MANDIBLE:
GROWTH,
DEVELOPMENT &
ANATOMICAL
CONSIDERATIONS
By
DR. ANTARLEENA SENGUPTA
I YR PG,
DEPTT. OF PERIODONTOLOGY &
IMPLANTOLOGY,
MCODS MANGALORE
CONTENTS
 INTRODUCTION
 ANATOMY
 BODY OF MANDIBLE
 OUTER SURFACE
 INNER SURFACE
 RAMUS
 LATERAL SURFACE
 MEDIAL SURFACE
 CORONOID PROCESS
 CONDYLAR PROCESS
 ATTACHMENTS & RELATIONS
 MUSCLE ATTACHMENTS
 FORAMINA
 BLOOD SUPPLY
 NERVE SUPPLY
 LYMPHATICS
 STRUCTURES RELATED TO MANDIBLE
2
contd.
 GROWTH OF MANDIBLE
 PRENATAL EMBRYOLOGY
 MECKEL’S CARTILAGE
 ENDOCHONDRAL BONE
FORMATION
 CONDYLAR PROCESS
 CORONOID PROCESS
 MENTAL REGION
 POST-NATAL EMBRYOLOGY
 RAMUS
 BODY
 ANGLE
 LINGUAL TUBEROSITY
 ALVEOLAR PROCESS
 CHIN
 CONDYLAR PROCESS
 CORONOID PROCESS
 THEORIES OF GROWTH
 ENLOW’S EXPANDING V PRINCIPLE
 ENLOW’S COUNTERPART
PRINCIPLE
 AGE CHANGES IN MANDIBLE
 INFANTS & CHILDREN
 ADULTS
 OLD AGE
 ANATOMIC CONSIDERATIONS
 DEVELOPMENTAL ANOMALIES
 SURGICAL CONSIDERATIONS
 ANATOMIC SPACES
 CONCLUSION
 REFERENCES
3
INTRODUCTION
4
ANTERIOR VIEW LATERAL VIEW
ANTERIOR VIEW LATERAL VIEW
5
POSTERIOR VIEW
6
7
ANTERIOR VIEW
8
MENTAL PROTUBERANCE MENTAL TUBERCLES
ANTERIOR VIEW
 BODY
 Each half of the body has outer and inner surfaces, and
upper and lower borders.
 The outer surface presents the following features:
1. The symphysis menti
2. The mental protuberance
3. The mental foramen
4. The oblique line
9
OUTER SURFACE
Anterior border
LATERAL VIEW
10
11
FRONTAL VIEW
symphysis menti
INNER SURFACE has the following
features:
 Mylohyoid Line
 Submandibular fossa
 Sublingual fossa
 Superior and Inferior genial tubercles
 Mylohyoid groove
12
INNER SURFACE
13
MEDIAL VIEW
UPPER BORDER
The upper or alveolar border bears sockets for the
teeth.
14
LOWER BORDER
15
The lower border of the mandible is also called the base. Near
the midline the base shows an oval depression called the
digastric fossa.
 RAMUS
16
ANTERIOR VIEW LATERAL VIEW
• LATERAL SURFACE of ramus is flat and bears a
number of oblique ridges.
U
L
P
A
MEDIAL SURFACE of ramus has the following:
 Mandibular foramen
 Lingula
 Mylohyoid groove
 Mandibular notch
 Angle of mandible
17
Lingula
Submandibular fossaMylohyoid
groove
Mandibular notch
 Coronoid process
 Condyloid process
 Head
 Neck
 Pterygoid fovea
18
Superior view Superior view
Pterygoid fovea
Neck
RADIOGRAPHIC VIEW OF MANDIBLE
19
ATTACHMENTS & RELATIONS OF
MANDIBLE
 MUSCLE ATTACHMENTS
20
LATERAL VIEW
21
MEDIAL VIEW
FORAMINA & RELATIONS OF MANDIBLE
22
Mandibular
foramen
Mental foramen
BLOOD SUPPLY
 Central blood supply
 Peripheral blood supply
23
via the INFERIOR ALVEOLAR
ARTERY except the coronoid
process, which is supplied by
temporalis muscle vessels.
via the PERIOSTEAL
VESSELS, which run parallel to
cortical surface of bone, giving
off NUTRIENT VESSELS those
penetrate cortical bone and
anastomose with the branches
of inferior alveolar artery.
NERVE SUPPLY
 Derived from mandibular branch (V3) of trigeminal
nerve.
1. Long Buccal Nerve – supplies mucosa opposite
the posterior-most mandibular molars(6,7,8) on
their buccal aspect.
2. Inferior Alveolar Nerve – supplies all lower jaw
teeth, lower lip, buccal mucosa from incisors to
premolar & the skin over the chin.
3. Lingual Nerve – sensory supply to anterior 2/3rd
of tongue, the mucosa on the lingual aspect of
lower teeth & floor of the mouth.
24
MANDIBULAR NERVE
25
LYMPHATICS
26
Submandibular lymph nodes
Submental lymph nodes
Jugulo-Omohyoid group of
deep cervical lymph nodes
Jugulo-Digastric group of
deep cervical lymph nodes
Mandible + lower teeth
Small wedge
in symphysis
Lower
incisors Extremely posterior
STRUCTURES RELATED TO
MANDIBLE
 SALIVARY GLANDS:
 Parotid
 Submandibular
 sublingual
 LYMPH NODES:
 Parotid
 Submandibular
 Submental
 ARTERIES:
 Maxillary
 Superficial temporal
 Masseteric
 Inferior alveolar
 Mylohyoid
 Mental
 Facial
27
 NERVES:
 Lingual
 Auriculotemporal
 Masseteric
 Inferior alveolar
 Mylohyoid
 Mental
 MoM: insertions of
 Temporalis
 Masseter
 Medial pterygoid
 Lateral pterygoid
 LIGAMENTS:
 Lateral ligament of TMJ
 Stylomandibular
 Sphenomandibular and
 Pterygomandibular raphe
28
PRENATAL DEVELOPMENT OF
MANDIBLE
 MECKEL’S CARTILAGE
 Derived from 1st branchial arch
 41st-45th day IU
 Extends from cartilaginous otic capsule to
midline(symphysis)
 Provides template for guiding growth of mandible
 MANDIBULAR DIVISION, TRIGEMINAL NERVE(V3)→
first structure to develop
 2 ossification centres: 1 for each half; arises 6th wk. IU
29
 Ossifying membrane located lateral to the
Meckel’s Cartilage
 Spreads below & around IAN and incisive
branch and upwards to form a trough to
accommodate developing tooth buds
 Dorsally and ventrally spreads to form body and
ramus of mandible
 Ossification continues→ Meckel’s cartilage
surrounded by bone→ invaded by bone→
ossification stops at lingula
30
Continued growth into middle ear : develops
auditory ossicles
To sphenoid bone to
form a remnant of
Meckel’s cartilage
Sphenomandibular ligament
 ENDOCHONDRAL BONE FORMATION
 THE CONDYLAR PROCESS:
 5th wk. IU
 10th- 14th wk. IU
 THE CORONOID PROCESS:
 10th – 14th wk. IU
 Accessory cartilage gets incorporated into
expanding ramus; disappears before birth.
31
Area of mesenchymal condensation
seen above ventral part of the
developing mandible
Develops into a cone-shaped
cartilage → replaced by mid-fetal
life; upper end persists into
adulthood
Secondary cartilage of coronoid
process grows in response to
developing temporalis muscle.
 MENTAL REGION:
 2 small cartilages appear on either side of
symphysis
 7th wk. IU
 1st yr. postnatal
32
Formation of mental ossicles
Incorporated in intramembranous
ossification
Complete ossification
POSTNATAL DEVELOPMENT OF MANDIBLE
 Divided into development of following
functional parts:
 Ramus
 Corpus or Body of mandible
 Angle of the mandible
 Lingual tuberosity
 Alveolar process
 Chin
 Condyle
 Coronoid process
33
THREE FORMS OF GROWTH can be seen in
the mandible:
o Vertical
o Transverse
o rotational
 VERTICAL GROWTH
of the mandible is quite
pronounced. The
mandible has to keep
pace with the descent of
the maxilla and must
also maintain the
interocclusal vertical
direction.
34
 TRANSVERSE
GROWTH of the
mandible is achieved
principally by the
divergence of the
condyles as they grow
posteriorly(Enlow’s V
principle)
 Buccal bone deposition
on the body and ramus
35
 In ROTATIONAL
GROWTH, the matrix
surrounding the
mandible acts to
moderate the shape
changes of the bone
rotating with it.
36
THEORIES OF GROWTH
 ENLOW’S EXPANDING ‘V’ PRINCIPLE
 ENLOW’S COUNTERPART PRINCIPLE
37
States that the growth of any given facial/cranial part
relates specifically to other structural and geometric
counterparts in the face and cranium.
The growth, movement &
enlargement of these
bones occur towards the
wide ends of the ‘V’ as a
result of differential
deposition & selective
resorption.
AGE CHANGES IN MANDIBLE
38
APPLIED ANATOMY
39
AGNATHIA
 Characterized by
hypoplasia or absence of
mandible.
 More commonly, only a
portion of jaw is missing.
 Partial absence of
mandible is more
common.
 Entire mandible on one
side may be missing or
more frequently, only the
condyle or the entire
ramus.
 Bilateral agenesis of
condyles and ramus have
also been reported.
40
MICROGNATHIA
Means small jaw, either the maxilla or
mandible may be involved.
True micrognathia is classified as:
 Congenital
 Acquired
41
CONGENITAL MICROGNATHIA
 Etiology:
I. Idiopathic
II. Assoc’d. with congenital heart disease
III. Pierre-Robin Syndrome
 Follows a hereditary pattern.
 Agenesis of condyles results in true
micrognathia.
 Such cases may be due to posterior
positioning of the mandible with regard to the
skull or to a steep mandibular angle resulting
in apparent retrusion of mandible.
42
ACQUIRED MICROGNATHIA
 Postnatal origin.
 Usually results from a
disturbance in the area of
TMJ.
 Since the normal growth of
the mandible depend on
normally developing
condyles as well as
muscles, condylar ankylosis
may result in deficient
mandible.
 Clinically it is characterized
by severe retrusion of the
chin, a steep mandibular
angle, and a deficient chin
button
43
MACROGNATHIA
 Macrognathia refers to
the condition of
abnormally large jaws.
 An increase in both the
jaws is frequently
proportional to
generalized increase in
entire skeleton.
 Often associated with
other conditions like:
 Paget’s disease of
bone
 Acromegaly
 Leontiasis ossea
44
 Etiology: unknown, although cases may
follow hereditary patterns.
 In many instances the prognathism is due to
disparity in the size of maxilla to mandible.
 The angle between the ramus and the body
influence the relation of mandible to maxilla.
 Thus prognathic patients tend to have long
rami which form a steep angle with the body
of the mandible.
45
FACIAL HEMIHYPERTROPHY
 One of the rare
developmental disorder.
 Asymmetric over growth of
one or more body parts.
 Represents hyperplasia
rather than hypertrophy.
 It is of 3 types, namely:
 Simple hyperplasia
 Complex hyperplasia
 Hemifacial hyperplasia
 F:M > 2:1, often affecting on
right side.
46
 Asymmetry starts at birth.
 Enlargement is more accentuated at the age of 6 and
continues ‘til the overall growth ceases.
 Enlargement of mandible and teeth on the affected side.
 The bone is wider and thicker.
 Premature shedding of the deciduous teeth.
 Roots of teeth are sometimes proportionately enlarged
but maybe short.
 Permanent teeth on the affected side is often enlarged,
most frequently involving cuspid, premolars, and 1st
molar.
 Permanent teeth on affected side develops more rapidly
and erupt before their counterpart on the uninvolved side.
 Macroglossia
47
48
PAGET’S DISEASE
 Characterized by
excessive growth and
abnormal remodeling of
bone.
 Results in bones which are
weak, enlarged and
extensively vascularized.
 Etiology: unknown, there
may be evidence of
genetic link.
 Possible etiologic factors:
 viral infections
 Inflammatory cause
 Autoimmune connective
tissue
49
o Recognized most commonly after
the age of 50 years.
o Its prevalence increases with age.
o Male:female> 1:1
o Jaws are involved more commonly.
o The most common complaint is
bone pain.
o This pain is perceived as dull
aching pain deep below the soft
tissues.
o It may persist or exacerbate during
the night.
o The involved bone becomes warm
to the touch due to increased
vascularity
CHERUBISM
 Autosomal dominant
 The gene is mapped to
chromosome 4p16.
 Facial appearance is similar to
plump-cheeked angels, hence the
name cherubism.
 First described in the year 1953 by
Jones.
 Jaw lesions are usually painless and
symmetric.
 Lesions which are firm and non-
tender to palpate involve molar to
coronoid regions, often associated
with cervical lymphadenopathy.
 This contributes to the characteristic
full-faced appearance.
50
CHERUBISM- RADIOGRAPHIC
APPEARANCE
 Bilateral multilocular
radiolucencies in the
posterior mandible.
 These lesions tend to
show varying degree of
remission and involution
after puberty.
 There maybe
displacement, rotation of
the teeth.
 Premature exfoliation,
delayed eruption.
51
EXOSTOSES
 Normal anatomic
variation.
 Hinders removal of
plaque by patient.
 May have to be removed
to improve the prognosis
of neighbouring teeth.
 Most common in lingual
area of canine and
premolars, above
mylohyoid muscle.
 Also found on
buccal/labial surfaces of
mandibular teeth.
52
ANATOMIC SPACES
 Several anatomic spaces or compartments
are found close to the operative field of
periodontal & implant surgery sites.
 Contain loose connective tissue– easily
distended by hemorrhage, inflammatory fluid,
and infection.
 Surgical invasion of these areas may result in
dangerous hemorrhage (intraoperative) or
infections (postoperative) & should be
carefully avoided.
53
ANATOMIC SPACES
54
A. SUBMENTAL SPACE: between mylohyoid muscle
superiorly and platysma inferiorly. Infection results in
swelling of the region; more dangerous as it proceeds
posteriorly.
B. MASTICATOR SPACE: contains masseter, pterygoid
(lat. and med.), tendon of insertion of temporalis, ramus
and posterior mandible. Infection leads to swelling of
face, severe trismus and pain.
C. SUBLINGUAL SPACE: below the oral mucosa in
anterior part of floor of mouth. Infection of this space
raises floor of mouth, displacing the tongue, resulting in
pain & difficulty swallowing.
D. SUBMANDIBULAR SPACE: external to sublingual
space below mylohyoid and hyoglossus muscle.
Contains the submandibular gland and connected with
sublingual space. Infections lead to obliterated
submandibular line+ pain in swallowing.
SURGICAL CONSIDERATIONS
 Surgical trauma (pressure, manipulation and
postsurgical swelling) to the mental nerve
produces paresthesia of lip– recovers slowly.
 Partial/complete cutting of the nerve can
result in permanent paresthesia/dysesthesia.
55
SURGICAL CONSIDERATIONS
 In partially/completely edentulous patients,
disappearance of alveolar portion brings
mandibular canal and mental foramen closer
to superior border.
 In such patients, during evaluation for
placement of implants, the distance
between canal and superior surface of the
bone as well as location of mental foramen
must be carefully determined to avoid surgical
injury to the nerve.
56
SURGICAL CONSIDERATIONS
 The lingual nerve lies close to the surface of
the oral mucosa in the third molar area and
goes deeper as it travels forward.
 It can be damaged during anesthetic
injections (and during extraction
procedures).
 It can be injured when a periodontal partial-
thickness flap is raised in third molar region
or when releasing incisions are made in the
area.
57
SURGICAL CONSIDERATIONS
 The alveolar process, which provides the supporting
bone to the teeth, has a narrower distal curvature than
the body of mandible, creating a flat surface in the
posterior area between the teeth and the anterior border
of the ramus.
 This results in the formation of external oblique ridge,
which runs downward and forward to region of
second/first molar, creating a shelflike bony area.
 Resective osseous therapy may be difficult in this area
because of the amount of bone that must be removed
distally toward ramus to achieve resection of a
periodontal osseous defect on the distal aspect of
mandibular 2nd/3rd molar.
58
SURGICAL CONSIDERATIONS
 Distal flap procedures in distal to the last molar
can be performed effectively only if there exists
sufficient space.
59
CONCLUSION
 Familiarity with the location and appearance
of the mental nerve reduces likelihood of
injury to the nerve.
 Determining the amount of available bone is
critical for placement of implants.
60
REFERENCES
1. B D Chaurasia’s Human Anatomy vol. 3
2. Human Embryology, Inderbir Singh
3. Clinical Periodontology by Carranza, 10th edition,
vol 2
4. Shafer’s Textbook of Oral Pathology
5. Contemporary Orthodontics by W R Profitt
6. Handbook of Local Anesthesia, S F Malamed
7. Image references:
 Wikimedia Commons
 Gray’s anatomy, 41st edition
61
62

GROWTH & DEVELOPMENT OF MANDIBLE

  • 1.
    MANDIBLE: GROWTH, DEVELOPMENT & ANATOMICAL CONSIDERATIONS By DR. ANTARLEENASENGUPTA I YR PG, DEPTT. OF PERIODONTOLOGY & IMPLANTOLOGY, MCODS MANGALORE
  • 2.
    CONTENTS  INTRODUCTION  ANATOMY BODY OF MANDIBLE  OUTER SURFACE  INNER SURFACE  RAMUS  LATERAL SURFACE  MEDIAL SURFACE  CORONOID PROCESS  CONDYLAR PROCESS  ATTACHMENTS & RELATIONS  MUSCLE ATTACHMENTS  FORAMINA  BLOOD SUPPLY  NERVE SUPPLY  LYMPHATICS  STRUCTURES RELATED TO MANDIBLE 2
  • 3.
    contd.  GROWTH OFMANDIBLE  PRENATAL EMBRYOLOGY  MECKEL’S CARTILAGE  ENDOCHONDRAL BONE FORMATION  CONDYLAR PROCESS  CORONOID PROCESS  MENTAL REGION  POST-NATAL EMBRYOLOGY  RAMUS  BODY  ANGLE  LINGUAL TUBEROSITY  ALVEOLAR PROCESS  CHIN  CONDYLAR PROCESS  CORONOID PROCESS  THEORIES OF GROWTH  ENLOW’S EXPANDING V PRINCIPLE  ENLOW’S COUNTERPART PRINCIPLE  AGE CHANGES IN MANDIBLE  INFANTS & CHILDREN  ADULTS  OLD AGE  ANATOMIC CONSIDERATIONS  DEVELOPMENTAL ANOMALIES  SURGICAL CONSIDERATIONS  ANATOMIC SPACES  CONCLUSION  REFERENCES 3
  • 4.
  • 5.
  • 6.
  • 7.
  • 8.
    8 MENTAL PROTUBERANCE MENTALTUBERCLES ANTERIOR VIEW
  • 9.
     BODY  Eachhalf of the body has outer and inner surfaces, and upper and lower borders.  The outer surface presents the following features: 1. The symphysis menti 2. The mental protuberance 3. The mental foramen 4. The oblique line 9
  • 10.
  • 11.
  • 12.
    INNER SURFACE hasthe following features:  Mylohyoid Line  Submandibular fossa  Sublingual fossa  Superior and Inferior genial tubercles  Mylohyoid groove 12
  • 13.
  • 14.
    UPPER BORDER The upperor alveolar border bears sockets for the teeth. 14
  • 15.
    LOWER BORDER 15 The lowerborder of the mandible is also called the base. Near the midline the base shows an oval depression called the digastric fossa.
  • 16.
     RAMUS 16 ANTERIOR VIEWLATERAL VIEW • LATERAL SURFACE of ramus is flat and bears a number of oblique ridges. U L P A
  • 17.
    MEDIAL SURFACE oframus has the following:  Mandibular foramen  Lingula  Mylohyoid groove  Mandibular notch  Angle of mandible 17 Lingula Submandibular fossaMylohyoid groove Mandibular notch
  • 18.
     Coronoid process Condyloid process  Head  Neck  Pterygoid fovea 18 Superior view Superior view Pterygoid fovea Neck
  • 19.
  • 20.
    ATTACHMENTS & RELATIONSOF MANDIBLE  MUSCLE ATTACHMENTS 20 LATERAL VIEW
  • 21.
  • 22.
    FORAMINA & RELATIONSOF MANDIBLE 22 Mandibular foramen Mental foramen
  • 23.
    BLOOD SUPPLY  Centralblood supply  Peripheral blood supply 23 via the INFERIOR ALVEOLAR ARTERY except the coronoid process, which is supplied by temporalis muscle vessels. via the PERIOSTEAL VESSELS, which run parallel to cortical surface of bone, giving off NUTRIENT VESSELS those penetrate cortical bone and anastomose with the branches of inferior alveolar artery.
  • 24.
    NERVE SUPPLY  Derivedfrom mandibular branch (V3) of trigeminal nerve. 1. Long Buccal Nerve – supplies mucosa opposite the posterior-most mandibular molars(6,7,8) on their buccal aspect. 2. Inferior Alveolar Nerve – supplies all lower jaw teeth, lower lip, buccal mucosa from incisors to premolar & the skin over the chin. 3. Lingual Nerve – sensory supply to anterior 2/3rd of tongue, the mucosa on the lingual aspect of lower teeth & floor of the mouth. 24
  • 25.
  • 26.
    LYMPHATICS 26 Submandibular lymph nodes Submentallymph nodes Jugulo-Omohyoid group of deep cervical lymph nodes Jugulo-Digastric group of deep cervical lymph nodes Mandible + lower teeth Small wedge in symphysis Lower incisors Extremely posterior
  • 27.
    STRUCTURES RELATED TO MANDIBLE SALIVARY GLANDS:  Parotid  Submandibular  sublingual  LYMPH NODES:  Parotid  Submandibular  Submental  ARTERIES:  Maxillary  Superficial temporal  Masseteric  Inferior alveolar  Mylohyoid  Mental  Facial 27
  • 28.
     NERVES:  Lingual Auriculotemporal  Masseteric  Inferior alveolar  Mylohyoid  Mental  MoM: insertions of  Temporalis  Masseter  Medial pterygoid  Lateral pterygoid  LIGAMENTS:  Lateral ligament of TMJ  Stylomandibular  Sphenomandibular and  Pterygomandibular raphe 28
  • 29.
    PRENATAL DEVELOPMENT OF MANDIBLE MECKEL’S CARTILAGE  Derived from 1st branchial arch  41st-45th day IU  Extends from cartilaginous otic capsule to midline(symphysis)  Provides template for guiding growth of mandible  MANDIBULAR DIVISION, TRIGEMINAL NERVE(V3)→ first structure to develop  2 ossification centres: 1 for each half; arises 6th wk. IU 29
  • 30.
     Ossifying membranelocated lateral to the Meckel’s Cartilage  Spreads below & around IAN and incisive branch and upwards to form a trough to accommodate developing tooth buds  Dorsally and ventrally spreads to form body and ramus of mandible  Ossification continues→ Meckel’s cartilage surrounded by bone→ invaded by bone→ ossification stops at lingula 30 Continued growth into middle ear : develops auditory ossicles To sphenoid bone to form a remnant of Meckel’s cartilage Sphenomandibular ligament
  • 31.
     ENDOCHONDRAL BONEFORMATION  THE CONDYLAR PROCESS:  5th wk. IU  10th- 14th wk. IU  THE CORONOID PROCESS:  10th – 14th wk. IU  Accessory cartilage gets incorporated into expanding ramus; disappears before birth. 31 Area of mesenchymal condensation seen above ventral part of the developing mandible Develops into a cone-shaped cartilage → replaced by mid-fetal life; upper end persists into adulthood Secondary cartilage of coronoid process grows in response to developing temporalis muscle.
  • 32.
     MENTAL REGION: 2 small cartilages appear on either side of symphysis  7th wk. IU  1st yr. postnatal 32 Formation of mental ossicles Incorporated in intramembranous ossification Complete ossification
  • 33.
    POSTNATAL DEVELOPMENT OFMANDIBLE  Divided into development of following functional parts:  Ramus  Corpus or Body of mandible  Angle of the mandible  Lingual tuberosity  Alveolar process  Chin  Condyle  Coronoid process 33 THREE FORMS OF GROWTH can be seen in the mandible: o Vertical o Transverse o rotational
  • 34.
     VERTICAL GROWTH ofthe mandible is quite pronounced. The mandible has to keep pace with the descent of the maxilla and must also maintain the interocclusal vertical direction. 34
  • 35.
     TRANSVERSE GROWTH ofthe mandible is achieved principally by the divergence of the condyles as they grow posteriorly(Enlow’s V principle)  Buccal bone deposition on the body and ramus 35
  • 36.
     In ROTATIONAL GROWTH,the matrix surrounding the mandible acts to moderate the shape changes of the bone rotating with it. 36
  • 37.
    THEORIES OF GROWTH ENLOW’S EXPANDING ‘V’ PRINCIPLE  ENLOW’S COUNTERPART PRINCIPLE 37 States that the growth of any given facial/cranial part relates specifically to other structural and geometric counterparts in the face and cranium. The growth, movement & enlargement of these bones occur towards the wide ends of the ‘V’ as a result of differential deposition & selective resorption.
  • 38.
    AGE CHANGES INMANDIBLE 38
  • 39.
  • 40.
    AGNATHIA  Characterized by hypoplasiaor absence of mandible.  More commonly, only a portion of jaw is missing.  Partial absence of mandible is more common.  Entire mandible on one side may be missing or more frequently, only the condyle or the entire ramus.  Bilateral agenesis of condyles and ramus have also been reported. 40
  • 41.
    MICROGNATHIA Means small jaw,either the maxilla or mandible may be involved. True micrognathia is classified as:  Congenital  Acquired 41
  • 42.
    CONGENITAL MICROGNATHIA  Etiology: I.Idiopathic II. Assoc’d. with congenital heart disease III. Pierre-Robin Syndrome  Follows a hereditary pattern.  Agenesis of condyles results in true micrognathia.  Such cases may be due to posterior positioning of the mandible with regard to the skull or to a steep mandibular angle resulting in apparent retrusion of mandible. 42
  • 43.
    ACQUIRED MICROGNATHIA  Postnatalorigin.  Usually results from a disturbance in the area of TMJ.  Since the normal growth of the mandible depend on normally developing condyles as well as muscles, condylar ankylosis may result in deficient mandible.  Clinically it is characterized by severe retrusion of the chin, a steep mandibular angle, and a deficient chin button 43
  • 44.
    MACROGNATHIA  Macrognathia refersto the condition of abnormally large jaws.  An increase in both the jaws is frequently proportional to generalized increase in entire skeleton.  Often associated with other conditions like:  Paget’s disease of bone  Acromegaly  Leontiasis ossea 44
  • 45.
     Etiology: unknown,although cases may follow hereditary patterns.  In many instances the prognathism is due to disparity in the size of maxilla to mandible.  The angle between the ramus and the body influence the relation of mandible to maxilla.  Thus prognathic patients tend to have long rami which form a steep angle with the body of the mandible. 45
  • 46.
    FACIAL HEMIHYPERTROPHY  Oneof the rare developmental disorder.  Asymmetric over growth of one or more body parts.  Represents hyperplasia rather than hypertrophy.  It is of 3 types, namely:  Simple hyperplasia  Complex hyperplasia  Hemifacial hyperplasia  F:M > 2:1, often affecting on right side. 46
  • 47.
     Asymmetry startsat birth.  Enlargement is more accentuated at the age of 6 and continues ‘til the overall growth ceases.  Enlargement of mandible and teeth on the affected side.  The bone is wider and thicker.  Premature shedding of the deciduous teeth.  Roots of teeth are sometimes proportionately enlarged but maybe short.  Permanent teeth on the affected side is often enlarged, most frequently involving cuspid, premolars, and 1st molar.  Permanent teeth on affected side develops more rapidly and erupt before their counterpart on the uninvolved side.  Macroglossia 47
  • 48.
  • 49.
    PAGET’S DISEASE  Characterizedby excessive growth and abnormal remodeling of bone.  Results in bones which are weak, enlarged and extensively vascularized.  Etiology: unknown, there may be evidence of genetic link.  Possible etiologic factors:  viral infections  Inflammatory cause  Autoimmune connective tissue 49 o Recognized most commonly after the age of 50 years. o Its prevalence increases with age. o Male:female> 1:1 o Jaws are involved more commonly. o The most common complaint is bone pain. o This pain is perceived as dull aching pain deep below the soft tissues. o It may persist or exacerbate during the night. o The involved bone becomes warm to the touch due to increased vascularity
  • 50.
    CHERUBISM  Autosomal dominant The gene is mapped to chromosome 4p16.  Facial appearance is similar to plump-cheeked angels, hence the name cherubism.  First described in the year 1953 by Jones.  Jaw lesions are usually painless and symmetric.  Lesions which are firm and non- tender to palpate involve molar to coronoid regions, often associated with cervical lymphadenopathy.  This contributes to the characteristic full-faced appearance. 50
  • 51.
    CHERUBISM- RADIOGRAPHIC APPEARANCE  Bilateralmultilocular radiolucencies in the posterior mandible.  These lesions tend to show varying degree of remission and involution after puberty.  There maybe displacement, rotation of the teeth.  Premature exfoliation, delayed eruption. 51
  • 52.
    EXOSTOSES  Normal anatomic variation. Hinders removal of plaque by patient.  May have to be removed to improve the prognosis of neighbouring teeth.  Most common in lingual area of canine and premolars, above mylohyoid muscle.  Also found on buccal/labial surfaces of mandibular teeth. 52
  • 53.
    ANATOMIC SPACES  Severalanatomic spaces or compartments are found close to the operative field of periodontal & implant surgery sites.  Contain loose connective tissue– easily distended by hemorrhage, inflammatory fluid, and infection.  Surgical invasion of these areas may result in dangerous hemorrhage (intraoperative) or infections (postoperative) & should be carefully avoided. 53
  • 54.
    ANATOMIC SPACES 54 A. SUBMENTALSPACE: between mylohyoid muscle superiorly and platysma inferiorly. Infection results in swelling of the region; more dangerous as it proceeds posteriorly. B. MASTICATOR SPACE: contains masseter, pterygoid (lat. and med.), tendon of insertion of temporalis, ramus and posterior mandible. Infection leads to swelling of face, severe trismus and pain. C. SUBLINGUAL SPACE: below the oral mucosa in anterior part of floor of mouth. Infection of this space raises floor of mouth, displacing the tongue, resulting in pain & difficulty swallowing. D. SUBMANDIBULAR SPACE: external to sublingual space below mylohyoid and hyoglossus muscle. Contains the submandibular gland and connected with sublingual space. Infections lead to obliterated submandibular line+ pain in swallowing.
  • 55.
    SURGICAL CONSIDERATIONS  Surgicaltrauma (pressure, manipulation and postsurgical swelling) to the mental nerve produces paresthesia of lip– recovers slowly.  Partial/complete cutting of the nerve can result in permanent paresthesia/dysesthesia. 55
  • 56.
    SURGICAL CONSIDERATIONS  Inpartially/completely edentulous patients, disappearance of alveolar portion brings mandibular canal and mental foramen closer to superior border.  In such patients, during evaluation for placement of implants, the distance between canal and superior surface of the bone as well as location of mental foramen must be carefully determined to avoid surgical injury to the nerve. 56
  • 57.
    SURGICAL CONSIDERATIONS  Thelingual nerve lies close to the surface of the oral mucosa in the third molar area and goes deeper as it travels forward.  It can be damaged during anesthetic injections (and during extraction procedures).  It can be injured when a periodontal partial- thickness flap is raised in third molar region or when releasing incisions are made in the area. 57
  • 58.
    SURGICAL CONSIDERATIONS  Thealveolar process, which provides the supporting bone to the teeth, has a narrower distal curvature than the body of mandible, creating a flat surface in the posterior area between the teeth and the anterior border of the ramus.  This results in the formation of external oblique ridge, which runs downward and forward to region of second/first molar, creating a shelflike bony area.  Resective osseous therapy may be difficult in this area because of the amount of bone that must be removed distally toward ramus to achieve resection of a periodontal osseous defect on the distal aspect of mandibular 2nd/3rd molar. 58
  • 59.
    SURGICAL CONSIDERATIONS  Distalflap procedures in distal to the last molar can be performed effectively only if there exists sufficient space. 59
  • 60.
    CONCLUSION  Familiarity withthe location and appearance of the mental nerve reduces likelihood of injury to the nerve.  Determining the amount of available bone is critical for placement of implants. 60
  • 61.
    REFERENCES 1. B DChaurasia’s Human Anatomy vol. 3 2. Human Embryology, Inderbir Singh 3. Clinical Periodontology by Carranza, 10th edition, vol 2 4. Shafer’s Textbook of Oral Pathology 5. Contemporary Orthodontics by W R Profitt 6. Handbook of Local Anesthesia, S F Malamed 7. Image references:  Wikimedia Commons  Gray’s anatomy, 41st edition 61
  • 62.