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CASE PRESENTATION
ON
CHOLELITHIASIS
BY ARUSHI NEGI
M.SC. NURSING 1ST YEAR
Identification Data:
• Client name: Mrs. Omi devi
• Age/sex: - 67 years / Female
• Father/spouse name: Mr. Ram meher
• Hospital registration number: 22030
• Ward: Surgery ward
• Bed No.- 45
• Address: Village sher, Panipat , Haryana
• Education: 3rd
• Occupation: House wife
Marital status: Married
Religion: Hindu
Date of admission:26/12/2021
Date of discharge: -
Diagnosis: Cholelithiasis
Surgery (if any): Laparascopic cholecystectomy
Date of Surgery: 28-12-2021
Date of interview :03-01-2022
Doctor In-charge: Dr. J.P singh
Informant : Patient herself.
History of Illness:
Chief complaints: patient was having pain in abdomen since
last 12 days.
History of fever 4-5 days
At present :Patient is well , and have pain at surgical site.
 Present medical history:
My patient was having epigastric pain since last 12 days so she
visited a nearby clinic at her residence who recommended an
ultrasound in which she was diagnosed with cholelithiasis. She
was recommended to go for higher facility for which she chose
Lal Bahadur Shastri Hospital
Past medical history:
Patient is diagnosed with diabetes since last 15 years and
was taking treatment from her nearby clinic.She has now
stopped taking medications .
Past surgical history:
Patient has undrgone hernioplasty 5 years back for hiatal
hernia nearby in a facility near her house.
FAMILY HISTORY
S. No . Name of
family
members
Age sex Occupation Relation of
family
Member to
patient
Education Health
status
1. Omi devi 67y F House wife Self 3rd Unhealthy
2. Ramprakash 70 y M Farmer Husband 5th Healthy
3. Indrajeet 45 y M Pvt job Son BA Healthy
4. Sushila 40 y F House wife Daughter in
law
10th Healthy
5. Rahul 20 y M Student Grandson BBA Healthy
6. Rakesh 18 y M Student Grandson 12th Healthy
7. Monika 44 y F House wife Daughter 12th Healthy
8. Rajeev 46 y M Pvt job Son in law Polytech Healthy
9. Shikha 16 y F Student Granddaught
er
10th Healthy
FAMILY TREE:
Female
Male
 Personal History:
No smoking and alchol consumption history.
 Menstrual history:
Patient attained menarche at the age of 14 yrs , and
menopause at 50 yrs,
She didn’t undergo any checkups during her pregnancy.
Had normal vaginal delivery.
 Dietary habits:
She consumes non vegetarian diet.
 Elimination pattern : She has constipation
PHYSICAL EXAMINATION
GENERALAPPERANCE:
 Body build - Endomorph
 Height- 151 cm
 Weight- 95 kg
Vital signs
 Temperature - 98.7 F
 Pulse: 85 b/m
 Respiration: 18 b/m
 B.P: 124/96 mmHg
Colour of skin: normal
Head:
Shape and size of skull: well rounded
Scalp: free from any lices
Face: sagging of skin due to old age
Eyes: no abnormality detected.
 Eye brow and eyelid: hair evenly distributed ,
symmetrical
Conjunctiva: no abnormality detected.
Sclera: slight yellow in color
Ear:
External ear: symmetrical
Hearing problem: no abnormality detected.
Nose:
External nares: straight and uniform
Mouth and pharynx:
Mouth: lips are pink in color
Teeth: no discoloration , lacks 3 teeth
Tongue: normal
Neck:
Thyroid gland: no abnormality detected
Lymph node: normal
Range of motion: No abnormalities.
Chest:
Breath sounds: no abnormality detected
Lungs: clear
Abdomen:
Inspection: scars of laproscopic cholecystectomy .
Auscultation: no abnormality detected.
Palpation: pain on touch
Extremities:
Upper: no abnormalities
Lower: no abnormalities
Back: no bed sores.
LAB INVESTIGATION:
LFT :
Bilirubin - 3 mg/dl
(0.1-1.2 mg/dl)
Direct -0.5 mg/dl
(0.0-0.2 mg/dl )
SGPT - 42 IU/L(
5-40 )
SGOT - 41
IU/L(5-40)
MEDICATIONS:
In my patient:
 VANCOMYCIN
 METRONIDAZOLE
 CIPLOXACIN
 PANTOPRAZOLE
 PARACETAMOL
 MONOCEFTRIAXONE
 NORMAL SALINE
METRONIDAZOLE
CHEMICAL
NAME
DOSE /ROUTE ACTION INDICATION CONTRA-
INDICATION
SIDE EFFECT NURSING
RESPONSIBILITI
ES
METRO NIDA
ZOLE
TRADE NAME -
METROGYL
DRUG CLASS -
ANTIBIOTICS
Adults—500 or 750
milligrams (mg) 3
times a day for 5 to
10 days.
Children—Dose is
based on body
weight and must be
determined by your
doctor. The dose is
usually 35 to 50
milligrams (mg) per
kilogram (kg) of
body weight per day,
divided into 3 doses,
for 10 days.
Metronidazole
interacts with the
microbial DNA to
break its strand and
helical structure
leading to inhibition
of protein synthesis,
degradation, and
cell death.
• Anaerobic
bacterial
infections.
• Amoebiasis
Bacterial
vaginosis
Trichomoniasis
• H. pylori
eradication
associated with
peptic ulcer
disease. Acute
dental infections.
• Prophylaxis of
post-op
anaerobic
bacterial
infections
• Hypersensitivity
to metronidazole
and other
nitroimidazoles.
• Concomitant
use with
disulfiram within
the last 14 days.
• Coadministratio
n with alcohol
or propylene
glycol
containing
products during
or 3 days after
therapy
discontinuation.
• Pregnancy
during the 1st
trimester in the
treatment of
trichomoniasis.
Severe neurological
disturbances,
encephalopathy,
convulsive seizures,
aseptic meningitis,
peripheral and optic
neuropathy,
paraesthesia;
superinfection (e.g.
fungal or bacterial
superinfection,
C. difficile-
associated diarrhoea.
Blood and
lymphatic system
disorders:
Leucopenia,
neutropenia. Cardiac
disorders: Chest
pain, tachycardia.
Ear and labyrinth
disorders: Tinnitu
Avoid use unless
necessary.
Metronidazole may
be carcinogenic.
Administer oral
doses with food.
report the adverse
effects to the
physicians.
VANCOMYCIN
CHEMICAL
NAME
DOSE /ROUTE ACTION INDICATION CONTRA-
INDICATION
SIDE EFFECT NURSING
RESPONSIBILITI
ES
VANCOMYCIN
HYDROCHLORID
E
TRADE NAME -
VANCOMYCIN
DRUG CLASS -
GLYCOPEPTIDE
ANTIBIOTICS
ADULT - 125MG
/ORAL
500MG / IV 6
HRLY
PEDIA : 40MG/KG
/IV
BACTERIOCIDAL
AND
BACTERIOSTATIC
IN ACTION
ACTS BY
INTERFERING
WITH CELL
MEMBRANES
SYNTHESIS IN
MULTIPLYING
ORGANISMS
IN LIFE
THREATNING
INFECTIONS.
IN CLOSTRIDIUM
DIFFICILE
COLITIS
HYPERSENSITIVI
TY TO
VANCOMYCIN
PREVIOUS
HEARING LOSS
USE OF
OTOTOXIC OR
NEPHROTOXIC
AGENTS
CNS: Dizziness,
nausea
Body as a whole:
Serious allergic
reactions
(anaphylactoidreacti
ons)
CV: Low blood
pressure
Respi: Wheezing
GI: Indigestion
Endo: Hives or
itching, Red Man
syndrome (due to
repid infusion of
Vancomycin
1. Administering
vancomycin include
ensuring a patent IV
line,
2. Planning for
administration of the
preoperative dose as
much as two hours
before the initial
incision is made.
3. Including
information about
the dose and timing
of preoperative
vancomycin
administration in the
surgical time out.
4. Report the
adverse effect to
physician.
DISEASE CONDITION
ANATOMY AND PHYSIOLOGY OF
GALL BLADDER
The gallbladder, a pear-shaped, hollow, sac like organ that is 7.5 to 10
cm (3 to 4 in) long, lies in a shallow depression on the inferior surface
of the liver, to which it is attached by loose connective tissue.
The capacity of the gallbladder is 30 to 50 ml of bile.
Its wall is composed largely of smooth muscle. The gallbladder is
connected to the common duct by the cystic duct .
Functions as a storage depot for bile.
Bile is composed of water and electrolytes along with
significant amounts of lecithin, fatty acids, cholesterol,
bilirubin, and bile salts.
The bile salts, together with cholesterol, assist in
emulsification of fats
Approximately half of the bilirubin is a
component of bile. It is converted into
urobilinogen, which is a highly soluble
substance.
 Urobilinogen is either excreted in the feces or returned
to the portal circulation.
If the flow of bile is impeded , bilirubin does not enter
the intestine.
As a result, blood levels of bilirubin increase resulting
increased renal excretion of urobilinogen and decreased
excretion in the stool.
These changes produce many of the signs and
symptoms seen in gallbladder disorders.
DEFINTION:
Cholelithiasis referes to calculi, or gallstones,
usually form in the gallbladder from the solid
constituents of bile; they vary greatly in size, shape,
and composition .
If gall stones migrate into ducts of biliary tract it is
known as choledocholithiasis.
TYPES OF GALL STONES :
Cholestrol stones
those composed pre
dominantly of cholesterol. If
excessive cholestrol is present
and insufficient bile acid is
secreted , bile becomes
supersaturated with cholestrol
and results in cholestrol
stones.
Pigment stones
probably form when
unconjugated
pigments in the bile
precipitate to form
stones.
Mixed stones
combination of
cholestrol and
pigment stones.
ETIOLOGY:
Excessive amounts of
cholesterol
Increased body weight and
older age with increased
cholesterol in the bile.
Bile contains too much
bilirubin
RISK FACTORS
Family
history Obesity
Women, especially
those who have
had multiple
pregnancies
Women of Native
American or U.S.
southwestern
Hispanic ethnicityy
Frequent
changes in
weight
Rapid weight
loss
Treatment with high
estrogen therapy
Cystic fibrosis
Diabetes
PATHOPHYSIOLOGY:
Decreased bile acid synthesis or incomplete and infrequent
emptying of gall bladder may cause the bile to become
overconcentrated.
Increased cholestrol synthesis in the liver
Super saturation of bile with cholestrol
Formation of precipitates
Gall stones( cholelithiasis)
CLINICAL MANIFESTATIONS
Gallstones may be silent, producing no pain and only mild Gl symptoms.
Pain and Biliary Colic
 If a gallstone obstructs the cystic duct, the gallbladder becomes distended,
inflamed, and eventually infected (acute cholecystitis).
 The patient may have biliary colic with excruciating upper right abdominal
pain that radiates to the back or right shoulder.
 Murphy’s sign - indicator of gall bladder inflammation ( acute pancreatitis
) pain on deep breath when finger on under the liver border at the bottom of
the rib cage.
Jaundice
 Found in patient usually with obstruction of the common
bile duct.
 The bile is absorbed by the blood and gives the skin and
mucous membranes a yellow color.
Changes in Urine and Stool Color
 The excretion of the bile pigments by the kidneys gives the urine a very
dark color.
 The feces, no longer colored with bile pigments, are grayish (like putty) or
clay colored.
Vitamin Deficiency
 Obstruction of bile flow interferes with
absorption of the fat soluble vitamins
A, D, E, and K.
 Patients may exhibit deficiencies of
these vitamins if biliary obstruction has
been prolonged. ( bleeding because of
vitamin K )
Patient Picture
In my patient pain in abdomen, and changes
in urine color was observed by patient during
the initial stages.
DIAGNOSTIC TESTS:
Abdominal X-Ray
Ultrasonography
Radionuclide Imaging or Cholescintigraphy
a radioactive agent is administered
intravenously (IV). The biliary tract is then
scanned, and images of the gallbladder and
biliary tract are obtained.
Cholescintigraphy
Cholecystography
 An iodide containing contrast is administered 10 to 12 hours
before the x-ray study.
 The normal gallbladder fills with this radiopaque substance.
If gallstones are present, they appear as shadows on the x-ray
film.
Endoscopic Retrograde
Cholangiopancreatography
ERCP permits direct visualization of
structures
This procedure examines the
hepatobiliary system via a side-
viewing flexible fiberoptic endoscope
inserted through the esophagus to the
descending duodenum .
Percutaneous
transhepatic
cholangiography
It invovles injection of
dye directly into biliary
tract and x-rays are
done,
Patient Picture
In my patient ultrasound was done which
identified cholelithiasis of size 45 × 40 × 35
mm.
CBC , LFT , KFT was also done.
MANAGEMENT:
Medical Management:
Nutritional and Supportive Therapy
 The diet immediately after an episode is usually low-fat liquids.
 These can include powdered supplements high in protein and
carbohydrate
 Cooked fruits, rice or tapioca, non gas-forming vegetables, bread,
coffee, or tea may be added as tolerated.
 The patient should avoid eggs, cream, fried foods, cheese, rich
dressings, and alcohol.
Pharmacologic Therapy
Ursodeoxycholic acid and chenodeoxycholic acid
have been used to dissolve small gallstones .
It acts by inhibiting the synthesis and secretion of
cholesterol, thereby desaturating bile.
Six to 12 months of therapy is required in many
patients to dissolve stones, and monitoring of the
patient for recurrence of symptoms or the occurrence
of side effects is required during this time.
Nonsurgical Removal of Gallstones
Dissolving Gallstones
 To dissolve gallstones by infusion of a solvent ( mono- octanoin or
methy tertiary butyl ether ) into the gallbladder.
The solvent can be infused through the following routes:
 through a tube or catheter inserted percutaneously directly into the
gallbladder,
 through a tube or drain inserted through a T-tube tract to dissolve
stones not removed at the time of surgery,
 endoscopically with ERCP;
 transnasal biliary catheter.
Stone Removal by Instrumentation
A catheter and instrument with a basket attached are threaded through the T-tube tract
, the basket is used to retrieve and remove the stones lodged in the common bile duct.
A second procedure involves the use of the ERCP endoscope . After the endoscope is
inserted, a cutting instrument is passed through the endoscope into the ampulla of
Vater of the common bile duct.
It may be used to cut the submucosal fibers, enlarg ing the opening, which may allow
the lodged stones to pass spontaneously into the duodenum.
Another instrument with a small basket or balloon at its tip may be inserted through the
endoscope to retrieve the stones .
Intracorporeal Lithotripsy
A laser pulse is directed under fluoroscopic guidance
with the use of devices that can distinguish between
stones and tissue.
The laser pulse produces rapid expansion and
disintegration of plasma on the stone surface, resulting
in a mechanical shock wave.
Repeated procedures may be necessary because of
stone size, local anatomy, bleeding, or technical
difficulty.
Extracorporeal Shock Wave Lithotripsy
Lithotripsy which is a noninvasive procedure, uses repeated
shock waves directed at the gallstones in the gallbladder or
common tripsy, bile duct to fragment the stones.
The waves are transmitted to the body through a fluid-filled
bag or by immersing the patient in a water bath.
After the stones are gradually broken up, the stone
fragments can be spontaneously passed from the gallbladder
or common bile duct, removed by endoscopy, or dissolved
with oral bile acid or solvents.
Surgical Management:
Laparoscopic Cholecystectomy
 Laparoscopic cholecystectomy is performed through a small
incision or puncture made through the abdominal wall at the
umbilicus.
 The fiberoptic scope is inserted through the small umbilical incision.
 Several additional punctures or small incisions are made in the
abdominal wall to introduce other surgical instruments into the
operative field.
 A camera attached to the laparoscope permits the surgeon to view
the intra-abdominal field and biliary system on a television monitor.
 The cystic artery is dissected free and clipped. The gallbladder is
separated from the hepatic bed and removed from the abdominal
cavity after bile and small stones are aspirated.
 Stone forceps also can be used to remove or crush larger stones.
Cholecystectomy
Gallbladder is removed
through an abdominal
incision after the cystic
duct and artery are
ligated.
A drain is placed close to
the gallbladder bed and
brought out through a
puncture wound if there
is a bile leak.
Small-Incision Cholecystectomy
A surgical procedure in which the gallbladder is
removed through a small abdominal incision..
If needed, the surgical incision is extended to
remove larger gallbladder stones.
Drains may or may not be used.
The short length hospital stay has been identified
as a major advantage of this type of procedure .
• It involves making an incision in the
common duct, usually for removal of
stones.
• After the stones have been evacuated, a
tube is usually inserted into the duct for
drainage of bile until edema subsides.
• This tube is connected to gravity
drainage tubing: the patient is monitored
closely, and a laparoscopic
cholecystectomy is planned for a future
date after acute inflammation has
resolved.
Choledochostomy
Percutaneous Cholecystostomy
 Under local anesthesia, a fine needle is inserted through the abdominal wall
and live edge into the gallbladder under the guidance of ultrasound or
computed tomography (CT).
 Bile is aspirated to ensure adequate placement of the needle, and a catheter
is inserted into the gallbladder to decompress the biliary tract.
Patient Picture
 IN MY PATIENT LAPROSCOPIC CHOLECYSTECTOMY WAS
DONE ON 28/12/2021
Complications:
 Chronic cholecystitis
 Acute cholecystitis
 Choledococholithiasis
 Gallstone pancreatitis
 Gallstone ileus
 Perforation of gall bladder
 Gallbladder carcinoma
Nursing Management:
ASSESSMENT NURSING
DIAGNOSIS
GOAL INTERVENTION IMPLEMENTATION
EVALUATION
subjective data -
mujhe pet me
dard hota hai
objective data -
facial
expression
acute pain and
discomfort related to
surgical incision.
to reduce pain assess the pain
score.
promote bed rest
provide pillows
around incision to
relieve pain.
encourage relaxation
techniques such as
deep breathing.
encourage walking
and using heat pad
to ease discomfort.
provide analgesics.
pain score assed as 3/10
bed rest is promoted
extra pillow provided
for comfort
deep breathing
promoted.
patient encouraged to
walk.
patient has slight
reduction in pain
as 2/10
according to
pain score.
ASSESSMENT NURSING
DIAGNOSIS
GOAL INTERVENTION IMPLEMENTATION EVALUATION
subjective data - pt
told , “mujhe khana
khane ka man nahi
krta.”
objective data -
weakness of patient
to perform activity.
loss of muscle tone
imbalanced nutrition
:less than body
requirements related
to inadequate bile
secretion.
to provide
optimal
nutritional intake.
asess the patient’s
nutritional status.
encourage patient to
have diet rich in
carbohydrates and
protein and low in
fats.
encorage patient to
follow this diet even
after getting discharge
for 4-6 weeks.
encorage to gradually
add the fat to the diet.
assess the elimination
pattern.
patient nutritional
status assessed as
inadequate.
patient was
encouraged to eat
protein rich food such
as daal and low fat
food such as oily food.
patient enocurage to
follow the diet.
elimination pattern
recorded as patient is
having constipation.
patient’s nutritional
satus is same.
patient promises to
follow the diet.
ASSESSMENT NURSING
DIAGNOSIS
GOAL INTERVENTION IMPLEMENTATION EVALUATION
subjective data -
patient told, “ mujhe
kuch samajh nahi aa
raha hai.
objective data -
patient looks
anxious.
deficient knowledge
about self care
activities related to
incision care and
dietary medication.
to provide
knowledge
regarding self care
routines.
asess the knowledge
level of patient.
educate patient about
the medication and
their action.
educate about the
symptoms to report
such as jaundice, dark
urine , pruritis.
edcuate about care of
wound.
clear all the doubts
encourage
questioning.
knowledge level
assessed as low.
educated patient about
the medication.
patient educated about
symptoms to look for.
patient is educated
about wound care.
all the doubts were
cleared.
patient ‘s knowledge
has been enhanced.
ASSESSMENT NURSING
DIAGNOSIS
GOAL INTERVENTION IMPLEMENTATION EVALUATION
subjective data -
mujhe surgery
ki jagh par
khujli krne ka
man krta hai
objective data -
surgical site is
slight red.
risk for infection
related to surgical
site
to reduce
chances of
infection.
Assess the condition
of surgical site.
monitor any signs of
infection
maintain strict
asepsis technique
while wound
dressing.
perform hand
hygiene .
educate client about
precautionary
measures.
surgical site assessed as
free from infection.
no signs of infection
witnessed.
aseptic technique
followed for dressing.
hand hygiene
performed.
health education
provided.
Risk for
infection
reduced.
ASSESSMEN
T
NURSING
DIAGNOSIS
GOAL INTERVENTION IMPLEMENTATION
EVALUATION
subjective data
- mujhe chalte
samay dard
hota hai
objective data -
pain while
walking.
Impaired physical
mobility related to
pain at surgical site.
to improve
physical
mobility
assess the level of
physical mobility.

assess the barriers
for mobility.
assess the strength
to perform ROM
.evaluate need for
assitive devices.
educate to walk
around a little .
level of physical
mobility is moderate.
pain is the barrier for
mobility.
patient has weakness to
perform ROM.
no need for assistive
devices.
patient was encouraged
for little walking like
going to washroom
with the help of family
member.
patient ‘s
physical
mobility is still
same
NURSE’S NOTES
Progress notes :
 Patient is well, general condition is fair . Vitals checked
and charted.
 T- 98.7 F
 P- 85 b/m
 R- 18 b/m
 B.P- 126/84 mmHg
 All due medications given
 Health education provided.
HEALTH EDUCATION :
• Sitting upright in bed or chair or walking may ease discomfort ,
Analgesic medication as needed and prescribed
• Report to surgeon if pain not relieved even with analgesic
Managing pain
• Light exercise ( walking ) immediately, Shower or bath after 1-2 days
,Avoid lifting heavy objects.
Resuming the
activity
• Check puncture sites daily for infection., Wash with soap and water
Caring for the
wound
• Resume normal diet
Resuming eating
• Report sign and symptoms of infection at puncture site., Report fever,
nausea , vomiting or abdominal pain.
Follow up care:
SUMMARY :
Today we dealt with case presntation on
cholelithiasis, my patient’s condition, about disease
condition, cholelithiasis, risk factors, etiology,
clinical manifestations, pathophysiology, lab
diagnostics, medcial management, surgical
management, nursing process, health education on
follow up care.
CONCLUSION
 Calculi, or gallstones, usually form in the gallbladder from
the solid constituents of bile; they vary greatly in size,
shape, and composition .
 They are uncommon in children and young adults but
become more prevalent with increasing age.
 If gall stones migrate into ducts of biliary tract it is known as
choledocholithiasis.
Bibliography:
 Hinkle Janice L. and Cheever H. Kerry , Brunner
and suddhart’s textbook of medical surgical nursing
, volume 2 , 13th edition,2014, wolters
lkluwer(india) private ltd. pg no - 1391-1401.
 Patient details from patient file and through data
collected on interview basis.
Presentation on cholelithiasis

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Presentation on cholelithiasis

  • 1. CASE PRESENTATION ON CHOLELITHIASIS BY ARUSHI NEGI M.SC. NURSING 1ST YEAR
  • 2. Identification Data: • Client name: Mrs. Omi devi • Age/sex: - 67 years / Female • Father/spouse name: Mr. Ram meher • Hospital registration number: 22030 • Ward: Surgery ward • Bed No.- 45 • Address: Village sher, Panipat , Haryana • Education: 3rd • Occupation: House wife
  • 3. Marital status: Married Religion: Hindu Date of admission:26/12/2021 Date of discharge: - Diagnosis: Cholelithiasis Surgery (if any): Laparascopic cholecystectomy Date of Surgery: 28-12-2021 Date of interview :03-01-2022 Doctor In-charge: Dr. J.P singh Informant : Patient herself.
  • 4. History of Illness: Chief complaints: patient was having pain in abdomen since last 12 days. History of fever 4-5 days At present :Patient is well , and have pain at surgical site.  Present medical history: My patient was having epigastric pain since last 12 days so she visited a nearby clinic at her residence who recommended an ultrasound in which she was diagnosed with cholelithiasis. She was recommended to go for higher facility for which she chose Lal Bahadur Shastri Hospital
  • 5. Past medical history: Patient is diagnosed with diabetes since last 15 years and was taking treatment from her nearby clinic.She has now stopped taking medications . Past surgical history: Patient has undrgone hernioplasty 5 years back for hiatal hernia nearby in a facility near her house.
  • 6. FAMILY HISTORY S. No . Name of family members Age sex Occupation Relation of family Member to patient Education Health status 1. Omi devi 67y F House wife Self 3rd Unhealthy 2. Ramprakash 70 y M Farmer Husband 5th Healthy 3. Indrajeet 45 y M Pvt job Son BA Healthy 4. Sushila 40 y F House wife Daughter in law 10th Healthy 5. Rahul 20 y M Student Grandson BBA Healthy 6. Rakesh 18 y M Student Grandson 12th Healthy 7. Monika 44 y F House wife Daughter 12th Healthy 8. Rajeev 46 y M Pvt job Son in law Polytech Healthy 9. Shikha 16 y F Student Granddaught er 10th Healthy
  • 8.  Personal History: No smoking and alchol consumption history.  Menstrual history: Patient attained menarche at the age of 14 yrs , and menopause at 50 yrs, She didn’t undergo any checkups during her pregnancy. Had normal vaginal delivery.  Dietary habits: She consumes non vegetarian diet.  Elimination pattern : She has constipation
  • 9. PHYSICAL EXAMINATION GENERALAPPERANCE:  Body build - Endomorph  Height- 151 cm  Weight- 95 kg Vital signs  Temperature - 98.7 F  Pulse: 85 b/m  Respiration: 18 b/m  B.P: 124/96 mmHg
  • 10. Colour of skin: normal Head: Shape and size of skull: well rounded Scalp: free from any lices Face: sagging of skin due to old age Eyes: no abnormality detected.  Eye brow and eyelid: hair evenly distributed , symmetrical Conjunctiva: no abnormality detected. Sclera: slight yellow in color
  • 11. Ear: External ear: symmetrical Hearing problem: no abnormality detected. Nose: External nares: straight and uniform Mouth and pharynx: Mouth: lips are pink in color Teeth: no discoloration , lacks 3 teeth Tongue: normal
  • 12. Neck: Thyroid gland: no abnormality detected Lymph node: normal Range of motion: No abnormalities. Chest: Breath sounds: no abnormality detected Lungs: clear
  • 13. Abdomen: Inspection: scars of laproscopic cholecystectomy . Auscultation: no abnormality detected. Palpation: pain on touch Extremities: Upper: no abnormalities Lower: no abnormalities Back: no bed sores.
  • 14. LAB INVESTIGATION: LFT : Bilirubin - 3 mg/dl (0.1-1.2 mg/dl) Direct -0.5 mg/dl (0.0-0.2 mg/dl ) SGPT - 42 IU/L( 5-40 ) SGOT - 41 IU/L(5-40)
  • 15. MEDICATIONS: In my patient:  VANCOMYCIN  METRONIDAZOLE  CIPLOXACIN  PANTOPRAZOLE  PARACETAMOL  MONOCEFTRIAXONE  NORMAL SALINE
  • 16. METRONIDAZOLE CHEMICAL NAME DOSE /ROUTE ACTION INDICATION CONTRA- INDICATION SIDE EFFECT NURSING RESPONSIBILITI ES METRO NIDA ZOLE TRADE NAME - METROGYL DRUG CLASS - ANTIBIOTICS Adults—500 or 750 milligrams (mg) 3 times a day for 5 to 10 days. Children—Dose is based on body weight and must be determined by your doctor. The dose is usually 35 to 50 milligrams (mg) per kilogram (kg) of body weight per day, divided into 3 doses, for 10 days. Metronidazole interacts with the microbial DNA to break its strand and helical structure leading to inhibition of protein synthesis, degradation, and cell death. • Anaerobic bacterial infections. • Amoebiasis Bacterial vaginosis Trichomoniasis • H. pylori eradication associated with peptic ulcer disease. Acute dental infections. • Prophylaxis of post-op anaerobic bacterial infections • Hypersensitivity to metronidazole and other nitroimidazoles. • Concomitant use with disulfiram within the last 14 days. • Coadministratio n with alcohol or propylene glycol containing products during or 3 days after therapy discontinuation. • Pregnancy during the 1st trimester in the treatment of trichomoniasis. Severe neurological disturbances, encephalopathy, convulsive seizures, aseptic meningitis, peripheral and optic neuropathy, paraesthesia; superinfection (e.g. fungal or bacterial superinfection, C. difficile- associated diarrhoea. Blood and lymphatic system disorders: Leucopenia, neutropenia. Cardiac disorders: Chest pain, tachycardia. Ear and labyrinth disorders: Tinnitu Avoid use unless necessary. Metronidazole may be carcinogenic. Administer oral doses with food. report the adverse effects to the physicians.
  • 17. VANCOMYCIN CHEMICAL NAME DOSE /ROUTE ACTION INDICATION CONTRA- INDICATION SIDE EFFECT NURSING RESPONSIBILITI ES VANCOMYCIN HYDROCHLORID E TRADE NAME - VANCOMYCIN DRUG CLASS - GLYCOPEPTIDE ANTIBIOTICS ADULT - 125MG /ORAL 500MG / IV 6 HRLY PEDIA : 40MG/KG /IV BACTERIOCIDAL AND BACTERIOSTATIC IN ACTION ACTS BY INTERFERING WITH CELL MEMBRANES SYNTHESIS IN MULTIPLYING ORGANISMS IN LIFE THREATNING INFECTIONS. IN CLOSTRIDIUM DIFFICILE COLITIS HYPERSENSITIVI TY TO VANCOMYCIN PREVIOUS HEARING LOSS USE OF OTOTOXIC OR NEPHROTOXIC AGENTS CNS: Dizziness, nausea Body as a whole: Serious allergic reactions (anaphylactoidreacti ons) CV: Low blood pressure Respi: Wheezing GI: Indigestion Endo: Hives or itching, Red Man syndrome (due to repid infusion of Vancomycin 1. Administering vancomycin include ensuring a patent IV line, 2. Planning for administration of the preoperative dose as much as two hours before the initial incision is made. 3. Including information about the dose and timing of preoperative vancomycin administration in the surgical time out. 4. Report the adverse effect to physician.
  • 19. ANATOMY AND PHYSIOLOGY OF GALL BLADDER
  • 20. The gallbladder, a pear-shaped, hollow, sac like organ that is 7.5 to 10 cm (3 to 4 in) long, lies in a shallow depression on the inferior surface of the liver, to which it is attached by loose connective tissue. The capacity of the gallbladder is 30 to 50 ml of bile. Its wall is composed largely of smooth muscle. The gallbladder is connected to the common duct by the cystic duct . Functions as a storage depot for bile.
  • 21. Bile is composed of water and electrolytes along with significant amounts of lecithin, fatty acids, cholesterol, bilirubin, and bile salts. The bile salts, together with cholesterol, assist in emulsification of fats Approximately half of the bilirubin is a component of bile. It is converted into urobilinogen, which is a highly soluble substance.
  • 22.  Urobilinogen is either excreted in the feces or returned to the portal circulation. If the flow of bile is impeded , bilirubin does not enter the intestine. As a result, blood levels of bilirubin increase resulting increased renal excretion of urobilinogen and decreased excretion in the stool. These changes produce many of the signs and symptoms seen in gallbladder disorders.
  • 23. DEFINTION: Cholelithiasis referes to calculi, or gallstones, usually form in the gallbladder from the solid constituents of bile; they vary greatly in size, shape, and composition . If gall stones migrate into ducts of biliary tract it is known as choledocholithiasis.
  • 24. TYPES OF GALL STONES : Cholestrol stones those composed pre dominantly of cholesterol. If excessive cholestrol is present and insufficient bile acid is secreted , bile becomes supersaturated with cholestrol and results in cholestrol stones. Pigment stones probably form when unconjugated pigments in the bile precipitate to form stones. Mixed stones combination of cholestrol and pigment stones.
  • 25. ETIOLOGY: Excessive amounts of cholesterol Increased body weight and older age with increased cholesterol in the bile. Bile contains too much bilirubin
  • 26. RISK FACTORS Family history Obesity Women, especially those who have had multiple pregnancies Women of Native American or U.S. southwestern Hispanic ethnicityy Frequent changes in weight Rapid weight loss Treatment with high estrogen therapy Cystic fibrosis Diabetes
  • 27. PATHOPHYSIOLOGY: Decreased bile acid synthesis or incomplete and infrequent emptying of gall bladder may cause the bile to become overconcentrated. Increased cholestrol synthesis in the liver Super saturation of bile with cholestrol Formation of precipitates Gall stones( cholelithiasis)
  • 28. CLINICAL MANIFESTATIONS Gallstones may be silent, producing no pain and only mild Gl symptoms. Pain and Biliary Colic  If a gallstone obstructs the cystic duct, the gallbladder becomes distended, inflamed, and eventually infected (acute cholecystitis).  The patient may have biliary colic with excruciating upper right abdominal pain that radiates to the back or right shoulder.  Murphy’s sign - indicator of gall bladder inflammation ( acute pancreatitis ) pain on deep breath when finger on under the liver border at the bottom of the rib cage.
  • 29.
  • 30. Jaundice  Found in patient usually with obstruction of the common bile duct.  The bile is absorbed by the blood and gives the skin and mucous membranes a yellow color. Changes in Urine and Stool Color  The excretion of the bile pigments by the kidneys gives the urine a very dark color.  The feces, no longer colored with bile pigments, are grayish (like putty) or clay colored.
  • 31. Vitamin Deficiency  Obstruction of bile flow interferes with absorption of the fat soluble vitamins A, D, E, and K.  Patients may exhibit deficiencies of these vitamins if biliary obstruction has been prolonged. ( bleeding because of vitamin K )
  • 32. Patient Picture In my patient pain in abdomen, and changes in urine color was observed by patient during the initial stages.
  • 33. DIAGNOSTIC TESTS: Abdominal X-Ray Ultrasonography Radionuclide Imaging or Cholescintigraphy a radioactive agent is administered intravenously (IV). The biliary tract is then scanned, and images of the gallbladder and biliary tract are obtained.
  • 35. Cholecystography  An iodide containing contrast is administered 10 to 12 hours before the x-ray study.  The normal gallbladder fills with this radiopaque substance. If gallstones are present, they appear as shadows on the x-ray film.
  • 36. Endoscopic Retrograde Cholangiopancreatography ERCP permits direct visualization of structures This procedure examines the hepatobiliary system via a side- viewing flexible fiberoptic endoscope inserted through the esophagus to the descending duodenum .
  • 37. Percutaneous transhepatic cholangiography It invovles injection of dye directly into biliary tract and x-rays are done,
  • 38. Patient Picture In my patient ultrasound was done which identified cholelithiasis of size 45 × 40 × 35 mm. CBC , LFT , KFT was also done.
  • 39. MANAGEMENT: Medical Management: Nutritional and Supportive Therapy  The diet immediately after an episode is usually low-fat liquids.  These can include powdered supplements high in protein and carbohydrate  Cooked fruits, rice or tapioca, non gas-forming vegetables, bread, coffee, or tea may be added as tolerated.  The patient should avoid eggs, cream, fried foods, cheese, rich dressings, and alcohol.
  • 40. Pharmacologic Therapy Ursodeoxycholic acid and chenodeoxycholic acid have been used to dissolve small gallstones . It acts by inhibiting the synthesis and secretion of cholesterol, thereby desaturating bile. Six to 12 months of therapy is required in many patients to dissolve stones, and monitoring of the patient for recurrence of symptoms or the occurrence of side effects is required during this time.
  • 41. Nonsurgical Removal of Gallstones Dissolving Gallstones  To dissolve gallstones by infusion of a solvent ( mono- octanoin or methy tertiary butyl ether ) into the gallbladder. The solvent can be infused through the following routes:  through a tube or catheter inserted percutaneously directly into the gallbladder,  through a tube or drain inserted through a T-tube tract to dissolve stones not removed at the time of surgery,  endoscopically with ERCP;  transnasal biliary catheter.
  • 42. Stone Removal by Instrumentation A catheter and instrument with a basket attached are threaded through the T-tube tract , the basket is used to retrieve and remove the stones lodged in the common bile duct. A second procedure involves the use of the ERCP endoscope . After the endoscope is inserted, a cutting instrument is passed through the endoscope into the ampulla of Vater of the common bile duct. It may be used to cut the submucosal fibers, enlarg ing the opening, which may allow the lodged stones to pass spontaneously into the duodenum. Another instrument with a small basket or balloon at its tip may be inserted through the endoscope to retrieve the stones .
  • 43. Intracorporeal Lithotripsy A laser pulse is directed under fluoroscopic guidance with the use of devices that can distinguish between stones and tissue. The laser pulse produces rapid expansion and disintegration of plasma on the stone surface, resulting in a mechanical shock wave. Repeated procedures may be necessary because of stone size, local anatomy, bleeding, or technical difficulty.
  • 44. Extracorporeal Shock Wave Lithotripsy Lithotripsy which is a noninvasive procedure, uses repeated shock waves directed at the gallstones in the gallbladder or common tripsy, bile duct to fragment the stones. The waves are transmitted to the body through a fluid-filled bag or by immersing the patient in a water bath. After the stones are gradually broken up, the stone fragments can be spontaneously passed from the gallbladder or common bile duct, removed by endoscopy, or dissolved with oral bile acid or solvents.
  • 45. Surgical Management: Laparoscopic Cholecystectomy  Laparoscopic cholecystectomy is performed through a small incision or puncture made through the abdominal wall at the umbilicus.  The fiberoptic scope is inserted through the small umbilical incision.  Several additional punctures or small incisions are made in the abdominal wall to introduce other surgical instruments into the operative field.  A camera attached to the laparoscope permits the surgeon to view the intra-abdominal field and biliary system on a television monitor.
  • 46.  The cystic artery is dissected free and clipped. The gallbladder is separated from the hepatic bed and removed from the abdominal cavity after bile and small stones are aspirated.  Stone forceps also can be used to remove or crush larger stones.
  • 47. Cholecystectomy Gallbladder is removed through an abdominal incision after the cystic duct and artery are ligated. A drain is placed close to the gallbladder bed and brought out through a puncture wound if there is a bile leak.
  • 48. Small-Incision Cholecystectomy A surgical procedure in which the gallbladder is removed through a small abdominal incision.. If needed, the surgical incision is extended to remove larger gallbladder stones. Drains may or may not be used. The short length hospital stay has been identified as a major advantage of this type of procedure .
  • 49. • It involves making an incision in the common duct, usually for removal of stones. • After the stones have been evacuated, a tube is usually inserted into the duct for drainage of bile until edema subsides. • This tube is connected to gravity drainage tubing: the patient is monitored closely, and a laparoscopic cholecystectomy is planned for a future date after acute inflammation has resolved. Choledochostomy
  • 50. Percutaneous Cholecystostomy  Under local anesthesia, a fine needle is inserted through the abdominal wall and live edge into the gallbladder under the guidance of ultrasound or computed tomography (CT).  Bile is aspirated to ensure adequate placement of the needle, and a catheter is inserted into the gallbladder to decompress the biliary tract.
  • 51. Patient Picture  IN MY PATIENT LAPROSCOPIC CHOLECYSTECTOMY WAS DONE ON 28/12/2021
  • 52. Complications:  Chronic cholecystitis  Acute cholecystitis  Choledococholithiasis  Gallstone pancreatitis  Gallstone ileus  Perforation of gall bladder  Gallbladder carcinoma
  • 53. Nursing Management: ASSESSMENT NURSING DIAGNOSIS GOAL INTERVENTION IMPLEMENTATION EVALUATION subjective data - mujhe pet me dard hota hai objective data - facial expression acute pain and discomfort related to surgical incision. to reduce pain assess the pain score. promote bed rest provide pillows around incision to relieve pain. encourage relaxation techniques such as deep breathing. encourage walking and using heat pad to ease discomfort. provide analgesics. pain score assed as 3/10 bed rest is promoted extra pillow provided for comfort deep breathing promoted. patient encouraged to walk. patient has slight reduction in pain as 2/10 according to pain score.
  • 54. ASSESSMENT NURSING DIAGNOSIS GOAL INTERVENTION IMPLEMENTATION EVALUATION subjective data - pt told , “mujhe khana khane ka man nahi krta.” objective data - weakness of patient to perform activity. loss of muscle tone imbalanced nutrition :less than body requirements related to inadequate bile secretion. to provide optimal nutritional intake. asess the patient’s nutritional status. encourage patient to have diet rich in carbohydrates and protein and low in fats. encorage patient to follow this diet even after getting discharge for 4-6 weeks. encorage to gradually add the fat to the diet. assess the elimination pattern. patient nutritional status assessed as inadequate. patient was encouraged to eat protein rich food such as daal and low fat food such as oily food. patient enocurage to follow the diet. elimination pattern recorded as patient is having constipation. patient’s nutritional satus is same. patient promises to follow the diet.
  • 55. ASSESSMENT NURSING DIAGNOSIS GOAL INTERVENTION IMPLEMENTATION EVALUATION subjective data - patient told, “ mujhe kuch samajh nahi aa raha hai. objective data - patient looks anxious. deficient knowledge about self care activities related to incision care and dietary medication. to provide knowledge regarding self care routines. asess the knowledge level of patient. educate patient about the medication and their action. educate about the symptoms to report such as jaundice, dark urine , pruritis. edcuate about care of wound. clear all the doubts encourage questioning. knowledge level assessed as low. educated patient about the medication. patient educated about symptoms to look for. patient is educated about wound care. all the doubts were cleared. patient ‘s knowledge has been enhanced.
  • 56. ASSESSMENT NURSING DIAGNOSIS GOAL INTERVENTION IMPLEMENTATION EVALUATION subjective data - mujhe surgery ki jagh par khujli krne ka man krta hai objective data - surgical site is slight red. risk for infection related to surgical site to reduce chances of infection. Assess the condition of surgical site. monitor any signs of infection maintain strict asepsis technique while wound dressing. perform hand hygiene . educate client about precautionary measures. surgical site assessed as free from infection. no signs of infection witnessed. aseptic technique followed for dressing. hand hygiene performed. health education provided. Risk for infection reduced.
  • 57. ASSESSMEN T NURSING DIAGNOSIS GOAL INTERVENTION IMPLEMENTATION EVALUATION subjective data - mujhe chalte samay dard hota hai objective data - pain while walking. Impaired physical mobility related to pain at surgical site. to improve physical mobility assess the level of physical mobility. assess the barriers for mobility. assess the strength to perform ROM .evaluate need for assitive devices. educate to walk around a little . level of physical mobility is moderate. pain is the barrier for mobility. patient has weakness to perform ROM. no need for assistive devices. patient was encouraged for little walking like going to washroom with the help of family member. patient ‘s physical mobility is still same
  • 59. Progress notes :  Patient is well, general condition is fair . Vitals checked and charted.  T- 98.7 F  P- 85 b/m  R- 18 b/m  B.P- 126/84 mmHg  All due medications given  Health education provided.
  • 60. HEALTH EDUCATION : • Sitting upright in bed or chair or walking may ease discomfort , Analgesic medication as needed and prescribed • Report to surgeon if pain not relieved even with analgesic Managing pain • Light exercise ( walking ) immediately, Shower or bath after 1-2 days ,Avoid lifting heavy objects. Resuming the activity • Check puncture sites daily for infection., Wash with soap and water Caring for the wound • Resume normal diet Resuming eating • Report sign and symptoms of infection at puncture site., Report fever, nausea , vomiting or abdominal pain. Follow up care:
  • 61. SUMMARY : Today we dealt with case presntation on cholelithiasis, my patient’s condition, about disease condition, cholelithiasis, risk factors, etiology, clinical manifestations, pathophysiology, lab diagnostics, medcial management, surgical management, nursing process, health education on follow up care.
  • 62. CONCLUSION  Calculi, or gallstones, usually form in the gallbladder from the solid constituents of bile; they vary greatly in size, shape, and composition .  They are uncommon in children and young adults but become more prevalent with increasing age.  If gall stones migrate into ducts of biliary tract it is known as choledocholithiasis.
  • 63. Bibliography:  Hinkle Janice L. and Cheever H. Kerry , Brunner and suddhart’s textbook of medical surgical nursing , volume 2 , 13th edition,2014, wolters lkluwer(india) private ltd. pg no - 1391-1401.  Patient details from patient file and through data collected on interview basis.