAGEAL FISTULA
Mrs.Shali
Associate professor
MCON,Khammam
4. Pediatric Differences in Anatomy
and Physiology
• Duodenum: digestion takes place
• Enzymes that aid in digestion:
– Amylase ( saliva; digests carbs)
– Lipase (enhances fat absorption)
– Trypsin (which breaks down protein into
polypeptides and some amino acids)
(Infants are deficient in these enzymes until
around 4-6 mos…therefore abdominal
distention from gas is common)
5. Pediatric Differences in Anatomy and
Physiology
• GI system is immature at birth
– Process of absorption and secretion do not
take place until after birth
– Sucking primitive reflex
– Voluntary swallow (at 6 weeks)
– Newborn’s stomach capacity is small at
birth Implications..
• (frequent feedings, freq bowel movements, and
intestinal motility is greater than in older kids
(peristalsis) therefore greater emptying time)
6. Pediatric Differences in Anatomy and
Physiology
• Liver function immature at birth and next few
weeks
• During first year of life
– Gluconeogenesis (formation of glycogen from
noncarbs)
– Plasma protein
– Ketone formation
– Vitamin storage
– Deamination
• GI structures in second year of life more mature
– Enlarged stomach capacity (to 3m m/day)
– Sphincter control (mylination of sc)
7. Signs/ symptoms of GI disorders in
infants/ children
• Vomiting/ regurgitation
• Irritability/ fussiness/Excessive crying
• Abdominal pain –Acute/severe pain, Recurrent colicky
pain, Recurrent or persistent dull aching pain/
distension/exaggerated or diminished bowel sound.
• Localized or generalized rigidity, feeding difficulty
• Weight loss
• Stool changes
• Abdominal pain
8. GI Assessment Techniques
• Subjective
– Lifestyle and family factors
• Including family hx
– Diet
• Gaining weight
• Thorough h/o feeding pattern,
• Allergies (lactose intolerant, celiac disease)
– Elimination patterns
• In take /out put
• Encorpresis/ constipation
10. GI Assessment Techniques
• Objective (con’t)
– Auscultation
• Hyper/hypo bowel sounds
– Percussion
• Tympany vs dullness
– Palpation
• Light vs deep
• Rebound tenderness…peritoneal
inflammation
• McBurney’s point
11. Disorders of the GI System
• Structural defects
• Disorders of motility
• Intestinal parasitic disorders
• Inflammatory disorders
• Disorders of malabsorption
• Hepatic disorders
• Injuries to the GI system
14. INTRODUCTION
Cleft lip and cleft palate are facial malformation
that occurs during embryonic development
and are the most common congenital deformities
15.
16. CLEFT LIP
It results from failure of fusion of maxillary
process with nose elevation on frontal
prominence.The extent of defect varies from
notch in the lip(incomplete cleft) to a large
cleft reaching the floor of nose (complete
cleft)It occur unilateral and bilateral.
17. Cleft palate is the failure of fusion of the hard
palate with each other and with the soft palate.Cleft
lip usually occurs with cleft palate.It may be
complete(involving hard and soft palate, or
Incomplete(hole in the roof of the mouth,usually in
soft palate
Definition
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40. Pathophysiology
The cleft lip and palate defects result from failure of the
maxillary and premaxillary processes to fuse during the
5th to 8th week of intrauterine life.
The cleft may be a simple notch in the vermilion line, or
it may extend up into the floor of the nose.
The child born with a cleft palate but with an intact lip
does not have the external disfigurement that may be so
distressing to the new parent; however, the problems
are more serious.
41. CONTINU..
In an 8-week old embryo, there is still no roof to the
mouth; the tissues that are to become the palate are
two shelves running from the front to the back of the
mouth and projecting vertically downward on either
side of the tongue.
The shelves move from a vertical position to a
horizontal position; their free edges meet and fuse in
the midline.
42. Later, bone forms within this tissue to form
the hard palate.
Normally the palate is intact by the 10th week
of fetal life.
Exactly what happens to prevent this closure
is not known for sure, leading to a cleft lip
and cleft palate.
43. Cleft Lip and Cleft Palate
• Clinical Manifestations
• Difficulty with feedings. The newborn easily
becomes choked on liquids.
Difficulty swallowing. The newborn has a hard time in
swallowing, with potential for liquids or foods to come
out the nose.
Nasal speaking voice. Due to the split in the palate,
the newborn has a nasal speaking voice.
– Otologic,Dental and orthodontic Developmental
problems
44.
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47.
48. FEEDING PROBLEMS
Due to a separation in the lip or
opening in the palate,sucking is
ineffective and the food and liquids can
pass from the mouth back through the
nose,There may be aspiration of feeds.
RESPIRATORY PROBLEMS
Aspiration of feeds may result in
respiratory infections like aspiration
pneumonia.
49. Diagnosis
• Assessment and Diagnostic Findings
• Inspection: Cleft lip is made at birth.
• Assess child’s ability to suck and swallow
• Defect in the upper lip also called hare lip. may
be unilateral or bilateral.
• Special feeding techniques if surgery is
delayed
61. Management
• Feeding a Child before Cleft Lip Repair
• Bottle with special nipple – longer and narrower
• Hold infant in upright position
• Large cross-cut hole in nipple to allow the child to get
food into back of throat without strong sucking
• Stimulate sucking by rubbing nipple on infant’s
lower lip
• Allow child to swallow and burp frequently
• ESSR method – Enlarge nipple, Stimulate sucking,
Swallow, Rest
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72. Surgical Management
• Cleft Lip Repair
– Usually done early (first few days to 1stmonth
of life) to improve parental bonding and
improve feeding
– Plastic surgery with a staggered suture line
(often in shape of letter “Z” to minimize
scarring)
– After surgery Logan Bar over child’s mouth
to reduce tension on suture line
73. CLEFT LIP
It may require one or two surgeries
depending on the severity of defect.The
initial surgery is usually performed at the
age of 3 months.Common procedures
include
1.Tennison-Randall Triangular Flap (Z –
Plasty)
2. Millard’s Rotational Advancement
Technique.
74.
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77. CLEFT PALATE
It often requires multiple surgeries
over the course of 18 years.The first
surgical repair usually occurs when
the baby is between 6-12 months.
- The initial surgery creates a
functional palate,reduces the
chance of fluid entering the
middle ears and helps in proper
development of teethand facial
bones
78. -Children with a cleft palate may
need a bone graft when they are
about 8 years old to fill in the upper
gum line so that it can support
permanent teeth and stabilize upper
jaw.
-Once the permanent teeth
grows,Braces may be put to
straighten the teeth.
85. Nursing care
• Pre-Op Care of the Child and
Parents
• Explain pre-op procedures to
parents
• Provide support and information
• Keep accurate record of child’s growth
and feeding schedule
• Infant:
– NPO X 4-6 hours pre-op,IV fluid.
86. Nursing Management
– Pre-Op
– Maintain adequate nutrition
– Positioning. If the cleft lip is unilateral, the nipple
should be aimed at the unaffected side; the infant
should be kept in an upright position during feeding.
• Imbalanced Nutrition: Less than body
requirements
• Risk for aspiration
• Impaired parenting
87. Post –op care
• Post-Op Care of Child and Family
• Encourage rooming-in
• Incision care: clean sutures with sterile cotton
swab and ½ strength H202 followed by saline
to prevent crusting (esp. after feeding). May
apply antibiotic ointment to suture line
• DO NOT DISPLACE LOGAN BAR
• Special feeder – syringe with rubber tubing
into side of mouth, Breck feeder
• Diet advance from clear to diet for age over 48
hours
• Elbow restraints
89. Cleft Palate
• Defect in the roof of the mouth and oral cavity
communicates with the nasal cavity.
• Can be unilateral or bilateral. May involve whole
plate or can be confined to soft palate.
• Repaired surgically between 6 months to 2
years prior to talking
– Maintain adequate nutrition
– Positioning. If the cleft lip is unilateral, the
nipple should be aimed at the unaffected side;
the infant should be kept in an upright position
during feeding.
90. Nursing care
Reduce family anxiety. Give the family
information about cleft repairs; encourage them
to ask questions and reassure them that any
question is valid.
Provide family teaching. Explain the usual
routine of preoperative, intraoperative, and post
operative care; written information is helpful,
but be certain the parents understand the
information.
91. Post –op care
• sutures in child’s mouth
• Keep straws, pacifiers, spoons away from
child’s mouth for 7-10 days post-op
• Elbow restraints and mittens
• Feeding – soft foods: baby food. Short nipples
may be used
• All feeding followed by rinsing mouth with
water to clean suture line
• No brushing teeth X 1-2 weeks
92. Nursing Diagnosis
– Pre-Op:
•Parental Knowledge Deficit
•Risk for Infection
•High Risk for Altered Family
Processes
– Post-Op:
•Imaparied Skin Integrity
•Child: Pain
96. INTRODUCTION
Congenital Esophageal atresia(EA) and
Tracheo-Esophageal fistula (TEF) are rare
malformation that represent a failure of
esophagus to develop as a continues passage
and trachea and esophagus to separate in to
deferent structure.
Incidence:1 in 1000live birth
Higher male , low birth weight
97. Tracheoesophageal Fistula (TEF)
• Defenition:
• Esophageal atresia and TEF
– a malformation that results from failure of
the esophagus to develop as a continuous
tube
– Foregut fails to lengthen, separate and fuse
in to 2 parallel tubes (at 4-5 weeks
gestation)
– Associated with maternal
polyhydramnios
98. atresia and tracheoesophageal fistula, the
upper segment of the esophagus ends in a
blind pouch connected to the trachea; the
fistula connects the lower segment to the
trachea.
99. Etiology
Trisomy 13, 18, or 21
Other digestive tract problems (such as
diaphragmatic hernia, duodenal atresia, or
imperforate anus)
Heart problems (such as ventricular septal
defect, tetralogy of Fallot, or patent ductus
arteriosus)
Kidney and urinary tract problems Muscular or
skeletal problems
VACTERL syndrome (which involves Vertebral,
Anal, Cardiac, TE fistula, Renal, and Limb
abnormalities)
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101.
102. CLINICAL FEATURES
• Apparent in immediate newborn period
– Respiratory Distress
– Difficulty feeding
– Excessive drooling
– Choking, coughing
– Cyanosis
• Esophageal Atresia is a surgical
emergency!
• Tx: Surgical correction
103.
104. DIAGNOSIS
– Confirmed by attempting to pass an NG
Tube into stomach
• Usually a 5 or 8 French tube
• Management –Medical
• Surgical
• Nursing Care
– Identify signs/symptoms of disease
– Careful Physical Assessment
105.
106.
107. Nursing Management
• Pre- and Post- Operative Care
• Pre-Op:
– NG tube to suction
– Prevent aspiration complication
• (increase HOB, NPO)
– Establish IV access: IVFs & IV abx
• Post-Op:
– Care of G-Tube
– Family teaching