RESUSCITATION
& THORACIC
TRAUMA
PRESENTERS: DR UMAR & DR MARYAM
SUPERVISOR: DR FAIZUL
OUTLINE
• INTRODUCTION
•LIFE THREATENING THORACIC INJURY
•COMMON CONDITION OF THORACIC INJURY
• TAKE HOME MESSAGES
INTRODUCTION
• Trauma causes 100,000 deaths and more than 9 million disability
injury annually in worldwide (6th
leading cause of death).
• Chest injuries are responsible for 20-25% of all trauma death.
• The World Health Ranking 2011 has ranked Malaysia at number 20
with the most deaths caused by road accidents.
• An average of 18 people were killed on Malaysian roads daily.
* Malaysian Institute of Road Safety (MIROS)
CLASIFICATION
•Anatomy
• Lungs, pleura and ribs
• Cardiac injury
• Trachea , oesophagus & major vessels
• Diaphragm
•Mechanism of injury
• Blunt
• Penetrating
• Shearing / acceleration-decelaration injury
•Cause / Aetiology
• MVA / trauma
• Fall from height
• Gunshot / sharp or blunt object
LIFETHREATENING
CONDITION
TENSION
PNEUMOTHORAX
TENSION PNEUMOTHORAX
Develops when air leak occurs
from the lung or through chest
wall.
Air is forced into thoracic
cavity without means of
escape
Creating a “one-way valve”.
Affected lung collapsed
Displaced mediastinum to
opposite site
Decrease venous return
Compressing the opposite
lung
TENSION PNEUMOTHORAX
SIGNS AND SYMPTOMS
TENSION PNEUMOTHORAX
RADIOGRAPHIC FINDINGS
Mediastinal
shift to the
right
TENSION
PNEUMOTHORAX
Tension pneumothorax is a clinical diagnosis.
Treatment should not be delayed to wait for radiologic confirmation.
TENSION PNEUMOTHORAX
Management
1. Initial Management: needle thoracostomy
- convert the injury to simple pneumothorax.
2. Definitive management: chest tube insertion
3. Supportive management:
- Analgesia
- Ventilatory support
- CXR monitoring
- Chest phyisotherapy
NEEDLE THORACOSTOMY
OPEN
PNEUMOTHORAX
OPEN PNEUMOTHORAX:
Pathophysiology
• Known as “sucking
chest wound.”
• Air allowed to enter
pleural space from the
outside.
• Ineffective ventilation
because air goes in and
out from the chest
wound, rather than
from trachea.
• Leading to hypoxia and
hypercarbia.
Open Pneumothorax
Clinical Findings
•A defect in the chest wall
with air coming in & out
•A sucking sound on
inhalation
•Tachycardia & tachypnea
•Respiratory distress
•Subcutaneous
emphysema
•Decreased breath sounds
on the affected side
OPEN PNEUMOTHORAX
MANAGEMENT
1. Initial management:
• 3 sided sterile occlusive
dressing
•Treat concurrent shock
2. Definitive management
•Chest tube insertion
3-sided occlusive dressing
FLAIL CHEST
& PULMONARY
CONTUSION
FLAIL CHEST
• Described as the
paradoxical movement
of a segment of chest
wall caused by
fractures of 3 or more
ribs in 2 or more
placed.
FLAIL CHEST
Severe hypoxia resulting from:
•The underlying lung injury  disturbance of ventilation & perfusion
•Restricted chest wall movement associated with pain  impaired ventilation
FLAIL CHEST
Clinical findings
INSPECTION
-Chest wall contusion
-Paradoxical chest wall
movement
-Respiratory distress
Palpation
-Crepitation of rib
FLAIL CHEST
Investigation
•CXR : multiple
ribs fracture
•ABG: respiratory
failure with
hypoxia
FLAIL CHEST
Management
1. Initial management:
- adequate ventilation
fluid resuscitation
In absence of systemic hypotension, fluid
resuscitation should be carefully controlled to
prevent overhydration.
FLAIL CHEST
Management
2. Definitive management
Positive-pressure ventilation may be needed.
•Reverses the mechanism of paradoxical chest wall
movement
•Restores the tidal volume
Adequate analgesic
•Reduces the pain of chest wall movement
Assess for the development of a pneumothorax
•May need chest tube insertion
MASSIVE
HEMOTHORAX
MASSIVE HEMOTHORAX
Defined as presence of
>1.5 liter
of blood drained from
the pleural space upon
chest tube insertion
or >200cc/hour in first
4 hours.
MASSIVE HEMOTHORAX
Clinical Findings
MASSIVE HEMOTHORAX
Management
Large caliber IV lines
Crystalloid infusion
Blood transfusion
Chest tube insertion
Consider for thoracotomy
MASSIVE HEMOTHORAX
Chest tube insertion
MASSIVE HEMOTHORAX
10 days after chest tube insertionOn admission
CARDIAC
TAMPONADE
CARDIAC TAMPONADE
•Suspect if injury within
the “box”.
•May need prompt
involvement of
cardiothoracic team
CARDIAC TAMPONADE
Pathophysiology
•A blunt or penetrating trauma may cause tears
in the myocardial walls, allowing blood to leak
from the heart.
If 150 to 200 mL of blood enters the pericardial
space acutely, pericardial tamponade can develops
CARDIAC TAMPONADE
CARDIAC TAMPONADE
CARDIAC TAMPONADE
Cardiac Tamponade
Management
•Airway and ventilation
•Circulation—IV fluid challenge
•Pericardiocentesis
•Prompt involvement of cardiothoracic team.
•Do not take out the penetrating object
CARDIAC TAMPONADE
Pericardocentesis
OTHER CHEST INJURIES
1. RIB FRACTURE
2. SIMPLE PNEUMOTHORAX
3. HEMOTHORAX
4. PULMONARY CONTUSION
5. TRACHEOBRONCHIAL TREE INJURIES
6. CARDIAC CONTUSION
7. TRAUMATIC MAJOR VESSEL DISRUPTION
8. DIAPHRAGMATIC INJURIES
*Hemorrhage should be excluded in all patients who are in shock after major trauma
†Neck vein distention may be absent in patients with hypovolemic shock.
GENERAL MANAGEMENT
•Primary & secondary survey
•Serial clinical assessment & SPO2 monitoring
•Adequate analgesia (pain control CPG)
•Oxygen therapy tailored to oxygenation status
•Chest tube insertion
•Intensive & vigorous chest physiotherapy, deep
breathing exercise & incentive spirometry
•Mucolytic & nebulizer
•Early referral to appropriate team (i.e. anaest, CTC)
•± Assisted ventilation or intubation
•± Thoracotomy / thorachoscopy and proceed
TAKE HOME MESSAGES
1. Life threatening condition in thoracic injury are
• Tension pneumothorax
• Open pneumothorax
• Massive hemothorax
• Flail chest
• Cardiac temponade.
2. Tension pneumothorax required emergent needle
thoracotomy without waiting for CXR if highly suspected
clinically
3. Do not remove the object causing the penetrating thoracic
injury
4. Open pneumothorax is managed with flutter-valve dressing or
three sided dressing
TAKE HOME MESSAGES
5. Flail chest is defined as segmental fractures in 2 or more
places of 3 or more consecutive ribs.
6. Massive hemothorax happen when
• more than 1.5 liters blood drained upon chest tube insertion
• Or more than 200cc/hour in 4 hours
5. All symptomatic traumatic pneumo/hemothorax require chest
tube insertion
6. Cardiac tamponade is recognized by presence of Beck’s Triad
which are
• Muffled heart sound
• Hypotension
• Distended neck veins
TAKE HOME MESSAGES
9. Key management in thoracic injury include
• Identifying the life threatening condition
• Resuscitation and oxygen therapy
• Chest tube insertion
• Adequate pain control and aggressive chest physiotherapy
• Ventilation and early associate team referral
REFERENCES
• ATLS for Doctors, 8th
Edition
• Bailey & Love Short Practice of Surgery, 25th
Editions
• Emergency Medicine Clinics of North America
- Volume 30, Issue 2 (May 2012)
• SRB’s Manual of Surgery 4th
edition

Resuscitation and thoracic trauma

  • 1.
    RESUSCITATION & THORACIC TRAUMA PRESENTERS: DRUMAR & DR MARYAM SUPERVISOR: DR FAIZUL
  • 2.
    OUTLINE • INTRODUCTION •LIFE THREATENINGTHORACIC INJURY •COMMON CONDITION OF THORACIC INJURY • TAKE HOME MESSAGES
  • 3.
    INTRODUCTION • Trauma causes100,000 deaths and more than 9 million disability injury annually in worldwide (6th leading cause of death). • Chest injuries are responsible for 20-25% of all trauma death. • The World Health Ranking 2011 has ranked Malaysia at number 20 with the most deaths caused by road accidents. • An average of 18 people were killed on Malaysian roads daily. * Malaysian Institute of Road Safety (MIROS)
  • 4.
    CLASIFICATION •Anatomy • Lungs, pleuraand ribs • Cardiac injury • Trachea , oesophagus & major vessels • Diaphragm •Mechanism of injury • Blunt • Penetrating • Shearing / acceleration-decelaration injury •Cause / Aetiology • MVA / trauma • Fall from height • Gunshot / sharp or blunt object
  • 5.
  • 6.
  • 7.
    TENSION PNEUMOTHORAX Develops whenair leak occurs from the lung or through chest wall. Air is forced into thoracic cavity without means of escape Creating a “one-way valve”. Affected lung collapsed Displaced mediastinum to opposite site Decrease venous return Compressing the opposite lung
  • 8.
  • 9.
    TENSION PNEUMOTHORAX RADIOGRAPHIC FINDINGS Mediastinal shiftto the right TENSION PNEUMOTHORAX Tension pneumothorax is a clinical diagnosis. Treatment should not be delayed to wait for radiologic confirmation.
  • 10.
    TENSION PNEUMOTHORAX Management 1. InitialManagement: needle thoracostomy - convert the injury to simple pneumothorax. 2. Definitive management: chest tube insertion 3. Supportive management: - Analgesia - Ventilatory support - CXR monitoring - Chest phyisotherapy
  • 11.
  • 12.
  • 13.
    OPEN PNEUMOTHORAX: Pathophysiology • Knownas “sucking chest wound.” • Air allowed to enter pleural space from the outside. • Ineffective ventilation because air goes in and out from the chest wound, rather than from trachea. • Leading to hypoxia and hypercarbia.
  • 14.
    Open Pneumothorax Clinical Findings •Adefect in the chest wall with air coming in & out •A sucking sound on inhalation •Tachycardia & tachypnea •Respiratory distress •Subcutaneous emphysema •Decreased breath sounds on the affected side
  • 15.
    OPEN PNEUMOTHORAX MANAGEMENT 1. Initialmanagement: • 3 sided sterile occlusive dressing •Treat concurrent shock 2. Definitive management •Chest tube insertion
  • 16.
  • 17.
  • 18.
    FLAIL CHEST • Describedas the paradoxical movement of a segment of chest wall caused by fractures of 3 or more ribs in 2 or more placed.
  • 19.
    FLAIL CHEST Severe hypoxiaresulting from: •The underlying lung injury  disturbance of ventilation & perfusion •Restricted chest wall movement associated with pain  impaired ventilation
  • 20.
    FLAIL CHEST Clinical findings INSPECTION -Chestwall contusion -Paradoxical chest wall movement -Respiratory distress Palpation -Crepitation of rib
  • 21.
    FLAIL CHEST Investigation •CXR :multiple ribs fracture •ABG: respiratory failure with hypoxia
  • 22.
    FLAIL CHEST Management 1. Initialmanagement: - adequate ventilation fluid resuscitation In absence of systemic hypotension, fluid resuscitation should be carefully controlled to prevent overhydration.
  • 23.
    FLAIL CHEST Management 2. Definitivemanagement Positive-pressure ventilation may be needed. •Reverses the mechanism of paradoxical chest wall movement •Restores the tidal volume Adequate analgesic •Reduces the pain of chest wall movement Assess for the development of a pneumothorax •May need chest tube insertion
  • 24.
  • 25.
    MASSIVE HEMOTHORAX Defined aspresence of >1.5 liter of blood drained from the pleural space upon chest tube insertion or >200cc/hour in first 4 hours.
  • 26.
  • 27.
    MASSIVE HEMOTHORAX Management Large caliberIV lines Crystalloid infusion Blood transfusion Chest tube insertion Consider for thoracotomy
  • 28.
  • 29.
    MASSIVE HEMOTHORAX 10 daysafter chest tube insertionOn admission
  • 30.
  • 31.
    CARDIAC TAMPONADE •Suspect ifinjury within the “box”. •May need prompt involvement of cardiothoracic team
  • 32.
    CARDIAC TAMPONADE Pathophysiology •A bluntor penetrating trauma may cause tears in the myocardial walls, allowing blood to leak from the heart. If 150 to 200 mL of blood enters the pericardial space acutely, pericardial tamponade can develops
  • 33.
  • 34.
  • 35.
  • 36.
    Cardiac Tamponade Management •Airway andventilation •Circulation—IV fluid challenge •Pericardiocentesis •Prompt involvement of cardiothoracic team. •Do not take out the penetrating object
  • 37.
  • 38.
    OTHER CHEST INJURIES 1.RIB FRACTURE 2. SIMPLE PNEUMOTHORAX 3. HEMOTHORAX 4. PULMONARY CONTUSION 5. TRACHEOBRONCHIAL TREE INJURIES 6. CARDIAC CONTUSION 7. TRAUMATIC MAJOR VESSEL DISRUPTION 8. DIAPHRAGMATIC INJURIES
  • 40.
    *Hemorrhage should beexcluded in all patients who are in shock after major trauma †Neck vein distention may be absent in patients with hypovolemic shock.
  • 41.
    GENERAL MANAGEMENT •Primary &secondary survey •Serial clinical assessment & SPO2 monitoring •Adequate analgesia (pain control CPG) •Oxygen therapy tailored to oxygenation status •Chest tube insertion •Intensive & vigorous chest physiotherapy, deep breathing exercise & incentive spirometry •Mucolytic & nebulizer •Early referral to appropriate team (i.e. anaest, CTC) •± Assisted ventilation or intubation •± Thoracotomy / thorachoscopy and proceed
  • 42.
    TAKE HOME MESSAGES 1.Life threatening condition in thoracic injury are • Tension pneumothorax • Open pneumothorax • Massive hemothorax • Flail chest • Cardiac temponade. 2. Tension pneumothorax required emergent needle thoracotomy without waiting for CXR if highly suspected clinically 3. Do not remove the object causing the penetrating thoracic injury 4. Open pneumothorax is managed with flutter-valve dressing or three sided dressing
  • 43.
    TAKE HOME MESSAGES 5.Flail chest is defined as segmental fractures in 2 or more places of 3 or more consecutive ribs. 6. Massive hemothorax happen when • more than 1.5 liters blood drained upon chest tube insertion • Or more than 200cc/hour in 4 hours 5. All symptomatic traumatic pneumo/hemothorax require chest tube insertion 6. Cardiac tamponade is recognized by presence of Beck’s Triad which are • Muffled heart sound • Hypotension • Distended neck veins
  • 44.
    TAKE HOME MESSAGES 9.Key management in thoracic injury include • Identifying the life threatening condition • Resuscitation and oxygen therapy • Chest tube insertion • Adequate pain control and aggressive chest physiotherapy • Ventilation and early associate team referral
  • 45.
    REFERENCES • ATLS forDoctors, 8th Edition • Bailey & Love Short Practice of Surgery, 25th Editions • Emergency Medicine Clinics of North America - Volume 30, Issue 2 (May 2012) • SRB’s Manual of Surgery 4th edition

Editor's Notes

  • #28 Indications for thoracotormy: 1. >1.5 liter of blood drained from the pleural space upon chest tube insertion or >200cc/hour in first 4 hours. 2. Initial output less than 1.5 L, but continues bleed and need transfusion 3. Site: medial to nipple line/scapula
  • #29 Indication for chest tube?
  • #38 Removal of as little as 20 mL of blood may drastically improve cardiac output.